Journal articles: 'Blood pressure;aircraft noise;child'

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Stansfeld, S. A., C. R. Clark, G. Turpin, L. M. Jenkins, and A. Tarnopolsky. "Sensitivity to noise in a community sample: II. Measurement of psychophysiological indices." Psychological Medicine 15, no. 2 (May 1985): 255–63. http://dx.doi.org/10.1017/s0033291700023539.

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SynopsisA sample of 77 women of high and low noise sensitivity in 1977, living in areas of high and low exposure to aircraft noise, were interviewed in the community in 1980. High, intermediate and low noise sensitive women were compared, using measures of blood pressure, heart rate, skin conductance, hearing threshold, uncomfortable loudness level and magnitude estimation of six tones. These physiological measures did not clearly distinguish different noise sensitivity groups, except that highly noise sensitive women had a consistently slower heart rate. Noise sensitivity was not related to auditory threshold. In the high aircraft noise area there were significantly more skin conductance responses than in the low aircraft noise area, irrespective of noise sensitivity. This may be the result of chronic exposure to high aircraft noise.

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Kourieh, Aboud, Lise Giorgis-Allemand, Liacine Bouaoun, Marie Lefèvre, Patricia Champelovier, Jacques Lambert, Bernard Laumon, and Anne-Sophie Evrard. "Incident hypertension in relation to aircraft noise exposure: results of the DEBATS longitudinal study in France." Occupational and Environmental Medicine 79, no. 4 (January 19, 2022): 268–76. http://dx.doi.org/10.1136/oemed-2021-107921.

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BackgroundAlthough several cross-sectional studies have shown that aircraft noise exposure was associated with an increased risk of hypertension, a limited number of longitudinal studies have addressed this issue. This study is part of the DEBATS (Discussion on the health effect of aircraft noise) research programme and aimed to investigate the association between aircraft noise exposure and the incidence of hypertension.MethodsIn 2013, 1244 adults living near three major French airports were included in this longitudinal study. Systolic and diastolic blood pressure, as well as demographic and lifestyle factors, were collected at baseline and after 2 and 4 years of follow-up during face-to-face interviews. Exposure to aircraft noise was estimated for each participant’s home address using noise maps. Statistical analyses were performed using mixed Poisson and linear regression models adjusted for potential confounding factors.ResultsA 10 dB(A) increase in aircraft noise levels in terms of Lden was associated with a higher incidence of hypertension (incidence rate ratio (IRR)=1.36, 95% CI 1.02 to 1.82). The association was also significant for Lday (IRR 1.41, 95% CI 1.07; to 1.85) and Lnight (IRR 1.31, 95% CI 1.01 to 1.71). Systolic and diastolic blood pressure increased with all noise indicators.ConclusionThese results strengthen those obtained from the cross-sectional analysis of the data collected at the time of inclusion in DEBATS, as well as those from previous studies conducted in other countries. Hence, they support the hypothesis that aircraft noise exposure may be considered as a risk factor for hypertension.

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Rojek, Marta, Marek W. Rajzer, Wiktoria Wojciechowska, Tomasz Drożdż, Paweł Skalski, Tomasz Pizoń, Andrzej Januszewicz, and Danuta Czarnecka. "Relationship among long-term aircraft noise exposure, blood pressure profile, and arterial stiffness." Journal of Hypertension 37, no. 7 (July 2019): 1350–58. http://dx.doi.org/10.1097/hjh.0000000000002060.

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Wojciechowska, Wiktoria, Andrzej Januszewicz, Tomasz Drożdż, Marta Rojek, Justyna Bączalska, Michał Terlecki, Karol Kurasz, et al. "Blood Pressure and Arterial Stiffness in Association With Aircraft Noise Exposure:Long-Term Observation and Potential Effect of COVID-19 Lockdown." Hypertension 79, no. 2 (February 2022): 325–34. http://dx.doi.org/10.1161/hypertensionaha.121.17704.

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In a cross-sectional analysis of a case-control study in 2015, we revealed the association between increased arterial stiffness (pulse wave velocity) and aircraft noise exposure. In June 2020, we evaluated the long-term effects, and the impact of a sudden decline in noise exposure during the coronavirus disease 2019 (COVID-19) lockdown, on blood pressure and pulse wave velocity, comparing 74 participants exposed to long-term day-evening-night aircraft noise level > 60 dB and 75 unexposed individuals. During the 5-year follow-up, the prevalence of hypertension increased in the exposed (42% versus 59%, P =0.048) but not in the unexposed group. The decline in noise exposure since April 2020 was accompanied with a significant decrease of noise annoyance, 24-hour systolic (121.2 versus 117.9 mm Hg; P =0.034) and diastolic (75.1 versus 72.0 mm Hg; P =0.003) blood pressure, and pulse wave velocity (10.2 versus 8.8 m/s; P =0.001) in the exposed group. Less profound decreases of these parameters were noticed in the unexposed group. Significant between group differences were observed for declines in office and night-time diastolic blood pressure and pulse wave velocity. Importantly, the difference in the reduction of pulse wave velocity between exposed and unexposed participants remained significant after adjustment for covariates (−1.49 versus −0.35 m/s; P =0.017). The observed difference in insomnia prevalence between exposed and unexposed individuals at baseline was no more significant at follow-up. Thus, long-term aircraft noise exposure may increase the prevalence of hypertension and accelerate arterial stiffening. However, even short-term noise reduction, as experienced during the COVID-19 lockdown, may reverse those unfavorable effects.

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Prasetya, Ekawaty, Zul Fikar Ahmad, and Siti Surya Indah Nurdin. "AIRPORT NOISE LEVEL AND ITS EFFECT ON BLOOD PRESSURE ON THE GORONTALO COMMUNITY." Jambura Journal of Health Sciences and Research 4, no. 1 (January 18, 2022): 500–509. http://dx.doi.org/10.35971/jjhsr.v4i1.12248.

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The rapidly increasing flight activity affects the health of the population living, traveling, and working around or at airports. Noise generated by aircraft causes physiological and psychological disturbances. This study aims to assess the effect of noise on increasing blood pressure. The research was conducted in Tolotio Village, Gorontalo Regency in 2019. Respondents were selected using a purposive sampling technique. The Noise was collected using a Sound Level Meter, and blood pressure was collected using a sphygmomanometer. Data were analyzed using One Way Anova statistical test. The results of the analysis showed that there was a significant difference in the systolic blood pressure of the respondents who lived in the three sampling locations with p-value = 0.016 = 0.05. However, for diastolic blood pressure, there was no significant difference between respondents who lived in the three sampling locations p-Value = 0.670 = 0.05. It is recommended to provide education about the dangers of airport noise for public health.

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Rojek, M., M. Rajzer, T. Pizon, P. Skalski, and D. Czarnecka. "[PP.06.27] THE EFFECT OF NIGHTTIME AIRCRAFT NOISE EXPOSURE ON CIRCADIAN BLOOD PRESSURE PROFILE." Journal of Hypertension 35 (September 2017): e136. http://dx.doi.org/10.1097/01.hjh.0000523357.90883.82.

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Evans, Gary W., Monika Bullinger, and Staffan Hygge. "Chronic Noise Exposure and Physiological Response: A Prospective Study of Children Living Under Environmental Stress." Psychological Science 9, no. 1 (January 1998): 75–77. http://dx.doi.org/10.1111/1467-9280.00014.

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Chronic exposure to aircraft noise elevated psychophysiological stress (resting blood pressure and overnight epinephrine and norepinephrine) and depressed quality-of-life indicators over a 2-year period among 9- to 11-year-old children. Data collected before and after the inauguration of a major new international airport in noise-impacted and comparison communities show that noise significantly elevates stress among children at ambient levels far below those necessary to produce hearing damage.

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Ancona, Carla, Chiara Badaloni, Valeria Fano, Tina Fabozzi, Francesco Forastiere, and Carlo Perucci. "Aircraft Noise and Blood Pressure in the Populations Living Near the Ciampino Airport in Rome." Epidemiology 20 (November 2009): S125—S126. http://dx.doi.org/10.1097/01.ede.0000362434.82215.9d.

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Aydin, Y., and M. Kaltenbach. "Noise perception, heart rate and blood pressure in relation to aircraft noise in the vicinity of the Frankfurt airport." Clinical Research in Cardiology 96, no. 6 (April 10, 2007): 347–58. http://dx.doi.org/10.1007/s00392-007-0507-y.

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Basner, Mathias, Maryam Witte, and Sarah McGuire. "Aircraft Noise Effects on Sleep—Results of a Pilot Study Near Philadelphia International Airport." International Journal of Environmental Research and Public Health 16, no. 17 (August 31, 2019): 3178. http://dx.doi.org/10.3390/ijerph16173178.

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Current objective data on aircraft noise effects on sleep are needed in the US to inform policy. In this pilot field study, heart rate and body movements were continuously measured during sleep of residents living in the vicinity of Philadelphia International Airport (PHL) and in a control region without aircraft noise with sociodemographic characteristics similar to the exposed region (N = 40 subjects each). The primary objective was to establish the feasibility of unattended field measurements. A secondary objective was to compare objective and subjective measures of sleep and health between control and aircraft noise exposed groups. For all measurements, there was less than 10% of data loss, demonstrating the feasibility of unattended home measurements. Based on 2375 recorded aircraft noise events, we found a significant (unadjusted p = 0.0136) exposure-response function between the maximum sound pressure level of aircraft noise events and awakening probability inferred from heart rate increases and body movements, which was similar to previous studies. Those living near the airport reported poorer sleep quality and poorer health than the control group in general, but when asked in the morning about their last night’s sleep, no significant difference was found between groups. Neither systolic nor diastolic morning blood pressures differed between study regions. While this study demonstrates the feasibility of unattended field study measurements, for a national study around multiple US airports refinements of the study design are necessary to further lower methodological expense and increase participation rates.

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Kalinovic, Sanela, Katie Frenis, Ahmad Al Zuabi, Matthias Oelze, Sebastian Steven, Miroslava Kvandova, Benjamin Ernst, et al. "Time‐dependent induction of vascular oxidative stress, inflammation, endothelial dysfunction and high blood pressure by aircraft noise exposure in mice." FASEB Journal 34, S1 (April 2020): 1. http://dx.doi.org/10.1096/fasebj.2020.34.s1.02202.

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Schmidt, Frank, Kristoffer Kolle, Katharina Kreuder, Boris Schnorbus, Philip Wild, Marlene Hechtner, Harald Binder, Tommaso Gori, and Thomas Münzel. "Nighttime aircraft noise impairs endothelial function and increases blood pressure in patients with or at high risk for coronary artery disease." Clinical Research in Cardiology 104, no. 1 (August 22, 2014): 23–30. http://dx.doi.org/10.1007/s00392-014-0751-x.

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Williams, Amber, Maureen Sanderson, Dejian Lai, Beatrice Selwyn, and Robert Lasky. "Intensive Care Noise and Mean Arterial Blood Pressure in Extremely Low-Birth-Weight Neonates." American Journal of Perinatology 26, no. 05 (December 11, 2008): 323–29. http://dx.doi.org/10.1055/s-0028-1104741.

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Wojciechowska, Wiktoria, Andrzej Januszewicz, Tomasz Drozdz, Marta Rojek, Justyna Baczalska, Michal Terlecki, Karol Kurasz, et al. "IMPACT OF REDUCTION IN AIRCRAFT NOISE EXPOSURE ON BLOOD PRESSURE AND ARTERIAL STIFFNESS DURING COVID-19 PANDEMIC: CROSS-SECTIONAL AND LONGITUDINAL STUDY." Journal of Hypertension 39, Supplement 1 (April 2021): e204. http://dx.doi.org/10.1097/01.hjh.0000746744.08145.f6.

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Ancona*, Carla, Martina Nicole Golini, Francesca Mataloni, Donatella Camerino, Gaetano Licitra, Marina Ottino, Salvatore Pisani, and Lorenzo Simonato. "Aircraft Noise Exposure and Blood Pressure Levels in the Populations Living Near Six Airports in Italy: Results of the SERA National Study." ISEE Conference Abstracts 2014, no. 1 (October 20, 2014): 2153. http://dx.doi.org/10.1289/isee.2014.p1-270.

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Brown, Gemma. "NICU Noise and the Preterm Infant." Neonatal Network 28, no. 3 (May 2009): 165–73. http://dx.doi.org/10.1891/0730-0832.28.3.165.

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Premature infants in the NICU are often exposed to continuous loud noise despite research documenting the presence and damaging effects of noise on the preterm infant’s development. Excessive auditory stimulation creates negative physiologic responses such as apnea and fluctuations in heart rate, blood pressure, and oxygen saturation. Preterm infants exposed to prolonged excessive noise are also at increased risk for hearing loss, abnormal brain and sensory development, and speech and language problems. Reducing noise levels in the NICU can improve the physiologic stability of sick neonates and therefore enlarge the potential for infant brain development. Recommendations include covering incubators with blankets, removing noisy equipment from the incubator environment, implementing a quiet hour, educating staff to raise awareness, and encouraging staff to limit conversation near infants.

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Mitchell, Pamela H., Catherine Kirkness, and Patricia A. Blissitt. "Cerebral Perfusion Pressure and Intracranial Pressure in Traumatic Brain Injury." Annual Review of Nursing Research 33, no. 1 (May 2015): 111–83. http://dx.doi.org/10.1891/0739-6686.33.111.

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Nearly 300,000 children and adults are hospitalized annually with traumatic brain injury (TBI) and monitored for many vital signs, including intracranial pressure (ICP) and cerebral perfusion pressure (CPP). Nurses use these monitored values to infer the risk of secondary brain injury. The purpose of this chapter is to review nursing research on the monitoring of ICP and CPP in TBI. In this context, nursing research is defined as the research conducted by nurse investigators or research about the variables ICP and CPP that pertains to the nursing care of the TBI patient, adult or child. A modified systematic review of the literature indicated that, except for sharp head rotation and prone positioning, there are no body positions or nursing activities that uniformly or nearly uniformly result in clinically relevant ICP increase or decrease. In the smaller number of studies in which CPP is also measured, there are few changes in CPP since arterial blood pressure generally increases along with ICP. Considerable individual variation occurs in controlled studies, suggesting that clinicians need to pay close attention to the cerebrodynamic responses of each patient to any care maneuver. We recommend that future research regarding nursing care and ICP/CPP in TBI patients needs to have a more integrated approach, examining comprehensive care in relation to short- and long-term outcomes and incorporating multimodality monitoring. Intervention trials of care aspects within nursing control, such as the reduction of environmental noise, early mobilization, and reduction of complications of immobility, are all sorely needed.

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Yashnikova, Maria V., Elena L. Poteriyaeva, Boris M. Doronin, Vladimir N. Maximov, Elena L. Smirnova, and Svetlana A. Karmanovskaya. "Clinical and phenotypical features of stroke and forecast in workers under exposure industrial noise." Russian Journal of Occupational Health and Industrial Ecology 61, no. 4 (May 25, 2021): 231–37. http://dx.doi.org/10.31089/1026-9428-2021-61-4-231-237.

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Introduction. The problem of preventing the development of stroke in workers under the influence of harmful production factors is urgent. The aim of the study was to study the characteristics of stroke in men under conditions of industrial noise exposure, to establish the prognostic value in the formation and course of the disease. Material and methods. The study involved 71 men with stroke from 30 to 65 years old, who worked in conditions of industrial noise in the professions of "pilot of civil aviation aircraft" and "adjuster of railway construction machines" for 5 years or more. In order to develop a mathematical model for predicting the development of stroke and to establish an individual risk, a group of 81 men from 30 to 65 years old, working under conditions of exposure to occupational noise for 5 years or more, who had no history of stroke data, were also examined. Using the method of sequential inclusion of the studied risk factors (Forward Stepwise), we selected those that made an independent significant contribution to the development of stroke. Based on the results obtained, a logistic regression formula was created, which determines the likelihood of stroke in men working under conditions of occupational noise. Results. In the group of patients with stroke, persons of middle age were found significantly more often - 53.5%. Ischemic stroke prevailed among clinical forms - 90.1%. Among the risk factors prevailed: arterial hypertension (AH) in 100.0% of cases, dyslipidemia factor in 78.9%, smoking factor in 59.1%. A direct moderate correlation was found between the level of systolic blood pressure, diastolic blood pressure and the index of the NIHSS 1 and NIHSS 2 scales. The influence of the following factors on the development of stroke was established: the level of total cholesterol (β-coefficient=1.0, p=0.001), the presence of atrial fibrillation factor (β-coefficient=2.9, p=0.004), the presence of a factor of alcohol abuse (β-coefficient=1.1, p=0.049), the patient has a diagnosis of grade 2 AH (β-coefficient=13, p=0.041) or a diagnosis of grade 3 hypertension (β-coefficient=1.8, p=0.008). A p(X) value exceeding 0.5 indicates a high risk of developing a stroke. The sensitivity of the proposed method was 76.1%, specificity - 74.1%. Conclusion. The proposed multivariate model is informative for the individual prediction of the development of stroke in men working in conditions of exposure to occupational noise, because reflects the degree of influence of one factor or another on the development of the disease.

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Kudish, G. "THE CASE OF ABORTION AT THE 5TH MONTH WITH A LIVING CHILD." Journal of obstetrics and women's diseases 6, no. 2 (August 18, 2020): 150–52. http://dx.doi.org/10.17816/jowd62150-152.

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On October 3, 1891, at 5 o'clock in the afternoon, a pregnant L.G., 24 years old, was admitted to the maternity hospital at the Hospital of the Society of Kremenchug Physicians, with complaints of pulling pains, as well as minor bloody expirations for 2 weeks. L. G. married 21/2 years, pregnant for the 3rd time: the 1st pregnancy ended in miscarriage in the 3rd month, 2nd urgent delivery 1 year and 3 months ago. The last regulations were in early May; L. G. felt fetal movement 5 days before admission to the clinic. Throughout her last pregnancy, L. G. suffered from time to time puffy pains and leucorrhoea, the latter, however, even before pregnancy. A pregnant woman is of good build, nutrition is not entirely satisfactory, thin, anemic, when listening to the heart, a noise is heard instead of the first tone. The abdomen is soft, the uterus is hard, sensitive to pressure, the bottom of it is 2 transverse fingers below the navel; the neck is soft, passes a finger; a little blood is shown from the genital tract.

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Rojek, M., M. Rajzer, W. Wojciechowska, P. Skalski, T. Pizon, and D. Czarnecka. "P2542Nighttime aircraft noise effect on blood pressure profile." European Heart Journal 39, suppl_1 (August 1, 2018). http://dx.doi.org/10.1093/eurheartj/ehy565.p2542.

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Rojek, Marta, Wiktoria Wojciechowska, Andrzej Januszewicz, Danuta Czarnecka, Paweł Skalski, and Marek Rajzer. "The relation of nocturnal exposure to aircraft noise and aircraft noise-induced insomnia with blood pressure." Polish Archives of Internal Medicine, December 14, 2020. http://dx.doi.org/10.20452/pamw.15716.

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Ba̧czalska, Justyna, Wiktoria Wojciechowska, Marta Rojek, Omar Hahad, Andreas Daiber, Thomas Münzel, and Marek Rajzer. "Cardiovascular consequences of aircraft noise exposure." Frontiers in Public Health 10 (December 2, 2022). http://dx.doi.org/10.3389/fpubh.2022.1058423.

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The results from epidemiological studies suggest that environmental noise including aircraft, railway, road traffic, wind turbine, and leisure-related noise is a growing public health concern. According to the WHO, at least 100 million people in the European Union are affected by traffic noise levels above the WHO-recommended thresholds. Environmental noise can adversely affect physical and mental health, as well as wellbeing. Chronic low-level noise exposure typical for most environmental sources is associated with psychophysiological stress causing non-auditory or indirect noise effects leading ultimately to cardiovascular diseases. Among all environmental noise sources, aircraft noise is considered the most annoying, and its leading mechanism of action is autonomic system activation such as increases in heart rate and blood pressure. Previously, we observed that long-term exposure to aircraft noise was associated with increased diastolic blood pressure, arterial stiffness (as assessed by pulse wave velocity), and impaired left ventricular diastolic function. All mentioned above effects are early, subclinical, and potentially reversible changes which preceded late noise effects in the cardiovascular system, that is, established cardiovascular diseases such as myocardial infarction, stroke, and heart failure. However, even a short-term reduction in aircraft noise exposure as observed during the COVID-19 lockdown may reverse these negative effects on arterial stiffness and blood pressure and may decrease the prevalence of insomnia. In this review, we aimed to critically discuss our obtained results considering recent studies on the influence of aircraft noise (and other traffic noises) on cardiovascular diseases in the context of the WHO Environmental Noise Guidelines for the European Region.

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Torjesen, Ingrid. "Long term aircraft noise is linked to incidence of high blood pressure." BMJ, June 13, 2017, j2872. http://dx.doi.org/10.1136/bmj.j2872.

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Khorshidi Behzadi, Yaser, Mohammad Ranjbarian, and Soheila Khodakarim. "Aircraft Noise and its Effects on Blood Pressure and Heart Rate at Mehrabad Airport Employees." Archives of Occupational Health, January 30, 2021. http://dx.doi.org/10.18502/aoh.v5i1.5266.

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Background: Sound is an inevitable part of everyday life these days. Hypertension is the most significant preventible risk of immature death all around the world. The main purpose of this study is to answer this question: Does aircraft noise affect blood pressure and heart rate? Methods: To achieve this objective, 100 personnel from the airport were selected. Additionally, 100 personnel were selected for the control group. In this case-control study the B&K dosimeter model 4444 was used to record noise in a long time duration (8 hours) Blood pressure and heart rates were measured before and after their shift and during their work shift by connecting a dosimeter device to their body and recording noise exposure. SPSS V.22 software was used for statistical analysis. Results: Analyses showed that the average noise intensity in the experimental group was 87.84 ± 2.76, and in the control group, it was 70.01 ± 4.01's. Compared with the control group, mean differences between systolic (P <0/001) and diastolic blood pressure (P=0.047) was significant, while the mean differences in heart rate after the shift not have a significant (P> 0.05). Conclusions: research indicates that unacceptable noise could be a hazardous agent for hypertension. It is suggested that monitoring blood pressure, training workers, and periodic examination to be practiced to decrease blood pressure

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Frenis, Katie, Sanela Kalinovic, Benjamin P. Ernst, Miroslava Kvandova, Ahmad Al Zuabi, Marin Kuntic, Matthias Oelze, et al. "Long-Term Effects of Aircraft Noise Exposure on Vascular Oxidative Stress, Endothelial Function and Blood Pressure: No Evidence for Adaptation or Tolerance Development." Frontiers in Molecular Biosciences 8 (January 31, 2022). http://dx.doi.org/10.3389/fmolb.2021.814921.

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Transportation noise is recognized as an important cardiovascular risk factor. Key mechanisms are noise-triggered vascular inflammation and oxidative stress with subsequent endothelial dysfunction. Here, we test for adaptation or tolerance mechanisms in mice in response to chronic noise exposure. C57BL/6J mice were exposed to aircraft noise for 0, 4, 7, 14 and 28d at a mean sound pressure level of 72 dB(A) and peak levels of 85 dB(A). Chronic aircraft noise exposure up to 28d caused persistent endothelial dysfunction and elevation of blood pressure. Likewise, reactive oxygen species (ROS) formation as determined by dihydroethidium (DHE) staining and HPLC-based measurement of superoxide formation in the aorta/heart/brain was time-dependently increased by noise. Oxidative burst in the whole blood showed a maximum at 4d or 7d of noise exposure. Increased superoxide formation in the brain was mirrored by a downregulation of neuronal nitric oxide synthase (Nos3) and transcription factor Foxo3 genes, whereas Vcam1 mRNA, a marker for inflammation was upregulated in all noise exposure groups. Induction of a pronounced hearing loss in the mice was excluded by auditory brainstem response audiometry. Endothelial dysfunction and inflammation were present during the entire 28d of aircraft noise exposure. ROS formation gradually increases with ongoing exposure without significant adaptation or tolerance in mice in response to chronic noise stress at moderate levels. These data further illustrate health side effects of long-term noise exposure and further strengthen a consequent implementation of the WHO noise guidelines in order to prevent the development of noise-related future cardiovascular disease.

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Steven, S., K. Frenis, S. Kroeller-Schoen, S. Kalinovic, J. Helmstaedter, M. Kvandova, M. Oelze, A. Daiber, and T. Munzel. "P4478Noise pollution exacerbates the development of arterial hypertension via additive oxidative stress and impairment of NO signaling." European Heart Journal 40, Supplement_1 (October 1, 2019). http://dx.doi.org/10.1093/eurheartj/ehz745.0873.

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Abstract Background Environmental noise pollution has been identified as a cardiovascular risk and is characterized by moderate hypertension, endothelial dysfunction, increased oxidative stress, and inflammation. We have gained insights into the mechanism by which these consequences occur by exposing mice lacking the critical NADPH oxidase subunit gp91phox to aircraft noise. Mice were protected from the effects of aircraft noise exposure. NADPH oxidase is believed to be the mediator by which angiotensin II increases oxidative stress, making investigation into the additive effect of noise and hypertension an important subject in modern cardiovascular health research. Methods and results C57Bl/6J mice were implanted with subcutaneous osmotic mini-pumps, delivering a moderate dose of 0.5mg/kg/d of angiotensin II for 7 days. Immediately following the implantation, half the mice were exposed to aircraft noise for 7 days at a maximum sound pressure level of 85 dB(A) and a mean sound pressure level of 72 dB(A), a level at which hearing loss does not occur*. Non-invasive blood pressure measurements revealed an additive increase in blood pressure in noise-exposed hypertensive mice. Following sacrifice, endothelial dysfunction was evaluated through isometric tension recordings of 3mm aortic ring segments. These recordings support the blood pressure measurements and indicate a more serious impairment in acetylcholine-induced vasorelaxation in hypertensive mice exposed to noise than the hypertensive or noise only controls. Whole blood stimulated with phorbol 12,13-dibutyrate (PDBu) or zymosan A showed an additive increase in oxidative burst in in noise-exposed hypertensive mice. Dihydroethidium (DHE) staining was used to assess the presence of vascular and cerebral oxidative stress, showing similar additive effects in mice with hypertension plus noise exposure. High performance liquid chromatography (HPLC) measurement of 2-hydroxyethidium further confirmed additive increase of oxidative stress in the aorta and brain. Western blot analysis of aortic tissue revealed highest levels of gp91phox in mice with hypertension plus noise exposure and indicated a decrease in the ratio of P-eNOSSer1177:eNOS as well as a decrease in the ratio of eNOS dimer:monomer, exposing eNOS uncoupling as a potential pathomechanism for endothelial dysfunction and gp91phox as a source for the oxidative stress.Ongoing immunohistochemical and flow cytometric investigations will characterize the role of immune cells in these adverse effects. Conclusion Herein, we present novel data demonstrating additive noise-induced cardiovascular consequences on developing hypertension. Noise has previously been established as a cardiovascular risk factor, but the effects have not been determined in pre-existing or developing cardiovascular disease. Our results show a cumulative effect between noise exposure and hypertension and forge an important link between environmental stressors and cardiovascular health. Acknowledgement/Funding Boehringer Ingelheim Foundation

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Barbara, Ohlenforst, Ligthart Seth, Aalmoes Roalt, Dirk Schreckenberg, and Maykel van Miltenburg. "Association Between Short-Term Annoyance and Several Physiological Parameters During Different Amounts of Nocturnal Aircraft Noise Exposure." Transactions on Aerospace Research, December 12, 2020. http://dx.doi.org/10.2478/tar-2020-0018.

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AbstractAnnoyance is the most prevalent community response to environmental noise. Observational and experimental lab studies have shown that exposure to environmental noise leads to annoyance, sleep disturbance, daytime sleepiness, increased heart rate and increased blood pressure. However, previous literature is preliminary based on controlled settings or experimental design, raising the question of the generalizability and applicability in daily life scenarios. This study aimed to investigate two main research questions. First, what is the relationship between short-term annoyance and different amounts of nocturnal aircraft noise exposure in daily life? Second, what is the relationship between physiological parameters, including heart rate, number of awakenings, sleep efficiency, sleep duration and different amounts of nocturnal aircraft noise exposure in daily life? This study also aimed to explore the suitability of non-invasive commercially available activity trackers to measure physiological metrics in a scientific way. During this field study, participants were wearing Fitbit Charge 3 activity trackers recording heart rate and different sleep-derived metrics (e.g. deep sleep duration, sleep efficiency and awakenings). The used activity trackers were readily available, non-intrusive, relatively cheap and easy to use by the participants. Simultaneously, a logbook was used by the participants to track the subjective perception and situational context of air traffic noise exposure. The noise levels corresponding to the exposure of air traffic of each participant were calculated based on the location of the participant and the corresponding radar track using an aircraft noise monitoring system.We hypothesize that a higher amount of exposure to aircraft noise in real life will be associated with increased annoyance, increased rest heartrate, higher number of awakenings, decreased sleep efficiency and decreased deep sleep duration.Preliminary results on the interactions between aircraft noise exposure, perceived annoyance and physiological metrics suggest increased nocturnal aircraft noise exposure seems to negatively affect sleep efficiency and deep sleep duration.

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Frenis, Katie, Johanna Helmstädter, Yue Ruan, Eva Schramm, Sanela Kalinovic, Swenja Kröller-Schön, Maria Teresa Bayo Jimenez, et al. "Ablation of lysozyme M-positive cells prevents aircraft noise-induced vascular damage without improving cerebral side effects." Basic Research in Cardiology 116, no. 1 (April 30, 2021). http://dx.doi.org/10.1007/s00395-021-00869-5.

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AbstractAircraft noise induces vascular and cerebral inflammation and oxidative stress causing hypertension and cardiovascular/cerebral dysfunction. With the present studies, we sought to determine the role of myeloid cells in the vascular vs. cerebral consequences of exposure to aircraft noise. Toxin-mediated ablation of lysozyme M+ (LysM+) myeloid cells was performed in LysMCreiDTR mice carrying a cre-inducible diphtheria toxin receptor. In the last 4d of toxin treatment, the animals were exposed to noise at maximum and mean sound pressure levels of 85 and 72 dB(A), respectively. Flow cytometry analysis revealed accumulation of CD45+, CD11b+, F4/80+, and Ly6G−Ly6C+ cells in the aortas of noise-exposed mice, which was prevented by LysM+ cell ablation in the periphery, whereas brain infiltrates were even exacerbated upon ablation. Aircraft noise-induced increases in blood pressure and endothelial dysfunction of the aorta and retinal/mesenteric arterioles were almost completely normalized by ablation. Correspondingly, reactive oxygen species in the aorta, heart, and retinal/mesenteric vessels were attenuated in ablated noise-exposed mice, while microglial activation and abundance in the brain was greatly increased. Expression of phagocytic NADPH oxidase (NOX-2) and vascular cell adhesion molecule-1 (VCAM-1) mRNA in the aorta was reduced, while NFκB signaling appeared to be activated in the brain upon ablation. In sum, we show dissociation of cerebral and peripheral inflammatory reactions in response to aircraft noise after LysM+ cell ablation, wherein peripheral myeloid inflammatory cells represent a dominant part of the pathomechanism for noise stress-induced cardiovascular effects and their central nervous counterparts, microglia, as key mediators in stress responses.

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"The Okinawa study: effects of chronic aircraft noise on blood pressure and some other physiological indices." Journal of Sound and Vibration 277, no. 3 (October 2004): 469–70. http://dx.doi.org/10.1016/j.jsv.2004.03.007.

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Siagian, Minarma, Bastaman Basuki, and Dede Kusmana. "High intensity interior aircraft noise increases the risk of high diastolic blood pressure in Indonesian Air Force pilots." Medical Journal of Indonesia, November 1, 2009, 276. http://dx.doi.org/10.13181/mji.v18i4.375.

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Akdeniz, Halil Yalcin, Mehmet Ziya Sogut, and Onder Turan. "Prediction and simulation of aircraft noise in the international Eskisehir Hasan Polatkan airport (LTBY)." Aircraft Engineering and Aerospace Technology ahead-of-print, ahead-of-print (December 4, 2020). http://dx.doi.org/10.1108/aeat-03-2020-0056.

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Purpose In recent years, in parallel with the increasing air traffic and the number of passengers in air transport, the number of people exposed to aircraft-induced noise has increased significantly. Especially people living in the areas close to the airports are affected by noise emission during the landing, take-off, taxi and ground operations. Negative effects of noise such as sleep disturbance, lack of concentration, anxiety and high blood pressure cardiac diseases were determined directly or indirectly for human health. For this reason, examining the noise effect caused by aircraft and determining the necessary measures to be taken is very important for the sustainable development of aviation. In the International Eskisehir Hasan Polatkan Airport (LTBY), this paper aims to calculate a noise mapping following international standards in line with the directives of the International Civil Aviation Organization (ICAO). Also, Annex 8, “Airworthiness of Aircrafts” and Annex 16, “Environmental Protection Volume 1 Aircraft Noise”, which were taken at the International Civil Aviation Convention, were proposed to determine the exposure caused by aircraft noise. Design/methodology/approach In this paper, noise levels for the day (07.00–19.00), evening (19.00–23.00) and night (23.00–07.00) period around LTBY were predicted and calculated by the use of the IMMI software according to the “ECAC Doc. 29-Interim” method for the prediction and computation of the aircraft noise. Findings According to the calculated/mapped values, in the 24 hours (Lden), the noise level is 65 dB (A) and above. In the day time zone, the noise level is 63 dB (A) and above. When the calculations for the evening time zone are examined, the noise level is above 58 dB (A). When the calculations for the night time frame are examined, it is calculated that there is no dwelling that is affected by the noise level above 53 dB (A). Practical implications Along with future improvements, it is recommended to be applied to other civil airports. Originality/value To the best of the authors’ knowledge, there is no previous research in the literature on aircraft noise mapping of LTBY. Also, unlike the software commonly used in other works in the literature, IMMI software was used in this study. Such investigations should be carried out in other civil airports in the coming years to struggle with noise emissions and noise control. If noise boundary values are exceeded, action plans should be developed for a sustainable aviation concept. Along with future improvements, it is recommended to be applied to other civil airports.

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Molitor, M., M. T. Bayo Jimenez, O. Hahad, C. Witzler, S. Finger, V. S. Garlapati, S. Rajlic, et al. "Aircraft noise exposure induces pro-inflammatory vascular conditioning and amplifies vascular dysfunction and impairment of cardiac function after myocardial infarction." Cardiovascular Research, January 26, 2023. http://dx.doi.org/10.1093/cvr/cvad021.

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Abstract Aims Traffic noise may play an important role in the development and deterioration of ischemic heart disease. Thus, we sought to determine the mechanisms of cardiovascular dysfunction and inflammation induced by aircraft noise in a mouse model of myocardial infarction (MI) and in humans with incident MI. Methods and Results C57BL/6J mice were exposed to noise alone (average sound pressure level 72 dB; peak level 85 dB) up to 4d, resulting in pro-inflammatory aortic gene expression in the myeloid cell adhesion/diapedesis pathways. Noise alone promoted adhesion and infiltration of inflammatory myeloid cells in vascular/cardiac tissue, paralleled by an increased percentage of leukocytes with a pro-inflammatory, reactive oxygen species (ROS)-producing phenotype and augmented expression of Nox-2/phospho-NFκB in peripheral blood. Ligation of the LAD resulted in worsening of cardiac function, pronounced cardiac infiltration of CD11b+ myeloid cells and Ly6Chigh monocytes and induction of interleukin (IL) 6, IL-1β, CCL-2 and Nox-2, being aggravated by noise exposure prior to MI. MI induced stronger endothelial dysfunction and more pronounced increases in vascular ROS in animals preconditioned with noise. Participants of the population-based Gutenberg Health Cohort Study (median follow-up:11.4y) with incident MI revealed elevated CRP at baseline and worse LVEF after MI in case of a history of noise exposure and subsequent annoyance development. Conclusion Aircraft noise exposure before MI substantially amplifies subsequent cardiovascular inflammation and aggravates ischemic heart failure, facilitated by a pro-inflammatory vascular conditioning. Our translational results suggest, that measures to reduce environmental noise exposure will be helpful in improving clinical outcome of subjects with MI.

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Liang, Xiaohua, Xian Tang, Mingliang Liu, Xiaoyue Liang, Li Chen, Xia Chen, Lei Zuo, Yanling Ren, and Guang Hao. "Associations of self-reported residential noise exposure with obesity and hypertension in children and adolescents." Frontiers in Pediatrics 10 (August 12, 2022). http://dx.doi.org/10.3389/fped.2022.902868.

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BackgroundEpidemiologic evidence linking environmental noise to obesity and hypertension remains scarce, especially in children, and the results remain inconclusive. This study aims to examine the cross-sectional associations of self-reported residential noise exposure with obesity and hypertension in children and adolescents.MethodsAs an ongoing study, a representative sample of the children aged 6–9 years in Chongqing were selected in 2014. In 2019, self-reported residential noise (answer categories: “very quiet,” “moderately quiet,” “slightly quiet,” and “not at all quiet”) data were collected, and 3,412 participants with completed data were included in the analyses.ResultsParticipants living in a quieter area had a significantly lower risk of obesity than those living in a noisy area (very quiet: OR = 0.50, 95%CI: 0.29–0.88, P = 0.015; moderately quiet: OR = 0.61, 95%CI: 0.36–1.02, P = 0.059). Similar associations were observed for abdominal obesity, although did not reach statistical significance. Consistently, residential noise exposure was significantly associated with body mass index (BMI) and waist-to-height ratio. Self-reported residential noise exposure was positively associated with systolic blood pressure (β = −1.808; 95%CI = −3.495, −0.110; P = 0.037). When sleep quality, study stress, BMI, and vegetable/fruits consumption were further adjusted, all effect estimates decreased, and no statistical association was observed between noise exposure and blood pressure. Furthermore, we found that the mediating effects of obesity on the associations of self-reported residential noise exposure with hypertension were 6.8% (% of total effect mediated = 0.068, 95%CI: −2.58, 3.99), although did not reach statistical significance.ConclusionsSelf-reported residential noise exposure was associated with a higher risk of obesity or abdominal obesity. Also, self-reported residential noise exposure was positively associated with hypertension, and obesity may partially mediate this association, but did not reach statistical significance.

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Nieden*, Anja zur, Doreen Ziedorn, Karin Römer, Jan Spilski, Ulrich Möhler, Susanne Harpel, Dirk Schreckenberg, and Thomas Eikmann. "Chronic exposure to aircraft noise and self-measured blood pressure – field study (adults) in the vicinity of a large airport in Germany." ISEE Conference Abstracts 2016, no. 1 (August 17, 2016). http://dx.doi.org/10.1289/isee.2016.3129.

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Roux, Stephane G., Nicolas B. Garnier, Patrice Abry, Nathan Gold, and Martin G. Frasch. "Distance to Healthy Metabolic and Cardiovascular Dynamics From Fetal Heart Rate Scale-Dependent Features in Pregnant Sheep Model of Human Labor Predicts the Evolution of Acidemia and Cardiovascular Decompensation." Frontiers in Pediatrics 9 (August 3, 2021). http://dx.doi.org/10.3389/fped.2021.660476.

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The overarching goal of the present work is to contribute to the understanding of the relations between fetal heart rate (FHR) temporal dynamics and the well-being of the fetus, notably in terms of predicting the evolution of lactate, pH and cardiovascular decompensation (CVD). It makes uses of an established animal model of human labor, where 14 near-term ovine fetuses subjected to umbilical cord occlusions (UCO) were instrumented to permit regular intermittent measurements of metabolites lactate and base excess, pH, and continuous recording of electrocardiogram (ECG) and systemic arterial blood pressure (to identify CVD) during UCO. ECG-derived FHR was digitized at the sampling rate of 1,000 Hz and resampled to 4 Hz, as used in clinical routine. We focused on four FHR variability features which are tunable to temporal scales of FHR dynamics, robustly computable from FHR sampled at 4 Hz and within short-time sliding windows, hence permitting a time-dependent, or local, analysis of FHR which helps dealing with signal noise. Results show the sensitivity of the proposed features for early detection of CVD, correlation to metabolites and pH, useful for early acidosis detection and the importance of coarse time scales (2.5–8 s) which are not disturbed by the low FHR sampling rate. Further, we introduce the performance of an individualized self-referencing metric of the distance to healthy state, based on a combination of the four features. We demonstrate that this novel metric, applied to clinically available FHR temporal dynamics alone, accurately predicts the time occurrence of CVD which heralds a clinically significant degradation of the fetal health reserve to tolerate the trial of labor.

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Journal articles on the topic 'Blood pressure;aircraft noise;child'

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