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Books on the topic 'Brain lipids'

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Author: Grafiati

Published: 4 September 2022

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1

Leray, Claude. Dietary Lipids for Healthy Brain Function. Other titles: Ces lipides qui stimulent notre cerveau. English Description: Boca Raton : Taylor & Francis, 2017.: CRC Press, 2017. http://dx.doi.org/10.1201/b21987.

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2

Ross Conference on Pediatric Research (103rd 1992 Adelaide). Lipids, learning, and the brain: Fats in infant formulas. Columbus, Ohio: Ross Laboratories, 1993.

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3

Farooqui, Akhlaq A. Lipid mediators and their metabolism in the brain. New York: Springer, 2011.

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4

Farooqui, Akhlaq A., Tahira Farooqui, and Lloyd A. Horrocks. Metabolism and Functions of Bioactive Ether Lipids in the Brain. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-77401-5.

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5

Tahira, Farooqui, and Horrocks Lloyd A, eds. Metabolism and functions of bioactive ether lipids in the brain. New York: Springer, 2008.

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6

Lajtha, Abel. Handbook of Neurochemistry and Molecular Neurobiology: Neural Lipids. Boston, MA: Springer US, 2009.

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7

Jumpsen, Jacqueline. Brain development: Relationship to dietary lipid and lipid metabolism. Champaign, Ill: AOCS Press, 1995.

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8

Stephens, L. The involvement of inositol lipids in the action of transmitters in the brain. Birmingham: University of Birmingham, 1985.

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9

McCleary, Larry. Feed your brain, lose your belly: A brain surgeon reveals the weight-loss secrets of the brain-belly connection. 2nd ed. Austin, Tex: Greenleaf Book Group Press, 2011.

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10

A, Horrocks Lloyd, ed. Glycerophospholipids in the brain: Phospholipases A2 in neurological disorders. New York, N.Y: Springer, 2007.

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11

Ross Conference on Pediatric Research (103rd 1992 Adelaide, S. Aust.). Lipids, learning, and the brain: Fats in infant formulas : report of the 103rd Ross Conference on Pediatric Research. Edited by Dobbing John and Benson John 1942-. Columbus, Ohio: Ross Laboratories, 1993.

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12

Melis, Miriam, ed. Endocannabinoids and Lipid Mediators in Brain Functions. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57371-7.

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13

Farooqui, Akhlaq A. Lipid Mediators and Their Metabolism in the Brain. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-9940-5.

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14

Augustin, Livia S. A. The effect of wheat bran particle size and wheat protein on serum lipids and colonic health. Ottawa: National Library of Canada, 1998.

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15

Albi, Elisabetta, Alice Vladimirovna Alessenko, Maria Dolores Ledesma, and Fanny M. Elahi, eds. Lipids in the Brain. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-986-1.

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16

Dietary Lipids for Healthy Brain Function. Taylor & Francis Group, 2017.

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17

Elliott, Katherine, and Julie Knight. Lipids, Malnutrition and the Developing Brain. Wiley & Sons, Incorporated, John, 2009.

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18

Roy, Skinner E., ed. Brain lipids and disorders in biological psychiatry. Amsterdam: Elsevier, 2002.

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19

Brain Lipids and Disorders in Biological Psychiatry. Elsevier, 2002. http://dx.doi.org/10.1016/s0167-7306(00)x0001-4.

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20

Brain Lipids in Synaptic Function and Neurological Disease. Elsevier, 2015. http://dx.doi.org/10.1016/c2013-0-09847-7.

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21

Fantini, Jacques, and Nouara Yahi. Brain Lipids in Synaptic Function and Neurological Disease: Clues to Innovative Therapeutic Strategies for Brain Disorders. Elsevier Science & Technology Books, 2015.

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22

Farooqui, Akhlaq A. Lipid Mediators and Their Metabolism in the Brain. Springer, 2011.

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23

Skinner, E. R. Brain Lipids and Disorders in Biological Psychiatry (New Comprehensive Biochemistry). Elsevier Science, 2002.

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24

Dobbing, John. Developing Brain Behaviour: The Role of Lipids in Infant Formula. Academic Press, 1997.

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25

Horrocks, Lloyd A., Tahira Farooqui, and Akhlaq A. A. Farooqui. Metabolism and Functions of Bioactive Ether Lipids in the Brain. Springer, 2010.

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26

Dobbing, John. Developing Brain Behaviour: The Role of Lipids in Infant Formula. Academic Press, 1997.

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27

John, Dobbing, ed. Developing brain and behaviour: The role of lipids in infant formula. San Diego: Academic Press, 1997.

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28

G, Bazán Nicholás, ed. Lipid mediators in ischemic brain damage and experimental epilepsy. Basel: Karger, 1990.

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29

McCleary, Larry. Feed Your Brain, Lose Your Belly: A Brain Surgeon Reveals the Weight-Loss Secrets of the Brain-Belly Connection. Greenleaf Book Group, 2011.

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30

Farooqui, Akhlaq, and L. A. Horrocks. Glycerophospholipids in brain: Phospholipase A2 in neurological disorders. Springer, 2006.

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31

Horrocks, Lloyd A., and Akhlaq A. Farooqui. Glycerophospholipids in the Brain: Phospholipases A2 in Neurological Disorders. Springer, 2014.

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32

Feed Your Brain Lose Your Belly. Teocalli, 2010.

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33

E, Vance Dennis, and Vance Jean E, eds. Biochemistry of lipids, lipoproteins, and membranes. Amsterdam: Elsevier, 1991.

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34

Chang, Michael Colin Joseph. The effect of temperature acclimation on the composition and distribution of lipids in mitochondrial membranes from goldfish brain. 1989.

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35

(Editor), Judith J. Wurtman, ed. Nutrition and the Brain. Raven Pr, 1990.

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36

Patterson, Marc C. Congenital Disorders of Glycosylation. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0066.

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Congenital disorders of glycosylation (CDG) comprise a family of multisystem diseases in which N- and O-linked glycosylation and glypiation of a variety of proteins and lipids is deficient. The hypoglycosylation of multiple glycoconjugates impairs normal development of the brain (and other organs), and is associated with both episodic and chronic organ dysfunction. Developmental disorders; seizures; strokelike episodes (and stroke); hearing and visual loss; peripheral neuropathy; coagulopathy; and immune, liver, endocrine, cardiac, and cutaneous manifestations may occur in varying combinations. Specific therapy is available for MPI-CDG and SLC35C2-CDG. Most forms of O-linked CDG affect muscle; these include congenital muscular dystrophies and limb girdle dystrophies.

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37

Melis, Miriam. Endocannabinoids and Lipid Mediators in Brain Functions. Springer, 2017.

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38

Farooqui, Akhlaq A. A. Lipid Mediators and Their Metabolism in the Brain. Springer, 2014.

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39

Arvanitis, Dina Nectaria. Protein and lipid assemblies in bovine brain myelin. 2004.

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40

Abdul Rohim, Gufran Ali Ibrahim, Buha Aritonang, Syihaabul Hudaa, Ahmad Bahtiar, Indrya Mulyaningsih, Mukhamdanah, et al. PROSIDING SEMINAR NASIONAL BAHASA DAN SASTRA KEBINEKAAN DALAM PERSPEKTIF BAHASA DAN SASTRA. Penerbit BRIN, 2022. http://dx.doi.org/10.55981/brin.305.

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Fenomena makin maraknya elemen destruktif yang berpotensi merusak kehidupan kebinekaan di tengah arus globalisasi dewasa ini seharusnya terus diimbangi dengan upaya untuk merawat tali kebinekaan. Bahasa dan Sastra ditengarai merupakan salah satu sarana yang mampu mewujudkan tugas dan tanggung jawab tersebut. Berbagai penelitian terkait bahasa dan sastra sebagai perspektif kebinekaan sehubungan dengan hal tersebut tertuang dalam prosiding ini. Prosiding ini memuat karya tulis dari berbagai hasil penelitian mengenai kebahasaan dan kesastraan yang berasal dari para peneliti di lingkungan Badan Pengembangan dan Pembinaan Bahasa, dosen dan mahasiswa beberapa perguruan tinggi, dan peneliti LIPI. Selain bertujuan untuk meningkatkan kesadaran kolektif terhadap realitas kebinekaan pada bidang bahasa dan sastra, prosiding ini diharapkan mampu menjadi referensi dalam pengembangan penelitian di masa mendatang, serta dapat dijadikan sebagai bahan pemikiran dan pemahaman terkait persoalan kebinekaan melalui khazanah bahasa dan sastra Indonesia.

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41

PS (PhosphatidylSerine) Nature's Brain Booster; a vital lipid nutrient for memory mood and stress. Science and Ingredients, Inc., 2005.

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42

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Choline in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0014.

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Choline is required for the structural integrity of cell membranes and is involved in methyl-group metabolism, neurotransmission, transmembrane signalling, and lipid and cholesterol transport and metabolism. Choline is critical during fetal and neonatal life to ensure optimal brain and cognitive development. There is an intersection of the pathways of choline, folate, and vitamin B12 in the formation of methionine from homocysteine. Maternal peri-conceptional deficiency for choline, like folate, is associated with an increased risk of neural tube defects in the offspring. It is recommended that pregnant women do not restrict fat severely from their diets, as choline is derived from the lipid content of food. Strict vegetarian or vegan diets may be low in choline. The high secretion rate of choline into breast milk means that lactating women have a high demand, and multivitamins containing choline may be helpful for both pregnancy and breastfeeding.

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43

Harney, Jacob P., Kathryn Gudsnuk, Ami Patel, Anantha R. Vellipuram, Sathyajit Bandaru, and David Butler. Endocrine and Reproductive Effects of Ketogenic Diets. Edited by Detlev Boison. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0025.

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This chapter reviews endocrine, behavioral, and reproductive outcomes of the ketogenic diet (KD) in rodent mouse and rat models. KD treatment can result in conditions seen in metabolic syndrome, including dyslipidemia and inflammation. Females raised on low protein KDs will experience delayed puberty onset. Results presented suggest an increase in ketones and a decrease in spatial memory as percent protein drops. Postpubertal female pups fed KD experienced similar cognitive decline to KD-fed dams, despite consuming only normal rodent chow since weaning. This finding is consistent with the development of anatomical differences in the brains of pups from KD-fed versus rodent chow–fed controls. A thorough evaluation of the effects of different lipid and protein profiles (amount and type) will be necessary if KDs are going to be safe and effective long-term therapies for both sexes and in neurological disorders besides pediatric intractable epilepsy.

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44

Burton, Derek, and Margaret Burton. Gas exchange. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0006.

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Oxygen intake for respiration, also carbon dioxide and, generally, ammonia elimination takes place across gas-exchange surfaces, usually the gills in fish. Water flows across gills, separated by the pharyngeal gill clefts, and supported by gill arches, and which possess highly folded surfaces covered by a very thin epithelium. Blood flow and water flow are separated only by the epithelium with a ‘countercurrent’ gas exchange between the two. A respiratory centre in the hind-brain is a respiratory rhythm pacemaker for the oral and pharyngeal ventilation movements creating water flow across the gills, although ‘ram ventilation’ occurs without such movements. The oxygen and carbon dioxide-carrying capacity of blood is increased considerably by temporary attachment to haemoglobin pigment in the erythrocytes. Some fish are air breathing, using lungs, swim bladder, skin or lips for gaseous exchange. Hypoxia, hypercapnia, supersaturation and high water temperatures present problems for fish respiration, which are discussed.

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