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Books on the topic 'Calcium channel drugs'

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Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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1

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. Boston: Kluwer Academic Publishers, 1989.

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2

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. 2nd ed. London: Kluwer, 1990.

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3

Opie, Lionel H., and William A. Coetzee. Clinical Use of Calcium Channel Antagonist Drugs. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8.

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4

Japan-U.S.A. Symposium on Cardiovascular Drugs (1989 Kahuku, Hawaii). Recent advances in calcium channels and calcium antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Edited by Yamada Kazuo and Shibata Shoji. New York: Pergamon Press, 1990.

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5

Drugs looking for diseases: Innovative drug research and the development of the beta blockers and the calcium antagonists. Dordrecht: Kluwer Academic Publishers, 1991.

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6

Albrecht, Fleckenstein, ed. Cardiovascular effects of dihydropyridine-type calcium antagonists and agonists. Berlin: Springer-Verlag, 1985.

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7

Godfraind, T. Calcium channel blockers. Boston, MA: Birkhauser Verlag, 2003.

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8

Calcium channel blockers. Basel: Birkhäuser Verlag, 2004.

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9

S, Meldrum Brian, Williams, Michael, 1947 Jan. 3-, and Princeton Drug Research Symposia (1st : 1989 : Princeton, N.J.), eds. Current and future trends in anticonvulsant, anxiety, and stroke therapy: Proceedings of a symposium held at Princeton, New Jersey, May 21-23, 1989. New York: Wiley-Liss, 1990.

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10

Roberto, Robles Nicolás, ed. Calcium channel blockers and renal disease. Hauppauge, NY: Nova Science Publishers, 2009.

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11

Sklar, Grant. A retrospective drug utilization review of the calcium channel blockers. [Ottawa]: Ottawa Civic Hospital, 1989.

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12

Kazuhide, Inoue, Watanabe Y, and Nihon Shinkei Seishin Yakuri Gakkai. Congress, eds. Calcium ion modulators: The new wave of psychotropic drugs. Australia: Harwood Academic Publishers, 1998.

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13

Howl, John. Calcium-mobilizing drugs and pathological changes in Mammalian muscle. Birmingham: University of Birmingham, 1988.

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14

Campbell, Anthony K. Intracellular calcium. Chichester, West Sussex: John Wiley & Sons, Ltd, 2014.

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15

M, Vanhoutte Paul, Paoletti Rodolfo, Govoni Stefano 1950-, Fondazione Giovanni Lorenzini, and New York Academy of Sciences., eds. Calcium antagonists: Pharmacology and clinical research. New York, N.Y: New York Academy of Sciences, 1988.

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16

S, Abraham, and Amitai G, eds. Calcium channel modulators in heart and smooth muscle: Basic mechanisms and pharmacological aspects : proceedings of the 33rd [i.e. 34th] Oholo Conference, Eilat, Israel, 1989. Rehovot, Israel: Balaban Publishers, 1990.

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17

L, Tranquilli A., ed. Calcium antagonists in the treatment of hypertension in pregnancy. Pearl River, NY: Parthenon Pub. Group, 1999.

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18

D, Nathan Richard, ed. Cardiac muscle: The regulation of excitation and contraction. Orlando: Academic Press, 1986.

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19

McDonough, Stefan I. Calcium Channel Pharmacology. Springer, 2012.

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20

McDonough, Stefan I. Calcium Channel Pharmacology. Springer London, Limited, 2011.

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21

I, McDonough Stefan, ed. Calcium channel pharmacology. New York: Kluwer Academic/Plenum Publishers, 2004.

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22

H, Opie Lionel. Clinical Use of Calcium Channel Antagonist Drugs. Springer, 2011.

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23

Clinical Use of Calcium Channel Antagonist Drugs. Springer, 2013.

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24

H, Opie Lionel. Clinical Use of Calcium Channel Antagonist Drugs. Springer London, Limited, 2012.

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25

Isbister, Geoffrey, and Colin Page. Management of β‎-blocker and calcium channel blocker poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0325.

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β‎-blocker and calcium channel-blockers can cause life-threatening toxicity due to cardiogenic shock. Both β‎-blockers and calcium channel-blockers are heterogenous groups of drugs and particular drugs, such as propranolol, diltiazem, and verapamil are far more toxic than the others in their class. The most important investigations in β‎-blocker and calcium channel-blocker overdose are an electrocardiogram, blood glucose measurement, and electrolytes. Like most overdoses, supportive treatment is the most important, with emphasis on the primary pathophysiology. Early decontamination should be considered based on the severity of the poisoning. Treatment of β‎-blockers and calcium channel-blockers poisoning, using absolute blood pressure as an endpoint can be misleading and measuring cardiac output can be more informative in gauging response to treatment. There are no specific antidotes, although β‎-agonists may be effective in β‎-blocker overdose and calcium has been shown to be effective in calcium channel-blocker overdose. The choice of inotropes and/or vasopressors will differ for β‎-blockers and calcium channel-blockers. These include isoprenaline, high dose insulin euglycaemia, phosphodiesterase inhibitors, and other catecholaminergic inotropes for β‎-blocker poisoning and adrenaline, high dose insulin euglycaemia and vasopressors for calcium channel-blocker poisoning.
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26

van, Zwieten P. A., ed. Clinical aspects of calcium entry blockers. Basel: Karger, 1989.

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27

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0019.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first line medication for patients with isolated systolic hypertension, black people, and during pregnancy, in the presence of intermittent claudication, asymptomatic atherosclerosis, or metabolic syndrome. A polypill is a combination pill in which multiple medications effective in the prevention of cardiovascular disease (for example statins, antihypertensives, and aspirin) are put together in a single pill.
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28

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0019_update_001.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first line medication for patients with isolated systolic hypertension, black people, and during pregnancy, in the presence of intermittent claudication, asymptomatic atherosclerosis, or metabolic syndrome. A polypill is a combination pill in which multiple medications effective in the prevention of cardiovascular disease (for example statins, antihypertensives, and aspirin) are put together in a single pill.
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29

Kurachi, Y., M. Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer London, Limited, 2012.

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30

Kurachi, Y., Masayoshi Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer Berlin / Heidelberg, 2012.

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31

Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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32

Hoffmeister, F., A. Fleckenstein, C. Van Breemen, and R. Groß. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer London, Limited, 2013.

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33

Hoffmeister, F., A. Fleckenstein, C. Van Breemen, and R. Groß. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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34

(Editor), M. Endo, Y. Kurachi (Editor), and M. Mishina (Editor), eds. Pharmacology of Ionic Channel Function: Activators and Inhibitors (Handbook of Experimental Pharmacology). Springer, 2000.

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35

Fleckenstein, A., C. Van Breemen, and R. Grob. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists (Bayer-Symposium//(Proceedings)). Springer, 1985.

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36

M, Bers D., and Baker Peter F. 1939-, eds. Calcium in drug actions. Berlin: Springer-Verlag, 1988.

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37

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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38

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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39

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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40

1933-, Aoki K., and Frohlich Edward D. 1931-, eds. Calcium in essential hypertension. Tokyo: Academic Press, 1989.

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41

Kazuo, Yamada. Recent Advances in Calcium Channels and Calcium Antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Pergamon, 1989.

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42

Kazuo, Yamada. Recent Advances in Calcium Channels and Calcium Antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Pergamon, 1989.

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43

Suri, Ajay, and Jean R. McEwan. Anti-anginal agents in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0037.

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Angina is chest pain resulting from the lack of blood supply to heart muscle most commonly due to obstructive atherosclerotic. Intensive care unit patients are subject to various stresses that will increase the demand on the heart and are in a pro-thrombotic state. Patients in an intensive treatment unit may be sedated and so cardiac ischaemia may be detected by electrocardiogram, haemodynamic monitoring, and echocardiographic imaging of function. These signs may indicate critical coronary perfusion heralding a myocardial infarction and are alleviated by anti-anginal drugs. Beta-blockers and calcium channel blockers are the usual first-line treatments for angina, but may not be ideal in the critically-ill patient. Nitrates reduce blood pressure without typically affecting heart rate. Nicorandil is a similar mechanism of action and tends to be given orally, while ivabridine, an If channel blocker, is a newer anti-anginal, which acts by reducing heart rate, while not affecting blood pressure. Ranolazine is the one of the newest anti-anginal agents and is believed to alter the transcellular late sodium current thereby decreasing sodium entry into ischaemic myocardial cells.
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44

1937-, Epstein Murray, ed. Calcium antagonists in clinical medicine. 2nd ed. Philadelphia: Hanley & Belfus, 1998.

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45

Cavanna, Andrea E. Antiepileptic drugs and behaviour: mechanisms of action. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0002.

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Antiepileptic drugs (AEDs) exert their pharmacological properties on both epileptic seizures and behaviour through different mechanisms of action. These include modulation of ion (mainly sodium and calcium) conductance through voltage-gated channels located within the neuronal membrane, as well as facilitation of inhibitory (GABAergic) neurotransmission and inhibition of excitatory (glutamatergic) neurotransmission, resulting in regulation of neuronal excitability.
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46

Godfraind, T. Pharmacological Control of Calcium and Potassium Homeostasis. Springer, 2012.

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47

Turney, Ben, and John Reynard. Prevention of idiopathic calcium stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0015.

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The main principles of idiopathic calcium oxalate stone prevention are to maintain dilute urine through increasing fluid intake and to reduce calcium and oxalate excretion. The influence of various urinary factors on the risk of stone formation has been quantified mathematically. Urine volume and urinary oxalate concentration are most influential on the risk of stone formation, while magnesium concentration contributes a small amount to risk. It is estimated that around 50% of stone formers will form another stone within five years. Some stone formers have frequent recurrences. Most stone formers ask how they can prevent future episodes. Advice can be generic or personalized, and treatment may include changes to diet, fluid intake, and addition of drugs to alter urine biochemistry.
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48

Gauci, Danielle Frances *. Drug transport by multidrug resistant cells and drug binding to P-glycoprotein: effects of calcium channel blockers. 1990.

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49

Subramanian, V. Bala. Calcium Antagonists in Chronic Stable Angina Pectoris: Current Status (Current Clinical Practice Series, 39). Excerpta Medica, 1986.

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50

Campbell, Anthony K. Intracellular Calcium: Universal Switch in Life and Death. Wiley & Sons, Limited, John, 2014.

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