Relevant bibliographies by topics / Bone dissection

Academic literature on the topic 'Bone dissection'

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Published: 14 March 2023

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Journal articles on the topic "Bone dissection"

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Prasad, K. C., Prathyusha K., Shreeharsha Maruvala, Harshita T. R., Indu Varsha Gopi, and Sumanth K. R. "Impact of temporal bone dissection on the understanding anatomy of the ear among medical students." International Journal of Otorhinolaryngology and Head and Neck Surgery 4, no. 6 (October 24, 2018): 1489. http://dx.doi.org/10.18203/issn.2454-5929.ijohns20184365.

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<p class="abstract">The aim was to study the impact of temporal bone dissection demonstrations on understanding anatomy of the ear among medical students. During a period of six months from October 2017 to March 2018, 10 cadaveric temporal bones dissections were demonstrated using ZEISS microscope and in the presence of medical students headed by a Consultant Otolaryngologist in the department of ENT, Sri Devaraj URS Medical College and Research Centre, Kolar. Anatomy of the middle ear and inner ear and various operative procedures were demonstrated. The students were divided into 2 groups. Group I comprised students who attended the temporal bone dissection and Group II included those who didn’t attend dissection. After the session both the groups were assessed by the consultant. Scores were given to the group individuals based on the ability to answer the questions. 10 Temporal bone dissections were demonstrated in 6 months period to medical students who were divided into 2 groups based on their attendance of the demonstration. The students of both groups were assessed. Scores were given by Likert scale-5point scale question. The results of our study proved that those students who attended the temporal bone dissection (Group-I) had better understanding of the anatomy and operative procedures of the ear as compared to students in group II. Demonstration of temporal bone dissection to the medical students had a good impact on their understanding of the three dimensional anatomy of the ear.</p>
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Golding-Wood, David G. "Temporal bone dissection for display." Journal of Laryngology & Otology 108, no. 1 (January 1994): 3–8. http://dx.doi.org/10.1017/s0022215100125691.

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Increasing concern with medicolegal issues has heightened the need for surgical simulation in training. Familiarity with the surgical anatomy of the temporal bone is essential for effective and safe otological surgery. Refinement of surgical technique and intimate knowledge of temporal bone anatomy can be gained by accurate dissection. The products of such endeavours are both illustrative and instructive. The issues, methods and techniques necessary for display of anatomical dissections are discussed.
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Natarajan, B., and A. Baxter. "Preparation of a temporal bone exhibit." Journal of Laryngology & Otology 108, no. 1 (January 1994): 9–12. http://dx.doi.org/10.1017/s0022215100125708.

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Temporal bone dissection forms an important aspect in the training of an otolaryngologist. The more dissection one does the more confident one is in the operating room. The aim of this paper is to advise in the preparation of temporal bones for the purpose of display, exhibition or competition. The practical aspects of dissection are deliberately avoided concentrating on the selection of bones pre- and post-dissection, preparation of the bones, fixing and display of vessels and nerves and also mounting and lighting for exhibition purposes.The temporal bone laboratory should be well equipped with a microscope, a power drill with a range of cutting and polishing burrs, a range of fine instruments, a suction machine with different sizes of suction tips, water source, a place to store the bones etc., thus creating an ideal environment for temporal bone dissection.
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Scott, A., S. A. A. Sadek, M. C. Garrido, and R. G. Courteney-Harris. "Temporal bone dissection: a possible route for prion transmission?" Journal of Laryngology & Otology 115, no. 5 (May 2001): 374–75. http://dx.doi.org/10.1258/0022215011907901.

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The aim of this study was to determine whether neural tissue is present in the bone ‘dust’ given off during temporal bone drilling. Bone ‘dust’ from three temporal bone dissections was collected and examined. Evidence of neural tissue was present in two out of the three specimens. Neural tissue is present in the bone dust given off during temporal bone drilling. This poses the question as to the risk of prion transmission during such dissection.
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Shlyakhtov, M. I., and K. G. Naumov. "Use of modern energetic methods of bone tissue removal during endoscopic dacryocystorhinostomy." Reflection 11, no. 1 (July 15, 2021): 61–67. http://dx.doi.org/10.25276/2686-6986-2021-1-61-67.

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The article presents the results of surgical treatment of 22 patients with chronic dacryocystitis accompanied by proximal obstruction of the nasolacrimal duct. A new method of nasolacrimal anastomosis bone window during endonasal endoscopic dacryocystorhinostomy using ultrasound bone dissection is described. The questions of operation technique using SONOCA 185 ultrasound bone dissector and specific features of postoperative treatment are discussed. The obtained data allow to conclude that low temperature process of bone dissection with ultrasound cavitation gives a possibility of adequate bone window formation, reduces surgical trauma of bone structures, surrounding soft tissues and nasal cavity vessels as well as reduces the risk of bleeding and operation time needed for its stopping. It also enables better healing of soft tissues in shorter terms, provides anatomical and functional success of operation in 91 % of cases. Key words: chronic dacryocystitis; endonasal endoscopic dacryocystorhinostomy; ultrasound bone dissection; dacryostoma.
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Hertzano, Ronna. "Temporal Bone Dissection Guide." Otology & Neurotology 32, no. 8 (October 2011): 1191. http://dx.doi.org/10.1097/mao.0b013e31822a1c99.

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Banerjee, Santanu. "Manual of temporal bone dissection." Indian Journal of Otolaryngology and Head and Neck Surgery 52, no. 1 (December 1999): 120. http://dx.doi.org/10.1007/bf02996461.

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Frithioff, Andreas, Martin Frendø, Kenneth Weiss, Søren Foghsgaard, David Bue Pedersen, Mads Sølvsten Sørensen, and Steven Arild Wuyts Andersen. "Effect of 3D-Printed Models on Cadaveric Dissection in Temporal Bone Training." OTO Open 5, no. 4 (October 2021): 2473974X2110650. http://dx.doi.org/10.1177/2473974x211065012.

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Objective Mastoidectomy is a cornerstone in the surgical management of middle and inner ear diseases. Unfortunately, training is challenged by insufficient access to human cadavers. Three-dimensional (3D) printing of temporal bones could alleviate this problem, but evidence on their educational effectiveness is lacking. It is largely unknown whether training on 3D-printed temporal bones improves mastoidectomy performance, including on cadavers, and how this training compares with virtual reality (VR) simulation. To address this knowledge gap, this study investigated whether training on 3D-printed temporal bones improves cadaveric dissection performance, and it compared this training with the already-established VR simulation. Study Design Prospective cohort study of an educational intervention. Setting Tertiary university hospital, cadaver dissection laboratory, and simulation center in Copenhagen, Denmark. Methods Eighteen otorhinolaryngology residents (intervention) attending the national temporal bone dissection course received 3 hours of mastoidectomy training on 3D-printed temporal bones. Posttraining cadaver mastoidectomy performances were rated by 3 experts using a validated assessment tool and compared with those of 66 previous course participants (control) who had received time-equivalent VR training prior to dissection. Results The intervention cohort outperformed the controls during cadaver dissection by 29% ( P < .001); their performances were largely similar across training modalities but remained at a modest level (~50% of the maximum score). Conclusion Mastoidectomy skills improved from training on 3D-printed temporal bone and seemingly more so than on time-equivalent VR simulation. Importantly, these skills transferred to cadaveric dissection. Training on 3D-printed temporal bones can effectively supplement cadaver training when learning mastoidectomy.
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George, A. P., and R. De. "Review of temporal bone dissection teaching: how it was, is and will be." Journal of Laryngology & Otology 124, no. 2 (December 3, 2009): 119–25. http://dx.doi.org/10.1017/s0022215109991617.

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AbstractObjective:We aimed to review the history of anatomical dissection, and to examine how modern educational techniques will change the way temporal bone dissection is taught to otolaryngology trainees.Method:Review of the literature using Medline, Embase and PubMed database searches.Results:Temporal bone anatomy has traditionally been taught using cadaveric specimens. However, resources such as three-dimensional reconstructed models and ‘virtual reality’ temporal bone simulators have a place in educating the otolaryngology trainee.Conclusion:We should encourage the use of fresh frozen cadaveric temporal bone specimens for future otologists. Artificial three-dimensional models and virtual reality temporal bone simulators can be used to educate junior trainees, thus conserving the scarce resource of cadaveric bones.
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Somjee, Shehnaz. "The Somjee-Crabtree temporal bone support clamp." Journal of Laryngology & Otology 111, no. 1 (January 1997): 54–55. http://dx.doi.org/10.1017/s0022215100136412.

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AbstractA new device is introduced for holding temporal bones during dissection. It is structurally very different, more practical and effective for securing the bone than the temporal bone holding bowl which has been in use so far.
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Dissertations / Theses on the topic "Bone dissection"

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Bryan, Jason Allen. "A virtual temporal bone dissection simulator." Connect to resource, 2001. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1133977208.

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Thesis (M.S.)--Ohio State University, 2001.
Advisor: Roger Crawfis, Dept. of Computer and Information Science. Includes bibliographical references (leaves 51-52). Available online via OhioLINK's ETD Center
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Wang, Cathy Ting-Peng. "Molecular dissection of RANKL signaling pathways in osteoclasts." University of Western Australia. School of Surgery and Pathology, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0037.

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[Truncated abstract] Bone remodeling is intricately regulated by osteoclast-mediated bone resorption and osteoblast-mediated bone formation. The elevation in osteoclast number and/or activity is a major hallmark of several common pathological bone disorders including post-menopausal osteoporosis, osteoarthritis, Paget's disease, and tumour-mediated osteolysis. Receptor activator of nuclear factor kappa B ligand (RANKL) is a key cytokine for osteoclast differentiation and activation. The association of RANKL to its cognate receptor, RANK, which is expressed on osteoclast precursors and mature osteoclasts, is essential for osteoclast formation and activation. The intimate interaction between RANKL and RANK triggers the activation of a cascade of signal transduction pathways including NF-κB, NFAT, MAPK and PI3 kinase. Although osteoclast signaling pathways have been intensively studied, the precise molecules and signaling events which underlie osteoclast differentiation and function remain unclear. In order to dissect the molecular mechanism(s) regulating osteoclast differentiation and activity, this thesis herein explores the key RANKL/RANK-mediated signaling pathways. Four truncation mutants within the TNF-like domain of RANKL [(aa160-302), (aa160-268), (aa239-318) and (aa246-318)] were generated to investigate their potential binding to RANK and the activation to RANK-signal transduction pathways. All were found to differentially impair osteoclastogenesis and bone resorption as compared to the wild-type RANKL. The impaired function of the truncation mutants of RANKL on osteoclast formation and function correlates with their reduced ability to activate crucial RANK signaling including NF-κB, IκBα, ERK and JNK. Further analysis revealed that the truncation mutants of RANKL exhibited differentially affinity to RANK as observed by in vitro pull-down assays. ... It is possible that Bryostatin 1 acts via upregulation of a fusion mechanism as the RANKL-induced OCLs are morphologically enlarged, exhibiting increased nuclei number expressing high level of DC-Stamp. Furthermore, Rottlerin was shown to inhibit NF-κB activity, whereas Bryostatin 1 showed the opposing effects. Both inhibitor and activator were also found to modulate other key osteoclastic signaling pathways including NFAT and total c-SRC. These findings implicate, for the first time, Protein Kinase C delta signaling pathways in the modulation of RANKL-induced osteoclast differentiation and activity. Taken together, the studies presented in this thesis provide compelling molecular, biochemical and morphological evidence to suggest that: (1) RANKL mutants might potentially serve as peptide mimic to inhibit RANKL-induced signaling, osteoclastogenesis and bone resorption. (2) A cross talk mechanism between extracellular Ca2+ and RANKL exist to regulate on osteoclast survival. (3) TPA suppressed RANKL-induced osteoclastogenesis predominantly during the early stage of osteoclast differentiation via modulation of NF-κB. (4) Selective inhibition of Protein Kinase C signaling pathways involved in osteoclastogenesis might be a potential treatment method for osteoclast-related bone diseases. (5) Protein Kinase C delta signaling pathways play a key role in regulating osteoclast formation and function.
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Lenz, Daniel. "Dissecting the heterogeneity of murine mesenchymal bone marrow stromal cells." Doctoral thesis, Humboldt-Universität zu Berlin, 2020. http://dx.doi.org/10.18452/21017.

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Knochenmarks-Stromazellen sind in den letzten Jahren in den Fokus der Forschung gerückt. Es konnte gezeigt werden, dass sie durch Bereitstellung von Überlebenssignalen essenziell für die Erhaltung hämatopoetischer Nischen sind. Stromales Interleukin-7 (IL-7) konnte dabei für T Zellen als Überlebenssignal identifiziert werden. Gemeinsam ist allen Stromazellen die Expression des Oberflächenmarkers CD106/VCAM-1. Ein effizientes Protokoll erlaubte die qualitative wie quantitative Isolation von Stromazellen aus dem murinen Knochenmark mit anschließender ex vivo Microarray-Analyse. Die auf diese Weise ermittelten Kandidaten-Marker wurden auf Proteinebene via Histologie und (Hochdurchsatz-) Durchflusszytometrie validier. Dazu gehören z.B. die Marker CD1d, gas6 oder ANXA2R. CD1d wurde als guter Interimsmarker für VCAM-1+PECAM-1- Stromazellen identifiziert, wohingegen die IL-7-Produzenten in der Population von CD200int/BP 1+/CD73+/CD105- angereichert sind. Gleiches gilt für den Transkriptionsfaktor Prrx1. CD55, BP-1 and Cadherin-11 zeigten eine Expressionsmuster in Abhängigkeit des verwendeten IL-7-Reportermaus-Haplotyps. Für BP-1 und Cadherin 11 konnte die Abwesenheit von reifen Lymphozyten als Ursache des Feedbacks ausgeschlossen werden. Die Haplotypen der Reportermaus legten auch eine monoallele Expression des IL-7 nahe. Die Ergebnisse dieser Arbeit zeigen VCAM-1+ (IL-7+/-) Stromazellen als heterogene Population, wenn es nach der Vielzahl der möglichen exprimierten Marker geht. Zwischen vielen dieser Marker gibt es aber wiederum auf Zelloberflächenebene einen großen Überlapp. Die funktionelle Relevanz dieser Oberflächenmarker-Diversität wird in weiteren Arbeiten zu klären sein, gibt aber den Stromazellen ein breites Repertoire vor, um Interaktionen mit Lymphozyten zu initiieren, modulieren und inhibieren. Abschließend ist zu erwarten, dass diese Erkenntnisse in die klinische Behandlung der Stroma-Nischen in Autoimmun-Fragestellungen einfließen.
Bone marrow stromal cells receive increasing amounts of attention lately. They have been shown to support survival of hematopoietic stem cells as well as memory lymphocytes which is of great importance when targeting the perseverance of autoimmune diseases. CD4+ memory T lymphocytes reside in the proximity of VCAM-1 expressing stromal cells which provide them with survival signals such as Interleukin-7. Herein, a protocol was developed to quantitatively obtain VCAM-1+ and VCAM-1+ IL-7+/- stromal cells via enzymatic/mechanic digestion and cytoskeleton-inhibition. Ex vivo gene expression analysis was performed from sorted, pure cells with good recovery. Candidate genes/markers were validated in (high-throughput) flow cytometry and histological analysis including subsequent semi-automated colocalization was performed. CD1d was found to be good surrogate marker for VCAM-1+PECAM-1- non-endothelial stroma while the population of CD200int/BP-1+/CD73+/CD105- stromal cells is greatly enriched in IL-7 producers which was equally true for the stromal transcription factor Prrx1. CD55, BP-1 and Cadherin-11 were found to be differentially expressed in differing IL-7 reporter mice haplotypes. The reporter mice haplotypes revealed monoallelic expression features of IL-7. All methodologies suggest that VCAM-1+ as well as IL-7+/- stromal cells are heterogeneous by marker expression yet don’t cluster extensively in flow cytometry co-stains. The functional relevance of the marker diversity described in this thesis remains to be tested but insinuates a broad repertoire for bone marrow stroma cells for new interaction pathways with lymphocyte subsets. Ultimately, this knowledge will hopefully feedback to clinical questions of autoimmunity for targeted treatment of stromal niches.
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Tang, Kai Dun. "Dissecting the prostate cancer stem cell niche inside the bone marrow." Thesis, Queensland University of Technology, 2015. https://eprints.qut.edu.au/88935/1/Kai%20Dun_Tang_Thesis.pdf.

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Prostate cancer frequently metastasizes to bone, which becomes incurable; yet how cancer cells manage to migrate and grow inside the bone remains unknown. In this study I have discovered that both bone and fat cells within the bone marrow actively promote the survival and expansion of prostate cancer cells, and have subsequently developed approaches that can effectively inhibit these processes. Therefore, my work offers opportunities for the development of new prognostic and therapeutic approaches against metastatic prostate cancer and have the potential for improving the treatment outcome of the patients.
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Xiao, Lan. "Dissecting the role of sphingosine 1-phosphate-sphingosine 1-phosphate receptor 1 in inflammatory bone remodelling." Thesis, Queensland University of Technology, 2017. https://eprints.qut.edu.au/105357/1/Lan_Xiao_Thesis.pdf.

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This thesis provides potential new therapeutic approaches for the prevention and treatment of destructive bone diseases. It investigates the cellular and molecular mechanisms of abnormal bone remodelling under inflammation, therefore sheds light on the pathogenesis of inflammation induced bone loss.
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Lenz, Daniel [Verfasser], Andreas [Gutachter] Radbruch, Andreas [Gutachter] Thiel, and Enrico [Gutachter] Klotzsch. "Dissecting the heterogeneity of murine mesenchymal bone marrow stromal cells / Daniel Lenz ; Gutachter: Andreas Radbruch, Andreas Thiel, Enrico Klotzsch." Berlin : Humboldt-Universität zu Berlin, 2020. http://d-nb.info/1203623933/34.

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Joun, George Louis. "Dissecting the Cellular and Molecular Regulation of Chemo-Resistance and Epithelial to Mesenchymal Transition in Human Ovarian Carcinoma." Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/28748.

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Ovarian carcinoma is the malignant transformation of ovarian or fallopian tube epithelial cells. High-grade serous carcinoma (HGSOC) diagnosis carries a 30% 5-year survival. The development of chemotherapy resistance is common leading to relapse of a resistant tumour. Due to inherent genetic/chromosomal instability in HGSOC, these tumours are multi-clonal leading to treatment resistance and metastasis. It was aimed to understand the clonal heterogeneity in HGSOC cell lines through single cell molecular and physiological dissection. Chapter 3 describes the use of single cell derived strains, single cell lineage tracking and single cell RNA sequencing of HGSOC cell line AOCS15 confirming a clonal fitness model of carboplatin resistance, establishing that molecular and physiological heterogeneity results in the development of platinum resistant clones in HGSOC. In Chapter 4, the chromatin proteome was compared between carboplatin treated resistant and sensitive single cell strains during the DNA synthesis phase. Major differences between sensitive and resistant clones included DNA repair, nuclear lamina, and DNA repair. In Chapter 5, the Bone Morphogenetic Protein (BMP) signalling pathway was targeted in combination therapy, Combination of DMH1 and Carboplatin increased the response of resistant clones through heightening replication stress by dampening the S/G2 checkpoint. In Chapter 6, BMP signalling correlated with an epithelial to mesenchymal transition (EMT) phenotype in ovarian cancer tumours. BMP inhibition in HGSOC cell lines led to down-regulation of EMT transcription factors and loss of cell migration in vitro and in vivo. In conclusion, the data presented confirms the presence of dynamic chemo-resistant subclones in ovarian cancer that may be underlined and targetable by BMP signalling.
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MACETTI, GIOVANNI. "TOPOLOGICAL DESCRIPTORS ENABLING NOVEL DISSECTIONS OF ELECTRON POSITION AND SPIN PROPERTIES IN COMPLEX MOLECULAR SYSTEMS." Doctoral thesis, Università degli Studi di Milano, 2019. http://hdl.handle.net/2434/615357.

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Macroscopic and microscopic properties of molecular and solid-state systems are intimately related to the their electronic structure. The electron position and spin densities, which represent the probability distributions to find all or unpaired electrons in the space, contain information concerning several chemical-relevant properties, such as the chemical bonding and the magnetic behaviour. Understanding the fine atomic-level mechanism behind these properties is a key step to design chemical modifications to properly tune and develop materials or molecules with specific features. Topological descriptors can be used to extract information from these electron distributions. In this work, novel applications of the source function descriptor have been developed to gain further insights on the electron and spin density-related properties. These developments, together with other topological descriptors, were used to get further insights on relevant chemical systems. Firstly, the source function reconstruction was enlarged to a multi-dimensional grid of points with a particular focus on the two-dimensional maps. This analysis allows to see the ability of chosen subsets of atoms to reconstruct the density in the selected area within a cause-effect relationship and to rationalise the chemical or magnetic behaviours. The source function partial reconstructed maps depict if in a molecular region the atomic contributions are important, modest or negligible. Besides, they may also be useful for a proper selection of the reference points and for a full understanding of the source function percentages analysis. In fact, the choice of the reference point where to reconstruct the studied density is neither easy nor objective for non-standard situations, such as for the spin density. This novel application was applied to the study of the spin density on a couple of azido Cu complexes. The source function partial reconstructed maps allow to unravel the different role played by the paramagnetic centre Cu and the ligand atoms and to explain the spin transmission mechanism at a molecular level. Moreover, they enable to highlight the nature of the spin density differences between the two complexes and among adopted computational approaches. DFT functionals tend to over-delocalise the spin density towards the ligand atoms introducing a biased spin-polarization mechanism between the Cu and the ligand atoms. The same descriptor was then applied to the study of the hydrogen bonds in the DNA base pairs. The source function reveals the delocalised nature of these interactions, highlighting that distant groups and rings have non-negligible effects on the reconstruction of the electron density in the intermolecular region. Besides, the analysis demonstrates that the purine and pyrimidine bases equally contribute to the reconstruction of the electron density at the hydrogen bond critical points. The source function also reveals that subtle variations of the atomic source contributions occur when the pairs are ionized, revealing that sources and sinks effects redistribution plays an important role in the stabilization of the DNA base pairs. The source function was also used to develop a method to extract full population matrices purely based on the electron density distribution and then amenable to experimental determination. The peculiar features of this descriptor, in particular the cause-effect relationship, assign a profound chemical meaning to the matrix elements in contrast with other population analyses such as the Mulliken's one, where the matrix elements are associated to orbital overlaps. The latest breakthroughs on the development of this method are shown together with some numerical examples on very simple compounds. The full population matrices obtained using the source function descriptor are able to retrieve the major chemical features. A detailed analysis on the intermolecular interactions involved in the in vivo molecular recognition of the antimalarial drug chloroquine with the heme moiety has been carried out using a combined topological-energetic analysis. This work reveals that charged-assisted hydrogen bonds set up between the lateral chains of the chloroquine and the propionate group of the heme are the most important interactions in the drug:substrate recognition process.
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Arnold, Amanda Suzanne. "Shift." Digital Archive @ GSU, 2007. http://digitalarchive.gsu.edu/english_theses/26.

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The following is a collection of original poetry. The manuscript consists of an introduction explaining influences and style, and four chapters of poems categorized by subject matter: object/nature, writing/creativity, relationships, and family/figures. INDEX WORDS: Poetry, Poem
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Fuente, Luis de la. "A molecular dissection of early skeletogenesis /." 2004. http://wwwlib.umi.com/dissertations/search.

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Books on the topic "Bone dissection"

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author, Niparko John K., ed. Temporal bone dissection guide. New York: Thieme, 2011.

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Barbara, M. Manual of temporal bone dissection. Hague, Netherlands: Kugler Publications, 2002.

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Fisch, Ugo. Temporal bone dissection: The Zurich guidelines. Tuttlingen: Endo-Press, 2006.

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Schaeffer, Rebecca. Not even bones. Boston: Houghton Mifflin Harcourt, 2018.

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The Tale of Raw Head and Bloody Bones. London: Chatto & Windus, 2013.

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L, Blakely Robert, and Harrington Judith M, eds. Bones in the basement: Postmortem racism in nineteenth-century medical training. Washington: Smithsonian Institution Press, 1997.

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Niparko, John K., and Howard W. Francis. Temporal Bone Dissection Guide. Thieme Medical Publishers, Incorporated, 2011.

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Niparko, John K., and Howard W. Francis. Temporal Bone Dissection Guide. Thieme Medical Publishers, Incorporated, 2016.

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Belsare, Gauri S. Step by Step: Temporal Bone Dissection. Jaypee Brothers,Medical Publishers Pvt. Ltd., 2014.

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Belsare Gauri, S. Step by Step� Temporal Bone Dissection. Jaypee Brothers Medical Publishers (P) Ltd., 2014. http://dx.doi.org/10.5005/jp/books/12134.

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Book chapters on the topic "Bone dissection"

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McCarthy, Edward F. "Bone." In Surgical Pathology Dissection, 90–95. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4757-2548-3_18.

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McCarthy, Edward F. "Bone." In Surgical Pathology Dissection, 114–19. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/0-387-21747-9_22.

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Hruban, Ralph H., William H. Westra, Timothy H. Phelps, and Christina Isacson. "Bone Marrow." In Surgical Pathology Dissection, 200–202. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4757-2548-3_40.

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Westra, William H., Timothy H. Phelps, Ralph H. Hruban, and Christina Isacson. "Bone Marrow." In Surgical Pathology Dissection, 232–34. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/0-387-21747-9_44.

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Hruban, Ralph H., William H. Westra, Timothy H. Phelps, and Christina Isacson. "Femoral Head/Bone Tumors." In Surgical Pathology Dissection, 23–24. New York, NY: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4612-2398-6_12.

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Hruban, Ralph H., William H. Westra, Timothy H. Phelps, and Christina Isacson. "Bone Marrow Aspirate/Lymph Nodes." In Surgical Pathology Dissection, 39–40. New York, NY: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4612-2398-6_20.

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Chinn, Steven B., and Randal S. Weber. "Parotidectomy and Neck Dissection for Temporal Bone Malignancy." In Temporal Bone Cancer, 293–304. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-74539-8_22.

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Graf, Daniel, and Aris N. Economides. "Dissection of bone morphogenetic protein signaling using genome-engineering tools." In Bone Morphogenetic Proteins: From Local to Systemic Therapeutics, 115–39. Basel: Birkhäuser Basel, 2008. http://dx.doi.org/10.1007/978-3-7643-8552-1_7.

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Ramírez Arroyo, Gabriela, Carolina Tella Vega, Daniella A. Monroy Llaguno, and Juan Carlos Cisneros Lesser. "Temporal Bone Diseases and Tumours and Its Related Surgery." In Head and Neck Surgery : Surgical Landmark and Dissection Guide, 377–405. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3854-2_16.

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Agus, Marco, Andrea Giachetti, Enrico Gobbetti, Gianluigi Zanetti, and Antonio Zorcolo. "Tracking the Movement of Surgical Tools in a Virtual Temporal Bone Dissection Simulator." In Surgery Simulation and Soft Tissue Modeling, 100–107. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-45015-7_10.

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Conference papers on the topic "Bone dissection"

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Bryan, J., D. Stredney, G. Wiet, and D. Sessanna. "Virtual temporal bone dissection: a case study." In Proceedings VIS 2001. Visualization 2001. IEEE, 2001. http://dx.doi.org/10.1109/visual.2001.964561.

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Tabrizi, Pooneh R., Hongqiang Sang, Hadi F. Talari, Diego Preciado, Reza Monfaredi, Brian Reilly, Sreekanth Arikatla, Andinet Enquobahrie, and Kevin Cleary. "Temporal bone dissection simulator for training pediatric otolaryngology surgeons." In SPIE Medical Imaging, edited by Robert J. Webster and Baowei Fei. SPIE, 2017. http://dx.doi.org/10.1117/12.2254168.

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Gorthey, Scott, Nathaniel Webb, and Maja Svrakic. "Micro-CT 3D Printed Temporal Bone Dissection: A Feasibility Study." In Special Virtual Symposium of the North American Skull Base Society. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1725554.

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Kumaresan, M., and Navin Bharath. "A New Method of Cadaver Dissection in the Temporal Bone." In 27th Annual National Conference of the Indian Society of Otology. Thieme Medical and Scientific Publishers Private Ltd., 2019. http://dx.doi.org/10.1055/s-0039-1700207.

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Aguiar, Vinícius de Queiroz, Gustavo Sales França, Bernardo Costa Berriel Abreu, Talles Henrique Caixeta, Alexandre Henrique de Azevedo Dias, Daniela de Paula Paraiso Alves, Juliana de Lena Souza Marques, and Maria Victória Quintas de Almeida. "Relato de caso: Dissecção Carotídea associada a Síndrome de Eagle." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.235.

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Context: Eagle syndrome is characterized by the elongation or disfiguration of the styloid process, which leads to a range of clinical manifestations resulting from the structures that are affected by the prolongation of the bone, and the classic presentation is composed of pain and foreign body sensation in the throat, otalgia, and dysphagia. Case report: We describe the case of a 60-year-old man with an ischemic stroke due to dissection of the left internal carotid artery, associated with compression resulting from Eagle syndrome. At clinical presentation, the patient presented right hemiparesis and severe dysphagia, with NIH=18, characterizing the stroke. An angiotomography of the skull and brain was performed, which showed an image compatible with dissection of the left internal carotid artery from the prolongation of the styloid process, characterizing Eagle syndrome stylocarotid syndrome. The patient was submitted to thrombolysis with rt-PA, presenting a partial response, and surgical bone reduction. Patient evolved with partial recovery, with NIH=10, and, at the time of discharge, presented RANKIN 1, symptoms without disabilities. Conclusion: The Eagle syndrome, while it occurs more frequently in women, in general, with the classic presentation, can evolve with a more severe picture, associated with vessel dissection and brain involvement.
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Santiago, Phellipe Caetano, João Vitor da Silva Chagas, and Aura Conci. "Developing Innovative Models for Learning in Social Isolation Environments: Exemplifying it for the Bone Anatomy Study." In Life Improvement in Quality by Ubiquitous Experiences Workshop. Brazilian Computing Society, 2021. http://dx.doi.org/10.5753/lique.2021.15717.

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Anatomy knowledge is essential for many professionals, such as: doctors, nurses, biologists, biochemical, physiotherapists and any professional in physical education. Over the years, the main anatomical teaching method, the dissection of cadavers becomes less common due to issues related to high costs associated with the maintenance of laboratories, ethical aspects and health risks related to exposure to formaldehyde vapor. This factor was maximized with the Covid-19 pandemic, since presentials accesses to laboratories have become unviable. In order to offer a complementary tool for the teaching of bone anatomy, we propose in this work a serious game in augmented reality, based on controls by analysis of manual gestures, to assist the learning of this discipline.
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Assunção, Silvaleide Ataides, Vinicius Lemos Nascimento, Bruno Henrique de Aguiar Brito, Carolina Daher de Alencar Neves, Laura Queiroz da Silva, Pedro Vinicyus Novais e. Souza, Fernando Santos de Azevedo, and Lanúscia Morais de Santana. "NTRK MUTATION IN ADENOID CYSTIC CARCINOMA: A RARE TYPE OF TRIPLE NEGATIVE." In Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2072.

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Introduction: Breast cancer is one of the neoplasms that most cause death in women. Among these, there are some subtypes of greater biological aggressiveness, such as triple negative and HER overexpressed, which are associated with greater recurrence and mortality. Adenoid cystic carcinoma (ACC), salivary gland type, represents less than 0.1% of primary breast carcinomas and has indolent biological behavior and favorable prognosis compared with nonspecial triplenegative types. Case Report: A 51-year-old woman diagnosed with locally advanced ACC in the right breast, with negative immunohistochemistry for hormone receptors and HER2, underwent quadrantectomy with upfront axillary dissection, followed by adjuvant radiotherapy. After 12 years of diagnosis, she presented significant back pain, with magnetic resonance imaging scan evidencing bone lesion without medullary involvement in D7 and L1 suggestive of the secondary implant. Anatomopathology revealed the same histology as the primary tumor. Re-evaluation of chest tomography showed progression of pulmonary disease, 5 months after diagnosis of the first metastasis, underwent segmentectomy, with descriptive pathology identical to the initial lesion. Due to the oligoprogression and tumor type, somatic genetic research of the lung material was requested, which revealed a mutation in the NTRK gene, patient is still waiting for Larotrectinib in court. Discussion: The tumor has an unusual histological type, rare occurrence, slow progression course, and the absence of lymph node metastasis; the average incidence is around age 60. In this case, a young patient presented an ACC tumor with lung and bone metastasis. Due to the rarity, there is no definitive consensus regarding the ideal treatment, with the literature referring to the choice of mastectomy. Conclusions: Although malignant breast neoplasms and nonspecial subtypes, such as ductal and triple negative, have a poor prognosis, breast carcinoma of this aforementioned type has a favorable prognosis. The search for driver mutations in cancers of special types, together with the advances in genetic medicine, allows satisfactory results with target-specific treatments.
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Rodau, V., and S. Knipping. "Komplikationen der Neck Dissection bei tumorchirurgischen Eingriffen." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685735.

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Rodau, V., and S. Knipping. "Complications of Neck dissection in tumor surgery interventions." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685752.

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Mansour, N., A. Knopf, and B. Hofauer. "Neck dissection Level II-IV – How we do it." In Abstract- und Posterband – 91. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Welche Qualität macht den Unterschied. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1710913.

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Reports on the topic "Bone dissection"

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Ehrlich, Marcelo, John S. Parker, and Terence S. Dermody. Development of a Plasmid-Based Reverse Genetics System for the Bluetongue and Epizootic Hemorrhagic Disease Viruses to Allow a Comparative Characterization of the Function of the NS3 Viroporin in Viral Egress. United States Department of Agriculture, September 2013. http://dx.doi.org/10.32747/2013.7699840.bard.

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Project Title: "Development of a plasmid-based reverse genetics system for the Bluetongue and Epizootic Hemorrhagic Disease viruses to allow comparative characterization of the function of the NS3 viroporin in viral egress". Project details: No - IS-4192-09; Participants – Ehrlich M. (Tel Aviv University), Parker J.S. (Cornell University), DermodyT.S. (Vanderbilt University); Period - 2009-2013. Orbiviruses are insect-borne infectious agents of ruminants that cause diseases with considerable economical impact in Israel and the United States. The recent outbreaks of BTV in Europe and of Epizootic Hemorrhagic Disease Virus (EHDV) in Israel, underscore the need for: (i) a better comprehension of the infection process of orbiviruses, (ii) the identification of unique vs. common traits among different orbiviruses, (iii) the development of novel diagnosis and treatment techniques and approaches; all aimed at the achievement of more effective control and treatment measures. It is the context of these broad goals that the present project was carried out. To fulfill our long-term goal of identifying specific viral determinants of virulence, growth, and transmission of the orbiviruses, we proposed to: (i) develop reverse genetics systems for BTV and EHDV2-Ibaraki; and (ii) identify the molecular determinants of the NS3 nonstructural protein related to viroporin/viral egress activities. The first objective was pursued with a two-pronged approach: (i) development of a plasmid-based reverse genetics system for BTV-17, and (ii) development of an "in-vitro" transcription-based reverse genetics system for EHDV2-Ibaraki. Both approaches encountered technical problems that hampered their achievement. However, dissection of the possible causes of the failure to achieve viral spread of EHDV2-Ibaraki, following the transfection of in-vitro transcribed genomic segments of the virus, revealed a novel characteristic of EHDV2-Ibaraki infection: an uncharacteristically low fold increase in titer upon infection of different cell models. To address the function and regulation of NS3 we employed the following approaches: (i) development (together with Anima Cell Metrology) of a novel technique (based on the transfection of fluorescently-labeledtRNAs) that allows for the detection of the levels of synthesis of individual viral proteins (i.e. NS3) in single cells; (ii) development of a siRNA-mediated knockdown approach for the reduction in levels of expression of NS3 in EHDV2-Ibaraki infected cells; (iii) biochemical and microscopy-based analysis of the localization, levels and post-translational modifications of NS3 in infected cells. In addition, we identified the altered regulation and spatial compartmentalization of protein synthesis in cells infected with EHDV2-Ibaraki or the mammalian reovirus. In EHDV2-Ibaraki-infected cells such altered regulation in protein synthesis occurs in the context of a cell stress reponse that includes the induction of apoptosis, autophagy and activation of the stressrelated kinase c-Jun N-terminal Kinase (JNK). Interestingly, inhibition of such stress-related cellular processes diminishes the production of infectious virions, suggesting that EHDV usurps these responses for the benefit of efficient infection. Taken together, while the present project fell short of the generation of novel reverse genetics systems for orbiviruses, the development of novel experimental approaches and techniques, and their employment in the analysis of EHDV-infected cells, yielded novel insights in the interactions of orbiviruses with mammalian cells.
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