Relevant bibliographies by topics / Bones – Growth

Academic literature on the topic 'Bones – Growth'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 February 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Contents

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Bones – Growth.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Bones – Growth"

1

Levine, Milton I. "Fragile Bones, Soft Bones, and Linear Growth Failure." Pediatric Annals 16, no. 12 (December 1, 1987): 943–45. http://dx.doi.org/10.3928/0090-4481-19871201-04.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Scheven, Ben A. A., and Nicola J. Hamilton. "Longitudinal bone growth in vitro: effects of insulin-like growth factor I and growth hormone." Acta Endocrinologica 124, no. 5 (May 1991): 602–7. http://dx.doi.org/10.1530/acta.0.1240602.

Full text
Abstract:
Abstract. Longitudinal growth was studied using an in vitro model system of intact rat long bones. Metatarsal bones from 18- and 19-day-old rat fetuses, entirely (18 days) or mainly (19 days) composed of chondrocytes, showed a steady rate of growth and radiolabelled thymidine incorporation for at least 7 days in serum-free media. Addition of recombinant human insulin-like growth factor-I to the culture media resulted in a direct stimulation of the longitudinal growth. Recombinant human growth hormone was also able to stimulate bone growth, although this was generally accomplished after a time lag of more than 2 days. A monoclonal antibody to IGF-I abolished both the IGF-I and GH-stimulated growth. However, the antibody had no effect on the growth of the bone explants in control, serum-free medium. Unlike the fetal long bones, bones from 2-day-old neonatal rats were arrested in their growth after 1-2 days in vitro. The neonatal bones responded to IGF-I and GH in a similar fashion as the fetal bones. Thus in this study in vitro evidence of a direct effect of GH on long bone growth via stimulating local production of IGF by the growth plate chondrocytes is presented. Furthermore, endogenous growth factors, others than IGFs, appear to play a crucial role in the regulation of fetal long bone growth.
APA, Harvard, Vancouver, ISO, and other styles
3

Hagan, Michael. "Sticks and Bones." Arithmetic Teacher 33, no. 1 (September 1985): 44–45. http://dx.doi.org/10.5951/at.33.1.0044.

Full text
Abstract:
The proliferation of both teacher-made and professionally prepared games might easily tempt a teacher to conclude that using them to kindle a child's interest in mathematics is an end in itself. And yet the full riches of many games may lie undiscovered until children are challenged to go beyond their interests, to think about their actions, and to elaborate on them. From this perspective, the most valuable games are those that interest children and that have rich variations to encourage their growth.
APA, Harvard, Vancouver, ISO, and other styles
4

Soliz, Mónica, María Jose Tulli, and Virginia Abdala. "Forelimb musculoskeletal-tendinous growth in frogs." PeerJ 8 (February 25, 2020): e8618. http://dx.doi.org/10.7717/peerj.8618.

Full text
Abstract:
The tendons unite and transmit the strength of the muscles to the bones, allowing movement dexterity, the distribution of the strength of the limbs to the digits, and an improved muscle performance for a wide range of locomotor activities. Tissue differentiation and maturation of the structures involved in locomotion are completed during the juvenile stage; however, few studies have investigated the ontogenetic variation of the musculoskeletal-tendinous system. We ask whether all those integrated tissues and limb structures growth synchronically between them and along with body length. We examined the ontogenetic variation in selected muscles, tendons and bones of the forelimbs in seventy-seven specimens belonging to seven anuran species of different clades and of three age categories, and investigate the relative growth of the forelimb musculoskeletal-tendinous structures throughout ontogeny. Ten muscles and nine tendons and their respective large bones (humerus and radioulna) were removed intact, and their length was measured and analyzed through a multivariate approach of allometry. We obtained an allometry coefficient, which indicates how the coefficient departures from isometry as well as allometric trends. Our data suggest that along with the post-metamorphic ontogeny, muscles tend to elongate proportionally to bone length, with a positive allometric trend. On the contrary, tendons show a negative allometric growth trend. Only two species show different patterns: Rhinella granulosa and Physalaemus biligonigerus, with an isometric and positive growth of muscles and bones, and most tendons being isometric.
APA, Harvard, Vancouver, ISO, and other styles
5

DeLuca, H. F. "TRIENNIAL GROWTH SYMPOSIUM— Vitamin D: Bones and beyond12." Journal of Animal Science 92, no. 3 (March 1, 2014): 917–29. http://dx.doi.org/10.2527/jas.2013-7237.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

McKee, Malcolm. "Growth deformities of the long bones in dogs." In Practice 32, no. 7 (July 2010): 282–91. http://dx.doi.org/10.1136/inp.c3914.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Deltoro, J., and Ana M. López. "Allometric growth patterns of limb bones in rabbits." Animal Production 46, no. 3 (June 1988): 461–67. http://dx.doi.org/10.1017/s0003356100019073.

Full text
Abstract:
ABSTRACTRelative growth of length and thickness of femur, tibia, humerus and radius-ulna was studied using data from 320 rabbits from both sexes of two lines (New Zealand White and California). A cross-sectional design was used with slaughter points fixed at weekly intervals from 1 to 20 weeks of age. Carcass length was chosen as independent variable and two models were fitted to the data. Model 1 assumed the existence of one allometric change at some moment of the post-natal development and model 2 of a continuous change of the allometric coefficient throughout the experimental period.Most of the bone measurement showed allometric changes and in all the cases they were not abrupt but continuous. The theoretical and practical implications of the existence of these changes are discussed. The allometric coefficients of all the bone measurements showed a decreasing rate of change except radius-ulna thickness. While the coefficients for bone lengths were positive tending to isometry, for bone thickness they were always negative.
APA, Harvard, Vancouver, ISO, and other styles
8

Efthimiou, J., and P. J. Barnes. "Effect of inhaled corticosteroids on bones and growth." European Respiratory Journal 11, no. 5 (May 1, 1998): 1167–77. http://dx.doi.org/10.1183/09031936.98.11051167.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Garzón-Alvarado, D. A., J. M. García-Aznar, and M. Doblaré. "A reaction–diffusion model for long bones growth." Biomechanics and Modeling in Mechanobiology 8, no. 5 (December 24, 2008): 381–95. http://dx.doi.org/10.1007/s10237-008-0144-z.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Lok, F., J. A. Owens, L. Mundy, J. S. Robinson, and P. C. Owens. "Insulin-like growth factor I promotes growth selectively in fetal sheep in late gestation." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 270, no. 5 (May 1, 1996): R1148—R1155. http://dx.doi.org/10.1152/ajpregu.1996.270.5.r1148.

Full text
Abstract:
Insulin-like growth factor I (IGF-I) is required for normal fetal growth and skeletal maturation in late gestation, because null mutations of the IGF-I gene in mice reduce fetal weight and retard ossification of bones. To determine if, conversely, increased abundance of IGF-I promotes fetal growth and skeletal maturation, fetal sheep were infused intravascularly with recombinant human IGF-I (n = 7) (26 +/- 3 micrograms. h-1.kg-1) from 120 to 130 days gestation and compared with controls (n = 15). IGF-I infusion increased plasma IGF-I concentrations by 140% (P = 0.002) and weights of fetal liver, lungs, heart, kidneys, spleen, pituitary, and adrenal glands by 16-50% (P < 0.05). Weights and/or lengths of the fetus, placenta, gastrointestinal tract, individual skeletal muscles, and long bones were unchanged by IGF-I. However, IGF-I increased the percentage of proximal epiphyses of long bones present (P < 0.05) and their cross-sectional areas by 15 to 38% (P < 0.05). These results show that IGF-I promotes growth of major fetal organs, endocrine glands, and skeletal maturation in vivo, consistent with IGF-I actively controlling and not merely facilitating fetal growth. The variable response of different tissues may partly reflect tissue specificity in growth requirements for additional factors.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Bones – Growth"

1

Yadav, Priti. "Modelling loading and growth of long bones Modelling loading and growth of long bones." Licentiate thesis, KTH, Biomekanik, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-177913.

Full text
Abstract:
The long bones grow by the process of endochondral ossification, which occurs at the growth plate. This process is regulated by biological factors and mechanical factors. The biological factors which contribute to endochondral ossification process are genes, hormones, nutrients etc. The mechanical factor is the load acting on the bone. The major forces on the bone are due to joint contact load and muscle forces, which induce stresses in the bone. Carter and Wong proposed in a theory that cyclic or intermittent octahedral shear stress promotes the bone growth and cyclic or intermittent hydrostatic compressive stress inhibits the bone growth. Previously this theory has been used to predict the morphological development of long bones, but with studies using simplified femur and growth plate models. Furthermore, the Carter and Wong theory has a limitation that it does not intrinsically incorporate the resulting growth direction.In the first study, the importance of a subject-specific growth plate over a simplified growth plate has been studied, and growth has been simulated using two different growth direction models: Femoral neck shaft deformation direction and minimum shear stress direction. This study favors the minimum shear stress growth direction model, as it is less sensitive to applied boundary condition than the femoral neck shaft deformation direction model.The second study aims to understand how different muscle groups affect the bone growth tendency. Subject-specific femur and growth plate models of able-bodied children were used. The muscle forces and associated hip contact force from specific muscle groups were applied, and neck shaft angle and femoral anteversion growth tendencies were predicted. This study indicated a tendency for reduction of neck shaft angle and femoral anteversion. Hip abductor muscle forces contribute most, and hip adductor muscle forces least, to bone growth rate.Accurate prediction of bone growth tendency and knowledge of the influence of different muscle groups on bone growth tendency may help in better treatment planning for children at risk of developing bone deformity problems.

QC 20151201

APA, Harvard, Vancouver, ISO, and other styles
2

Chim, Shek Man. "Identification and characterization of novel secreted factors involved in bone remodeling." University of Western Australia. School of Surgery, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0110.

Full text
Abstract:
[Truncated abstract] Bone remodeling is an important process to maintain mechanical integrity. It is accomplished by two important steps, bone resorption followed by new bone formation. Osteoclasts and osteoblasts are the principal cells in bone resorption and bone formation, respectively. A multitude of local and systemic factors regulates this process by controlling the cellular activities in bone remodeling compartments (BRC). An imbalance of osteoblastic bone formation and osteoclastic bone destruction will result in the development of skeletal diseases. Recent studies suggested that angiogenesis is closely associated with bone remodeling. The vasculature in bone is important for skeletal development, growth and repair. During endochondral ossification, cartilage is invaded by blood vessels which bring in osteoblast and osteoclast precursor cells, nutrients, growth factors and differentiation factors. During fracture repair, it has been demonstrated that mature osteoclasts produce heparanase which can degrade heparin sulfate proteoglycans, a major component in extracellular matrix (ECM). The process leads to the release of heparin-binding growth factors including vascular endothelial growth factor (VEGF), a potent angiogenic factor which contributes largely to local angiogenesis. In recent studies, endothelial cells have been found to produce bone morphogenetic protein (BMP)-2 and BMP-4 when they are subjected to mechanical stimuli, or a hypoxia environment. Conversely, inhibition of angiogenesis has been shown to prevent fracture healing. In a distraction osteogenesis model, either inhibition of angiogenesis or disruption of the mechanical environment prevents normal osteogenesis and results in fibrous nonunion. .... A total of 42 mice from F1 and F2 generations were genotyped as transgene positive. Preliminary analysis using radiography did not reveal any difference between the gross structures of transgenic and wild type mice. Interestingly, the preliminary histology revealed a decrease in trabecular bone and an increase of lipid space in metaphysis of transgenic mice overexpressing EGFL6. However, further studies will need to be carried out to investigate the role of EGFL6 in angiogenesis and adipogenesis using a transgenic mice model. This will be a prime focus of future work. Collectively, the results presented in this thesis have identified EGFL6, a member of the EGF-like family, as a potential angiogenic factor which may play an important role in bone remodeling. EGFL6 has been found to be expressed highly in calvarial osteoblasts and upregulated during primary murine osteoblast differentiation. EGFL6 has been 8 characterized to be a secreted homomeric complex. More importantly, EGFL6 has been shown to induce angiogenic activity in endothelial cell migration, tube formation and in vivo chick embryo chorioallantoic membrane assay. Furthermore, conditioned medium containing the EGFL6 recombinant protein was shown to induce phosphorylation of ERK in endothelial cells. Inhibition of ERK impaired EGFL6-induced ERK activation and endothelial cell migration. Taken together these studies raise the possibility that EGFL6 has a potential role in angiogenesis, and mediates a paracrine mechanism of cross-talk between vascular endothelial cells and osteoblasts during osteogenesis. An understanding of this process offers the potential to facilitate the development of therapeutic treatments for bone disease.
APA, Harvard, Vancouver, ISO, and other styles
3

Ma, Li, and 马丽. "The influence of nicotine on angiogenesis and osteogenesis in bone regeneration." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508440.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Oest, Megan Elizabeth. "Dual Osteogenic and Angiogenic Growth Factor Delivery as a Treatment for Segmental Bone Defects." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16264.

Full text
Abstract:
A new model of a critically-sized segmental femoral bone defect in rats was developed to enable in vivo imaging and facilitate post-mortem mechanical testing of samples. The critically-sized nature of the model was assessed and confirmed. The efficacy of sustained co-delivery of osteogenic (BMP-2 and TGF- Ò3) and angiogenic (VEGF) growth factors in promoting functional bone repair was assessed. Effects of scaffold modification in terms of geometry and composition were evaluated. The results indicated that co-delivery of BMP-2 and TGF- Ò3 resulted in a dose-dependent improvement in functional bone repair. Modification of the polylactide scaffold to include an absorbable ceramic component and a cored out geometry enhanced rate of union. Addition of VEGF to the scaffold treatment did not significantly impact revascularization of the defect site or functional repair of the bone defect. These data demonstrate that the complex environment of an acute bone defect requires different treatment strategies than simple ectopic models would suggest. A positive predictive correlation between bone repair parameters measured in vivo and mechanical functionality was established. The novel defect model demonstrated robustness and reproducibility. Implications for further research are discussed.
APA, Harvard, Vancouver, ISO, and other styles
5

關健明 and Kin-ming Kwan. "Defining the function of type X collagen in skeletal development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237162.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Foster, Bruce Kristian. "Epiphyseal plate repair using fat interposition to reverse physeal deformity : an experimental study." Title page, contents and summary only, 1989. http://web4.library.adelaide.edu.au/theses/09MD/09mdf754.pdf.

Full text
Abstract:
Bibliography: leaves 169-197. Hypothesises that the physis has an internal mechanism of repair to restore physeal function. Aims to establish a defined degree of deformity by partial growth plate excision, then to examine different methods of reversal of such deformity to observe the process of growth plate repair. A secondary aim was to define the percentage of physis that could be resected yet still enable reversal of deformity.
APA, Harvard, Vancouver, ISO, and other styles
7

Chayanupatkul, Atinooch. "Bone formation in the temporomandibular joint in response to forward mandibular positioning." Thesis, Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25598776.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Cha, Ming Chuan 1955. "The effect of zinc deficiency on the growth promoting actions of growth hormone and insulin-like growth factor-I /." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55484.

Full text
Abstract:
The effect of zinc deficiency on the growth promoting effect of circulating IGF-I and the direct growth effect of GH on long bone growth were investigated. Food intake was decreased by lack of zinc in the diet. Tissue zinc content and plasma alkaline phosphatase activity were reduced by zinc deficiency. Systemic administration of human IGF-I increased the body weight, tail length and tibia epiphyseal cartilage width of control animals. This somatogenic action was impaired by zinc deficiency, as evidenced by continued weight loss, no increase in tail length and decreased tibial epiphyseal cartilage width of zinc deficient animals. Unilateral arterial infusion of GH increased the tibial epiphyseal width of the treated limb but not of the non-treated limb in control rats. However, no difference was found between the infused and the non-infused limb of zinc deficient animals, suggesting the occurrence of GH resistance on long bone growth in zinc deficiency. We conclude that zinc deficiency inhibits the growth promoting action of circulating IGF-I and the direct growth effect of GH on long bone growth.
APA, Harvard, Vancouver, ISO, and other styles
9

Dai, Zhijie, and 戴志洁. "The role of sodium/myo-inositol cotransporter 1 and myo-inositol in osteogenesis and bone formation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43783533.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Dahlgren, Emma. "Effects of Different Load Magnitudes on Longitudinal Growth of Immature Bones." Thesis, KTH, Skolan för kemi, bioteknologi och hälsa (CBH), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-230885.

Full text
Abstract:
In vivo studies of mechanical loading on bone have suggested that load magnitude is one of the parameters that play a vital role in bone adaptation. This study examined how longitudinal growth of immature rat metatarsals is affected by different load magnitudes. The main hypotheses were that the longitudinal growth of immature bone would decrease with increased compressive load magnitude, and that the longitudinal growth would be more decelerated the higher the load mag- nitude. The three middle metatarsal bones in the back paws of 19-20 days old Sprague-Dawley rat fetuses were extracted. Metatarsal bones were loaded with 0.05 N, 0.25 N, 1.25 N and 6.25 N. Loading rate and number of cycles were constant at 0.01 mm/sec and 10 cycles respectively. Length measurements occurred every 2-3 day. Concluded from the study was that a load magni- tude of 0.05 N resulted in an increased longitudinal growth, compared to unloaded bones. For the other load magnitudes the results were insufficient and inconsistent and therefore nothing could be suggested for them. The problem remained as before and further studies are needed.
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Bones – Growth"

1

J, Massaro Edward, and Rogers John M, eds. The skeleton: Biochemical, genetic, and molecular interactions in development and homeostasis. Totowa, N.J: Humana Press, 2004.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
2

M, Shapiro Irving, Boyan Barbara, and Anderson H. Clarke, eds. The growth plate. Amsterdam: IOS Press, 2002.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

1946-, Revell William J., ed. Blood supply of bone: Scientific aspects. London: Springer, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Enlow, Donald H. Facial growth. 3rd ed. Philadelphia: Saunders, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

Bones and cartilage: Developmental and evolutionary skeletal biology. Australia: Elsevier Academic Press, 2005.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

The complete book of bone health. Amherst, N.Y: Prometheus Books, 2011.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Mone, Zaidi, ed. Skeletal development and remodeling in health, disease, and aging. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

Lin, Yunfeng. Osteogenesis. Rijeka, Croatia: InTech, 2012.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

NATO Advanced Study Institute on Advances in Bone Regulatory Factors: Morphology, Biochemistry, Physiology, and Pharmacology (1989 Erice, Italy). Bone regulatory factors: Morphology, biochemistry, physiology, and pharmacology. New York: Plenum Press, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Mone, Zaidi, New York Academy of Sciences, and Mount Sinai School of Medicine, eds. Skeletal biology and medicine. Boston, Mass: Blackwell Pub., on behalf of the New York Academy of Sciences, 2007.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Bones – Growth"

1

Uhthoff, H. K., and J. J. Robichon. "The Growth of Tubular Bones." In The Embryology of the Human Locomotor System, 15–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75310-7_3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Snell, E. J. "Growth of bones from chick embryos." In Applied Statistics, 56–60. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-011-6946-2_12.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Snell, E. J., and H. R. Simpson. "Growth of bones from chick embryos." In Applied Statistics, 50–53. Boca Raton: Chapman and Hall/CRC, 2021. http://dx.doi.org/10.1201/9781003059844-11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Kleber, Bernd-Michael, and Karl Hecht. "Influence of Hypokinesia and Weightlessness on Jaw Bones and Teeth." In Humoral Factors in the Regulation of Tissue Growth, 244–66. New York, NY: Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4613-9272-9_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Karbowski, A., J. Herwig, H. H. Matthiaß, and E. Buddecke. "Effects of Mechanical Factors on Structure and Function of the Growth Plate of Long Bones." In Generalized Bone Diseases, 337–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73346-8_31.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Wilson, Helen, Diana Calcraft, Cai Neville, Susan Lanham-New, and Louise R. Durrant. "Bone Health, Fragility and Fractures." In Perspectives in Nursing Management and Care for Older Adults, 115–34. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63892-4_9.

Full text
Abstract:
AbstractAchieving and maintaining skeletal health throughout the life trajectory is essential for the prevention of bone diseases such as rickets, osteomalacia and osteoporosis. Rickets and osteomalacia are usually a result of calcium and/or vitamin D deficiency, causing softening of bones and bone pain, and both conditions are treatable with calcium and vitamin D supplementation. Osteoporosis is a multifaceted disease mainly affecting older people, and its pathogenesis (and hence treatment) is more complex. Untreated osteoporosis results in fragility fractures causing morbidity and increased mortality.Nutrition is one of many factors that influence bone mass and risk of bone disease. Developing a nutritional sciences approach is a feasible option for improving bone health.The importance of adequate calcium and vitamin D in ensuring skeletal integrity throughout the life course has a sound evidence base. Poor vitamin D status in population groups of all ages is widespread across many countries (including affluent and non-affluent areas). Public health approaches are required to correct this given the fact that vitamin D is not just required for musculoskeletal health but also for other health outcomes.Dietary protein may be beneficial for bone due to its effect of increasing insulin-like growth-factor-1 (IGF-1). Recent meta-analyses show that dietary protein has a beneficial role to play in bone health at all ages.Other nutritional factors and nutrients (such as potassium, magnesium, vitamin K and acid-base balance) are also likely to have an important role in bone health, though the literature is less clear in terms of the association/relationship and more research is required.
APA, Harvard, Vancouver, ISO, and other styles
7

Kim, Su-Jung, and Ki Beom Kim. "Craniofacial Morphology Related to Obstructive Sleep Apnea: Growth of Craniofacial Bones and the Upper Airway." In Management of Obstructive Sleep Apnea, 105–33. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-54146-0_9.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Brookes, Murray, and William J. Revell. "Growth cartilages." In Blood Supply of Bone, 152–76. London: Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-1543-4_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Bartl, Reiner, and Christoph Bartl. "Growth and Ageing of Bone." In Bone Disorders, 39–41. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29182-6_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Anderson, H. Clarke, and Irving M. Shapiro. "The Epiphyseal Growth Plate." In Bone and Development, 39–64. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-822-3_3.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Bones – Growth"

1

Askew, Michael J., Gary B. Schneider, Kristina J. Grecco, Jason Hsu, Emily Mugler, and Donald A. Noe. "Effect of Pharmaceutical Bone Growth Stimulation With Novel Anabolic Peptides: Biomechanical and Bone Density Measurements in a Rat Model." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43044.

Full text
Abstract:
Pharmaceutical bone growth stimulation holds promise for prevention and treatment bone disorders, and the enhancement of fracture healing. Bone growth hormones have begun to have limited clinical use, but can illicit adverse side effects. Recent studies have shown that short peptides (less than 15 amino acids) derived from the protein sequence of Vitamin D Binding Protein (DBP), can enhance bone formation (osteogenesis). These peptides may have potential as controllable bone growth stimulators without the adverse side effects and cost of bone growth hormones. Rats, injected every other day for two weeks with DBP-based peptide fragments ranging from 3 to 13 amino acids in length, were euthanized and the tibias and femurs were scanned by peripheral quantitative computerized tomography (pQCT) to determine bone density and cross-sectional geometric properties. The bones were then tested in three-point bending to determine strength and bending modulus. Injection of DBP-based peptides over only a 2-week period resulted in significant (p&lt;0.05) increases in bone density and material properties in the experimental rat bones in comparison to controls injected with saline. The short length of these effective peptides suggests their use not only in systemic injections but also as clinically convenient pills taken orally for pharmaceutically induced bone growth stimulation.
APA, Harvard, Vancouver, ISO, and other styles
2

Vincitorio, F. M., N. Budini, C. Mulone, M. Spector, C. Freyre, A. J. López Díaz, and A. Ramil. "Detection of fungi colony growth on bones by dynamic speckle." In 8th Ibero American Optics Meeting/11th Latin American Meeting on Optics, Lasers, and Applications, edited by Manuel Filipe P. C. Martins Costa. SPIE, 2013. http://dx.doi.org/10.1117/12.2025550.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Kłodowski, Adam, Antti Valkeapa¨a¨, and Aki Mikkola. "Craig-Bampton Modal Reduction Applied to Human Tibia Tradeoff Between Accuracy and Speed." In ASME 2011 International Mechanical Engineering Congress and Exposition. ASMEDC, 2011. http://dx.doi.org/10.1115/imece2011-63618.

Full text
Abstract:
Human bones adapt to external loading through bone growth and resorption processes (1). Strains within specific range induced by physical loading can lead to strengthening of affected bones. On the other hand, when external forces are too high, it can lead to bone fracture (2) or cause significant loads in the joints, which in turn can be damaging for the cartilage surfaces (3) and ligaments. Optimization of the gym equipment as well as the techniques of exercising is necessary to achieve bone growth stimulation without overloading the bones or the joints. This issue has been recently addressed with the use of flexible multibody simulations supported by modal reduction techniques. Although the strain output of the simulations is sound (4), it is necessary to understand the tradeoffs between accuracy and speed of the modal reduction methods. This paper presents a comparison of the tibial strains, stresses and global displacements obtained from modal representation of the bone and results obtained from the initial finite element model. Strains are obtained at the same nodes in both models during various static case loadings. Efficiency of both methods is compared by correlating computation times. Accuracy of modal representation is verified by using bending, torsion, tension and compression tests, which represent the possible physical loading conditions of tibia. Influence of the material models as well as discretization level has also been taken into account. Finally conclusions are drawn from the results providing guidelines for future work.
APA, Harvard, Vancouver, ISO, and other styles
4

Hart, Stephen A., and Marcelo J. Dapino. "Accelerated Bone Growth Remotely Induced by Magnetic Fields and Smart Materials." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175966.

Full text
Abstract:
Bone structure is exquisitely matched to its physical loading environment. From the cartilaginous skeletal framework formed in the embryo to the aging skeleton, bone architecture is directly related to function. Bone is a dynamic system, constantly remodeling itself by absorbing old tissue and forming new tissue. This capability allows bone architecture to become optimized to the loading environment. Julius Wolff [1] first postulated that bone structure adapts to changing stress environments in 1892. Exact understanding of the process of mechanotransduction, however, has remained elusive. In addition to normal remodeling, bone growth has been shown to occur along the diaphysis, or shaft portion, of long bones such as the femur when placed in dynamic bending. Bone in this region is dense and is known as cortical bone. A bending moment placed on a long bone will cause the bone to curve creating a region of tension on one side and a region of compression on the opposing side. As the bending moment is cycled, fluid within the bone will flow from the region of compression to the region of tension creating fluid shear on cell walls within the bone which promotes the anabolic response of growth [2]. Growth from such stimuli is thought to be mediated by fluid flow around quiescent bone cells, osteocytes, and their canalicular process coursing through the bone structure [3]. Growth in this manner is directed in a latitudinal direction creating a thicker and stronger diaphysis.
APA, Harvard, Vancouver, ISO, and other styles
5

Lerner, Amy L., David L. Gushue, and Emily A. Gedbaw. "Mechanical Stress Patterns in the Human Proximal Tibial Growth Plate During the Stance Phase of Normal Gait." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-32609.

Full text
Abstract:
Since the mid 1800’s clinicians and researchers have considered the effect of mechanical stresses on bone growth and the development of growth disorders [1]. However, the specifics of this relationship remain poorly understood. Both clinical and experimental evidence support the concept that magnitude, frequency and duration of loads are critical in defining the response of cartilage to pressures [2, 3]. Yet, the range of physiologic pressures in the growth plate has not been identified, and most models of growing bones have considered single quasi-static loading conditions and/or elastic material models that can not accurately represent time dependence [4, 5]. It was the goal of this study to implement loading conditions representing an entire stance phase of gait in a two-dimensional model of the proximal tibia of a normal child. A poroelastic material model was used in order to identify the variations in growth plate pressures in both time and location, and investigate the potential for fluid flow within the growth plate.
APA, Harvard, Vancouver, ISO, and other styles
6

Chou, Kathy, Grace Kim, and Marjolein C. H. van der Meulen. "The Effects of Vitamin D Deficiency on Histomorphometry and Strength of Rat Vertebrae." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53519.

Full text
Abstract:
Vitamin D3 is integral to both bone remodeling and calcium homeostasis [1]. With vitamin D deficiency, rickets develops during growth and osteomalacia results in adulthood [2]; in both cases, mineralization is altered and bones are more prone to fracture. Although the degenerative effects of vitamin D deficiency on trabecular architecture have been studied, investigations examining both compromised tissue material properties and mechanical properties in the vertebrae of growing animals are scarce. Therefore, the objective of this study was to investigate cancellous bone architecture and mechanical property changes caused by altered mineralization through vitamin D deficiency in growing rats.
APA, Harvard, Vancouver, ISO, and other styles
7

Yanoso, Laura, Justin Jacobson, Tulin Dadali, David Reynolds, and Hani Awad. "Evaluation of Polylactic Acid/Beta-Tricalcium Phosphate Scaffolds as Segmental Bone Graft Substitutes." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192978.

Full text
Abstract:
The use of processed structural allografts for treatment of massive segmental defects in long bones can be complicated by poor incorporation and remodeling of the devitalized graft, foreign-body reaction and micro-damage accumulation which often leads to catastrophic graft failure [1]. It is therefore useful to develop a bioengineered, biodegradable scaffold that is able to stimulate healing of the defect region. The use of bioengineered scaffolds has been limited due to their poor mechanical strength that does not permit withstanding large in vivo loads and due to their poor osteoinductive properties. We therefore investigated the use of rigid polylactic acid/beta-tricalcium phosphate (PLA/βTCP) composites used in conjunction with osteoinductive factors such as growth hormones (parathyroid hormone (PTH)) and growth factors (bone morphogenic protein-2 (BMP-2) & vascular endothelial growth factor (VEGF)) to stimulate bone formation and vessel ingrowth in the segmental defect region. We examined the physical characteristics of the scaffolds, and evaluated their osteoinductive potential in a clinically-relevant mouse model of a femoral segmental defect with or without PTH treatment. Finally, we used an ectopic bone formation model to assess the efficacy of the scaffold in site-specific delivery of bone anabolic factors.
APA, Harvard, Vancouver, ISO, and other styles
8

Nowlan, Niamh C., Paula Murphy, and Patrick J. Prendergast. "Mechanical Stimuli Resulting From Embryonic Muscle Contractions Promote Avian Periosteal Bone Collar Formation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-172077.

Full text
Abstract:
Mechanical forces due to muscle contractions play an essential role in embryonic skeletal development. In neuromuscular conditions such as congenital myotonic dystrophy, where movement of the fetus in utero is reduced or absent, the bones and joints of the newborn often show malformations [1]. In this paper, we examine the effect of muscle contractions on embryonic bone development. We propose the hypothesis that mechanical loading due to muscle contractions promotes periosteal ossification and we test this hypothesis using computational and experimental methods. A set of FE analyses were performed using anatomically realistic morphologies and loading conditions, at several timepoints during development, in order to identify biophysical stimuli active during bone formation. Avian immobilization experiments were performed to examine bone growth in the absence of skeletal muscle contractions.
APA, Harvard, Vancouver, ISO, and other styles
9

Charoenphan, Saiphon, and Apiwon Polchai. "Finite Element Modeling for Energy Release Rate in Human Cortical Bone." In ASME 7th Biennial Conference on Engineering Systems Design and Analysis. ASMEDC, 2004. http://dx.doi.org/10.1115/esda2004-58307.

Full text
Abstract:
The energy release rates in human cortical bone are investigated using a hybrid method of experimental and finite element modeling techniques. An explicit finite element analysis was implemented with an energy release rate calculation for evaluating this important fracture property of bones. Comparison of the critical value of the energy release rate, Gc, shows good agreement between the finite element models and analytical solutions. The Gc was found to be approximately 820–1150 J/m2 depending upon the samples. Specimen thickness appears to have little effect on the plane strain condition and pure mode I assumption. Therefore the energy release rate can be regarded as a material constant and geometry independent and can be determined with thinner specimens. In addition, the R curve resulting from the finite element models during slow crack growth shows slight ductility of the bone specimen that indicates an ability to resist crack propagation. Oscillations were found at the onset of the crack growth due to the nodal releasing application in the models. In this study light mass-proportional damping was used to suppress the noises. Although this techniques was found to be efficient for this slow crack growth simulation, other methods to continuously release nodes during the crack growth would be recommended for rapid crack propagation.
APA, Harvard, Vancouver, ISO, and other styles
10

Uddin, Sardar M. Zia, and Yi-Xian Qin. "Anabolic Effects of Ultrasound as Countermeasures of Simulated Microgravity in In-Vitro and In-Vivo Functional Disuse Models." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53796.

Full text
Abstract:
Microgravity (MG) during space flight has been known to cause adverse effect on bone quality. Data collected from studies done on spaceflights show loss of 1–1.6% bone mineral density (BMD) per space-flight-month[1]. Most BMD has been recorded in load-bearing bones [2]. Some studies has considered using drugs and different growth factors to maintain bone mass in microgravity conditions but it can be too expensive to maintain over longer periods of time besides the systematic effects of such treatments [3]. Considering the effects of microgravity are partially attributed to lack of mechanical force on bone tissue, which alters gene expression, reduction in transcription factors and growth factors. Furthermore, lack of gravity effects cell growth, proliferation, differentiation, cytoskeleton polymerization and cellular morphology [4, 5]. Thus to reverse these adverse effects on bone physiology, it is important to provide cells with mechanical stimulus which can provide essential mechanical signal for cells to counter the effects of microgravity. Ultrasound acoustic vibrations can be readily applied in, in vivo and human studies and has shown anabolic effects on osteopenic bone tissue [6]. Furthermore, ultrasound is a non-invasive and more target specific treatment relative to cyclic strain and vibration. The objective of this study is to see effects of low intensity pulsed ultrasound (LIPUS) on disused bone model and osteogenic activity of osteoblast cells cultures in simulated microgravity. This will help us understand that effects of ultrasound on microgravity and mechanotransduction pathway responsible for anabolic effect on bone cells.
APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "Bones – Growth"

1

Tao, Yang, Victor Alchanatis, and Yud-Ren Chen. X-ray and stereo imaging method for sensitive detection of bone fragments and hazardous materials in de-boned poultry fillets. United States Department of Agriculture, January 2006. http://dx.doi.org/10.32747/2006.7695872.bard.

Full text
Abstract:
As Americans become increasingly health conscious, they have increased their consumptionof boneless white and skinless poultry meat. To the poultry industry, accurate detection of bonefragments and other hazards in de-boned poultry meat is important to ensure food quality andsafety for consumers. X-ray imaging is widely used for internal material inspection. However,traditional x-ray technology has limited success with high false-detection errors mainly becauseof its inability to consistently recognize bone fragments in meat of uneven thickness. Today’srapid grow-out practices yield chicken bones that are less calcified. Bone fragments under x-rayshave low contrast from meat. In addition, the x-ray energy reaching the image detector varieswith the uneven meat thickness. Differences in x-ray absorption due to the unevenness inevitablyproduce false patterns in x-ray images and make it hard to distinguish between hazardousinclusions and normal meat patterns even by human visual inspection from the images.Consequently, the false patterns become camouflage under x-ray absorptions of variant meatthickness in physics, which remains a major limitation to detecting hazardous materials byprocessing x-ray images alone.Under the support of BARD, USDA, and US Poultry industries, we have aimed todeveloping a new technology that uses combined x-ray and laser imaging to detect bonefragments in de-boned poultry. The technique employs the synergism of sensors of differentprinciples and has overcome the deficiency of x-rays in physics of letting x-rays work alone inbone fragment detection. X-rays in conjunction of laser-based imaging was used to eliminatefalse patterns and provide higher sensitivity and accuracy to detect hazardous objects in the meatfor poultry processing lines.Through intensive research, we have met all the objectives we proposed during the researchperiod. Comprehensive experiments have proved the concept and demonstrated that the methodhas been capable of detecting frequent hard-to-detect bone fragments including fan bones andfractured rib and pulley bone pieces (but not cartilage yet) regardless of their locations anduneven meat thickness without being affected by skin, fat, and blood clots or blood vines.
APA, Harvard, Vancouver, ISO, and other styles
2

Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2005. http://dx.doi.org/10.21236/ada443762.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada429530.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Gilsanz, Vicente. Bone Growth Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2003. http://dx.doi.org/10.21236/ada430393.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2002. http://dx.doi.org/10.21236/ada416003.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Gilsanz, Vicente. Bone Growth, Mechanical Stimulus and IGF-I. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada481973.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Nickerson, Nicole. Transforming Growth Factor Beta Signaling in Growth of Estrogen-Insensitive Metastatic Bone Lesions. Fort Belvoir, VA: Defense Technical Information Center, January 2012. http://dx.doi.org/10.21236/ada558405.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Sumrall, Jr, and George L. Enlistment and Reenlistment Bonus Debts: Can the Growth Be Curbed? Fort Belvoir, VA: Defense Technical Information Center, April 1989. http://dx.doi.org/10.21236/ada209070.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Knudsen, Beatrice S. Hepatocyte Growth Factor and Interleukin-6 in Prostate Cancer Bone Metastasis. Fort Belvoir, VA: Defense Technical Information Center, March 2005. http://dx.doi.org/10.21236/ada435856.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Knudsen, Beatrice S. Hepatocyte Growth Factor and Interleukin-6 in Prostate Cancer Bone Metastasis. Fort Belvoir, VA: Defense Technical Information Center, March 2004. http://dx.doi.org/10.21236/ada428439.

Full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Bones – Growth' for particular source types:
Journal articles Dissertations / Theses Books
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography