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Journal articles on the topic 'Blood – Diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 July 2024

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1

G, Kapatia. "Role of Peripheral Blood Film in Renal Diseases." Journal of Human Anatomy 7, no. 1 (February 20, 2023): 1–5. http://dx.doi.org/10.23880/jhua-16000173.

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Peripheral blood smear examination is a diagnostic tool used in the assessment of various clinical conditions, including kidney diseases. While it is not a primary test for diagnosing kidney diseases, it can provide valuable insights into the presence of anemia, hemolysis, platelet abnormalities, infections, and other systemic changes that can be associated with kidney disorders. It is important to note that peripheral blood smear examination is just one aspect of a comprehensive diagnostic evaluation for kidney diseases and is not a direct method for diagnosing kidney diseases. Other tests, such as blood chemistry, urinalysis, kidney function tests imaging studies (e.g., ultrasound, CT scan), are essential for a complete assessment and accurate diagnosis. In this article, we have stressed on the role of simple and cheap test- peripheral blood film in renal diseases. We have discussed various renal diseases which show different findings on peripheral blood film. All the hematologists, nephrologists and hematopathologists should be aware of these findings.

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2

SHIBATA, AKIRA. "The blood.1.Interferon and blood diseases." Nihon Naika Gakkai Zasshi 82, no. 3 (1993): 450–54. http://dx.doi.org/10.2169/naika.82.450.

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3

Carey, Francis A., and Mary N. Sheppard. "Diseases of blood vessels." Surgery (Oxford) 39, no. 5 (May 2021): 241–47. http://dx.doi.org/10.1016/j.mpsur.2021.03.005.

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4

Rossiyskiy, D. M. "Organotherapy for blood diseases." Kazan medical journal 29, no. 8-9 (January 12, 2022): 730–36. http://dx.doi.org/10.17816/kazmj89827.

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Hormones of various endocrine glands, being powerful regulators of growth, tissue nutrition and metabolism in the body and affecting both groups of the autonomic nervous system, the autonomic and sympathetic nervous system, at the same time have either an intensifying or inhibitory effect on the activity of the hematopoietic apparatus.

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5

Cluzeau, Thomas, Guillaume Robert, Alexandre Puissant, Arnaud Jacquel, Frédéric Luciano, and Patrick Auberger. "Autophagy and blood diseases." Hématologie 21, no. 2 (March 2015): 107–16. http://dx.doi.org/10.1684/hma.2015.0982.

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6

Lindop, George B. M. "Diseases of blood vessels." Surgery (Oxford) 27, no. 8 (August 2009): 313–19. http://dx.doi.org/10.1016/j.mpsur.2009.06.009.

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7

Sheppard, Mary N. "Diseases of blood vessels." Surgery (Oxford) 30, no. 8 (August 2012): 370–76. http://dx.doi.org/10.1016/j.mpsur.2012.05.017.

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8

Sheppard, Mary N. "Diseases of blood vessels." Surgery (Oxford) 33, no. 7 (July 2015): 295–301. http://dx.doi.org/10.1016/j.mpsur.2015.04.006.

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9

Carey, Francis A., and Mary N. Sheppard. "Diseases of blood vessels." Surgery (Oxford) 36, no. 6 (June 2018): 259–64. http://dx.doi.org/10.1016/j.mpsur.2018.03.011.

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10

Queral, Luis A. "Diseases of blood vessels." Journal of Vascular Surgery 3, no. 6 (June 1986): A1. http://dx.doi.org/10.1016/s0741-5214(86)70013-x.

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11

Queral, Luis A. "Diseases of blood vessels." Journal of Vascular Surgery 3, no. 6 (June 1986): 940. http://dx.doi.org/10.1016/0741-5214(86)90438-6.

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12

Abramson, David I. "Diseases of Blood Vessels." JAMA: The Journal of the American Medical Association 254, no. 14 (October 11, 1985): 2000. http://dx.doi.org/10.1001/jama.1985.03360140162051.

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13

Kalandarov, R. S., and L. L. Golovkina. "Blood groups and oncological diseases." Russian journal of hematology and transfusiology 66, no. 3 (October 25, 2021): 417–23. http://dx.doi.org/10.35754/0234-5730-2021-66-3-417-423.

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Introduction. The study of the possible impact of the blood group system ABO and other antigenic systems of red blood cells for cancer patients is currently directed at a number of distinct paths, including the study of changes in the expression of a group of antigens in tumors; the appearance of new antigens on tumor cells; the risk of developing tumors depending on the blood group; the possible infl uence of blood group on the prognosis of the disease, etc.Aim — to examine the current state of the problem of the relationship between blood groups and cancer.Main findings. There is evidence that cancer is less common in people with blood type O than in people with other blood groups of the ABO system. According to other data, this dependence is noted only for some types of cancer. In general, the results of work on this topic can be assessed as insuffi cient and sometimes contradictory.

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14

Mills, John A. "Inflammatory Diseases of Blood Vessels." Mayo Clinic Proceedings 77, no. 12 (December 2002): 1399. http://dx.doi.org/10.4065/77.12.1398.

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15

Hu, Mengyu, Yuxia Yang, Zhi Ji, and Jianyuan Luo. "RBM15 Functions in Blood Diseases." Current Cancer Drug Targets 16, no. 7 (August 12, 2016): 579–85. http://dx.doi.org/10.2174/1568009616666160112105706.

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16

Hossfeld, D. K. "Neoplastic Diseases of the Blood." Annals of Oncology 16, no. 1 (January 2005): 175. http://dx.doi.org/10.1093/annonc/mdi005.

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17

Hashimoto, Ryota. "Blood biomarkers for neuropsychiatric diseases." Psychiatry and Clinical Neurosciences 72, no. 3 (March 2018): 139. http://dx.doi.org/10.1111/pcn.12624.

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18

Hokland, M., and P. Hokland. "Interferons for malignant blood diseases?" European Journal of Haematology 38, no. 3 (April 24, 2009): 209–12. http://dx.doi.org/10.1111/j.1600-0609.1987.tb01165.x.

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19

Anderson, Kenneth C. "Neoplastic Diseases of the Blood." Mayo Clinic Proceedings 71, no. 8 (August 1996): 822. http://dx.doi.org/10.1016/s0025-6196(11)64851-9.

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20

McIntyre, O. Ross. "Neoplastic Diseases of the Blood." Mayo Clinic Proceedings 60, no. 6 (June 1985): 425. http://dx.doi.org/10.1016/s0025-6196(12)60855-6.

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21

Jokelainen, Kalle, Katja S. Salmela, Kari Humaloja, Risto Roine, Seppo Autio, Maria Arvio, Irma Järvelä, Irma Nykänen, Jorma Palo, and Mikko Salaspuro. "Blood dolichol in lysosomal diseases." Biochemistry and Cell Biology 70, no. 6 (June 1, 1992): 481–85. http://dx.doi.org/10.1139/o92-074.

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Highly elevated serum total dolichol (free dolichol + dolichyl ester) concentrations have recently been found in two lysosomal storage diseases, aspartylglucosaminuria (AGU) and mannosidosis. The present study demonstrates that the increase of serum dolichol in AGU patients is caused by an increase of serum free dolichol. In 15 patients the mean serum level of free dolichol (227 ± 16 ng/mL) was 1.9 times higher (p < 0.001) than that in healthy controls (120 ± 6 ng/mL), while the amounts of dolichol fatty acid esters were similar in the patients and controls (110 ± 9 vs. 118 ± 6 ng/mL). In contrast, 10 patients with neuronal ceroid-lipofuscinosis (NCL) (three with infantile, four with juvenile, and three with variant late infantile NCL) had significantly (p < 0.01) lower mean serum levels of both free (79 ± 5 ng/mL) and total (159 ± 6 ng/mL) dolichol than age-adjusted healthy controls (free, 100 ± 6 ng/mL; total, 206 ± 14 ng/mL). Decreased blood dolichol has not been reported earlier for any other disease. We conclude that the increased serum free dolichol in AGU reflects disturbed lysosomal function and that the decreased free and esterified dolichols in NCLs speak against their presumed primary lysosomal nature.Key words: serum dolichol, aspartylglucosaminuria, neuronal ceroid-lipofuscinosis, lysosomal function, lysosomal storage disease.

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22

Cheng, Tsung O. "Diseases That Lower Blood Pressure." Southern Medical Journal 82, no. 10 (October 1989): 1320. http://dx.doi.org/10.1097/00007611-198910000-00039.

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23

Geary, C. G. "Neoplastic Diseases of the Blood." British Journal of Cancer 53, no. 4 (April 1986): 581–82. http://dx.doi.org/10.1038/bjc.1986.95.

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24

BIEGUS, JAN, JAN SKÓRA, and ZBIGNIEW RYBAK. "Angiogenesis in blood vessels diseases." PRZEGLĄD FLEBOLOGICZNY 13, no. 5 (October 13, 2005): 1–8. http://dx.doi.org/10.1066/s10003050036.

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25

KNUUTILA, SAKARI, MARTTI SIIMES, and PEKKA VUOPIO. "Chromosome pulverization in blood diseases." Hereditas 95, no. 1 (February 12, 2009): 15–24. http://dx.doi.org/10.1111/j.1601-5223.1981.tb01323.x.

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26

Kohn, Donald B. "Gene therapy for blood diseases." Current Opinion in Biotechnology 60 (December 2019): 39–45. http://dx.doi.org/10.1016/j.copbio.2018.11.016.

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27

Hocking, William G. "Neoplastic Diseases of the Blood." JAMA: The Journal of the American Medical Association 267, no. 4 (January 22, 1992): 580. http://dx.doi.org/10.1001/jama.1992.03480040132050.

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28

Hocking, William G. "Neoplastic Diseases of the Blood." JAMA: The Journal of the American Medical Association 254, no. 2 (July 12, 1985): 280. http://dx.doi.org/10.1001/jama.1985.03360020114039.

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29

Nomura, Shosaku. "Extracellular vesicles and blood diseases." International Journal of Hematology 105, no. 4 (January 27, 2017): 392–405. http://dx.doi.org/10.1007/s12185-017-2180-x.

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30

TOOYAMA, KAORU. "Apoptosis and internal medicine diseases. Blood diseases and apoptosis." Nihon Naika Gakkai Zasshi 86, no. 9 (1997): 1597–601. http://dx.doi.org/10.2169/naika.86.1597.

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31

UEDA, TAKANORI. "Blood transfusion therapy of internal medical field. Blood diseases and blood transfusion." Nihon Naika Gakkai Zasshi 85, no. 6 (1996): 857–61. http://dx.doi.org/10.2169/naika.85.857.

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32

Cildag, Songul, Yasemin Kara, and Taskin Senturk. "ABO blood groups and rheumatic diseases." European Journal of Rheumatology 4, no. 4 (December 19, 2017): 250–53. http://dx.doi.org/10.5152/eurjrheum.2017.17044.

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33

Sato, N., S. Kawano, S. Tsuji, T. Ogihara, and S. Yamada. "Gastric Blood Flow in Ulcer Diseases." Scandinavian Journal of Gastroenterology 30, sup208 (January 1995): 14–20. http://dx.doi.org/10.3109/00365529509107756.

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34

Tomasik, Jaromir, and Grzegorz Władysław Basak. "Inflammasomes—New Contributors to Blood Diseases." International Journal of Molecular Sciences 23, no. 15 (July 23, 2022): 8129. http://dx.doi.org/10.3390/ijms23158129.

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Inflammasomes are intracellular multimeric complexes that cleave the precursors of the IL-1 family of cytokines and various proteins, found predominantly in cells of hematopoietic origin. They consist of pattern-recognition receptors, adaptor domains, and the enzymatic caspase-1 domain. Inflammasomes become activated upon stimulation by various exogenous and endogenous agents, subsequently promoting and enhancing inflammatory responses. To date, their function has been associated with numerous pathologies. Most recently, many studies have focused on inflammasomes’ contribution to hematological diseases. Due to aberrant expression levels, NLRP3, NLRP1, and NLRC4 inflammasomes were indicated as predominantly involved. The NLRP3 inflammasome correlated with the pathogenesis of non-Hodgkin lymphomas, multiple myeloma, acute myeloid leukemia, lymphoid leukemias, myelodysplastic neoplasms, graft-versus-host-disease, and sickle cell anemia. The NLRP1 inflammasome was associated with myeloma and chronic myeloid leukemia, whereas NLRC4 was associated with hemophagocytic lymphohistiocytosis. Moreover, specific gene variants of the inflammasomes were linked to disease susceptibility. Despite the incomplete understanding of these correlations and the lack of definite conclusions regarding the therapeutic utility of inflammasome inhibitors, the available results provide a valuable basis for clinical applications and precede upcoming breakthroughs in the field of innovative treatments. This review summarizes the latest knowledge on inflammasomes in hematological diseases, indicates the potential limitations of the current research approaches, and presents future perspectives.

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35

Mustjoki, Satu. "Somatic mutations in “benign” blood diseases." Seminars in Hematology 59, no. 3 (July 2022): 121–22. http://dx.doi.org/10.1053/j.seminhematol.2022.08.003.

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36

Urabe, Akio. "Treatment of blood diseases and DDS." Drug Delivery System 19, no. 2 (2004): 90–94. http://dx.doi.org/10.2745/dds.19.90.

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37

Weatherall, D. J. "Prenatal Diagnosis of Inherited Blood Diseases." Clinics in Haematology 14, no. 3 (October 1985): 747–74. http://dx.doi.org/10.1016/s0308-2261(21)00503-8.

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38

Lang, Calvin A., Betty J. Mills, Walter Mastropaolo, and Marcia C. Liu. "Blood glutathione decreases in chronic diseases." Journal of Laboratory and Clinical Medicine 135, no. 5 (May 2000): 402–5. http://dx.doi.org/10.1067/mlc.2000.105977.

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39

Finch, Stuart C. "Book ReviewNeoplastic Diseases of the Blood." New England Journal of Medicine 312, no. 21 (May 23, 1985): 1395. http://dx.doi.org/10.1056/nejm198505233122121.

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40

Fowlkes, Laura, and Jay M. Sullivan. "Estrogens, Blood Pressure, and Cardiovascular Diseases." Cardiology in Review 3, no. 2 (March 1995): 106–14. http://dx.doi.org/10.1097/00045415-199503000-00006.

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41

Zhang, Hanrui, Ciarán J. Mooney, and Muredach P. Reilly. "ABO Blood Groups and Cardiovascular Diseases." International Journal of Vascular Medicine 2012 (2012): 1–11. http://dx.doi.org/10.1155/2012/641917.

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ABO blood groups have been associated with various disease phenotypes, particularly cardiovascular diseases. Cardiovascular diseases are the most common causes of death in developed countries and their prevalence rate is rapidly growing in developing countries. There have been substantial historical associations between non-O blood group status and an increase in some cardiovascular disorders. Recent GWASs have identified ABO as a locus for thrombosis, myocardial infarction, and multiple cardiovascular risk biomarkers, refocusing attention on mechanisms and potential for clinical advances. As we highlight in this paper, more recent work is beginning to probe the molecular basis of the disease associations observed in these observational studies. Advances in our understanding of the physiologic importance of various endothelial and platelet-derived circulating glycoproteins are elucidating the mechanisms through which the ABO blood group may determine overall cardiovascular disease risk. The role of blood group antigens in the pathogenesis of various cardiovascular disorders remains a fascinating subject with potential to lead to novel therapeutics and prognostics and to reduce the global burden of cardiovascular diseases.

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42

LI, HuiYuan, and RenChi YANG. "Research progress of hereditary blood diseases." SCIENTIA SINICA Vitae 47, no. 12 (December 1, 2017): 1306–12. http://dx.doi.org/10.1360/n052017-00271.

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43

Peake, I. "The Molecular Basis of Blood Diseases." Journal of Medical Genetics 25, no. 11 (November 1, 1988): 789–90. http://dx.doi.org/10.1136/jmg.25.11.789-a.

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44

Takeshita, Yukio, and Richard M. Ransohoff. "Blood-brain barrier and neurological diseases." Clinical and Experimental Neuroimmunology 6, no. 4 (July 27, 2015): 351–61. http://dx.doi.org/10.1111/cen3.12229.

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45

Polli, Elio, and Agostino Cortelezzi. "Therapy with Interferons in Blood Diseases." Acta Haematologica 78, no. 1 (1987): 64–69. http://dx.doi.org/10.1159/000205905.

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46

Law, Malcolm. "Salt, Blood Pressure and Cardiovascular Diseases." European Journal of Cardiovascular Risk 7, no. 1 (February 2000): 5–8. http://dx.doi.org/10.1177/204748730000700102.

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47

Bordignon, Claudio. "Stem-cell therapies for blood diseases." Nature 441, no. 7097 (June 2006): 1100–1102. http://dx.doi.org/10.1038/nature04962.

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48

Glomstein, A. "Self-sufficiency and blood transmitted diseases." Blood Coagulation & Fibrinolysis 6, Sup 2 (July 1995): S23—S26. http://dx.doi.org/10.1097/00001721-199506002-00006.

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49

Yoder, Linda H. "Diseases treated with blood cell transplants." Seminars in Oncology Nursing 13, no. 3 (August 1997): 164–71. http://dx.doi.org/10.1016/s0749-2081(97)80032-0.

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50

Baxter, B. Timothy. "Heritable diseases of the blood vessels." Cardiovascular Pathology 14, no. 4 (July 2005): 185–88. http://dx.doi.org/10.1016/j.carpath.2005.04.003.

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