Relevant bibliographies by topics / Biomechanics of articular cartilage

Academic literature on the topic 'Biomechanics of articular cartilage'

Author: Grafiati
Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Biomechanics of articular cartilage"

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June, R. K., and D. P. Fyhrie. "Temperature effects in articular cartilage biomechanics." Journal of Experimental Biology 213, no. 22 (October 29, 2010): 3934–40. http://dx.doi.org/10.1242/jeb.042960.

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Takashima, Tatsuki, Yoshitaka Nakanishi, and Hidehiko Higaki. "Material and Wear Characteristics of Artificial Articular Cartilage(Orthopaedic Biomechanics)." Proceedings of the Asian Pacific Conference on Biomechanics : emerging science and technology in biomechanics 2004.1 (2004): 171–72. http://dx.doi.org/10.1299/jsmeapbio.2004.1.171.

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Reiter, Mary Pat, Shawn H. Ward, Barbara Perry, Adrian Mann, Joseph W. Freeman, and Moti L. Tiku. "Intra-articular injection of epigallocatechin (EGCG) crosslinks and alters biomechanical properties of articular cartilage, a study via nanoindentation." PLOS ONE 17, no. 10 (October 25, 2022): e0276626. http://dx.doi.org/10.1371/journal.pone.0276626.

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Osteoarthritis and rheumatoid arthritis are debilitating conditions, affecting millions of people. Both osteoarthritis and rheumatoid arthritis degrade the articular cartilage (AC) at the ends of long bones, resulting in weakened tissue prone to further damage. This degradation impairs the cartilage’s mechanical properties leading to areas of thinned cartilage and exposed bone which compromises the integrity of the joint. No preventative measures exist for joint destruction. Discovering a way to slow the degradation of AC or prevent it would slow the painful progression of the disease, allowing millions to live pain-free. Recently, that the articular injection of the polyphenol epigallocatechin-gallate (EGCG) slows AC damage in an arthritis rat model. It was suggested that EGCG crosslinks AC and makes it resistant to degradation. However, direct evidence that intraarticular injection of EGCG crosslinks cartilage collagen and changes its compressive properties are not known. The aim of this study was to investigate the effects of intraarticular injection of EGCG induced biomechanical properties of AC. We hypothesize that in vivo exposure EGCG will bind and crosslink to AC collagen and alter its biomechanical properties. We developed a technique of nano-indentation to investigate articular cartilage properties by measuring cartilage compressive properties and quantifying differences due to EGCG exposure. In this study, the rat knee joint was subjected to a series of intraarticular injections of EGCG and contralateral knee joint was injected with saline. After the injections animals were sacrificed, and the knees were removed and tested in an anatomically relevant model of nanoindentation. All mechanical data was normalized to the measurements in the contralateral knee to better compare data between the animals. The data demonstrated significant increases for reduced elastic modulus (57.5%), hardness (83.2%), and stiffness (17.6%) in cartilage treated with injections of EGCG normalized to those treated with just saline solution when compared to baseline subjects without injections, with a significance level of alpha = 0.05. This data provides evidence that EGCG treated cartilage yields a strengthened cartilage matrix as compared to AC from the saline injected knees. These findings are significant because the increase in cartilage biomechanics will translate into resistance to degradation in arthritis. Furthermore, the data suggest for the first time that it is possible to strengthen the articular cartilage by intraarticular injections of polyphenols. Although this data is preliminary, it suggests that clinical applications of EGCG treated cartilage could yield strengthened tissue with the potential to resist or compensate for matrix degradation caused by arthritis.
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Hartmann, Bastian, Gabriele Marchi, Paolo Alberton, Zsuzsanna Farkas, Attila Aszodi, Johannes Roths, and Hauke Clausen-Schaumann. "Early Detection of Cartilage Degeneration: A Comparison of Histology, Fiber Bragg Grating-Based Micro-Indentation, and Atomic Force Microscopy-Based Nano-Indentation." International Journal of Molecular Sciences 21, no. 19 (October 6, 2020): 7384. http://dx.doi.org/10.3390/ijms21197384.

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We have determined the sensitivity and detection limit of a new fiber Bragg grating (FBG)-based optoelectronic micro-indenter for biomechanical testing of cartilage and compared the results to indentation-type atomic force microscopy (IT-AFM) and histological staining. As test samples, we used bovine articular cartilage, which was enzymatically degraded ex vivo for five minutes using different concentrations of collagenase (5, 50, 100 and 500 µg/mL) to mimic moderate extracellular matrix deterioration seen in early-stage osteoarthritis (OA). Picrosirius Red staining and polarization microscopy demonstrated gradual, concentration-dependent disorganization of the collagen fibrillar network in the superficial zone of the explants. Osteoarthritis Research Society International (OARSI) grading of histopathological changes did not discriminate between undigested and enzymatically degraded explants. IT-AFM was the most sensitive method for detecting minute changes in cartilage biomechanics induced by the lowest collagenase concentration, however, it did not distinguish different levels of cartilage degeneration for collagenase concentrations higher than 5 µg/mL. The FBG micro-indenter provided a better and more precise assessment of the level of cartilage degeneration than the OARSI histological grading system but it was less sensitive at detecting mechanical changes than IT-AFM. The FBG-sensor allowed us to observe differences in cartilage biomechanics for collagenase concentrations of 100 and 500 µg/mL. Our results confirm that the FBG sensor is capable of detecting small changes in articular cartilage stiffness, which may be associated with initial cartilage degeneration caused by early OA.
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He, Chuan, Wu He, Fuke Wang, Lu Tong, Zhengguang Zhang, Di Jia, Guoliang Wang, Jiali Zheng, Guangchao Chen, and Yanlin Li. "Biomechanics of Knee Joints after Anterior Cruciate Ligament Reconstruction." Journal of Knee Surgery 31, no. 04 (June 30, 2017): 352–58. http://dx.doi.org/10.1055/s-0037-1603799.

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AbstractThis study aimed to investigate the biomechanical properties of anterior cruciate ligament (ACL); tibial, femoral articular cartilage; and meniscus in knee joints receiving computer-aided or conventional ACL reconstruction. Three-dimensional (3D) knee joint finite element models were established for healthy volunteers (normal group) and patients receiving computer-aided surgery (CAS) or conventional (traditional surgery [TS]) ACL reconstruction. The stress and stress distribution on the ACL, tibial, femoral articular cartilage, and meniscus were examined after force was applied on the 3D knee joint finite element models. No significant differences were observed in the stress on ACL among normal group, CAS group, and TS group when a femoral backward force was loaded. However, when a vertical force of 350 N was loaded on the knee joints, TS group had significant higher stress on the articular cartilage and meniscus than the other two groups at any flexion angle of 0, 30, 60, and 90 degrees. However, no significant differences were observed between CAS group and normal group. In conclusion, computer-aided ACL reconstruction has advantages over conventional surgery approach in restoring the biomechanical properties of knee joints, thus reducing the risk of damage to the knee joint cartilage and meniscus after ACL reconstruction.
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Sergienko, R. A., S. S. Strafun, S. I. Savosko, and A. M. Makarenko. "Analysis of the dynamics of the structural changes development in the humerus of guinea pigs under modeling biomechanical disturbances." Reports of Morphology 25, no. 3 (September 19, 2019): 33–39. http://dx.doi.org/10.31393/morphology-journal-2019-25(3)-06.

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Today, the role of the traumatic factor and inflammation in the development and progression of osteoarthrosis is generally recognized, but the available research results do not allow to establish the role of impaired biomechanics as a monofactor in the development of deforming ostearthrosis of the shoulder joint. Violation of the function of the bone and bone-cartilage elements of the joint, which is compensated by soft tissue formations, leads to overloads of the joints, upsets the normal balance of the load forces in the joint, creates abnormal biomechanics and the resulting pathological manifestations of deforming osteoarthrosis. The aim of the study is research of the dynamics of the disturbed biomechanics influence of the shoulder joint on the development of deformation osteoarthrosis and the features of the development of its structural changes. The experiments were conducted on guinea pigs weighing 380-420 grams at the age of 5 months. A model of surgical restriction of joint mobility was reproduced, which caused the formation of contracture. Using the methods of histology and scanning electron microscopy, we studied the relief of the articular surface, the topography of degenerative changes, and structural changes in the articular cartilage and subchondral bone. A statistical evaluation of the obtained data samples was carried out using Student t-test. The results were considered reliable at р<0.05. The results of an experimental study demonstrated a decrease in the thickness and structure of articular cartilage when modeling deforming osteoarthrosis and confirmed the hypothesis that pathological limitation of the mobility of the shoulder joint and violation of biomechanics is an independent factor in the formation of osteoarthrosis. After surgery on day 30, degenerative changes and their progression with the formation of contracture on day 90 of observation were found in the articular cartilage. The features of the development of articular surface degeneration, the dynamics of the pathological changes and topography, which can expand the understanding of the pathogenesis of the disease, were established. The loss of the superficial zone caused the progression of dystrophic changes in the articular cartilage and sclerosis of the subchondral bone at 60 and 90 days.
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PENG, XIONGQI, GENG LIU, and ZAOYANG GUO. "FINITE ELEMENT CONTACT ANALYSIS OF A HUMAN SAGITTAL KNEE JOINT." Journal of Mechanics in Medicine and Biology 10, no. 02 (June 2010): 225–36. http://dx.doi.org/10.1142/s0219519410003423.

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Articular cartilage is a vital component of human knee joints by providing a low-friction and wear-resistant surface in knee joints and distributing stresses to tibia. The degeneration or damage of articular cartilage will incur acute pain on the human knee joints. Hence, to understand the mechanism of normal and pathological functions of articular cartilage, it is very important to investigate the contact mechanics of the human knee joints. Experimental research has difficulties in reproducing the physiological conditions of daily activities and measuring the key factors such as contact-stress distributions inside knee joint without violating the physiological environment. On the other hand, numerical approaches such as finite element (FE) analysis provide a powerful tool in the biomechanics study of the human knee joint. This article presents a two-dimensional (2D) FE model of the human knee joints that includes the femur, tibia, patella, quadriceps, patellar tendon, and cartilages. The model is analyzed with dynamic loadings to study stress distribution in the tibia and contact area during contact with or without articular cartilage. The results obtained in this article are very helpful to find the pathological mechanism of knee joint degeneration or damage, and thus guide the therapy of knee illness and artificial joint replacement.
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Wilk, Kevin E., Leonard C. Macrina, and Michael M. Reinold. "Rehabilitation following Microfracture of the Knee." CARTILAGE 1, no. 2 (March 19, 2010): 96–107. http://dx.doi.org/10.1177/1947603510366029.

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Postoperative rehabilitation programs following articular cartilage repair procedures will vary greatly among patients and need to be individualized based on the nature of the lesion, the unique characteristics of the patient, and the type and detail of each surgical procedure. These programs are based on knowledge of the basic science, anatomy, and biomechanics of articular cartilage as well as the biological course of healing following surgery. The goal is to restore full function in each patient as quickly as possible by facilitating a healing response without overloading the healing articular cartilage. The purpose of this article is to overview the principles of rehabilitation following microfracture procedures of the knee.
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Eschweiler, Joerg, Nils Horn, Bjoern Rath, Marcel Betsch, Alice Baroncini, Markus Tingart, and Filippo Migliorini. "The Biomechanics of Cartilage—An Overview." Life 11, no. 4 (April 1, 2021): 302. http://dx.doi.org/10.3390/life11040302.

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Articular cartilage (AC) sheathes joint surfaces and minimizes friction in diarthrosis. The resident cell population, chondrocytes, are surrounded by an extracellular matrix and a multitude of proteins, which bestow their unique characteristics. AC is characterized by a zonal composition (superficial (tangential) zone, middle (transitional) zone, deep zone, calcified zone) with different mechanical properties. An overview is given about different testing (load tests) methods as well as different modeling approaches. The widely accepted biomechanical test methods, e.g., the indentation analysis, are summarized and discussed. A description of the biphasic theory is also shown. This is required to understand how interstitial water contributes toward the viscoelastic behavior of AC. Furthermore, a short introduction to a more complex model is given.
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Marks, Ray. "Osteoarthritis and Articular Cartilage: Biomechanics and Novel Treatment Paradigms." Advances in Aging Research 03, no. 04 (2014): 297–309. http://dx.doi.org/10.4236/aar.2014.34039.

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Dissertations / Theses on the topic "Biomechanics of articular cartilage"

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Gratz, Kenneth R. "Biomechanics of articular cartilage defects." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2007. http://wwwlib.umi.com/cr/ucsd/fullcit?p3284116.

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Thesis (Ph. D.)--University of California, San Diego, 2007.
Title from first page of PDF file (viewed January 9, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
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Olsen, Sigb. "Modelling of articular cartilage load-carriage biomechanics." Thesis, Queensland University of Technology, 2003.

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Kerin, Alexander James. "The mechanical failure of articular cartilage." Thesis, University of Bristol, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265315.

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Stewart, Kevin Matthew. "MECHANICAL SIMULATION OF ARTICULAR CARTILAGE BASED ON EXPERIMENTAL RESULTS." DigitalCommons@CalPoly, 2009. https://digitalcommons.calpoly.edu/theses/93.

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Recently, a constituent based cartilage growth finite element model (CGFEM) was developed in order to predict articular cartilage (AC) biomechanical properties before and after growth. Previous research has noted limitations in the CGFEM such as model convergence with growth periods greater than 12 days. The main aims of this work were to address these limitations through (1) implementation of an exact material Jacobian matrix definition using the Jaumann-Kirchhoff (J-K) method and (2) quantification of elastic material parameters based upon research findings of the Cal Poly Cartilage Biomechanics Group (CPGBG). The J-K method was successfully implemented into the CGFEM and exceeded the maximum convergence strains for both the “pushed forward, then differentiated” (PFD) and “differentiated, then pushed forward” (DPF) methods, while maintaining correct material stress responses. Elastic parameters were optimized for confined compression (CC), unconfined compression (UCC), and uniaxial tension (UT) protocols. This work increases the robustness of the CGFEM through the J-K method, as well as defines an accurate starting point for AC growth based on the optimized material parameters.
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Wong, Benjamin L. "Biomechanics of cartilage articulation effects of degeneration, lubrication, and focal articular defects /." Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p3356137.

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Thesis (Ph. D.)--University of California, San Diego, 2009.
Title from first page of PDF file (viewed June 15, 2009). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
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Kashani, Jamal. "An innovative agent-based cellular automata framework for simulating articular cartilage biomechanics." Thesis, Queensland University of Technology, 2017. https://eprints.qut.edu.au/107203/1/Jamal_Kashani_Thesis.pdf.

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Mammalian tissues and organs are complex in structure and function and can degenerate because of diseases. It is not possible to study in full their characteristics with traditional laboratory methods making it difficult to extend our knowledge of tissue function in vivo. This thesis presents a novel computational cellular automata agent and new rules of interaction that can simulate behaviours within and outside such biological components in computational simulations. The new computational methodology was applied to articular cartilage to simulate its complex porous single-phase osmosis-governed structure and characteristics. The results demonstrate that this new computational agent can be used to study other single-phase multi-component materials.
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Motavalli, Sayyed Mostafa. "DEPTH-DEPENDENT BIAXIAL MECHANICAL BEHAVIOR OF NATIVE AND TISSUE ENGINEERING ARTICULAR CARTILAGE." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1390313405.

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Arabshahi, Zohreh. "New mechanical indentation framework for functional assessment of articular cartilage." Thesis, Queensland University of Technology, 2018. https://eprints.qut.edu.au/119696/1/Zohreh_Arabshahi_Thesis.pdf.

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In this research, two new mechanical indentation frameworks were established where the two different indenters (cylindrical and ring-shaped flat-ended indenters) were integrated with ultrasound for assessing functional properties of articular cartilage during loading/unloading. The aim of establishing these framewoks was to address some of the limitations to the conventional indentation techniques. Two new parameters within these frameworks were developed and their capacity to distinguish between normal and different types of cartilage degeneration models during deformation/recovery, was investigated. The ring-shaped flat-ended indenter, integrated with an ultrasound transducer, was shown to be capable of distinguishing normal from artificially degraded bovine osteochondral samples.
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Schöne, Martin. "Possibilities of Articular Cartilage Quantification Based on High-Frequency Ultrasound Scans and Ultrasound Palpation." Doctoral thesis, Humboldt-Universität zu Berlin, 2020. http://dx.doi.org/10.18452/21781.

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In der Diagnostik und Reparatur von hyalinem Gelenkknorpel sind neue Methoden zur Quantifizierung von Struktur und mechanischer Belastbarkeit gefragt, um die Behandlung von Knorpelschäden an Millionen von Patienten weltweit zu verbessern. Mittels hochfrequentem, fokussierten Ultraschall werden Oberflächenparameter für Reflektivität und Rauheit an Gelenkknorpel bestimmt. Es wird gezeigt wie die Oberflächenneigung kontrolliert werden kann. Die Ergebnisse vermitteln ein besseres Verständnis über die Zusammensetzung der Ultraschallsignale aus reflektierten und gestreuten Komponenten. 3D Ultraschallscans von Knorpelregeneraten erlauben die Defektstellen volumetrisch zu Quantifizieren. Die Proben wurden zusätzlich nach etablierten Bewertungssystemen benotet, welche auf makroskopischer Beurteilungen, MRT-Scans und Histologie basieren. Die ultraschallbasierten Volumendaten zeigten dabei gute Korrelationen mit den Punktwertungen. Die im Labor verwendeten Messaufbauten zur biomechanischen Charakterisierung von Gelenkknorpel können am Patienten nicht angewandt werden. Daher können Ärzte die Festigkeit von Knorpel bisher nur mittels manueller Palpation abschätzen. Diese Arbeit entwickelt eine Methode der Ultraschall-Palpation (USP), die es erlaubt, die während der manuellen Palpation erzeugte Kraft und Deformation, basierend auf Ultraschallechos, aufzunehmen. Es wurde einen Prototyp entwickelt womit gezeigt werden konnte, dass USP eine ausreichende Genauigkeit und Reproduzierbarkeit aufweist. Wiederholte Messungen können zusätzlich zeitabhängige biomechanische Parameter von Knorpel ableiten. Zusammenfassend zeigt diese Arbeit verbesserte und neue Möglichkeiten zur strukturellen und biomechanischen Charakterisierung von hyalinem Gelenkknorpel bzw. den Ergebnissen von Knorpelreparatur basierend auf Ultraschalldaten. Diese Methoden haben das Potenzial die Diagnostik von Gelenkknorpel und die Quantifizierung von Knorpelreparatur zu verbessern.
In the diagnostics and repair of hyaline articular cartilage, new methods to quantify structure and mechanical capacity are required to improve the treatment of cartilage defects for millions of patients worldwide. This thesis uses high frequency focused ultrasound to derive surface parameters for reflectivity and roughness from articular cartilage. It is shown how to control the inclination dependency to gain more reliable results. Furthermore, the results provided a better understanding of the composition of ultrasonic signals from reflected and scattered components. 3D ultrasound scans of cartilage repair tissue were performed to quantify defect sites after cartilage repair volumetrically. The samples were also graded according to established scoring systems based on macroscopic evaluation, MRI scans and histology. The ultrasound-based volumetric parameters showed good correlation with these scores. Complex biomechanical measurement setups used in laboratories cannot be applied to the patient. Therefore, currently physicians have to estimate the stiffness of cartilage by means of manual palpation. In the last part of this thesis, a method denoted as ultrasound palpation is developed, which allows for measuring the applied force and strain during manual palpation in real time, solely based on the evaluation of the time of flight of ultrasound pulses. A prototype was developed and its measurement accuracy and reproducibility were characterized. It could be shown that ultrasound palpation has sufficient accuracy and reproducibility. Additionally, by repeated measurements it was possible to derive time-dependent biomechanical parameters of cartilage. In summary, this work shows improved and new possibilities for structural and biomechanical characterization of hyaline articular cartilage and the outcomes of cartilage repair based on ultrasound data. The methods have the potential to improve the diagnostics of articular cartilage and quantification of its repair.
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Mouw, Janna Kay. "Mechanoregulation of chondrocytes and chondroprogenitors the role of TGF-BETA and SMAD signaling /." Diss., Available online, Georgia Institute of Technology, 2005, 2005. http://etd.gatech.edu/theses/available/etd-11232005-103041/.

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Thesis (Ph. D.)--Bioengineering, Georgia Institute of Technology, 2006.
Harish Radhakrishna, Committee Member ; Christopher Jacobs, Committee Member ; Andres Garcia, Committee Member ; Marc E. Levenston, Committee Chair ; Barbara Boyan, Committee Member.
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Books on the topic "Biomechanics of articular cartilage"

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E, Kuettner Klaus, Schleyerbach Rudolf, and Hascall Vincent C, eds. Articular cartilage biochemistry. New York: Raven Press, 1986.

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Smith, David W., Bruce S. Gardiner, Lihai Zhang, and Alan J. Grodzinsky. Articular Cartilage Dynamics. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-1474-2.

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Cole, Brian J., and M. Mike Malek. Articular Cartilage Lesions. New York, NY: Springer New York, 2004. http://dx.doi.org/10.1007/978-0-387-21553-2.

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Athanasiou, K. A. Articular cartilage tissue engineering. San Rafael, Calif. (1537 Fourth Street, San Rafael, CA 94901 USA): Morgan & Claypool Publishers, 2010.

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Gahunia, Harpal K., Allan E. Gross, Kenneth P. H. Pritzker, Paul S. Babyn, and Lucas Murnaghan, eds. Articular Cartilage of the Knee. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-7587-7.

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Argatov, Ivan, and Gennady Mishuris. Contact Mechanics of Articular Cartilage Layers. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-20083-5.

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Rodrìguez-Merchán, E. Carlos, ed. Articular Cartilage Defects of the Knee. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2727-5.

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1964-, Hendrich Christian, Nöth Ulrich 1967-, and Eulert Jochen, eds. Cartilage surgery and future perspectives. Berlin: Springer, 2003.

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Cartilage tympanoplasty. Stuttgart: Thieme, 2009.

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Shakibaei, Mehdi, Constanze Csaki, and Ali Mobasheri. Diverse Roles of Integrin Receptors in Articular Cartilage. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-78771-6.

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Book chapters on the topic "Biomechanics of articular cartilage"

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Ferretti, Mário, Lauro Augusto Veloso Costa, and Noel Oizerovici Foni. "Articular Cartilage: Functional Biomechanics." In Cartilage Injury of the Knee, 1–9. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-78051-7_1.

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Wong, M., and E. B. Hunziker. "Articular Cartilage Biology and Biomechanics." In Gelenkknorpeldefekte, 15–28. Heidelberg: Steinkopff, 2001. http://dx.doi.org/10.1007/978-3-642-57716-1_2.

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Oka, M., and Y. Kotoura. "Mechanical Properties of the Articular Cartilage." In Biomechanics: Basic and Applied Research, 279–84. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3355-2_36.

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Haider, Mansoor A., Brandy A. Benedict, Eunjung Kim, and Farshid Guilak. "Computational Modeling of Cell Mechanics in Articular Cartilage." In Computational Modeling in Biomechanics, 329–52. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3575-2_11.

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Torzilli, P. A., E. Askari, and J. T. Jenkins. "Water Content and Solute Diffusion Properties in Articular Cartilage." In Biomechanics of Diarthrodial Joints, 363–90. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3448-7_13.

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Hou, J. S., M. H. Holmes, W. M. Lai, and V. C. Mow. "Squeeze Film Lubrication for Articular Cartilage with Synovial Fluid." In Biomechanics of Diarthrodial Joints, 347–67. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3450-0_14.

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Semitela, Â., J. Couto, A. Capitão, A. F. Mendes, P. A. A. P. Marques, and A. Completo. "Bilayered arcade-like scaffolds for articular cartilage repair." In Advances and Current Trends in Biomechanics, 182–86. London: CRC Press, 2021. http://dx.doi.org/10.1201/9781003217152-41.

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Hart, J. A. L., and R. K. Miller. "Articular Cartilage: Biomechanics, Injury, and Surgical Treatment of Defects." In Biomechanics and Biomaterials in Orthopedics, 229–46. London: Springer London, 2004. http://dx.doi.org/10.1007/978-1-4471-3774-0_24.

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Jacob, George, Kazunori Shimomura, David A. Hart, Hiromichi Fujie, and Norimasa Nakamura. "Mechanical and Biologic Properties of Articular Cartilage Repair Biomaterials." In Orthopaedic Biomechanics in Sports Medicine, 57–71. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81549-3_5.

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Semitela, Â., A. L. Pereira, A. Capitão, A. F. Mendes, P. A. A. P. Marques, and A. Completo. "Automated fabrication of 3D chondrocyte-laden anisotropic scaffolds for articular cartilage." In Advances and Current Trends in Biomechanics, 187–91. London: CRC Press, 2021. http://dx.doi.org/10.1201/9781003217152-42.

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Conference papers on the topic "Biomechanics of articular cartilage"

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Murakami, Teruo, Nobuo Sakai, Yoshinori Sawae, Itaru Ishikawa, Natsuko Hosoda, Emiko Suzuki, and Jun Honda. "Biomechanical Aspects of Natural Articular Cartilage and Regenerated Cartilage." In In Commemoration of the 1st Asian Biomaterials Congress. WORLD SCIENTIFIC, 2008. http://dx.doi.org/10.1142/9789812835758_0028.

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Blackburn, Brecken J., Mostafa Motavalli, Matthew R. Ford, Jean F. Welter, Joseph M. Mansour, and Andrew M. Rollins. "Ex vivo measurement of biaxial strain distribution in articular cartilage with optical coherence tomography (Conference Presentation)." In Optical Elastography and Tissue Biomechanics VI, edited by Kirill V. Larin and Giuliano Scarcelli. SPIE, 2019. http://dx.doi.org/10.1117/12.2509681.

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Travascio, Francesco, Roberto Serpieri, and Shihab Asfour. "Articular Cartilage Biomechanics Modeled via an Intrinsically Compressible Biphasic Model: Implications and Deviations From an Incompressible Biphasic Approach." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14082.

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Biphasic continuum models have been extensively deployed for modeling macroscopic articular cartilage biomechanics [1,2]. This consolidated theoretical approach schematizes tissue as a mixture of an elastic solid matrix embedded in a fluid phase. In physiological conditions, intrinsic compressibility of each phase is very limited when compared to the whole tissue macroscopic counterpart. Based on such experimental evidence, intrinsic phase compressibility is generally reasonably neglected [3]. Hence, traditionally, cartilage biomechanics models have been developed on the basis of incompressible biphasic formulations [3–5], often referred to as Incompressible Theories of Mixtures (ITM). Alternatively, a more general biphasic model for cartilage biomechanics, accounting for full intrinsic compressibility of phases, may be considered. A consistent theoretical formulation of this type has been recently made available [6,7], hereby referred to as Theory of Microscopically Compressible Porous Media (TMCPM). In the present contribution, a new model for articular cartilage biomechanics, based on TMCPM, was developed. Predictions of this new model, and its deviations from a traditional ITM approach were studied. In particular, deviations between compressible and incompressible theoretical frameworks were investigated with a specific focus on the repercussions on models’ capability of characterizing fundamental tissue properties, such as hydraulic permeability, via established experimental testing procedures.
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Haut, Tammy L., Maury L. Hull, and Stephen M. Howell. "A High Accuracy Three-Dimensional Coordinate Digitizing System for Reconstructing the Geometry of Diarthrodial Joints." In ASME 1997 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-0206.

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Abstract Finite element modeling (FEM) in orthopaedic biomechanics requires a measurement technique to obtain accurate, three-dimensional (3-D) representations of diarthrodial joints [5], Accurate representations of joints, including soft tissue, articular cartilage, and bone are needed for FEM to be useful in the study of joint behavior and orthopaedic implants. Based on measuring the thickness of articular cartilage, the accuracy requirement for the measurement technique is 20 μm for a relative error of 1 percent.
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Petrtyl, M., J. Danesova, J. Lisal, J. Sejkotova, Jane W. Z. Lu, Andrew Y. T. Leung, Vai Pan Iu, and Kai Meng Mok. "Biomechanical Properties of Peripheral Layer in Articular Cartilage." In PROCEEDINGS OF THE 2ND INTERNATIONAL SYMPOSIUM ON COMPUTATIONAL MECHANICS AND THE 12TH INTERNATIONAL CONFERENCE ON THE ENHANCEMENT AND PROMOTION OF COMPUTATIONAL METHODS IN ENGINEERING AND SCIENCE. AIP, 2010. http://dx.doi.org/10.1063/1.3452080.

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Batista, Michael, Hadi T. Nia, Karen Cox, Christine Ortiz, Alan J. Grodzinsky, Dick Heinegård, and Lin Han. "Effects of Chondroadherin on Cartilage Nanostructure and Biomechanics via Murine Model." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14516.

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While small leucine rich proteins/proteoglycans (SLRPs) are present in very low concentrations in the extracellular matrix (ECM), they have been shown to be critical determinants of the proper ECM assembly and function in connective tissues [1] including bone [2], cornea [3], and cartilage [4]. However, their direct and indirect roles in matrix biomechanics and the potential for osteoarthritis-related dysfunction of cartilage remain unclear. With the advent of new high resolution nanotechnological tools, the direct quantification of cartilage biomechanical properties using murine models can provide important insights into how secondary ECM molecules, such as SLRPs, affect the function and pathology of cartilage [5]. Previous nanoindentation studies of murine cartilage have assessed the effects of maturation and osteoarthritis-like degradation of cartilage on its biomechanical properties [6, 7]. Recently, murine models have received increased attention because of the availability of specific gene-knockout and gene alteration technologies [8]. For example, chondroadherin (CHAD) is a non-collagenous small leucine-rich proteoglycan (SLRP) with α-helix and β-sheet secondary structure, spatially localized in the territorial matrix (MW = 38 kDa) [9]. In articular cartilage, CHAD is distributed non-uniformly with depth [10], and binds to type II collagen and the α2β1 integrin and is hypothesized to function in the communication between chondrocytes and their surrounding matrix, as well as in the regulation of collagen fibril assembly [11, 12] (Fig. 1). The objective of the present study is to explore the role of CHAD and its depletion on the structure and nanomechanical properties of both superficial and middle/deep zone cartilage. The current methods thereby enabled depth-dependent analysis of cartilage nanostructure and dynamic energy-dissipative mechanisms.
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Vahdati, Ali, and Diane R. Wagner. "Influence of Calcified Cartilage Zone Permeability in Mechanical Behavior of Articular Cartilage: A Finite Element Study." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206512.

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Articular cartilage (AC) disease and especially osteoarthrithis (OA) are debilitating conditions that are associated with huge social and economic burdens. To understand the factors involved in initiation and progression of OA, the mechanical state of the cartilage tissue must be first understood [1]. Biphasic and triphasic models developed by Mow and coworkers relate AC structure with its mechanical behavior and provided researchers with valuable models for AC biomechanics [2, 3]. Although much is known about AC and its mechanical properties, the zone of calcified cartilage (ZCC) has been sparsely studied. ZCC is very thin and highly interdigitated with subchondral bone (SB) which makes it very difficult to isolate for independent study [4]. It is well known that SB plays an important role in both initiation and/or progression of OA [5], thus ZCC may also be an important player in the pathology of the disease [6]. A few studies have investigated mechanical properties of ZCC, but conflicting results have been published on ZCC permeability. Although ZCC has been mainly assumed to be impermeable [7], recently Hwang et al. [8] suggested that ZCC may have even higher permeability than cartilage itself. We studied the effect of ZCC permeability on mechanical behavior of AC using a finite element (FE) model.
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Pottle, Jonathan E., and J. K. Francis Suh. "An in Situ Dual Indentation and Optimization Method to Determine Mechanical Properties of Articular Cartilage." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176601.

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The efficacy of the biphasic poroviscoelastic (BPVE) theory [1] in constitutive modeling of articular cartilage biomechanics is well-established [2–4]. Indeed, this model has been used to simultaneously predict stress relaxation force across confined compression, unconfined compression, and indentation protocols [2,3]. Previous works have also demonstrated success in simultaneously curve-fitting the BPVE model to reaction force and lateral deformation data gathered from stress relaxation tests of articular cartilage in unconfined compression [4]. However, a potential limitation of practical applications of such a successful model is seen in some commonly-employed mechanical testing methods for articular cartilage, such as confined compression and unconfined compression. These methods require the excision of a disk of cartilage from its underlying subchondral base, which likely would compromise the structural integrity of the tissue, causing swelling and curling artifacts of the sample [5]. Indentation represents a testing protocol that can be used with an intact cartilage layer. This results in a specimen more closely resembling cartilage in vivo. Using an agarose gel construct, our previous study [6] has demonstrated that a unique set of the six BPVE model parameters of a soft tissue can be determined readily from in situ dual indentation method using stress relaxation and creep viscoelastic protocols. The objective of the current study is to validate the efficacy of this technique as a means to determine the BPVE material parameters of articular cartilage.
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Manda, Krishnagoud, and Anders Eriksson. "Simulating Metal Implants in Full Thickness Cartilage Defects." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53235.

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Damage or degeneration in the articular cartilage is a major problem that affects millions of people in the world. The biomechanical forces at a site of damage in the cartilage may make the tissue more susceptible to continued long-term degeneration. Various biological treatments are currently available, but all have drawbacks. Alternatively, a contoured articular resurfacing implant is developed to offer a treatment to such full thickness chondral defects [1,3,4]. The main goal of using metal implants, to fill the degenerated portion of the cartilage, is to seal the surrounding cartilage so that further damage can be prevented, and to re-establish the integrity of the joint articulating surface. Many researchers have studied the safety, feasibility and reliability of the metal implants in animal models from a biological point of view [3,4]. They showed promising results. Till date, the mechanical behavior of cartilages surrounding the implant has not been studied, even in animal models. It is essential to understand the time dependent behavior of the cartilages due to biphasic nature of cartilage. Any protrusion of metal implant into the joint cavity damages the opposing soft tissue [1]. In order to avoid this, the positioning of implant together with the behavior of the cartilages immediately surrounding the implant have to be studied.
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Shin, Daehwan, Jian-Hua Lin, and Kyriacos Athanasiou. "Microindentation of the Individual Layers of Articular Cartilage." In ASME 1997 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1997. http://dx.doi.org/10.1115/imece1997-0275.

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Abstract Articular cartilage is known to have three distinct layers (superficial, middle, and deep) along its depth. Each layer has different biochemical, structural, and cellular characteristics. In the superficial layer, collagen fibrils are densely arranged along a tangential direction to the articular surface. Collagen fibrils are randomly oriented in the middle layer and vertically toward the subchondral bone in the deep layer. The superficial layer has a relatively low concentration of proteoglycans while the matrix in the middle and deep zones contains higher concentrations. The chondrocytes are flattened and most densely populated in the superficial layer, are rounded in the middle zone, and are larger, elongated, and arranged vertically between extracellular matrix in the deep layer [1, 2]. Based on the depth-related differences in the structural, biochemical, and cellular compositions, it is reasonable to assume that intrinsic mechanical properties of articular cartilage vary with depth. However, little information is available on the variation of the biomechanical properties in different layers of articular cartilage. The objectives of this study were 1) to test the applicability of the microindenter for testing cartilage at the microscopic level and 2) to obtain the intrinsic biomechanical properties of rabbit articular cartilage as a function of depth.
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Reports on the topic "Biomechanics of articular cartilage"

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Huard, Johnny. Articular Cartilage Repair Through Muscle Cell-Based Tissue Engineering. Fort Belvoir, VA: Defense Technical Information Center, March 2011. http://dx.doi.org/10.21236/ada552048.

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de Sousa, Eduardo, Renata Matsui, Leonardo Boldrini, Leandra Baptista, and José Mauro Granjeiro. Mesenchymal stem cells for the treatment of articular cartilage defects of the knee: an overview of systematic reviews. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0114.

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Review question / Objective: Population: adults (aged between 18 and 50 years) with traumatic knee lesions who underwent treatment with mesenchymal stem cells; Intervention: defined by the treatment with mesenchymal stem cells; The comparison group: treatment with autologous chondrocytes or microfracture treatments; Primary outcome: formation of cartilage neo tissue in the defect area, determined by magnetic resonance imaging (MRI) or by direct visualization in second-look knee arthroscopy.; Secondary outcomes: based on clinical scores such as visual analog scale (VAS) for pain, Western Ontario and McMaster universities score (WOMAC), knee society score (KSS), Tegner and Lysholm.
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