Dissertations / Theses on the topic 'Blood endothelial cells'
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Al-Malki, Aysha Ibrahim. "Detection of endothelial cells in whole blood donations." Thesis, University of Sheffield, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531130.
Full textYuan, Yifan. "Enhancing Blood Outgrowth Endothelial Cells for Optimal Coating of Blood Contacting Surfaces." Thesis, Université d'Ottawa / University of Ottawa, 2017. http://hdl.handle.net/10393/36837.
Full textLester, Elizabeth Ann. "Consequences of biomaterial activation of blood cells on endothelial cell proinflammatory phenotype." Thesis, Georgia Institute of Technology, 2001. http://hdl.handle.net/1853/11869.
Full textPrasad, Raju. "Endothelial progenitor cells, vascular function, and exercise." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 59 p, 2009. http://proquest.umi.com/pqdweb?did=1654501181&sid=4&Fmt=2&clientId=8331&RQT=309&VName=PQD.
Full textZolle, Lapuente Olga C. "Cyclic GMP and calcium homeostasis in endothelial cells." Thesis, University of Liverpool, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367654.
Full textDoty, Sherry D. "Fluid shear stress effects on fibronectin in endothelial cells." Thesis, Georgia Institute of Technology, 1989. http://hdl.handle.net/1853/19073.
Full textTretiach, Marina Louise. "Bovine Models of Human Retinal Disease: Effect of Perivascular Cells on Retinal Endothelial Cell Permeability." University of Sydney, 2005. http://hdl.handle.net/2123/1153.
Full textBackground: Diabetic vascular complications affect both the macro- and microvasculature. Microvascular pathology in diabetes may be mediated by biochemical factors that precipitate cellular changes at both the gene and protein levels. In the diabetic retina, vascular pathology is found mainly in microvessels, including the retinal precapillary arterioles, capillaries and venules. Macular oedema secondary to breakdown of the inner blood-retinal barrier is the most common cause of vision impairment in diabetic retinopathy. Müller cells play a critical role in the trophic support of retinal neurons and blood vessels. In chronic diabetes, Müller cells are increasingly unable to maintain their supportive functions and may themselves undergo changes that exacerbate the retinal pathology. The consequences of early diabetic changes in retinal cells are primarily considered in this thesis. Aims: This thesis aims to investigate the effect of perivascular cells (Müller cells, RPE, pericytes) on retinal endothelial cell permeability using an established in vitro model. Methods: Immunohistochemistry, cell morphology and cell growth patterns were used to characterise primary bovine retinal cells (Müller cells, RPE, pericytes and endothelial cells). An in vitro model of the blood-retinal barrier was refined by coculturing retinal endothelial cells with perivascular cells (Müller cells or pericytes) on opposite sides of a permeable Transwell filter. The integrity of the barrier formed by endothelial cells was assessed by transendothelial electrical resistance (TEER) measurements. Functional characteristics of endothelial cells were compared with ultrastructural morphology to determine if different cell types have barrier-enhancing effects on endothelial cell cultures. Once the co-culture model was established, retinal endothelial cells and Müller cells were exposed to different environmental conditions (20% oxygen, normoxia; 1% oxygen, hypoxia) to examine the effect of perivascular cells on endothelial cell permeability under reduced oxygen conditions. Barrier integrity was assessed by TEER measurements and permeability was measured by passive diffusion of radiolabelled tracers from the luminal to the abluminal side of the endothelial cell barrier. A further study investigated the mechanism of laser therapy on re-establishment of retinal endothelial cell barrier integrity. Müller cells and RPE, that comprise the scar formed after laser photocoagulation, and control cells (Müller cells and pericytes, RPE cells and ECV304, an epithelial cell line) were grown in long-term culture and treated with blue-green argon laser. Lasered cells were placed underneath confluent retinal endothelial cells growing on a permeable filter, providing conditioned medium to the basal surface of endothelial cells. The effect of conditioned medium on endothelial cell permeability was determined, as above. Results: Co-cultures of retinal endothelial cells and Müller cells on opposite sides of a permeable filter showed that Müller cells can enhance the integrity of the endothelial cell barrier, most likely through soluble factors. Low basal resistances generated by endothelial cells from different retinal isolations may be the result of erratic growth characteristics (determined by ultrastructural studies) or the selection of vessel fragments without true âbarrier characteristicsâ in the isolation step. When Müller cells were co-cultured in close apposition to endothelial cells under normoxic conditions, the barrier integrity was enhanced and permeability was reduced. Under hypoxic conditions, Müller cells had a detrimental effect on the integrity of the endothelial cell barrier and permeability was increased in closely apposed cells. Conditioned medium from long-term cultured Müller cells and RPE that typically comprise the scar formed after lasering, enhanced TEER and reduced permeability of cultured endothelial cells. Conclusions: These studies confirm that bovine tissues can be used as a suitable model to investigate the role of perivascular cells on the permeability of retinal endothelial cells. The dual effect of Müller cells on the retinal endothelial cell barrier under different environmental conditions, underscores the critical role of Müller cells in regulating the blood-retinal barrier in health and disease. These studies also raise the possibility that soluble factor(s) secreted by Müller cells and RPE subsequent to laser treatment reduce the permeability of retinal vascular endothelium. Future studies to identify these factor(s) may have implications for the clinical treatment of macular oedema secondary to diseases including diabetic retinopathy.
Ahmed, Syed Rumel. "Characterising lymphocyte trafficking across blood vascular and lymphatic endothelial cells." Thesis, University of Birmingham, 2012. http://etheses.bham.ac.uk//id/eprint/3846/.
Full textHelmlinger, Gabriel. "Effects of pulsatile laminar shear stress on cultured vascular endothelial cells." Thesis, Georgia Institute of Technology, 1990. http://hdl.handle.net/1853/16738.
Full textSeetharaman, Seeta Lakshmy. "Multidrug transporter expression in endothelial cells of the blood-brain barrier." Thesis, University of Cambridge, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621690.
Full textJoseph, Laurie B. "Comparison of stressed human endothelial cells derived from different vascular beds /." The Ohio State University, 1986. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487266691093865.
Full textJiang, Liying. "Exposure of endothelial cells to shear stress stimulates protein tryosine phosphorylation." Thesis, Georgia Institute of Technology, 1996. http://hdl.handle.net/1853/25421.
Full textAfzal-Ahmed, Iram. "Redox regulation in circulating blood cells and foetal endothelial cells in normotensive and pre-eclamptic pregnancies." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.497287.
Full textMagid, Richard. "Engineering molecular reporters to investigate the effects of shear stress upon endothelial cells." Thesis, Georgia Institute of Technology, 2000. http://hdl.handle.net/1853/13754.
Full textEnsley, Ann Elizabeth. "Functional evaluation of circulating endothelial progenitor cells for vascular tissue engineering." Diss., Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-04042006-145611/.
Full textVito, Raymond, Committee Member ; Nerem, Robert, Committee Chair ; Eskin, Suzanne, Committee Member ; Hanson, Stephen, Committee Member ; Gibbons, Gary, Committee Member.
Tan, Kevin S. "Role of Circulating Peripheral Blood-Derived Endothelial Colony-Forming Cells in Patients with Proliferative Diabetic Retinopathy." Case Western Reserve University School of Graduate Studies / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=case1238452739.
Full textMitchell, Andrew Joseph. "Understanding the role of endothelial progenitor cells in vascular injury and repair." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/33310.
Full textRobinson, Scott Thomas. "Determining the role of endothelial progenitor cells in post-natal neovascularization." Diss., Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/37178.
Full textLafleur, Marc Andre. "The involvement of matrix metalloproteinases in angiogenesis." Thesis, University of East Anglia, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.327541.
Full textKrenning, Guido. "Endothelial progenitor cells in vascular regenerative medicine towards 'designer blood vessels' and 'therapeutic neovascularization' /." [S.l. : [Groningen : s.n.] ; University of Groningen] [Host], 2009. http://irs.ub.rug.nl/ppn/.
Full textHelmlinger, Gabriel. "Effect of pulsatile flow on the intracellular free calcium concentration of cultured vascular endothelial cells." Diss., Georgia Institute of Technology, 1994. http://hdl.handle.net/1853/16707.
Full textRajani, Rikesh Mukesh. "Is small vessel disease a disease of the blood brain barrier?" Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25866.
Full textJenkins, Meredith E. "An examination of the human fibrinogen-like protein2 sequence variations and genetic expression by human endothelial cells /." unrestricted, 2005. http://etd.gsu.edu/theses/available/etd-07222005-093438/.
Full textTitle from title screen. Roberta Attanasio, committee chair; P.C. Tai, W.C. Hooper, committee members. Electronic text (57 p. : col. ill.) : digital, PDF file. Description based on contents viewed Aug. 15, 2007. Includes bibliographical references (p. 55-57).
Thomas, Kelly Angelique. "The effects of Ethanol and Aspalathus linearis on immortalized mouse brain endothelial cells (bEnd5)." Thesis, University of the Western Cape, 2015. http://hdl.handle.net/11394/4959.
Full textThe blood brain barrier (BBB) is a signaling interface between the blood and the central nervous system (CNS), which prohibits the entry of harmful blood-borne substances into the brain micro-environment, thus maintaining brain homeostasis. The crucial role of the BBB is protecting the CNS, which may adversely be affected by alcohol. The central component of the BBB, endothelial cells (ECs), regulates BBB transport by regulating the permeability both transcellularly and through their paracellular junctions, by structures called tight junctions (TJs) that are composed of proteins. The aim of this study was to investigate the in vitro effects of ethanol (EtOH) and fermented rooibos (Rf) on a monolayer of bEnd5 mouse brain ECs, by determining the effects of EtOH and Rf on bEnd5 (i) cell viability (ii) cell proliferation (iii) rate of cell division (iv) cell toxicity (v) claudin-5 transcription (vi) permeability across a monolayer of bEnd5 ECs and (vii) morphology, for a selected experimental timeline of 24, 48, 72, and 96hrs. We then investigated if the simultaneous exposure of Rf and EtOH could reverse or alleviate the EtOHinduced effects on the bEnd5 ECs. EtOH metabolism induces oxidative stress and results in a range of adverse physiological effects. Aspathalus linearis (rooibos) contains many phenolic compounds, of which the main antioxidant activity is attributed to aspalathin. Our underlining hypothesis is that the antioxidants in an aqueous rooibos extract may therefore protect against the potential oxidant damaging effects of alcohol on the BBB. Cells were exposed for 24hrs to selected concentrations of EtOH (25mM and 100mM), a concentration of Rf containing equivalent of 1.9nM aspalathin, and the combinations of EtOH and Rf. Cell viability and cell toxicity was determined, while cell proliferation and rate of cell division was estimated using the trypan blue exclusion assay. Real time quantitative PCR was implemented to quantify claudin-5 transcription, normalized against housekeeping genes, GAPDH and HPRT. Transepithelial electrical resistance (TEER) was measured using the Ohm Millicell-electrical resistance system, while bEnd5 monolayer morphology was analysed using the Zeiss scanning electron microscope. Both concentrations of EtOH led to an overall decrease in cell viability, and a decreased number of live cells across 72hrs. Consistent with this, EtOH resulted in increased cell toxicity across the 96hr experimental timeframe and a diminished rate of cell division. The transcription of claudin-5 in bEnd5 ECs exposed to 25mM and 100mM EtOH varied dramatically across the 96hr timeframe. While 25mM EtOH resulted in an overall decrease in TEER, cells exposed to 100mM EtOH only decreased TEER between 48 and 96hrs. Morphologically, both concentrations of EtOH led to compromised paracellular spaces as endorsed by high definition SEM analysis. The administration of Rf on its own resulted in an initial decrease in viability, followed by recovery between 72 and 96hrs. Exposure to Rf diminished live cell numbers at 72 and 96hrs, accompanied by a compromised rate of cell division and an overall increase in cell toxicity. In addition, Rf down-regulated claudin-5 transcription across the course of the experiment, particularly between 24 and 48hrs. In alignment with this, Rf also led to an increase in BBB permeability from 24 to 96hrs. However, SEM studies were not able to discriminate any differences between control and Rf treated cells. Our study showed that the BBB could be protected against the adverse effects of EtOH, and this at the plasma concentration induced by 500ml’s of Rooibos tea. The simultaneous exposure of Rf and EtOH was able to negate the effects of EtOH on cell viability, cell proliferation, and cell toxicity but exacerbated the effects of EtOH on claudin-5 transcription and paracellular permeability. Morphologically, co-exposure with Rf only reversed the effects of 25mM EtOH while exacerbating the effects of 100mM EtOH at 96hrs. In conclusion, EtOH was shown to be detrimental to the integrity of bEnd5 ECs, and the addition of a minuscule quantity of t h e Rf extract was able to partially alleviate excess ROS-induced effects.
National Research Foundation (NRF)
Plant, Stuart D. "The response of human umbilical vein endothelial cells and blood platelets to modified NiTi surfaces." Thesis, University of Nottingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275630.
Full textCardinal, Kristen O'Halloran. "Development and Utilization of a Tissue Engineered Blood Vessel Mimic to Assess the Neointimal Response to Intravascular Stents." Diss., The University of Arizona, 2007. http://hdl.handle.net/10150/195387.
Full textZiegler, Thierry. "Co-culture of endothelial cells with smooth muscle cells in a matrix of collagen : Effect of a steady, laminar stress on the cell behavior." Diss., Georgia Institute of Technology, 1994. http://hdl.handle.net/1853/17618.
Full textArief, Melissa Suen. "Human Tissue Engineered Small Diameter Blood Vessels." Yale University, 2010. http://ymtdl.med.yale.edu/theses/available/etd-03152010-144428/.
Full textBraddon, Linda Greer. "The effect of shear stress on a co-culture of endothelial cells and fibroblasts in a biodegradable polymer scaffold." Diss., Georgia Institute of Technology, 1997. http://hdl.handle.net/1853/17850.
Full textSuidan, Georgette Leila. "CD8 T cells mediate CNS vascular permeability under neuroinflammatory conditions." University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1242912012.
Full textJohnson, Tiffany Lynn. "Endothelial Cell Function Using a Tissue Engineered Blood Vessel Model: A Case Study of Cell-Cell Communication." Diss., Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-04032006-120602/.
Full textPollman, Matthew, Committee Member ; Galis, Zorina, Committee Member ; McIntire, Larry, Committee Member ; Taylor, W Robert, Committee Member ; Jo, Hanjoong, Committee Member ; Nerem, Robert, Committee Chair.
Okyere, Benjamin. "Eph-mediated restriction of cerebrovascular arteriogenesis." Diss., Virginia Tech, 2019. http://hdl.handle.net/10919/89222.
Full textDoctor of Philosophy
Stroke is the fifth leading cause of death in the United States. Ischemic stroke is the most common type of stroke and occurs when blood flow to part of the brain is impeded. Lack of blood results in cell death and tissue damage in the brain. In an effort to restore blood flow, specialized blood vessels in the brain called collaterals remodel and become larger to allow re-routed blood to the blood-deprived region of the brain. The duration it takes to remodel these remarkable blood vessels and re-route blood varies in humans, and sometimes is not able to prevent adequate tissue damage. The current work explores novel therapeutic targets to accelerate collateral remodeling in an effort to reduce tissue loss after stroke. We present studies which show that a protein called EphA4, found on endothelial cells restricts remodeling, and when inhibited in the brain can increase collateral remodeling and reduced adverse effects after ischemic stroke.
Lacroix-Desmazes, Sebastien. "Influence of flow environment on the production and secretion of metalloproteinases and urokinase-type plasminogen activator by cultured bovine aortic endothelial cells." Thesis, Georgia Institute of Technology, 1993. http://hdl.handle.net/1853/15826.
Full textSun, Bing. "Characterization of CRGRP and 5-HT receptors in vascular tissues, and expression of CGRP in cultured human endothelial cells." Thesis, University of Cambridge, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260599.
Full textNguyen, Hieu Thi Minh. "The effect of cardiolipin synthase deficiency on the mitochondrial function and barrier properties of human cerebral capillary endothelial cells." Elsevier, 2014. http://hdl.handle.net/1993/30184.
Full textKojima, Hidenobu. "Establishment of practical recellularized liver graft for blood perfusion using primary rat hepatocytes and liver sinusoidal endothelial cells." Kyoto University, 2018. http://hdl.handle.net/2433/233836.
Full textArzhangi, Zahra S. "Collagen biomaterials for the delivery of FVIII-producing blood outgrowth endothelial cells in the treatment of Haemophilia A." Thesis, University of Ottawa (Canada), 2011. http://hdl.handle.net/10393/28921.
Full textBiernacki, Katarzyna. "Blood-brain barrier endothelial cells as regulators of immune-mediated injury and repair in the central nervous system." Thesis, McGill University, 2004. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85129.
Full textTo examine the mechanisms that regulate the migration of immune cells that participate in CNS injury and/or repair, I have used human adult brain-derived endothelial cells (HBECs) grown in Boyden chambers. I compared the migration of Th1 lymphocytes, disease-inducing cells, and Th2 lymphocytes, cells induced by several immuno-modulatory therapies aimed at reducing neuroinflammation, across an in vitro model of the BBB. I demonstrate that Th2 cells can efficiently cross the BBB, and do so at significantly higher rates compared to Th1 lymphocytes. This selectivity is dependent on HBEC chemokine CCL2, and the selective expression of the receptor CCR2 on Th2 cells. Adhesion molecules are not implicated in selective T cell recruitment, although migration of both subsets can be blocked with anti-ICAM-1 antibody. I also explored the consequences of T cell:HBEC interactions on HBEC properties. I show that supernatants derived from Th1 polarized cells significantly up-regulate a range of pro-inflammatory chemokines and adhesion molecules in HBECs. Conversely, Th2 supernatants had no such effects and did not suppress the effects induced by Thl lymphocytes. Together, this suggests that Th2 cell-mediated bystander suppression does not occur at the level of the BBB. I also show that Th1 and Th2 cell migration can enhance subsequent passage of cells and molecules across HBEC barriers. Furthermore, I demonstrate that lymphocyte contact induced HBEC production of the neurotrophin NGF but not BDNF, an effect potentiated by IFNbeta. The secretion of NGF was specific to HBECs; neither astrocytes nor microglia could be induced to secrete this neurotrophin. I also demonstrate an inverse correlation between HBEC NGF production induced by MS patient lymphocytes and measures of clinical disability and brain atrophy suggesting that HBECs may have a neuroprotective role in MS. Collectively, my data indicate that the BBB is a dynamic structure involved in the regula
Alamu, Olufemi Akinyinka. "Differential toxicity of two murine endothelial cells to ROS duress: Understanding oxidative stress-induced blood-brain barrier dysfunction." University of the Western Cape, 2020. http://hdl.handle.net/11394/7876.
Full textThe blood-brain barrier (BBB) is a critical interface between the blood circulation and brain tissue which performs critical selection of circulating molecules that gain access to the brain tissue. Its unique ability to adjust to changes in the constituents of the blood circulation confer in the BBB a dynamic nature enabling changes in its properties to suit the homeostatic needs of the brain. Dysfunction of the BBB has been established to be pivotal to the initiation and/or maintenance of an array of neurological disorders, most of which involve the production of excess reactive oxygen species (ROS) and oxidative stress in their pathophysiology. Thus, clinical trials of exogenous antioxidant agents have been proposed and initiated, with most results being inconclusive. Extensive studies of the impact, capacity and plasticity of endogenous antioxidants in the cells that constitute the blood-brain barrier, especially the brain endothelial cells, therefore, became necessary for the rational choice, timing, and the mode of application of antioxidants in the management of oxidative stress-mediated neurological diseases.
Al-Waili, Daniah I. "In Vitro Functional Study of YES-Associated Protein (YAP) in Murine Brain Endothelial Cells under Normal and Ischemic Conditions." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1448037357.
Full textHedigan, Conor Charles. "Evaluation of Human Umbilical Vein Endothelial Cells in Blood Vessel Mimics Through Changes in Gene Expression and Caspase Activity." DigitalCommons@CalPoly, 2019. https://digitalcommons.calpoly.edu/theses/2081.
Full textChen, Lin Min. "Angiogenic activities of Drynaria fortunei-derived extract and isolated compounds on zebrafish in vivo and human umbilical vein endothelial cells in vitro." Thesis, University of Macau, 2017. http://umaclib3.umac.mo/record=b3690926.
Full textMehra, Anupriya. "NMDA receptor of the blood brain barrier : mechanism of action and interaction with tPA." Thesis, Normandie, 2017. http://www.theses.fr/2017NORMC404/document.
Full textNeuroinflammation is a common denominator of several central nervous system disorders. Inflammatory reactions are often mediated by several signaling pathways which lead to the opening of the blood brain barrier. Tissue plasminogen activator (tPA) is a serine protease induces opening of the blood brain barrier. In recent years, it has also been shown that NMDA receptors located in endothelial cells can play a crucial role in propagation of inflammatory reaction. My doctoral study focused on the finding the underlying mechanisms of action(s) by which NMDA receptor mediates tPA induced opening of the blood brain barrier. In our first study we show that endothelial NMDA receptors are potential therapeutic targets to prevent EAE mediated immune cell infiltration and inflammation. We show that NMDA receptor specific mouse monoclonal antibody Glunomab could prevent the brain spinal cord barrier from inflammatory damage. We also show that NMDA receptors are expressed in close association of tight junction proteins in cerebral endothelial cells. In our second study, we show for the first time that, neuroendothelial NMDA receptors can exhibit metabotropic mode of action during inflammation. We also highlight that these receptors are indeed GluN3A expressing non-conventional NMDA receptors. In addition, we report that tPA accelerates the opening of blood brain barrier in presence of an uncommon agonist glycine by RhoA activation dependent mechanism.My project results provide a nouvelle insight for the role of metabotropic NMDA receptors in cerebral endothelial cells. In addition it also provides more precise details of blood brain barrier opening mediated by tissue plasminogen activator
Punshon, G. "Development of an improved two-stage seeding process for a nanocomposite vascular graft using human peripheral-blood derived endothelial cells." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/1560130/.
Full textMohd, Nasir Mohd Hamzah. "Activation of endothelial cells and its potential involvement in blood-brain barrier damage in cerebral malaria : an in vitro study." Thesis, Keele University, 2015. http://eprints.keele.ac.uk/3252/.
Full textRennel, Emma. "Molecular Mechanisms in Endothelial Cell Differentiation." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-4059.
Full textChen, Miao. "Endothelial Cell-Specific Knockout of Meis1 Protects Ischemic Hindlimb Through Vascular Remodeling." Diss., Virginia Tech, 2018. http://hdl.handle.net/10919/96188.
Full textPHD
Djassemi, Navid. "Tissue Engineering A Blood Vessel Mimic While Monitoring Contamination Through Sterility Assurance Testing." DigitalCommons@CalPoly, 2012. https://digitalcommons.calpoly.edu/theses/831.
Full textLu, Lingge. "Roles of the Shb and Cbl Proteins in Signal Transduction and Blood Vessel Formation." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3491.
Full textLam, In Kei. "Anti-angiogenic activities of flavonoids from Pericarpium Citri Reticulatae on human umbilical vein endothelial cells (HUVECs) and zebrafish." Thesis, University of Macau, 2010. http://umaclib3.umac.mo/record=b2447360.
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