Log in

Relevant bibliographies by topics / Bioenergetic pathways

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers

Academic literature on the topic 'Bioenergetic pathways'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Bioenergetic pathways.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Bioenergetic pathways"

1

Sandage, Mary J., and Audrey G. Smith. "Muscle Bioenergetic Considerations for Intrinsic Laryngeal Skeletal Muscle Physiology." Journal of Speech, Language, and Hearing Research 60, no. 5 (May 24, 2017): 1254–63. http://dx.doi.org/10.1044/2016_jslhr-s-16-0192.

Full text

Abstract:

PurposeIntrinsic laryngeal skeletal muscle bioenergetics, the means by which muscles produce fuel for muscle metabolism, is an understudied aspect of laryngeal physiology with direct implications for voice habilitation and rehabilitation. The purpose of this review is to describe bioenergetic pathways identified in limb skeletal muscle and introduce bioenergetic physiology as a necessary parameter for theoretical models of laryngeal skeletal muscle function.MethodA comprehensive review of the human intrinsic laryngeal skeletal muscle physiology literature was conducted. Findings regarding intrinsic laryngeal muscle fiber complement and muscle metabolism in human models are summarized and exercise physiology methodology is applied to identify probable bioenergetic pathways used for voice function.ResultsIntrinsic laryngeal skeletal muscle fibers described in human models support the fast, high-intensity physiological requirements of these muscles for biological functions of airway protection. Inclusion of muscle bioenergetic constructs in theoretical modeling of voice training, detraining, fatigue, and voice loading have been limited.ConclusionsMuscle bioenergetics, a key component for muscle training, detraining, and fatigue models in exercise science, is a little-considered aspect of intrinsic laryngeal skeletal muscle physiology. Partnered with knowledge of occupation-specific voice requirements, application of bioenergetics may inform novel considerations for voice habilitation and rehabilitation.

APA, Harvard, Vancouver, ISO, and other styles

2

Cotter, David G., Rebecca C. Schugar, and Peter A. Crawford. "Ketone body metabolism and cardiovascular disease." American Journal of Physiology-Heart and Circulatory Physiology 304, no. 8 (April 15, 2013): H1060—H1076. http://dx.doi.org/10.1152/ajpheart.00646.2012.

Full text

Abstract:

Ketone bodies are metabolized through evolutionarily conserved pathways that support bioenergetic homeostasis, particularly in brain, heart, and skeletal muscle when carbohydrates are in short supply. The metabolism of ketone bodies interfaces with the tricarboxylic acid cycle, β-oxidation of fatty acids, de novo lipogenesis, sterol biosynthesis, glucose metabolism, the mitochondrial electron transport chain, hormonal signaling, intracellular signal transduction pathways, and the microbiome. Here we review the mechanisms through which ketone bodies are metabolized and how their signals are transmitted. We focus on the roles this metabolic pathway may play in cardiovascular disease states, the bioenergetic benefits of myocardial ketone body oxidation, and prospective interactions among ketone body metabolism, obesity, metabolic syndrome, and atherosclerosis. Ketone body metabolism is noninvasively quantifiable in humans and is responsive to nutritional interventions. Therefore, further investigation of this pathway in disease models and in humans may ultimately yield tailored diagnostic strategies and therapies for specific pathological states.

APA, Harvard, Vancouver, ISO, and other styles

3

Bettinazzi, Stefano, Liliana Milani, Pierre U. Blier, and Sophie Breton. "Bioenergetic consequences of sex-specific mitochondrial DNA evolution." Proceedings of the Royal Society B: Biological Sciences 288, no. 1957 (August 18, 2021): 20211585. http://dx.doi.org/10.1098/rspb.2021.1585.

Full text

Abstract:

Doubly uniparental inheritance (DUI) represents a notable exception to the general rule of strict maternal inheritance (SMI) of mitochondria in metazoans. This system entails the coexistence of two mitochondrial lineages (F- and M-type) transmitted separately through oocytes and sperm, thence providing an unprecedented opportunity for the mitochondrial genome to evolve adaptively for male functions. In this study, we explored the impact of a sex-specific mitochondrial evolution upon gamete bioenergetics of DUI and SMI bivalve species, comparing the activity of key enzymes of glycolysis, fermentation, fatty acid metabolism, tricarboxylic acid cycle, oxidative phosphorylation and antioxidant metabolism. Our findings suggest reorganized bioenergetic pathways in DUI gametes compared to SMI gametes. This generally results in a decreased enzymatic capacity in DUI sperm with respect to DUI oocytes, a limitation especially prominent at the terminus of the electron transport system. This bioenergetic remodelling fits a reproductive strategy that does not require high energy input and could potentially link with the preservation of the paternally transmitted mitochondrial genome in DUI species. Whether this phenotype may derive from positive or relaxed selection acting on DUI sperm is still uncertain.

APA, Harvard, Vancouver, ISO, and other styles

4

Protasoni, M., and J. W. Taanman. "Remodelling of bioenergetic pathways in human fibroblasts with carbohydrates." Neuromuscular Disorders 27 (March 2017): S19. http://dx.doi.org/10.1016/s0960-8966(17)30273-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Busch, Karin B., Gabriele Deckers-Hebestreit, Guy T. Hanke, and Armen Y. Mulkidjanian. "Dynamics of bioenergetic microcompartments." Biological Chemistry 394, no. 2 (February 1, 2013): 163–88. http://dx.doi.org/10.1515/hsz-2012-0254.

Full text

Abstract:

Abstract The vast majority of life on earth is dependent on harvesting electrochemical potentials over membranes for the synthesis of ATP. Generation of membrane potential often relies on electron transport through membrane protein complexes, which vary among the bioenergetic membranes found in living organisms. In order to maximize the efficient harvesting of the electrochemical potential, energy loss must be minimized, and this is achieved partly by restricting certain events to specific microcompartments, on bioenergetic membranes. In this review we will describe the characteristics of the energy-converting supramolecular structures involved in oxidative phosphorylation in mitochondria and bacteria, and photophosphorylation. Efficient function of electron transfer pathways requires regulation of electron flow, and we will also discuss how this is partly achieved through dynamic re-compartmentation of the membrane complexes into different supercomplexes. In addition to supercomplexes, the supramolecular structure of the membrane, and in particular the role of water layers on the surface of the membrane in the prevention of wasteful proton escape (and therefore energy loss), is discussed in detail. In summary, the restriction of energetic processes to specific microcompartments on bioenergetic membranes minimizes energy loss, and dynamic rearrangement of these structures allows for regulation.

APA, Harvard, Vancouver, ISO, and other styles

6

Hippler, Michael, Kevin Redding, and Jean-David Rochaix. "Chlamydomonas genetics, a tool for the study of bioenergetic pathways." Biochimica et Biophysica Acta (BBA) - Bioenergetics 1367, no. 1-3 (October 1998): 1–62. http://dx.doi.org/10.1016/s0005-2728(98)00136-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Haq, Rizwan, David E. Fisher, and Hans R. Widlund. "Molecular Pathways: BRAF Induces Bioenergetic Adaptation by Attenuating Oxidative Phosphorylation." Clinical Cancer Research 20, no. 9 (March 7, 2014): 2257–63. http://dx.doi.org/10.1158/1078-0432.ccr-13-0898.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Cheng, Gang, Jacek Zielonka, Joy Joseph, and Balaraman Kalyanaraman. "Synergistic Lethality of Inhibitors of Bioenergetic Pathways in Pancreatic Cells." Free Radical Biology and Medicine 51 (November 2011): S120. http://dx.doi.org/10.1016/j.freeradbiomed.2011.10.312.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Maimouni, Sara, Mi-Hye Lee, and Stephen Byers. "2427." Journal of Clinical and Translational Science 1, S1 (September 2017): 9–10. http://dx.doi.org/10.1017/cts.2017.49.

Full text

Abstract:

OBJECTIVES/SPECIFIC AIMS: The goal of this study is to examine bioenergetic phenotype of retinoic acid receptor responder 1 (RARRES1)-depleted epithelial cells and to facilitate the discovery of personalized metabo-therapeutics in the context of cancers characterized with loss of or low expression of RARRES1. METHODS/STUDY POPULATION: Anoikis assay and annexinV labeling were used to assess drug resistance and apoptotic phenotype in RARRES1-depleted epithelial cells. Metabolomics, AMP kinase activity, mito-tracker, and extracellular flux assays were used to examine the bioenergetic profile of RARRES1-depleted epithelial cells. Extracellular flux assays were used to assess the phenotype of RARRES1-depleted epithelial cells treated with or without metformin. RESULTS/ANTICIPATED RESULTS: RARRES1 is a major regulator of mitochondrial function. Its depletion in tumors induces an oxidative phosphorylation dependent phenotype and subsequently increases ATP abundance in the cell, enhances anabolic pathways and increases survival. Treatment with FDA approved mitochondrial respiration inhibitor, metformin, reversed the metabolic phenotype of RARRES1 depleted-epithelial cells. Metformin could be the ideal therapeutics to reduce tumor burden in cancers with loss of or low expression of RARRES1. DISCUSSION/SIGNIFICANCE OF IMPACT: Bioenergetic dynamics are emerging as a basis for understanding the pathology of cancer. The malignancy progresses as its metabolic pattern and mitochondrial respiration become more dysfunctional. The regulatory pathways of bioenergetic dynamics are currently poorly understood, and the characterization of proteins implicated in those processes must be assessed. One understudied protein and tumor suppressor is RARRES1. RARRES1 is induced by retinoic acid (a major metabolic regulator) and functions as a putative carboxypeptidase inhibitor. Understanding the connection between this carboxypeptidase inhibitor and intermediary metabolism will enlighten our understanding of the bioenergetic profile of cells and facilitate the discovery of personalized metabo-therapeutics in the context of cancer.

APA, Harvard, Vancouver, ISO, and other styles

10

Riddle, Ryan C., and Thomas L. Clemens. "Bone Cell Bioenergetics and Skeletal Energy Homeostasis." Physiological Reviews 97, no. 2 (April 2017): 667–98. http://dx.doi.org/10.1152/physrev.00022.2016.

Full text

Abstract:

The rising incidence of metabolic diseases worldwide has prompted renewed interest in the study of intermediary metabolism and cellular bioenergetics. The application of modern biochemical methods for quantitating fuel substrate metabolism with advanced mouse genetic approaches has greatly increased understanding of the mechanisms that integrate energy metabolism in the whole organism. Examination of the intermediary metabolism of skeletal cells has been sparked by a series of unanticipated observations in genetically modified mice that suggest the existence of novel endocrine pathways through which bone cells communicate their energy status to other centers of metabolic control. The recognition of this expanded role of the skeleton has in turn led to new lines of inquiry directed at defining the fuel requirements and bioenergetic properties of bone cells. This article provides a comprehensive review of historical and contemporary studies on the metabolic properties of bone cells and the mechanisms that control energy substrate utilization and bioenergetics. Special attention is devoted to identifying gaps in our current understanding of this new area of skeletal biology that will require additional research to better define the physiological significance of skeletal cell bioenergetics in human health and disease.

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Bioenergetic pathways"

1

Spickett, Corinne Michelle. "NMR studies of cellular bioenergetics." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257961.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Martins, Vicente de Paulo. "Caracterização bioenergética da forma leveduriforme de P. \'brasiliensis\': estudos bioquímicos e moleculares de vias mitocondriais alternativas." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/60/60135/tde-11052007-101615/.

Full text

Abstract:

O fungo dimórfico Paracoccidioides brasiliensis, é o agente etiológico da paracoccidioidomicose, uma das micoses sistêmicas humanas mais prevalentes na América Latina. Componentes da cadeia respiratória mitocondrial são potenciais alvos quimioterapêuticos. Nesse sentido, em nosso trabalho demonstramos diferenças entre a cadeia respiratória do fungo P. brasiliensis e do hospedeiro mamífero. A respiração, potencial de membrana e fosforilação oxidativa mitocondrial de esferoplastos de P. brasiliensis foram avaliados in situ, os quais demonstraram a presença de uma cadeia respiratória funcional. A adição de ADP induziu a transição da respiração do estado de repouso para o fosforilativo, em esferoplastos energizados com succinato, NADH, NAD+ e substratos ligados ao complex I. A presença de uma NADH-ubiquinona oxidoredutase foi demonstrada pela capacidade das células oxidarem NADH exógeno, assim como, pela respiração insensível a rotenona e sensível a flavona. Além disso, a respiração mantida por NAD+ foi sensível a rotenona e flavona, sugerindo que vias metabólicas citosolicas contribuem para a produção de NADH e substratos ligados ao complexo I. Foi demonstrado também que os níveis de expressão do complexo I e da NADH desidrogenase alternativa alternam-se durante a curva de crescimento de leveduras de P. brasiliensis. A respiração induzida por NADH ou succinato foi parcialmente inibida por KCN ou antimicina A e sensível à inibição por BHAM, indicando a presença de uma oxidase alternativa. A fim de se caracterizar esta enzima oxidase alternativa, o seu gene foi clonado e expressado em xii S. cerevisiae e E. coli, o qual conferiu uma respiração resistente a cianeto e sensível a BHAM. S. cerevisiae expressando oxidase alternativa mostraram uma menor taxa de multiplicação celular e diminuição na geração de EROs. Nós também observamos que inibidores da cadeia transportadora de elétrons retardaram a transição de micélio para levedura em culturas de P. brasiliensis. Além disso, estes inibidores e outras drogas geradoras de EROs aumentaram a expressão do gene da oxidase alternativa, assim como a produção de EROs em leveduras de P. brasiliensis.
Paracoccidioides brasiliensis, a thermically dimorphic fungus, is the etiological agent of endemic paracoccidioidomycosis, one of the most prevalent human systemic mycosis in Latin America. Components of the respiratory chain constitute potential pharmacological targets, and here are reported differences between the respiratory chain of the mammalian host and the fungus P. brasiliensis. Respiration, membrane potential and oxidative phosphorylation of mitochondria from P. brasiliensis spheroplasts were evaluated in situ, which demonstrated the existence of a functional respiratory chain. Adenosine 5\'-diphosphate (ADP) induced a transition from resting to phosphorylating respiration in mitochondria energized by succinate, NADH, NAD+ and complex I linked substrates. The presence of an alternative NADH-ubiquinone oxidoreductase was indicated by: the ability of the fungus to oxidize exogenous NADH; the insensitivity substrate-supported respiration to rotenone and sensitivity of this respiration to flavone. In addition, the sensitivity of NAD+-supported respiration to rotenone and flavone suggest that citosolic pathways contribute to NADH and complex I linked substrates production. Moreover, it was demonstrated that expression levels of complex I and alternative NADH dehydrogenase change during P. brasiliensis growth curve. The partial sensitivity of NADH or succinate-supported respiration to antimycin A and cyanide, as well as the sensitivity to BHAM, indicates the presence of an alternative oxidase. Therefore, to characterize the alternative oxidase its gene was cloned and heterologously xii expressed in S. cerevisiae and E. coli, wich confered, a cyanide resistant respiration and BHAM sensitivity. Moreover, S. cerevisiae that expressed alternative oxidase showed slower growth rate and decreased ROS generation. We also observed that electron transport pathways inhibitors delayed the P. brasiliensis mycelium to yeast transition in cultures. Besides, these inhibitors and other ROS generated drugs increase the ROS production and altenative oxidase expression.

APA, Harvard, Vancouver, ISO, and other styles

3

Sünwoldt, Juliane [Verfasser]. "Neuronal culture microenvironments determine preferences in bioenergetic pathway use / Juliane Sünwoldt." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2018. http://d-nb.info/1170814611/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Books on the topic "Bioenergetic pathways"

1

Skulachev, V. P. Principles of bioenergetics. Heidelberg: Springer, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Knott, Andrew B., and Ella Bossy-Wetzel. Mitochondrial Changes and Bioenergetics in Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0012.

Full text

Abstract:

Mitochondria are dynamic organelles that are of critical importance for cellular survival and health. Because mitochondria play central roles in energy production and synaptic maintenance, neurons are believed to be particularly vulnerable to mitochondrial dysfunction. The discovery that genetic mutations in genes coding for mitochondrial proteins cause neurodegenerative conditions further hinted at the likelihood that mitochondrial dysfunction is a key pathway of neurodegeneration. Indeed, a wealth of research has identified mitochondrial dysfunction as an early and shared event of all common neurodegenerative diseases, both genetic and sporadic in origin. Specific types of mitochondrial dysfunction that have been observed in most neurodegenerative diseases include bioenergetic failure, increased oxidative stress, mitochondrial DNA mutations, defective calcium handling, impaired mitochondrial dynamics, defective mitophagy, and decreased mitochondrial biogenesis. The search for drugs that successfully target these pathways of mitochondrial dysfunction in neurodegeneration is ongoing.

APA, Harvard, Vancouver, ISO, and other styles

3

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0022.

Full text

Abstract:

Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocation has been positively selected as an adaptive programme for transient serious, albeit non-life-threatening, inflammatory episodes. In CARDs, mistakenly, the adaptive programmes are used again but for a much longer time leading to systemic disease sequelae with endocrine (and also neuronal) abnormalities. The major endocrine alterations are depicted in the following list: mild activation of the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system, inadequate secretion of ACTH and cortisol relative to inflammation, loss of androgens, inhibition of the hypothalamic-pituitary-gonadal axis and fertility problems, high serum levels of oestrogens relative to androgens, fat deposits adjacent to inflamed tissue, increase of serum prolactin, and hyperinsulinaemia (and the metabolic syndrome). Neuroendocrine abnormalities are demonstrated using this framework that can explain many CARD-related endocrine disturbances. This chapter gives an overview on pathophysiology of neuroendocrine alterations in the context of energy regulation.

APA, Harvard, Vancouver, ISO, and other styles

4

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0022_update_002.

Full text

Abstract:

Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocation has been positively selected as an adaptive programme for transient serious, albeit non-life-threatening, inflammatory episodes. In CARDs, mistakenly, the adaptive programmes are used again but for a much longer time leading to systemic disease sequelae with endocrine (and also neuronal) abnormalities. The major endocrine alterations are depicted in the following list: mild activation of the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system, inadequate secretion of ACTH and cortisol relative to inflammation, loss of androgens, inhibition of the hypothalamic-pituitary-gonadal axis and fertility problems, high serum levels of oestrogens relative to androgens, fat deposits adjacent to inflamed tissue, increase of serum prolactin, and hyperinsulinaemia (and the metabolic syndrome). Neuroendocrine abnormalities are demonstrated using this framework that can explain many CARD-related endocrine disturbances. This chapter gives an overview on pathophysiology of neuroendocrine alterations in the context of energy regulation.

APA, Harvard, Vancouver, ISO, and other styles

5

Straub, Rainer H. Neuroendocrine system. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199642489.003.0022_update_003.

Full text

Abstract:

Endocrine abnormalities are very common in patients with chronic autoimmune rheumatic diseases (CARDs) due to the systemic involvement of the central nervous system and endocrine glands. In recent years, the response of the endocrine (and also neuronal) system to peripheral inflammation has been linked to overall energy regulation of the diseased body and bioenergetics of immune cells. In CARDs, hormonal and neuronal pathways are outstandingly important in partitioning energy-rich fuels from muscle, brain, and fat tissue to the activated immune system. Neuroendocrine regulation of fuel allocation has been positively selected as an adaptive programme for transient serious, albeit non-life-threatening, inflammatory episodes. In CARDs, mistakenly, the adaptive programmes are used again but for a much longer time leading to systemic disease sequelae with endocrine (and also neuronal) abnormalities. The major endocrine alterations are depicted in the following list: mild activation of the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system, inadequate secretion of ACTH and cortisol relative to inflammation, loss of androgens, inhibition of the hypothalamic-pituitary-gonadal axis and fertility problems, high serum levels of oestrogens relative to androgens, fat deposits adjacent to inflamed tissue, increase of serum prolactin, and hyperinsulinaemia (and the metabolic syndrome). Neuroendocrine abnormalities are demonstrated using this framework that can explain many CARD-related endocrine disturbances. This chapter gives an overview on pathophysiology of neuroendocrine alterations in the context of energy regulation.

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Bioenergetic pathways"

1

Minagawa, Jun. "Chlamydomonas: Bioenergetic Pathways—Regulation of Photosynthesis." In Chlamydomonas: Molecular Genetics and Physiology, 135–54. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66365-4_5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Gäbelein, Philipp, Laura Mosebach, and Michael Hippler. "Bioenergetic Pathways in the Chloroplast: Photosynthetic Electron Transfer." In Chlamydomonas: Molecular Genetics and Physiology, 97–134. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66365-4_4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Massoz, Simon, Pierre Cardol, Diego González-Halphen, and Claire Remacle. "Mitochondrial Bioenergetics Pathways in Chlamydomonas." In Chlamydomonas: Molecular Genetics and Physiology, 59–95. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-66365-4_3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Singer, M. "Dysfunction of the Bioenergetic Pathway." In Update in Intensive Care and Emergency Medicine, 299–310. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/3-540-30328-6_21.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Burgot, Jean-Louis. "The Pentose Phosphates Pathway—Glucogenesis." In Thermodynamics in Bioenergetics, 266–73. Boca Raton, FL : CRC Press, 2019. | “A science publishers book.”: CRC Press, 2019. http://dx.doi.org/10.1201/9781351034227-37.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Kramer, Jeffrey F., Daniel H. Pope, and John C. Salerno. "Pathways of Electron Transfer in Desulfovibrio." In Advances in Membrane Biochemistry and Bioenergetics, 249–58. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-8640-7_24.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Blum, Jacob J., and Michael S. Rabkin. "Quantitation of Fluxes in the Gluconeogenic, Glycolytic, and Pentose Phosphate Pathways in Isolated Rat Hepatocytes: Energetic Considerations." In Myocardial and Skeletal Muscle Bioenergetics, 255–70. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5107-8_19.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Hollman, Gregory A., and Waldemar E. Storm. "Cellular Bioenergetic Pathways and Processes." In Pediatric Critical Care, 1054–67. Elsevier, 2006. http://dx.doi.org/10.1016/b978-032301808-1.50070-5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Rasola, Andrea. "Chaperones and protein quality control in the neoplastic process." In Oxford Textbook of Cancer Biology, edited by Francesco Pezzella, Mahvash Tavassoli, and David J. Kerr, 239–54. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198779452.003.0017.

Full text

Abstract:

Maintenance of proteome quality control in cells is a vital and extremely complex task, which requires fine-tuning among synthesis, folding, and degradation of proteins and is controlled by an integrated network of subcellular components. A pivotal role in this process is played by chaperones, molecular machines that take part in nearly all cellular functions and make possible the optimal activity of proteins by assisting their folding, conformational changes, and subcellular trafficking, and by controlling protein degradation following unfolding, misfolding, or aggregation. Neoplastic cells undergo major changes in the homeostasis of their proteome, or proteostasis, as a consequence of a profound rewiring of their metabolic circuitries and of exposure to stressful environmental stimuli, such as hypoxia or nutritional and pH fluctuations. These stress conditions also affect protein folding in the endoplasmic reticulum and mitochondrial bioenergetic functions, leading to activation of organelle-restricted, protective signalling pathways called unfolded protein responses, which can subtly regulate the equilibrium among death, dormancy, and aggressiveness of tumour cells. In most cancer types molecular chaperones are overexpressed and exploited to cope with these stress stimuli and to underpin pro-oncogenic biological routines, including cell growth, proliferation, invasion, metastasis, and escape to death stimuli. Chaperone induction has been associated with cancer progression, resistance to chemotherapy, and poor prognosis; therefore, development of chaperone-targeting drugs has emerged as a promising antineoplastic strategy.

APA, Harvard, Vancouver, ISO, and other styles

10

Bhatti, Jasvinder Singh, Paras Pahwa, P. Hemachandra Reddy, and Gurjit Kaur Bhatti. "Impaired mitochondrial bioenergetics and signaling pathways." In Clinical Bioenergetics, 61–79. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-819621-2.00002-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Bioenergetic pathways"

1

Cheng, Gang, Jacek Zielonka, Joy Joseph, and Balaraman Kalyanaraman. "Abstract 1136: Synergistic inhibition of bioenergetic pathways in pancreatic cancer cells by mitochondria-targeted nitroxide and glycolytic inhibitors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1136.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Orr, C., T. Mcgarry, S. Wade, M. Biniecka, S. Wade, DJ Veale, and U. Fearon. "FRI0020 Altered bioenergetics, mitochondrial function and pro-inflammatory pathways in ra synovium in response to tofacitinib." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.6209.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Orr, Carl, Trudy McGarry, Sarah Wade, Monika Biniecka, Siobhan Wade, Douglas Veale, and Ursula Fearon. "02.33 Altered bioenergetics, mitochondrial function and pro-inflammatory pathways in ra synovium in response to tofacitinib." In 37th European Workshop for Rheumatology Research 2–4 March 2017 Athens, Greece. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2016-211050.33.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Kang, Rui, Daolin Tang, Nicole E. Schapiro, Michael T. Lotze, and Herbert J. Zeh. "Abstract B96: The HMGB1/RAGE inflammatory pathway promotes pancreatic tumor growth by regulating mitochondrial bioenergetics." In Abstracts: AACR Special Conference on Pancreatic Cancer: Progress and Challenges; June 18-21, 2012; Lake Tahoe, NV. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.panca2012-b96.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!