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Books on the topic 'Blood samples and stains'

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Published: 6 September 2023

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1

Copyright Paperback Collection (Library of Congress), ed. Blood stains. New York: Kensington Pub. Corp., 2002.

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Blood stains. Waterville, Me: Thorndike Press, 2011.

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3

Loewen, Arley. Blood stains: A novel Afghan story. Kabul: Rahmat Publications, 2013.

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4

Blood stains on the veils of the kaaba. [Cairo]: State Publishing House, 1996.

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5

Miller, Tobin L. The use of search warrants to obtain blood samples from juveniles in "drunk driving" cases. Lansing, MI (P.O. Box 30205 Lansing 48909): Michigan Judicial Institute, 2000.

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6

Institute of Medicine (U.S.). Roundtable on Translating Genomic-Based Research for Health, ed. Challenges and opportunities in using residual newborn screening samples for translational research: Workshop summary. Washington, D.C: National Academies Press, 2010.

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7

F, Siems David, and Geological Survey (U.S.), eds. Analyses and descriptions of geochemical samples, Tray Mountain Wilderness, Chattachoochee Roadless Area, and Blood Mountain Wilderness, northeastern Georgia. [Reston, Va.?]: U.S. Dept. of the Interior, Geological Survey, 1988.

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8

Hunter, Connie. Blood Stains. Lulu.com, 2019.

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9

Blood Stains. MIRA, 2011.

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10

Blood Stains. Harlequin Enterprises, Limited, 2011.

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11

Blood Stains. Kensington Publishing Corporation, 2011.

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12

Blood Stains in Paradise. Selah Publishing Group, 2011.

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13

He, Anju, and Linna Zhou. Blood Stains of History. Asian Culture Press, LLC, 2022.

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14

First Story Group Staff Ark Putney Academy and Dawson Juno. Blood and Spit Samples. First Story Limited, 2018.

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15

Merritt. Obtaining Blood Samples, Video 2 (Mosby's Blood Video). C.V. Mosby, 1995.

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16

Bauer, Patrick L. Goodloe: Blood Stains the Fury of the Coming Storm. Xlibris Corporation LLC, 2007.

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17

Bauer, Patrick L. Goodloe: Blood Stains the Fury of the Coming Storm. Xlibris Corporation LLC, 2007.

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18

Lindsay, Douglas. Blood That Stains Your Hands: DS Hutton Book 3. Independently Published, 2017.

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19

Jamaica. Blood Stains of a Shotta 3: Always Us, Never Them. Lock Down Publications, 2020.

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20

Blood Stains A Child Of Africa Reclaims Her Human Rights. Uncut/Voices Press, 2010.

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21

Ink Clots, Tear Stains, Blood on Cross: Where America Finds True Freedom Again. iUniverse, Incorporated, 2008.

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22

Fawcett, John. A study of free drug assays in human blood samples by G.L.C. 1985.

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23

Edwards, Rachel G. Evaluation of dried blood spot samples for the investigation of prolonged neonatal jaundice. 1996.

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24

A Note upon serum diagnosis by means of dried blood samples in (experimental) cholera. [S.l.]: D. Appleton, 1985.

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25

A note upon serum diagnosis by means of dried blood samples in (experimental) cholera. [S.l.]: D. Appleton, 1985.

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26

Assessing Anticoagulant Resistance in Rats and Coagulation Effects in Birds Using Small-Volume Blood Samples. Not Avail, 2005.

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27

SINGH, Uttam, and A. Study T. O. Estimate Acetone I. N. Insulin Dependent A. N. D. Insulin Independent Diabetic (KETOACIDOSIS) Persons Using Blood Samples R. Study to Estimate Acetone in Insulin Dependent and Insulin Independent Diabetic (ketoacidosis) Persons Using Blood Samples. Independently Published, 2018.

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28

Singh, Uttam. Study to Estimate Acetone in Insulin Dependent and Insulin Independent Diabetic (ketoacidosis) Persons Using Blood Samples. Independently Published, 2018.

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29

Sklar, Larry A., ed. Flow Cytometry for Biotechnology. Oxford University Press, 2005. http://dx.doi.org/10.1093/oso/9780195183146.001.0001.

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Flow cytometry is a sensitive and quantitative platform for the measurement of particle fluorescence. In flow cytometry, the particles in a sample flow in single file through a focused laser beam at rates of hundreds to thousands of particles per second. During the time each particle is in the laser beam, on the order of ten microseconds, one or more fluorescent dyes associated with that particle are excited. The fluorescence emitted from each particle is collected through a microscope objective, spectrally filtered, and detected with photomultiplier tubes. Flow cytometry is uniquely capable of the precise and quantitative molecular analysis of genomic sequence information, interactions between purified biomolecules and cellular function. Combined with automated sample handling for increased sample throughput, these features make flow cytometry a versatile platform with applications at many stages of drug discovery. Traditionally, the particles studied are cells, especially blood cells; flow cytometry is used extensively in immunology. This volume shows how flow cytometry is integrated into modern biotechnology, dealing with issues of throughput, content, sensitivity, and high throughput informatics with applications in genomics, proteomics and protein-protein interactions, drug discovery, vaccine development, plant and reproductive biology, pharmacology and toxicology, cell-cell interactions and protein engineering.
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30

Verification--development of a portable trichothecene sensor kit for the detection of T-2 mycotoxin in human blood samples. [Ottawa, Ont: s.n.], 1987.

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31

Sarah, Tribuiani. Blood Stains Are Red Ultraviolet Lights Are Blue Funny Gift: Ruled Blank 6x9 Cute Notebook, Original Appreciation Gag Gift for Graduation, College, High School, Congratulations Funny Journal for Your Favorite Graduate, Students. Independently Published, 2020.

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32

Hansoti, Bhakti. Pulmonary Tuberculosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0028.

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Mycobacterium tuberculosis (TB) is most commonly known for its manifestations in the lungs; symptoms include fever and chest pain (retrosternal pain and/or dull intracapsular pain). In the reactivation stage of TB, typical symptoms may include cough, weight loss, fatigue, fever, night sweats, chest pain, dyspnea, and/or hemoptysis. Symptoms may remain undiagnosed for several years. Poverty, HIV, and drug resistance are major contributors to the resurging global TB epidemic. Two kinds of tests are used to detect TB: the tuberculin skin test or a TB blood test. These tests only tell you if a person has been infected with the bacteria. The do not differentiate between latent TB infection and active TB. This distinction clinically suspected when the clinical picture of active TB matches with initial investigations (such as acid-fast bacilli stains, chest x-ray, or CT) and is definitively confirmed by the growth of M. tuberculosis in a clinical specimen.
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33

Kahn, S. Lowell. Catheter Modification Techniques for Venous Sampling. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0062.

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Venous sampling is critically important in the diagnosis and localization of pituitary, parathyroid, renal, adrenal, and ovarian endocrine tumors and conditions. Catheterization of smaller veins can present a challenge and may be responsible for technical failures, particularly with adrenal vein, parathyroid, and inferior petrosal sinus venous sampling. Beyond the inherent challenges of catheterization posed by small veins, obtaining adequate blood samples can be difficult because the return of blood from a small vein may be exceedingly slow. This chapter discusses techniques to enhance the return of venous blood flow from a diagnostic catheter in a small vein. These techniques are applicable to all venous sampling, but they are particularly beneficial when sampling small-caliber veins.
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34

Duran, Marinus, and Isabel Tavares de Almeida. Interpretation of Acylcarnitine Analysis Results. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0085.

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The analysis of acylcarnitines in plasma or blood spot samples by tandem mass spectrometry will detect all 15 defects of mitochondrial fatty acid beta-oxidation, although false negative results may occur in well-fed, non-fasting patients. Moreover, more than 20 organic acidemias can be detected by this methodological approach. An acylcarnitine profile should be part of the work-up of patients presenting with rhabdomyolysis and/or hypoglycemia and adults with an unexplained leukoencephalopathy. Cases with abnormal acylcarnitines require an analysis of urine organic acids as well as enzyme activity evaluation and molecular investigations to confirm the inherited defect.
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35

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 45-Year-Old Male with Toxin Exposure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0004.

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A patient presents with a sensorimotor neuropathy and believes he has been poisoned. The approach to the differential diagnosis of arsenic toxicity is presented. Comparisons with mimics of this entity are made, and clinical clues to its early detection are provided. There are typical skin and nail changes with can occur with arsenic poisoning. Arsenic poisoning can appear similar to Guillain-Barre syndrome with gastrointestinal symptoms and later an ascending paralysis. Urine arsenic levels are more reliable than blood levels. Hair and nail samples are very useful in confirming the diagnosis. Electrodiagnostic testing confirmed an axonal polyneuropathy. Treatment of arsenic poisoning is discussed. The recent lead contamination in Flint Michigan points out that heavy metal poisoning still occurs despite public health awareness.
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36

Smith, Rebecca. Smallpox. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0063.

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Symptoms of the smallpox virus include fever and a progressive papular rash that becomes vesicular and then pustular. A systemic inflammatory response syndrome (SIRS) leads to septic shock and death in 30% of cases. The definitive diagnosis can be confirmed via blood samples, lesion contents, or scrapings from crusts analyzed using electron microscopy, viral antigen immunohistochemistry, or polymerase chain reaction. The suspicion of a single smallpox case should lead to immediate notification of local public health authorities and the hospital epidemiologist. Because the disease does not exist in nature, smallpox should be considered the result of a bioterrorist attack until proven otherwise. An epidemiologic investigation is essential for determining the perimeter of the initial release so that tracking and quarantine of those exposed can be completed. Patients are extremely contagious and must be placed on contact, droplet, and airborne precautions in a negative pressure room.
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37

Fye, W. Bruce. The Expansion of Open-Heart Surgery and Cardiac Catheterization. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199982356.003.0011.

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Several groups began performing open-heart surgery during the late 1950s as simpler and less expensive heart-lung machines were marketed. Some surgeons attempted to develop operations to treat obstructed or leaking heart valves. Doctors who hoped to invent operations to treat diseased aortic and mitral valves (on the left side of the heart) stimulated the invention of new cardiac catheterization techniques. Until the mid-1950s, catheterization was limited to the right side of the heart. Catheterizing the left side of the heart presented several problems that were eventually solved. This procedure improved the accuracy of preoperative diagnosis, which contributed to better surgical outcomes. Cardiac catheters that were used to withdraw blood samples or measure intracardiac pressures could also be used to inject radiopaque contrast into the heart. This technique, angiocardiography, produced shadow pictures of the heart’s chambers that complemented data derived from catheterization and traditional clinical methods.
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38

Clark, Phillip. Haematology of Australian Mammals. CSIRO Publishing, 2004. http://dx.doi.org/10.1071/9780643091030.

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Haematology of Australian Mammals is a valuable guide to collecting and analysing the blood of Australian mammals for haematological studies and diagnosis and monitoring of disease. It outlines general principles for selecting sites for blood collection and for handling and analysing samples to achieve quality results. Chapters then describe the morphology and function of haematological cells, with reference to the known characteristics of Australian mammals in health and the changes that may be encountered in response to common diseases. Haemoparasites that have been encountered in Australian mammals are discussed next, along with comments on their pathogenicity. Lastly, haematological values from previously published studies are compiled into species-specific tables, providing a convenient reference to compare to the results of clinical cases. Written descriptions and colour photomicrographs of haematological cells from more than 100 species aid the identification of cells and the detection of abnormalities. Information is provided throughout for representative species from all the major groups of native Australian mammals including monotremes, polyprotodont marsupials, diprotodont marsupials, rats and mice, bats and marine mammals.
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39

Brimioulle, Serge. Pathophysiology, causes, and management of metabolic alkalosis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0257.

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Metabolic alkalosis occurs in up 51% of abnormal acid-base samples in the hospital. It is characterized by a primary increase in bicarbonate concentration and is always associated with chloride depletion. In critically-ill patients, it is most often generated by diuretic administration, digestive losses, alkali administration, or rapid correction of hypercapnia. Even after all causal factor are removed, it can be maintained by blood volume depletion and potassium depletion. Metabolic alkalosis results in hypercapnia, hypoxaemia, cardiac arrhythmias, altered consciousness, and neuromuscular hyperexcitability. It is first treated by removing the causal factors, whenever possible. Maintaining factors must be reversed by sodium chloride and/or potassium chloride administration. Acetazolamide and renal replacement therapy, when given for specific indications, can also correct the alkalosis. Lysine and arginine chloride are no longer used. If metabolic alkalosis is severe or when other treatments are contraindicated or ineffective, hydrochloric acid infusion is useful. Dilute hydrochloric acid can be infused safely, provided adequate precautions are taken to prevent extravascular leakage, vessel damage, and tissue necrosis.
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40

Mesquita, Emersom C., and Fernando A. Bozza. Diagnosis and management of viral haemorrhagic fevers in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0293.

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In a globalized scenario where widespread international travel allows viral agents to migrate from endemic to non-endemic areas, health care providers and critical care specialists must be able to readily recognize a suspected case of viral haemorrhagic fever (VHF). Early suspicion is pivotal for improving patient outcome and to ensure that appropriate biosafety measures be applied. VHFs are acute febrile illnesses marked by coagulation disorders and organ specific syndromes. VHFs represent a great medical challenge because diseases are associated with a high mortality rate and many VHFs have the potential for person-to-person transmission (Filoviruses, Arenavioruses, and Bunyaviroses). Dengue is the most frequent haemorrhagic viral disease and re-emergent infection in the world and, due to its public health relevance, severe dengue will receive special attention in this chapter. The diagnosis of VHFs is made by detecting specific antibodies, viral antigens (ELISA) and viral nucleic acid (RT-PCR) on blood samples. Supportive care is the cornerstone in the treatment of VHFs. Ribavirin should be started as soon as a case of VHF is suspected and discontinued if a diagnosis of Filovirus or Flavivirus infection is established. Adjunctive antimicrobial therapy is usually implemented to treat co-existing or secondary infections. Antimalarial treatment should also be initiated if a malaria test (thick blood films) is not quickly available and/or reliable and patients travel history is compatible. It is always recommended to apply appropriate biosafety measures and notify local infection control unit and state and national authorities.
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41

Gu, Wenduo, Yao Xie, and Qingbo Xu. Animal models to study pathophysiology of the vasculature. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0005.

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Animal models are designed to be preliminary tools for a better understanding of the pathogenesis, improvement in diagnosis, prevention, and therapy of vascular diseases in humans. Animal models are easily manageable, as compounding effects of dietary and environmental factors can be controlled experimentally. Blood vessel samples can be taken for detailed experimental and biomolecular examination. A thorough understanding of the animal models used is necessary and complete analysis must be validated so that the data can be extrapolated to humans. There are several species that are used for studying vascular pathophysiology, including mice, rats, rabbits, and pigs. Attracted by the well-defined genetic systems, a number of investigators have begun to use the mouse as an experimental system for arteriosclerosis research. Because vascular disorder is a complicated disease, which includes spontaneous (native) atherosclerosis, transplant arteriosclerosis, vein graft atherosclerosis, and angioplasty-induced restenosis, several models for studying all types of vascular disease have recently been established. Using these animal models, much knowledge concerning the pathogenesis of the disease and therapeutic intervention has been gained. This chapter will not attempt to cover all aspects of animal models, but will rather focus on the major progress in understanding the pathophysiology of the vasculature, the (dis)advantages of a variety of models, and how specific models can be appropriately chosen for different purposes of study.
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