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Journal articles on the topic 'Bradycardia'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Gautam, Binod, Ashmita Maharjan, and Suson Ghimire. "Bradycardia during laparoscopic surgeries: A cross-sectional study." Journal of Kathmandu Medical College 9, no. 1 (2020): 5–12. http://dx.doi.org/10.3126/jkmc.v9i1.33515.

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Background: Bradycardia occurring during laparoscopic surgery potentially leads to cardiac arrest and adverse outcomes. Apart from the vagal reflex for its genesis, the knowledge on frequency and risk factors is limited.  Objectives: To identify the bradycardia frequency and time points for its occurrence during laparoscopic surgeries. Methodology: In this hospital-based cross-sectional study, anaesthesia-related incident reports on bradycardia were collected from January to December 2019. Bradycardias (heart rate less than 60/minute) that occurred during laparoscopic surgeries wer

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2

Rozloznik, Miroslav, Julian F. R. Paton, and Mathias Dutschmann. "Repetitive paired stimulation of nasotrigeminal and peripheral chemoreceptor afferents cause progressive potentiation of the diving bradycardia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 1 (2009): R80—R87. http://dx.doi.org/10.1152/ajpregu.00806.2007.

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Hallmarks of the mammalian diving response are protective apnea and bradycardia. These cardiorespiratory adaptations can be mimicked by stimulation of the trigeminal ethmoidal nerve (EN5) and reflect oxygen-conserving mechanisms during breath-hold dives. Increasing drive from peripheral chemoreceptors during sustained dives was reported to enhance the diving bradycardia. The underlying neuronal mechanisms, however, are unknown. In the present study, expression and plasticity of EN5-bradycardias after paired stimulation of the EN5 and peripheral chemoreceptors was investigated in the in situ wo

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3

Cummings, Kevin J., Aihua Li, Evan S. Deneris, and Eugene E. Nattie. "Bradycardia in serotonin-deficient Pet-1−/− mice: influence of respiratory dysfunction and hyperthermia over the first 2 postnatal weeks." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 5 (2010): R1333—R1342. http://dx.doi.org/10.1152/ajpregu.00110.2010.

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Neonatal rodents deficient in medullary serotonin neurons have respiratory instability and enhanced spontaneous bradycardias. This study asks if, in Pet-1−/− mice over development: 1) the respiratory instability leads to hypoxia; 2) greater bradycardia is related to the degree of hypoxia or concomitant hypopnea; and 3) hyperthermia exacerbates bradycardias. Pet-1+/+, Pet-1+/−, and Pet-1−/− mice [postnatal days (P) 4–5, P11–12, P14–15] were held at normal body temperature (Tb) and were then made 2°C hypo- and hyperthermic. Using a pneumotach-mask system with ECG, we measured heart rate, metabol

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4

García-Domingo, Mónica, José Ángel García-Pedraza, Juan Francisco Fernández-González, Cristina López, María Luisa Martín, and Asunción Morán. "Fluoxetine Treatment Decreases Cardiac Vagal Input and Alters the Serotonergic Modulation of the Parasympathetic Outflow in Diabetic Rats." International Journal of Molecular Sciences 23, no. 10 (2022): 5736. http://dx.doi.org/10.3390/ijms23105736.

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Comorbid diabetes and depression constitutes a major health problem, worsening associated cardiovascular diseases. Fluoxetine’s (antidepressant) role on cardiac diabetic complications remains unknown. We determined whether fluoxetine modifies cardiac vagal input and its serotonergic modulation in male Wistar diabetic rats. Diabetes was induced by alloxan and maintained for 28 days. Fluoxetine was administered the last 14 days (10 mg/kg/day; p.o). Bradycardia was obtained by vagal stimulation (3, 6 and 9 Hz) or i.v. acetylcholine administrations (1, 5 and 10 μg/kg). Fluoxetine treatment diminis

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5

Maltais-Bilodeau, Camille, Maryse Frenette, Geneviève Morissette, et al. "2 Systemic glucocorticoids and bradycardia in critically ill children: a retrospective study." Paediatrics & Child Health 25, Supplement_2 (2020): e1-e1. http://dx.doi.org/10.1093/pch/pxaa068.001.

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Abstract Background Glucocorticoids are widely used in the pediatric population. They are associated with numerous side effects including repercussions on the cardiovascular system. The impact on heart rate is not well known, but bradycardia has been reported, mostly with high doses. Objectives We described the occurrence of bradycardias and the variation of heart rate in critically ill children receiving glucocorticoids. Design/Methods We conducted a retrospective study including 1 month old to 18 year old children admitted to the Pediatric Intensive Care Unit between 2014 and 2017, who recei

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6

Fortrat, Jacques-Olivier. "Zipf’s Law of Vasovagal Heart Rate Variability Sequences." Entropy 22, no. 4 (2020): 413. http://dx.doi.org/10.3390/e22040413.

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Cardiovascular self-organized criticality (SOC) has recently been demonstrated by studying vasovagal sequences. These sequences combine bradycardia and a decrease in blood pressure. Observing enough of these sparse events is a barrier that prevents a better understanding of cardiovascular SOC. Our primary aim was to verify whether SOC could be studied by solely observing bradycardias and by showing their distribution according to Zipf’s law. We studied patients with vasovagal syncope. Twenty-four of them had a positive outcome to the head-up tilt table test, while matched patients had a negati

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7

Colombari, E., L. G. Bonagamba, and B. H. Machado. "Mechanisms of pressor and bradycardic responses to L-glutamate microinjected into the NTS of conscious rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 266, no. 3 (1994): R730—R738. http://dx.doi.org/10.1152/ajpregu.1994.266.3.r730.

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Microinjection of increasing doses of L-glutamate (L-Glu, 0.03-5.0 nmol/100 nl) into the nucleus tractus solitarii (NTS) produced a dose-related pressor and bradycardic response. Prazosin virtually abolished the pressor response but produced no changes in the bradycardic response to L-Glu, indicating that bradycardia is not reflex in origin. The bradycardic response was blocked by atropine. In three different groups of rats, excitatory amino acid receptors in the NTS were blocked by increasing doses of kynurenic acid (0.5, 2.0, and 10.0 nmol/100 nl) and the pressor and bradycardic responses to

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8

Porta-García, Miguel Ángel, Alberto Quiroz-Salazar, Eric Alonso Abarca-Castro, and José Javier Reyes-Lagos. "Bradycardia May Decrease Cardiorespiratory Coupling in Preterm Infants." Entropy 25, no. 12 (2023): 1616. http://dx.doi.org/10.3390/e25121616.

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Bradycardia, frequently observed in preterm infants, presents significant risks due to the immaturity of their autonomic nervous system (ANS) and respiratory systems. These infants may face cardiorespiratory events, leading to severe complications like hypoxemia and neurodevelopmental disorders. Although neonatal care has advanced, the influence of bradycardia on cardiorespiratory coupling (CRC) remains elusive. This exploratory study delves into CRC in preterm infants, emphasizing disparities between events with and without bradycardia. Using the Preterm Infant Cardio-Respiratory Signals (PIC

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9

Ahmad, Munir, Muhammad Yasir, and Sehar Fatima. "DRUG INDUCED BRADYCARDIA." Professional Medical Journal 25, no. 06 (2018): 908–13. http://dx.doi.org/10.29309/tpmj/2018.25.06.280.

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Background: Bradycardia in patients on rate slowing drugs i.e. beta blockers,digoxin and non dihydropyridine calcium channel blockers is common after discontinuation ofrate slowing drugs. Bradycardia persists in majority of patients, so bradycardia is not truly druginduced but due to underlying conduction system disease. Objectives: To determine the outcomein patients with bradycardia after discontinuation of rate slowing drugs in terms of frequencyof persistent bradycardia. Study Design: Descriptive cross-sectional. Place and Duration ofStudy: Cardiology Department, Faisalabad Institute of Ca

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10

Cardinal, René, Pierre Pagé, Michel Vermeulen, et al. "Spinal cord stimulation suppresses bradycardias and atrial tachyarrhythmias induced by mediastinal nerve stimulation in dogs." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 291, no. 5 (2006): R1369—R1375. http://dx.doi.org/10.1152/ajpregu.00056.2006.

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Spinal cord stimulation (SCS) applied to the dorsal aspect of the cranial thoracic cord imparts cardioprotection under conditions of neuronally dependent cardiac stress. This study investigated whether neuronally induced atrial arrhythmias can be modulated by SCS. In 16 anesthetized dogs with intact stellate ganglia and in five with bilateral stellectomy, trains of five electrical stimuli were delivered during the atrial refractory period to right- or left-sided mediastinal nerves for up to 20 s before and after SCS (20 min). Recordings were obtained from 191 biatrial epicardial sites. Before

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11

Higuchi, S., A. Takeshita, H. Higashi, et al. "Lowering calcium in the nucleus tractus solitarius causes hypotension and bradycardia." American Journal of Physiology-Heart and Circulatory Physiology 250, no. 2 (1986): H226—H230. http://dx.doi.org/10.1152/ajpheart.1986.250.2.h226.

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It has been shown that saline microinjected into the region of the nucleus tractus solitarius (NTS) causes, but artificial cerebrospinal fluid (CSF) in the same volume does not cause, hypotension and bradycardia. This study was done to examine the possibility that the difference in effects between saline and artificial CSF may be due to the lack of calcium ions in saline. In anesthetized rats, saline or artificial CSF with or without calcium ions was microinjected into the region of the NTS. Saline microinjected in volumes of 0.2 and 0.5 microliter produced the volume-dependent decreases in ar

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12

Vaswani, Zameer G., Sarah M. Smith, Anthony Zapata, Erin A. Gottlieb, and Paul W. Sheeran. "Bradycardic Arrest in a Child with Complex Congenital Heart Disease Due to Sugammadex Administration." Journal of Pediatric Pharmacology and Therapeutics 28, no. 7 (2023): 667–70. http://dx.doi.org/10.5863/1551-6776-28.7.667.

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The neuromuscular blocking drugs rocuronium and vecuronium are often used during general anesthesia. These drugs temporarily paralyze the patient and thus both facilitate placement of an endotracheal tube and prevent any patient movement during surgery. Reversal of neuromuscular blockade is necessary at the end of surgery to avoid postoperative weakness and adverse respiratory events in the recovery room. Neostigmine, the traditional reversal agent, may not completely restore muscle strength. Sugammadex is a reversal agent that is more effective and quicker acting than neostigmine. In adults,

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13

Chitravanshi, Vineet C., and Hreday N. Sapru. "Microinjections of urocortin1 into the nucleus ambiguus of the rat elicit bradycardia." American Journal of Physiology-Heart and Circulatory Physiology 300, no. 1 (2011): H223—H229. http://dx.doi.org/10.1152/ajpheart.00391.2010.

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Urocortins are members of the hypothalamic corticotropin-releasing factor (CRF) peptide family. Urocortin1 (UCN1) mRNA has been reported to be expressed in the brainstem neurons. The present investigation was carried out to test the hypothesis that microinjections of UCN1 into the nucleus ambiguus (nAmb) may elicit cardiac effects. Urethane-anesthetized, artificially ventilated, adult male Wistar rats, weighing between 300–350 g, were used. nAmb was identified by microinjections of l-glutamate (5 mM, 30 nl). Microinjections (30 nl) of different concentrations (0.062, 0.125, 0.25, and 0.5 mM) o

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14

Kemeç, Zeki, and Ali Gürel. "Acute kidney injury and sinus bradycardia associated with near-drowning." Ukrainian Journal of Nephrology and Dialysis, no. 4(68) (August 5, 2020): 18–22. http://dx.doi.org/10.31450/ukrjnd.4(68).2020.03.

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Acute kidney injury (AKI) occurs in different situations and may have a variable prognosis due to underlying cause, clinical setting and comorbidity. Near-drowning is known to lead to bradycardic rhythms which can lead to hypoxia because of hypoperfusion. AKI has a high risk of mortality and morbidity. However, sequelae of sinus bradycardia are related to its underlying etiology. Urinary, cardiovascular and respiratory disorders are more frequently seen after near-drowning. Near-drowning related AKI and sinus bradycardia are not reported together in the literature. We aimed to emphasize these

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15

Deshmukh, Amrish, and Cevher Ozcan. "Symptomatic Long Pauses and Bradycardia due to Massive Multinodular Goiter." Case Reports in Cardiology 2017 (2017): 1–3. http://dx.doi.org/10.1155/2017/4201942.

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Sinus node dysfunction with symptomatic bradycardia or chronotropic incompetence is generally an indication for pacemaker implantation. However, in patients with symptomatic sinus bradycardia, the identification and treatment of underlying pathologies may avoid the need for permanent pacemaker implantation. We present a case of carotid sinus syndrome and severe obstructive sleep apnea due to a massive multinodular goiter in a patient who presented with recurrent sinus pauses and syncope. The patient was managed without pacemaker implantation but instead with thyroidectomy resulting in decompre

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16

Kitchen, Amy M., Donal S. O'Leary, and Tadeusz J. Scislo. "Sympathetic and parasympathetic component of bradycardia triggered by stimulation of NTS P2X receptors." American Journal of Physiology-Heart and Circulatory Physiology 290, no. 2 (2006): H807—H812. http://dx.doi.org/10.1152/ajpheart.00889.2005.

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We have previously shown that activation of P2X purinoceptors in the subpostremal nucleus tractus solitarius (NTS) produces a rapid bradycardia and hypotension. This bradycardia could occur via sympathetic withdrawal, parasympathetic activation, or a combination of both mechanisms. Thus we investigated the relative roles of parasympathetic activation and sympathetic withdrawal in mediating this bradycardia in chloralose-urethane anesthetized male Sprague-Dawley rats. Microinjections of the selective P2X purinoceptor agonist α,β-methylene ATP (25 pmol/50 nl and 100 pmol/50 nl) were made into th

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17

Cardoso, Leonardo Máximo, Débora Simões de Almeida Colombari, José V. Menani, Glenn M. Toney, Deoclécio Alves Chianca, and Eduardo Colombari. "Cardiovascular responses to hydrogen peroxide into the nucleus tractus solitarius." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 297, no. 2 (2009): R462—R469. http://dx.doi.org/10.1152/ajpregu.90796.2008.

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The nucleus tractus solitarius (NTS), a major hindbrain area involved in cardiovascular regulation, receives primary afferent fibers from peripheral baroreceptors and chemoreceptors. Hydrogen peroxide (H2O2) is a relatively stable and diffusible reactive oxygen species (ROS), which acting centrally, may affect neural mechanisms. In the present study, we investigated effects of H2O2 alone or combined with the glutamatergic antagonist kynurenate into the NTS on mean arterial pressure (MAP) and heart rate (HR). Conscious or anesthetized (urethane and α-chloralose) male Holtzman rats (280–320 g) w

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18

Pregerson, Brady. "BradyCardia." Emergency Medicine News 42, no. 10 (2020): 27. http://dx.doi.org/10.1097/01.eem.0000719132.08202.f7.

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19

Hayes, Denise D. "Bradycardia." Nursing 34 (May 2004): 4–12. http://dx.doi.org/10.1097/00152193-200405001-00002.

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20

MEE, CHERYL L. "Bradycardia." Nursing 26, no. 4 (1996): 25. http://dx.doi.org/10.1097/00152193-199604000-00009.

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MEE, CHERYL L. "Bradycardia." Nursing 26, no. 4 (1996): 25. http://dx.doi.org/10.1097/00152193-199626040-00009.

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22

Nandi, P. R., and B. Astley. "Bradycardia." Anaesthesia 40, no. 11 (1985): 1140. http://dx.doi.org/10.1111/j.1365-2044.1985.tb10635.x.

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23

BURKE, LAURA J. "BRADYCARDIA." Nursing 18, no. 9 (1988): 102–5. http://dx.doi.org/10.1097/00152193-198809000-00030.

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24

Kim, Albert M., and Nova Goldschlager. "Bradycardia." Journal of Electrocardiology 41, no. 3 (2008): 206. http://dx.doi.org/10.1016/j.jelectrocard.2008.02.021.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 12 (2021): 21. http://dx.doi.org/10.1097/01.eem.0000804960.62369.62.

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Pregerson, Brady, and Stephen W. Smith. "BradyCardia." Emergency Medicine News 44, no. 2 (2022): 15. http://dx.doi.org/10.1097/01.eem.0000820896.22016.24.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 9 (2021): 10. http://dx.doi.org/10.1097/01.eem.0000791932.61152.d4.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 44, no. 11 (2022): 9. http://dx.doi.org/10.1097/01.eem.0000898204.53578.87.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 3 (2022): 17. http://dx.doi.org/10.1097/01.eem.0000824156.06215.e7.

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Pregerson, Brady, and Stephen W. Smith. "Bradycardia." Emergency Medicine News 44, no. 4 (2022): 17. http://dx.doi.org/10.1097/01.eem.0000827696.00238.94.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 45, no. 2 (2023): 18. http://dx.doi.org/10.1097/01.eem.0000920084.51582.cf.

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32

Pregerson, Brady. "BradyCardia." Emergency Medicine News 45, no. 1 (2023): 14. http://dx.doi.org/10.1097/01.eem.0000911912.07201.c8.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 42, no. 12 (2020): 32. http://dx.doi.org/10.1097/01.eem.0000724624.87486.46.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 8 (2022): 28. http://dx.doi.org/10.1097/01.eem.0000855856.59055.0c.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 1 (2022): 17. http://dx.doi.org/10.1097/01.eem.0000815556.43364.9c.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 42, no. 11 (2020): 10. http://dx.doi.org/10.1097/01.eem.0000722396.53647.75.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 42, no. 8 (2020): 25. http://dx.doi.org/10.1097/01.eem.0000695636.38819.b0.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 5 (2021): 18. http://dx.doi.org/10.1097/01.eem.0000751892.02437.9e.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 11 (2021): 18. http://dx.doi.org/10.1097/01.eem.0000800516.31882.4e.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 6 (2021): 14. http://dx.doi.org/10.1097/01.eem.0000754844.33982.26.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 43, no. 2 (2021): 26. http://dx.doi.org/10.1097/01.eem.0000734628.59992.04.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 10 (2022): 27. http://dx.doi.org/10.1097/01.eem.0000891164.34217.c3.

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Pregerson, Brady. "BradyCardia." Emergency Medicine News 43, no. 3 (2021): 26. http://dx.doi.org/10.1097/01.eem.0000737520.79662.7a.

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Pregerson, Brady, and Stephen W. Smith. "BradyCardia." Emergency Medicine News 44, no. 5 (2022): 11. http://dx.doi.org/10.1097/01.eem.0000831232.82848.7a.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 6 (2022): 13–14. http://dx.doi.org/10.1097/01.eem.0000834128.27769.ca.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 9 (2022): 8. http://dx.doi.org/10.1097/01.eem.0000874676.29582.52.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 4 (2021): 25. http://dx.doi.org/10.1097/01.eem.0000743232.89664.41.

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Pregersen, Brady. "BradyCardia." Emergency Medicine News 44, no. 12 (2022): 11. http://dx.doi.org/10.1097/01.eem.0000904628.02623.b6.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 43, no. 10 (2021): 19. http://dx.doi.org/10.1097/01.eem.0000795788.01992.0f.

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Pregerson, Brady. "Bradycardia." Emergency Medicine News 44, no. 7 (2022): 16. http://dx.doi.org/10.1097/01.eem.0000852624.46973.ca.

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