Log in

Relevant bibliographies by topics / Bulimia nervosa / Journal articles

To see the other types of publications on this topic, follow the link: Bulimia nervosa.

Journal articles on the topic 'Bulimia nervosa'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Bulimia nervosa.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Bossert, S., R. Laessle, and M. Junker. "Anamnestic similarities in bulimic inpatients with and without a history of anorexia nervosa." Psychiatry and Psychobiology 4, no. 2 (1989): 107–10. http://dx.doi.org/10.1017/s0767399x00002947.

Full text

Abstract:

SummaryThe significance of a history of anorexia nervosa as regards the diagnosis and treatment outcome for bulimia is unclear. In a retrospective analysis of medical records of 59 inpatients with bulimia (DSM-III), variables related to personal and psychiatric family history did not reveal any differences in bulimics subtyped according to previous anorexia nervosa as defined in the criteria of Russell (1979). These anamnestic data support the results of studies indicating that no specific clinical and outcome variables are correlated with a history of anorexia nervosa in bulimia. The lower body weight and longer duration of bulimia found in bulimic inpatients with a history of anorexia nervosa, however, should be further examined.

APA, Harvard, Vancouver, ISO, and other styles

2

Cooper, Peter J., Deborah J. Charnock, and Melanie J. Taylor. "The Prevalence of Bulimia Nervosa." British Journal of Psychiatry 151, no. 5 (November 1987): 684–86. http://dx.doi.org/10.1192/bjp.151.5.684.

Full text

Abstract:

There have been reports of a high prevalence of bulimic episodes and the syndromes of bulimia nervosa and DSM-III bulimia in community samples. A group of American authors recently compared the findings of a contemporary survey with those of a survey they had conducted previously and reported a three-fold increase in the prevalence of DSM-III bulimia. The present study replicates a community survey conducted four years ago in Britain. The prevalence of bulimic episodes, self-induced vomiting and bulimia nervosa found in the present survey was very similar to that found in the earlier study.

APA, Harvard, Vancouver, ISO, and other styles

3

Lacey, J. Hubert, and G. Smith. "Bulimia Nervosa." British Journal of Psychiatry 150, no. 6 (June 1987): 777–81. http://dx.doi.org/10.1192/bjp.150.6.777.

Full text

Abstract:

This study examines the impact of pregnancy on the reported eating behaviour of 20 untreated normal body weight bulimia nervosa women; it also reports foetal and obstetric abnormalities and indicates the initial eating habits of the infants. The prevalence of binge-eating and self-induced vomiting reduced sequentially during each trimester of pregnancy. By the third trimester 15 women (75%) had stopped all bulimic behaviour and in the remainder the disturbed eating was less severe. Symptoms tended to return in the Puerperium and in nearly half the sample abnormal eating was more disturbed after delivery than before conception. However, the improvement associated with the pregnancy described by seven patients was maintained and for five it appears to have been curative. The common fear among pregnant bulimics that their abnormal eating behaviour may damage their unborn child cannot be dispelled by this study; the incidence of foetal abnormality (including cleft palate and cleft lip), multiple pregnancies and obstetric complications (including breech presentation and surgical intervention) was high. The nutrition and development of the infants was good although three mothers (15%) reported slimming their babies down within the first year.

APA, Harvard, Vancouver, ISO, and other styles

4

Zeeck, Hartmann, Sandholz, and Joos. "Bulimia nervosa." Therapeutische Umschau 63, no. 8 (August 1, 2006): 535–38. http://dx.doi.org/10.1024/0040-5930.63.8.535.

Full text

Abstract:

Die Bulimia nervosa ist durch Essanfälle und Verhaltensweisen gekennzeichnet, welche einer Gewichtszunahme entgegensteuern sollen (Erbrechen, Laxantienabusus, Hungerphasen u.a.). Sie ist eine multifaktoriell bedingte psychische Erkrankung, welche vor allem junge Frauen betrifft. Die Bulimie kann zu gravierendem Folgen auf körperlicher, psychischer und sozialer Ebene führen und bedarf in der Regel einer spezialisierten, psychotherapeutischen Behandlung. Diese kann in den meisten Fällen ambulant erfolgen, es muss jedoch die häufige Komorbidität mit weiteren psychischen Erkrankungen berücksichtigt werden. Auch eine psychopharmakologische Mitbehandlung kann hilfreich sein. Nach 5–10 Jahren zeigen rund 50% der Patientinnen eine Vollremission, 30% Teilremissionen und etwa 20% einen chronischen Verlauf. Hausärzte, Zahnärzte und Gynäkologen sollten über Anzeichen einer oft von den Betroffenen selbst aus Schamgefühl verheimlichten Bulimia nervosa informiert sein.

APA, Harvard, Vancouver, ISO, and other styles

5

Schumaker, John F., William G. Warren, Gwenda S. Schreiber, and Craig C. Jackson. "DISSOCIATION IN ANOREXIA NERVOSA AND BULIMIA NERVOSA." Social Behavior and Personality: an international journal 22, no. 4 (January 1, 1994): 385–92. http://dx.doi.org/10.2224/sbp.1994.22.4.385.

Full text

Abstract:

The present study employed the Riley Questionnaire of Experiences of Dissociation in order to assess degree of dissociation in females diagnosed with anorexia nervosa and bulimia. The subjects consisted of 26 anorexic and 18 bulimic females, and a non eating-disordered control group of 22 females. Results indicated that eating disordered subjects, considered together, had significantly higher dissociation scores than the non eating-disordered control group. Additionally, when considered separately, both the anorexic and bulimic groups had significantly higher dissociation scores than the control subjects. No significant difference was found in the level of dissociation between anorexic and bulimic groups. These findings are discussed in relation to previous investigations and implications for possible future research and treatment.

APA, Harvard, Vancouver, ISO, and other styles

6

Fichter, M. M., N. Quadflieg, and W. Rief. "Course of multi-impulsive bulimia." Psychological Medicine 24, no. 3 (August 1994): 591–604. http://dx.doi.org/10.1017/s0033291700027744.

Full text

Abstract:

SynopsisThirty-two consecutively admitted females with bulimia nervosa (purging type) according to DSM-IV and additional impulsive behaviours (multi-impulsive bulimia (MIB)) and 32 age-matched female controls with DSM-IV bulimia nervosa (purging type) (uni-impulsive bulimia (UIB)) were assessed longitudinally on admission and at discharge following in-patient therapy and at a 2-year follow-up. Multi-impulsive bulimics were defined as presenting at least three of the six of the following impulsive behaviours in their life-time in addition to their bulimic symptoms at admission: (a) suicidal attempts, (b) severe autoaggression, (c) shop lifting (other than food), (d) alcohol abuse, (e) drug abuse, or (f) sexual promiscuity. Multi-impulsive bulimics were more frequently separated or divorced, had less schooling and held less-skilled jobs. Except for interoceptive awareness (EDI), which was more disturbed in multi-impulsive bulimics, there were no differences concerning scales measuring eating disturbances and related areas. Multi-impulsive bulimics showed more general psychopathology – anxiety, depression, anger and hostility, psychoticism – differed in several personality scales from uni-impulsive bulimics (e.g. increased excitability and anger/hostility) and had overall a less favourable course of illness. Multi-impulsive bulimics also received more in- and out-patient therapy previous to the index treatment and during the follow-up period. The data support the notion that ‘multi-impulsive bulimia’ or ‘multi-impulsive disorder’ should be classified as a distinct diagnostic group on axis I or that an ‘Impulsive Personality Disorder’ should be introduced on axis II. The development of more effective treatment for multi-impulsive bulimia is warranted.

APA, Harvard, Vancouver, ISO, and other styles

7

Bossert-Zaudig, S., M. Zaudig, M. Junker, M. Wiegand, and J.-C. Krieg. "Psychiatric comorbidity of bulimia nervosa inpatients: relationship to clinical variables and treatment outcome." European Psychiatry 8, no. 1 (1993): 15–23. http://dx.doi.org/10.1017/s0924933800001504.

Full text

Abstract:

SummaryExperimental evidence suggesting that psychiatric comorbidity has important clinical and prognostic implications in bulimia nervosa has mostly been based on outpatient studies investigating a selection of co-existing psychopathological features with rather unstructured and not standardized diagnostic instruments. Using structured instruments (SCID-P, MDCL) for the diagnoses of DSM III-R axis I disorders and clinical interviews for the diagnosis of DSM III-R axis II disorders in 24 hospitalized bulimics, the present study demonstrated that more than half of the patients had two or three axis I disorders in addition to bulimia nervosa and almost half of the patients met criteria of at least one personality disorder. Subgroups of patients classified according to the type of psychiatric comorbidity did not differ significantly with respect to clinical features; regarding measures of hospital behavior therapy outcome. However, the findings provided evidence for a negative impact of anxiety disorder in addition to bulimia nervosa on the improvement of bulimic behavior and possibly also on self-rated depression.

APA, Harvard, Vancouver, ISO, and other styles

8

Wade, Tracey D., Cynthia M. Bulik, and Kenneth S. Kendler. "Reliability of lifetime history of bulimia nervosa." British Journal of Psychiatry 177, no. 1 (July 2000): 72–76. http://dx.doi.org/10.1192/bjp.177.1.72.

Full text

Abstract:

BackgroundPrevious studies have found that the reliability of the lifetime prevalence of bulimia nervosa is low to moderate. However, the reasons for poor reliability remain unknown.AimsWe investigated the ability of a range of variables to predict reliability, sensitivity, and specificity of reporting of both bulimia nervosa and major depression.MethodTwo interviews, approximately 5 years apart, were completed with 2163 women from the Virginia Twin Registry.ResultsAfter accounting for different base rates, bulimia nervosa was shown to be as reliably reported as major depression. Consistent with previous studies of major depression, improved reliability of bulimia nervosa reporting is associated with more severe bulimic symptomatology.ConclusionsFrequent binge eating and the presence of salient behavioural markers such as vomiting and laxative misuse are associated with more reliable reporting of bulimia nervosa. In the absence of the use of fuller forms of assessment, brief interviews should utilise more than one prompt question, thus increasing the probability that memory of past disorders will be more successfully activated and accessed.

APA, Harvard, Vancouver, ISO, and other styles

9

Robinson, P. H., S. A. Checkley, and G. F. M. Russell. "Suppression of Eating by Fenfluramine in Patients with Bulimia Nervosa." British Journal of Psychiatry 146, no. 2 (February 1985): 169–76. http://dx.doi.org/10.1192/bjp.146.2.169.

Full text

Abstract:

SummaryFifteen patients with bulimia nervosa received fenfluramine (60 mg po) or placebo under double-blind, randomly ordered conditions. Two hours later food was presented. Significantly less food was eaten after fenfluramine and the quantity eaten was inversely correlated with serum fenfluramine levels. Significantly fewer patients reported bulimic symptoms during the test after fenfluramine, but no significant effect was demonstrated after leaving the ward. Fenfluramine caused drowsiness but did not reduce hunger ratings. Similarly, eating failed to reduce hunger ratings normally in the patients. These findings suggest that in patients with bulimia nervosa, hunger is reported abnormally and eating is suppressed by fenfluramine. Bulimic symptoms were probably reduced by fenfluramine, which may prove to be a useful treatment for bulimia nervosa.

APA, Harvard, Vancouver, ISO, and other styles

10

Fahy, Thomas, and Ivan Eisler. "Impulsivity and Eating Disorders." British Journal of Psychiatry 162, no. 2 (February 1993): 193–97. http://dx.doi.org/10.1192/bjp.162.2.193.

Full text

Abstract:

Sixty-seven patients with bulimia nervosa and 29 patients with anorexia nervosa completed the Impulsiveness Questionnaire and questionnaires detailing severity of eating disorder. Bulimic patients had higher impulsivity scores than anorexic patients. Bulimics with high impulsivity scores did not have more severe eating disorders than low scorers. When 39 bulimics and 25 anorexics were interviewed about other impulsive behaviour, 51 % of bulimics and 28% of anorexics reported at least one other impulsive behaviour. Patients with so-called ‘multiimpulsive’ bulimia reported more severe eating disturbance, but this was not reflected on more reliable measures of symptoms. Thirty-nine bulimics entered an eight-week treatment trial and their progress was assessed at eight weeks, 16 weeks and one year. ‘Non-impulsive’ bulimics had a more rapid response than ‘impulsives' during treatment, but there was no difference at follow-up. There was no evidence of an association between high impulsivity trait scores and poor treatment response. It is concluded that impulsivity may shape the expression of eating disorders, but that ‘multi-impulsives' do not constitute a categorically distinct subgroup of bulimics.

APA, Harvard, Vancouver, ISO, and other styles

11

LESTER, N. A., P. K. KEEL, and S. F. LIPSON. "Symptom fluctuation in bulimia nervosa: relation to menstrual-cycle phase and cortisol levels." Psychological Medicine 33, no. 1 (December 23, 2002): 51–60. http://dx.doi.org/10.1017/s0033291702006815.

Full text

Abstract:

Background. Individuals with bulimia nervosa report significant symptom fluctuation, and some studies have suggested a premenstrual exacerbation of binge frequency. The purpose of this study is to explore the hormonal correlates of symptom fluctuation in bulimia nervosa.Method. For five consecutive weeks (one full menstrual cycle), eight women with bulimia nervosa and eight non-eating-disordered control women collected morning saliva samples and recorded several mood characteristics; the bulimic women also recorded binge and purge episodes. Subsequently, salivary cortisol and androgen levels were determined by radioimmunoassay.Results. Bulimic symptoms were exacerbated in both the mid-luteal and premenstrual phases, when compared with the follicular and ovulatory phases (F(3,21)=3·76, P=0·026; contrast analysis t(7)=3·47, P<0·01). Fluctuation in cortisol was closely correlated with fluctuation of bulimic symptoms, with elevated cortisol secretion following symptom exacerbation (r(24)=0·64, P=0·001).Conclusions. Bulimic symptom fluctuation appears to be related to two hormonal phenomena – phase of the menstrual cycle and cortisol secretion – with menstrual-cycle phase influencing bulimic symptom severity, and bulimic symptom severity effecting increases in cortisol secretion. Improved understanding of the hormonal causes and consequences of symptom fluctuation may lead to improved psychological and pharmacological treatments for bulimia nervosa.

APA, Harvard, Vancouver, ISO, and other styles

12

Huemer, Julia, Maria Haidvogl, Fritz Mattejat, Gudrun Wagner, Gerald Nobis, Fernando Fernandez-Aranda, David A. Collier, Janet L. Treasure, and Andreas F. K. Karwautz. "Perception of Autonomy and Connectedness Prior to the Onset of Anorexia Nervosa and Bulimia Nervosa." Zeitschrift für Kinder- und Jugendpsychiatrie und Psychotherapie 40, no. 1 (January 2012): 61–68. http://dx.doi.org/10.1024/1422-4917/a000150.

Full text

Abstract:

Objective: This study examines retrospective correlates of nonshared family environment prior to onset of disease, by means of multiple familial informants, among anorexia and bulimia nervosa patients. Methods: A total of 332 participants was included (anorexia nervosa, restrictive type (AN-R): n = 41 plus families); bulimic patients (anorexia nervosa, binge-purging type; bulimia nervosa: n = 59 plus families). The EATAET Lifetime Diagnostic Interview was used to establish the diagnosis; the Subjective Family Image Test was used to derive emotional connectedness (EC) and individual autonomy (IA). Results: Bulimic and AN-R patients perceived significantly lower EC prior to onset of disease compared to their healthy sisters. Bulimic patients perceived significantly lower EC prior to onset of disease compared to AN-R patients and compared to their mothers and fathers. A low family sum – sister pairs sum comparison – of EC had a significant influence on the risk of developing bulimia nervosa. Contrary to expectations, AN-R patients did not perceive significantly lower levels of IA compared to their sisters, prior to onset of disease. Findings of low IA in currently ill AN-R patients may represent a disease consequence, not a risk factor. Conclusions: Developmental child psychiatrists should direct their attention to disturbances of EC, which may be present prior to the onset of the disease.

APA, Harvard, Vancouver, ISO, and other styles

13

Morgan, John Farnill, J. Hubert Lacey, and Philip M. Sedgwick. "Impact of pregnancy on bulimia nervosa." British Journal of Psychiatry 174, no. 2 (February 1999): 135–40. http://dx.doi.org/10.1192/bjp.174.2.135.

Full text

Abstract:

BackgroundBulimia nervosa affects women at a peak age of reproductive functioning, but few studies have examined the impact of pregnancy on bulimia.AimTo examine the impact of pregnancy on symptoms of bulimia nervosa and associated psychopathology.MethodWomen actively suffering from bulimia nervosa during pregnancy (n=94) were interviewed using the eating disorder examination (12th edn) and structured clinical interview for DSM–III–R, with additional structured questions. Behaviours were recorded at conception, each trimester and postnatally. Relative risks were calculated for prognostic factors.ResultsBulimic symptoms improved throughout pregnancy. After delivery, 57% had worse symptoms than pre-pregnancy, but 34% were no longer bulimic. Relapse was predicted by behavioural severity and persistence, previous anorexia nervosa (‘Type II’ bulimia), gestational diabetes and ‘unplanned’ pregnancy. Unplanned pregnancies were the norm, usually resulting from mistaken beliefs about fertility. ‘Postnatal depression’ was suggested in one-third of the sample, and in two-thirds of those with ‘Type II’ bulimia, and was predicted by alcohol misuse, symptom severity and persistence.ConclusionsPostnatal treatment intervention should focus on women ‘at risk’ of relapse, but all women with bulimia should be assessed for postnatal depression.

APA, Harvard, Vancouver, ISO, and other styles

14

Fitzgerald, Barbara A., Jesse H. Wright, and Katherine D. Atala. "Bulimia nervosa." Postgraduate Medicine 84, no. 2 (August 1988): 119–23. http://dx.doi.org/10.1080/00325481.1988.11700368.

Full text

APA, Harvard, Vancouver, ISO, and other styles

15

Rushing, Jona M., Laura E. Jones, and Caroline P. Carney. "Bulimia Nervosa." Primary Care Companion to The Journal of Clinical Psychiatry 05, no. 05 (October 1, 2003): 217–24. http://dx.doi.org/10.4088/pcc.v05n0505.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Mehler, Philip S. "Bulimia Nervosa." New England Journal of Medicine 349, no. 9 (August 28, 2003): 875–81. http://dx.doi.org/10.1056/nejmcp022813.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Fairburn, C. G. "Bulimia nervosa." BMJ 300, no. 6723 (February 24, 1990): 485–87. http://dx.doi.org/10.1136/bmj.300.6723.485.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Svaldi, Jennifer, and Brunna Tuschen-Caffier. "Bulimia nervosa." PSYCH up2date 12, no. 05 (September 2018): 415–31. http://dx.doi.org/10.1055/a-0498-3661.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Russell, Gerald, and Janet Treasure. "Bulimia nervosa." British Journal of Psychiatry 201, no. 1 (July 2012): 19. http://dx.doi.org/10.1192/bjp.bp.112.109645.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Lilly, R. Z. "Bulimia nervosa." BMJ 327, no. 7411 (August 16, 2003): 380–81. http://dx.doi.org/10.1136/bmj.327.7411.380.

Full text

APA, Harvard, Vancouver, ISO, and other styles

21

POWERS, PAULINE S. "Bulimia Nervosa." American Journal of Psychiatry 148, no. 8 (August 1991): 1082—a—1083. http://dx.doi.org/10.1176/ajp.148.8.1082-a.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Erpen, Heinrich. "Bulimia nervosa." Daseinsanalyse 4, no. 4 (1987): 285–313. http://dx.doi.org/10.1159/000456200.

Full text

APA, Harvard, Vancouver, ISO, and other styles

23

LACEY, J. HUBERT, and G. SMITH. "Bulimia Nervosa." Obstetrical & Gynecological Survey 44, no. 3 (March 1989): 191–92. http://dx.doi.org/10.1097/00006254-198903000-00006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

24

Sedghizadeh, Parish P. "Bulimia Nervosa." New England Journal of Medicine 368, no. 13 (March 28, 2013): 1238. http://dx.doi.org/10.1056/nejmicm1207495.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

Hall, Richard C. W., Robert E. Blakey, and Anne Klassen Hall. "Bulimia Nervosa." Psychosomatics 33, no. 4 (November 1992): 428–36. http://dx.doi.org/10.1016/s0033-3182(92)71947-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

26

Herpertz, Stephan. "Bulimia nervosa." PiD - Psychotherapie im Dialog 2, no. 2 (June 2001): 139–53. http://dx.doi.org/10.1055/s-2001-15586.

Full text

APA, Harvard, Vancouver, ISO, and other styles

27

Mendhekar, D. N., Rajiv Mehta, and P. K. Srivastav. "Bulimia nervosa." Indian Journal of Pediatrics 71, no. 9 (September 2004): 861–62. http://dx.doi.org/10.1007/bf02730730.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Đurović, Marija. "Bulimia: Modern man challenge." Galenika Medical Journal 1, no. 1 (2022): 78–86. http://dx.doi.org/10.5937/galmed2201078d.

Full text

Abstract:

The fact, that man has always been focused on external beauty is never surprising. The base of our identity is our body. However, it is worrying to know that whether they are obese, thin, or normally fed, people of both sexes and all age groups have never been as dissatisfied with their bodies as they are today. Many do not perceive eating disorders as serious diseases, but as someone's whim, the desire to be thin, and some even go so far as to accept a disturbed diet as healthy. We notice that healthy people talk about eating disorders in an affirmative tone - they don't understand that it is a disease. Avoiding a normal diet is considered "normal" and even desirable. Unfortunately, eating disorders are culturally normalized. The most commonly diagnosed eating disorders are anorexia nervosa and bulimia nervosa. Unlike anorexia nervosa, bulimia nervosa is much more difficult to detect. It can remain unrecognized for a long time even from the immediate environment, because a person suffering from bulimia is usually of normal body weight, and bulimic crises usually take place in secret. And then, even when the disease is discovered, its meaning and severity are often unrecognized. Unfortunately, bulimia nervosa is often experienced as a whim of a spoiled child from a rich society. In etiological terms, bulimia nervosa is a multifactorial disease. In its origin and development, the role is played by various factors that interact with each other: genetic, neurobiological, psychological and socio-cultural. There are many interpretations of the psychodynamics of bulimia nervosa, the significance and symbolism of symptoms. The most common comorbidities are depression, anxiety disorders, PTSD, and substance abuse. The consequences of the disease are in the sphere of mental and physical health. Recognition and treatment of bulimia nervosa is extremely important, and psychotherapy and pharmacotherapy are used in the treatment of patients.

APA, Harvard, Vancouver, ISO, and other styles

29

FRANKO, D. L., P. K. KEEL, D. J. DORER, M. A. BLAIS, S. S. DELINSKY, K. T. EDDY, V. CHARAT, R. RENN, and D. B. HERZOG. "What predicts suicide attempts in women with eating disorders?" Psychological Medicine 34, no. 5 (July 2004): 843–53. http://dx.doi.org/10.1017/s0033291703001545.

Full text

Abstract:

Background. Suicide is a common cause of death in anorexia nervosa and suicide attempts occur often in both anorexia nervosa and bulimia nervosa. No studies have examined predictors of suicide attempts in a longitudinal study of eating disorders with frequent follow-up intervals. The objective of this study was to determine predictors of serious suicide attempts in women with eating disorders.Method. In a prospective longitudinal study, women diagnosed with either DSM-IV anorexia nervosa (n=136) or bulimia nervosa (n=110) were interviewed and assessed for suicide attempts and suicidal intent every 6–12 months over 8·6 years.Results. Fifteen percent of subjects reported at least one prospective suicide attempt over the course of the study. Significantly more anorexic (22·1%) than bulimic subjects (10·9%) made a suicide attempt. Multivariate analyses indicated that the unique predictors of suicide attempts for anorexia nervosa included the severity of both depressive symptoms and drug use over the course of the study. For bulimia nervosa, a history of drug use disorder at intake and the use of laxatives during the study significantly predicted suicide attempts.Conclusions. Women with anorexia nervosa or bulimia nervosa are at considerable risk to attempt suicide. Clinicians should be aware of this risk, particularly in anorexic patients with substantial co-morbidity.

APA, Harvard, Vancouver, ISO, and other styles

30

Palmer, Teresa A. "Anorexia Nervosa, Bulimia Nervosa." Nurse Practitioner 15, no. 4 (April 1990): 12???21. http://dx.doi.org/10.1097/00006205-199004000-00007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

31

Cooper, Peter J., and Melanie J. Taylor. "Body Image Disturbance in Bulimia Nervosa." British Journal of Psychiatry 153, S2 (1988): 32–36. http://dx.doi.org/10.1192/s0007125000298966.

Full text

Abstract:

Bulimia nervosa is a disorder which has only recently been identified and described (Russell, 1979). It is a condition, found almost exclusively in young women, in which episodes of excessive uncontrolled eating are the central feature. Patients with bulimia nervosa usually present at normal weight because they compensate for the bulimic episodes in a variety of ways, such as by inducing vomiting, abusing purgatives and exercising extreme dietary restraint. In addition to these disturbed eating habits, patients with bulimia nervosa have attitudes to their shape and weight similar to those found in anorexia nervosa. They also present with a wide range of neurotic symptoms, particularly of a depressive nature (Russell, 1979; Fairburn & Cooper, 1984). It is generally accepted that these neurotic symptoms are usually a secondary reaction to the core eating disorder rather than of primary diagnostic significance (Fairburn et al, 1985; Cooper & Fairburn, 1986).

APA, Harvard, Vancouver, ISO, and other styles

32

Srivastav, Yash, Mohd Faijan Mansoori, Aditya Srivastav, and Aniket Kumar. "Schematic Brief Outline: Bulimia Nervosa and its Medical-Based Management." International Neuropsychiatric Disease Journal 21, no. 4 (May 30, 2024): 61–65. http://dx.doi.org/10.9734/indj/2024/v21i4442.

Full text

Abstract:

The disease known as bulimia nervosa, which is typified by purging and binge eating, usually starts in adolescence and peaks at the age of 18. The ratio of female to male patients varies from 10:1 to 20:1, with a lifetime frequency of 3 per cent. The majority of bulimic individuals also suffer from other mental illnesses, such as depression or anxiety. Additionally, there is a correlation between substance misuse and promiscuity. Bulimia nervosa was initially identified as a "chronic phase of anorexia nervosa" in 1979 by British psychiatrist Gerald Russell. During this stage, patients overeat and resort to compensatory methods such as self-induced vomiting, laxatives, or extended periods of deprivation. For three months, bingeing and purging episodes occurring at least once a week are typically associated with a bulimia diagnosis. However, even infrequent binge and purge behaviours can be harmful and require medical attention. The severity of the bulimia increases with the frequency of the bouts. Family therapy and individual treatment are frequently used to treat bulimia. The goal is to address any dietary issues and modify your behaviour. The relationship between your thoughts, feelings, and behaviours is examined in therapy. We go over the aetiology, epidemiology, current treatment, and state of bulimia nervosa in this review study.

APA, Harvard, Vancouver, ISO, and other styles

33

Naessén, Sabine, Kjell Carlström, Rolf Glant, Hans Jacobsson, and Angelica Lindén Hirschberg. "Bone mineral density in bulimic women – influence of endocrine factors and previous anorexia." European Journal of Endocrinology 155, no. 2 (August 2006): 245–51. http://dx.doi.org/10.1530/eje.1.02202.

Full text

Abstract:

Objective: Data concerning bone mineral density (BMD) in bulimia nervosa are contradictory and include both low and normal values. The aim of the present study was to elucidate possible endocrine-and nutrition-related factors predicting BMD in bulimic women. Design: Cross-sectional study. Methods: Seventy-seven bulimic patients and 56 age- and body mass index (BMI)-matched healthy controls were examined with respect to BMD (dual energy X-ray absorptiometry) and to serum levels of hormones and metabolic factors. Results: Bulimics had significantly lower spinal BMD and higher frequency of osteopenia in the total body than controls. Furthermore, bulimic women had significantly lower levels of estradiol-17β and free thyroxine and significantly higher cortisol levels compared with controls. Among the bulimics, 31.2% had present menstrual disturbance, 51.9% had a history of amenorrhea and 23.4% had previous anorexia nervosa. Subgroups of bulimics with a history of amenorrhea and previous anorexia nervosa had significantly lower total and spinal BMD than controls, whereas those without such history did not differ from the controls. In univariate analysis, a history of amenorrhea, cortisol, testosterone, previous anorexia nervosa, and BMI showed significant associations with spinal BMD. Multiple regression analysis including all significant variables revealed previous anorexia nervosa to be the strongest determinant of spinal BMD, accounting for 34% of the variance, while associations between endocrine factors and BMI disappeared. Conclusions: Low bone mass in bulimics may be explained by previous anorexia nervosa, whereas endocrine variables related to BMD seem to be secondary determinants that are dependent on previous anorexia nervosa and BMI.

APA, Harvard, Vancouver, ISO, and other styles

34

MONTELEONE, P., F. BRAMBILLA, F. BORTOLOTTI, and M. MAJ. "Serotonergic dysfunction across the eating disorders: relationship to eating behaviour, purging behaviour, nutritional status and general psychopathology." Psychological Medicine 30, no. 5 (September 2000): 1099–110. http://dx.doi.org/10.1017/s0033291799002330.

Full text

Abstract:

Background. Several recent studies have pointed to a dysfunction of serotonin transmission in patients with eating disorders. Notwithstanding, it is not known whether serotonergic abnormalities are related primarily to eating and/or purging behaviour, nutritional status or general psychopathological dimensions. Therefore, by using a validated neuroendocrine strategy, we investigated central serotonergic function in patients with anorexia nervosa, bulimia nervosa or binge-eating disorder who differ on the above parameters.Methods. Plasma prolactin response to D-fenfluramine (30 mg p.o.) or placebo was measured in 58 drug-free female volunteers, comprising 15 underweight anorexic women, 18 bulimic women, 10 women with binge-eating disorder and 15 female healthy controls. Behavioural assessment included ratings of eating disorder symptoms, depression, aggression and food-related obsessions and compulsions.Results. A significantly decreased prolactin response to D-fenfluramine was found in underweight anorexic women and in bulimics with high frequency bingeing ([ges ]2 binge episodes/day), but not in patients with binge-eating disorder or in bulimics with low frequency bingeing ([les ]1 binge episode/day). In the whole bulimic group, a negative correlation emerged between frequency of bingeing and prolactin response. No significant correlation was found between physical or psychopathological measures and the hormonal response in any group.Conclusions. These results confirm our previous findings of an impaired serotonergic transmission in underweight anorexics and in bulimics with high frequency bingeing, but not in patients with less severe bulimia nervosa. Moreover, they show, for the first time, that the hypothalamic serotonergic system is not altered in women with binge-eating disorder.

APA, Harvard, Vancouver, ISO, and other styles

35

Schmidt, Ulrike, Jane Tiller, and Janet Treasure. "Setting the scene for eating disorders: childhood care, classification and course of illness." Psychological Medicine 23, no. 3 (August 1993): 663–72. http://dx.doi.org/10.1017/s0033291700025447.

Full text

Abstract:

SynopsisThe aim of this study was to determine whether the childhood experiences of patients with anorexia nervosa and bulimia nervosa differ and affect the course of the illness. A semistructured interview developed by Harris et al. (1986) was used to assess the childhood family environment of 64 patients with restricting anorexia nervosa (RAN), 23 patients with bulimic anorexia nervosa (BAN), 37 bulimic patients with a history of anorexia nervosa (BN/HistAN) and 79 patients with normal weight bulimia nervosa (BN).There were no significant differences between groups in terms of parental mental disorder, low parental control or childhood sexual abuse. BN patients had had significantly more family arrangements and had experienced more parental indifference, excessive parental control, physical abuse, and violence against other family members than RAN patients with the BAN and BN/HistAN group being intermediate. There was a trend for BN-patients to have had more intra-familial discord than the other groups. Different aspects of adversity tended to cluster in the same patients and 65% of the bulimic group had experienced two or more types of childhood adversity. These results suggest that childhood experiences contribute to the form of eating disorder which later develops.

APA, Harvard, Vancouver, ISO, and other styles

36

Rodin, Gary M., Liane E. Johnson, Paul E. Garfinkel, Denis Daneman, and Anne B. Kenshole. "Eating Disorders in Female Adolescents with Insulin Dependent Diabetes Mellitus." International Journal of Psychiatry in Medicine 16, no. 1 (March 1987): 49–57. http://dx.doi.org/10.2190/hulh-ctpr-4v17-383c.

Full text

Abstract:

Recent case reports have suggested an association between anorexia nervosa and/or bulimia with insulin-dependent diabetes mellitus (IDDM). Fifty-eight females aged fifteen to twenty-two with IDDM for more than one year were assessed for the presence of eating disorders. Patients were screened for eating and weight pathology using the Eating Disorder Inventory (EDI) and Eating Attitudes Test-26 (EAT-26). Glycosylated hemoglobin (HbAl) was measured to assess metabolic control. Subjects who scored above the cut-off points associated with eating and weight pathology were interviewed. Clinically significant eating and weight pathology was found in 20.7 percent of the population. Of these subjects, anorexia nervosa was found in 6.9 percent and the syndrome of bulimia, based on DSM-III criteria, was found in 6.9 percent. In patients with bulimia, there was a strong inverse correlation between bulimic symptoms and metabolic control. These findings suggest that anorexia nervosa may be more common in female adolescents with IDDM than in nondiabetic populations and that bulimic symptoms may be a risk factor for poor metabolic control.

APA, Harvard, Vancouver, ISO, and other styles

37

Sullivan, Patrick F., Cynthia M. Bulik, Frances A. Carter, and Peter R. Joyce. "The Significance of a History of Childhood Sexual Abuse in Bulimia Nervosa." British Journal of Psychiatry 167, no. 5 (November 1995): 679–82. http://dx.doi.org/10.1192/bjp.167.5.679.

Full text

Abstract:

BackgroundChildhood sexual abuse (CSA) is found to have occurred to a substantial minority of women with bulimia nervosa. Its clinical significance is unclear.MethodWe studied 87 bulimic women in a clinical trial. Structured interviews determined the presence of CSA, DSM–III–R disorders, global functioning, and depressive and bulimic symptoms.ResultsForty-four per cent reported a history of CSA. Bulimic women with CSA reported earlier onset of bulimia, greater depressive symptoms, worse global functioning and more suicide attempts, and were more likely to meet criteria for bipolar II disorder, alcohol and drug dependence, conduct disorder and avoidant personality disorder.ConclusionsAlthough those with CSA had greater comorbidity, it was not an important modifier of bulimic symptoms.

APA, Harvard, Vancouver, ISO, and other styles

38

Turnbull, Sue, Anne Ward, Janet Treasure, Hershel Jick, and Laura Derby. "The Demand for Eating Disorder Care." British Journal of Psychiatry 169, no. 6 (December 1996): 705–12. http://dx.doi.org/10.1192/bjp.169.6.705.

Full text

Abstract:

BackgroundAn epidemiological study of anorexia nervosa and bulimia nervosa in primary care was performed using the General Practice Research Database (GPRD).MethodThe GPRD was screened between 1988 and 1994 for newly diagnosed cases of anorexia nervosa and bulimia nervosa. The validity of the computer diagnosis was established by obtaining clinical details from a random sample of the general practitioners (GPs).ResultsIncidence rates for detection of cases by GPs in 1993 was 4.2 per 100 000 population for anorexia nervosa and 12.2 per 100 000 for bulimia nervosa The relative risks of females to males was 40:1 for anorexia nervosa and 47:1 for bulimia nervosa A threefold increase in the recording of bulimia nervosa was found from 1988 to 1993. Eighty per cent of anorexia nervosa cases and 60% of bulimia nervosa cases were referred to secondary care.ConclusionThere is a continuing expansion of service need for bulimia nervosa The majority of cases of eating disorders are referred to secondary services. There is scope for more effective management of bulimia nervosa in primary care.

APA, Harvard, Vancouver, ISO, and other styles

39

Krauß, Eva, and Martina de Zwaan. "Anorexia nervosa und Bulimia nervosa." Psychiatrie und Psychotherapie up2date 1, no. 4 (July 2007): 273–89. http://dx.doi.org/10.1055/s-2007-970824.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Treasure, J. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 3, no. 2 (April 1990): 211–14. http://dx.doi.org/10.1097/00001504-199004000-00007.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Woodside, D. Blake. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 3, no. 4 (August 1990): 453–56. http://dx.doi.org/10.1097/00001504-199008000-00006.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Treasure, Janet. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 4, no. 2 (April 1991): 236–41. http://dx.doi.org/10.1097/00001504-199104000-00009.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Treasure, Janet. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 5, no. 2 (April 1992): 228–33. http://dx.doi.org/10.1097/00001504-199204000-00009.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Smith, Jane Ellen, and Denise E. Laframboise. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 8, no. 6 (November 1995): 419–23. http://dx.doi.org/10.1097/00001504-199511000-00016.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Müller, Astrid, and Martina de Zwaan. "Anorexia nervosa und Bulimia nervosa." Geburtshilfe und Frauenheilkunde 65, no. 6 (June 2005): R105—R120. http://dx.doi.org/10.1055/s-2005-865792.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

Krauß, E., and M. de Zwaan. "Anorexia nervosa und Bulimia nervosa." Frauenheilkunde up2date 2, no. 04 (August 2008): 347–68. http://dx.doi.org/10.1055/s-2008-1076910.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

Støylen, Inge Jarl, and Jon Chr Laberg. "Anorexia Nervosa and Bulimia Nervosa." Acta Psychiatrica Scandinavica 82 (June 28, 2008): 52–58. http://dx.doi.org/10.1111/j.1600-0447.1990.tb11086.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

Huber, M. "Anorexia nervosa und Bulimia nervosa." Der Nephrologe 13, no. 1 (November 2, 2017): 12–18. http://dx.doi.org/10.1007/s11560-017-0205-3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Şaban, Havva, and Esra Baki. "ANOREXIA NERVOSA AND BULIMIA NERVOSA." Vision International Refereed Scientific Journal 1 (2024): 27–46. http://dx.doi.org/10.55843/ivisum241027sh.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

SULLIVAN, P. F., C. M. BULIK, and K. S. KENDLER. "The epidemiology and classification of bulimia nervosa." Psychological Medicine 28, no. 3 (May 1998): 599–610. http://dx.doi.org/10.1017/s0033291798006576.

Full text

Abstract:

Background. We sought to determine whether there was empirical support for the diagnostic thresholds of DSM-IV bulimia nervosa (BN) and whether an empirically derived typology resembled the diagnostic categories of DSM-IV.Methods. Detailed information about bulimic behaviours were assessed via personal interview in a population-based sample of 1897 Caucasian female twins. We assessed the lifetime prevalence of the component bulimic behaviours and DSM-IV and DSM-III-R BN. Latent class analysis of nine separate bulimic symptoms was used to develop an empirical typology of bulimic behaviour.Results. Although the lifetime prevalences of bingeing (23·6%) and vomiting (4·8%) were relatively common, DSM-IV BN was distinctly uncommon (0·5%). The criterion that specified the frequency and duration of bingeing and vomiting was an important limiting condition. Analysis of alternative thresholds found little support for the DSM-IV thresholds requiring an average of twice per week for 3 months. Latent class analysis yielded an interpretable four class solution that had little overlap with the DSM-IV typology.Conclusions. As in other studies of unselected samples of women, the lifetime presence of bulimic behaviours are relatively high. Our results suggest that the DSM-IV approach to categorizing bulimic behaviour inadequately captures the spectrum of lifetime bulimic behaviours in the general population.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!