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1

1940-, Tabuchi K., ed. Biological aspects of brain tumors: Proceedings of the 8th Nikko Brain Tumor Conference, Karatsu (Saga) 1990. Tokyo: Springer-Verlag, 1991.

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2

Brain tumors: Leaving the Garden of Eden : a survival guide to diagnosis, learning the basics, getting organized and finding your medical team. Encino, Cal: Shilysca Press, 2004.

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3

Gillespie, Joanne. Brave heart. London: Century, 1989.

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4

Disrupted: On fighting death & keeping faith. Eugene, Or: Cascade Books, 2011.

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5

Kressel, Duffner Patricia, ed. Brain tumors in children: Principles of diagnosis and treatment. 2nd ed. New York: Raven Press, 1994.

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6

Kline, Jerry D. An unremarkable man: A survivor's story. Enumclaw, WA: Pleasant Word, a division of WinePress Group, 2010.

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7

Károly, Lapis, Eckhardt S, and International Union Against Cancer, eds. Radiotherapy, paediatric oncology, neurooncology. Budapest: Akadémiai Kiadó, 1987.

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8

Gutiérrez, Lucía M. Neuro-oncology and cancer targeted therapy. New York: Nova Biomedical Books, 2010.

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9

Iwata, Takanobu. Isha ga makki gan kanja ni natte wakatta koto: Aru nōgekai ga nōshōshō to tatakata seizetsu na hibi. Tōkyō: Chūkei Shuppan, 1998.

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10

Virginia. General Assembly. Joint Legislative Audit & Review Commission. Evaluation of House Bill 2156: mandated coverage of second opinions for primary malignant brain tumor patients at NCI Comprehensive Cancer Center. Richmond, Va: Commonwealth of Virginia, 2007.

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11

Iwata, Takanobu. Zoku isha ga makki gan kanja ni natte wakatta koto: Kazoku no tatakai to kango no kiroku. Tōkyō: Chūkei Shuppan, 1998.

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12

Quelques mois pour l'aimer. Paris: Fixot, 1994.

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13

Craig, the boy who lives. Dublin: Gill & Macmillan, 2011.

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14

Gunther, John. Death be not proud: A memoir. New York: HarperPerennial, 1992.

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15

Gunther, John. Death be not proud. Hampton, New Hampshire: Curley, 1992.

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16

T, Thomas D. G., and Graham David I, eds. Malignant brain tumours. London: Springer-Verlag, 1995.

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17

CancerBACUP. Understanding Brain Tumours. CancerBACUP (British Association of Cancer United, 1995.

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18

Understanding Brain Tumours. CancerBACUP (British Association of Cancer United, 2000.

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19

Peter, Chambers, and BACUP, eds. Understanding brain tumours. London: BACUP, 1994.

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20

CancerBACUP. Understanding Brain Tumours. CancerBACUP (British Association of Cancer United, 1997.

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21

Preusser, Matthias, Gabriele Schackert, and Brigitta G. Baumert. Metastatic brain tumours. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0019.

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Brain metastasis is a common clinical challenge in cancer patients, particularly those with lung cancer, breast cancer, and melanoma. The prognosis is poor, with median overall survival times measured in months for most patient populations. Established treatments include neurosurgical resection, radiotherapy (including stereotactic radiosurgery and stereotactic radiotherapy, whole-brain radiotherapy, and new radiation techniques), and supportive care measures. Recently, more and more targeted therapies such as EGFR inhibitors, HER2 antagonists, BRAF inhibitors, ALK inhibitors, and immune checkpoint inhibitors are demonstrating some efficacy in brain metastasis patients and should be considered in the clinical setting.
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22

Sara, Booth, and Bruera Eduardo, eds. Palliative care consultations in primary and metastatic brain tumours. Oxford: Oxford University Press, 2004.

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23

Ajithkumar, Thankamma, Ann Barrett, Helen Hatcher, and Natalie Cook. CNS tumours. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235636.003.0005.

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Tumours of the CNS are characterized by a remarkable diversity of pathological type and behaviour. Although rare, they affect people of all ages and hence have a disproportionate influence on the number of years of life lost to cancer. With few exceptions there is a marked consistency of incidence worldwide. Some types of brain tumour are increasing in incidence and, after many years of therapeutic stagnation, there have recently been some important improvements in treatment and outcome. For both of these reasons the subdiscipline of neuro-oncology is of increasing relevance in cancer medicine....
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24

L, Domellöf, ed. Drug delivery in cancer treatment III: Home care--symptom control, economy, brain tumours. Berlin: Springer-Verlag, 1990.

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25

Drug Delivery in Cancer Treatment III: Home Care - Symptom Control, Economy, Brain Tumours. Springer, 2011.

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26

Hodgkiss, Andrew. Biological Psychiatry of Cancer and Cancer Treatment. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.001.0001.

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As long-term cancer survival becomes a widely shared experience, the quality of life of people living with and beyond a cancer diagnosis is increasingly important. Optimizing the prevention and treatment of any psychiatric consequences of certain tumours and treatments is now central to high-quality cancer care. This book—a rather original addition to the oncology and psycho-oncology literature—aims to equip oncology clinicians with the knowledge to more expertly prevent, detect, and manage the ‘organic’ psychiatric disorders experienced by people with cancer. It will also serve as a valuable introduction to contemporary oncology for psychiatrists.The psychiatry of cancer is a distinct subject within the wider field of psycho-oncology. Psychiatric disorders arising through direct biological mechanisms from particular tumours or cancer treatments is a narrower topic still, but one in which oncologists are required to have expertise. This book considers in detail the psychiatric aspects of pro-inflammatory cytokines, endocrine paraneoplastic syndromes, onconeuronal antibodies, brain irradiation, hormone deprivation, glucocorticoid treatment, conventional chemotherapies, and molecularly targeted agents.
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27

Glasper, Edward Alan, Gillian McEwing, and Jim Richardson, eds. Oncology. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780198569572.003.0018.

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Related physiology 596The child with cancer 598Paediatric brain tumours 600Bone tumours 602Neuroblastoma 604Rhabdomyosarcoma (RMS) 606Acute myeloid leukaemia (AML) 608Acute lymphoblastic leukaemia (ALL) 610Wilms tumour 612T-cell acute lymphoblastic leukaemia 614Lymphoma 616Bone-marrow transplantation 618Related skills...
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28

Domellof, L. Drug Delivery in Cancer Treatment III: Home Care Symptom Control, Economy, Brain Tumours (Eso Monographs (European School of Oncology)). Springer, 1990.

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29

Michaud, Dominique, David Savitz, and Lorelei Mucci. Brain Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0024.

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cBrain tumors constitute an array of histologic types, the most common being meningioma and glioma. Unlike other cancers, both benign and malignant brain tumors are concerning for survival because of their anatomic location. Two-thirds of brain tumors are benign. The most well established risk factor is high dose ionizing radiation, based on studies of atomic bomb survivors as well as children treated for tinea capitis. In contrast, nonionizing radiation including from cellular telephones, is not a risk factor. Tobacco use does not appear to be associated with glioma or meningioma. There is fairly consistent evidence of an inverse association between allergies and asthma with risk of glioma, potentially through levels of IgE. Finally, occupational epidemiology studies suggest potential positive associations with specific exposures. The identification of modifiable risk factors for brain tumors has been challenging, due in part to the diversity of tumor subtypes.
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30

Hodgkiss, Andrew. Psychiatric consequences of particular cancers. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198759911.003.0004.

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Certain tumour types can cause psychopathology through direct biological mechanisms such as metastatic spread to the brain, release of onconeuronal antibodies, ectopic hormone secretion, or release of pro-inflammatory cytokines. Lung cancers, adenocarcinoma of the pancreas, brain tumours, and ovarian tumours are considered in detail. Confusional states due to brain metastases, syndrome of inappropriate ADH secretion, hypercalcaemia of malignancy, and anti-Hu encephalitis are found in lung cancers. Severe depression, due to interleukin-6 release and its actions on the HPA axis and tryptophan metabolism, is common in adenocarcinoma of the pancreas. Anti-NMDA-receptor limbic encephalitis, clinically indistinguishable from acute schizophrenia, can complicate teratomas. Gliomas, pituitary tumours, and thyroid, adrenal, and testicular tumours can also disrupt mental health through various biological mechanisms described here.
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31

Michael, Prados, and American Cancer Society, eds. Brain cancer. Hamilton, Ont: B.C. Decker, 2002.

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32

Brain Tumors (Contemporary Cancer Research). Humana Press, 2004.

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33

Ali-Osman, Francis. Brain Tumors (Contemporary Cancer Research). Humana Press, 2003.

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34

Thomas, David G. Malignant Brain Tumors. Springer, 1995.

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35

Translational Immunotherapy of Brain Tumors. Elsevier Science & Technology Books, 2017.

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36

Pediatric Cancer Volume 2 Pediatric Cancer. Springer, 2012.

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37

1931-, Jellinger K., ed. Therapy of malignant brain tumors. Wien: Springer-Verlag, 1987.

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38

Goldfarb, Ronald H. Brain Tumor Invasiveness. Springer, 2012.

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39

Brain Tumor Invasiveness. Springer, 2011.

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40

H, Goldfarb Ronald, ed. Brain tumor invasiveness. Dordrecht: Kluwer Academic, 1994.

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41

(Editor), Jörg-Christian Tonn, Manfred Westphal (Editor), James T. Rutka (Editor), and Stuart A. Grossman (Editor), eds. Neuro-Oncology of CNS Tumors. Springer, 2005.

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42

Smedley, Julia, Finlay Dick, and Steven Sadhra. Neurological disorders. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199651627.003.0012.

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Brain cancer 288Acute narcosis 290Parkinsonism 292Compression neuropathies 294Peripheral neuropathy 296Organophosphate poisoning 298Hand–arm vibration syndrome 300Noise-induced hearing loss 302Primary brain cancers are relatively rare with an annual incidence in UK adults of 7/100,000 population.• Although relatively rare, p brain tumour is the eighth most common tumour in people of working age...
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43

Salvatti, Elena K. Brain Cancer, Tumor Targeting, and Cervical Cancer. Nova Science Publishers, Incorporated, 2011.

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44

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, Gareth Morris-Stiff, and Madhumita Bhattacharyya. Skin cancers. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0023_update_001.

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Tumours of the central nervous system examines the epidemiology, aetiology, genetics and pathology of these heterogeneous tumours. Clinical presentation reflects the site of origin and rate of growth. Investigation usually comprises imaging (MRI superior to CT for most), and biopsy; requirement for additional staging depends on pathology. The treatment of low-grade gliomas may be delayed if small with few symptoms, otherwise surgery and/or radiotherapy. High grade gliomas may be managed with surgery, radiotherapy, and temozolomide chemotherapy in fit patients. Unfit patients should be offered supportive care only. Brief summaries are provided for management of ependymoma, pineal tumours, meningioma, germ-cell CNS tumours, pituitary tumours, CNS lymphoma, acoustic neuroma, medulloblastoma, and spinal cord tumours. Radiotherapy for primary CNS tumours is described along with its side effects, and chemotherapy for these diseases is reviewed. Brain metastases far outnumber primary brain tumours, with generally poor prognosis, but this relates both to the pathology and patient performance status. Appropriate treatment may include surgery, radiotherapy, and/or chemotherapy.
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45

Woolf, Eric C., and Adrienne C. Scheck. Ketogenic Diet as Adjunctive Therapy for Malignant Brain Cancer. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0013.

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Malignant brain tumors are devastating, and increased survival requires new therapeutic modalities. Metabolic dysregulation results in an increased need for glucose in tumor cells, suggesting that reduced tumor growth could be achieved with decreased glucose availability either through pharmacological means or use of a high-fat, low-carbohydrate ketogenic diet (KD). KD provides increased blood ketones to support energy needs of normal tissues and has been shown to reduce tumor growth, angiogenesis, inflammation, peritumoral edema, migration, and invasion. Furthermore, this diet can enhance the activity of radiation and chemotherapy in a mouse model of glioma, thus increasing survival. In vitro studies indicate that increasing ketones in the absence of glucose reduction can also inhibit cell growth and potentiate the effects of radiation. Thus, emerging data provide strong support for the use of KD in the treatment of malignant gliomas and thus far has led to a limited number of clinical trials.
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46

Tumors and Cancers: Brain - Central Nervous System. Taylor & Francis Group, 2017.

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47

Tumors and Cancers: Brain - Central Nervous System. Taylor & Francis Group, 2017.

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48

Hayat, M. A. Brain Metastases from Primary Tumors: Epidemiology, Biology, and Therapy. Elsevier Science & Technology Books, 2015.

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49

Michael, Salcman, and Congress of Neurological Surgeons, eds. Neurobiology of brain tumors. Baltimore: Williams & Wilkins, 1991.

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50

Chief complaint: Brain tumor. Sunstone Press, 2012.

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