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Relevant bibliographies by topics / Brain damage/social cognition

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Academic literature on the topic 'Brain damage/social cognition'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Brain damage/social cognition"

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Batista, Sonia, Carolina Alves, Otília C. d’Almeida, Ana Afonso, Ricardo Félix-Morais, João Pereira, Carmo Macário, et al. "Disconnection as a mechanism for social cognition impairment in multiple sclerosis." Neurology 89, no. 1 (May 31, 2017): 38–45. http://dx.doi.org/10.1212/wnl.0000000000004060.

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Objective:To assess the contribution of microstructural normal-appearing white matter (NAWM) damage to social cognition impairment, specifically in the theory of mind (ToM), in multiple sclerosis (MS).Methods:We enrolled consecutively 60 patients with MS and 60 healthy controls (HC) matched on age, sex, and education level. All participants underwent ToM testing (Eyes Test, Videos Test) and 3T brain MRI including conventional and diffusion tensor imaging sequences. Tract-based spatial statistics (TBSS) were applied for whole-brain voxel-wise analysis of fractional anisotropy (FA) and mean diffusivity (MD) on NAWM.Results:Patients with MS performed worse on both tasks of ToM compared to HC (Eyes Test 58.7 ± 13.8 vs 81.9 ± 10.4, p < 0.001, Hedges g −1.886; Videos Test 75.3 ± 9.3 vs 88.1 ± 7.1, p < 0.001, Hedges g −1.537). Performance on ToM tests was correlated with higher values of FA and lower values of MD across widespread white matter tracts. The largest effects (≥90% of voxels with statistical significance) for the Eyes Test were body and genu of corpus callosum, fornix, tapetum, uncinate fasciculus, and left inferior cerebellar peduncle, and for the Videos Test genu and splenium of corpus callosum, fornix, uncinate fasciculus, left tapetum, and right superior fronto-occipital fasciculus.Conclusions:These results indicate that a diffuse pattern of NAWM damage in MS contributes to social cognition impairment in the ToM domain, probably due to a mechanism of disconnection within the social brain network. Gray matter pathology is also expected to have an important role; thus further research is required to clarify the neural basis of social cognition impairment in MS.

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Adolphs, Ralph, Simon Baron-Cohen, and Daniel Tranel. "Impaired Recognition of Social Emotions following Amygdala Damage." Journal of Cognitive Neuroscience 14, no. 8 (November 1, 2002): 1264–74. http://dx.doi.org/10.1162/089892902760807258.

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Lesion, functional imaging, and single-unit studies in human and nonhuman animals have demonstrated a role for the amygdala in processing stimuli with emotional and social significance. We investigated the recognition of a wide variety of facial expressions, including basic emotions (e.g., happiness, anger) and social emotions (e.g., guilt, admiration, flirtatiousness). Prior findings with a standardized set of stimuli indicated that recognition of social emotions can be signaled by the eye region of the face and is disproportionately impaired in autism (Baron-Cohen, Wheelwright, & Jolliffe, 1997). To test the hypothesis that the recognition of social emotions depends on the amygdala, we administered the same stimuli to 30 subjects with unilateral amygdala damage (16 left, 14 right), 2 with bilateral amygdala damage, 47 brain-damaged controls, and 19 normal controls. Compared with controls, subjects with unilateral or bilateral amygdala damage were impaired when recognizing social emotions; moreover, they were more impaired in recognition of social emotions than in recognition of basic emotions, and, like previously described patients with autism, they were impaired also when asked to recognize social emotions from the eye region of the face alone. The findings suggest that the human amygdala is relatively specialized to process stimuli with complex social significance. The results also provide further support for the idea that some of the impairments in social cognition seen in patients with autism may result from dysfunction of the amygdala.

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Henry, Audrey, Ayman Tourbah, Marie-Pierre Chaunu, Lucien Rumbach, Michèle Montreuil, and Serge Bakchine. "Social Cognition Impairments in Relapsing-Remitting Multiple Sclerosis." Journal of the International Neuropsychological Society 17, no. 6 (October 7, 2011): 1122–31. http://dx.doi.org/10.1017/s1355617711001147.

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AbstractTheory of Mind (ToM) is the ability to attribute independent mental states to self and others to explain and predict behavior. Impairment of ToM is well established in developmental pathologies. In neurological populations, investigation of ToM is still rare but data suggest that ToM impairment could contribute to behavioral and social disturbances. In addition to neurological signs, multiple sclerosis (MS) presents with disorders of cognition and behavior directly related to brain damage. The aim of this study was to assess ToM abilities and recognition of facial emotional expression in adults with MS. We compared 64 patients with relapsing MS and 30 matched healthy controls on three levels of ToM tasks, a facial emotion recognition task, and a neuropsychological assessment. MS patients performed significantly worse than controls in emotion recognition and all ToM tasks (p < .02). These deficits were not correlated with demographic variables or neuropsychological test performance. These findings underscore the importance of assessing ToM and facial recognition in MS, as dysfunction in these areas may impact upon social interaction and, thus, impair quality of life for both patients with MS and their families. (JINS, 2011, 17, 1122–1131)

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Hoskinson, Kristen R., Erin D. Bigler, Tracy J. Abildskov, Maureen Dennis, H. Gerry Taylor, Kenneth Rubin, Cynthia A. Gerhardt, Kathryn Vannatta, Terry Stancin, and Keith Owen Yeates. "The mentalizing network and theory of mind mediate adjustment after childhood traumatic brain injury." Social Cognitive and Affective Neuroscience 14, no. 12 (December 2019): 1285–95. http://dx.doi.org/10.1093/scan/nsaa006.

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Abstract Childhood traumatic brain injury (TBI) affects over 600 000 children per year in the United States. Following TBI, children are vulnerable to deficits in psychosocial adjustment and neurocognition, including social cognition, which persist long-term. They are also susceptible to direct and secondary damage to related brain networks. In this study, we examine whether brain morphometry of the mentalizing network (MN) and theory of mind (ToM; one component of social cognition) mediates the effects of TBI on adjustment. Children with severe TBI (n = 15, Mage = 10.32), complicated mild/moderate TBI (n = 30, Mage = 10.81) and orthopedic injury (OI; n = 42, Mage = 10.65) completed measures of ToM and executive function and underwent MRI; parents rated children’s psychosocial adjustment. Children with severe TBI demonstrated reduced right-hemisphere MN volume, and poorer ToM, vs children with OI. Ordinary least-squares path analysis indicated that right-hemisphere MN volume and ToM mediated the association between severe TBI and adjustment. Parallel analyses substituting the central executive network and executive function were not significant, suggesting some model specificity. Children at greatest risk of poor adjustment after TBI could be identified based in part on neuroimaging of social brain networks and assessment of social cognition and thereby more effectively allocate limited intervention resources.

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Karafin, Matthew S., Daniel Tranel, and Ralph Adolphs. "Dominance Attributions Following Damage to the Ventromedial Prefrontal Cortex." Journal of Cognitive Neuroscience 16, no. 10 (December 2004): 1796–804. http://dx.doi.org/10.1162/0898929042947856.

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Damage to the human ventromedial prefrontal cortex (VM) can result in dramatic and maladaptive changes in social behavior despite preservation of most other cognitive abilities. One important aspect of social cognition is the ability to detect social dominance, a process of attributing from particular social signals another person's relative standing in the social world. To test the role of the VM in making attributions of social dominance, we designed two experiments: one requiring dominance judgments from static pictures of faces, the second requiring dominance judgments from film clips. We tested three demographically matched groups of subjects: subjects with focal lesions in the VM (n = 15), brain-damaged comparison subjects with lesions excluding the VM (n = 11), and a reference group of normal individuals with no history of neurological disease (n = 32). Contrary to our expectation, we found that subjects with VM lesions gave dominance judgments on both tasks that did not differ significantly from those given by the other groups. Despite their grossly normal performance, however, subjects with VM lesions showed more subtle impairments specifically when judging static faces: They were less discriminative in their dominance judgments, and did not appear to make normal use of gender and age of the faces in forming their judgments. The findings suggest that, in the laboratory tasks we used, damage to the VM does not necessarily impair judgments of social dominance, although it appears to result in alterations in strategy that might translate into behavioral impairments in real life.

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Butti, Niccolò, Rosario Montirosso, Lorenzo Giusti, Luigi Piccinini, Renato Borgatti, and Cosimo Urgesi. "Early Brain Damage Affects Body Schema and Person Perception Abilities in Children and Adolescents with Spastic Diplegia." Neural Plasticity 2019 (August 18, 2019): 1–17. http://dx.doi.org/10.1155/2019/1678984.

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Early brain damage leading to cerebral palsy is associated to core motor impairments and also affects cognitive and social abilities. In particular, previous studies have documented specific alterations of perceptual body processing and motor cognition that are associated to unilateral motor deficits in hemiplegic patients. However, little is known about spastic diplegia (SpD), which is characterized by motorial deficits involving both sides of the body and is often associated to visuospatial, attentional, and social perception impairments. Here, we compared the performance of a sample of 30 children and adolescents with SpD (aged 7-18 years) and of a group of age-matched controls with typical development (TD) at two different tasks tapping on body representations. In the first task, we tested visual and motor imagery abilities as assessed, respectively, by the object-based mental rotation of letters and by the first-person transformations for whole-body stimuli. In the second task, we administered an inversion effect/composite illusion task to evaluate the use of configural/holistic processing of others’ body. Additionally, we assessed social perception abilities in the SpD sample using the NEPSY-II battery. In line with previously reported visuospatial deficits, a general mental imagery impairment was found in SpD patients when they were engaged in both object-centered and first-person mental transformations. Nevertheless, a specific deficit in operating an own-body transformation emerged. As concerns body perception, while more basic configural processing (i.e., inversion effect) was spared, no evidence for holistic (i.e., composite illusion) body processing was found in the SpD group. NEPSY-II assessment revealed that SpD children were impaired in both the theory of mind and affect recognition subtests. Overall, these findings suggested that early brain lesions and biased embodied experience could affect higher-level motor cognition and perceptual body processing, thus pointing to a strict link between motor deficits, body schema alterations, and person processing difficulties.

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Shamay-Tsoory, S. G., R. Tomer, B. D. Berger, and J. Aharon-Peretz. "Characterization of Empathy Deficits following Prefrontal Brain Damage: The Role of the Right Ventromedial Prefrontal Cortex." Journal of Cognitive Neuroscience 15, no. 3 (April 1, 2003): 324–37. http://dx.doi.org/10.1162/089892903321593063.

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Impaired empathic response has been described in patients following brain injury, suggesting that empathy may be a fundamental aspect of the social behavior disturbed by brain damage. However, the neuroanatomical basis of impaired empathy has not been studied in detail. The empathic response of patients with localized lesions in the prefrontal cortex (n = 25) was compared to responses of patients with posterior (n = 17) and healthy control subjects (n = 19). To examine the cognitive processes that underlie the empathic ability, the relationships between empathy scores and the performance on tasks that assess processes of cognitive flexibility, affect recognition, and theory of mind (TOM) were also examined. Patients with prefrontal lesions, particularly when their damage included the ventromedial prefrontal cortex, were significantly impaired in empathy as compared to patients with posterior lesions and healthy controls. However, among patients with posterior lesions, those with damage to the right hemisphere were impaired, whereas those with left posterior lesions displayed empathy levels similar to healthy controls. Seven of nine patients with the most profound empathy deficit had a right ventromedial lesion. A differential pattern regarding the relationships between empathy and cognitive performance was also found: Whereas among patients with dorsolateral prefrontal damage empathy was related to cognitive flexibility but not to TOM and affect recognition, empathy scores in patients with ventromedial lesions were related to TOM but not to cognitive flexibility. Our findings suggest that prefrontal structures play an important part in a network mediating the empathic response and specifically that the right ventromedial cortex has a unique role in integrating cognition and affect to produce the empathic response.

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Corden, Ben, Hugo D. Critchley, David Skuse, and Raymond J. Dolan. "Fear Recognition Ability Predicts Differences in Social Cognitive and Neural Functioning in Men." Journal of Cognitive Neuroscience 18, no. 6 (June 2006): 889–97. http://dx.doi.org/10.1162/jocn.2006.18.6.889.

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By testing the facial fear-recognition ability of 341 men in the general population, we show that 8.8% have deficits akin to those seen with acquired amygdala damage. Using psychological tests and functional magnetic resonance imaging (fMRI) we tested the hypothesis that poor fear recognition would predict deficits in other domains of social cognition and, in response to socially relevant stimuli, abnormal activation in brain regions that putatively reflect engagement of the “social brain.” On tests of “theory of mind” ability, 25 “low fear scorers” (LFS) performed significantly worse than 25 age- and IQ-matched “normal (good) fear scorers” (NFS). In fMRI, we compared evoked activity during a gender judgement task to neutral faces portraying different head and eye gaze orientations in 12 NFS and 12 LFS subjects. Despite identical between-group accuracy in gender discrimination, LFS demonstrated significantly reduced activation in amygdala, fusiform gyrus, and anterior superior temporal cortices when viewing faces with direct versus averted gaze. In a functional connectivity analysis, NFS show enhanced connectivity between the amygdala and anterior temporal cortex in the context of direct gaze; this enhanced coupling is absent in LFS. We suggest that important individual differences in social cognitive skills are expressed within the healthy male population, which appear to have a basis in a compromised neural system that underpins social information processing.

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Place, Charlie. "Alcohol related brain damage – a case of neglect?" Advances in Dual Diagnosis 7, no. 3 (August 12, 2014): 129–36. http://dx.doi.org/10.1108/add-02-2014-0008.

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Purpose – The purpose of this paper is to argue that alcohol-related brain damage (ARBD) is a neglected problem. ARBD is a term that has begun to be used over the past decade to describe prolonged cognitive impairment caused by alcohol use, including Wernicke's encephalopathy and Korsakoff syndrome, alcohol dementia and alcohol-related brain injury. Design/methodology/approach – The paper provides an overview of ARBD describing the research around its prevalence and prognosis. There is a consensus in the literature that there is little research and a lack of awareness of this condition. The author uses case studies from his own experience working with people with ARBD to describe the difficulties in accessing appropriate assessment and care for this group, and suggests that they are often excluded in a way that is familiar from the experience of the person with “dual diagnosis”. Findings – Recommendations are made including raising awareness, improving screening for cognitive impairment and developing specialist services. Originality/value – ARBD appears to have been neglected in the “dual diagnosis” world and this paper attempts to address this, and so should be of interest to a wide range of professionals working with substance use, mental health, homelessness and social work.

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Koscik, Timothy R., and Daniel Tranel. "Abnormal Causal Attribution Leads to Advantageous Economic Decision-making: A Neuropsychological Approach." Journal of Cognitive Neuroscience 25, no. 8 (August 2013): 1372–82. http://dx.doi.org/10.1162/jocn_a_00398.

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People tend to assume that outcomes are caused by dispositional factors, for example, a person's constitution or personality, even when the actual cause is due to situational factors, for example, luck or coincidence. This is known as the “correspondence bias.” This tendency can lead normal, intelligent persons to make suboptimal decisions. Here, we used a neuropsychological approach to investigate the neural basis of the correspondence bias, by studying economic decision-making in patients with damage to the ventromedial pFC (vmPFC). Given the role of the vmPFC in social cognition, we predicted that vmPFC is necessary for the normal correspondence bias. In our experiment, consistent with expectations, healthy (n = 46) and brain-damaged (n = 30) comparison participants displayed the correspondence bias during economic decision-making and invested no differently when given dispositional or situational information. By contrast, vmPFC patients (n = 17) displayed a lack of correspondence bias and invested more when given dispositional than situational information. The results support the conclusion that vmPFC is critical for normal social inference and the correspondence bias. The findings help clarify the important (and sometimes disadvantageous) role of social inference in economic decision-making.

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Dissertations / Theses on the topic "Brain damage/social cognition"

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Peng, Catherine Yee-yuen. "Decoding facial expressions of emotion." Thesis, University of Oxford, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253287.

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Gupta, Rupa. "The effects of ventromedial prefrontal cortex damage on interpersonal coordination in social interaction." Diss., University of Iowa, 2012. https://ir.uiowa.edu/etd/2883.

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Conversation is a highly interactive and coordinated effort between interactants. For example, interactants often mimic the behaviors and speech of one another and coordinate the timing of behaviors, or interactional synchrony. Despite being affected in certain neurological and psychiatric disorders, the neural mechanisms underlying these processes are not understood. The goal of this study is to understand the role of the ventromedial prefrontal cortex (vmPFC), an area of the brain involved in social and emotional behavior, for interpersonal coordination, including mimicry and interactional synchrony. To test the role of the vmPFC for mimicry, normal comparison (NC), brain damaged comparison (BDC), and participants with vmPFC damage interacted in two sessions with a research assistant (RA) who was performing a target behavior (1st session: nodding, 2nd session: face touching). The amount of time the participants spent nodding or touching their face in each session was recorded. NC and BDC participants tended to mimic the partner and nodded slightly more in the session in which the RA was nodding, and touched their face slightly more in the session in which the RA was touching their face. In contrast, vmPFC patients showed no difference in their behaviors in either session, suggesting that they were not influenced by the partner's behaviors and did not mimic them. In a second experiment, all of the above participant groups had a naturalistic conversation with an unfamiliar interactional partner. The conversational data were analyzed for numerous aspects of interpersonal coordination, including convergence of number of words, words per turn and backchannels, reciprocity of self-disclosures, the use of questions, interactional synchrony, and a time series analysis of response latency and speech rate. The vmPFC participants performed consistently worse than NC participants on convergence of words and words per turn, self-disclosures and asking questions. All brain-damaged participants were impaired on aspects of interactional synchrony, and no conclusive results were found for the time series analysis of response latency and speech rate. This study provides support for the hypothesis that the vmPFC is important for interpersonal coordination as the vmPFC group differed significantly from the NC group on the majority of the analyses. The final goal of this study was to understand the effects of traumatic brain injury (TBI) on interpersonal coordination. TBI patients participated in all of the experiments described above and preliminary results showed that they also seemed to be impaired on the mimicry task, and they performed slightly worse than NC participants on many of the interpersonal coordination analyses of the conversational data. This suggests that TBI also does seem to affect certain aspects of interpersonal coordination.

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Wong, Pauline P. "Mathematical models of cognitive recovery and impairment profile after severe traumatic brain injury." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0003/NQ43457.pdf.

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McLaughlin, Karen A. "Initial investigation of a collaborative intervention model for individuals with brain injury and their families /." view abstract or download file of text, 2001. http://wwwlib.umi.com/cr/uoregon/fullcit?p3003997.

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Thesis (Ph. D.)--University of Oregon, 2001.
Typescript. Includes vita and abstract. Includes bibliographical references (leaves 111-115). Also available for download via the World Wide Web; free to University of Oregon users.

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Kastuk, Donald John. "Social skills training for the traumatic brain injured." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0002/NQ43434.pdf.

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Ip, Ka-yan. "Social cognition deficits in frontal lesion patients." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43224325.

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Sundström, Anna. "Mild head injury : relation to cognition, dementia, fatigue & genetics /." Umeå : Department of Psychology, Umeå University, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-852.

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Ehlhardt, Laurie Anne. "E-steps : evaluation of an instructional sequence for persons with impaired memory and executive functions /." view abstract or download file of text, 2003. http://wwwlib.umi.com/cr/uoregon/fullcit?p3095242.

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Thesis (Ph. D.)--University of Oregon, 2003.
Typescript. Includes vita and abstract. Includes bibliographical references (leaves 124-128). Also available for download via the World Wide Web; free to University of Oregon users.

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Preece, Megan H. W. "The contribution of pre-existing depression to the acute cognitive sequelae of mild traumatic brain injury /." [St. Lucia, Qld.], 2006. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19733.pdf.

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Laske, Kate M. "Correlation of assessment measures in a rehabilitation program for individuals with traumatic brain injury." Oxford, Ohio : Miami University, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=miami1082685973.

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Books on the topic "Brain damage/social cognition"

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Dikengil, Angela Tipton. Building functional social skills: Group activities for adults. Tucson, Ariz: Therapy Skill Builders, 1992.

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Hartley, Leila L. Cognitive-communicative abilities following brain injury: A functional approach. San Diego, Calif: Singular Pub. Group, 1995.

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S, Hecht Jeffrey, and Calub Connie J, eds. Educating families of the head injured: A guide to medical, cognitive, and social issues. Gaithersburg, Md: Aspen Publishers, 1991.

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Right hemisphere damage: Disorders of communication and cognition. San Diego: Singular Pub., 1999.

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J, Herrmann Douglas, ed. Retraining cognition: Techniques and applications. 3rd ed. Austin, Tex: PRO-ED, Inc., 2010.

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J, Herrmann Douglas, ed. Retraining cognition: Techniques and applications. Gaithersburg, Md: Aspen Publishers, 1996.

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Parenté, Rick. Retraining cognition: Techniques and applications. 3rd ed. Austin, Tex: PRO-ED, Inc., 2010.

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Stuss, Donald T. Cognitive neurorehabilitation: Evidence and application. 2nd ed. Cambridge: Cambridge University Press, 2008.

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Anderson, Kathleen. Lessons for the right brain. Austin, Texas: Pro-Ed, 1985.

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Anderson, Kathleen. Lessons for the right brain. Austin, Tex: Pro-Ed, 1985.

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Book chapters on the topic "Brain damage/social cognition"

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Berndt, Rita Sloan. "Brain/Cognition Relationships: Interpreting the Effects of Focal Brain Damage." In Neurodevelopment, Aging and Cognition, 272–91. Boston, MA: Birkhäuser Boston, 1992. http://dx.doi.org/10.1007/978-1-4684-6805-2_16.

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Bijleveld, H., and A. M. Simon. "A Case of Acquired Stuttering Following Brain Damage." In Neuropsychology and Cognition, 163–69. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5776-6_10.

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Kenemans, Leon, and Nick Ramsey. "Social Cognition and Communication." In Psychology in the Brain, 244–62. London: Macmillan Education UK, 2013. http://dx.doi.org/10.1007/978-1-137-29614-6_11.

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Schulte-Rüther, Martin, and Ellen Greimel. "Brain Networks Supporting Empathy." In From DNA to Social Cognition, 47–61. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118101803.ch3.

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Shultz, Susanne, and Robin IM Dunbar. "Social Cognition and Cortical Function." In Action, Perception and the Brain, 43–67. London: Palgrave Macmillan UK, 2012. http://dx.doi.org/10.1057/9780230360792_3.

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Van Borsel, J., and V. Cappaert. "Aggravation or Recurrence of Pre-Existing Stuttering Following Brain Damage Suffered in Adulthood." In Neuropsychology and Cognition, 171–84. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5776-6_11.

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Kumfor, Fiona, and Skye McDonald. "Research methodologies, brain correlates, cross-cultural perspectives." In Clinical Disorders of Social Cognition, 52–80. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003027034-2.

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Río, J. A. Del, F. de Castro, and E. Soriano. "Axon Guidance and Repulsion. The Molecular Code of Social Life in the Brain." In Brain Damage and Repair, 165–79. Dordrecht: Springer Netherlands, 2004. http://dx.doi.org/10.1007/1-4020-2541-6_11.

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Berger, Andrea. "Self-regulation in social contexts." In Self-regulation: Brain, cognition, and development., 91–103. Washington: American Psychological Association, 2011. http://dx.doi.org/10.1037/12327-005.

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Wearne, Travis, Michelle Kelly, and Skye McDonald. "Disorders of social cognition in adults with acquired brain injury." In Clinical Disorders of Social Cognition, 145–77. London: Routledge, 2021. http://dx.doi.org/10.4324/9781003027034-5.

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Conference papers on the topic "Brain damage/social cognition"

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Zhu, Banghe, and Anuradha Godavarty. "Brain activation and connectivity of social cognition using diffuse optical imaging." In SPIE BiOS: Biomedical Optics, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, Reza S. Malek, Brian J. Wong, Justus F. R. Ilgner, Kenton W. Gregory, et al. SPIE, 2009. http://dx.doi.org/10.1117/12.809942.

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Kaczmarek, Bożydar L. J. "The embodied brain: cultural aspects of cognition." In 2nd International Neuropsychological Summer School named after A. R. Luria “The World After the Pandemic: Challenges and Prospects for Neuroscience”. Ural University Press, 2020. http://dx.doi.org/10.15826/b978-5-7996-3073-7.15.

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Our thinking is grounded in our sensory, motor, affective, and interpersonal experience. Recent psychological studies confirmed that our cognition is not only embodied but also embedded since it arises from interactions with its social and cultural environments, which makes it possible to create image schemas and conceptual metaphors. Those schemas facilitate acting in everyday, routine situations, but make it difficult to depart from them since they are frames that limit our ability to see the alternatives. They are intricately linked to our world view and, therefore, resistant to changes because the latter threaten the feeling of security. This paper is aimed at evaluating people’s ability to change the existing schema. In the study, participants were asked to create a completely new story based on two well.known stories in which they had previously inserted the missing words. It was found that most participants exhibited considerable difficulties in departing from the formerly established schemas. Moreover, the emotionally loaded story proved to be more difficult to change.

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Matejkova, Andrea. "COORDINATED REHABILITATION FROM PATIENT'S PERSPECTIVE AFTER BRAIN DAMAGE." In 5th SGEM International Multidisciplinary Scientific Conferences on SOCIAL SCIENCES and ARTS SGEM2018. STEF92 Technology, 2018. http://dx.doi.org/10.5593/sgemsocial2018h/31/s13.076.

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Bartova, Marie. "NEEDS OF FAMILIES OF PATIENTS AFTER BRAIN DAMAGE." In 5th SGEM International Multidisciplinary Scientific Conferences on SOCIAL SCIENCES and ARTS SGEM2018. STEF92 Technology, 2018. http://dx.doi.org/10.5593/sgemsocial2018h/31/s13.085.

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Vackova, Jitka. "COORDINATED REHABILITATION OF PATIENTS AFTER BRAIN DAMAGE - FIRST STUDY RESULTS." In 4th International Multidisciplinary Scientific Conference on Social Sciences and Arts SGEM2017. Stef92 Technology, 2017. http://dx.doi.org/10.5593/sgemsocial2017/hb31/s13.055.

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Mandatova, Katerina. "POSITIVE AND NEGATIVE ASPECTS OF SOCIAL WORK WITH PATIENTS AFTER BRAIN DAMAGE FROM THE PERSPECTIVE OF SOCIAL WORKER." In 5th SGEM International Multidisciplinary Scientific Conferences on SOCIAL SCIENCES and ARTS SGEM2018. STEF92 Technology, 2018. http://dx.doi.org/10.5593/sgemsocial2018h/31/s13.088.

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Praskova, Anna. "COORDINATED REHABILITATION AND ITS IMPLEMENTATION IN ORGANIZATIONS WORKING WITH PERSONS AFTER ACQUIRED BRAIN DAMAGE." In 5th SGEM International Multidisciplinary Scientific Conferences on SOCIAL SCIENCES and ARTS SGEM2018. STEF92 Technology, 2018. http://dx.doi.org/10.5593/sgemsocial2018h/31/s13.075.

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