Journal articles: 'Blood volume'

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IIJIMA, Takehiko. "Static Blood Volume and Dynamic Blood Volume." JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA 34, no. 1 (2014): 139–44. http://dx.doi.org/10.2199/jjsca.34.139.

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CONVERTINO, VICTOR A. "Blood volume." Medicine & Science in Sports & Exercise 23, no. 12 (December 1991): 1338???1348. http://dx.doi.org/10.1249/00005768-199112000-00004.

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McGuire, Lynn, Michael R. Williamson, and Charles M. Boyd. "Red Cell Blood Volume, Plasma Volume, and Whole Blood Volume Calculation." Laboratory Medicine 18, no. 10 (October 1, 1987): 704–5. http://dx.doi.org/10.1093/labmed/18.10.704.

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Barker, Steven J. "Blood Volume Measurement." Anesthesiology 89, no. 6 (December 1, 1998): 1310–12. http://dx.doi.org/10.1097/00000542-199812000-00006.

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Isbister, James P. "Blood volume regulation." Emergency Medicine 8 (August 26, 2009): 1–14. http://dx.doi.org/10.1111/j.1442-2026.1996.tb00536.x.

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Dasselaar, Judith J., Marjolijn N. Lub-de Hooge, Jan Pruim, Hugo Nijnuis, Anneke Wiersum, Paul E. de Jong, Roel M. Huisman, and Casper F. M. Franssen. "Relative Blood Volume Changes Underestimate Total Blood Volume Changes during Hemodialysis." Clinical Journal of the American Society of Nephrology 2, no. 4 (May 9, 2007): 669–74. http://dx.doi.org/10.2215/cjn.00880207.

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Kim, Kyoungbo, and Sunggyun Park. "Validation of the Accuracy of Automatic Measurement of Blood Volume in Culture Bottles for Blood Culture." Diagnostics 13, no. 16 (August 15, 2023): 2685. http://dx.doi.org/10.3390/diagnostics13162685.

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Several manufacturers have developed systems that automatically measure the amount of blood in culture bottles. We compared the volumes measured automatically by the Virtuo instrument (bioMérieux, France) (height-based volumes) and those calculated by weighing the bottles. In all, 150 pairs of aerobic and anaerobic blood culture bottles (BacT/ALERT FA/FN Plus, bioMérieux) were randomly selected over two periods to compare the height- and weight-based volumes and analyze the effect of foam. We also estimated the limit of detection (LOD) and the cut-off value for 5 mL equine blood. The mean height-based volume was approximately 0.67 mL greater than the weight-based volume, particularly in anaerobic culture bottles. Foam did not have a significant effect. The LOD for the automatic height-based volume of equine blood was 0.2–0.4 mL. The 5 mL cut-off was 4–4.2 mL. Therefore, when reporting or monitoring blood volume within culture bottles in the laboratory, these performance characteristics should be adequately considered.

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Bealer, S. L., and E. G. Schneider. "Plasma corticosterone and volume after preoptic recess lesions and volume depletion." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 248, no. 2 (February 1, 1985): R161—R165. http://dx.doi.org/10.1152/ajpregu.1985.248.2.r161.

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The effects of extracellular fluid volume depletion on plasma corticosterone concentration (Pcort) and plasma volume in rats were determined after recovery from either electrolytic ablation of the periventricular tissue surrounding the anteroventral third ventricle (AV3V region) or control surgery. Rats received either furosemide injections and sodium-free chow or isotonic saline injections and continued access to sodium-replete food. One week after these injections some animals were decapitated and trunk blood collected for analysis of Pcort by radioimmunoassay. The remainder of the rats were implanted with femoral arterial catheters to obtain blood samples for measurement of plasma and blood volumes by calculating dilution of 125I-labeled serum albumin. Volume-replete rats with AV3V lesions had significantly higher Pcort concentrations and smaller plasma and blood volumes than volume-replete control-operated animals. Furthermore, volume depletion induced by furosemide caused a significant increase in Pcort concentration only in rats with AV3V ablations, whereas plasma and blood volumes were significantly lowered in both groups. These data demonstrate that AV3V periventricular ablation results in a chronic elevation of Pcort in the volume-replete animals and an exaggerated glucocorticoid response to volume depletion. These data show that decreased PV characteristic of animals with AV3V lesions is not due to glucocorticoid insufficiency.

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Hagberg, James M., Andrew P. Goldberg, Loretta Lakatta, Frances C. O’Connor, Lewis C. Becker, Edward G. Lakatta, and Jerome L. Fleg. "Expanded blood volumes contribute to the increased cardiovascular performance of endurance-trained older men." Journal of Applied Physiology 85, no. 2 (August 1, 1998): 484–89. http://dx.doi.org/10.1152/jappl.1998.85.2.484.

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To determine whether expanded intravascular volumes contribute to the older athlete’s higher exercise stroke volume and maximal oxygen consumption (V˙o 2 max), we measured peak upright cycle ergometry cardiac volumes (99mTc ventriculography) and plasma (125I-labeled albumin) and red cell (NaCr51) volumes in 7 endurance-trained and 12 age-matched lean sedentary men. The athletes had ∼40% higherV˙o 2 max values than did the sedentary men and larger relative plasma (46 vs. 38 ml/kg), red cell (30 vs. 26 ml/kg), and total blood volumes (76 vs. 64 ml/kg) (all P < 0.05). Athletes had larger peak cycle ergometer exercise stroke volume indexes (75 vs. 57 ml/m2, P < 0.05) and 17% larger end-diastolic volume indexes. In the total group,V˙o 2 maxcorrelated with plasma, red cell, and total blood volumes ( r = 0.61–0.70, P < 0.01). Peak exercise stroke volume was correlated directly with the blood volume variables ( r = 0.59–0.67, P < 0.01). Multiple regression analyses showed that fat-free mass and plasma or total blood volume, but not red cell volume, were independent determinants ofV˙o 2 max and peak exercise stroke volume. Plasma and total blood volumes correlated with the stroke volume and end-diastolic volume changes from rest to peak exercise. This suggests that expanded intravascular volumes, particularly plasma and total blood volumes, contribute to the higher peak exercise left ventricular end-diastolic volume, stroke volume, and cardiac output and hence the higherV˙o 2 max in master athletes by eliciting both chronic volume overload and increased utilization of the Frank-Starling effect during exercise.

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Henrick, Basil, Paul Keartland, Annette McCarthy, Liam Daly, and A. E. Wood. "Residual Blood in Neonatal Oxygenators After Drainage." Journal of ExtraCorporeal Technology 30, no. 4 (December 1998): 190–92. http://dx.doi.org/10.1051/ject/1998304190.

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The objective of this investigation was to measure the quantity of residual blood remaining in neonatal cardiopulmonary bypass (CPB) circuits after they had been drained and to assess the overall significance with regards to total patient blood volume. The residual blood volume left in three infant/neonatal CPB circuits-Medtronic Minimax 3381 (Group MM; n=5), Polystan Safe Micro (Group SM; n=6), and Terumo Capiox 308 (Group CX; n=3)-after they had been drained was determined. This was done by using an electronic scale to weigh the circuit before setup and after CPB when all possible blood was recovered from it. Total priming volume, estimated patient blood volume, residual blood volume, surgical blood loss in theater, and autogeneic blood usage were recorded in each case. Mean residual blood volumes measured were MM=161ml (SD 27ml), SM=103ml (SD 19ml), and CX=133ml (SD 15ml). These volumes were significant, because calculations show that the volume of red cells lost in the circuit is equivalent to fourteen percent of the total patient blood volume. In view of this, neonatal oxygenator design should be minimized to reduce the priming volume and more consideration should be given to ease of residual blood recovery.

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Ogilvie, Richard Ian, and Danuta Zborowska-Sluis. "Vascular capacitance and cardiac output in pacing-induced canine models of acute and chronic heart failure." Canadian Journal of Physiology and Pharmacology 73, no. 11 (November 1, 1995): 1641–50. http://dx.doi.org/10.1139/y95-726.

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The relationship between stressed and total blood volume, total vascular capacitance, central blood volume, cardiac output (CO), and pulmonary capillary wedge pressure (Ppcw) was investigated in pacing-induced acute and chronic heart failure. Acute heart failure was induced in anesthetized splenectomized dogs by a volume load (20 mL/kg over 10 min) during rapid right ventricular pacing at 250 beats/min (RRVP) for 60 min. Chronic heart failure was induced by continuous RRVP for 2–6 weeks (average 24 ± 2 days). Total vascular compliance and capacitance were calculated from the mean circulatory filling pressure (Pmcf) during transient circulatory arrest after acetylcholine at three different circulating volumes. Stressed blood volume was calculated as a product of compliance and Pmcf, with the total blood volume measured by a dye dilution. Central blood volume (CBV) and CO were measured by thermodilution. Central (heart and lung) vascular capacitance was estimated from the plot of Ppcw against CBV. Acute volume loading without RRVP increased capacitance and CO, whereas after volume loading with RRVP, capacitance and CO were unaltered from baseline. Chronic RRVP reduced capacitance and CO. All interventions, volume ± RRVP or chronic RRVP, increased stressed and central blood volumes and Ppcw. Acute or chronic RRVP reduced central vascular capacitance. Cardiac output was increased when stressed and unstressed blood volumes increased proportionately as during volume loading alone. When CO was reduced and Ppcw increased, as during chronic RRVP or acute RRVP plus a volume load, stressed blood volume was increased and unstressed blood volume was decreased. Thus, interventions that reduced CO and increased Ppcw also increased stressed and reduced unstressed blood volume and total vascular capacitance.Key words: vascular capacitance, vascular compliance, central blood volume, rapid ventricular pacing, dogs, heart failure.

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Tarazi, R. C. "Chapter 8: Blood Volume." European Heart Journal 6, suppl C (October 2, 1985): 41–42. http://dx.doi.org/10.1093/eurheartj/6.suppl_c.41.

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Secher, Niels H. "Inhale your blood volume!" Experimental Physiology 99, no. 10 (October 1, 2014): 1285. http://dx.doi.org/10.1113/expphysiol.2014.081844.

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Ito, Hiroshi, and Yasuhiro Nakajima. "Method of analyzing and displaying blood volume using myocardial blood volume map." Journal of the Acoustical Society of America 123, no. 4 (2008): 1830. http://dx.doi.org/10.1121/1.2909070.

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Altez, Maria S. Rueda, Lamia Soghier, Joseph M. Campos, James Bost, Jiaxiang Gai, and Rana F. Hamdy. "1341. Blood Volume Collected for Blood Cultures in Infants with Suspected Neonatal Sepsis in the NICU." Open Forum Infectious Diseases 7, Supplement_1 (October 1, 2020): S682. http://dx.doi.org/10.1093/ofid/ofaa439.1523.

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Abstract Background Blood cultures have high sensitivity to detect bacteremia in septic neonates when >=1 ml of blood is collected. Neonatologists often cite low confidence in microbiologic sampling as rationale for continuing antibiotics without a focus of infection despite negative blood cultures, resulting in prolonged antimicrobial therapy. We aim to describe the blood culture sample volumes in NICU patients, to identify factors associated with sample volumes < 1ml, and to compare the sample volumes of patients treated for culture-negative sepsis with those with bloodstream infections and those treated for a ≤72-hour sepsis rule-out Methods Data from this observational cohort study were collected retrospectively and prospectively from NICU patients with blood cultures obtained from September 2018 to February 2019. Clinical data were collected through chart review. All inoculated culture bottles were weighed for volume calculation. We determined the association of age, weight, sample source, and time of collection with volume < 1mL. Continuous variables were analyzed using Wilcoxon-Mann-Whitney, and categorical variables using chi-squared test. For aim 3, the volumes of the groups were compared using analysis of variance. Results A total of 310 blood cultures were identified, corresponding to 159 patients. Of these, 49 (16%) were positive. Among the negative blood cultures, 86% were collected in patients who subsequently received antibiotics (Figure 1). Median inoculated volume was 0.6 ml (IQR: 0.1-2.4). Weight and age at time of culture collection, source of sample, and time of collection were not significantly associated with the inoculation of < 1ml of blood. Median volume of blood was 0.6ml (0.3-0.6) for sepsis rule-out, 0.6ml (0.2-0.6) for bloodstream infection, and 0.6ml (0.6-1.4) for culture-negative sepsis. No difference was found among the three groups (p=0.54) Figure 1. Classification of blood cultures identified during study period Conclusion The blood volume collected for cultures in the NICU is lower than recommended. Clinical and environmental characteristics are not significantly associated with the inoculated volume. The volume of blood sampled does not differ in patients with culture-negative sepsis, bloodstream infection and sepsis rule-out, and should not be a justification for longer duration of antibiotic therapy Disclosures All Authors: No reported disclosures

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Stewart, Julian M., and Leslie D. Montgomery. "Regional blood volume and peripheral blood flow in postural tachycardia syndrome." American Journal of Physiology-Heart and Circulatory Physiology 287, no. 3 (September 2004): H1319—H1327. http://dx.doi.org/10.1152/ajpheart.00086.2004.

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Variants of postural tachycardia syndrome (POTS) are associated with increased [“high-flow” POTS (HFP)], decreased [“low-flow” POTS (LFP)], and normal [“normal-flow” POTS (NFP)] blood flow measured in the lower extremities while subjects were in the supine position. We propose that postural tachycardia is related to thoracic hypovolemia during orthostasis but that the patterns of peripheral blood flow relate to different mechanisms for thoracic hypovolemia. We studied 37 POTS patients aged 14–21 yr: 14 LFP, 15 NFP, and 8 HFP patients and 12 healthy control subjects. Peripheral blood flow was measured in the supine position by venous occlusion strain-gauge plethysmography of the forearm and calf to subgroup patients. Using indocyanine green techniques, we showed decreased cardiac index (CI) and increased total peripheral resistance (TPR) in LFP, increased CI and decreased TPR in HFP, and unchanged CI and TPR in NFP while subjects were supine compared with control subjects. Blood volume tended to be decreased in LFP compared with control subjects. We used impedance plethysmography to assess regional blood volume redistribution during upright tilt. Thoracic blood volume decreased, whereas splanchnic, pelvic, and leg blood volumes increased, for all subjects during orthostasis but were markedly lower than control for all POTS groups. Splanchnic volume was increased in NFP and LFP. Pelvic blood volume was increased in HFP only. Calf volume was increased above control in HFP and LFP. The results support the hypothesis of (at least) three pathophysiologic variants of POTS distinguished by peripheral blood flow related to characteristic changes in regional circulations. The data demonstrate enhanced thoracic hypovolemia during upright tilt and confirm that POTS is related to inadequate cardiac venous return during orthostasis.

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Stewart, Julian M., Leslie D. Montgomery, June L. Glover, and Marvin S. Medow. "Changes in regional blood volume and blood flow during static handgrip." American Journal of Physiology-Heart and Circulatory Physiology 292, no. 1 (January 2007): H215—H223. http://dx.doi.org/10.1152/ajpheart.00681.2006.

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Increased blood pressure (BP) and heart rate during exercise characterizes the exercise pressor reflex. When evoked by static handgrip, mechanoreceptors and metaboreceptors produce regional changes in blood volume and blood flow, which are incompletely characterized in humans. We studied 16 healthy subjects aged 20–27 yr using segmental impedance plethysmography validated against dye dilution and venous occlusion plethysmography to noninvasively measure changes in regional blood volumes and blood flows. Static handgrip while in supine position was performed for 2 min without postexercise ischemia. Measurements of heart rate and BP variability and coherence analyses were used to examine baroreflex-mediated autonomic effects. During handgrip exercise, systolic BP increased from 120 ± 10 to 148 ± 14 mmHg, whereas heart rate increased from 60 ± 8 to 82 ± 12 beats/min. Heart rate variability decreased, whereas BP variability increased, and transfer function amplitude was reduced from 18 ± 2 to 8 ± 2 ms/mmHg at low frequencies of ∼0.1 Hz. This was associated with marked reduction of coherence between BP and heart rate (from 0.76 ± 0.10 to 0.26 ± 0.05) indicative of uncoupling of heart rate regulation by the baroreflex. Cardiac output increased by ∼18% with a 4.5% increase in central blood volume and an 8.5% increase in total peripheral resistance, suggesting increased cardiac preload and contractility. Splanchnic blood volume decreased reciprocally with smaller decreases in pelvic and leg volumes, increased splanchnic, pelvic and calf peripheral resistance, and evidence for splanchnic venoconstriction. We conclude that the exercise pressor reflex is associated with reduced baroreflex cardiovagal regulation and driven by increased cardiac output related to enhanced preload, cardiac contractility, and splanchnic blood mobilization.

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Greenway, C. V. "Effects of hemorrhage and hepatic nerve stimulation on venous compliance and unstressed volume in cat liver." Canadian Journal of Physiology and Pharmacology 65, no. 11 (November 1, 1987): 2168–74. http://dx.doi.org/10.1139/y87-342.

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Intrahepatic blood volume–pressure relationships were studied using plethysmography to measure hepatic blood volume and a hepatic venous long-circuit to control intrahepatic pressure. In cats anesthetized with pentobarbital or with ketamine–chloralose, hemorrhage (to reduce hepatic blood flow to 60% of control) caused marked reductions in hepatic blood volume and intrahepatic pressure but did not significantly change hepatic blood volume–pressure relationships. We were unable to demonstrate an active reflex venous response to hemorrhage in these preparations, although a large passive response occurred. The volume–pressure relationships in innervated livers were different from those in denervated livers: apparent venous compliance was much greater and apparent unstressed volume was zero or negative. Hepatic nerve stimulation in denervated livers caused a marked decrease in hepatic blood volume at low intrahepatic pressures but failed to alter hepatic blood volumes at high intrahepatic pressures (15 mmHg) (1 mmHg = 133.3 Pa). This resulted in large apparent compliances and apparently negative unstressed volumes, as seen in the innervated livers. Thus blood volume–pressure relationships in innervated livers may not give valid measurements of compliance and unstressed volume. A remarkable feature in all these experiments was the linearity of the relationship between hepatic blood volume and intrahepatic pressure. Exudation of fluid begins at higher intrahepatic pressures in innervated compared with denervated livers.

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Todd, M. M., J. B. Weeks, and D. S. Warner. "Cerebral blood flow, blood volume, and brain tissue hematocrit during isovolemic hemodilution with hetastarch in rats." American Journal of Physiology-Heart and Circulatory Physiology 263, no. 1 (July 1, 1992): H75—H82. http://dx.doi.org/10.1152/ajpheart.1992.263.1.h75.

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The influence of isovolemic hemodilution with 6% hetastarch [hematocrits (Hct) ranging from 43 to 20%] on cerebral blood flow (CBF), cerebral red blood cell and plasma volumes, total cerebral blood volume (CBV), and cerebral Hct was examined in normothermic, normocarbic, halothane-anesthetized Sprague-Dawley rats. CBF was measured via the indicator-fractionation method ([3H]nicotine), red blood cell volume was measured using 99mTc-labeled red blood cells, while plasma volume was measured using [14C]dextran. Brain tissue was fixed in situ by microwave irradiation. All data plots (e.g., CBF vs. Hct) were fitted by linear regression methods. Hemodilution was associated with a progressive increase in forebrain CBF (from a fitted value of 78 ml.100 g-1.min-1 at Hct = 43%, to 171 ml.100 g-1.min-1 at 20%). Cerebral plasma volume also rose, while red blood cell volume decreased. Total CBV (i.e., the sum of red blood cell and plasma volumes) increased in parallel with CBF (from 2.51 ml/100 g at Hct = 43 to 4.94 ml/100 g at Hct = 20%). This increase is larger than can be explained by a simple increase in the diameter of arterial/arteriolar resistance vessels and may be due to either capillary recruitment or to an increase in the volume of postarteriolar structures. Calculated cerebral tissue hematocrit decreased. The magnitude of this decrease was larger than the reduction in arterial Hct; the ratio of cerebral to arterial Hct decreased from 0.780 at an arterial Hct equaling 43% to 0.458 at Hct equaling 20%.(ABSTRACT TRUNCATED AT 250 WORDS)

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Matte, J. J., and C. L. Girard. "Changes of serum and blood volumes during gestation and lactation in multiparous sows." Canadian Journal of Animal Science 76, no. 2 (June 1, 1996): 263–66. http://dx.doi.org/10.4141/cjas96-039.

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Changes of serum and plasma volumes were determined in 36 gestating sows and 20 lactating sows at their second parity. There was no change (P > 0.05) in blood and serum volumes between 4 wk pre-mating and 1 wk post-mating. During gestation, blood and serum volumes increased by approximately 25%, with most of this increase occurring between 11 and 14 wk of pregnancy (P < 0.006). From parturition to weaning at 4 wk of lactation, serum and blood volumes decreased linearly (P < 0.02) by approximately 9%. Key words: Serum volume, blood volume, packed red cell volume, sow

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Keiver, K. M., M. Chandler, R. J. Frank, and K. Ronald. "Plasma and blood volumes of the hooded seal (Cystophora cristata)." Canadian Journal of Zoology 65, no. 7 (July 1, 1987): 1866–67. http://dx.doi.org/10.1139/z87-286.

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Plasma volumes and haematocrits were determined in six hooded seals (Cystophora cristata) and blood volumes were estimated. Expressed on a total body weight basis, plasma volume was found to range from 39 to 109 mL∙kg−1 and blood volume from 93 to 222 mL∙kg−1. Logarithms of the values for plasma and blood volume varied directly with the total body weight of the seals.

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Gonçalves, Rilvani C., Carlos Alberto Buschpigell, and Antonio Augusto Lopes. "Circulating blood volumes in pulmonary hypertension associated with erythrocytosis – the effects of therapeutic hemodilution." Cardiology in the Young 13, no. 6 (December 2003): 544–50. http://dx.doi.org/10.1017/s1047951103001148.

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In the Eisenmenger syndrome, indirect estimation of blood volumes may provide quite inaccurate information when seeking to define therapeutic strategies. With this in mind, we analyzed directly the red cell mass, plasma volume, and total blood volume in patients with pulmonary hypertension associated with congenital cardiac defects and erythrocytosis, comparing the results with the respective estimated volumes, and examining the changes induced by therapeutic hemodilution.Thus, we studied 17 patients with the Eisenmenger syndrome, aged from 15 to 53 years, in the basal condition, studying 12 of them both before and after hemodilution. We also investigated five individuals with minimal cardiac lesions, aged from 14 to 42 years, as controls. Red cell mass and plasma volumes were measured using [51 chromium]-sodium chromate and [131iodine]-albumin respectively. Hemodilution was planned so as to exchange 10% of the total blood volume, using 40,000 molecular weight dextran simultaneously to replace the removed volume. The mean values of the red cell mass, plasma volume and total blood volume as assessed by radionuclide techniques were 32%, 31% and 32% higher than the respective volumes as estimated using empirical mathematical formulas (p < 0.002). The measured total blood volume was also 19% higher in the patients compared with controls. Following a period of 5 days after hemodilution, we noted a 13% reduction in red cell mass (p = 0.046), and 10% reduction in total blood volume (p = 0.02), albeit with no changes in the plasma volume.We conclude that direct measurement of blood volumes is useful for proper management of these patients, and provides results that are considerably different from those obtained by empirical estimations.

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Dale, Jane C., and Stephen G. Ruby. "Specimen Collection Volumes for Laboratory Tests." Archives of Pathology & Laboratory Medicine 127, no. 2 (February 1, 2003): 162–68. http://dx.doi.org/10.5858/2003-127-162-scvfl.

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Abstract Context.—Unnecessary tests, inefficient ordering practices, and collection of more blood than is required for testing contribute to iatrogenic anemia in hospitalized patients. Laboratories accredited by the College of American Pathologists are expected to review phlebotomy practices for specimen collection volumes periodically. Objective.—To report specimen collection, analytic, and discard volumes for routine laboratory tests and to identify practice variables associated with overcollection and blood wastage. Design.—Clinical laboratories participating in the College of American Pathologists Q-Probes laboratory improvement program recorded collection container size, laboratory-defined requested volume, manufacturer-defined analytic volume, and average discard volume for routine complete blood cell counts and electrolyte panels ordered for patients in intensive care units. Participants provided information about their specimen collection, processing, and analytic practices in a questionnaire. Setting and Participants.—A total of 140 public and private institutions. Main Outcome Measures.—Overcollections for routine collections and for situations in which a reduced volume of specimen is collected, and average discard volume per tube. Results.—Laboratories collected a median of 2.76 mL (or 8.5 times) more than their instrument's analytic volume for routine complete blood cell counts and 1.75 mL (or 12 times) more than their instrument's analytic volume for routine electrolyte panels. For clinical situations in which reduced collection volumes were necessary, overcollection for the same analytes was 0.5 mL (3 times) and 0.44 mL (4.2 times), respectively. The median discard volume was 2.8 mL/tube for complete blood cell counts and 2.0 mL/tube for electrolyte panels. Specimen collection container size was directly associated with overcollections and discard volumes. Instrument analytic volume was not a determinant of blood wastage. Conclusions.—Most laboratories can decrease collection volumes without compromising the ability of the laboratory to report a reliable and timely result. Use of smaller collection tubes can help reduce blood wastage.

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LEENDERS, K. L., D. PERANI, A. A. LAMMERTSMA, J. D. HEATHER, P. BUCKINGHAM, T. JONES, M. J. R. HEALY, et al. "CEREBRAL BLOOD FLOW, BLOOD VOLUME AND OXYGEN UTILIZATION." Brain 113, no. 1 (1990): 27–47. http://dx.doi.org/10.1093/brain/113.1.27.

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Wright, I. M., and S. R. Goodall. "Blood pressure and blood volume in preterm infants." Archives of Disease in Childhood - Fetal and Neonatal Edition 70, no. 3 (May 1, 1994): F230—F231. http://dx.doi.org/10.1136/fn.70.3.f230-a.

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Birchard, Geoffrey F., and S. M. Tenney. "Relationship between blood-oxygen affinity and blood volume." Respiration Physiology 83, no. 3 (March 1991): 365–73. http://dx.doi.org/10.1016/0034-5687(91)90055-n.

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Rehm, Markus, Victoria Orth, Uwe Kreimeier, Manfred Thiel, Mathias Haller, Heinz Brechtelsbauer, and Udilo Finsterer. "Changes in Intravascular Volume during Acute Normovolemic Hemodilution and Intraoperative Retransfusion in Patients with Radical Hysterectomy." Anesthesiology 92, no. 3 (March 1, 2000): 657–64. http://dx.doi.org/10.1097/00000542-200003000-00008.

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Background Changes in blood volume during acute normovolemic hemodilution (ANH) and their consequences for the perioperative period have not been investigated sufficiently. Methods In 15 patients undergoing radical hysterectomy, preoperative ANH to a hematocrit of 24% was performed using 5% albumin solution. Intraoperatively, saline 0.9% solution was used for volume substitution, and intraoperative retransfusion was started at a hematocrit of 20%. Plasma volume (indocyanine green dilution technique), hematocrit, and plasma protein concentration were measured before and after ANH, before retransfusion, and postoperatively. Red cell volume (labeling erythrocytes with fluorescein) was determined before and after ANH and postoperatively. Results Mean normal plasma volumes (1,514 +/- 143 ml/m2) and reduced red cell volumes (707 +/- 79 ml/m2) were measured preoperatively. Blood (1,150 +/- 196 ml) was removed and replaced with 1,333 +/- 204 ml of colloid. Blood volume before and after ANH was equal and amounted to 3,740 ml. Intraoperatively, plasma volume did not increase until retransfusion despite infusing 3,389 +/- 1,021 ml of crystalloid (corrected for urine output) to compensate for an estimated surgical blood loss of 727 +/- 726 mi. Postoperatively, after retransfusion of all autologous blood, blood volume was 255 +/- 424 ml higher than preoperatively before ANH. Despite mean calculated blood loss of 1,256 +/- 892 ml, only one patient received allogeneic blood. Conclusions During ANH, normovolemia was exactly maintained. After surgical blood loss of 1,256 +/- 892 ml, crystalloid and colloid supplies of 5,752 +/- 1,462 ml and 1,667 +/- 548 ml, respectively, and complete intraoperative retransfusions of autologous blood in every patient, mean blood volume was 250 ml higher than preoperatively before ANH.

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Taneja, Indu, Christopher Moran, Marvin S. Medow, June L. Glover, Leslie D. Montgomery, and Julian M. Stewart. "Differential effects of lower body negative pressure and upright tilt on splanchnic blood volume." American Journal of Physiology-Heart and Circulatory Physiology 292, no. 3 (March 2007): H1420—H1426. http://dx.doi.org/10.1152/ajpheart.01096.2006.

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Upright posture and lower body negative pressure (LBNP) both induce reductions in central blood volume. However, regional circulatory responses to postural changes and LBNP may differ. Therefore, we studied regional blood flow and blood volume changes in 10 healthy subjects undergoing graded lower-body negative pressure (−10 to −50 mmHg) and 8 subjects undergoing incremental head-up tilt (HUT; 20°, 40°, and 70°) on separate days. We continuously measured blood pressure (BP), heart rate, and regional blood volumes and blood flows in the thoracic, splanchnic, pelvic, and leg segments by impedance plethysmography and calculated regional arterial resistances. Neither LBNP nor HUT altered systolic BP, whereas pulse pressure decreased significantly. Blood flow decreased in all segments, whereas peripheral resistances uniformly and significantly increased with both HUT and LBNP. Thoracic volume decreased while pelvic and leg volumes increased with HUT and LBNP. However, splanchnic volume changes were directionally opposite with stepwise decreases in splanchnic volume with LBNP and stepwise increases in splanchnic volume during HUT. Splanchnic emptying in LBNP models regional vascular changes during hemorrhage. Splanchnic filling may limit the ability of the splanchnic bed to respond to thoracic hypovolemia during upright posture.

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Doerschuk, C. M., and H. S. Sekhon. "Pulmonary blood volume and edema in postpneumonectomy lung growth in rats." Journal of Applied Physiology 69, no. 3 (September 1, 1990): 1178–82. http://dx.doi.org/10.1152/jappl.1990.69.3.1178.

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After pneumonectomy in young animals, the contralateral lung undergoes compensatory growth and generally attains the same weight and air space volume as both lungs in age-matched controls. In this study, we determined the contribution of lung edema and increased blood volume to the weight gain in rats. Three weeks after pneumonectomy (n = 18) or sham pneumonectomy (n = 17), the pulmonary blood volume and the extravascular water and albumin were evaluated by use of 51Cr-labeled erythrocytes and 125I-labeled albumin. The air space volume, blood-free lung weights, and DNA and protein content were also compared. The data show that the total pulmonary blood volumes and the blood volume per gram of blood-free dry lung were similar in pneumonectomized and age-matched sham controls. The total extravascular albumin and the extravascular albumin per gram of blood-free dry lung were also similar as well as the extravascular lung water, wet-to-dry weight ratios, DNA and protein content, and air space volumes. These data indicate that the increased weight of the postpneumonectomy lung was due to cellular and stromal proliferation. The blood volume and interstitial fluid increased in proportion to the increase in lung parenchyma. Neither vascular congestion nor increased extravascular protein and water contributed to the observed weight gain.

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Ito, Hiroshi, Masanobu Ibaraki, Iwao Kanno, Hiroshi Fukuda, and Shuichi Miura. "Changes in the Arterial Fraction of Human Cerebral Blood Volume during Hypercapnia and Hypocapnia Measured by Positron Emission Tomography." Journal of Cerebral Blood Flow & Metabolism 25, no. 7 (February 16, 2005): 852–57. http://dx.doi.org/10.1038/sj.jcbfm.9600076.

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Hypercapnia induces cerebral vasodilation and increases cerebral blood volume (CBV), and hypocapnia induces cerebral vasoconstriction and decreases CBV. Cerebral blood volume measured by positron emission tomography (PET) is the sum of three components, that is, arterial, capillary, and venous blood volumes. Changes in arterial blood volume ( Va) and CBV during hypercapnia and hypocapnia were investigated in humans using PET with H215O and 11CO. Arterial blood volume was determined from H215O PET data by means of a two-compartment model that takes Va into account. Baseline CBV and values during hypercapnia and hypocapnia in the cerebral cortex were 0.034 ± 0.003, 0.038 ± 0.003, and 0.031 ± 0.003 mL/mL (mean ± s.d.), respectively. Baseline Va and values during hypercapnia and hypocapnia were 0.015 ± 0.003, 0.025 ± 0.011, and 0.007 ± 0.003 mL/mL, respectively. Cerebral blood volume changed significantly owing to changes in PaCO2, and Va changed significantly in the direction of CBV changes. However, no significant change was observed in venous plus capillary blood volume (= CBV- Va). This indicates that changes in CBV during hypercapnia and hypocapnia are caused by changes in arterial blood volume without changes in venous and capillary blood volume.

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Lima, Ana Paula Souza de, Flávia Giron Camerini, Vanessa Galdino de Paula, Karla Biancha Silva de Andrade, and Cintia Silva Fassarella. "Descarte sanguíneo em sistema aberto de pressão arterial invasiva." Revista Recien - Revista Científica de Enfermagem 11, no. 34 (June 27, 2021): 23–32. http://dx.doi.org/10.24276/rrecien2021.11.34.23-32.

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Analisar o volume de solução a ser desprezado do cateter de pressão arterial invasiva a partir de quatro volumes mínimos para obtenção de amostra sanguínea com hemoconcentração eficaz para análise laboratorial. Estudo observacional transversal, com análise laboratorial, realizado em uma unidade de terapia intensiva no Estado do Rio de Janeiro. Os dados coletados foram armazenados no software Microsoft Excel® e analisados no SPSS Statistics®20.0, por análise estatística descritiva e de variância usando ANOVA e Tukey. Foram analisadas 157 amostra e percebeu-se que mesmo em comparações múltiplas não há diferença significativa entre os quatro volumes analisados, isto é, os volumes comparados não evidenciaram diferença dos resultados dos valores de hematócrito e hemoglobina. Concluiu-se que é possível recomendar o menor volume a ser desprezado do cateter de pressão arterial invasiva para uma amostra sanguínea eficaz, sendo este o volume de 1,5ml para cateteres arteriais radiais ou femorais.Descritores: Monitorização Hemodinâmica, Coleta de Amostras Sanguíneas, Cuidados Críticos. Blood disposal in an open invasive blood pressure systemAbstract: Analyze the volume of solution to be discarded from the invasive blood pressure catheter from four minimum volumes to obtain blood samples with effective hemoconcentration for laboratory analysis. Cross-sectional observational study, with laboratory analysis, carried out in an intensive care unit in the State of Rio de Janeiro. The collected data were stored in Microsoft Excel® software and analyzed using SPSS Statistics®20.0, using descriptive and variance statistical analysis using ANOVA and Tukey. 157 samples were analyzed and it was noticed that even in multiple comparisons there is no significant difference between the four volumes analyzed, that is, the volumes compared did not show any difference in the results of the hematocrit and hemoglobin values. It was concluded that it is possible to recommend the smallest volume to be discarded from the invasive blood pressure catheter for an effective blood sample, this being the volume of 1.5 ml for radial or femoral arterial catheters.Descriptors: Hemodynamic Monitoring, Blood Specimen Collection, Critical Care. Eliminación de sangre en un sistema abierto de presión arterial invasivaResumen: Analice el volumen de solución que se desechará del catéter invasivo de presión arterial a partir de cuatro volúmenes mínimos para obtener muestras de sangre con hemoconcentración efectiva para análisis de laboratorio. Estudio observacional transversal, con análisis de laboratorio, realizado en una unidad de cuidados intensivos en el estado de Río de Janeiro. Los datos recopilados se almacenaron en el software Microsoft Excel® y se analizaron usando SPSS Statistics®20.0, usando análisis estadísticos descriptivos y de varianza usando ANOVA y Tukey. Se analizaron 157 muestras y se observó que, incluso en comparaciones múltiples, no existe una diferencia significativa entre los cuatro volúmenes analizados, es decir, los volúmenes comparados no mostraron ninguna diferencia en los resultados de los valores de hematocrito y hemoglobina. Se concluyó que es posible recomendar que se descarte el volumen más pequeño del catéter invasivo de presión arterial para obtener una muestra de sangre efectiva, siendo este el volumen de 1,5 ml para catéteres arteriales radiales o femorales.Descriptores: Monitorización Hemodinâmica, Recolección de Muestras de Sangre, Cuidados Críticos.

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Wang, Y., C. Drakonakis, J. L. Alderman, and D. L. Rutlen. "Changes in regional vascular capacitance during prostacyclin-induced cardiac vagal reflex in pigs." American Journal of Physiology-Heart and Circulatory Physiology 267, no. 2 (August 1, 1994): H535—H539. http://dx.doi.org/10.1152/ajpheart.1994.267.2.h535.

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The purpose of this study was to determine the effects of the prostaglandin I2 (prostacyclin; PGI2)-induced cardiac vagal reflex on intestinal and liver blood volumes and the intestinal vascular pressure-volume (P-V) relationship. In anesthetized pigs, blood volumes were measured by blood-pool scintigraphy. Portal venous pressure was varied by graded inflation of a portal vein constrictor to determine the intestinal vascular P-V relationship. Proximal right coronary infusion of PGI2 at a rate of 0.15 micrograms.kg-1.min-1 for 6 min increased intestinal blood volume by 7.0 +/- 1.2% (P < 0.01, means +/- SE) and shifted the intestinal vascular P-V relationship away from the pressure axis (i.e., a volume increase at a given venous pressure). This change was associated with decreases in liver blood volume and left ventricular end-diastolic pressure by 4.5 +/- 1.2 (P < 0.01) and 17 +/- 2% (P < 0.05), respectively. PGI2 also reduced central venous pressure by 16 +/- 2% from 3.2 +/- 0.5 mmHg (P < 0.05) and portal venous pressure by 7.0 +/- 0.6% from 7.6 +/- 0.6 mmHg (P < 0.05). These responses were abolished by bilateral vagotomy. The results demonstrate that intracoronary PGI2 infusion increases intestinal blood volume. This increase is mediated by a cardiac vagal reflex. The PGI2-induced shift in the intestinal vascular P-V relationship suggests that intestinal blood volume increases by an active change in vascular capacitance, whereas reductions in liver blood volume and left ventricular end-diastolic pressure appear to be due to passive mechanisms related to the shift of blood volume to the intestinal circulation.

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33

Blaber, Andrew P., Helmut Hinghofer-Szalkay, and Nandu Goswami. "Blood Volume Redistribution During Hypovolemia." Aviation, Space, and Environmental Medicine 84, no. 1 (January 1, 2013): 59–64. http://dx.doi.org/10.3357/asem.3424.2013.

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34

Ihara, A., T. Nishiura, Y. Uchida, K. Abe, F. Tanaka, K. Matsui, and S. Shouno. "PLATELET VOLUME IN CORD BLOOD." Journal of Thrombosis and Haemostasis 5 (July 2007): P—T—289—P—T—289. http://dx.doi.org/10.1111/j.1538-7836.2007.tb02047.x.

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35

Tarazi, R. C. "Chapter 9: Pulmonary Blood Volume." European Heart Journal 6, suppl C (October 2, 1985): 43. http://dx.doi.org/10.1093/eurheartj/6.suppl_c.43.

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36

Armitage, Sue. "Cord Blood Processing: Volume Reduction." Cell Preservation Technology 4, no. 1 (March 2006): 9–16. http://dx.doi.org/10.1089/cpt.2006.4.9.

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37

Shulman, T. S., A. P. Heidenheim, S. M. Shulman, and R. M. Lindsay. "PRESERVING CENTRAL BLOOD VOLUME [CBV]." ASAIO Journal 47, no. 2 (March 2001): 153. http://dx.doi.org/10.1097/00002480-200103000-00210.

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38

Santoro, Antonio, Elena Mancini, Francesco Paolini, Giovanni Cavicchioli, Antonio Bosetto, and Pietro Zucchelli. "Blood Volume Regulation During Hemodialysis." American Journal of Kidney Diseases 32, no. 5 (November 1998): 739–48. http://dx.doi.org/10.1016/s0272-6386(98)70128-3.

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39

BERLIN, RAGNAR. "Blood Volume in Chronic Leukemia." Acta Medica Scandinavica 164, no. 3 (April 24, 2009): 257–62. http://dx.doi.org/10.1111/j.0954-6820.1959.tb00188.x.

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40

Santoro, A., E. Mancini, F. Paolini, and P. Zucchelli. "Blood volume monitoring and control." Nephrology Dialysis Transplantation 11, supp2 (January 1, 1996): 42–47. http://dx.doi.org/10.1093/ndt/11.supp2.42.

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41

Rubin, Jonathan M. "Angle-Independent Blood Volume Flow." Ultrasound in Medicine & Biology 45 (2019): S2. http://dx.doi.org/10.1016/j.ultrasmedbio.2019.07.420.

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42

Sano, Y., A. Sakamoto, Y. Oi, and R. Ogawa. "Anaesthesia and circulating blood volume." European Journal of Anaesthesiology 22, no. 4 (April 2005): 258–62. http://dx.doi.org/10.1017/s0265021505000438.

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43

Dalmau, Rafael. "The “stressed blood volume” revisited." Canadian Journal of Anesthesia/Journal canadien d'anesthésie 65, no. 9 (June 7, 2018): 1070–71. http://dx.doi.org/10.1007/s12630-018-1154-7.

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44

Jones, B. G., J. G. Jones, R. B. Beattie, B. M. Holland, R. F. Wynne, and C. M. Wardrop. "Measurements of fetoplacental blood volume." Early Human Development 45, no. 1-2 (July 1996): 159–60. http://dx.doi.org/10.1016/0378-3782(96)81343-0.

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Sweeting, J. G. "Central blood volume in cirrhosis." Gastroenterology 104, no. 6 (June 1993): 1878–79. http://dx.doi.org/10.1016/0016-5085(93)90679-7.

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46

Buhre, W., K. Bendyk, A. Weyland, S. Kazmaier, M. Schmidt, K. Mursch, and H. Sonntag. "Assessment of intrathoracic blood volume." Der Anaesthesist 47, no. 1 (January 30, 1998): 51–53. http://dx.doi.org/10.1007/s001010050522.

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Vaz, Tina, and Georgene Singh. "Large-volume Epidural Blood Patch." Journal of Neurosurgical Anesthesiology 29, no. 3 (July 2017): 359–60. http://dx.doi.org/10.1097/ana.0000000000000285.

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Sánchez, M., M. Jiménez-Lendínez, M. Cidoncha, M. J. Asensio, E. Herrero, A. Collado, and M. Santacruz. "Comparison of Fluid Compartments and Fluid Responsiveness in Septic and Non-Septic Patients." Anaesthesia and Intensive Care 39, no. 6 (November 2011): 1022–29. http://dx.doi.org/10.1177/0310057x1103900607.

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Our objective was to study the response to a fluid load in patients with and without septic shock, the relationship between the response and baseline fluid distributions and the ratios of the various compartments. A total of 18 patients with septic shock and 14 control patients without pathologies that increase capillary permeability were evaluated prospectively. We used transpulmonary thermodilution to measure the extravascular lung water index, intrathoracic blood volume index and pulmonary blood volume. For the measurement of the initial distribution volume of glucose, plasma volume and extracellular water, we used dilutions of glucose, indocyanine green and sinistrin respectively. Transpulmonary thermodilution and dilutions of glucose were repeated 75 minutes after the beginning of the fluid load.The patients in the septic group had higher volumes of extracellular water (median 295 vs 234 ml/kg, P <0.001), lower intrathoracic blood volume index (median 894 vs 1157 ml/m2, P <0.003), higher pulmonary permeability ratios (extravascular lung water/pulmonary blood volume) (P <0.003) and higher systemic permeability ratios (interstitial/plasma volume) (P <0.04). The intrathoracic blood volume index increase after fluid loading was lower in the septic group (10 vs 145 ml/m2). The pulmonary permeability ratios did not correlate with the systemic permeability ratios, and in the septic group, the percentage volume retained in the intrathoracic blood volumes after fluid loading did not correlate with the systemic permeability ratios. Septic shock can cause a redistribution of fluids. Fluid administration in these patients produced a minimal increase in intrathoracic blood volume, and the percentage of volume retained in this space was not correlated with the interstitial/plasma volume ratio.

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Stewart, Julian M., and Leslie D. Montgomery. "Reciprocal splanchnic-thoracic blood volume changes during the Valsalva maneuver." American Journal of Physiology-Heart and Circulatory Physiology 288, no. 2 (February 2005): H752—H758. http://dx.doi.org/10.1152/ajpheart.00717.2004.

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The Valsalva maneuver is frequently used to test autonomic function. Previous work demonstrated that the blood pressure decrease during the Valsalva maneuver relates to thoracic hypovolemia, which may preclude pressure recovery during phase II, even with normal resting peripheral vasoconstriction. We hypothesized that increased regional blood volume, specifically splanchnic hypervolemia, accounts for the degree of thoracic hypovolemia during the Valsalva maneuver. We studied 17 healthy volunteers aged 15–22 yr. All had normal blood volumes by dye dilution. Subjects also had normal vascular resistance while supine as well as normal vasoconstrictor responses during 35° upright tilt. We assessed changes in estimated splanchnic, pelvic-thigh, and lower leg blood volume, along with thoracic blood volume shifts, by impedance plethysmography before and during the Valsalva maneuver performed in the supine position. Early increases in splanchnic blood volume dominated the regional vascular changes during the Valsalva maneuver. The increase in splanchnic blood volume correlated well ( r2 = 0.65, P < 0.00001) with the decrease in thoracic blood volume, there was less correlation of the increase in pelvic blood volume ( r2 = 0.21, P < 0.03), and there was no correlation of the increase in leg blood volume ( r2 = 0.001, P = 0.9). There was no relation of thoracic hypovolemia with blood volume or peripheral resistance in supine or upright positions. Thoracic hypovolemia during the Valsalva maneuver is closely related to splanchnic hyperemia and weakly related to regional changes in blood volume elsewhere. Changes in baseline splanchnic vascular properties may account for variability in thoracic blood volume changes during the Valsalva maneuver.

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Levine, Nicole L., Yidan Zhang, Bang H. Hoang, Rui Yang, Zachary H. Jurkowski, Michael E. Roth, Jonathan B. Gill, et al. "LigaSure Use Decreases Intraoperative Blood Loss Volume and Blood Transfusion Volume in Sarcoma Surgery." Journal of the American Academy of Orthopaedic Surgeons 27, no. 22 (November 2019): 841–47. http://dx.doi.org/10.5435/jaaos-d-18-00144.

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Journal articles on the topic 'Blood volume'

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