Relevant bibliographies by topics / Blood pressure variability

Academic literature on the topic 'Blood pressure variability'

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Published: 4 June 2021
Last updated: 9 March 2023

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Journal articles on the topic "Blood pressure variability"

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Ostroumova, O. D., E. V. Borisova, and E. E. Pavleeva. "Blood Pressure Variability. Visit-to-Visit Blood Pressure Variability." Kardiologiia 17, no. 11 (2017): 68–75. http://dx.doi.org/10.18087/cardio.2017.11.10056.

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Cao, H., P. Norris, N. Mowery, R. Donahue, and J. Morris. "BLOOD PRESSURE VARIABILITY." Shock 25, Supplement 1 (June 2006): 54. http://dx.doi.org/10.1097/00024382-200606001-00163.

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Chatellier, G., Le Thi Huong Du, M. A. Dufloux, P. Corvol, and J. M??nard. "Blood Pressure Variability." Journal of Hypertension 3, no. 5 (October 1985): 535. http://dx.doi.org/10.1097/00004872-198510000-00024.

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Mancia, G. "Blood Pressure Variability." Journal of Cardiovascular Pharmacology 16 (January 1990): S1—S6. http://dx.doi.org/10.1097/00005344-199000006-00002.

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Mancia, G. "Blood Pressure Variability." Journal of Cardiovascular Pharmacology 16 (January 1990): S1—S6. http://dx.doi.org/10.1097/00005344-199006166-00002.

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Rabkin, Simon W. "Blood Pressure Variability." Hypertension 75, no. 5 (May 2020): 1161–62. http://dx.doi.org/10.1161/hypertensionaha.119.14279.

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Krakoff, Lawrence R., and Robert A. Phillips. "Blood Pressure Variability." Journal of the American College of Cardiology 68, no. 13 (September 2016): 1387–88. http://dx.doi.org/10.1016/j.jacc.2016.07.721.

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Dolan, Eamon, and Eoin O'Brien. "Blood Pressure Variability." Hypertension 56, no. 2 (August 2010): 179–81. http://dx.doi.org/10.1161/hypertensionaha.110.154708.

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Schillaci, Giuseppe, Giacomo Pucci, and Gianfranco Parati. "Blood Pressure Variability." Hypertension 58, no. 2 (August 2011): 133–35. http://dx.doi.org/10.1161/hypertensionaha.111.175752.

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Goldsmith, David, and Adrian Cork. "Blood pressure variability measurements." Nephrology Dialysis Transplantation 15, no. 1 (January 1, 2000): 131–32. http://dx.doi.org/10.1093/ndt/15.1.131.

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Dissertations / Theses on the topic "Blood pressure variability"

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Soueidan, Karen. "Augmented blood pressure measurement through the estimation of physiological blood pressure variability." Thesis, University of Ottawa (Canada), 2010. http://hdl.handle.net/10393/28828.

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Current noninvasive blood pressure (BP) measurement methods estimate the systolic and diastolic blood pressure (SBP and DBP) at two random instants in time. The BP variability and its serious consequences on the measurement are not recognized by most physicians. The standard for automated BP devices sets a maximum allowable system error of +/- 5 mmHg, even though natural BP variability often exceeds these limits. This thesis characterizes the variability of SBP and DBP and proposes a new approach to augment the conventional noninvasive measurement using simultaneous recordings of the oscillometric and continuous arterial pulse waveforms by providing: 1) The mean SBP (or DBP) over the measurement interval, 2) Their respective standard deviations, and 3) An indicator as to whether or not the oscillometric reading is an outlier. Recordings with healthy subjects showed that the approach has prominent potential and does not suffer from bias relative to the conventional method.
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Svensson, Per. "Blood pressure, blood pressure variability and myocardial ischemia : studies in patients with peripheral arterial disease and matched control subjects /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-826-2.

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Dawson, Suzanne L. "Blood pressure variability and cerebral autoregulation in acute ischaemic stroke." Thesis, University of Leicester, 2000. http://hdl.handle.net/2381/29609.

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This thesis examines the haemodynamic changes that occur following acute ischaemic stroke with reference to beat-to-beat blood pressure (BP) levels and variability as well as static and dynamic cerebral autoregulation (CA). Elevated 24h BP levels following acute stroke are associated with a poor outcome, but whether shorter durations of recording using beat-to-beat measurements or other BP parameters such as variability have the same prognostic significance is unknown. A single 10 minute non-invasive beat-to-beat BP monitoring period following acute cerebral infarction showed that increasing BP levels and beat-to-beat beat variability of mean arterial and diastolic BP were associated with a worse prognosis in terms of post ictal death/disability. These initial prognostic findings for BP variability might be explained if CA responses to rapid changes in systemic BP were impaired post stroke. The second part of the thesis set out to measure dynamic and static CA using novel non-invasive techniques employing transcranial Doppler ultrasonography to measure cerebral blood flow velocity in response to non-pharmacologically induced pressor and depressor BP stimuli. The initial study found that in normal controls, the reproducibility and actual values obtained for the autoregulatory indices for both static and dynamic tests varied according to the BP stimulus used. As a result of these initial studies thigh cuff release was used as the stimulus for dynamic CA, and isometric hand grip and thigh cuff inflation as the stimuli for static CA. When the CA results for a study group of acute ischaemic stroke patients were compared to an age and sex matched control group dynamic CA was significantly reduced in the patients' affected and non-affected hemispheres, whereas static CA was unimpaired.
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Terathongkum, Sangthong. "Relationships Among Stress, Blood Pressure, and Heart Rate Variability in Meditators." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1497.

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PROBLEM STATEMENT: Growing evidence indicates that psychological stress contributes to cardiovascular diseases through complex neuroendocrine mechanisms. Psychological stress leads to several physiological responses including increased heart rate (HR) and blood pressure (BP) as well as decreased heart rate variability (HRV) through alterations in the autonomic nervous system (ANS), specifically increased sympathetic nervous system (SNS) activity and decreased parasympathetic nervous system (PNS) activity. Meditation is thought to induce an innate relaxation response leading to reduced psychological stress. Findings from past studies have provided inconclusive evidence regarding the direction and strength of relationships among stress, BP, HRV, and meditation practice. PROCEDURES: A cross-sectional descriptive-correlational design was used to examine relationships among perceived stress, BP, HRV and meditation practice in meditators. A convenience sample of 71 meditators at two meditation centers in the southeast United States was used. Sample size was based on a power analysis. Each participant was asked to complete meditation, perceived stress, and demographic questionnaires. Participants' BP was measured before meditation and HRV was recorded during a 30 minute meditation session. Finally, BP was recorded after meditation. RESULTS: Participants were predominantly female (55%), Caucasian/white (94%), and Buddhist (76%), with 93% having at least college graduate. Most participants practiced soto zen or vipassana meditation (45% and 30%, respectively). The average length of total meditation practice was 103.66 months. Participants practiced meditation an average of once a day for 4 days a week with mean session duration of 34 minutes. Most participants had a low level of perceived stress and normal HRV. There was a statistically significant decrease in mean systolic BP after meditation (t = 5.31, p CONCLUSIONS: The results suggested meditators had low levels of perceived stress and that meditation had an effect on systolic BP and perceived current stress. Future research needs to include longitudinal studies to elucidate the cumulative effects of consistent meditation practice on psychological and physiological outcomes.
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Rantala, M. (Maire). "Dietary modification and genetic variability of atherosclerosis risk factors." Doctoral thesis, University of Oulu, 2000. http://urn.fi/urn:isbn:9514256522.

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Abstract The risk factors for atherosclerosis and coronary heart disease (CHD) are multiple and may interact with each other. Diet has a significant role among the main risk factors for atherosclerosis, as it regulates the levels of plasma lipids and lipoproteins, their oxidative modification or protection from oxidation, blood pressure, energy balance, and thrombogenesis. Nutrients can transfer their effects directly through plasma concentrations or modify the cell transduction or gene expression of important regulatory genes. The response to dietary modification varies between individuals. The plasma cholesterol response induced by dietary modification is at least partly regulated genetically and some of the variation is explained by other environmental factors. Apolipoprotein E (apo E) and apolipoprotein B (apo B) are the key regulatory proteins in cholesterol and lipoprotein metabolism. The genetic variation of apo E is associated with the plasma lipid levels and the CHD risk. The polymorphic variation of the apo B gene is also associated with increased plasma cholesterol and CHD risk. Obesity is associated with increased morbidity and mortality. Plasma lipid abnormalities, impaired glucose metabolism and increased blood pressure caused by obesity are the main reasons for increased CHD mortality among obese subjects. To study the magnitude of the response to dietary modification, genetically selected groups were investigated. Dietary modification had a significant impact on plasma total, LDL, and HDL cholesterol concentrations, and the individual response in plasma LDL cholesterol varied from 3 to 100%. The role of genetic variation in the apo E gene was not significant in the lipid response, but the blood pressure response was more distinct among subjects with the ε 4 allele than those with the ε 3 allele. The determination of apo B EcoRI and MspI gene polymorphisms revealed subjects with a greater response to diet, a finding which may have clinical importance in the future for the attempt to identify subjects for effective dietary counselling. The effect of caloric restriction on gene expression was studied in obese gallstone patients. Moderate weight reduction during caloric restriction was associated with reduced lipoprotein lipase gene expression, while the cholesteryl ester transfer protein gene expression remained unchanged. Some of the beneficial changes in plasma lipids and lipoproteins during and after weight reduction may be followed by altered transcription of their modifying genes. Meta-analysis is a modern and generally accepted method. Many clinical uncertainties can be solved by combining all the data available to a quantitative and objective analysis. However, the use of meta-analysis do not resolve the problem of the effect of publication bias.
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Manning, Lisa Suzanne. "Blood pressure variability : its definition, measurement, and prognostic significance in acute stroke." Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37832.

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Background: How best to manage elevated blood pressure (BP) in the acute stroke period remains unclear. Blood pressure variability (BPV), that is variability in BP over a period of time, may predict outcome in acute stroke, and could explain the ongoing uncertainty surrounding acute stroke BP management. Moreover, it presents a potentially modifiable therapeutic target. Evidence of the effect of BPV on outcome following stroke is scarce, and how best to measure or define BPV in acute stroke is unknown. Objectives: This thesis aimed to determine the prognostic significance of BPV in acute stroke, and to investigate how best to measure and define BPV in the immediate post-stroke period. Methods: The following studies were undertaken: a systematic review and meta‐analysis of observational studies to assess the effect of BPV on outcome in acute stroke; a feasibility study to assess the ability of a novel BP measurement device to capture very‐short‐term BPV; post‐hoc analyses of three acute stroke randomised controlled trials to assess the effect of BPV on outcome using individual patient data; a prospective observational study to measure acute stroke BPV using a variety of measurement techniques. Results: This thesis has demonstrated that greater BPV is associated with poor outcome in acute stroke. However, the presence and strength of this relationship is influenced by a number of factors including: frequency, timing, and nature of BP measurements; time from stroke onset; BPV parameters; use of thrombolytic therapy; and the timing of outcome measures. For the first time in acute stroke, the observational study describes and compares beat‐to‐beat, casual cuff, and ambulatory BP monitoring (ABPM) derived BPV, showing that it is feasible and acceptable to patients to measure BPV using casual cuff and beat‐to‐beat measurements, and reports significant associations between beat‐to‐beat and casual cuff BPV parameters and outcome.
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Seydnejad, Saeid Reza. "Analysis of heart rate variability and blood pressure variation by nonlinear modelling techniques." Thesis, Imperial College London, 1998. http://hdl.handle.net/10044/1/7814.

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Schurtz-Bouissou, Camille. "Relations entre la variabilité tensionnelle et la rigidité des gros troncs artériels chez le rat : Etudes dans trois modèles expérimentaux." Thesis, Paris Est, 2014. http://www.theses.fr/2014PEST0070.

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La rigidité artérielle ayant une valeur prédictive forte et indépendante d'évènements cardiovasculaires, nous émettons l'hypothèse que l'accumulation de variations de contraintes hémodynamiques altère la fonction et la structure des gros troncs artériels, indépendamment du niveau de pression artérielle. Nous avons donc mesuré l'impact de la variabilité tensionnelle sur la rigidité et la structure artérielles dans différents modèles de variabilité tensionnelle chez le rat.Chez le rat barodénervé et le rat sympathectomisé par la guanéthidine, 2 modèles de variabilité tensionnelle à court terme, une augmentation de la rigidité artérielle est associée à des altérations tissulaires différentes. En effet chez les rats barodénervés, une hypertrophie aortique est couplée à une augmentation du collagène et des attachements cellule-matrice (fibronectine et intégrine α5). Au contraire, chez les rats sympathectomisés, une hypotrophie vasculaire est associée à une diminution de l'élastine et une augmentation des attachements via l'intégrine αv.Nous avons ensuite créé, caractérisé et validé un modèle de variabilité tensionnelle à long terme, le rat spontanément hypertendu traité de façon discontinue par un antihypertenseur. Le traitement discontinu réduit la pression artérielle systolique tout en augmentant isolément la variabilité tensionnelle à long terme. La rigidité artérielle, élevée sous traitement discontinu, est associée à une hypertrophie vasculaire avec augmentation des attachements (fibronectine et intégrine αv) et sans modification du rapport élastine/collagène.En conclusion, l'élevation de variabilité tensionnelle engendre de la rigidité artérielle, et ce à pression artérielle constante. Les altérations structurales dans les modèles de variabilité tensionnelle étudiés impliquent des mécanismes différents reposant sur des modifications des relations cellule-matrice, mettant en jeu la fibronectine et les intégrines α5 et αv
Arterial stiffness is nowadays accepted as a strong and independent predictor of cardiovascular disease. We hypothesized that increased blood pressure variability (BPV) may lead to arterial damage, independently of the blood pressure level. We thus aimed investigating the relationship between BPV and arterial stiffness and composition of the aorta in different rat models of increased BPV.In a first study performed in two models of increased short term BPV, sinoaortic denervated and chemically sympathectomized rats, an increase in wall stiffness was associated with different modifications of cell-extracellular matrix adhesion. Indeed in sinoaortic denervated rats, increased media cross-sectional area was coupled with an increased collagen content and muscle cell attachments to its cell-extracellular matrix (fibronectin and its α5β1 integrin). In contrast, chemically sympathectomized rats were characterized by a reduced media cross-sectional area associated to a reduction of elastin content and upregulation of αvβ3 integrin.In a second study, we created, characterized and validated a new experimental model of long term BPV by discontinuously treating spontaneously hypertensive rats with valsartan. Discontinuous treatment reduced systolic blood pressure level but increased long term BPV. In addition, this treatment regimen failed to reduce arterial stiffness and induced a vascular hypertrophy without modification of elastin/collagen ratio. Discontinuous treatment also highly increased vascular fibronectin in parallel to αv integrin.In conclusion, a rise of both short- and long-term BPV leads to an increase in arterial stiffness, independently of blood pressure level. The structural changes at the origin of this increase in arterial rigidity involve different mechanisms, in which fibronectin and integrin α5 and αv play a key role
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Gagnon, Marie-Claude. "Sympathovagal influences on heart rate and blood pressure variability in highly trained endurance athletes." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=24080.

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The evidence for an increase in resting vagal tone to explain the lower heart rate of endurance athletes remains controversial. This study used spectral analysis of heart rate (HRV) and blood pressure (BPV) variability to examine the vagal and sympathovagal influences on the sinus node in 12 endurance-trained athletes (A) and 10 age and sex-matched control subjects (C) (age: 26(1.2) yrs; VO2max: A: 68.2(2.1) vs C: 41.4(2.0) ml/kg/min; p $<$ 0.05). Continuous ECG and BP recordings were obtained during supine rest, sitting, controlled respiration (12 breaths/min), standing, exercise at heart rates of 100 and 130 beats/min, and after 5 and 15 minutes of seated recovery. No differences were observed between A and C in the vagal or high frequency (HF) components (48.0(4.0) vs 44.2(6.9) nu), in the low frequency (LF) components (55.8(6.9) vs 52.0(4.0) nu), or in the LF/HF ratios (1.72(0.4) vs 1.22(0.2)) of the HRV spectral components at rest or other experimental conditions, despite the lower resting heart rates of the A (53.1(1.8) vs 65.1(2.1) beats/min; p $<$ 0.05). Standing and exercise decreased the HF components and increased the LF/HF ratios similarily in both the A and C, controlled respiration induced similar rises in the HF component of HRV. Despite a significantly higher systolic BP in athletes, no difference was observed between A and C in the HF or LF components of either systolic or diastolic BPV. Exercise induced an increase in the HF component of BPV which was associated with the rise in breathing frequency. These results indicate a similar sinoatrial sympathovagal balance in A and C suggesting that training-induced bradycardia could result from an adaptation in the intrinsic sinoatrial properties. Also, endurance training does not appear to influence the beat-to-beat BPV.
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Diaz, Keith M. "Blood Pressure Variability: Relationship with Endothelial Health and Effects of an Exercise Training Intervention." Diss., Temple University Libraries, 2012. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/183386.

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Kinesiology
Ph.D.
Purpose: Evidence has accumulated to show that blood pressure variability (BPV) has a striking relationship with cardiovascular (CV) risk. Despite the mounting evidence implicating BPV as a CV risk factor, scant attention has been paid to: (1) the mechanisms by which high BPV confers greater CV risk; and (2) the efficacy of non-pharmacologic treatment modalities in the attenuation of BPV. In order to address these two unresolved questions, the purpose of this dissertation was twofold. The purpose of study #1 was to investigate the association between measures of short-term BPV (24-hour BPV) and long-term BPV (visit-to-visit BPV) with markers of endothelial health in a cohort of African Americans in order to determine if increased BPV may confer greater CV risk by eliciting injury to the endothelium. The purpose of study #2 was to investigate the effects of a 6-month aerobic exercise training (AEXT) intervention on visit-to-visit BPV and 24-hour BPV in the same cohort of African Americans in order to provide the first available data on the efficacy of a non-pharmacologic treatment modality in the lowering of BPV. Methods: We recruited 72 African Americans who were sedentary, non-diabetic, non-smoking, and free of CV and renal disease. Before and after a 6-month AEXT intervention, office blood pressure (BP) was measured at 3 separate visits and 24-hour ambulatory BP monitoring (ABPM) was conducted to measure visit-to-visit BPV and 24-hour BPV, respectively. Right brachial artery diameter was assessed at rest, during flow-mediated dilation (FMD), and after nitroglycerin-mediated dilation (NMD). Peak and area under the curve (AUC) were calculated as measures of FMD and NMD, and the FMD/NMD ratio was calculated as a measure of endothelial function normalized by smooth muscle function. Fasted blood samples were obtained and were analyzed for circulating EMPs expressed as CD31+CD42- and CD62E+ EMPs. Results: In study #1, participants with higher 24-hour diastolic BPV (DBPV) had significantly lower CD31+CD42- EMPs compared to participants with lower 24-hour DBPV. When categorized according to visit-to-visit DBPV, participants with higher visit-to-visit DBPV had a significantly lower FMD/NMD ratio, and significantly higher %NMDpeak and NMDAUCs compared to participants with lower visit-to-visit DBPV. When analyzed as continuous variables, 24-hour mean arterial pressure variability (MAPV) was inversely associated with CD31+CD42- EMPs visit-to-visit DBPV was inversely associated with the FMD/NMD ratio and positively associated with %NMDpeak and NMDAUC; and 24-hour DBPV was positively associated with NMDAUC. All associations were independent of age, gender, BMI, mean BP, and pulse pressure. In study #2 investigating the effects of AEXT in 33 participants who completed the study, 24-hour DBPV and 24-hour MAPV were significantly increased after AEXT. The increase in 24-hour DBPV was independent of changes in BMI, mean BP, and self-reported sleep time. Heart rate variability (HRV) derived from ABPM was associated with the changes in 24-hour DBPV and 24-hour MAPV. There were no significant changes in visit-to-visit BPV after AEXT. Conclusions: The results from study #1 provide evidence that BPV is associated with vascular health as endothelial function was decreased in participants with high visit-to-visit DBPV, while smooth muscle function was increased in participants with higher visit-to-visit and 24-hour DBPV. The findings from study #2 show that 6-months of AEXT do not elicit beneficial changes in BPV. The finding of an association between changes in 24-hour BPV with HRV could indicate, however, that changes in activity levels during ABPM, in part, contributed to the observed changes in 24-hour BPV.
Temple University--Theses
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Books on the topic "Blood pressure variability"

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M, Di Rienzo, ed. Blood pressure and heart rate variability: Computer analysis, modelling and clinical applications. Amsterdam ; Washington: IOS Press, 1993.

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Jakovljević, Miroljub. Heart rate, blood pressure and respiration variability during inhalation of sub-anaesthetic levels of nitrous oxide. Portsmouth: University of Portsmouth, 2001.

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Carl, Pieper, and Schechter Clyde B, eds. Ambulatory monitoring and blood pressure variability. Science Press, 1991.

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Ambulatory Monitoring and Blood Pressure Variability. Science Press, 1991.

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Pickering, T. G. Ambulatory Monitoring and Blood Pressure Variability. 2nd ed. Science Press Ltd, 1999.

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Blood Pressure Monitoring: An International Journal Devoted to Research in Blood Pressure Monitoring and Variability (Periodicals). Lippincott Williams & Wilkins, 2003.

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Rienzo, M. Di, G. Parati, and G. Mancia. Blood Pressure and Heart Rate Variability: Computer Analysis, Methodology and Clinical Applications. IOS Press, Incorporated, 1993.

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Rienzo, M. Di. Blood Pressure and Heart Rate Variability, (Studies in Health Technology and Informatics, V). IOS Press, 1993.

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Korhonen, Ilkka. Methods for the analysis of short-term variability of heart rate and blood pressure in frequency domain. 1997.

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Takalo, Reijo. Variability of Blood Pressure and Heart Rate in Borderline and Mild Hypertension: With Special Reference to Spectral Analysis (Comprehensive Summaries of Uppsala Dissertations, 854). Uppsala Universitet, 1999.

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Book chapters on the topic "Blood pressure variability"

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Parati, Gianfranco, and Juan Eugenio Ochoa. "Blood Pressure Variability." In Updates in Hypertension and Cardiovascular Protection, 395–417. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-75310-2_28.

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Palatini, P., L. Mos, P. Mormino, A. Di Marco, L. Munari, M. Del Torre, F. Valle, A. C. Pessina, and C. Dal Palù. "Blood pressure variability during athletics." In Blood Pressure Measurements, 225–40. Heidelberg: Steinkopff, 1990. http://dx.doi.org/10.1007/978-3-642-72423-7_24.

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Parati, Gianfranco, and Juan Eugenio Ochoa. "Blood Pressure Variability and Blood Pressure Load." In Updates in Hypertension and Cardiovascular Protection, 31–58. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-93320-7_3.

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Parati, Gianfranco, Juan Eugenio Ochoa, Yutaka Imai, Anastasios Kollias, Efstathios Manios, Takayoshi Ohkubo, Kazuomi Kario, George S. Stergiou, and Grzegorz Bilo. "Home Blood Pressure Variability." In Updates in Hypertension and Cardiovascular Protection, 143–54. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23065-4_14.

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Harshfield, G. A., T. G. Pickering, G. D. James, and S. G. Blank. "Blood pressure variability and reactivity in the natural environment." In Blood Pressure Measurements, 241–51. Heidelberg: Steinkopff, 1990. http://dx.doi.org/10.1007/978-3-642-72423-7_25.

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Öney, T., W. Meyer-Sabellek, and H. Weitzel. "Variability of arterial blood pressure in normal and in hypertensive pregnancy." In Blood Pressure Measurements, 217–23. Heidelberg: Steinkopff, 1990. http://dx.doi.org/10.1007/978-3-642-72423-7_23.

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Otsuka, Kuniaki, Germaine Cornelissen, and Franz Halberg. "Diagnosing Vascular Variability Disorders and Vascular Variability Syndromes." In Chronomics and Continuous Ambulatory Blood Pressure Monitoring, 459–79. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-54631-3_16.

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González-Juanàtey, José Ramón, and Carlos González-Juanàtey. "Pronostic Implications of Blood Pressure Variability." In Advances in Noninvasive Electrocardiographic Monitoring Techniques, 371–79. Dordrecht: Springer Netherlands, 2000. http://dx.doi.org/10.1007/978-94-011-4090-4_36.

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Salvi, Paolo. "Arterial Stiffness and Blood Pressure Variability." In Pulse Waves, 69–78. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40501-8_3.

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Veerman, D. P. "Various aspects of blood pressure variability." In Blutdruckselbstmessung, 75–81. Heidelberg: Steinkopff, 1994. http://dx.doi.org/10.1007/978-3-642-85428-6_8.

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Conference papers on the topic "Blood pressure variability"

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Avolio, Alberto P., Ke Xu, and Mark Butlin. "Effect of large arteries on blood pressure variability." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6610441.

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Tsoi, Kelvin KF, Max WY Lam, Felix CH Chan, Hoyee Hirai, Baker KK Bat, Samuel YS Wong, and Helen ML Meng. "Classification of Visit-to-Visit Blood Pressure Variability." In DH '17: International Conference on Digital Health. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3079452.3079454.

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Murray, Alan, Dingchang Zheng, Chengyu Liu, David Graham, Jeff Neasham, Adrian Cossor, and Clive Griffiths. "Variability in Blood Pressure Measurements from Recorded Auscultation Sounds." In 2019 Computing in Cardiology Conference. Computing in Cardiology, 2019. http://dx.doi.org/10.22489/cinc.2019.183.

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Corino, V. D. A., L. T. Mainardi, S. Belletti, and F. Lombardi. "Spectral analysis of blood pressure variability in atrial fibrillation." In 2008 35th Annual Computers in Cardiology Conference. IEEE, 2008. http://dx.doi.org/10.1109/cic.2008.4749171.

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Mojon, Hermida, Fernandez, and Ayala. "Circadian And Ultradian Blood Pressure Variability Assessed By Periodic Regression." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.595835.

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Mojon, A., R. C. Hermida, J. R. Fernandez, and D. E. Ayala. "Circadian and ultradian blood pressure variability assessed by periodic regression." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761201.

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Holme, Nathalie L., Maria Skytioti, Signe Sovik, and Maja Elstad. "Induced respiratory variability in arterial blood pressure lowers cerebral blood flow in healthy humans." In 2020 11th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2020. http://dx.doi.org/10.1109/esgco49734.2020.9158010.

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Nikolic, Dragana, Anthony A. Birch, Ronney B. Panerai, and David M. Simpson. "Assessing cerebral blood flow control from variability in blood pressure and arterial CO2 levels." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7318725.

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Avolio, Alberto P., Ke Xu, and Mark Butlin. "Application of cardiovascular models in comparative physiology and blood pressure variability." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609476.

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Škorić, Iva, Ivana Trutin, Karolina Kramarić, Ivica Škoro, and Matej Šapina. "380 What influences blood pressure variability in children with essential hypertension?" In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.380.

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