Relevant bibliographies by topics / Burkholderia pseudomallei

Academic literature on the topic 'Burkholderia pseudomallei'

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Published: 4 June 2021
Last updated: 21 September 2024

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Journal articles on the topic "Burkholderia pseudomallei"

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Duangurai, Taksaon, Nitaya Indrawattana, and Pornpan Pumirat. "Burkholderia pseudomalleiAdaptation for Survival in Stressful Conditions." BioMed Research International 2018 (May 27, 2018): 1–11. http://dx.doi.org/10.1155/2018/3039106.

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Burkholderia pseudomalleiis a Gram-negative bacterium that causes melioidosis, which can be fatal in humans. Melioidosis is prevalent in the tropical regions of Southeast Asia and Northern Australia. Ecological data have shown that this bacterium can survive as a free-living organism in environmental niches, such as soil and water, as well as a parasite living in host organisms, such as ameba, plants, fungi, and animals. This review provides an overview of the survival and adaptation ofB. pseudomalleito stressful conditions induced by hostile environmental factors, such as salinity, oxidation, and iron levels. The adaptation ofB. pseudomalleiin host cells is also reviewed. The adaptive survival mechanisms of this pathogen mainly involve modulation of gene and protein expression, which could cause alterations in the bacteria’s cell membrane, metabolism, and virulence. Understanding the adaptations of this organism to environmental factors provides important insights into the survival and pathogenesis ofB. pseudomallei, which may lead to the development of novel strategies for the control, prevention, and treatment of melioidosis in the future.
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Williams, Natasha L., Jodie L. Morris, Catherine M. Rush, and Natkunam Ketheesan. "Migration of Dendritic Cells Facilitates Systemic Dissemination of Burkholderia pseudomallei." Infection and Immunity 82, no. 10 (July 28, 2014): 4233–40. http://dx.doi.org/10.1128/iai.01880-14.

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ABSTRACTBurkholderia pseudomallei, the etiological agent for melioidosis, is an important cause of community-acquired sepsis in northern Australia and northeast Thailand. Due to the rapid dissemination of disease in acute melioidosis, we hypothesized that dendritic cells (DC) could act as a vehicle for dissemination ofB. pseudomallei. Therefore, this study investigated the effect ofB. pseudomalleiinfection on DC migration capacity and whether migration of DC enabled transportation ofB. pseudomalleifrom the site of infection.B. pseudomalleistimulated significantly increased migration of bone marrow-derived DC (BMDC), bothin vitroandin vivo, compared to uninfected BMDC. Furthermore, migration of BMDC enabled significantly increasedin vitrotrafficking ofB. pseudomalleiandin vivodissemination ofB. pseudomalleito secondary lymphoid organs and lungs of C57BL/6 mice. DC within the footpad infection site of C57BL/6 mice also internalizedB. pseudomalleiand facilitated dissemination. Although DC have previously been shown to kill intracellularB. pseudomalleiin vitro, the findings of this study demonstrate thatB. pseudomallei-infected DC facilitate the systemic spread of this pathogen.
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Inglis, Timothy J. J., Dorothee R. Hahne, Adam J. Merritt, and Michael W. Clarke. "Volatile-Sulfur-Compound Profile Distinguishes Burkholderia pseudomallei from Burkholderia thailandensis." Journal of Clinical Microbiology 53, no. 3 (January 7, 2015): 1009–11. http://dx.doi.org/10.1128/jcm.03644-14.

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Solid-phase microextraction gas chromatography-mass spectrometry (SPME-GCMS) was used to show that dimethyl sulfide produced byBurkholderia pseudomalleiis responsible for its unusual truffle-like smell and distinguishes the species fromBurkholderia thailandensis. SPME-GCMS can be safely used to detect dimethyl sulfide produced by agar-grownB. pseudomallei.
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Martin de Frémont, Grégoire, Marie Gominet, Aurore Bousquet, Alban Gervaise, Dinaherisoa Andriamanantena, and Cécile Ficko. "Burkholderia pseudomallei." AIDS 33, no. 8 (July 2019): 1403–4. http://dx.doi.org/10.1097/qad.0000000000002205.

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BRETT, P. J., D. DESHAZER, and D. E. WOODS. "Characterization of Burkholderia pseudomallei and Burkholderia pseudomallei-like strains." Epidemiology and Infection 118, no. 2 (April 1997): 137–48. http://dx.doi.org/10.1017/s095026889600739x.

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Previous reports in the literature suggest that Burkholderia pseudomallei strains can be differentiated on the basis of animal virulence. Twenty environmentally and clinically derived isolates of Burkholderia pseudomallei were examined for the production of exoenzymes, morphological and biochemical phenotypes and virulence for Syrian golden hamsters. The partial sequence of the 16S ribosomal RNA [rRNA] genes from a number of these strains was also determined. Based upon these observations, it is suggested that highly virulent Burkholderia pseudomallei strains are true Burkholderia pseudomallei strains. The DNA sequences of the 16S rRNA genes of the true Burkholderia pseudomallei strains were identical to the published sequences for Burkholderia pseudomallei while differences were revealed between the published sequences and those of the lowly virulent strains. Thus, these latter strains have been designated as Burkholderia pseudomallei-like organisms since they demonstrate significant differences in exoenzyme production, hamster virulence and 16S rRNA gene sequences.
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Rao, P. Sugandhi, Reetika Dhawan, and P. G. Shivananda. "Burkholderia Pseudomallei Infections." Tropical Doctor 32, no. 3 (July 2002): 174–75. http://dx.doi.org/10.1177/004947550203200321.

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Dance, D. A. B. "Burkholderia pseudomallei Infections." Clinical Infectious Diseases 30, no. 1 (January 1, 2000): 235–36. http://dx.doi.org/10.1086/313577.

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Chen, K. J., M. H. Sun, C. H. Hou, C. C. Sun, and T. L. Chen. "Burkholderia pseudomallei Endophthalmitis." Journal of Clinical Microbiology 45, no. 12 (October 3, 2007): 4073–74. http://dx.doi.org/10.1128/jcm.01467-07.

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Wang, Guanbo, Paulina Zarodkiewicz, and Miguel A. Valvano. "Current Advances in Burkholderia Vaccines Development." Cells 9, no. 12 (December 11, 2020): 2671. http://dx.doi.org/10.3390/cells9122671.

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The genus Burkholderia includes a wide range of Gram-negative bacterial species some of which are pathogenic to humans and other vertebrates. The most pathogenic species are Burkholderia mallei, Burkholderia pseudomallei, and the members of the Burkholderia cepacia complex (Bcc). B. mallei and B. pseudomallei, the cause of glanders and melioidosis, respectively, are considered potential bioweapons. The Bcc comprises a subset of Burkholderia species associated with respiratory infections in people with chronic granulomatous disease and cystic fibrosis. Antimicrobial treatment of Burkholderia infections is difficult due to the intrinsic multidrug antibiotic resistance of these bacteria; prophylactic vaccines provide an attractive alternative to counteract these infections. Although commercial vaccines against Burkholderia infections are still unavailable, substantial progress has been made over recent years in the development of vaccines against B. pseudomallei and B. mallei. This review critically discusses the current advances in vaccine development against B. mallei, B. pseudomallei, and the Bcc.
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Novem, Vidhya, Guanghou Shui, Dongling Wang, Anne K. Bendt, Siew Hoon Sim, Yichun Liu, Tuck Weng Thong, et al. "Structural and Biological Diversity of Lipopolysaccharides from Burkholderia pseudomallei and Burkholderia thailandensis." Clinical and Vaccine Immunology 16, no. 10 (August 19, 2009): 1420–28. http://dx.doi.org/10.1128/cvi.00472-08.

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ABSTRACT Burkholderia pseudomallei, the etiological agent of melioidosis, is a facultative intracellular pathogen. As B. pseudomallei is a gram-negative bacterium, its outer membrane contains lipopolysaccharide (LPS) molecules, which have been shown to have low-level immunological activities in vitro. In this study, the biological activities of B. pseudomallei LPS were compared to those of Burkholderia thailandensis LPS, and it was found that both murine and human macrophages produced levels of tumor necrosis factor alpha, interleukin-6 (IL-6), and IL-10 in response to B. pseudomallei LPS that were lower than those in response to B. thailandensis LPS in vitro. In order to elucidate the molecular mechanisms underlying the low-level immunological activities of B. pseudomallei LPS, its lipid A moiety was characterized using mass spectrometry. The major lipid A species identified in B. pseudomallei consists of a biphosphorylated disaccharide backbone, which is modified with 4-amino-4-deoxy-arabinose (Ara4N) at both phosphates and penta-acylated with fatty acids (FA) C14:0(3-OH), C16:0(3-OH), and either C14:0 or C14:0(2-OH). In contrast, the major lipid A species identified in B. thailandensis was a mixture of tetra- and penta-acylated structures with differing amounts of Ara4N and FA C14:0(3-OH). Lipid A species acylated with FA C14:0(2-OH) were unique to B. pseudomallei and not found in B. thailandensis. Our data thus indicate that B. pseudomallei synthesizes lipid A species with long-chain FA C14:0(2-OH) and Ara4N-modified phosphate groups, allowing it to evade innate immune recognition.
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Dissertations / Theses on the topic "Burkholderia pseudomallei"

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Jones, Amanda L. "Burkholderia pseudomallei, host interactions." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq20744.pdf.

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Lowe, Carolyn Ann. "Iron regulation in Burkholderia cepacia and Burkholderia pseudomallei." Thesis, University of Sheffield, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246990.

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Ellis, Jill Frances. "Antibody detection of Burkholderia pseudomallei and Burkholderia mallei." Thesis, Aston University, 2000. http://publications.aston.ac.uk/10974/.

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Monoclonal and polyclonaI antibodies have been produced for use in immunological assays for the detection of Burkholderia pseudomallei and Burkholderia mallei. Monoclonal antibodies recognising a high molecular weight polysaccharide material found in some strains of both species have been shown to be effective in recognising B. pseudomallei and B. mallei and distinguishing them from other organisms. The high molecular weight polysaccharide material is thought to be the capsule of B. pseudomallei and B. mallei and may have important links with virulence. B. pseudomallei and B. mallei are known to be closely related, sharing many epitopes, but antigenic variation has been demonstrated within both the species. The lipopolysaccharide from strains of B. pseudomal/ei and B. mallei has been isolated and the silver stain profiles found to be visually very similar. A monoclonal antibody raised to B. mallei LPS has been found to recognise both B. mallei and B. pseudomallei strains. However, in a small number of B. pseudomallei strains a visually atypical LPS profile has been demonstrated. A monoclonal ant ibody rai sed against this atypical LPS showed no recognition of the typical LPS profile of either B. mallei or B. pseudomallei. This atypical LPS structure has not been reported and may be immunologically distinct from the typical LPS. Molecular biology and antibody engineering techniques have been used in an attempt to produce single-chain antibody fragments reactive to B. pseudomallei. Sequencing of one of the single-chain antibody fragments produced showed high homology with murine immunoglobulin genes, but none of the single-chain antibody fragments were found to be specific to B. pselldomallei.
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Senkiw, Michelle D. "Intracellular existence of Burkholderia pseudomallei." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0016/MQ49655.pdf.

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Atkins, Timothy Philip. "Virulence determinants of Burkholderia pseudomallei." Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.325608.

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張健文 and Kin-man Cheung. "B-lactamases in Burkholderia pseudomallei." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31225792.

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Davies, Clare. "Molecular characterisation of Burkholderia pseudomallei." Thesis, University of Plymouth, 2001. http://hdl.handle.net/10026.1/2281.

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A programme of research was carried out to attempt the molecular characterisation of the human and animal pathogen, Burkholderia pseudomallei, the causative agent of melioidosis and the newly described avirulent species, B.thailandensis for comparative purposes. Melioidosis is still little understood, and so the clinical approach to the prevention and control of melioidosis must ultimately rest upon the basic understanding of the causative organism, particularly the pathogenic properties of B.pseudomallei. A range of B.pseudomallei and B.thailandensis isolates were cultured and the extracellular products were isolated and concentrated and an initial study conducted to identify potential target molecules for cloning. Those isolates tested were shown to have somewhat differing ECP profiles when analysed with SDS-PAGE and antigenic profiles when subject to irnmunoblotting using convalescent human serum although isolates within and between species shared a number of common bands. The ECPs were also tested for a range of activities and it was established that both species had proteolytic and phospholipase activities neither had a haemolytic activity and only isolates of B.pseudomallei had a hexosaminidase activity a putative pathogenicity determinant. Genomic DNA of B.pseudomallei was used to construct genomic libraries in a range of E. coli host vector systems. A λGTII genomic library was screened with antisera for the presence of B.pseudomallei antigens and a number of natural and synthetic substrates for the presence of haemolytic and proteolytic components. Screening yielded one stable immunopositive clone with a novel positive reaction in the form of a "halo" of reaction around the plaque. The 5 kbp cloned fragment was subcloned into a plasmid vector, and the resulting recombinant molecule, pBPGT2 was DNA sequenced and found to contain a putative pilin gene. Attempts were made to determine the size of the recombinant antigen and to further express the pilin gene product all of which were unsuccessful. A southern blot procedure confirmed the fidelity of the cloning procedure proving that the fragment was from the host organism, B.pseudomallei. A further southern blot procedure tested for the presence of the pilin sequence in a range of B.pseudomallei and B.thailandensis isolates proving the presence of the gene in only isolates of B.pseudomallei. PCR primers were designed to amplify the DNA encoding the active site of the ADP-ribosylating toxin (ET A) of Pseudomonas aeruginosa and a PCR reaction was carried out on a number of B.pseudomallei and B.thailandensis isolates. The reaction yielded a 500 bp product in only B.pseudomallei isolates and DNA sequencing of the product revealed no obvious homology to ETA of P.aeruginosa but was used as a probe to isolate a larger fragment of DNA which was found to encode a number of interesting putative genes. These included one with homology to a porin similar to that of the pathogen Neisseria gonorrhoea, with a role in virulence. During attempts to digest the genomic DNA of B.pseudomallei isolate 4845 with the restriction enzyme Sau3A two 12 kbp bands of DNA were resistant to the endonuclease activity. Attempts were made to clone these bands into a range of plasmid vectors with two clones containing deleted products. DNA sequencing proved inadequate with only a small amount of sequence information obtained. However, towards the final stages of the research project sequence information from the B.pseudomallei genome sequencing project facilitated the recognition of a 38 kbp fragment containing the sequence information from one of the clones, which encodes an alkaline protease and a putative haemagglutinin and is postulated to be a Pathogenicity Island encoding secreted virulence factors. The sequencing project also facilitated the isolation of two putative hexosaminidase genes postulated to be responsible for the activities observed when testing the B.pseudomallei isolates concentrated ECPs. Future studies for the putative genes identified and other components of B.pseudomallei are discussed.
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Cheung, Kin-man. "B-lactamases in Burkholderia pseudomallei /." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25205158.

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梁嘉玲 and Ka-ling Leung. "Novel molecular targets of Burkholderia pseudomallei." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2001. http://hub.hku.hk/bib/B31224726.

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Leung, Ka-ling. "Novel molecular targets of Burkholderia pseudomallei /." Hong Kong : University of Hong Kong, 2001. http://sunzi.lib.hku.hk/hkuto/record.jsp?B23440144.

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Books on the topic "Burkholderia pseudomallei"

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Burkholderia Pseudomallei dan GSK3:: Antara Pantogen dan Enzim Pengisyaratan. Bangi, Selangor, Malaysia: Penerbit Universiti Kebangsaan Malaysia, 2013.

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Ramachandran, Raja, and Vivekanand Jha. Renal involvement in other infections. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0198_update_001.

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Renal involvement has been described in patients with many other infections and this chapter discusses several of these.Water-borne infections are a common cause of acute kidney injury (AKI) worldwide but especially in tropical regions. Cholera is notoriously dangerous but any other cause of fluid-depletion may achieve the same. Typhoid fever is more likely to cause AKI from its complications than directly, but a small proportion of patients have glomerulonephritis.Meliodosis is caused by the intracellular organism Burkholderia pseudomallei. It typically affects workers in paddy (rice) fields in the rainy season, and may cause a local, genitourinary infection or an acute melioidosis septicaemia with a high incidence of AKI and mortality. Those with other chronic conditions are at greatest risk.Obstetric infections as a result of unsafe deliberate abortion or post-partum are a very common (often the most common) cause of AKI in developing countries, and a major cause of avoidable death in young women.
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Book chapters on the topic "Burkholderia pseudomallei"

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Pflughoeft, Kathryn J., Derrick Hau, Peter Thorkildson, and David P. AuCoin. "Burkholderia pseudomallei." In Defense Against Biological Attacks, 185–211. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03071-1_8.

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Sprague, Lisa D., and Mandy C. Elschner. "Burkholderia pseudomallei: Melioidosis." In BSL3 and BSL4 Agents, 303–5. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch18.

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Sprague, Lisa D., and Mandy C. Elschner. "Burkholderia pseudomallei: Melioidosis." In BSL3 and BSL4 Agents, 47–56. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2012. http://dx.doi.org/10.1002/9783527645114.ch4.

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Galeas-Pena, Michelle, and Lisa A. Morici. "Vaccine Development Against Melioidosis." In Vaccines for Neglected Pathogens: Strategies, Achievements and Challenges, 329–44. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-24355-4_15.

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AbstractMelioidosis in an infectious disease of humans and animals caused by the bacterium Burkholderia pseudomallei. Melioidosis is often considered a disease of the tropics, but recent data suggests that B. pseudomallei is distributed worldwide and the disease is likely largely underestimated. B. pseudomallei is inherently resistant to many antibiotics, which complicates treatment, particularly in low-resource countries. There is currently no licensed vaccine to prevent melioidosis. Fortunately, there has been significant progress over the last decade in our understanding of B. pseudomallei pathogenesis and host immunity. This has been paralleled by the discovery and testing of promising vaccine candidates against melioidosis. Collectively, these scientific advances spark optimism that licensure of a safe and effective vaccine is achievable.
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Samy, Ramar Perumal, Gautam Sethi, Bradley G. Stiles, Sok Lin Foo, Octavio Luiz Franco, Frank Arfuso, Lina H. K. Lim, and P. Gopalakrishnakone. "Burkholderia pseudomallei Toxins and Clinical Implications." In Toxinology, 31–49. Dordrecht: Springer Netherlands, 2018. http://dx.doi.org/10.1007/978-94-007-6449-1_12.

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Ramar, Perumal Samy, Gautam Sethi, G. Stiles Bradley, Sok Lin Foo, Octavio Luiz Franco, Arfuso F., Lina H. K. Lim, and Gopalakrishnakone P. "Burkholderia pseudomallei Toxins and Clinical Implications." In Microbial Toxins, 1–19. Dordrecht: Springer Netherlands, 2016. http://dx.doi.org/10.1007/978-94-007-6725-6_12-1.

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Samy, Ramar Perumal, Gautam Sethi, Bradley G. Stiles, Sok Lin Foo, Octavio Luiz Franco, Frank Arfuso, Lina H. K. Lim, and P. Gopalakrishnakone. "Burkholderia pseudomallei Toxins and Clinical Implications." In Toxinology, 1–19. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-007-6725-6_12-2.

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Hussein, Mansour F. "Melioidosis (Whitmore Disease; Burkholderia pseudomallei Infection)." In Infectious Diseases of Dromedary Camels, 159–61. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-79389-0_25.

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Vermaak, Samantha, Samantha Sampson, and Helen McShane. "The VALIDATE Network: Accelerating Vaccine Development for Tuberculosis, Leishmaniasis, Melioidosis and Leprosy." In Vaccines for Neglected Pathogens: Strategies, Achievements and Challenges, 11–25. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-24355-4_2.

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AbstractEstablished in June 2017, VALIDATE is an international network of researchers working to accelerate vaccine development for four neglected intracellular pathogens that cause significant mortality and morbidity globally: Mycobacterium tuberculosis, Leishmania spp., Burkholderia pseudomallei and Mycobacterium leprae. In 5 years, VALIDATE has grown to have more than 550 members from over 250 institutes across 72 countries and has had several successes and important outputs. This chapter discusses VALIDATE’s origins, achievements and future direction.
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Christodoulides, Myron. "Introduction." In Vaccines for Neglected Pathogens: Strategies, Achievements and Challenges, 1–9. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-24355-4_1.

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AbstractNeglected Tropical Diseases (NTDs) are caused by a diversity of pathogens including viruses, bacteria, parasites, fungi, and toxins, which affect 2–3 billion people globally who live in the least developed countries (LDC) and low-to-middle income countries (LMIC). The World Health Organization classifies around 24 NTDs that are prevalent mainly in tropical and sub-tropical areas and these conditions impact enormously on personal and population health, with debilitating social and economic consequences to communities and countries. The overall focus of the book is vaccines for NTDs caused by the organisms studied by the VALIDATE (VAccine deveLopment for complex Intracellular neglecteD pAThogEns) network, i.e. Mycobacterium tuberculosis, M. leprae, Leishmania spp. and Burkholderia pseudomallei.
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Conference papers on the topic "Burkholderia pseudomallei"

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Eng, Su Anne, and Sheila Nathan. "Screening for potential anti-infective agents towards Burkholderia pseudomallei infection." In THE 2014 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2014 Postgraduate Colloquium. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4895249.

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Yusoff, Nur Syamimi, Nadzirah Damiri, and Mohd Firdaus-Raih. "Identification of the conserved hypothetical protein BPSL0317 in Burkholderia pseudomallei K96243." In THE 2014 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2014 Postgraduate Colloquium. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4895258.

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Munyati-Othman, Noor, Ahmad Luqman Abdul Fatah, Mohd Al Akmarul Fizree Bin Md Piji, Effirul Ikhwan Ramlan, and Mohd Firdaus Raih. "Identification and cloning of four riboswitches from Burkholderia pseudomallei strain K96243." In THE 2015 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2015 Postgraduate Colloquium. AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4931239.

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Akhir, Nor Azurah Mat, Nurul Nadzirin, Rahmah Mohamed, and Mohd Firdaus-Raih. "Screening and expression of selected taxonomically conserved and unique hypothetical proteins in Burkholderia pseudomallei K96243." In THE 2015 UKM FST POSTGRADUATE COLLOQUIUM: Proceedings of the Universiti Kebangsaan Malaysia, Faculty of Science and Technology 2015 Postgraduate Colloquium. AIP Publishing LLC, 2015. http://dx.doi.org/10.1063/1.4931238.

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Rufino Leão, Bruna, Bruna Aurora Nunes Cavalcante Castro, Maria das Graças Barbosa Sousa, Kharen Assunção Bezerra Galdino, and Yarla Catarina Antão de Alencar. "OSTEOMYELITIS IN SACROILIAC AND SEPSIS BY Burkholderia pseudomallei: MELIOIDOSIS IN PATIENT WITH SYSTEMIC LUPUS ERYTHEMATOSUS – CASE REPORT." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1952.

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Nawaz, T., B. W. Fouty, and V. Solodushko. "Expression of Burkholderia Pseudomallei Antigens Hcp1 and TssM in Engrafted Tumor Cells Leads to Activation of In Vivo Cytotoxic Immune Response in BALB/c Mice." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7734.

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Reports on the topic "Burkholderia pseudomallei"

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Gardner, S., and C. Jaing. Interim report on updated microarray probes for the LLNL Burkholderia pseudomallei SNP array. Office of Scientific and Technical Information (OSTI), March 2012. http://dx.doi.org/10.2172/1047245.

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Terwilliger, Thomas, Hung Li-Wei, Chang Kim, Kim Huengbok, Kato Takaaki, Yamashita Eeiki, Okada Ui, Herbert Schweizer, and Murakami Satoshi. Crystal Structures of Multidrug Efflux Transporters from 2 Burkholderia pseudomallei suggest details of transport mechanism. Office of Scientific and Technical Information (OSTI), August 2022. http://dx.doi.org/10.2172/2377987.

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Harvey, Steven P. Literature Review of DNA-Based Subspecies Analysis of Bacillus Anthracis Burkholderia Pseudomallel Burkholderia Mallei, and Yersinia Pestis. Fort Belvoir, VA: Defense Technical Information Center, October 1999. http://dx.doi.org/10.21236/ada370205.

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