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Relevant bibliographies by topics / Cancer aetiology

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Academic literature on the topic 'Cancer aetiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Cancer aetiology"

1

Saw, K. C. "Aetiology of pancreatic cancer." British Journal of Surgery 78, no. 3 (March 1991): 377. http://dx.doi.org/10.1002/bjs.1800780334.

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Manchande, Jean-Pierre, Tony Mets, Amy Klion, ValdaM Craddock, D. I. Thurnham, Nubia Munoz, J. Wahrendorf, and M. Crespi. "AETIOLOGY OF OESOPHAGEAL CANCER." Lancet 329, no. 8548 (June 1987): 1499–500. http://dx.doi.org/10.1016/s0140-6736(87)92259-8.

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Craddock, V. M. "Aetiology of oesophageal cancer." European Journal of Cancer Prevention 1, no. 2 (February 1992): 89–104. http://dx.doi.org/10.1097/00008469-199202000-00002.

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Haddock, G., and D. C. Carter. "Aetiology of pancreatic cancer." British Journal of Surgery 77, no. 10 (October 1990): 1159–66. http://dx.doi.org/10.1002/bjs.1800771025.

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Kinlen, L. J. "Fat and cancer aetiology." Progress in Lipid Research 25 (January 1986): 527–31. http://dx.doi.org/10.1016/0163-7827(86)90107-4.

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Ogden, G. R., and A. J. Wight. "Aetiology of oral cancer: alcohol." British Journal of Oral and Maxillofacial Surgery 36, no. 4 (August 1998): 247–51. http://dx.doi.org/10.1016/s0266-4356(98)90707-0.

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Newby, John A., and C. Vyvyan Howard. "Environmental influences in cancer aetiology." Journal of Nutritional & Environmental Medicine 15, no. 2-3 (January 2005): 56–114. http://dx.doi.org/10.1080/13590840500535396.

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Griffiths, K., C. L. Eaton, and P. Davies. "Prostatic Cancer: Aetiology and Endocrinology." Hormone Research 32, no. 1 (1989): 38–43. http://dx.doi.org/10.1159/000181307.

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Tapio, Soile, and Bernd Grosche. "Arsenic in the aetiology of cancer." Mutation Research/Reviews in Mutation Research 612, no. 3 (June 2006): 215–46. http://dx.doi.org/10.1016/j.mrrev.2006.02.001.

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Ariyaratne, MHJ, and WL Dilesha. "New concepts of breast cancer aetiology." Sri Lanka Journal of Surgery 27, no. 2 (July 22, 2010): 31. http://dx.doi.org/10.4038/sljs.v27i2.2097.

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Dissertations / Theses on the topic "Cancer aetiology"

1

Al-Dabbagh, S. A. "Nitrates in the aetiology of gastric cancer." Thesis, University of Oxford, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.353051.

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Key, Timothy J. A. "Studies in the epidemiology of sex hormones and cancer." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.238136.

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Richiardi, Lorenzo. "New evidence on germ-cell testicular cancer aetiology /." Stockholm, 2003. http://diss.kib.ki.se/2004/91-7349-733-9/.

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Waller, Josephine. "The viral aetiology of cervical cancer : psychosocial issues." Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1446882/.

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This work stems from the discovery that certain sexually transmitted types of human papillomavirus (HPV) are the main causal agents in cervical carcinogenesis. The thesis sets out to explore the psychosocial issues that arise from linking a sexually transmitted infection with cervical cancer. Four studies were carried out. Study 1 was a survey of women attending a well-woman clinic (n=1032) and assessed awareness and knowledge about HPV. Study 2 used a population representative sample of men and women (n=1937) to assess beliefs about the risk factors for cervical cancer. Study 3 used in-depth interviews to explore the beliefs and experiences of 74 women who had taken part in HPV testing. Study 4 was a continuation of Study 3, in which 30 women were interviewed following participation in their second HPV test, a year after the first. Awareness of HPV and its link with cervical cancer was found to be low. Although there was higher awareness of sexual activity as a risk factor for cervical cancer, this was far from universal. Women testing positive for HPV who understood that it was sexually transmitted frequently reported negative emotional and social responses, different from those that have been found among women with abnormal smear test results. Leventhal's Common Sense Model of self-regulation in health and illness provided a useful framework within which to conceptualise the relationship between women's cognitive representations of HPV and their responses to the infection. It seemed that women were also engaged in the self-regulation of their relationships and were motivated to develop representations of HPV that did not impugn their current partners. Diagnosis with persistent HPV infection was associated with higher levels of anxiety about health and with the desire for immediate further investigation by colposcopy, rather than continued surveillance. The introduction of HPV testing and vaccination should be accompanied by widespread public education. If information provision is not handled in a sensitive way, it could cause confusion and stigmatise cervical cancer. More research is needed to develop ways to communicate information about HPV effectively.

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Hammond, Victoria Naomi. "The role of genetic factors in breast cancer aetiology." Thesis, University of Southampton, 2010. https://eprints.soton.ac.uk/162737/.

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Breast cancer is the most common cancer in women and is also the leading cause of cancer mortality in women. There are several known risk factors for breast cancer including genetic factors which account for at least 25% of the incidence of breast cancer, although only a small proportion of this is a result of mutations in known high penetrance susceptibility genes. The majority of genetic risk is now thought to be due to common genetic variants, for example single nucleotide polymorphisms (SNPs). We investigated whether SNPs in candidate genes, with a biological reason for being of interest to study in relation to breast cancer, were correlated with the development of tumours with a certain phenotype, such as grade, lymph node involvement, oestrogen receptor status and the presence of distant metastases. We genotyped 206 SNPs across 30 candidate genes in 1001 patients. Association was performed using Cochran-Armitage trend test and 2-by-3 tables of disease by genotype. We replicated observations from previous studies such as the association of SNPs in FGFR2, TNRC9 and ATM with oestrogen receptor status and identified novel associations of SNPs in the oestrogen receptor gene and matrix metalloproteinase-9 gene (MMP-9) with grade and presence of distant metastasis respectively. The function of two promoter SNPs in MMP-9 were further investigated using luciferase reporter gene assays. The C allele of rs3918242 had a 1.5 fold increase in MMP-9 expression in MDA-MB- 231 cells and the A allele of rs3918241 showed a slight increase in MMP-9 expression in MCF-7 and NIH-3T3 cell lines although not significant. The novel results identified need to be replicated for validation but this study provides evidence that common genetic variants play a role in predisposing to certain tumour types.

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McKinney, P. "An examination of antenatal factors in the aetiology of childhood malignancies." Thesis, University of Leeds, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.378056.

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Cooper, Sheldon Charles. "Studies of the aetiology of oesophageal adenocarcinoma." Thesis, University of Birmingham, 2013. http://etheses.bham.ac.uk//id/eprint/4112/.

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Oesophageal adenocarcinoma (OAC), a cancer with dismal prognosis, has been increasing rapidly in incidence over the last 30 years, nowhere more so than in the UK. Intriguingly, it is a disease predominantly among white males, but there is a paucity of data from England. In performing a range of epidemiological studies, it has been confirmed that OAC has risen five-fold in the West Midlands, UK, five times more common among men, and predominantly a disease among Caucasians. A reduced incidence of OAC was identified among subjects with prostate cancer, suggesting a protective effect of anti-androgen therapy. Examination of a general practice database revealed a negative association with aspirin, non-steroidal anti-inflammatories and statins with OAC, and a positive association with inhaled steroids, increasing number of drugs with a side effect of reducing the lower oesophageal sphincter, and drugs used for asthma/COPD. Finally, a region wide case-control study, confirmed the positive association seen with increasing body mass index, waist circumference, smoking and reflux symptoms, with negative associations seen with a diet high in fruit and vegetables. This work has identified potentially modifiable risk factors that may be employed to reduce the incidence of oesophageal adenocarcinoma, and better help stratify those most likely to benefit from endoscopic surveillance.

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Bergh, Drott Johanna. "The role of microorganisms in prostate cancer development." Doctoral thesis, Umeå universitet, Institutionen för klinisk mikrobiologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-59987.

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Prostate cancer is the most common cancer among Swedish men, but the aetiology of this disease is largely unknown. There is evidence for a linkage between chronic inflammation and prostate cancer. The mechanisms causing prostate inflammation and how this could promote tumour development and progression are however largely unknown. Chronic inflammatory infiltrates are common findings in prostate tissue samples and infection is proposed to be one possible cause for this inflammation. Inflammatory cells release free radicals, cytokines, and growth factors that facilitate increased cell proliferation, DNA damage, mutations, and angiogenesis. However, the present literature on the presence of microbes in prostate tissue and their possible linkage to inflammation and cancer development is limited. Therefore, the aim of this thesis was to investigate if microorganisms are present in prostate tissue and to evaluate their role in inducing prostatitis and prostate epithelial neoplasia. The presence of microorganisms (virus, bacteria and fungi) was studied in clinical prostate tissue samples to evaluate whether or not the occurrences of microorganisms were different in patients that later developed cancer compared with matched controls that did not. Viruses, bacteria and fungi were found in prostate tissues. Out of eight different viruses investigated, EBV and JC virus were detected, but there were no differences in occurrence in the case group compared to the control group. The fungus Candida albicans was present in a very small proportion of the prostate tissue samples. The predominant bacterium was Propionibacterium acnes and the second most prevalent was Escherichia coli. The presence of Propionibacterium acnes was associated with inflammation and subsequent prostate cancer development. Propionibacterium acnes was further evaluated for its capacity to induce an inflammatory response both in vitro and in vivo. Live Propionibacterium acnes induced a strong immune reaction in prostate epithelial cells in vitro with up-regulation of inflammatory genes and secretion of pro-inflammatory cytokines. Infection with Propionibacterium acnes in rat prostate resulted in a lobe specific inflammation with the most intense inflammation in the dorso-lateral prostate, lasting up to 3 months post-inoculation. Propionibacterium acnes inflammation was also associated with altered epithelial cell morphology, signs of DNA damage and increased cell proliferation. Taken together, this thesis shows that different viruses and bacteria can be found in prostate tissue. Propionibacterium acnes, the most abundant among the bacteria detected and more prevalent in the cancer than in the control group, exhibits strong prostatitis promoting properties both in vitro and in vivo. In addition, Propionibacterium acnes can induce some of the epithelial changes known to occur during prostate neoplasia formation. This thesis therefore suggests that Propionibacterium acnes induced chronic prostatitis could promote prostate cancer development. Further studies are needed to elucidate the molecular interplay linking Propionibacterium acnes induced inflammation and the formation of a pre-neoplastic state that could evolve into prostate cancer.

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Jackson, Mark B. "The aetiology of cryptorchidism and its relationship to testicular cancer." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236296.

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Lund, Elizabeth Kay. "The role of iron in the aetiology of colon cancer." Thesis, Anglia Ruskin University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.323076.

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Books on the topic "Cancer aetiology"

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Miller, Anthony B., ed. Diet and the Aetiology of Cancer. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74376-4.

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2

Choi, Alice. The role of occupational factors in brain cancer aetiology: An epidemiological study. Birmingham: University of Birmingham, 1995.

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3

Meanwell, Clive Arthur. Age and cervical cancer: An investigation of incidence, aetiology and prognosis in young women. Birmingham: University of Birmingham, 1987.

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4

MacKie, Rona M. Skin cancer: An illustrated guide to the aetiology, clinical features, pathology and management of benign and malignant cutaneous tumours. London: M. Dunitz, 1989.

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MacKie, Rona M. Skin cancer: An illustrated guide to the aetiology, clinical features, pathology and management of benign and malignant cutaneous tumors. London: Dunitz, 1989.

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Miller, Anthony B. Diet and the Aetiology of Cancer. Springer, 2012.

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B, Miller A., ed. Diet and the aetiology of cancer. Berlin: Springer-Verlag, 1989.

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8

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff. Aetiology and epidemiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0001.

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Describes how cancer care is structured and explains rationale and components of multidisciplinary teams. This runs through cancer care from diagnosis to terminal care and has resulted in better quality and outcomes for patients

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Patel, Nilay, David Cranston, and Mark Sullivan. The aetiology, epidemiology, clinical features, and investigation of kidney cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0083.

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Over 270,000 patients worldwide are diagnosed with renal cancer every year. It is the most lethal of all urological malignancies, with 33–44% of patients dying as a result of the disease. The past three decades has seen the incidence of renal cancer increasing by approximately 2% per year. This increased incidence has predominantly been within localized tumours, detected incidentally due to the increased use of cross-sectional imaging in medical practice. Despite an increase in the number of patients undergoing surgery for renal cancer, mortality rates have continued to rise. There is some evidence to suggest this may be a consequence of the overdiagnosis and overtreatment of small renal masses. At present, there is no justification for national screening programmes for renal cancer.

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Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff. Anal cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0016.

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Outlines the symptoms, epidemiology, aetiology, pathology and metastatic patterns of the disease. Guides to diagnosis, staging and planning therapy. Outlines surgical, radiotherapy and chemotherapy options for both early stage and metastatic disease. Describes the more commonly used chemotherapy regimens and the rationale for choice between them. Illustrates some of the controversial aspects of care.

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Book chapters on the topic "Cancer aetiology"

1

Hansen, Heine H. "Epidemiology and Aetiology." In Lung Cancer, 15–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-76031-0_15.

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Beral, V., and M. Booth. "Occurrence and Aetiology." In Ovarian Cancer, 14–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-69695-4_2.

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Miller, R. W. "Aetiology and Epidemiology." In Cancer in Children, 3–8. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-96889-1_1.

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Terracini, B. "Aetiology and Epidemiology." In Cancer in Children, 3–11. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-84722-6_1.

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Kurukkal, Santhosh Narayana, and Salim Said Al-Busaidy. "Epidemiology and Aetiology." In Urological Cancer Management, 121–24. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16301-7_12.

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Riccabona, Georg. "Aetiology of Thyroid Cancer." In Thyroid Cancer, 39–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71210-4_5.

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Boyle, P., F. E. Alexander, L. Luchini, and T. Bishop. "Aetiology of Prostate Cancer." In Epidemiology of Prostate Disease, 202–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-78826-0_19.

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Hardcastle, Jack Donald. "Epidemiology and Aetiology of Colorectal Cancer." In Colorectal Cancer, 5–10. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78225-1_3.

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Baylis, M. S., W. J. Henderson, C. G. Pierrepoint, and K. Griffiths. "The Aetiology of Ovarian Cancer." In Gynaecological Oncology, 157–65. London: Springer London, 1986. http://dx.doi.org/10.1007/978-1-4471-1389-8_16.

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Alleemudder, Adam, Amlesh Seth, and Vinod H. Nargund. "Renal Cancer – Epidemiology and Aetiology." In Urological Oncology, 345–52. London: Springer London, 2014. http://dx.doi.org/10.1007/978-0-85729-482-1_18.

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Conference papers on the topic "Cancer aetiology"

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Rocha, WM, CF Baez, LA Afonso, FN Carestiato, RB Varella, and SMB Cavalcanti. "P3.236 Study of genital cancer aetiology: association of human papilomavirus (HPV) and merkel cell polyomavyrus (MCPYV)." In STI and HIV World Congress Abstracts, July 9–12 2017, Rio de Janeiro, Brazil. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/sextrans-2017-053264.471.

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Simpson, P., K. Nones, J. Johnson, F. Newell, A.-M. Patch, H. Thorne, S. Kazakoff, et al. "Abstract P5-10-01: Using whole genome sequencing and somatic mutation signatures to unravel insight into familial breast cancer aetiology." In Abstracts: 2018 San Antonio Breast Cancer Symposium; December 4-8, 2018; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p5-10-01.

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Osorio, F., A. Rosendahl Huber, F. Camargo, and R. Van Boxtel. "30 Whole-genome sequencing of normal stem cells provides novel insights into human native hematopoiesis and leukaemia aetiology." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.30.

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Ranasinghe, Jagath, and Mette Jorgensen. "111 Femur fractures in children with cancers. What could be the aetiology?" In GOSH Conference 2020 – Our People, Our Patients, Our Hospital. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2020. http://dx.doi.org/10.1136/archdischild-2020-gosh.111.

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