Relevant bibliographies by topics / Cancer epigenetics

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cancer epigenetics'

Author: Grafiati
Published: 8 March 2025
Last updated: 26 July 2025

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Journal articles on the topic "Cancer epigenetics"

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Youness, Eman. "Overview on Epigenetics and Cancer." Clinical Medical Reviews and Reports 2, no. 3 (2020): 01–06. http://dx.doi.org/10.31579/2690-8794/015.

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Epigenetics is considered as the science of hereditary phenotype which does not encompass amendment in the DNA. This occurs through chemical processes that modify the phenotype, without altering the genotype. A large number of studies showed that metabolic diseases are highly associated with epigenetic alterations suggesting that epigenetic factors may play a central role in cancer. Recent advancements in the rapidly evolving field of cancer epigenetics have shown extensive reprogramming of every component of the epigenetic machinery in cancer including DNA methylation, histone modifications,
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Li, Jiaqiu, Hongchuan Jin, and Xian Wang. "Epigenetic Biomarkers: Potential Applications in Gastrointestinal Cancers." ISRN Gastroenterology 2014 (March 6, 2014): 1–10. http://dx.doi.org/10.1155/2014/464015.

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Genetics and epigenetics coregulate the cancer initiation and progression. Epigenetic mechanisms include DNA methylation, histone modification, chromatin remodeling, and noncoding RNAs. Aberrant epigenetic modifications play a fundamental role in the formation of gastrointestinal cancers. Advances in epigenetics offer a better understanding of the carcinogenesis and provide new insights into the discovery of biomarkers for diagnosis, and prognosis prediction of human cancers. This review aims to overview the epigenetic aberrance and the clinical applications as biomarkers in gastrointestinal c
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Pathak, Ayush, Sarthak Tomar, and Sujata Pathak. "Epigenetics and Cancer: A Comprehensive Review." Asian Pacific Journal of Cancer Biology 8, no. 1 (2023): 75–89. http://dx.doi.org/10.31557/apjcb.2023.8.1.75-89.

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Cancer is a disease with extraordinary clinical significance, with much of medical research being devoted to it. Innumerable factors are relevant in fully understanding cancer but the epigenetic aspect stands out. Epigenetics is the study of changes, often germ-line, to the genome affecting the gene expression by silencing certain genes and modifying the gene expression. The three primary mechanisms for epigenetic changes are DNA methylation, histone modification and non-coding RNA (ncRNA) associated gene silencing. While epigenetics is a pivotal mechanism for the regular maintenance of a myri
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Chou, PoChung Jordan, Rebecca Mary Peter, Ahmad Shannar, et al. "Epigenetics of Dietary Phytochemicals in Cancer Prevention." Cancer Journal 30, no. 5 (2024): 320–28. http://dx.doi.org/10.1097/ppo.0000000000000742.

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Abstract Cancer development takes 10 to 50 years, and epigenetics plays an important role. Recent evidence suggests that ~80% of human cancers are linked to environmental factors impinging upon genetics/epigenetics. Because advanced metastasized cancers are resistant to radiation/chemotherapeutic drugs, cancer prevention by relatively nontoxic “epigenetic modifiers” will be logical. Many dietary phytochemicals possess powerful antioxidant and anti-inflammatory properties that are hallmarks of cancer prevention. Dietary phytochemicals can regulate gene expression of the cellular genome via epig
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Rahman, Md, HM Jamil, Naznin Akhtar, et al. "Cancer epigenetics and epigenetical therapy." Journal of Experimental and Integrative Medicine 6, no. 3 (2016): 143. http://dx.doi.org/10.5455/jeim.270616.rw.016.

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Yadav, Rinki, Ashish Srivastava, Suresh Chandra, and A.K. Rai. "ROLE OF EPIGENETIC MECHANISMS IN VARIOUS CANCER THERAPIES." Pharmaceutical and Biological Evaluations 3, no. 2 (2016): 178–84. https://doi.org/10.5281/zenodo.51060.

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Epigenetics play a role not just in the normal functioning of the cell and its development, but also in diseases like neurological diseases and cancer. Epigenetic therapies can help to resolve non-identical problems of these pathophysiological conditions. Cancer is a complex disease with both genetic and epigenetic origins. The importance of epigenetics in cancer has been identified, and the field has emerged rapidly in recent years. Epigenetic and genetic alterations contribute to the initiation and progression of cancer. Epigenetic modifications introduced genetic changes, and usually occur
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Hatzimichael, Eleftheria, and Tim Crook. "Cancer Epigenetics: New Therapies and New Challenges." Journal of Drug Delivery 2013 (February 26, 2013): 1–9. http://dx.doi.org/10.1155/2013/529312.

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Cancer is nowadays considered to be both a genetic and an epigenetic disease. The most well studied epigenetic modification in humans is DNA methylation; however it becomes increasingly acknowledged that DNA methylation does not work alone, but rather is linked to other modifications, such as histone modifications. Epigenetic abnormalities are reversible and as a result novel therapies that work by reversing epigenetic effects are being increasingly explored. The biggest clinical impact of epigenetic modifying agents in neoplastic disorders thus far has been in haematological malignancies, and
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Maio, Michele. "Introduction: Cancer Epigenetics and Epigenetic Treatment of Cancer." Seminars in Oncology 32, no. 5 (2005): 435–36. http://dx.doi.org/10.1053/j.seminoncol.2005.08.001.

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Kanwal, Rajnee, and Sanjay Gupta. "Epigenetics and cancer." Journal of Applied Physiology 109, no. 2 (2010): 598–605. http://dx.doi.org/10.1152/japplphysiol.00066.2010.

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Epigenetic modifications are central to many human diseases, including cancer. Traditionally, cancer has been viewed as a genetic disease, and it is now becoming apparent that the onset of cancer is preceded by epigenetic abnormalities. Investigators in the rapidly expanding field of epigenetics have documented extensive genomic reprogramming in cancer cells, including methylation of DNA, chemical modification of the histone proteins, and RNA-dependent regulation. Recognizing that carcinogenesis involves both genetic and epigenetic alterations has led to a better understanding of the molecular
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van Engeland, Manon, Sarah Derks, Kim M. Smits, Gerrit A. Meijer, and James G. Herman. "Colorectal Cancer Epigenetics: Complex Simplicity." Journal of Clinical Oncology 29, no. 10 (2011): 1382–91. http://dx.doi.org/10.1200/jco.2010.28.2319.

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Colorectal cancer (CRC) has predominantly been considered a genetic disease, characterized by sequential accumulation of genetic alterations. Growing evidence indicates that epigenetic alterations add an additional layer of complexity to the pathogenesis of CRC, and characterize a subgroup of colorectal cancers with a distinct etiology and prognosis. Epigenetic dysregulation in colorectal cancer is organized at multiple levels, involving DNA methylation, histone modifications, nucleosomal occupancy and remodeling, chromatin looping, and noncoding RNAs. Interactions between these processes and
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Dissertations / Theses on the topic "Cancer epigenetics"

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Donovan, Micah Gerard, and Micah Gerard Donovan. "Breast Cancer Epigenetics: Modification by Genistein." Thesis, The University of Arizona, 2017. http://hdl.handle.net/10150/624144.

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Breast cancer it is the most common type of cancer and leading cause of cancer mortality among women worldwide. Women who inherit mutations in the breast cancer 1 susceptibility gene (BRCA1) are five times more likely to develop breast cancer than women who do not. However, only ~5-10% of breast cancer cases are due to germline mutations in tumor suppressor genes. There are currently no targeted therapies available triple negative breast cancers (TNBC), which often lack BRCA1 expression. BRCA1 is epigenetically silenced by the activated aryl-hydrocarbon receptor (AhR), suggesting that dietary
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Giger, O. T. "Epigenetics in regulation of oesophageal cancer stromal myofibroblasts." Thesis, University of Liverpool, 2017. http://livrepository.liverpool.ac.uk/3007985/.

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Cancer is the 2nd most common cause of death in our society and is associated with high morbidity and costs. The word ‘cancer’ amalgamates the complex interplay between cells which have acquired genetic alterations leading to uncontrolled proliferation, i.e. the malignant cells, and genetically ‘normal’ host cells, i.e. stromal cells, vascular cells and inflammatory cells which all acquire modified biological phenotypes in the presence of malignant cells. This community of cells and their secreted proteins defines the tumour microenvironment. Stromal cells in the tumour microenvironment displa
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Tufegdžić-Vidaković, Ana. "Epigenetic determinants of context specificity in breast cancer." Thesis, University of Cambridge, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708671.

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Simpson, Louise. "Epigenetics and breast cancer : a candidate gene association study." Thesis, University of Aberdeen, 2014. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=225333.

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Cooper, Matthew L. "Selenium and the Genetics and Epigenetics of Prostate Cancer." Thesis, University of Surrey, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499409.

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Muñoz-Rodríguez, José Luis. "Postpartum Breast Cancer in Hispanic Women: Epigenetics and microRNAs." Diss., The University of Arizona, 2015. http://hdl.handle.net/10150/603490.

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The risk of breast cancer transiently increases immediately following pregnancy. Hispanic women have one of the highest rates of postpartum breast cancers of all racial/ethnic minority groups in the US. The biology that underlies this risk window and the effect on the natural history of the disease is unknown. MicroRNAs (miRNAs) are small non-coding RNAs that have been shown to be dysregulated in breast cancer. In this study, we measured the miRNA expression of 56 tumors from a case series of multiparous Hispanic women and assessed the pattern of expression by time since last full-term pregnan
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Hinshelwood, Rebecca Garvan Institute of Medical Research UNSW. "Epigenetic changes in breast cancer." Publisher:University of New South Wales. Garvan Institute of Medical Research, 2009. http://handle.unsw.edu.au/1959.4/43633.

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Changes in the epigenetic landscape are widespread in neoplasia, with de novo methylation and histone repressive marks commonly occurring in association with gene silencing. However, understanding the dynamics of epigenetic changes is often hindered due to the absence of adequate in vitro model systems that accurately reflect events occurring in vivo. Human mammary epithelial cells (HMECs) grown under standard culture conditions enter a growth arrest termed selection, but a subpopulation is able to escape from arrest and continue to proliferate. These cells, called post-selection cells, hav
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Custodio, Rojo Joaquín. "Epigenetic mechanisms in colorectal cancer." Doctoral thesis, Universitat Autònoma de Barcelona, 2014. http://hdl.handle.net/10803/287891.

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Mitjançant tècniques d’anàlisi de tot el genoma hem sigut capaços d'identificar regions diferencialment metilades entre teixit de colon normal i tumoral provinents del mateix pacient. Un dels gens més importants és AKR1B1, l'illa CpG associada al promotor d’aquest gen esdevé hipermetilada en un 90% dels càncers colorectals estudiats (més de 200), aquesta troballa ha estat confirmada en dos sets de mostres independents. Sorprenentment, la hipermetilació no està acompanyada per una clara baixada de l’expressió d’AKR1B1, de fet vam observar que aquesta hipermetilació estava associada amb el silen
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Kutanzi, Kristy, and University of Lethbridge Faculty of Arts and Science. "The role of epigenetics in the rat mammary gland." Thesis, Lethbridge, Alta. : University of Lethbridge, Dept. of Biological Sciences, c2010, 2010. http://hdl.handle.net/10133/2492.

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Epigenetics plays an important role in carcinogenesis with heritable changes in DNA methylation and histone modifications intricately linked to the initiation, promotion, and progression of cancer. Evidence shows that a number of chemical and physical agents can induce epigenetic changes during carcinogenesis. Two such agents, estrogen and ionizing radiation, are generally recognized as being carcinogenic. Yet the epigenetic repercussions of these carcinogens remain relatively unknown. More importantly, the combined effect of these carcinogens has never been addressed in vivo from an epigeneti
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Wu, Jiejun. "Genome-wide analysis of epigenetics and alternative promoters in cancer cells." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1187019769.

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Books on the topic "Cancer epigenetics"

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Dumitrescu, Ramona G., and Mukesh Verma, eds. Cancer Epigenetics. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-612-8.

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Verma, Mukesh, ed. Cancer Epigenetics. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-1804-1.

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Giordano, Antonio, and Marcella Macaluso, eds. Cancer Epigenetics. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118005743.

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O, Tollefsbol Trygve, ed. Cancer epigenetics. CRC Press/Taylor & Francis Group, 2009.

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Kalkan, Rasime, ed. Cancer Epigenetics. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-42365-9.

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Sarkar, Fazlul H., ed. Epigenetics and Cancer. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-6612-9.

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Mehdipour, Parvin, ed. Epigenetics Territory and Cancer. Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-017-9639-2.

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Kabekkodu, Shama Prasada, Aniruddha Chatterjee, and Sanjiban Chakrabarty, eds. Epigenetics of Cervical Cancer. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-96-3985-4.

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Antonio, Giordano. Cancer epigenetics: Biomolecular therapeutics in human cancer. Wiley-Blackwell, 2011.

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Berger, Nathan A., ed. Epigenetics, Energy Balance, and Cancer. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-41610-6.

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Book chapters on the topic "Cancer epigenetics"

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Carlberg, Carsten, and Ferdinand Molnár. "Cancer Epigenetics." In Human Epigenetics: How Science Works. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22907-8_8.

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Demircan, Berna, and Kevin Brown. "Cancer Epigenetics." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_807-2.

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Demircan, Berna, and Kevin Brown. "Cancer Epigenetics." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_807.

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Schulz, Wolfgang A. "Cancer Epigenetics." In Molecular Biology of Human Cancers. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-16286-2_8.

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Carlberg, Carsten. "Cancer Epigenetics." In Gene Regulation and Epigenetics. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-68730-3_15.

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Paro, Renato, Ueli Grossniklaus, Raffaella Santoro, and Anton Wutz. "Epigenetics and Cancer." In Introduction to Epigenetics. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68670-3_8.

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AbstractAlterations in chromatin function and epigenetic mechanisms are a hallmark of cancer. The disruption of epigenetic processes has been linked to altered gene expression and to cancer initiation and progression. Recent cancer genome sequencing projects revealed that numerous epigenetic regulators are frequently mutated in various cancers. This information has not only started to be utilized as prognostic and predictive markers to guide treatment decisions but also provided important information for the understanding of the molecular mechanisms of epigenetic regulation in both physiologic
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Montanari, Micaela, Antonio Giordano, Marcella Cintorino, and Marcella Macaluso. "Epigenetic Modulation of Cell Cycle: An Overview." In Cancer Epigenetics. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118005743.ch1.

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Fratta, Elisabetta, Luca Sigalotti, Alessia Covre, et al. "Epigenetic Mechanisms in Cancer Formation and Progression." In Cancer Epigenetics. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118005743.ch10.

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Romano, Gaetano. "Recent Advances in the Field of Stem Cell Research: Toward the Definition of the Epigenetic and Genetic Codes of Pluripotency." In Cancer Epigenetics. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118005743.ch11.

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Otvos, Laszlo. "Potential of Heat Shock Protein Targeting for Human Therapy." In Cancer Epigenetics. John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118005743.ch12.

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Conference papers on the topic "Cancer epigenetics"

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Andrade, Gustavo Moreira, Antonio Márcio Teodoro Cordeiro Silva, Amanda Hasan Figueiredo, Ana Luiza Gomes Monteiro, Leonardo Chaves de Oliveira Moraes, and Giovanna Silva Quirino. "The use of epigenetics in the treatment of triplenegative breast cancer, focusing on IncRNA." In Brazilian Breast Cancer Symposium 2023. Mastology, 2023. http://dx.doi.org/10.29289/259453942023v33s1032.

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Objective: In triple-negative breast cancer (CMTN), the standard therapeutic procedure is usually not very effective due to the aggressiveness of the disease. Therefore, it is important to identify and characterize new forms of treatment for this neoplasm. In this context, the study of the genetic material of diseases has gained notoriety among alternative forms of therapy, as long non-coding RNAs (IncRNAs) have been identified in neoplastic cells. Therefore, the aim of this study was to evaluate the use of epigenetics in the treatment of CMTN, with emphasis on lncRNAs. Methodology: A systemat
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Marques, Flavia Araújo Marques, and Matheus Moreira. "EPIGENETICS IN COLORECTAL CANCER." In Congresso Online de Atualização em Oncologia. Congresse.me, 2023. http://dx.doi.org/10.54265/mobq8354.

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Raj, Utkarsh, Saurav Kumar Mishra, Chakshu Bhatia, Pritish Kumar Varadwaj, and Gaurav Harit. "A Comprehensive Knowledgebase on Cancer Epigenetics." In 2018 International Conference on Bioinformatics and Systems Biology (BSB). IEEE, 2018. http://dx.doi.org/10.1109/bsb.2018.8770626.

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Jones, Peter A. "Abstract IA01: The epigenetics of cancer." In Abstracts: AACR Special Conference on Bladder Cancer: Transforming the Field; May 18-21, 2019; Denver, CO. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1557-3265.bladder19-ia01.

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Miyamoto, K., and T. Ushijima. "MicroRNAs and epigenetics in human breast cancer." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-4051.

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Waterland, Robert A. "Abstract PL02-01: Nutrition, epigenetics, and cancer." In Abstracts: Thirteenth Annual AACR International Conference on Frontiers in Cancer Prevention Research; September 27 - October 1, 2014; New Orleans, LA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1940-6215.prev-14-pl02-01.

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Zhao, Xinyue. "Review of Role of Epigenetics in Cancer Research." In ICBET '21: 2021 11th International Conference on Biomedical Engineering and Technology. ACM, 2021. http://dx.doi.org/10.1145/3460238.3460263.

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Brown, Myles. "Abstract IA15: Epigenetics of hormone dependence." In Abstracts: AACR Special Conference on Chromatin and Epigenetics in Cancer - June 19-22, 2013; Atlanta, GA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.cec13-ia15.

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Riggins, Karen, Benjamin Musher, Michael Scheurer, et al. "Abstract 1251: Epigenetics in early onset colorectal cancer (EOCRC)." In Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-1251.

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Thakkar, Kaushik N., Shiming Jiang, Sabrina Stratton, and Michelle Barton. "Abstract B17: TRIM24 links epigenetics and metabolism in cancer." In Abstracts: AACR Special Conference: Metabolism and Cancer; June 7-10, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.metca15-b17.

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Reports on the topic "Cancer epigenetics"

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Wang, Ying yuan, Zechang Chen, Luxin Zhang, et al. A systematic review and network meta-analysis: Role of SNPs in predicting breast carcinoma risk. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.2.0092.

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Review question / Objective: P: Breast cancer patient; I: Single nucleotide polymorphisms associated with breast cancer risk; C: Healthy person; O: By comparing the proportion of SNP mutations in the tumor group and the control group, the effect of BREAST cancer risk-related SNP was investigated; S: Case-control study. Condition being studied: Breast cancer (BC) is one of the most common cancers among women, and its morbidity and mortality have continued to increase worldwide in recent years, reflecting the strong invasiveness and metastasis characteristics of this cancer. BC is a complex dise
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Murphy, Susan K. Epigenetic Characterization of Ovarian Cancer. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada452440.

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Murphy, Susan K. Epigenetic Characterization of Ovarian Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada509791.

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Margueron, Raphael F. An Epigenetic Link to Prostate Cancer. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada484222.

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Reich, Norbert R. An Epigenetic Approach to Breast Cancer Treatment. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada429260.

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Williams, Kristin P. Epigenetic Control of Tamoxifen-Resistant Breast Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada581650.

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Euhus, David. Epigenetic Testing for Breast Cancer Risk Stratification. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada582383.

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Williams, Kristin P. Epigenetic Control of Tamoxifen-Resistant Breast Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada601260.

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Liao, Jianhua, Jingting Liu, Baoqing Liu, Chunyan Meng, and Peiwen Yuan. Effect of OIP5-AS1 on clinicopathological characteristics and prognosis of cancer patients: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.10.0118.

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Review question / Objective: According to recent studies, long non-coding RNA (lncRNAs) i.e., OPA-interacting protein 5 antisense RNA 1 (OIP5-AS1) has an important role in various carcinomas. However, its role in the cancer is contradictory. Therefore, we aimed to evaluate the link between OIP5-AS1 and cancer patients' clinicopathological characteristics and prognosis to better understand OIP5-AS1's role in cancer. Condition being studied: Reported studies have revealed that long non-coding RNA (lncRNAs) are considerably involved in crucial physiological events in several carcinomas, it can in
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Simon, Jeffrey A., and Carol A. Lange. Histone Methylation and Epigenetic Silencing in Breast Cancer. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada491094.

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