Relevant bibliographies by topics / Bone formation

Academic literature on the topic 'Bone formation'

Author: Grafiati
Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Bone formation"

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Stützle, H., K. Hallfeldt, H. Mandelkow, S. Keßler, and L. Schweiberer. "Bone substitutes and bone formation." Der Orthopäde 27, no. 2 (February 1998): 118–25. http://dx.doi.org/10.1007/pl00003477.

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Tsartsalis, Athanasios, Charalambos Dokos, Georgia Kaiafa, Dimitris Tsartsalis, Antonios Kattamis, Apostolos Hatzitolios, and Christos Savopoulos. "Statins, bone formation and osteoporosis: hope or hype?" HORMONES 11, no. 2 (April 15, 2012): 126–39. http://dx.doi.org/10.14310/horm.2002.1339.

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Bellingham, F. Richard. "Endometrial Bone Formation." Australian and New Zealand Journal of Obstetrics and Gynaecology 36, no. 1 (February 1996): 109–10. http://dx.doi.org/10.1111/j.1479-828x.1996.tb02943.x.

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PUZAS, J. EDWARD, MICHAEL D. MILLER, and RANDY N. ROSIER. "Pathologic Bone Formation." Clinical Orthopaedics and Related Research &NA;, no. 245 (August 1989): 269???281. http://dx.doi.org/10.1097/00003086-198908000-00042.

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Schiergens, Tobias S., Angela Reichelt, Wolfgang E. Thasler, and Markus Rentsch. "Abdominal Bone Formation." Journal of Gastrointestinal Surgery 19, no. 3 (January 9, 2015): 579–80. http://dx.doi.org/10.1007/s11605-014-2737-4.

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Habal, Mutaz B. "Bone Engineering, Bone Formation, or just Refined Bone Regeneration." Journal of Craniofacial Surgery 14, no. 3 (May 2003): 265. http://dx.doi.org/10.1097/00001665-200305000-00001.

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Luriya, E. A., M. Owen, A. Ya Fridenshtein, S. A. Kuznetsov, E. N. Genkina, and V. V. Gosteva. "Bone formation in bone marrow organ cultures." Bulletin of Experimental Biology and Medicine 101, no. 4 (April 1986): 520–24. http://dx.doi.org/10.1007/bf00834432.

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Draenert, M. E., C. Martini, D. C. Watts, K. Draenert, and A. Wittig-Draenert. "Bone augmentation by replica-based bone formation." Dental Materials 36, no. 11 (November 2020): 1388–96. http://dx.doi.org/10.1016/j.dental.2020.08.005.

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Hoggard, Nathan K., and Linden E. Craig. "Medullary bone in male budgerigars (Melopsittacus undulatus) with testicular neoplasms." Veterinary Pathology 59, no. 2 (January 8, 2022): 333–39. http://dx.doi.org/10.1177/03009858211069126.

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Medullary bone is a calcium-rich, labile bone normally occurring in female birds with each egg-laying cycle. The stimulus for formation of medullary bone is, in part, the cyclic increase in serum estrogens produced by preovulatory ovarian follicles. Increased bone density due to formation of medullary bone, particularly in pneumatic bones, has been termed polyostotic hyperostosis, even if physiologic. This study investigated the formation of medullary bone in nonpneumatic (femur) and pneumatic (humerus) bones in sexually mature male budgerigars submitted for autopsy. Of the 21 sexually mature male budgerigars submitted for autopsy, 7 (33%) had medullary bone in 1 or more bones examined. All 7 male budgerigars with medullary bone had a testicular neoplasm, which was morphologically consistent with a testicular sustentacular cell tumor, seminoma, or interstitial cell tumor. Medullary bone was not present in the 14 cases with other diseases. Medullary bone formation in pneumatic and nonpneumatic bones can occur in male budgerigars with testicular neoplasms. Radiographic increases in medullary bone density, particularly in the humerus, could provide antemortem indication of testicular neoplasia in male budgerigars.
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Garrett, I., G. Gutierrez, and G. Mundy. "Statins and Bone Formation." Current Pharmaceutical Design 7, no. 8 (May 1, 2001): 715–36. http://dx.doi.org/10.2174/1381612013397762.

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Dissertations / Theses on the topic "Bone formation"

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Samizadeh, S. "Bone formation on calcium phosphate bone substitute materials." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19891/.

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A large number of bone substitute materials are available; for which some authors claim osteoconductivity and some osteoinductivity. In order to rank these materials an in vivo analysis was carried out. These materials were chosen based on their availability and claimed mode of action. Silicon substituted Hydroxyapatite (SiHA), Hydroxyapatite (HA), Resorbable Calcium Phosphate Silicon, Skelite [siliconstabilized tricalcium phosphate-based bone substitute], Pro Osteon 500R [coralline HA], BiIonic [Yttrium stabilized SiHA] and two non-calcium phosphate, Dimeneralised Bone Matrix (DBM) based biomaterials: Accell Connexus DBM putty and Grafton crunch DBM were implanted in sheep femoral condyle defects for 6 weeks. Implanted calcium phosphate (CaP) based biomaterials demonstrated superior bone formation in comparison with the DBM samples. Silicon within CaPs increased the rate of bone formation in vivo. Silicon substituted HA showed increased proliferation rate (P<0.05) of human marrow stromal cells compared to pure HA in vitro. Expression of osteoblastic marker genes RUNX2, Osterix and Osteopontin within the hMSCs indicated the differentiation of preosteoblasts into osteoblasts, and osteogenic development on both HA and SiHA. Expression of osteocalcin and bone sialoprotein genes on HA and SiHA samples indicated the activation of mineralisation process. Differentiation of hMSCs into osteoblasts in vitro suggested a role in promotion of osteoinduction by both HA and SiHA. Implantation of porous SiHA and HA in paraspinous muscle of sheep, exhibited new bone formation through osteoinduction. SiHA indicated significantly higher new bone formation (P<0.01) compared to HA. SiHA and HA biomaterials with higher strut porosity (30%) indicated greater bone formation (P<0.05). In conclusion, CaP based biomaterials demonstrate superior bone formation in comparison with DBM biomaterials. Silicon substitution within HA enhances the cellular activity of hMSCs. Osteoinduction was greatest on SiHA with higher strut porosity. This result is believed to be due to a combination of the effect of interconnected porosity and chemical composition of the bone substitute.
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Chen, Jinbiao Prince of Wales Clinical School UNSW. "In vitro and in vivo bone formation - assessment and application." Awarded by:University of New South Wales. Prince of Wales Clinical School, 2006. http://handle.unsw.edu.au/1959.4/24922.

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Background: Bone-grafting materials are required in orthopaedic surgery to treat bone defects. Bone formation assessment is required for the development of new strategies and approaches and for quality assurance and quality control of currently available materials. Approaches to the assessment of bone formation are yet to be systematically established, quantified and standardized. Aims: the overall aim of this study was to establish a set of comprehensive quantitative approaches for the assessment of bone formation and to evaluate the role of osteoblastic cells, growth factors, and scaffolds on this process. Materials & methods: both in vitro and in vivo parameters for osteoblast phenotype and bone formation were tested in osteosarcoma cell lines, Saos-2 and U2OS cells, mesenchymal cell line, C2C12 cells, primary adipose derived stromal cells (ADSCs), platelet rich plasma (PRP), and morselized bone grafts. The in vitro parameters used were measurement of alkaline phosphatase (ALP) activity, detection of bone nodules and biomineralization, and quantification of immunocytochemistry and conventional RT-PCR of osteoblast genotyping. In vivo parameters involved ectopic bone formation in nude mice and nude rats and a tibial defect model in nude rats. Histomorphometric and quantitative immunohistochemical analyses were also performed. Results: The in vitro characterization and ectopic bone formation capabiltity of Saos-2 and U2OS cells have been established. Saos-2 cell line, which presents many osteoblast genotype and phenotype, is a stable positive control for both in vitro and in vivo bone formation assessments. The measurement of ALP activity in both solid and liquid phases has been standardized. Both the genotype and phenotype of osteoblast lineage cells has been quantitatively assessed during the capability testing of ADSCs and PRP. Quantitative assessment of new bone formation and related protein markers in vivo has been successfully established through the testing of the biological properties of gamma irradiated morselized bone grafts. Conclusion: A comprehensive knowledge of the assessment of bone regeneration and formation in vitro and in vivo has been integrated and developed through years of study. A whole set of in vitro and in vivo approaches for the assessment of bone formation has been modified and standardized to best suit the different clinical applications. This thesis provides an outline of both in vitro and in vivo bone formation assessment and their clinical applications.
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Hrit, Manuela. "Acceleration of bone formation in distraction osteogenesis by bone morphogenetic protein-7." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=101142.

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The manipulation of the molecular mechanisms that govern distraction osteogenesis (DO) in order to increase the biomechanical strength of new bone and to accelerate its synthesis has been the topic of intense research during the past decades.
Bone morphogenetic proteins (BMPs) play an important role in bone formation. In this study, using a rabbit model of DO, the expressions of BMP's major intracellular signalling molecules, Smad proteins, was analyzed and correlated with the expression of BMP ligands and receptors. Based on these results, which confirmed post-receptor activity for the BMP signalling pathway during DO, we hypothesized that exogenous BMPs injected early in the distraction phase will accelerate bone formation in cases of DO. The cellular changes induced by local application of rhBMP-7 (OP-1) on bone formation were therefore investigated, as well as the possible pathways through which OP-1 was responsible for these effects.
The present study reveals that acceleration of bone formation can be attained after injection of OP-1 early during the distraction protocol. The enhanced bone formation, which occurs through the activation of numerous pathways, most likely depends on a non-vascular mechanism.
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Hu, Kai. "VEGF-Dependent Mechanisms Controlling Osteoblast Differentiation and Bone Formation During Bone Repair." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17467316.

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Osteoblast-derived vascular endothelial growth factor (VEGF) is important for bone development and postnatal bone homeostasis. Several studies have demonstrated that VEGF affects bone repair and regeneration; however, the cellular mechanisms by which it works are not fully understood. In this study, we investigated the functions of osteoblast-derived VEGF in healing of a cortical bone defect. In addition, how VEGF signaling modulates BMP2 functions during bone healing was also examined. To define the roles of osteoblast-derived VEGF in bone repair, a mouse tibial monocortical defect model was used. The effects of deleting Vegfa or Vefgr2 in osteoblast precursors and their descendants on the bone repair process were analyzed at various time points after surgery. To study how VEGF modulates the osteogenic activity of BMP2, BMP2, with or without the soluble VEGFR (sFlt1, VEGF decoy receptor), was delivered to the cortical defects in VE-cadherin-cre;tdTomato mice. The results indicate that osteoblast-derived VEGF is important at various stages during healing of the cortical defect. In the inflammation phase, osteoblast-derived VEGF controls neutrophil release into the circulation and macrophage-related angiogenic responses. VEGF is required, at optimal levels, for angiogenesis-osteogenesis coupling in areas where repair occurs by intramembranous ossification (IO). In this role, VEGF likely functions as a paracrine factor since deletion of Vegfr2 in osteoblast precursors and their progeny enhances osteoblastic maturation and mineralization. Furthermore, osteoblast- and hypertrophic chondrocyte-derived VEGF stimulates recruitment of blood vessels and osteoclasts, and promotes cartilage resorption at the repair site during the periosteal endochondral ossification stage. Finally, osteoblast-derived VEGF stimulates osteoclast formation in the final remodeling phase of the repair process. Our data also indicate that skeletal stem cells at different locations respond differently to BMP2, and that the osteogenic activity of BMP2 is modulated by extracellular VEGF. In the cortical defect, delivery of recombinant BMP2 inhibits intramembranous bone formation in the intramedullary space while it enhances endochondral bone formation in the injured periosteum. Inhibition of extracellular VEGF by sFlt1 reverses the inhibitory effects of BMP2 on intramembranous ossification-mediated bone repair. These findings add to the understanding of VEGF functions and provide a basis for clinical strategies to improve bone regeneration and treat cases of compromised bone healing.
Biological Sciences in Dental Medicine
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Williams, Bristol Marie. "Effects of tricalcium phosphate coated titanium on adjacent early bone formation." View the abstract Download the full-text PDF version, 2007. http://etd.utmem.edu/ABSTRACTS/2007-005-Williams-index.html.

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Thesis (M.S. )--University of Tennessee Health Science Center, 2007
Title from title page screen (viewed on July 28, 2008). Research advisor: Joo L. Ong, Ph.D. Document formatted into pages (iv, 36 p. : ill.). Vita. Abstract. Includes bibliographical references (p. 33-36).
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Lean, Jennifer Maree. "Mechanical stimulation of bone formation in the rat." Thesis, St George's, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263682.

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Moroz, Adam. "Reduced order modelling of bone resorption and formation." Thesis, De Montfort University, 2011. http://hdl.handle.net/2086/5409.

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The bone remodelling process, performed by the Bone Multicellular Unit (BMU) is a key multi-hierarchically regulated process, which provides and supports various functionality of bone tissue. It is also plays a critical role in bone disorders, as well as bone tissue healing following damage. Improved modelling of bone turnover processes could play a significant role in helping to understand the underlying cause of bone disorders and thus develop more effective treatment methods. Moreover, despite extensive research in the field of bone tissue engineering, bonescaffold development is still very empirical. The development of improved methods of modelling the bone remodelling process should help to develop new implant designs which encourage rapid osteointegration. There are a number of limitations with respect to previous research in the field of mathematical modelling of the bone remodelling process, including the absence of an osteocyte loop of regulation. It is within this context that this research presented in this thesis utilises a range of modelling methods to develop a framework for bone remodelling which can be used to improve treatment methods for bone disorders. The study concentrated on dynamic and steady state variables that in perspective can be used as constraints for optimisation problem considering bone remodelling or tissue remodelling with the help of the grafts/scaffolds.The cellular and combined allosteric-regulation approaches to modelling of bone turnover, based on the osteocyte loop of regulation, have been studied. Both approaches have been studied different within wide range of rate parameters. The approach to the model validation has been considered, including a statistical approach and parameter reduction approach. From a validation perspective the cellular class of modes is preferable since it has fewer parameters to validate. The optimal control framework for regulation of remodelling has been studied. Future work in to improve the models and their application to bone scaffold design applications have been considered. The study illustrates the complexity of formalisation of the metabolic processes and the relations between hierarchical subsystems in hard tissue where a relatively small number of cells are active. Different types/modes of behaviour have been found in the study: relaxational, periodical and chaotic modes. All of these types of behaviour can be found, in bone tissue. However, a chaotic or periodic modes are ones of the hardest to verify although a number of periodical phenomena have been observed empirically in bone and skeletal development. Implementation of the allosteric loop into cellular model damps other types of behaviour/modes. In this sense it improves the robustness, predictability and control of the system. The developed models represent a first step in a hierarchical model of bone tissue (system versus local effects). The limited autonomy of any organ or tissue implies differentiation on a regulatory level as well as physiological functions and metabolic differences. Implementation into the cellular phenomenological model of allosteric-like loop of regulation has been performed. The results show that the robustness of regulation can be inherited from the phenomenological model. An attempt to correlate the main bone disorders with different modes of behaviour has been undertaken using Paget’s disorder in bone, osteoporosis and some more general skeleton disorders which lead to periodical changes in bone mass, reported by some authors. However, additional studies are needed to make this hypothesis significant. The study has revealed a few interesting techniques. When studying a multidimensional phenomenon, as a bone tissue is, the visualisation and data reduction is important for analysis and interpretation of results. In the study two novel technical methods have been proposed. The first is the graphical matrix method to visualise/project the multidimensional phase space of variables into diagonal matrix of regular combination of two-dimensional graphs. This significantly simplifies the analysis and, in principle, makes it possible to visualise the phase space higher than three-dimensional. The second important technical development is the application of the Monte-Carlo method in combination with the regression method to study the character and stability of the equilibrium points of a dynamic system. The advantage of this method is that it enables the most influential parameters that affect the character and stability of the equilibrium point to be identified from a large number of the rate parameters/constants of the dynamic system. This makes the interpretation of parameters and conceptual verification of the model much easier.
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Gundle, Roger. "Microscopical and biochemical studies of mineralised matrix production by bone-derived cells." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282203.

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Wang, Jason Lee. "Effects of aging and remodeling on bone microdamage formation." Thesis, Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/37114.

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Skeletal fragility is characterized by low bone mass, negative changes in bone microarchitecture, and compromised tissue matrix properties, including accumulation of microdamage. Microdamage accumulates in vivo from daily physiological loading and is targeted for repair through a normal remodeling process, thus preventing microcrack growth and potential fracture. However, impaired remodeling is associated with aging and osteoporosis, resulting in an increased accumulation of microdamage which contributes to reduced bone mechanical properties. The current clinical method for assessing increased risk of fracture involves measuring bone mineral density (BMD) of the hip and spine, locations of trabecular bone where high rates of remodeling occur. The bisphosphonate alendronate (ALN) reduces clinical risk for fracture by significantly increasing BMD, but studies have shown a concomitant reduction in intrinsic properties that may be the underlying cause for recent reports of spontaneous fractures with long-term alendronate use. Another anti-resorptive agent called raloxifene (RAL) is a selective estrogen receptor modulator (SERM) and has been shown to modestly improve BMD while decreasing fracture risk to a similar degree as alendronate. The combination of RAL and ALN as a treatment for osteoporosis may provide the benefits of each drug without the negative effects of ALN. Therefore, the overall goal of this thesis was to address the effects of aging and anti-resorptive agents on the properties of bone through the formation of microdamage. Assessment of age-related effects on bone was conducted through quantification of microdamage progression. It was found that old bone results in greater incidences of microdamage progression, reflecting a compromised tissue matrix with reduced resistance to crack growth. Effects of combination treatment with RAL and ALN were evaluated through biomechanical testing, micro-CT imaging, and microdamage quantification. Results showed improved trabecular bone volume and ultimate load with positive effects on trabecular architecture. Combination treatment reduced the proportion of microdamage that may lead to catastrophic fracture, indicating an improvement in the local tissue matrix properties.
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Lozano-Carrascal, Naroa. "Topical Application of Bisphosphonates to Enhance Alveolar New Bone Formation." Doctoral thesis, Universitat Internacional de Catalunya, 2017. http://hdl.handle.net/10803/456485.

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This PhD thesis is a compendium of three publications, which sets out to broaden our knowledge and understanding of the topical application of bisphosphonates alone or mixed with a bone graft in alveolar bone defects, to evaluate the potential capacity of them to preserve/enhance alveolar new bone formation. In recent years, research has focused on improving bone substitutes to achieve faster and better regeneration by morphologic and biochemical modification. Bisphosphonates are a group of drugs that reduce bone resorption by inhibiting the formation, recruitment activity of mature osteoclasts; and promoting their apoptosis. In addition, some bisphosphonates enhance osteoblast differentiation and activity. Thence, it has been demonstrated that topical application of a bisphosphonates can minimize the bone resorption following muco-periostial flap surgery or in peri- implantitis; improve the osteoconductive and regenerative capacity of a biomaterial; prevent the surface resorption of onlay bone grafts; or reduce post-extraction dimensional changes. Mandibular second premolars (P2) and first molars (M1) were extracted from six Fox-Hound dogs. P2 were categorized as small defects (SD) and M1 as large defects (LD). Four random groups were created: SC (small control defects with MP3®), ST (small test defects MP3® + pamidronate), LC (large control defects with MP3®), and LT (large test defects MP3® + pamidronate). At four and eight weeks of healing the percentages of new bone formation (NB), residual grafts (RG) and connective tissue (CT) were analysed by histology and histomorphometric analysis. To complement the information already obtained from histological analysis, the samples were evaluated through scanning electron microscopy (SEM), and Energy dispersive X-ray spectroscopy (EDX), to identify the chemical elements present into the biomaterial and surrounding tissues, for understanding the biomaterial’s degradation process. The study was complemented with a systemic literature review of the articles published between January 2000 and December 2016, that evaluated in vivo the effects of the topical application of bisphosphonates on bone regeneration/preservation in alveolar defects. A total of 154 abstracts were identified, of which 18 potentially relevant articles were selected; a final total of nine papers were included for analysis. Histomorphometric and histologic analysis of the present pilot study demonstrated that after 4 and 8 weeks of healing, higher new bone formation for test groups (ST and LT) treated topically with pamidronate, compared with SC and LC respectively; residual graft was significantly higher in both control groups (SC and LC) compared to test (ST and LT) groups; and connective tissue percentage was higher in large defects (LC and LT) compared to small defects (SC and ST). SEM analysis revealed more mineralized bone in test groups (ST and LT) compared with control groups, demonstrated by higher percentages of Ca obtained from EDX spectroscopy. Within the limitations of this experimental study, the findings suggest that porcine xenografts (MP3®) modified with pamidronate favours the new bone formation and increased the porcine xenograft substitution/replacement after 4 and 8 weeks of healing. These results are in accordance with the conclusions obtained from the systematic review. Despite the comparison of the findings of the selected studies was made difficult by the heterogeneity of the articles, the topical application of bisphosphonate solution would appear to favour new bone formation in alveolar defects, and boosts the regenerative capacities of biomaterials resulting in increased bone density.
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Books on the topic "Bone formation"

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Bronner, Felix, and Mary C. Farach-Carson. Bone Formation. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1.

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Percival, Christopher J., and Joan T. Richtsmeier, eds. Building Bones: Bone Formation and Development in Anthropology. Cambridge: Cambridge University Press, 2017. http://dx.doi.org/10.1017/9781316388907.

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Rosen, Vicki. The cellular and molecular basis of bone formation and repair. New York: Springer, 1995.

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1957-, Thies Robert Scott, ed. The cellular and molecular basis of bone formation and repair. Austin: R.G. Landes, 1995.

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Zhou, Hong. Cement line formation in rat femoral bone. [Toronto: s.n.], 1993.

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Zhou, Hong. Cement line formation in rat femoral bone. Ottawa: National Library of Canada, 1993.

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Ellies, Lesley Gae *. The effects of interleukin-1 on bone formation in vitro. [Toronto: Faculty of Dentistry, University of Toronto], 1991.

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Peel, Sean A. F. The influence of substratum modification on interfacial bone formation in vitro. [Toronto: Faculty of Dentistry, University of Toronto], 1995.

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Hsieh, Yao-Dung. Factors affecting bone formation following molar tooth extraction in the rat. Manchester: University of Manchester, 1995.

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Gottlow, Jan. New attachment formation by guided tissue regeneration. Göteborg, Sweden: University of Göteborg, Faculty of Odontology, Dept. of Periodontology, 1986.

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Book chapters on the topic "Bone formation"

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Bukka, Prasanna, Marc D. McKee, and Andrew C. Karaplis. "Molecular Regulation of Osteoblast Differentiation." In Bone Formation, 1–17. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_1.

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Karin, Norman J., and Mary C. Farach-Carson. "In Vitro Regulation of Osteoblast Activity." In Bone Formation, 18–43. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_2.

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Hurley, Marja M., and Joseph A. Lorenzo. "Systemic and Local Regulators of Bone Remodeling." In Bone Formation, 44–70. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_3.

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Puzas, J. Edward. "Site-specific Mineralized Matrix Formation by Osteoblasts." In Bone Formation, 71–78. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_4.

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Turner, Charles H. "Biomechanical Aspects of Bone Formation." In Bone Formation, 79–105. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_5.

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Martin, T. John, and Ego Seeman. "Reduced Bone Formation in the Pathogenesis of Bone Fragility." In Bone Formation, 106–19. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_6.

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McLean, William, and Bjorn R. Olsen. "Diseases of Excess Bone Formation." In Bone Formation, 120–37. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_7.

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Peterlik, Meinrad. "Targeting the Osteoblast for Prevention and Treatment of Bone Diseases." In Bone Formation, 138–53. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3777-1_8.

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Krstić, Radivoj V. "Bony Tissue. Secondary Bone Formation." In General Histology of the Mammal, 214–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70420-8_105.

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Order, Stanley E., and Sarah S. Donaldson. "Heterotopic Bone Formation." In Radiation Therapy of Benign Diseases, 144–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-58719-1_54.

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Conference papers on the topic "Bone formation"

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Mouchantaf, Fares G., Patrick Scanlon, and Andrew Villanueva. "Abnormal Bone Formation Within The Lung." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4498.

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Dry, Carolyn M. "Polymer-ceramic composite that mimics bone formation." In 1999 Symposium on Smart Structures and Materials, edited by Yoseph Bar-Cohen. SPIE, 1999. http://dx.doi.org/10.1117/12.349705.

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Jabbari, E. "Engineering bone formation with peptidomimetic hybrid biomaterials." In 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5332681.

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Nowlan, Niamh C., Paula Murphy, and Patrick J. Prendergast. "Mechanical Stimuli Resulting From Embryonic Muscle Contractions Promote Avian Periosteal Bone Collar Formation." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-172077.

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Mechanical forces due to muscle contractions play an essential role in embryonic skeletal development. In neuromuscular conditions such as congenital myotonic dystrophy, where movement of the fetus in utero is reduced or absent, the bones and joints of the newborn often show malformations [1]. In this paper, we examine the effect of muscle contractions on embryonic bone development. We propose the hypothesis that mechanical loading due to muscle contractions promotes periosteal ossification and we test this hypothesis using computational and experimental methods. A set of FE analyses were performed using anatomically realistic morphologies and loading conditions, at several timepoints during development, in order to identify biophysical stimuli active during bone formation. Avian immobilization experiments were performed to examine bone growth in the absence of skeletal muscle contractions.
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Abbassi, Sam, Ming Ding, Feifei Li, Yaojuan Lu, Guojun Wu, and Qiping Zheng. "Abstract 3919: Potential roles of oncogenic ΔNP63α during bone and bone cancer formation." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3919.

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Hao, Sijie, Gong Cheng, Yuan Wan, Yiqiu Xia, Donna M. Sosnoski, Andrea M. Mastro, and Si-Yang Zheng. "A bone-on-a-chip microdevice for long-term spontaneous 3D bone tissue formation and cancer bone metastasis." In 2017 19th International Conference on Solid-State Sensors, Actuators and Microsystems (TRANSDUCERS). IEEE, 2017. http://dx.doi.org/10.1109/transducers.2017.7994514.

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Ito, A., K. Ojima, K. Kawamura, N. Ichinose, P. Layrolle, K. Hayashi, and T. Tateishi. "ZINC-RELEASING CALCIUM PHOSPHATE CERAMICS STIMULATING BONE FORMATION." In Proceedings of the 12th International Symposium on Ceramics in Medicine. WORLD SCIENTIFIC, 1999. http://dx.doi.org/10.1142/9789814291064_0136.

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Morinobu, M., M. Ishijima, S. R. Rittling, K. Tsuji, H. Yamamoto, A. Nifuji, D. T. Denhardt, and M. Noda. "OSTEOPONTIN-DEFICIENCY REDUCES BONE FORMATION UNDER MECHANICAL STRESS." In 3rd International Conference on Osteopontin and SIBLING (Small Integrin-Binding Ligand, N-linked Glycoprotein) Proteins, 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.319.

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Siviero, Bethania C. T., Elizabeth Rega, Arthur V. Chadwick, and Leonard R. Brand. "PSEUDOPATHOLOGIES IN EDMONTOSAURUS ANNECTENS BONES: BIOGENETIC AND DIAGENETIC BONE ALTERATIONS FROM A MONOSPECIFIC BONE BED IN THE LANCE FORMATION, WYOMING." In GSA Annual Meeting in Phoenix, Arizona, USA - 2019. Geological Society of America, 2019. http://dx.doi.org/10.1130/abs/2019am-341376.

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Simmons, Craig A., Shaker A. Meguid, and Robert M. Pilliar. "Modelling of Mechanically Regulated Tissue Formation Around Bone-Interfacing Implants." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2495.

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Abstract The clinical success of bone-interfacing orthopaedic and dental implants is dependent on adequate fixation of the implant by mechanical interlock with ingrown bone tissue (i.e., functional osseointegration). The rate and reliability with which osseointegration is achieved are influenced by a number of factors, including the surface geometry of the implant (Thomas and Cook, 1985; Simmons et al., 1999). However, the mechanisms by which implant surface geometry influences initial bone formation remain unresolved. Identifying the factors that allow bone-interfacing implants to osseointegrate more rapidly and reliably should lead to improvements in their use and design.
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Reports on the topic "Bone formation"

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Kim, Yongbaek. Participation of Bone Marrow-Derived Cells in the Formation of Tumor-Associated Stroma During Lung Cancer. Fort Belvoir, VA: Defense Technical Information Center, June 2011. http://dx.doi.org/10.21236/ada552887.

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Baylink, David J. Molecular Mechanisms of Soft Tissue Regeneration and Bone Formation in Mice: Implications in Fracture Repair and Wound Healing in Humans. Fort Belvoir, VA: Defense Technical Information Center, October 2003. http://dx.doi.org/10.21236/ada420947.

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Baylink, David J. Molecular Mechanisms of Soft Tissue Regeneration and Bone Formation in Mice: Implications in Fracture Repair and Wound Healing in Humans. Fort Belvoir, VA: Defense Technical Information Center, October 2000. http://dx.doi.org/10.21236/ada391335.

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Mohan, Subburaman. Molecular Mechanisms of Soft Tissue Regeneration and Bone Formation in Mice: Implication in Fracture Repair and Wound Healing in Humans. Fort Belvoir, VA: Defense Technical Information Center, April 2008. http://dx.doi.org/10.21236/ada482393.

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Canellas, João Vitor, Luciana Drugos, Fabio Ritto, Ricardo Fischer, and Paulo Jose Medeiros. What grafting materials produce greater new bone formation in maxillary sinus floor elevation surgery? A systematic review and network meta-analysis protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0106.

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Sela, Shlomo, and Michael McClelland. Investigation of a new mechanism of desiccation-stress tolerance in Salmonella. United States Department of Agriculture, January 2013. http://dx.doi.org/10.32747/2013.7598155.bard.

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Low-moisture foods (LMF) are increasingly involved in foodborne illness. While bacteria cannot grow in LMF due to the low water content, pathogens such as Salmonella can still survive in dry foods and pose health risks to consumer. We recently found that Salmonella secretes a proteinaceous compound during desiccation, which we identified as OsmY, an osmotic stress response protein of 177 amino acids. To elucidate the role of OsmY in conferring tolerance against desiccation and other stresses in Salmonella entericaserovarTyphimurium (STm), our specific objectives were: (1) Characterize the involvement of OsmY in desiccation tolerance; (2) Perform structure-function analysis of OsmY; (3) Study OsmY expression under various growth- and environmental conditions of relevance to agriculture; (4) Examine the involvement of OsmY in response to other stresses of relevance to agriculture; and (5) Elucidate regulatory pathways involved in controlling osmY expression. We demonstrated that an osmY-mutant strain is impaired in both desiccation tolerance (DT) and in long-term persistence during cold storage (LTP). Genetic complementation and addition of a recombinantOsmY (rOsmY) restored the mutant survival back to that of the wild type (wt). To analyze the function of specific domains we have generated a recombinantOsmY (rOsmY) protein. A dose-response DT study showed that rOsmY has the highest protection at a concentration of 0.5 nM. This effect was protein- specific as a comparable amount of bovine serum albumin, an unrelated protein, had a three-time lower protection level. Further characterization of OsmY revealed that the protein has a surfactant activity and is involved in swarming motility. OsmY was shown to facilitate biofilm formation during dehydration but not during bacterial growth under optimal growth conditions. This finding suggests that expression and secretion of OsmY under stress conditions was potentially associated with facilitating biofilm production. OsmY contains two conserved BON domains. To better understand the role of the BON sites in OsmY-mediated dehydration tolerance, we have generated two additional rOsmY constructs, lacking either BON1 or BON2 sites. BON1-minus (but not BON2) protein has decreased dehydration tolerance compared to intact rOsmY, suggesting that BON1 is required for maximal OsmY-mediated activity. Addition of BON1-peptide at concentration below 0.4 µM did not affect STm survival. Interestingly, a toxic effect of BON1 peptide was observed in concentration as low as 0.4 µM. Higher concentrations resulted in complete abrogation of the rOsmY effect, supporting the notion that BON-mediated interaction is essential for rOsmY activity. We performed extensive analysis of RNA expression of STm undergoing desiccation after exponential and stationary growth, identifying all categories of genes that are differentially expressed during this process. We also performed massively in-parallel screening of all genes in which mutation caused changes in fitness during drying, identifying over 400 such genes, which are now undergoing confirmation. As expected OsmY is one of these genes. In conclusion, this is the first study to identify that OsmY protein secreted during dehydration contributes to desiccation tolerance in Salmonella by facilitating dehydration- mediated biofilm formation. Expression of OsmY also enhances swarming motility, apparently through its surfactant activity. The BON1 domain is required for full OsmY activity, demonstrating a potential intervention to reduce pathogen survival in food processing. Expression and fitness screens have begun to elucidate the processes of desiccation, with the potential to uncover additional specific targets for efforts to mitigate pathogen survival in desiccation.
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Koay, Chun Giok, Teng Fung Looi, and Rohit Kunnath Menon. Systematic review of studies evaluating the microbiome of periimplantitis using next generation sequencing techniques. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0111.

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Review question / Objective: This systematic review aims to summarize and critically analyse the methodology and findings of studies which have utilized sequencing techniques to elucidate the microbial profiles of peri-implantitis. Condition being studied: Peri-implantitis is defined as an infection of the peri-implant tissues accompanied by suppuration and clinically significant progressing crestal bone loss after the adaptive phase, leading to decreased osseointegration and pocket formation. Eligibility criteria: Original studies investigating the microbiome of peri-implant tissues through next-generation DNA sequencing methods will be included. Culture-based study, conference papers, review articles, studies regarding peri-implantitis associated with other systematic factors (smoking, diabetes mellitus, etc.), articles that examine only specific microorganisms will be excluded from this systematic review. Non-English language articles and research conducted on non-human specimens will be excluded.
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I. A. Parshikov, Igor A. OXIDATION OF GERANYL-N-PHENYLCARBAMATE BY FUNGUS BEAUVERIA BASSIANA WITH AIM TO OBTANING OF NEW ANTI-CANCER DRUGS. Intellectual Archive, October 2020. http://dx.doi.org/10.32370/iaj.2427.

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The microbial oxidation of geranyl-N-phenylcarbamate by fungus Beauveria bassiana was investigated. Oxidation of the C3 – C4 double bond of the parent molecule leads to regioselective formation of O-3,4-epoxyheranyl-N-phenylcarbamate in 30 % yield
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Roth, Justine P. Isotopic Studies of O-O Bond Formation During Water Oxidation (SISGR). Office of Scientific and Technical Information (OSTI), March 2015. http://dx.doi.org/10.2172/1171645.

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Jayatissa, Kuruppu. A Metal-Free Approach to Biaryl Compounds: Carbon-Carbon Bond Formation from Diaryliodonium Salts and Aryl Triolborates. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.2226.

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