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Journal articles on the topic 'Calcium'

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Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Vázquez-Calvo, Carmen, Monica Álvarez De Buergo, Rafael Fort, and Asuncion De Los Rios. "Detection of calcium phosphates in calcium oxalate patinas." European Journal of Mineralogy 24, no. 6 (November 16, 2012): 1031–45. http://dx.doi.org/10.1127/0935-1221/2012/0024-2240.

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2

Oliynyk, Mykola, and Anatoliy Shestozub. "Research of calcium oxide hydration in calcium nitrate solutions." Odes’kyi Politechnichnyi Universytet. Pratsi, no. 2 (August 20, 2016): 71–75. http://dx.doi.org/10.15276/opu.2.49.2016.16.

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3

Izer, Alaz, Tugce Nefise Kahyaoglu, and Devrim Balköse. "Calcium Soap Lubricants." Vestnik Volgogradskogo gosudarstvennogo universiteta. Serija 10. Innovatcionnaia deiatel’nost’, no. 1 (March 2014): 16–25. http://dx.doi.org/10.15688/jvolsu10.2014.1.3.

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4

Kuzel, Hans-Jürgen, and Herbert Baier. "Hydration of calcium aluminate cements in the presence of calcium carbonate." European Journal of Mineralogy 8, no. 1 (February 22, 1996): 129–42. http://dx.doi.org/10.1127/ejm/8/1/0129.

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5

Eberhard, Marc, and Paul Erne. "Calcium binding to fluorescent calcium indicators: Calcium green, calcium orange and calcium crimson." Biochemical and Biophysical Research Communications 180, no. 1 (October 1991): 209–15. http://dx.doi.org/10.1016/s0006-291x(05)81278-1.

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6

Sahu, Dr Rita Rani. "Study of Serum Calcium, Microalbuminuria, Urinary Calcium/Creatinine Ratio in Postmenopausal Women." Journal of Medical Science And clinical Research 05, no. 03 (March 26, 2017): 19347–53. http://dx.doi.org/10.18535/jmscr/v5i3.162.

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7

Triggle, David J. "Calcium, calcium channels, and calcium channel antagonists." Canadian Journal of Physiology and Pharmacology 68, no. 11 (November 1, 1990): 1474–81. http://dx.doi.org/10.1139/y90-224.

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Voltage-dependent Ca2+ channels are an important pathway for Ca2+ influx in excitable cells. They also represent an important site of action for a therapeutic group of agents, the Ca2+ channel antagonists. These drugs enjoy considerable use in the cardiovascular area including angina, some arrhythmias, hypertension, and peripheral vascular disorders. The voltage-dependent Ca2+ channels exist in a number of subclasses characterized by electrophysiologic, permeation, and pharmacologic criteria. The Ca2+ channel antagonists, including verapamil, nifedipine, and diltiazem, serve to characterize the L channel class. This channel class has been characterized as a pharmacologic receptor, since it possesses specific drug-binding sites for both antagonists and activators and it is regulated by homologous and heterologous influences. The Ca2+ channels of both voltage- and ligand-regulated classes are likely to continue to be major research targets for new drug design and action.Key words: calcium, calcium channels, calcium antagonists, 1,4-dihydropyridines, channel regulation, receptor regulation.

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8

AL DORAZI, SHOROK ALI, and WASEEM AL TALALWAH. "A SYSTEMATIC REVIEW OF MATERNAL CALCIUM SUPPLEMENTATION AND PAEDIATRIC HEALTH." Fetal and Maternal Medicine Review 25, no. 3-4 (November 2014): 178–96. http://dx.doi.org/10.1017/s0965539515000042.

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Calcium is an important element of body composition1,2 as well as normal physiological functions3. A neonate's body has around 20–30 g calcium present at birth2,4–6 and this amount has to be supplied by the mother since human body cannot synthesise calcium2. Therefore, if the mother has a shortage of calcium, the foetus might be affected.

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9

Wheeler, Glen, Katherine Helliwell, and Colin Brownlee. "Calcium signalling in algae." Perspectives in Phycology 6, no. 1-2 (July 1, 2019): 1–10. http://dx.doi.org/10.1127/pip/2018/0082.

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10

&NA;. "Acenocoumarol/calcium/calcium carbonate." Reactions Weekly &NA;, no. 1343 (March 2011): 5. http://dx.doi.org/10.2165/00128415-201113430-00014.

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11

Wrighton, Katharine H. "Calcium: Mitochondria channel calcium." Nature Reviews Molecular Cell Biology 12, no. 9 (August 3, 2011): 550. http://dx.doi.org/10.1038/nrm3170.

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12

Roderick, H. Llewelyn, Michael J. Berridge, and Martin D. Bootman. "Calcium-induced calcium release." Current Biology 13, no. 11 (May 2003): R425. http://dx.doi.org/10.1016/s0960-9822(03)00358-0.

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13

&NA;. "Alfacalcidol/calcium/calcium lactate." Reactions Weekly &NA;, no. 1420 (September 2012): 7–8. http://dx.doi.org/10.2165/00128415-201214200-00025.

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14

Hanzlik, Robert P., Stephen C. Fowler, and Daniel H. Fisher. "Relative Bioavailability of Calcium from Calcium Formate, Calcium Citrate, and Calcium Carbonate." Journal of Pharmacology and Experimental Therapeutics 313, no. 3 (February 25, 2005): 1217–22. http://dx.doi.org/10.1124/jpet.104.081893.

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15

Ueda, Yukari, and Zenei Taira. "Pharmacokinetic Characterization of Calcium from Three Calcium Salts (Calcium Chloride, Calcium Acetate and Calcium Ascorbate) in Mice." Journal of Hard Tissue Biology 21, no. 3 (2012): 291–98. http://dx.doi.org/10.2485/jhtb.21.291.

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16

Ogasawara, Tomoyasu, Ken-ichi Mori, Teruo Tominaga, Fumitaka Tsukihashi, and Nobuo Sano. "The Activity of Calcium in Calcium-Calcium Halide Fluxes." ISIJ International 36, Suppl (1996): S30—S33. http://dx.doi.org/10.2355/isijinternational.36.suppl_s30.

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17

Naftel, S. J., T. K. Sham, Y. M. Yiu, and B. W. Yates. "CalciumL-edge XANES study of some calcium compounds." Journal of Synchrotron Radiation 8, no. 2 (March 1, 2001): 255–57. http://dx.doi.org/10.1107/s0909049500019555.

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18

Zamay, A. S., O. S. Kolovskaya, N. M. Titova, L. L. Chesnokova, E. A. Maltceva, and T. N. Zamay. "CALCIUM SIGNALING IN TUMOR CELLS." Siberian Medical Review, no. 3 (2016): 27–33. http://dx.doi.org/10.20333/25000136-2016-3-27-33.

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19

Patil, Alka, Nitin Kulkarni, and Rahul V. Patil. "Calcium: Nutrient in Adolescent Girls." Indian Journal of Trauma and Emergency Pediatrics 9, no. 4 (December 15, 2017): 235–40. http://dx.doi.org/10.21088/ijtep.2348.9987.9417.6.

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Bone health is important to health and quality of life. It is important to optimize bone health throughout life cycle. Adolescence is a transitional journey from childhood to adult life along with physical development. This may be considered as physical, psychological and emotional rebirth. Nutrition in adolescence has preventive role related to diet causing chronic diseases like cardiovascular disease, cancer and osteoporosis. Diet and physical factors play a major role in promoting bone health, which begins in childhood and continues into old age. Calcium and protein in the diet with sunlight exposure and weight bearing exercises are important for building the peak bone mass in adolescents. Calcium as nutrient in adolescence is critical for prevention of osteoporosis and improving quality of life.

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20

Cote', Charles J., Lambertus J. Drop, Alfred L. Daniels, and David C. Hoaglin. "Calcium Chloride Versus Calcium Gluconate." Anesthesiology 66, no. 4 (April 1, 1987): 465–70. http://dx.doi.org/10.1097/00000542-198704000-00003.

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21

&NA;. "Calcium, calcium antagonists and BP." Inpharma Weekly &NA;, no. 834 (April 1992): 12. http://dx.doi.org/10.2165/00128413-199208340-00020.

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22

Henry, P. D. "Atherosclerosis, calcium, and calcium antagonists." Circulation 72, no. 3 (September 1985): 456–59. http://dx.doi.org/10.1161/01.cir.72.3.456.

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23

Webb, Joanna K. "CALCIUM EDTA (EDETATE CALCIUM DISODIUM)." Journal of Exotic Pet Medicine 27, no. 3 (July 2018): 54–57. http://dx.doi.org/10.1053/j.jepm.2018.04.001.

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24

Genazzani, Armando A., and Pete Thorn. "Calcium Signalling: Calcium Goes Global." Current Biology 12, no. 12 (June 2002): R432—R433. http://dx.doi.org/10.1016/s0960-9822(02)00918-1.

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25

Nancollas, George H., and Zachary J. Henneman. "Calcium oxalate: calcium phosphate transformations." Urological Research 38, no. 4 (July 13, 2010): 277–80. http://dx.doi.org/10.1007/s00240-010-0292-3.

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26

Nordin, BEC, AG Need, HA Morris, M. Horowitz, TC Durbridge, and DB Cleghorn. "Calcium requirement and calcium therapy." Journal of Bone and Mineral Metabolism 11, no. 2 (December 1993): S3—S8. http://dx.doi.org/10.1007/bf02383534.

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27

Kerkut, G. A. "Calcium and calcium binding proteins." Comparative Biochemistry and Physiology Part A: Physiology 92, no. 1 (January 1989): 152. http://dx.doi.org/10.1016/0300-9629(89)90768-8.

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28

Fullmer, Curtis S. "Intestinal Calcium Absorption: Calcium Entry." Journal of Nutrition 122, suppl_3 (March 1, 1992): 644–50. http://dx.doi.org/10.1093/jn/122.suppl_3.644.

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29

CHARLES, P. "Calcium absorption and calcium bioavailability." Journal of Internal Medicine 231, no. 2 (February 1992): 161–68. http://dx.doi.org/10.1111/j.1365-2796.1992.tb00519.x.

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30

Henry, Philip D. "Atherogenesis, calcium and calcium antagonists." American Journal of Cardiology 66, no. 21 (December 1990): 3–6. http://dx.doi.org/10.1016/0002-9149(90)91256-6.

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31

Jaffe, Lionel F. "Stretch-activated calcium channels relay fast calcium waves propagated by calcium-induced calcium influx." Biology of the Cell 99, no. 3 (March 2007): 175–84. http://dx.doi.org/10.1042/bc20060031.

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32

Weinreich, Thomas, A. Colombi, H. H. Echterhoff, G. Mielke, M. Nebel, C. Ziegelmayer, and J. Passlick-Deetjen. "Transperitoneal Calcium Mass Transfer using Dialv Sate with a Low Calcium Concentration (1.0 mM)." Peritoneal Dialysis International: Journal of the International Society for Peritoneal Dialysis 13, no. 2_suppl (January 1993): 467–70. http://dx.doi.org/10.1177/089686089301302s117.

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Lower dialysate calcium concentrations were recently proposed to overcome the risk of hypercalcemia In continuous ambulatory peritoneal dialysis (CAPD) patients on calcium containing phosphate binders and/or vitamin D metabolites using the standard dialysate calcium concentration (sCa) of 1.75 mM. To assess transperitoneal calcium mass transfer (CaMT) in CAPD patients using a dialysate with a low calcium concentration (LCa, 1.00 mM), 18 stable patients were randomly allocated to receive either LCa or SCa. CaMT was assessed over 4 hours using 2L dialysate bags with three different dialysate glucose concentrations (1.5%, 2.3%, 4.25%). Total serum calcium (tCa), Ionized calcium (iCa), and the exact dialysate volume were measured before and after the 4-hour dwell. A sample of the drained dialysate was obtained to measure the dialysate calcium concentration. The tCa and iCa levels were not significantly different In both groups prior to and did not change throughout the test. CaMT (median/range) was .0.64 mmol/exchange (0.35 –1.29 mmol/exchange) using LCa with 1.5% glucose compared to 0.23 mmol (.0.18 -0.87 mmol) with SCa (p<0.0001). CaMT was negatively correlated to ICa and ultrafiltration volume [4.25%: LCa -1.22 (.0.84 -1.9); SCa .0.43 (-1.35 -0.13); p<0.001]. In summary, LCa results in a loss of calcium into the dialysate even at low ultrafiltration volumes and serum ICa levels. This might facilitate the prevention and therapy of renal osteodystrophy with calclum-containing phosphate binders and calcitriol. However, patients using LCa must be carefully monitored for calcium homeostasis and bone turnover.

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33

Hämäläinen, Mauri M. "Bone Repair in Calcium-Deficient Rats: Comparison of Xylitol + Calcium Carbonate with Calcium Carbonate, Calcium Lactate and Calcium Citrate on the Repletion of Calcium." Journal of Nutrition 124, no. 6 (June 1, 1994): 874–81. http://dx.doi.org/10.1093/jn/124.6.874.

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34

DeHaven, Wayne I., Jeremy T. Smyth, Rebecca R. Boyles, and James W. Putney. "Calcium Inhibition and Calcium Potentiation of Orai1, Orai2, and Orai3 Calcium Release-activated Calcium Channels." Journal of Biological Chemistry 282, no. 24 (April 23, 2007): 17548–56. http://dx.doi.org/10.1074/jbc.m611374200.

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35

S, Ramani, Vijaya B, and Nithya B. "Efficacy of calcium carbonate in reducing the incidence of pre-eclampsia vis-a-vis calcium lactate." New Indian Journal of OBGYN 8, no. 2 (February 2022): 278–84. http://dx.doi.org/10.21276/obgyn.2022.8.2.23.

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36

Saleh Balubaid, Waleed, Mohammed Fouad Garanbish, Bayader Baher Alawi Kurdi, Roaa Mohammed Altumaihi, Noura Nabil Naamani, Rakan Ibrahim Qutob, Bashayer Mansour Bukhari, Faisal A. Halawani, Abeer Abdullatif Alomarey, and Wiam Talal Mohammad Sagr. "Calcium Ions Released from Light - Cured Calcium Hydroxide Cement using Argon - Based Induction Coupled Mass Spectroscopy." International Journal of Science and Research (IJSR) 12, no. 9 (September 5, 2023): 41–44. http://dx.doi.org/10.21275/sr23829180533.

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37

&NA;. "Calcium." Reactions Weekly &NA;, no. 1384 (January 2012): 15–16. http://dx.doi.org/10.2165/00128415-201213840-00054.

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38

Weaver, Connie M., and Munro Peacock. "Calcium." Advances in Nutrition 2, no. 3 (April 30, 2011): 290–92. http://dx.doi.org/10.3945/an.111.000463.

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39

Haworth, David, and J. Wark. "Calcium." Australian Prescriber 27, no. 4 (August 1, 2004): 82–83. http://dx.doi.org/10.18773/austprescr.2004.068.

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40

&NA;. "Calcium." Reactions Weekly &NA;, no. 807 (June 2000): 7. http://dx.doi.org/10.2165/00128415-200008070-00015.

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41

&NA;. "Calcium." Reactions Weekly &NA;, no. 1199 (April 2008): 15. http://dx.doi.org/10.2165/00128415-200811990-00038.

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42

ISHAQ, MUHAMMAD, ISRAR AHMED AKHUND, MOULA BUX LAGHARI, and Muhammad Sabir. "CALCIUM." Professional Medical Journal 17, no. 04 (December 10, 2010): 698–701. http://dx.doi.org/10.29309/tpmj/2010.17.04.3027.

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Aims & Objectives: To evaluate the effects of Serum Calcium and Urinary Calcium excretion on upper urinary tract stone diseases in the Peshawar (a high stone incidence belt). Subjects & Methods: One hundred patients (age 20-60years) who were suffering severely from upper urinary tract stone disease were selected from LRH and Hayatabad Medical Complex Hospitals of Peshawar, same numbers of healthy controls from the same region were also selected for the study. Results: When results were summed up and testParameters were compared, it was seen that mean Serum Calcium in stone formers was greater than that of non-stone formers (P<0.001). Same pattern was also observed (P< 0.001) in both groups regarding mean urinary calcium excretion. Conclusions: We concluded that calcium is a definitive risk factor in upper urinary tract stone disease. However we suggest further work and research on wide scale population inorder to evaluate this relation.

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43

DeCristofaro, Joseph D., and Reginald C. Tsang. "Calcium." Emergency Medicine Clinics of North America 4, no. 2 (May 1986): 207–21. http://dx.doi.org/10.1016/s0733-8627(20)30996-2.

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44

&NA;. "Calcium." Reactions Weekly &NA;, no. 1308 (July 2010): 12. http://dx.doi.org/10.2165/00128415-201013080-00027.

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45

Eich, Torsten, Magnus Ståhle, Bengt Gustafsson, Rune Horneland, Marko Lempinen, Torbjörn Lundgren, Ehab Rafael, et al. "Calcium." Cell Transplantation 27, no. 7 (June 26, 2018): 1031–38. http://dx.doi.org/10.1177/0963689718779350.

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Background: Effective digestive enzymes are crucial for successful islet isolation. Supplemental proteases are essential because they synergize with collagenase for effective pancreatic digestion. The activity of these enzymes is critically dependent on the presence of Ca2+ ions at a concentration of 5–10 mM. The present study aimed to determine the Ca2+ concentration during human islet isolation and to ascertain whether the addition of supplementary Ca2+ is required to maintain an optimal Ca2+ concentration during the various phases of the islet isolation process. Methods: Human islets were isolated according to standard methods and isolation parameters. Islet quality control and the number of isolations fulfilling standard transplantation criteria were evaluated. Ca2+ was determined by using standard clinical chemistry routines. Islet isolation was performed with or without addition of supplementary Ca2+ to reach a Ca2+ of 5 mM. Results: Ca2+ concentration was markedly reduced in bicarbonate-based buffers, especially if additional bicarbonate was used to adjust the pH as recommended by the Clinical Islet Transplantation Consortium. A major reduction in Ca2+ concentration was also observed during pancreatic enzyme perfusion, digestion, and harvest. Additional Ca2+ supplementation of media used for dissolving the enzymes and during digestion, perfusion, and harvest was necessary in order to obtain the concentration recommended for optimal enzyme activity and efficient liberation of a large number of islets from the human pancreas. Conclusions: Ca2+ is to a large extent consumed during clinical islet isolation, and in the absence of supplementation, the concentration fell below that recommended for optimal enzyme activity. Ca2+ supplementation of the media used during human pancreas digestion is necessary to maintain the concentration recommended for optimal enzyme activity. Addition of Ca2+ to the enzyme blend has been implemented in the standard isolation protocols in the Nordic Network for Clinical Islet Transplantation.

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46

Angood, Peter B., Loren D. Nelson, Mary Murtha, and Bernard steele. "CALCIUM." Critical Care Medicine 15, no. 4 (April 1987): 430. http://dx.doi.org/10.1097/00003246-198704000-00182.

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47

Chernow, Bart. "Calcium." Critical Care Medicine 18, no. 8 (August 1990): 895. http://dx.doi.org/10.1097/00003246-199008000-00024.

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48

Vetanovetz, R. P., and J. C. Peterson. "Calcium." Journal of Plant Nutrition 10, no. 9 (June 1987): 1889–95. http://dx.doi.org/10.1080/01904168709363732.

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49

Cline, R. A. "Calcium." Journal of Plant Nutrition 10, no. 9 (June 1987): 1897–905. http://dx.doi.org/10.1080/01904168709363733.

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50

Anderson, C. A. "Calcium." Journal of Plant Nutrition 10, no. 9 (June 1987): 1907–16. http://dx.doi.org/10.1080/01904168709363734.

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