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1

Cercle d'étude de la déportation et de la Shoah-Amicale d'Auschwitz. Témoigner de la déportation dans les classes: Film-débat du 9 avril 2008, Il faudra raconter, en présence de Daniel Cling, co-réalisateur du film, Sam Braun, rescapé d'Auschwitz, et Claude Dumond, professeur d'histoire. Paris: Cercle d'étude de la déportation et de la Shoah-Amicale d'Auschwitz, 2008.

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2

Odagaki, Yuji, and Dasiel O. Borroto-Escuela, eds. Co-Immunoprecipitation Methods for Brain Tissue. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8985-0.

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3

Frank, Rösler, ed. Lifespan development and the brain: The perspective of biocultural co-constructivism. Cambridge: Cambridge University Press, 2010.

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4

B, Baltes Paul, Reuter-Lorenz Patricia Ann 1958-, and Rösler Frank, eds. Lifespan development and the brain: The perspective of biocultural co-constructivism. New York: Cambridge University Press, 2006.

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5

East Sussex, Brighton and Hove Health Authority. and Headway (Organisation), eds. Acquired brain injury in East Sussex: Incidence, services & recommendations for change : report by the Acquired Brain Injury Co-ordinator (East Sussex), 1999-2001. [Nottingham?]: Headway, 2001.

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6

United States. National Aeronautics and Space Administration., ed. "CO₂-O₂ interactions in extension of tolerance to acute hypoxia": Final report. [Washington, DC]: National Aeronautics and Space Administration, 1995.

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7

United States. National Aeronautics and Space Administration., ed. "CO₂-O₂ interactions in extension of tolerance to acute hypoxia": Final report. [Washington, DC]: National Aeronautics and Space Administration, 1995.

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8

Katayama, Yusuke. Prolonged release of brain-derived neurotrophic factor from poly(lactide-co-glycolide) microspheres dispersed within a polyethylene glycol hydrogel. Ottawa: National Library of Canada, 2003.

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9

Richter, Jochen. Rasse, Elite, Pathos: Eine Chronik zur medizinischen Biographie Lenins und zur Geschichte der Elitegehirnforschung in Dokumenten ; a study in co-operation with the Stalin Era Research and Archives Project of the University of Toronto. Herbolzheim: Centaurus, 2000.

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10

Co-Immunoprecipitation Methods for Brain Tissue. Humana, 2018.

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11

Talairach, Jean, and P. Tournoux. Co-planar Stereotaxic Atlas of the Human Brain. Thieme Publishing Group, 1988.

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12

Seeck, Margitta, L. Spinelli, Jean Gotman, and Fernando H. Lopes da Silva. Combination of Brain Functional Imaging Techniques. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0046.

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Several tools are available to map brain electrical activity. Clinical applications focus on epileptic activity, although electric source imaging (ESI) and electroencephalography-coupled functional magnetic resonance imaging (EEG–fMRI) are also used to investigate non-epileptic processes in healthy subjects. While positron-emission tomography (PET) reflects glucose metabolism, strongly linked with synaptic activity, and single-photon-emission computed tomography (SPECT) reflects blood flow, fMRI (BOLD) signals have a hemodynamic component that is a surrogate signal of neuronal (synaptic) activity. The exact interpretation of BOLD signals is not completely understood; even in unifocal epilepsy, more than one region of positive or negative BOLD is often observed. Co-registration of medical images is essential to answer clinical questions, particularly for presurgical epilepsy evaluations. Multimodal imaging can yield information about epileptic foci and underlying networks. Co-registering MRI, PET, SPECT, fMRI, and ESI (or magnetic source imaging) provides information to estimate the epileptogenic zone and can help optimize surgical results.
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13

Deacon, Terrence W. Symbolic Species: The Co-Evolution of Language and the Brain. Penguin Books, Limited, 1998.

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14

Deacon, Terrence W. Symbolic Species: The Co-Evolution of Language and the Brain. Norton & Company, Incorporated, W. W., 1998.

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15

Deacon, Terrence W. The Symbolic Species: The Co-Evolution of Language and the Brain. W. W. Norton & Company, 1998.

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16

Schechter, Elizabeth. Self and Other in the Split-Brain Subject. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0007.

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This chapter concerns self-consciousness in split-brain subjects. I argue, first, that R and L are both capable of thinking I-thoughts: thoughts containing the mental or conceptual analogue of the English word “I.” On the other hand, R’s and L’s self-consciousness differs, in its operative dynamics, from self-consciousness in, say, my sister and me. First of all, neither R nor L recognizes the existence of a second thinker sharing its body. I call this lack of mutual recognition. Second, L seems to assume that its I-thoughts refer to S, and R seems to assume the same of its I-thoughts. I call this (subjective) co-identification as S. I then argue that lack of mutual recognition and co-identification as S are explained by the fact that R and L lack the capacity for self-distinction: neither can first-personally distinguish itself from the other.
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17

Baltes, Paul B., Frank Rösler, and Patricia A. Reuter-Lorenz. Lifespan Development and the Brain: The Perspective of Biocultural Co-Constructivism. Cambridge University Press, 2009.

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18

Deacon, Terrence W. The Symbolic Species: The Co-Evolution of Language and the Brain. W. W. Norton & Company, 1998.

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19

Lifespan Development and the Brain: The Perspective of Biocultural Co-Constructivism. Cambridge University Press, 2006.

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20

Reuter-Lorenz, Patricia Ann, Frank Ro¨sler, and Paul B. Baltes. Lifespan Development and the Brain: The Perspective of Biocultural Co-constructivism. Cambridge University Press, 2006.

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21

The symbolic species: The co-evolution of language and the brain. New York: W.W. Norton, 1997.

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22

Baltes, Paul B., Frank Rösler, and Patricia A. Reuter-Lorenz. Lifespan Development and the Brain: The Perspective of Biocultural Co-Constructivism. Cambridge University Press, 2006.

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23

Baltes, Paul B., Frank Rösler, and Patricia A. Reuter-Lorenz. Lifespan Development and the Brain: The Perspective of Biocultural Co-Constructivism. Cambridge University Press, 2002.

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24

Baltes, Paul B., Frank Rösler, and Patricia A. Reuter-Lorenz. Lifespan Development and the Brain: The Perspective of Biocultural Co-Constructivism. Cambridge University Press, 2006.

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25

Jay, Omolove. 400+ Math Puzzles & Logic Problems - The Ultimate Brain Teaser Math Puzzles Co. Createspace Independent Publishing Platform, 2017.

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26

Diamond, Pamela M. Traumatic brain injury. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199360574.003.0053.

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During the past decade, traumatic brain injury (TBI) has become a frequent topic in the media. It has been a decade of expanding awareness, increased research, and growing concern about TBI of all severity levels. Consistent with this increased attention, researchers and policymakers have made strides toward greater understanding of the risks of TBI, the scope and complexity of the symptom profiles seen after TBI, and the types of treatments that optimize recovery. Recent studies have confirmed a 50 to 60% prevalence of TBI among prisoners. Most have experienced multiple injuries and experienced their first TBI in their mid-teens. Routine screening for TBI is rarely done in these settings in spite of there being a number of tested instruments available. The cognitive deficits associated with mild to moderate TBI are often indistinguishable from those associated with many mental illnesses and substance abuse. Etiology is difficult to establish; nevertheless, the common symptom patterns often make adjustment to jail or prison difficult. Educational interventions designed to improve staff knowledge of the prevalence of TBI and frequent symptom patterns are important first steps. Training staff how to modify their behavior and facilitate communication with inmates expressing these symptoms may reduce episodes of misunderstanding and potential aggression. Similarly, current programming may be modified to accommodate the cognitive deficits suffered by inmates with TBI as well as other disorders. This chapter reviews the prevalence of TBI in correctional settings, its impact on co-occurring mental illness and substance use, and opportunities to recognize, intervene, and treat patients with TBI.
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27

Schechter, Elizabeth. How Many Minds? Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0004.

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The previous chapters argued that within a split-brain subject there are two subjects of conscious experience and intentional agents, R and L. This chapter explains who these two thinking beings are and how it is possible for two thinkers to be co-embodied. The basis of the 2-thinkers claim is, naturally, that R and L think, feel, decide, and so on, independently of each other. Of course, this does not mean that they do not causally interact; since they are co-embodied, they interact all the time. What split-brain experiments show, however, is that R’s mental activities interact with L’s largely only indirectly: one of them acts or reacts in some way, and the other senses or perceives this re/action. Mental activities are causal activities whose psychological kinds are defined by their powers to interact directly. Thus the thinking things in the split-brain case are R and L, and only derivatively S.
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28

Mildred Carolyn White 1898- Brann. Descendants of Nicholas Brann; a Revolutionary War Soldier of Westmourland [sic] Co. , Va. Creative Media Partners, LLC, 2021.

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29

Schechter, Elizabeth. Bodies and Being One. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198809654.003.0006.

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This chapter concerns the relationship between the split-brain case and the non-split case. In the first half of the chapter, I consider arguments to the effect that if split-brain subjects have two minds apiece, then so do non-split subjects. Sometimes these arguments have taken the form of a reductio against the 2-thinkers claim for split-brain subjects. These arguments do not work: that a split-brain subject has two minds does not mean that I have two minds, although it does mean that I could. The second half of the chapter offers my own proposal for the respect in which R’s and L’s co-embodiment as one animal, S, makes a split-brain subject one of us: I argue that S must be the single object of both R’s and L’s implicit bodily self-awareness.
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30

Számadó, Szabolcs, and Eörs Szathmáry. Evolutionary biological foundations of the origin of language: the co‐evolution of language and brain. Oxford University Press, 2011. http://dx.doi.org/10.1093/oxfordhb/9780199541119.013.0014.

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31

Art, Publishing. My Brain Has Too Many Tabs Open: Blank Lined Journal Co-Worker Notebook Sarcastic Quotes. Independently Published, 2021.

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32

Sharma, Hari Shanker, Aruna Sharma, and Lars Wiklund. Nanowired Delivery of Drugs and Antibodies for Neuroprotection in Brain Diseases with Co-Morbidity Factors. Elsevier Science & Technology Books, 2023.

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33

Art, Publishing. My Brain Has Too Many Tabs Open: Blank Lined Journal Co-Worker Notebook Sarcastic Quotes. Independently Published, 2021.

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34

Nanowired Delivery of Drugs and Antibodies for Neuroprotection in Brain Diseases with Co-Morbidity Factors. Elsevier Science & Technology, 2023.

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35

Couples Bucket Listbook : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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36

Pangarkar, Sanjog S. Pain and Addiction in Patients with Traumatic Brain Injury (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190265366.003.0027.

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Distinct from Chapter 24, on co-occurring psychiatric disorders, this chapter addresses common physical comorbidities that give rise to chronic pain and are notorious for associated substance use disorders. The concept of “pseudo-addiction” is explored as one of several contributors to common misperceptions of the analgesic needs of such patients. Examples of entities discussed are chronic low back pain, sleep apnea, chronic pancreatitis, cirrhosis, and HIV infection or AIDS-related pain. While not intrinsically painful, sleep apnea merits inclusion as it arises in conjunction with sedative-hypnotic, opioid, or nicotine use. Cirrhosis likewise creates obstacles to successful pain or addiction management resulting from altered metabolism of medications and enhanced susceptibility to potentially lethal syndromes (hepato-renal syndrome, gastric hemorrhage, etc.). The management of neuropathic pain in HIV infection (Chapter 15) is amplified here.
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37

Adults Floral Coloring Book : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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38

The Symbolic Species: The Co-evolution of Language and the Human Brain (Allen Lane Science S.). Allen Lane, 1997.

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39

"CO₂-O₂ interactions in extension of tolerance to acute hypoxia": Final report. [Washington, DC]: National Aeronautics and Space Administration, 1995.

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40

Horse Coloring Book for Adults : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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41

Inspirational Coloring Book for Adults : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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42

Butterfly Coloring Book for Adults : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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43

Fashion Coloring Book for Adults : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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44

Gallagher, Shaun. Perception without Inferences. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198794325.003.0006.

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This chapter examines inference models of perception, including predictive coding models, and offers an enactivist alternative. It explores how the enactivist approach can respond to issues related to cognitive penetration and the effects of culture on perception. The enactivist view appeals to an account of the co-variant coupling of brain–body–environment, structured by the physical aspects of neuronal processes, bodily movements, affects, anatomy and function, and environmental regularities. Changes in any of these factors mean that perception changes. Processes involved in neural plasticity (where the brain is ‘set up to be set off’), and metaplasticity, where material and cultural practices define environmental regularities, mitigate the need to think that subpersonal perceptual processes are inferential.
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45

Noël, Marie-Pascale. When Number Processing and Calculation is Not Your Cup of Tea. Edited by Roi Cohen Kadosh and Ann Dowker. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199642342.013.62.

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This section of this volume deals with the study of numerical impairment occurring either after brain damage (i.e., acquired acalculia) or during development without any known brain damage (i.e., dyscalculia). The chapters in this section will report the research aiming at characterizing those difficulties. The study of atypical number processing and calculation in acalculia has contributed importantly to the understanding of how our brain is structured to process number and to calculate. The study of dyscalculia has shed light on the numerical bases for arithmetic learning. This research has also helped us in determining how other cognitive functions such as working memory, visuospatial processing, or phonological awareness have an impact on numerical cognition. These relations between different cognitive domains could partly explain the co-morbidities that are often observed in developmental disorders. Finally, this section also reviews the few attempts that have been made to enhance those numerical capacities.
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46

Nanowired Delivery of Drugs and Antibodies for Neuroprotection in Brain Diseases with Co-Morbidity Factors Part B. Elsevier Science & Technology Books, 2023.

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47

Co-Planar Stereotaxic Atlas of the Human Brain: 3-Dimensional Proportional System : An Approach to Cerebral Imaging. Thieme Medical Publishers, 1988.

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48

Nanowired Delivery of Drugs and Antibodies for Neuroprotection in Brain Diseases with Co-Morbidity Factors Part B. Elsevier Science & Technology, 2023.

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49

Sugar Skull Coloring Book for Adults : Brain Activities and Coloring Book for Brain Health with Fun and Relaxing: Gift for Men Women Colleague Co-Workers. Independently Published, 2021.

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50

Gaitanis, John, Phillip L. Pearl, and Howard Goodkin. The EEG in Degenerative Disorders of the Central Nervous System. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0013.

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Nervous system alterations can occur at any stage of prenatal or postnatal development. Any of these derangements, whether environmental or genetic, will affect electrical transmission, causing electroencephalogram (EEG) alteration and possibly epilepsy. Genetic insults may be multisystemic (for example, neurocutaneous syndromes) or affect only the brain. Gene mutations account for inborn errors of metabolism, channelopathies, brain malformations, and impaired synaptogenesis. Inborn errors of metabolism cause seizures and EEG abnormalities through a variety of mechanisms, including disrupted energy metabolism (mitochondrial disorders, glucose transporter defect), neuronal toxicity (amino and organic acidopathies), impaired neuronal function (lysosomal and peroxisomal disorders), alteration of neurotransmitter systems (nonketotic hyperglycinemia), and vitamin and co-factor dependency (pyridoxine-dependent seizures). Environmental causes of perinatal brain injury often result in motor or intellectual impairment (cerebral palsy). Multiple proposed etiologies exist for autism, many focusing on synaptic development. This chapter reviews the EEG findings associated with this myriad of pathologies occurring in childhood.
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