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Journal articles on the topic 'Cancer aetiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Saw, K. C. "Aetiology of pancreatic cancer." British Journal of Surgery 78, no. 3 (March 1991): 377. http://dx.doi.org/10.1002/bjs.1800780334.

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2

Manchande, Jean-Pierre, Tony Mets, Amy Klion, ValdaM Craddock, D. I. Thurnham, Nubia Munoz, J. Wahrendorf, and M. Crespi. "AETIOLOGY OF OESOPHAGEAL CANCER." Lancet 329, no. 8548 (June 1987): 1499–500. http://dx.doi.org/10.1016/s0140-6736(87)92259-8.

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3

Craddock, V. M. "Aetiology of oesophageal cancer." European Journal of Cancer Prevention 1, no. 2 (February 1992): 89–104. http://dx.doi.org/10.1097/00008469-199202000-00002.

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4

Haddock, G., and D. C. Carter. "Aetiology of pancreatic cancer." British Journal of Surgery 77, no. 10 (October 1990): 1159–66. http://dx.doi.org/10.1002/bjs.1800771025.

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5

Kinlen, L. J. "Fat and cancer aetiology." Progress in Lipid Research 25 (January 1986): 527–31. http://dx.doi.org/10.1016/0163-7827(86)90107-4.

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6

Ogden, G. R., and A. J. Wight. "Aetiology of oral cancer: alcohol." British Journal of Oral and Maxillofacial Surgery 36, no. 4 (August 1998): 247–51. http://dx.doi.org/10.1016/s0266-4356(98)90707-0.

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7

Newby, John A., and C. Vyvyan Howard. "Environmental influences in cancer aetiology." Journal of Nutritional & Environmental Medicine 15, no. 2-3 (January 2005): 56–114. http://dx.doi.org/10.1080/13590840500535396.

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8

Griffiths, K., C. L. Eaton, and P. Davies. "Prostatic Cancer: Aetiology and Endocrinology." Hormone Research 32, no. 1 (1989): 38–43. http://dx.doi.org/10.1159/000181307.

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9

Tapio, Soile, and Bernd Grosche. "Arsenic in the aetiology of cancer." Mutation Research/Reviews in Mutation Research 612, no. 3 (June 2006): 215–46. http://dx.doi.org/10.1016/j.mrrev.2006.02.001.

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10

Ariyaratne, MHJ, and WL Dilesha. "New concepts of breast cancer aetiology." Sri Lanka Journal of Surgery 27, no. 2 (July 22, 2010): 31. http://dx.doi.org/10.4038/sljs.v27i2.2097.

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11

Oliver, Richard. "Challenging The Aetiology of Oral Cancer." Dental Update 28, no. 1 (January 2, 2001): 28. http://dx.doi.org/10.12968/denu.2001.28.1.28a.

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12

Elmasry, Karim, and Simon A. Gayther. "Ovarian cancer aetiology: facts and fiction." Journal of Family Planning and Reproductive Health Care 32, no. 2 (April 1, 2006): 82–86. http://dx.doi.org/10.1783/147118906776276297.

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13

Scully, C. "Viruses in the aetiology of cancer." British Dental Journal 164, no. 11 (June 1988): 362–64. http://dx.doi.org/10.1038/sj.bdj.4806457.

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14

Cocco, Pierluigi, and Mary H. Ward. "Occupational exposures and gastric cancer aetiology." European Journal of Gastroenterology & Hepatology 6, no. 12 (December 1994): 1089–96. http://dx.doi.org/10.1097/00042737-199412000-00004.

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15

Hill, M. J. "Aetiology of colorectal cancer: current concepts." Baillière's Clinical Gastroenterology 3, no. 3 (July 1989): 567–92. http://dx.doi.org/10.1016/0950-3528(89)90018-3.

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16

Barber, Miriam, Rebecca C. Fitzgerald, and Carlos Caldas. "Familial gastric cancer – aetiology and pathogenesis." Best Practice & Research Clinical Gastroenterology 20, no. 4 (January 2006): 721–34. http://dx.doi.org/10.1016/j.bpg.2006.03.014.

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17

McMullin, Mary Frances, and Lesley Ann Anderson. "Aetiology of Myeloproliferative Neoplasms." Cancers 12, no. 7 (July 6, 2020): 1810. http://dx.doi.org/10.3390/cancers12071810.

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Myeloproliferative neoplasms (MPNs) have estimated annual incidence rates for polycythemia vera (PV), essential thrombocythemia (ET), and primary myelofibrosis of 0.84, 1.03, and 0.47 per 100,000. Prevalence is much higher, particularly for PV and ET, as mortality rates are relatively low. Patients are often concerned about why they developed an MPN and epidemiological studies enable the identification of potential causative factors. Previous work in small heterogeneous studies has identified a variety of risk factors associated with MPNs including family history of MPN, autoimmune conditions, some occupational exposures, and blood donation. At a population level, germline predisposition factors in various populations have been associated with MPNs. The pilot MOSAICC (Myeloproliferative Neoplasm: An In-depth Case-Control) study is one of the largest epidemiological studies in MPN ever carried out to date. It demonstrated the most effective methods for carrying out a significant epidemiological study in this patient group including the best way of recruiting controls, as well as how to evaluate occupational and lifestyle exposures, evaluate symptoms, and collect biological samples. Significant results linked to MPNs in the pilot study of 106 patients included smoking, obesity, and childhood socioeconomic status. The methodology is now in place for a much larger ongoing MOSAICC study which should provide further insight into the potential causes of MPNs.
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18

Gerson, Stanton L. "MGMT: its role in cancer aetiology and cancer therapeutics." Nature Reviews Cancer 4, no. 4 (April 2004): 296–307. http://dx.doi.org/10.1038/nrc1319.

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19

McNally, Richard JQ. "The Epidemiology of Childhood Cancers." European Oncology & Haematology 05, no. 01 (2009): 79. http://dx.doi.org/10.17925/eoh.2009.05.1.79.

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In this article, the recent epidemiological literature on childhood cancer is reviewed. This includes findings from descriptive, case-control and cohort studies. The aetiology of most childhood cancers is unclear. Both genetic and environmental factors are likely to contribute. Increasing incidence, findings of clustering and seasonality in the incidence of certain cancers support a role for environmental agents in aetiology. The evidence concerning putative risk factors is considered and suggests that the aetiology is likely to be multifactorial and involve a number of different agents. These include infections, ionising radiation, certain chemical exposures, parental smoking, parental alcohol consumption and hair dyes. Conversely, breastfeeding and certain dietary supplements may convey protection. Recent findings regarding electromagnetic fields suggest that this factor is not likely to have a major role in aetiology.
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20

Muñoz, Nubia. "Human Papillomavirus in the Aetiology and Prevention of Genital Cancers." European Review 24, no. 1 (February 2016): 31–38. http://dx.doi.org/10.1017/s106279871500040x.

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In the first part of this paper the molecular epidemiological evidence linking HPV to cervical cancer and other genital cancers is reviewed based mainly on the research work of the author. In the second part, the implications of the epidemiological findings for the prevention of cervical cancer are discussed in two areas: in primary prevention by the use of prophylactic HPV vaccines and in secondary prevention by increasing the accuracy of cervical cancer screening.
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21

Cumberbatch, Marcus G. K., and Aidan P. Noon. "Epidemiology, aetiology and screening of bladder cancer." Translational Andrology and Urology 8, no. 1 (March 2019): 5–11. http://dx.doi.org/10.21037/tau.2018.09.11.

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22

Banning, Maggi. "Advanced breast cancer: aetiology, pathogenesis and treatment." Cancer Nursing Practice 6, no. 3 (April 2007): 35–39. http://dx.doi.org/10.7748/cnp2007.04.6.3.35.c4206.

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23

Rowley, K. H. M., and M. D. Mason. "The aetiology and pathogenesis of prostate cancer." Clinical Oncology 9, no. 4 (January 1997): 213–18. http://dx.doi.org/10.1016/s0936-6555(97)80003-9.

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24

Key, Timothy. "Micronutrients and cancer aetiology: the epidemiological evidence." Proceedings of the Nutrition Society 53, no. 3 (November 1994): 605–14. http://dx.doi.org/10.1079/pns19940069.

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25

Allam, M. F., and R. A. Lucena. "Aetiology of sex differences in colorectal cancer." European Journal of Cancer Prevention 10, no. 3 (June 2001): 299–300. http://dx.doi.org/10.1097/00008469-200106000-00016.

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26

Gui, Gerald. "Male breast cancer: aetiology and clinical features." Trends in Urology & Men's Health 3, no. 3 (May 2012): 29–31. http://dx.doi.org/10.1002/tre.266.

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27

VAN DEN BRANDT, P. A., R. A. GOLDBOHM, P. VAN T VEER, R. J. J. HERMUS, and F. STURMANS. "Dietary Habits and the Aetiology of Cancer." International Journal of Epidemiology 17, no. 2 (1988): 472. http://dx.doi.org/10.1093/ije/17.2.472.

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28

ZARIDZE, D. G., and P. BOYLE. "Cancer of the Prostate: Epidemiology and Aetiology." British Journal of Urology 59, no. 6 (June 1987): 493–502. http://dx.doi.org/10.1111/j.1464-410x.1987.tb04862.x.

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29

Salabe, G. B. "Aetiology of thyroid cancer: An epidemiological overview." Biomedicine & Pharmacotherapy 47, no. 6-7 (November 1993): 244. http://dx.doi.org/10.1016/0753-3322(93)90131-4.

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30

Holmes, Susan. "Xerostomia: aetiology and management in cancer patients." Supportive Care in Cancer 6, no. 4 (June 1998): 348–55. http://dx.doi.org/10.1007/s005200050176.

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31

Holloway, Susan M. "Coefficients of Relationship by Isonymy among Scottish Males with Multiple Primary Cancers." Disease Markers 12, no. 3 (1994): 205–14. http://dx.doi.org/10.1155/1994/843108.

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Patients with heritable cancer syndromes often develop multiple primary cancers (mpc) affecting one or more organs or tissues. However, it is less clear whether the presence of mpc in a patient implies that genetic factors have an important role in aetiology. We have investigated this by comparing the surname distributions of 11205 males with mpc on the Scottish Cancer Register, with corresponding distributions in male controls and in males with individual cancers, by calculating coefficients of relationship by isonymy. Our results suggest that although genetic factors may have a role in the aetiology of mpc, particularly if the cancers affect different tissues or organs, their effect is small compared with that of environmental factors.
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32

Alvi, Qurratulann, Gul Muhammad Baloch, Karuthan Chinna, and Ali Dabbagh. "Lifestyle and reproductive health: the aetiology of ovarian cancer in Pakistan." F1000Research 9 (August 4, 2020): 901. http://dx.doi.org/10.12688/f1000research.24866.1.

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Ovarian cancer is a fatal gynaecological cancer and eighth most common cancer in women globally. Lifestyle, reproductive and sociodemographic factors are among the influential parameters that may significantly affect the risk of ovarian cancer and its mortality rate. However, the epidemiological investigations have shown that the risk of ovarian cancers associated with these factors is different in varied geographical distributions. Lifestyle and reproductive factors have not been investigated thoroughly across a wide cultural diversity. The objective of this study is to investigate the association of these factors with ovarian cancer in Pakistan. This investigation will focus on the lifestyle effects of fat intake, intake of tea, habitual exercise, use of talc, personal hygiene, habit of holding urine for long time, obesity on ovarian cancer among Pakistani women. Reproductive variables will include age at menarche, natural menopausal age, parity, nulliparity (miscarriages, abortion, stillbirths), infertility, fertility treatment, tubal ligation, oral contraceptive use, and family history of breast or ovarian cancer. Sociodemographic variables will include effect of age, income, education, and geographical location. A case-control study will be conducted in the major cancer hospitals of Pakistan and the patients will also be interviewed. The controls will be recruited outside the hospital. For controls the same age limit and residency requirements will be applied. The information gained from this research will be an important contribution to develop programs for health promotion, with a focus on ovarian cancer prevention and women’s health. The findings could be used for health policies and planning to prevent ovarian cancer. The research will pave the way for a public policy and interventions to reduce the burden of ovarian cancer in Pakistan.
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33

Stringer, Susan. "Aetiology and management of non-melanoma skin cancer." Cancer Nursing Practice 9, no. 1 (February 9, 2010): 20–24. http://dx.doi.org/10.7748/cnp2010.02.9.1.20.c7547.

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34

Heys, Steven D., Ian C. Smith, and Oleg Eremin. "Hypercalcaemia in patients with cancer: aetiology and treatment." European Journal of Surgical Oncology (EJSO) 24, no. 2 (April 1998): 139–42. http://dx.doi.org/10.1016/s0748-7983(98)91589-x.

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35

Stang, Andreas, and Rita K. Schmutzler. "The Epidemiology and Aetiology of Female Breast Cancer." Breast Care 4, no. 2 (2009): 73–74. http://dx.doi.org/10.1159/000212103.

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36

Kelemen, Linda E., Thomas A. Sellers, and Celine M. Vachon. "Can genes for mammographic density inform cancer aetiology?" Nature Reviews Cancer 8, no. 10 (September 5, 2008): 812–23. http://dx.doi.org/10.1038/nrc2466.

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37

Banning, Maggi. "Advanced breast cancer: aetiology, treatment and psychosocial features." British Journal of Nursing 16, no. 2 (January 2007): 86–90. http://dx.doi.org/10.12968/bjon.2007.16.2.22765.

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38

Ruiz-Rebollo, María Lourdes, Lourdes del Olmo-Martínez, Benito Velayos-Jiménez, Maria Fe Muñoz, María Álvarez-Quiñones-Sanz, and José Manuel González-Hernández. "Aetiology and prevalence of post-colonoscopy colorectal cancer." Gastroenterología y Hepatología (English Edition) 39, no. 10 (December 2016): 647–55. http://dx.doi.org/10.1016/j.gastre.2016.01.006.

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39

Smith, C. J. "Oral cancer and precancer: background, epidemiology and aetiology." British Dental Journal 167, no. 11 (December 1989): 377–83. http://dx.doi.org/10.1038/sj.bdj.4807055.

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40

Blenkiron, Cherie, and Eric A. Miska. "miRNAs in cancer: approaches, aetiology, diagnostics and therapy." Human Molecular Genetics 16, R1 (April 15, 2007): R106—R113. http://dx.doi.org/10.1093/hmg/ddm056.

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41

ADAMI, H. "The aetiology and pathogenesis of human breast cancer." Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis 333, no. 1-2 (December 1995): 29–35. http://dx.doi.org/10.1016/0027-5107(95)00128-x.

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42

Boyle, Peter. "Diet in the aetiology of cancer editorial comment." European Journal of Cancer 30, no. 2 (January 1994): 133. http://dx.doi.org/10.1016/0959-8049(94)90070-1.

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43

Miller, A. B., F. Berrino, M. Hill, P. Pietinen, E. Riboli, and J. Wahrendorf. "Diet in the aetiology of cancer: a review." European Journal of Cancer 30, no. 2 (January 1994): 207–20. http://dx.doi.org/10.1016/0959-8049(94)90088-4.

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44

Elwood, J. M. "The epidemiology and aetiology of melanoma." Melanoma Research 3, no. 1 (March 1993): 3. http://dx.doi.org/10.1097/00008390-199303000-00001.

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45

Mansouri, Siham, Ayoub Bounsir, Laila Benzekri, and Badr Hassam. "Toe necrosis: report of uncommon aetiology." BMJ Case Reports 12, no. 7 (July 2019): e230322. http://dx.doi.org/10.1136/bcr-2019-230322.

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Toe necrosis has many aetiologies. The most common one is ischaemia. In fact, both direct and indirect toe necrosis, as well as related lower limb gangrene, could be secondary to mechanical trauma, infections, pharmacological sensitivity, cancer, blue toe syndrome and other granulomatous diseases. We report a case of toes necrosis caused by an uncommon aetiology.
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46

Snow, Anson, Charite Ricker, and Gino K. In. "Two synchronous malignancies: nodular melanoma and renal cell carcinoma in a patient with an underlying germline BRCA2 mutation." BMJ Case Reports 12, no. 6 (June 2019): e227625. http://dx.doi.org/10.1136/bcr-2018-227625.

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Modernised genetic testing among patients with cancer has led to an increasing wealth of knowledge regarding cancer biology and aetiology. Furthermore, some germline mutations have the potential to direct therapeutic approaches as well. While BRCA1/2 mutations are well-established risk factors for breast and ovarian cancers, their impact on other cancers is less understood. We describe a patient with a germline BRCA2 mutation who developed synchronous melanoma and renal cell carcinoma, but responded well to treatment and is now cancer free.
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47

Jones, Oliver, Stephanie Hackett, Despoina Chatzistavrianou, and David Newsum. "Head and Neck cancer part 1: diagnosis and classification." Dental Update 46, no. 8 (September 2, 2019): 722–29. http://dx.doi.org/10.12968/denu.2019.46.8.722.

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Head and neck cancer is a group of cancers that affects the oropharynx, nasopharynx, laryngopharynx and neck. With improving survival rates for patients, general dental practitioners (GDPs) are becoming increasingly likely to engage in the care of affected patients. Therefore, a sound clinical knowledge of the aetiology, treatment and rehabilitation for head and neck cancer is essential for all practitioners who may be involved in the care pathways for such patients. CPD/Clinical Relevance: This paper provides an update on the prevalence, risk factors, diagnosis and classification of head and neck cancer.
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48

Kluttig, Alexander, and Andrea Schmidt-Pokrzywniak. "Established and Suspected Risk Factors in Breast Cancer Aetiology." Breast Care 4, no. 2 (2009): 82–87. http://dx.doi.org/10.1159/000211368.

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49

Collins, Andrew, Susan Duthie, and Marion Ross. "Micronutrients and oxidative stress in the aetiology of cancer." Proceedings of the Nutrition Society 53, no. 1 (March 1994): 67–75. http://dx.doi.org/10.1079/pns19940011.

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50

Ponz de Leon, M. "Aetiology of colorectal cancer and relevance of monogenic inheritance." Gut 53, no. 1 (January 1, 2004): 115–22. http://dx.doi.org/10.1136/gut.53.1.115.

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