Log in

Relevant bibliographies by topics / Bladder disease / Journal articles

Journal articles on the topic 'Bladder disease'

To see the other types of publications on this topic, follow the link: Bladder disease.

Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Bladder disease.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Yu, Zhenyuan, Jinling Liao, Yang Chen, Chunlin Zou, Haiying Zhang, Jiwen Cheng, Deyun Liu, et al. "Single-Cell Transcriptomic Map of the Human and Mouse Bladders." Journal of the American Society of Nephrology 30, no. 11 (August 28, 2019): 2159–76. http://dx.doi.org/10.1681/asn.2019040335.

Full text

Abstract:

BackgroundHaving a comprehensive map of the cellular anatomy of the normal human bladder is vital to understanding the cellular origins of benign bladder disease and bladder cancer.MethodsWe used single-cell RNA sequencing (scRNA-seq) of 12,423 cells from healthy human bladder tissue samples taken from patients with bladder cancer and 12,884 cells from mouse bladders to classify bladder cell types and their underlying functions.ResultsWe created a single-cell transcriptomic map of human and mouse bladders, including 16 clusters of human bladder cells and 15 clusters of mouse bladder cells. The homology and heterogeneity of human and mouse bladder cell types were compared and both conservative and heterogeneous aspects of human and mouse bladder evolution were identified. We also discovered two novel types of human bladder cells. One type is ADRA2A+ and HRH2+ interstitial cells which may be associated with nerve conduction and allergic reactions. The other type is TNNT1+ epithelial cells that may be involved with bladder emptying. We verify these TNNT1+ epithelial cells also occur in rat and mouse bladders.ConclusionsThis transcriptomic map provides a resource for studying bladder cell types, specific cell markers, signaling receptors, and genes that will help us to learn more about the relationship between bladder cell types and diseases.

APA, Harvard, Vancouver, ISO, and other styles

2

Jones, Emily, John Alawneh, Mary Thompson, Chiara Palmieri, Karen Jackson, and Rachel Allavena. "Predicting Diagnosis of Australian Canine and Feline Urinary Bladder Disease Based on Histologic Features." Veterinary Sciences 7, no. 4 (November 27, 2020): 190. http://dx.doi.org/10.3390/vetsci7040190.

Full text

Abstract:

Anatomic pathology is a vital component of veterinary medicine but as a primarily subjective qualitative or semiquantitative discipline, it is at risk of cognitive biases. Logistic regression is a statistical technique used to explain relationships between data categories and outcomes and is increasingly being applied in medicine for predicting disease probability based on medical and patient variables. Our aims were to evaluate histologic features of canine and feline bladder diseases and explore the utility of logistic regression modeling in identifying associations in veterinary histopathology, then formulate a predictive disease model using urinary bladder as a pilot tissue. The histologic features of 267 canine and 71 feline bladder samples were evaluated, and a logistic regression model was developed to identify associations between the bladder disease diagnosed, and both patient and histologic variables. There were 102 cases of cystitis, 84 neoplasia, 42 urolithiasis and 63 normal bladders. Logistic regression modeling identified six variables that were significantly associated with disease outcome: species, urothelial ulceration, urothelial inflammation, submucosal lymphoid aggregates, neutrophilic submucosal inflammation, and moderate submucosal hemorrhage. This study demonstrated that logistic regression modeling could provide a more objective approach to veterinary histopathology and has opened the door toward predictive disease modeling based on histologic variables.

APA, Harvard, Vancouver, ISO, and other styles

3

Giridharan, Bhanumati, and Madhivadhanam Madhivadhanam. "Clinical analysis on Gall Bladder Disease Cholecystitis and Cholelitheasis." Scholars Journal of Applied Medical Sciences 4, no. 7 (July 2016): 2480–83. http://dx.doi.org/10.21276/sjams.2016.4.7.36.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

David, Rowan, Luke Traeger, and Christopher McDonald. "Gall bladder torsion: a disease of the elderly." BMJ Case Reports 12, no. 10 (October 2019): e227324. http://dx.doi.org/10.1136/bcr-2018-227324.

Full text

Abstract:

We describe the case of a gall bladder torsion in an elderly female patient, which was discovered during laparoscopic exploration for presumed acute cholecystitis. The rising incidence of this relatively uncommon process can be attributed to increasing life expectancy. Gall bladder torsion typically manifests in septuagenarians and octogenarians of the female gender, as seen in the presented case. It is thought that local mesenteric redundancy predisposes to the development of mechanical organoaxial torsion along the gall bladder’s longitudinal axis involving the cystic duct and artery. Clinicians must have a high index of suspicion for gall bladder torsion, as a mimicker of acute cholecystitis, in the described patient demographic. Preoperative diagnosis is challenging with the vast majority of reported cases being diagnosed intraoperatively, and only five cases preoperatively. Prompt surgical intervention results in an overall mortality rate of approximately 5%, while a delay in diagnosis can lead to catastrophic patient outcomes.

APA, Harvard, Vancouver, ISO, and other styles

5

Peters, C. A., M. R. Freeman, C. A. Fernandez, J. Shepard, D. G. Wiederschain, and M. A. Moses. "Dysregulated proteolytic balance as the basis of excess extracellular matrix in fibrotic disease." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 272, no. 6 (June 1, 1997): R1960—R1965. http://dx.doi.org/10.1152/ajpregu.1997.272.6.r1960.

Full text

Abstract:

To investigate mechanisms of tissue fibrosis, we developed a model of ovine fetal bladder fibrosis due to surgically induced obstruction. Tissues were analyzed for matrix metalloproteinases (MMPs) and their inhibitors, the tissue inhibitors of metalloproteinases (TIMPs). Active MMP-2 was not detected in obstructed bladders, while latent and active forms were detected in normal bladders. MMP-1 (interstitial collagenase) activity was lower in obstructed bladders. MMP inhibitory activity was increased with obstruction, as were levels of TIMP mRNA and protein. These results indicate that the proteins responsible for collagen degradation are present in the developing bladder, and a shift in the proteolytic balance favoring inhibition of degradation occurs in a model of obstruction-induced fibrosis. This altered proteolytic balance favors accumulation of extracellular matrix and decreased tissue compliance characteristic of this and perhaps other fibrotic conditions.

APA, Harvard, Vancouver, ISO, and other styles

6

Scremin, Luciano Henrique Gazoni, Carlos Eduardo Pereira Corbett, Márcia Dallastra Laurenti, Elizabeth Visone Nunes, Joaquim José Gama-Rodrigues, and Masayuki Okumura. "Megabladder in experimental Chagas disease: pathological features of the bladder wall." Revista do Hospital das Clínicas 54, no. 2 (April 1999): 43–46. http://dx.doi.org/10.1590/s0041-87811999000200003.

Full text

Abstract:

Mega-organs, primarily in the digestive tract, are well known to occur in chronic Chagas disease. Acute experimental infection with Trypanosoma cruzi results in parasitism of a wide range of cells, tissues, and organs, including the urinary bladder. Infection of BALB/c mice with 100,000 bloodstream forms of the Y strain of T. cruzi induced acute infection with intense parasitism of all layers of the urinary bladder. Parasites were found in the mucosa, lamina propria, muscular, adventitial connective, and fat tissue. Desquamate epithelial cells with amastigotes in the bladder lumen were also found. After 60 days of infection, mice inoculated with 50 bloodstream forms developed dilated, thin-walled bladders that had inflammatory infiltrates and foci of fibrosis replacing areas of damaged muscular layer. These lesions result from direct damage to the muscle fibers by the T. cruzi, leading to myosites, muscle damage, and scarring. Direct damage of paraganglia cells secondary to parasitism, leading to dilatation, damage of muscle fibers, and scarring with replacement of muscular tissue with connective tissue, should also be considered as a cause of functional disturbance of the urinary bladder.

APA, Harvard, Vancouver, ISO, and other styles

7

Li, Huixi, Guiting Lin, and Tom F. Lue. "Potential application of adipose tissue-derived stem cells for urological Disease." Bladder 1, no. 1 (November 4, 2014): 2. http://dx.doi.org/10.14440/bladder.2014.23.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Shah, Navin C. "Atlas of Bladder Disease." JAMA 304, no. 1 (July 7, 2010): 99. http://dx.doi.org/10.1001/jama.2010.912.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Speich, John E., Jordan B. Southern, Sheree Henderson, Cameron W. Wilson, Adam P. Klausner, and Paul H. Ratz. "Adjustable passive stiffness in mouse bladder: regulated by Rho kinase and elevated following partial bladder outlet obstruction." American Journal of Physiology-Renal Physiology 302, no. 8 (April 15, 2012): F967—F976. http://dx.doi.org/10.1152/ajprenal.00177.2011.

Full text

Abstract:

Detrusor smooth muscle (DSM) contributes to bladder wall tension during filling, and bladder wall deformation affects the signaling system that leads to urgency. The length-passive tension ( L-Tp) relationship in rabbit DSM can adapt with length changes over time and exhibits adjustable passive stiffness (APS) characterized by a L-Tpcurve that is a function of both activation and strain history. Muscle activation with KCl, carbachol (CCh), or prostaglandin E2at short muscle lengths can increase APS that is revealed by elevated pseudo-steady-state Tpat longer lengths compared with prior Tpmeasurements at those lengths, and APS generation is inhibited by the Rho Kinase (ROCK) inhibitor H-1152. In the current study, mouse bladder strips exhibited both KCl- and CCh-induced APS. Whole mouse bladders demonstrated APS which was measured as an increase in pressure during passive filling in calcium-free solution following CCh precontraction compared with pressure during filling without precontraction. In addition, CCh-induced APS in whole mouse bladder was inhibited by H-1152, indicating that ROCK activity may regulate bladder compliance during filling. Furthermore, APS in whole mouse bladder was elevated 2 wk after partial bladder outlet obstruction, suggesting that APS may be relevant in diseases affecting bladder mechanics. The presence of APS in mouse bladder will permit future studies of APS regulatory pathways and potential alterations of APS in disease models using knockout transgenetic mice.

APA, Harvard, Vancouver, ISO, and other styles

10

Bales, Gregory T., Francis H. Straus, II, and Glenn S. Gerber. "Crohn's Disease and Urinary Bladder Mass." Diagnostic and Therapeutic Endoscopy 1, no. 4 (January 1, 1995): 233–36. http://dx.doi.org/10.1155/dte.1.233.

Full text

Abstract:

The presence of a bladder mass in a patient with inflammatory bowel disease poses a diagnostic dilemma. We present the case of a 26-year-old male with a bladder mass who had not previously been diagnosed with Crohn's disease. Initial biopsies of the bladder mass were consistent with inflammatory changes, but superficial transitional cell carcinoma could not be reliably excluded. Subsequent evaluation confirmed the presence of Crohn's disease with bladder involvement, and the patient underwent bowel resection and partial cystectomy. Pathologic evaluation demonstrated Crohn’s disease and no evidence of malignancy. Accurate differentiation of benign and malignant bladder masses in patients with inflammatory bowel disease may be difficult and requires cooperation between pathologists and clinicians.

APA, Harvard, Vancouver, ISO, and other styles

11

Jones, Emily, John Alawneh, Mary Thompson, and Rachel Allavena. "Association between case signalment and disease diagnosis in urinary bladder disease in Australian cats and dogs." Journal of Veterinary Diagnostic Investigation 33, no. 3 (April 2, 2021): 498–505. http://dx.doi.org/10.1177/10406387211004008.

Full text

Abstract:

Urinary bladder diseases are common in dogs and cats; however, there is little published work on urinary bladder disease in Australian pets. We identified pathology records of Australian dogs and cats with urinary bladder tissue submitted to the University of Queensland Veterinary Laboratory Service during 1994–2016 ( n = 320). We described the proportion of bladder diseases in dogs and cats, and applied the less-commonly used logistic regression procedure to quantify associations between signalment variables and disease diagnosis that were evident using descriptive statistics alone. After preliminary analysis, both species were combined because of similar results. Spayed/castrated animals were 74% less likely to be diagnosed with cystitis compared with intact animals. Animals 4–11 y old were also at lower risk of being diagnosed with cystitis compared with younger or older animals. Male animals were at increased risk of neoplasia compared to females, which contrasts with reports from North America and Europe. There was increased risk for developing neoplasia with progressive age, with up to 20 times higher odds in the > 11-y age group. Logistic regression modeling provided unique insight into proportionate morbidity of urinary bladder diseases in Australian dogs and cats.

APA, Harvard, Vancouver, ISO, and other styles

12

Tabakin, Alexandra L., and Hari S. G. R. Tunuguntla. "Does deep brain stimulation improve Parkinson’s disease-related lower urinary tract symptoms and voiding dysfunction?" Bladder 8, no. 2 (August 27, 2021): e46. http://dx.doi.org/10.14440/bladder.2021.825.

Full text

Abstract:

Parkinson’s disease (PD), caused by degeneration of dopaminergic neurons, leads to motor and autonomic symptoms. A large proportion of PD patients experience lower urinary tract symptoms (LUTS) and voiding dysfunction, associated with poor quality of life. Deep brain stimulation (DBS) is an adjunctive therapy used in combination with medication for Parkinsonian motor symptoms and is currently being explored to treat PD-related LUTS. This review discusses the current literature on the effects of DBS on lower urinary tract function and whether this modality can be used to modulate PD-related LUTS and voiding dysfunction.

APA, Harvard, Vancouver, ISO, and other styles

13

Aiba, Yosuke, Ryuji Sakakibara, Fang-Ching Lee, and Fuyuki Tateno. "Urodynamic Assessment of Neuronal Intranuclear Inclusion Disease." European Neurology 83, no. 3 (2020): 312–16. http://dx.doi.org/10.1159/000508746.

Full text

Abstract:

Neuronal intranuclear inclusion disease (NIID) is a disease that causes leukoencephalopathy (dementia) and peripheral neuropathy (variable manifestation including bladder dysfunction). This is the first urodynamic report to show that bladder dysfunction in NIID is a combination of detrusor overactivity, decreased bladder sensation, large post-void residual, and neurogenic changes in the sphincter electromyogram. This report will help managing bladder dysfunction in NIID.

APA, Harvard, Vancouver, ISO, and other styles

14

Pasternak, Grzegorz, Dorota Bartusik-Aebisher, David Aebisher, and Rafał Filip. "Crohn’s disease complicated with a bladder-fistula – a case report." European Journal of Clinical and Experimental Medicine 19, no. 1 (2021): 76–80. http://dx.doi.org/10.15584/ejcem.2021.1.10.

Full text

Abstract:

Introduction. Entero-bladder fistula (fistula entero-vesicalis) is a pathological connection between the lumen of the gastrointestinal tract and the bladder. Entero-bladder fistulas are not a common condition. The main reason for the formation of entero- bladder fistulas are intestinal diseases occurring within the intestinal loop adjacent to the bladder resulting in the formation of an abnormal channel, the connection between the above structures Aim. The aim is to present the causes of the fistulas can be divided into congenital and acquired (intestinal infection, cancer, Crohn’s disease, resulting from trauma and iatrogenic). Clinical manifestations of the biliary-bullous fistulae may be from the digestive or urinary tract. The most characteristic ailments are pneumaturia, fecuria, urge to urinate, frequent urination, lower abdominal pain, hematuria, urinary tract infection. Description of the case. The article discusses the case of a patient with Leśniowski-Crohn disease complicated with a bladder- fistula. The treatment of entero-bladder fistulas is primarily surgical, it consists in resection of the fistula together with resection of the affected intestine and bladder wall fragment. Conclusion. The test confirming the presence of an entero-bladder fistula is a test with oral administration of poppies, although it happens that the test result may be negative, especially in the case of a bladder-follicular fistula. Among the tests useful in the diagnosis of entero-bladder fistula include abdominal ultrasound, computed tomography, magnetic resonance imaging, endoscopic tests (colonoscopy or cystoscopy).

APA, Harvard, Vancouver, ISO, and other styles

15

Kim, Mi-hwa, Won Chan Kim, and Dong-Su Park. "Neurogenic Bladder in Lyme Disease." International Neurourology Journal 16, no. 4 (2012): 201. http://dx.doi.org/10.5213/inj.2012.16.4.201.

Full text

APA, Harvard, Vancouver, ISO, and other styles

16

Basra, Ramandeep, and Con Kelleher. "Disease Burden of Overactive Bladder." PharmacoEconomics 25, no. 2 (2007): 129–42. http://dx.doi.org/10.2165/00019053-200725020-00005.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Merine, Dimitri, Elliot K. Fishman, Janet E. Kuhlman, Bronwyn Jones, Theodore M. Bayless, and Stanley Siegelman. "Bladder Involvement in Crohn Disease." Journal of Computer Assisted Tomography 13, no. 1 (January 1989): 90–93. http://dx.doi.org/10.1097/00004728-198901000-00019.

Full text

APA, Harvard, Vancouver, ISO, and other styles

18

Woodhouse, D. R., and B. Haylen. "Gall Bladder Disease Complicating Pregnancy." Australian and New Zealand Journal of Obstetrics and Gynaecology 25, no. 3 (August 1985): 233–37. http://dx.doi.org/10.1111/j.1479-828x.1985.tb00652.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

19

Dincer, Murat, Omer Onur Cakir, Onur Fikri, and Engin Kandirali. "Bladder Dysfunction in Behçet’s Disease." Journal of Academic Research in Medicine 7, no. 2 (August 24, 2017): 86–88. http://dx.doi.org/10.5152/jarem.2016.972.

Full text

APA, Harvard, Vancouver, ISO, and other styles

20

Mizutani, Yoichi, and Haruhito Azuma. "Prognostic significance of second mitochondria-derived activator of caspase expression in bladder cancer and target for therapy." Journal of Clinical Oncology 30, no. 15_suppl (May 20, 2012): e15000-e15000. http://dx.doi.org/10.1200/jco.2012.30.15_suppl.e15000.

Full text

Abstract:

e15000 Background: Although the expression of second mitochondria-derived activator of caspase (Smac/DIABLO) has been reported in various cancers, little is known about its clinical significance in bladder cancer. The present study was designed to evaluate the relationship between progression of disease and Smac/DIABLO expression by clinical pathological analysis of patients with bladder cancer. Methods: The level of Smac/DIABLO expression was quantified by western blot analysis using non-fixed fresh frozen tissues derived from patients with bladder cancer. Results: All normal bladders expressed Smac/DIABLO. However, 64/84 ( 76% ) of bladder cancers expressed Smac/DIABLO and 24% were negative. In Ta and T1 superficial bladder cancers, 98% expressed Smac/DIABLO, whereas only 41% expressed Smac/DIABLO in muscle-invasive bladder cancers. Smac/DIABLO expression inversely correlated with the grade of bladder cancer. Patients with Ta and T1 superficial bladder cancer with higher Smac/DIABLO expression had a longer postoperative recurrence-free period than those with lower Smac/DIABLO expression after transurethral resection in the 5-year follow-up. Patients with invasive bladder cancer expressing Smac/DIABLO had a longer postoperative disease-specific survival than those without Smac/DIABLO expression after radical cystectomy in the 5-year follow-up. The cisplatin-resistant T24 bladder cancer line ( T24/CDDP ) and the adriamycin-resistant T24 line ( T24/ADR ) showed lower level of Smac/DIABLO expression, compared with the T24 parental line. Conclusions: The present study demonstrates for the first time that Smac/DIABLO expression was downregulated in bladder cancer, especially in high grade muscle-invasive bladder cancer, and that lower Smac/DIABLO expression in bladder cancer predicted a worse prognosis. In addition, the cisplatin-resistant T24/CDDP line and the adriamycin-resistant T24/ADR line expressed lower level of Smac/DIABLO expression. These results suggest that Smac/DIABLO expression in bladder cancer may be used as a prognostic parameter, and that low Smac/DIABLO expression in bladder cancer may be associated with resistance to chemotherapy.

APA, Harvard, Vancouver, ISO, and other styles

21

Iacovelli, Elisa, Francesca Gilio, Giuseppe Meco, Francesco Fattapposta, Nicola Vanacore, Livia Brusa, Elena Giacomelli, et al. "Bladder symptoms assessed with overactive bladder questionnaire in Parkinson's disease." Movement Disorders 25, no. 9 (March 22, 2010): 1203–9. http://dx.doi.org/10.1002/mds.23093.

Full text

APA, Harvard, Vancouver, ISO, and other styles

22

Keihani, Sorena, and Abdol-Mohammad Kajbafzadeh. "Bladder rupture after voiding cystourethrography: A case report and literature review on pitfalls and bladder volume estimation." Canadian Urological Association Journal 9, no. 11-12 (November 4, 2015): 826. http://dx.doi.org/10.5489/cuaj.3234.

Full text

Abstract:

Voiding cystourethrography (VCUG) is a common diagnostic study in pediatric urology. Although generally considered a safe procedure, VCUG can incur some complications. Bladder rupture following VCUG is a very rare complication and mostly happens in the setting of unused bladders, previous surgeries, or underlying disease. The rupture is almost always intraperitoneal needing prompt surgical treatment. We present a unique case of extraperitoneal bladder rupture after VCUG in an 8-month-old boy. The rupture remained unnoticed and the patient recovered spontaneously without medical or surgical intervention. To the best of our knowledge, this is the first such case report in the English literature. The information provided may guide clinicians on the proper use of bladder volume formulas in children and also help them in getting a proper VCUG study.

APA, Harvard, Vancouver, ISO, and other styles

23

Tyson, Mark D., Sam S. Chang, and Krik A. Keegan. "Role of consolidative surgical therapy in patients with locally advanced or regionally metastatic bladder cancer." Bladder 3, no. 2 (August 31, 2016): e26. http://dx.doi.org/10.14440/bladder.2016.89.

Full text

Abstract:

The effect of radical cystectomy and extended pelvic lymph node dissection (RC/PLND) on the survival of patients with locally advanced and/or regionally metastatic bladder cancer is unknown. However, emerging evidence suggests that there may be survival benefit to a subset of select patients with this disease who demonstrate a response to chemotherapy. This article will review the current literature on the role of RC/PLND in the consolidative treatment of locally advanced and regionally metastatic bladder cancer.

APA, Harvard, Vancouver, ISO, and other styles

24

B. J., Sharath Chandra, and Rahul Bose. "Study of the degree of gall bladder wall thickness and its impact on outcomes following laparoscopic cholecystectomy in JSS Hospital." International Surgery Journal 5, no. 4 (March 23, 2018): 1417. http://dx.doi.org/10.18203/2349-2902.isj20181122.

Full text

Abstract:

Background: Elective laparoscopic cholecystectomy (LC) is presently the gold standard for management of symptomatic gall stone disease, replacing open cholecystectomy. The objective of this study was to measure gall bladder wall thickness preoperatively on ultrasound in patients with symptomatic gallstone disease and to establish its role as a prognostic indicator for complications during or following laparoscopic cholecystectomy.Methods: Gall bladder wall thickness was measured by ultrasonography in 151 patients who presented with Gallstone disease and underwent laparoscopic cholecystectomy for gallstone disease in the JSS Hospital in the given time period. They were then divided into 4 groups depending upon the wall thickness. Normal (upto 2 mm), mildly thickened (>2 to 4 mm), moderately thickened (>4 to 6mm), severely thickened (>6 mm). The incidence of intra and post-operative complications were monitored and compared between the four groups.Results: The incidence of complications was found to be significantly higher in patients with mildly and moderately thickened gall bladder walls (53.1% and 83.3% respectively) as compared to gall bladders with normal wall thickness (10.5%). Average postoperative length of stay in hospital was significantly higher in patients with thickened walls as compared to patients with normal thickness.Conclusions: With wall thickness of greater than 2 mm, the complication and conversion rates are extremely high. An increase in the thickness of the gall bladder wall leads to an increased risk of complications and conversions as well as an increased length of stay in hospital post operatively. Patients with thickened gall bladder walls accounted for only 30% of the study population but they experienced the maximum number of complications (72.5%) and conversions to open (71.4%).

APA, Harvard, Vancouver, ISO, and other styles

25

Genç, Mine, Berhan Genç, Serap Karaarslan, Aynur Solak, and Musa Saraçoglu. "Endometriosis localized to urinary bladder wall mimicking urinary bladder carcinoma." Archivio Italiano di Urologia e Andrologia 86, no. 3 (September 30, 2014): 233. http://dx.doi.org/10.4081/aiua.2014.3.233.

Full text

Abstract:

Although endometriosis is a common disease in women of reproductive age, urinary system endometriosis is an exceedingly rare disease that may cause important clinical problems. In this paper we discussed a 42-year-old woman who had urinary bladder endometriosis misdiagnosed as urinary bladder tumor in imaging modalities. The diagnosis of endometriosis was made by histopathological examination of the operative material after partial resection of the urinary bladder. Urinary bladder endometriosis causes nonspecific signs and symptoms in many patients. In female patients presenting with unexplained urinary symptoms the differential diagnosis should include urinary bladder endometriosis that may mimic urinary bladder cancer and lead to difficulties in making definitive preoperative diagnosis.

APA, Harvard, Vancouver, ISO, and other styles

26

Rousseau, Matthieu, Conan J. O. O’Brien, Eduardo Antequera, Hana Zdimerova, Dilay Cansever, Tracy Canton, Anna Zychlinsky Scharff, and Molly A. Ingersoll. "Identification of Sex Differences in Tumor-Specific T Cell Infiltration in Bladder Tumor-Bearing Mice Treated with BCG Immunotherapy." Bladder Cancer 6, no. 4 (December 14, 2020): 507–24. http://dx.doi.org/10.3233/blc-200384.

Full text

Abstract:

BACKGROUND: Bladder cancer is the fourth most common cancer for men. However, women are often diagnosed with later stage disease and have poorer outcomes. Whether immune-based sex differences contribute to this discrepancy is unclear. In addition, models to investigate tumor-specific immunity in bladder cancer, in the context of tumor development or response to therapy, are lacking. OBJECTIVE: To address this specific unmet need, we incorporated a commonly used model antigen, ovalbumin, into two well-established models of bladder cancer; the orthotopic MB49 cell line model and the carcinogenic BBN bladder cancer model. METHOD: We tested the utility of these models to investigate tumor-specific immunity in the context of immunotherapy in both sexes. RESULTS: We found that BCG vaccination, prior to weekly BCG instillation does not impart an immune-specific benefit to tumor-bearing mice in the context of multiple BCG instillations. Furthermore, tumors developed in the testes in male mice, precluding the use of the MB49 model to directly investigate sex-based immune differences. In the BBN model, we observed that more tumor antigen-specific CD8+ T cells infiltrated male bladders compared to female bladders in the context of BCG immunotherapy whereas regulatory T cells had higher levels of the exhaustion marker PD-1 in female mice. CONCLUSIONS: We propose our modified BBN model will contribute to our understanding of how tumor-specific immunity arises in bladder cancer. Additionally, the BBN bladder cancer model may help to uncover sex differences in tumor-specific immunity, which would provide valuable information for the development of new treatments or combination therapies for bladder cancer in women and men.

APA, Harvard, Vancouver, ISO, and other styles

27

Krishnanand, Dr Krishnanand, and Dr Narendra Singh Kurmi. "Study of gall bladder disease with incidence of gall bladder malignancy." Surgical Update: International Journal of Surgery and Orthopedics 5, no. 1 (March 31, 2019): 54–59. http://dx.doi.org/10.17511/ijoso.2019.i01.09.

Full text

APA, Harvard, Vancouver, ISO, and other styles

28

Klee, Nicole S., Robert S. Moreland, and Derek M. Kendig. "Detrusor contractility to parasympathetic mediators is differentially altered in the compensated and decompensated states of diabetic bladder dysfunction." American Journal of Physiology-Renal Physiology 317, no. 2 (August 1, 2019): F388—F398. http://dx.doi.org/10.1152/ajprenal.00178.2019.

Full text

Abstract:

Diabetic bladder dysfunction (DBD) affects up to 50% of all patients with diabetes, characterized by symptoms of both overactive and underactive bladder. Although most diabetic bladder dysfunction studies have been performed using models with type 1 diabetes, few have been performed in models of type 2 diabetes, which accounts for ~90% of all diabetic cases. In a type 2 rat model using a high-fat diet (HFD) and two low doses of streptozotocin (STZ), we examined voiding measurements and functional experiments in urothelium-denuded bladder strips to establish a timeline of disease progression. We hypothesized that overactive bladder symptoms (compensated state) would develop and progress into symptoms characterized by underactive bladder (decompensated state). Our results indicated that this model developed the compensated state at 1 wk after STZ and the decompensated state at 4 mo after STZ administration. Diabetic bladders were hypertrophied compared with control bladders. Increased volume per void and detrusor muscle contractility to exogenous addition of carbachol and ATP confirmed the development of the compensated state. This enhanced contractility to carbachol was not due to increased levels of M3 receptor expression. Decompensation was characterized by increased volume per void, number of voids, and contractility to ATP but not carbachol. Thus, progression from the compensated to decompensated state may involve decreased contractility to muscarinic stimulation. These data suggest that the compensated state of DBD progresses temporally into the decompensated state in the male HFD/STZ model of diabetes; therefore, this male HFD/STZ model can be used to study the progression of DBD.

APA, Harvard, Vancouver, ISO, and other styles

29

Jesudasan, Ajantha, and Mohammed A. Rana. "Urinary bladder problems in Parkinson's disease." Journal of Parkinsonism & Restless Legs Syndrome 2, no. 1 (April 2012): 30–31. http://dx.doi.org/10.7157/jprls.2012.v2n1pp30-31.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Mudoni, Anna, Francesco Caccetta, Maurizio Caroppo, Fernando Musio, Antonella Accogli, Maria Dolores Zacheo, Domenica Maria Lucia Burzo, Giancamillo Carluccio, and Vitale Nuzzo. "Bladder neck disease and kidney damage." Archivio Italiano di Urologia e Andrologia 86, no. 4 (December 30, 2014): 391. http://dx.doi.org/10.4081/aiua.2014.4.391.

Full text

Abstract:

Primary bladder neck obstruction (PBNO) was first described in men by Marion in 1933. The precise cause of PBNO has not been clearly elucidated. This paper review the theories on etiology, clinical presentation, diagnostic evaluation and treatments for PBNO. Also this paper focuses on management of patients with complications like acute urine retention, hydroureteronephrosis and severe renal failure. The treatment options for men and women with PBNO include careful clinical evaluation, pharmacotherapy with alpha-blockers and surgical intervention.

APA, Harvard, Vancouver, ISO, and other styles

31

Smith, Thomas J., Victoria Andrews, Seok Cho, Nicholas Drinnan, and William Dunsmuir. "Bladder problems associated with neurological disease." InnovAiT: Education and inspiration for general practice 8, no. 5 (April 2, 2015): 291–97. http://dx.doi.org/10.1177/1755738015578935.

Full text

APA, Harvard, Vancouver, ISO, and other styles

32

EVANS, R. H. "Crohn's Disease Mimicking Primary Bladder Tumour." British Journal of Urology 65, no. 3 (March 1990): 299–300. http://dx.doi.org/10.1111/j.1464-410x.1990.tb14735.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Cuschieri, A. "Laparoscopic treatment of gall-bladder disease." Minimally Invasive Therapy 1, no. 2 (January 1992): 115–23. http://dx.doi.org/10.3109/13645709209152933.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Saito, Motoaki, and Ikuo Miyagawa. "Bladder Dysfunction due to Behçet’s Disease." Urologia Internationalis 65, no. 1 (2000): 40–42. http://dx.doi.org/10.1159/000064832.

Full text

APA, Harvard, Vancouver, ISO, and other styles

35

Evans, Claire Elizabeth, and Mark Peter Tighe. "Crohn's disease presenting as bladder mass." Archives of Disease in Childhood 99, no. 3 (December 13, 2013): 256. http://dx.doi.org/10.1136/archdischild-2013-305500.

Full text

APA, Harvard, Vancouver, ISO, and other styles

36

Lockhart, Jorge L., Bert Vorstman, David Weinstein, and Victor A. Politano. "Sphincterotomy Failure in Neurogenic Bladder Disease." Journal of Urology 135, no. 1 (January 1986): 86–89. http://dx.doi.org/10.1016/s0022-5347(17)45525-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Efstathiou, Jason A., Anthony L. Zietman, Donald S. Kaufman, Niall M. Heney, John J. Coen, and William U. Shipley. "Bladder-sparing approaches to invasive disease." World Journal of Urology 24, no. 5 (November 3, 2006): 517–29. http://dx.doi.org/10.1007/s00345-006-0114-y.

Full text

APA, Harvard, Vancouver, ISO, and other styles

38

Höglund, Mattias. "Bladder cancer, a two phased disease?" Seminars in Cancer Biology 17, no. 3 (June 2007): 225–32. http://dx.doi.org/10.1016/j.semcancer.2006.02.002.

Full text

APA, Harvard, Vancouver, ISO, and other styles

39

Sung, Bong Mo, Dong-Jin Oh, Moon Hee Choi, and Hye Min Choi. "Chronic kidney disease in neurogenic bladder." Nephrology 23, no. 3 (February 20, 2018): 231–36. http://dx.doi.org/10.1111/nep.12990.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Williamson, R. C. "Acalculous disease of the gall bladder." Gut 29, no. 6 (June 1, 1988): 860–72. http://dx.doi.org/10.1136/gut.29.6.860.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Saha, Sumona, Alessandro Fichera, Gregory Bales, Eugene Greenberg, and Sunanda Kane. "Metastatic Crohnʼs disease of the bladder." Inflammatory Bowel Diseases 14, no. 1 (January 2008): 140–42. http://dx.doi.org/10.1002/ibd.20254.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Winge, Kristian, and Kurt K. Nielsen. "Bladder dysfunction in advanced Parkinson's disease." Neurourology and Urodynamics 31, no. 8 (April 6, 2012): 1279–83. http://dx.doi.org/10.1002/nau.22237.

Full text

APA, Harvard, Vancouver, ISO, and other styles

43

Ryoo, Jae Wook, and Jae Min Cho. "Multiple bladder diverticula in Menkes disease." Pediatric Radiology 38, no. 5 (January 22, 2008): 595. http://dx.doi.org/10.1007/s00247-007-0745-1.

Full text

APA, Harvard, Vancouver, ISO, and other styles

44

Sharma, B. C., D. K. Agarwal, S. S. Baijal, T. S. Negi, G. Choudhuri, and V. A. Saraswat. "Effect of endoscopic sphincterotomy on gall bladder bile lithogenicity and motility." Gut 42, no. 2 (February 1, 1998): 288–92. http://dx.doi.org/10.1136/gut.42.2.288.

Full text

Abstract:

Background—Endoscopic sphincterotomy has been shown to inhibit stone formation in the gall bladder of experimental animals.Aims—To investigate the alterations in bile composition and gall bladder motility after endoscopic sphincterotomy.Patients—A study was performed of gall bladder bile composition and gall bladder motility in patients with gallstone disease ((n = 20; age 40–60 years, median age 55 years: seven men), with gall bladder calculi (n = 12) and with diseased gall bladder (chronic inflammation) without gall bladder calculi (n = 8)), who had received endoscopic sphincterotomy for common bile duct stones. Age and sex matched disease controls comprised 20 patients with gallstone disease but without stones and an intact sphincter of Oddi (with gall bladder calculi (n = 10) and diseased gall bladder without gall bladder calculi (n = 10)).Methods—Gall bladder motility was assessed by ultrasound. Duodenal bile collected by nasoduodenal tube after stimulation of gall bladder by intravenous ceruletid infusion was analysed for cholesterol, phospholipid, and bile acid concentrations, cholesterol saturation index, and nucleation time.Results—There was a significant reduction in mean (SEM) fasting volume (12.5 (1.7) ml v 26.4 (2.5) ml; p<0.001) and mean (SEM) residual volume (4.34 (0.9) ml v14.7 (0.98) ml; p<0.001), and increase in mean (SEM) ejection fraction (65.7 (4.2)% v 43.6 (5.52)%; p<0.001) and mean (SEM) rate constant of gall bladder emptying (−0.031/min v−0.020/min; p<0.01) in patients who had been subjected to endoscopic sphincterotomy. Median nucleation time was significantly longer (17 days v 6 days; p<0.006) in treated patients. There was a reduction in total mean (SEM) lipid concentrations (6.73 (0.32) g/dlv 7.72 (0.84) g/dl; p<0.05), cholesterol (5.6 (1.5) mmol/l v 10.3 (2.23) mmol/l; p<0.001) and CSI (0.72 (0.15) v 1.32 (0.31); p<0.001). There was no significant change in mean (SEM) phospholipid (25.6 (3.5) mmol/l v23.4 (6.28) mmol/l) and bile acid (93.7 (7.31) mmol/l v105.07 (16.6) mmol/l) concentrations.Conclusions—After endoscopic sphincterotomy there was enhanced contractility of the gall bladder, accompanied by a prolongation of nucleation time and reduction in cholesterol saturation index.

APA, Harvard, Vancouver, ISO, and other styles

45

Arora, Bhavinder K., Rachit Arora, and Akshit Arora. "Laparoscopic cholecystectomy in wall echo complex gall stone disease: a study." International Surgery Journal 4, no. 4 (March 25, 2017): 1309. http://dx.doi.org/10.18203/2349-2902.isj20171133.

Full text

Abstract:

Background: There are so many subtitles for difficult laproscopic cholecystectomy. Stone in the neck of gall bladder constitutes one of the entities. Wall echo complex is an ultrasound terminology used for cholelithiasis. It has three layers, first the pericholecystic fat between gallbladder and liver. Second layer consists of gall bladder wall. The third layer consists of echogenic stone itself. Wall echo complex is one of the entities which constitute difficult laproscopic cholecystectomy. Wall echo complex in the neck of the gall bladder is particularly difficult gall bladder where the conversion rates are high.Methods: The study was conducted in Department of Surgery. Standard four port cholecystectomy was done in 50 patients. Wall echo complex in all these patients was reported by ultrasonologist. Difficulties in operating wall echo complex cholelithiasis consisted of dissection of neck of gall bladder due to adhesions and a sleeve of fat covering the calot’s triangle. The difficulty of wall echo cholelithiasis was managed by opening the neck of gall bladder and evacuating the stones into a separate latex bag. By this procedure the difficult wall echo cholelithiasis was managed in all cases. Ligaclips were used for ligation of cystic duct, cystic artery and pericholecystic veins.Results: The evacuation of stones from the neck of gall bladder led to an easy cholecystectomy in 46 patients while the four patients had conversion to open cholecystectomy.Conclusions: Wall echo complex although an ultrasonologists entity but is a difficult gall bladder for laproscopic cholecystectomy.

APA, Harvard, Vancouver, ISO, and other styles

46

Parkin, Cameron James, George Acland, Ban Sulaiman, Mark Louie Johnsun, and Edward Latif. "Malakoplakia, a malignant mimic." Bladder 7, no. 3 (September 2, 2020): e44. http://dx.doi.org/10.14440/bladder.2020.818.

Full text

Abstract:

Malakoplakia is a chronic inflammatory condition that affects multiple systems, most commonly the urogenital tract. Its clinical presentation is often non-specific, but is typically characterized by recurrent urinary tract infections and haematuria. We report a rare case of intravesical malakoplakia mimicking an aggressive transitional cell carcinoma both in its clinical presentation and in its macroscopic appearance on cystoscopy in an 82-year patient, the oldest reported case in the literature. Malakoplakia has been described in the literature as a benign disease process presenting typically in younger patients. This case demonstrates its ability to cause obstructive uropathy and affect elderly patients. Thus, this case serves as a reminder to consider malakoplakia as a differential in the evaluation of suspected bladder malignancy in patients of all ages.

APA, Harvard, Vancouver, ISO, and other styles

47

Joseph, John, Aswin Kumar, Susan Mathews, Jagathnath K. M. Krishna, and Francis V. James. "Clinical outcome of muscle invasive carcinoma urinary bladder patients treated with bladder preservation protocol." International Journal of Research in Medical Sciences 9, no. 1 (December 28, 2020): 79. http://dx.doi.org/10.18203/2320-6012.ijrms20205656.

Full text

Abstract:

Background: To evaluate the outcome in terms of bladder preservation in muscle invasive urinary bladder patients treated with radical radiotherapy.Methods: A total of sixty patients with muscle invasive bladder cancer who were treated between 2011 and 2013 with bladder preservation protocol were identified. Thirty-two patients were stage T2 disease, rest were T3 and T4. Initial TURBT was reported complete in 24 of the total 60 patients. All patients received 60 to 64Gy to the whole bladder.Results: The follow-up range was 3 to 72 months. One year follow-up proportion was 78%. The median progression free survival was 40months. Early stage (T2) and advanced stage (T3 and T4) had progression free survival of 87.8% and 48.5% respectively at one year follow-up with a p value of 0.001. Thirty-six patients who had an incomplete Transurethral resection of bladder tumor (TURBT) initially had a statistically significant lower PFS compared to those patients who had a complete TURBT with a p value of 0.029. Twenty-nine patients were disease free with an intact bladder during the follow-up period providing a bladder preservation rate of 48%.Conclusions: A bladder-conserving protocol with radiotherapy provides encouraging results with nearly half of the patients able to retain a disease-free bladder. Proper patient selection with early stage disease and those with complete TURBT may further improve the bladder preservation rates.

APA, Harvard, Vancouver, ISO, and other styles

48

Cruz, Alejandro Gutierrez, Mafalda S. L. Aresta Branco, Brian A. Perrino, Kenton M. Sanders, and Violeta N. Mutafova-Yambolieva. "Urinary ATP Levels Are Controlled by Nucleotidases Released from the Urothelium in a Regulated Manner." Metabolites 13, no. 1 (December 24, 2022): 30. http://dx.doi.org/10.3390/metabo13010030.

Full text

Abstract:

Adenosine 5′-triphosphate (ATP) is released in the bladder lumen during filling. Urothelial ATP is presumed to regulate bladder excitability. Urinary ATP is suggested as a urinary biomarker of bladder dysfunctions since ATP is increased in the urine of patients with overactive bladder, interstitial cystitis or bladder pain syndrome. Altered urinary ATP might also be associated with voiding dysfunctions linked to disease states associated with metabolic syndrome. Extracellular ATP levels are determined by ATP release and ATP hydrolysis by membrane-bound and soluble nucleotidases (s-NTDs). It is currently unknown whether s-NTDs regulate urinary ATP. Using etheno-ATP substrate and HPLC-FLD detection techniques, we found that s-NTDs are released in the lumen of ex vivo mouse detrusor-free bladders. Capillary immunoelectrophoresis by ProteinSimple Wes determined that intraluminal solutions (ILS) collected at the end of filling contain ENTPD3 > ENPP1 > ENPP3 ≥ ENTPD2 = NT5E = ALPL/TNAP. Activation of adenylyl cyclase with forskolin increased luminal s-NTDs release whereas the AC inhibitor SQ22536 had no effect. In contrast, forskolin reduced and SQ22536 increased s-NTDs release in the lamina propria. Adenosine enhanced s-NTDs release and accelerated ATP hydrolysis in ILS and lamina propria. Therefore, there is a regulated release of s-NTDs in the bladder lumen during filling. Aberrant release or functions of urothelial s-NTDs might cause elevated urinary ATP in conditions with abnormal bladder excitability.

APA, Harvard, Vancouver, ISO, and other styles

49

Kachnic, L. A., D. S. Kaufman, N. M. Heney, A. F. Althausen, P. P. Griffin, A. L. Zietman, and W. U. Shipley. "Bladder preservation by combined modality therapy for invasive bladder cancer." Journal of Clinical Oncology 15, no. 3 (March 1997): 1022–29. http://dx.doi.org/10.1200/jco.1997.15.3.1022.

Full text

Abstract:

PURPOSE To update the efficacy of a selective multimodality bladder-preserving approach by transurethral resection (TURBT), systemic chemotherapy, and radiation therapy. PATIENTS AND METHODS From 1986 through 1993, 106 patients with muscle-invading clinical stage T2 to T4a,Nx,M0 bladder cancer were treated with induction by maximal TURBT and two cycles of chemotherapy (methotrexate, cisplatin, vinblastine [MCV]) followed by 39.6-Gy pelvic irradiation with concomitant cisplatin. Patients with a negative postinduction therapy tumor site biopsy and cytology (a T0 response, 70 patients) plus those with less than a T0 response but medically unfit for cystectomy (six patients), received consolidative chemoradiation to a total of 64.8 Gy. Surgical candidates with less than a T0 response (13 patients) and patients who could not tolerate the chemoradiation (six patients) went to immediate cystectomy. The median follow-up duration is 4.4 years. RESULTS The 5-year actuarial overall survival and disease-specific survival rates of all patients are 52% and 60%, respectively. For clinical stage T2 patients, the actuarial overall survival rate is 63%, and for T3-4, 45%. Thirty-six patients (34%) underwent cystectomy, all with evidence of tumor activity, including 17 with an invasive recurrence. The 5-year overall survival rate with an intact functioning bladder is 43%. Among 76 patients who completed bladder-preserving therapy, the 5-year rate of freedom from an invasive bladder relapse is 79%. No patient required cystectomy for treatment-related bladder morbidity. CONCLUSION Combined modality therapy with TURBT, chemotherapy, radiation, and selection for organ-conservation by response has a 52% overall survival rate. This result is similar to cystectomy-based studies for patients of similar age and clinical stages. The majority of the long-term survivors retain fully functional bladders.

APA, Harvard, Vancouver, ISO, and other styles

50

Fabiano, Marco, Alfonso Califano, Francesco Chiancone, Antonio D’Antonio, Francesco Maiorino, Davide Simeone, Gianmarco Silvestre, and Vincenzo Altieri. "Bladder schistosomiasis in Italy: A case report." Urologia Journal 87, no. 4 (March 5, 2020): 191–93. http://dx.doi.org/10.1177/0391560320910647.

Full text

Abstract:

Introduction: Human schistosomiasis is a snail-borne disease caused by parasitic blood-dwelling flukes. A long-term infection can lead to the risk of liver damage, kidney failure, infertility, or bladder cancer. The most common sign is hematuria with the blood first seen in the terminal urine, but in severe cases the whole urine sample can be dark colored. We analyze the case of a healthy African child living in Italy since birth, harboring a hidden debilitating disease that was picked up during ultrasonography. Case Report: A 11-year-old African child was admitted to our emergency department with macroscopic hematuria, dysuria, and frequency for 2 months. Ultrasonography revealed a solid mass involving bladder’s right wall. Non-contrast and contrast-enhanced scans of computerized tomography showed a mass of 45 mm x 15 mm on the right bladder wall. A bipolar transurethral resection of bladder was performed. The pathological examination showed findings consistent with Schistosoma haematobium. Discussion: The clinical manifestations of schistosomiasis depend on the inflammatory response to the parasitic infection. In particular, it can manifest in the bladder as painless dysuria, urinary incontinence and urinary frequency, hematuria, or even urinary retention if the trigone is involved. Utilization of ultrasonography for diagnostic evaluation of schistosomiasis is mandatory. For treatment, the World Health Organization recommends praziquantel which has an efficacy of up to 90%.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!