Relevant bibliographies by topics / Bioenergetic

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Bioenergetic'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 March 2023

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Journal articles on the topic "Bioenergetic"

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Bowden, Pye. "Including the body in psychotherapy." Ata: Journal of Psychotherapy Aotearoa New Zealand 7, no. 1 (July 30, 2001): 65–71. http://dx.doi.org/10.9791/ajpanz.2001.06.

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This paper describes the development of Bioenergetic Analysis, one of the more recent psychotherapies to arrive in New Zealand. Bioenergetics opens up psychoanalytic theory and practice to include all aspects of the self: the mind, the body, emotion, energy and relationship. In doing so it provides a holistic psychotherapy for the twenty-first century. The paper describes Bioenergetic's beginnings with Wilhelm Reich, a contemporary of Freud, and its establishment by Alexander Lowen. It critiques Bioenergetic's association with the 'cathartic' approach of the 1960s and describes how Bioenergetics is integrating Reich's and Lowen's work with current thinking about the therapeutic relationship.
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Sandage, Mary J., and Audrey G. Smith. "Muscle Bioenergetic Considerations for Intrinsic Laryngeal Skeletal Muscle Physiology." Journal of Speech, Language, and Hearing Research 60, no. 5 (May 24, 2017): 1254–63. http://dx.doi.org/10.1044/2016_jslhr-s-16-0192.

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PurposeIntrinsic laryngeal skeletal muscle bioenergetics, the means by which muscles produce fuel for muscle metabolism, is an understudied aspect of laryngeal physiology with direct implications for voice habilitation and rehabilitation. The purpose of this review is to describe bioenergetic pathways identified in limb skeletal muscle and introduce bioenergetic physiology as a necessary parameter for theoretical models of laryngeal skeletal muscle function.MethodA comprehensive review of the human intrinsic laryngeal skeletal muscle physiology literature was conducted. Findings regarding intrinsic laryngeal muscle fiber complement and muscle metabolism in human models are summarized and exercise physiology methodology is applied to identify probable bioenergetic pathways used for voice function.ResultsIntrinsic laryngeal skeletal muscle fibers described in human models support the fast, high-intensity physiological requirements of these muscles for biological functions of airway protection. Inclusion of muscle bioenergetic constructs in theoretical modeling of voice training, detraining, fatigue, and voice loading have been limited.ConclusionsMuscle bioenergetics, a key component for muscle training, detraining, and fatigue models in exercise science, is a little-considered aspect of intrinsic laryngeal skeletal muscle physiology. Partnered with knowledge of occupation-specific voice requirements, application of bioenergetics may inform novel considerations for voice habilitation and rehabilitation.
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Lowen, Alexander. "Al Lowen riflette sugli aspetti teorici dell'analisi bioenergetica e sulla sua esperienza negli ultimi quarant'anni." GROUNDING, no. 2 (July 2009): 17–32. http://dx.doi.org/10.3280/gro2008-002003.

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- Alexander Lowen recounts his meeting with Reich, its therapeutic experience with him and the reasons that led him to decide to continue the work of his teacher developing the bioenergetic analysis. As in his costume, biographical and theoretical elements are closely related.Key words: Wilhelm Reich, body, vegetoteraphy, bioenergetic analysisParole chiave: Wilhelm Reich, corpo, vegetoterapia, analisi bioenergetica
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Mahapatra, Gargi, Zhengrong Gao, James Bateman, Jenny L. Gonzalez-Armenta, Allison Amick, Ramon Casanova, Suzanne Craft, and Anthony J. A. Molina. "Systemic Bioenergetic Capacity Changes with Cognitive Status and Insulin Sensitivity in Older Adults." Innovation in Aging 5, Supplement_1 (December 1, 2021): 638. http://dx.doi.org/10.1093/geroni/igab046.2423.

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Abstract Systemic mitochondrial dysfunction is reported with AD progression, suggesting that peripheral blood cells may be used to investigate systemic mitochondrial alterations related to cognitive decline. We aimed to identify bioenergetic signatures associated with AD-related dementia and differences in insulin sensitivity associated with AD risk. We analyzed mitochondrial bioenergetics in peripheral blood cells collected from 365 older adults with varying cognitive status (normal, mild cognitive impairment, and AD) and insulin sensitivity. Normoglycemic individuals exhibited lower maximal bioenergetic capacity with AD (PBMCs: 239.6 pmol·min−1, p = 0.02; Platelets: 151.7 pmol·min−1, p = 0.06) compared to normal cognition (PBMCs: 271.5 pmol·min−1; Platelets: 171.7 pmol·min−1). Individuals with impaired insulin sensitivity exhibited lower maximal bioenergetic capacity in platelets with AD (171.6 pmol·min−1, p = 0.008) compared to normal cognition (210.6 pmol.min−1). Participants with impaired insulin sensitivity also exhibited unique bioenergetic profiles exemplified by overall higher levels of mitochondrial respiration, indicating that comorbidities such as diabetes can significantly influence bioenergetic capacity. We observed strong positive associations between maximal respiration in normoglycemic individuals with cognitive function, as measured by Modified Preclinical Alzheimer’s Cognitive Composite (mPACC5) (p = 0.06), and fatty acid oxidation in individuals with impaired insulin sensitivity with cortical thickness (p = 0.05). This study demonstrates that circulating cells may provide a cost-effective and minimally invasive way to monitor systemic bioenergetic changes associated with AD risk, progression, and insulin sensitivity. These findings also suggest that blood-based bioenergetics are related to key features of AD development and progression and should be further developed as a potential biomarker.
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Coven, Arnold B. "The Bioenergetic Approach to Rehabilitation Counseling." Journal of Applied Rehabilitation Counseling 16, no. 2 (June 1, 1985): 6–10. http://dx.doi.org/10.1891/0047-2220.16.2.6.

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The current focus on assisting the severely disabled confronts rehabilitation counselors with the demand of being more effective behavioral change agents. This article suggests that counselors try out Bioenergetics, a mindbody counseling approach. An overview of Bioenergetics theory is presented with examples of how it can be applied to the impaired. Guidelines for using Bioenergetic techniques are identified along with the necessary precautions.
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Chacko, Balu K., Philip A. Kramer, Saranya Ravi, Gloria A. Benavides, Tanecia Mitchell, Brian P. Dranka, David Ferrick, et al. "The Bioenergetic Health Index: a new concept in mitochondrial translational research." Clinical Science 127, no. 6 (May 29, 2014): 367–73. http://dx.doi.org/10.1042/cs20140101.

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Bioenergetics has become central to our understanding of pathological mechanisms, the development of new therapeutic strategies and as a biomarker for disease progression in neurodegeneration, diabetes, cancer and cardiovascular disease. A key concept is that the mitochondrion can act as the ‘canary in the coal mine’ by serving as an early warning of bioenergetic crisis in patient populations. We propose that new clinical tests to monitor changes in bioenergetics in patient populations are needed to take advantage of the early and sensitive ability of bioenergetics to determine severity and progression in complex and multifactorial diseases. With the recent development of high-throughput assays to measure cellular energetic function in the small number of cells that can be isolated from human blood these clinical tests are now feasible. We have shown that the sequential addition of well-characterized inhibitors of oxidative phosphorylation allows a bioenergetic profile to be measured in cells isolated from normal or pathological samples. From these data we propose that a single value–the Bioenergetic Health Index (BHI)–can be calculated to represent the patient's composite mitochondrial profile for a selected cell type. In the present Hypothesis paper, we discuss how BHI could serve as a dynamic index of bioenergetic health and how it can be measured in platelets and leucocytes. We propose that, ultimately, BHI has the potential to be a new biomarker for assessing patient health with both prognostic and diagnostic value.
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Strope, Taylor A., Cole J. Birky, and Heather M. Wilkins. "The Role of Bioenergetics in Neurodegeneration." International Journal of Molecular Sciences 23, no. 16 (August 16, 2022): 9212. http://dx.doi.org/10.3390/ijms23169212.

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Bioenergetic and mitochondrial dysfunction are common hallmarks of neurodegenerative diseases. Decades of research describe how genetic and environmental factors initiate changes in mitochondria and bioenergetics across Alzheimer’s disease (AD), Parkinson’s disease (PD), and amyotrophic lateral sclerosis (ALS). Mitochondria control many cellular processes, including proteostasis, inflammation, and cell survival/death. These cellular processes and pathologies are common across neurodegenerative diseases. Evidence suggests that mitochondria and bioenergetic disruption may drive pathological changes, placing mitochondria as an upstream causative factor in neurodegenerative disease onset and progression. Here, we discuss evidence of mitochondrial and bioenergetic dysfunction in neurodegenerative diseases and address how mitochondria can drive common pathological features of these diseases.
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Patón, Mauricio, and Jorge Rodríguez. "Integration of bioenergetics in the ADM1 and its impact on model predictions." Water Science and Technology 80, no. 2 (July 15, 2019): 339–46. http://dx.doi.org/10.2166/wst.2019.279.

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Abstract In this work, the integration of dynamic bioenergetic calculations in the IWA Anaerobic Digestion Model No. 1 (ADM1) is presented. The impact of bioenergetics on kinetics was addressed via two different approaches: a thermodynamic-based inhibition function and variable microbial growth yields based on dynamic Gibbs free energy calculations. The dynamic bioenergetic calculations indicate that the standard ADM1 predicts positive reaction rates under thermodynamically unfeasible conditions. The dissolved hydrogen inhibition approach used in ADM1 is, however, deemed as adequate, offering the trade-off of not requiring dynamic bioenergetics computation despite the need of hydrogen inhibition parameters. Simulations of the model with bioenergetics showed the low amount of energy available in butyrate and propionate oxidation, suggesting that microbial growth on these substrates must be very limited or occur via alternative mechanisms rather than dissolved hydrogen.
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Lehrer, H. Matthew, Lauren Chu, Martica Hall, and Kyle Murdock. "009 Self-Reported Sleep Efficiency and Duration are Associated with Systemic Bioenergetic Function in Community-Dwelling Adults." Sleep 44, Supplement_2 (May 1, 2021): A4. http://dx.doi.org/10.1093/sleep/zsab072.008.

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Abstract Introduction Sleep is important for aging, health, and disease, but its cellular role in these outcomes is poorly understood. Basic research suggests that disturbed and insufficient sleep impair mitochondrial bioenergetics, which is involved in numerous aging-related chronic conditions. However, the relationship between sleep and bioenergetics has not been examined in humans. We examined associations of self-reported sleep with systemic bioenergetic function in peripheral blood mononuclear cells (PBMCs) of community-dwelling adults. Methods N = 43 adults (79% female) ages 48–70 (M = 61.63, SD = 5.99) completed the Pittsburgh Sleep Quality Index (PSQI) from which key components of sleep (satisfaction, alertness, timing, efficiency, and duration) were calculated. Participants provided blood samples from which PBMCs were isolated and measured for bioenergetics using extracellular flux analysis. Associations of sleep components with bioenergetic parameters, including the Bioenergetic Health Index (BHI), were examined. Results In bivariate analyses, lower sleep efficiency was associated with lower maximal respiration, spare capacity, and BHI (ps < 0.05). Longer sleep duration was associated with lower BHI (p < 0.01) and later sleep timing was associated with higher basal respiration, ATP-linked respiration, maximal respiration, spare capacity, and non-mitochondrial respiration (ps < 0.05). After adjustment for age, sex, and body mass index, lower sleep efficiency (β = 0.52, p < 0.01) and longer sleep duration (β = -0.43, p < 0.01) were associated with lower BHI. Conclusion Self-reported indices of sleep efficiency and duration are related to systemic bioenergetic function in humans, suggesting a possible cellular pathway linking sleep to health. Support (if any) T32HL082610
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Hill, Bradford G., Gloria A. Benavides, Jack R. Lancaster, Scott Ballinger, Lou Dell’Italia, Jianhua Zhang, and Victor M. Darley-Usmar. "Integration of cellular bioenergetics with mitochondrial quality control and autophagy." Biological Chemistry 393, no. 12 (December 1, 2012): 1485–512. http://dx.doi.org/10.1515/hsz-2012-0198.

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Abstract Bioenergetic dysfunction is emerging as a cornerstone for establishing a framework for understanding the pathophysiology of cardiovascular disease, diabetes, cancer and neurodegeneration. Recent advances in cellular bioenergetics have shown that many cells maintain a substantial bioenergetic reserve capacity, which is a prospective index of ‘healthy’ mitochondrial populations. The bioenergetics of the cell are likely regulated by energy requirements and substrate availability. Additionally, the overall quality of the mitochondrial population and the relative abundance of mitochondria in cells and tissues also impinge on overall bioenergetic capacity and resistance to stress. Because mitochondria are susceptible to damage mediated by reactive oxygen/nitrogen and lipid species, maintaining a ‘healthy’ population of mitochondria through quality control mechanisms appears to be essential for cell survival under conditions of pathological stress. Accumulating evidence suggest that mitophagy is particularly important for preventing amplification of initial oxidative insults, which otherwise would further impair the respiratory chain or promote mutations in mitochondrial DNA (mtDNA). The processes underlying the regulation of mitophagy depend on several factors, including the integrity of mtDNA, electron transport chain activity, and the interaction and regulation of the autophagic machinery. The integration and interpretation of cellular bioenergetics in the context of mitochondrial quality control and genetics is the theme of this review.
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Dissertations / Theses on the topic "Bioenergetic"

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Sullivan, Courtney R. "Bioenergetic Abnormalities in Schizophrenia." University of Cincinnati / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1523629996205968.

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Quirk, P. G. "NMR studies of bioenergetic systems." Thesis, University of Oxford, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.379927.

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Hamraz, Minoo. "Bioenergetic consequences of the hyperosmotic shock." Thesis, Sorbonne Paris Cité, 2019. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2332&f=17549.

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L'inflammation est associée à des modifications du métabolisme cellulaire avec une glycolyse (libération de lactate) accrue accompagnée d'une baisse de la phosphorylation oxydative mitochondriale. L'inflammation cause l'hyperosmolarité du milieu extracellulaire. Cette thèse examine les effets de l'hyperosmolarité sur le métabolisme énergétique cellulaire. Nous avons mesuré la consommation d'oxygène cellulaire (OCR) et la production d'acide (PPR) c'est à dire de lactate libéré dans le milieu extérieur avec deux approches expérimentales : l'oxygraphie haute résolution (O2k Oroboros instrument) pour l'OCR et l'analyseur de flux extracellulaires (Seahorse Agilent) pour l'OCR et le PPR. L'exposition de cellules à des conditions hyperosmolaires (600 mOsmoles au lieu de la valeur normale 300) cause une répression de la consommation d'oxygène qui s'établit en quelques minutes et dure des heures (indéfiniment?) et à la longue affecte la viabilité cellulaire. Cet effet a été retrouvé sur plusieurs types cellulaires: CHO (épithélium ovarien), HT29 (colonocytes), HEK293 (rein embryonnaire), SH-SY5Y (neuroblastome). Il est reproduit avec trois osmolytes différents: le mannitol, le polyéthylène glycol et le chlorure de sodium. Un stress osmotique plus modéré (450 mOsm) cause une même chute de la respiration mais de durée limitée (une-deux heures). Une recherche des mécanismes à l'origine de cette inhibition montre que l'hyperosmolarité altère la fonction mitochondriale de différentes manières. Un premier effet est une inhibition du système enzymatique de production d'ATP. En présence de glucose cette inhibition s'accompagne d'une importante augmentation de la glycolyse qui cause une inhibition mitochondriale supplémentaire qui repose sur l'amplification de l'effet Crabtree (inhibition de la respiration par le glucose) dont la cible sont les complexes respiratoires. En l'absence de glucose le turnover cellulaire e l'ATP est sérieusement diminué mais de façon inattendue la survie cellulaire est plutôt meilleure. Ces résultats posent la question de la contribution des conditions hyperosmotiques liées à l'inflammation dans l'établissement d'un profil métabolique de type inflammatoire
Metabolic alterations associated with inflammation include increased recruitment of glycolysis (lactate release) and repression of mitochondrial oxidative phosphorylation. Inflammation causes hyperosmolar conditions in the extracellular medium. This thesis examines the consequences of hyperosmolarity on cellular bioenergetics. For this purpose we measured the cellular oxygen consumption rate (OCR) and proton production rate (PPR) for lactate release in the external medium. Two methodologies were used the high-resolution respirometer (O2k Oroboros Instruments) for OCR and the extracellular flux analyzer (Seahorse, Agilent) for OCR and PPR. The exposure cells to hypertonic conditions (600 milliOsmoles while normal value is 300) causes within few minutes a decrease in OCR (cellular respiration) that lasts for hours (indefinitely) and in the long term impact on cellular viability. This effect was observed with four different cell lines CHO (ovarian epithelial), HT29 (colonocytes), HEK293 (Embryonic kidney) and SH-SY5Y (Neuroblastoma). It was shown to be caused by three different osmolytes: Mannitol, polyethylene glycol, sodium chloride. A milder osmotic challenge (450 mOsm) caused a similar initial decrease but with restoration of initial OCR within few hours. The mechanisms underlying this effect have been investigated, hyperosmolarity impacts on mitochondrial respiration at different steps. A first effect is the inhibition of the mitochondrial ATP production step. In presence of glucose this is accompanied by a large increase in glycolysis (lactate release) that causes further mitochondrial inhibition by a second mechanism, which is likely to represent an enhancement of the Crabtree effect (inhibition of respiration by glycolysis) that impacts on respiratory complexes. In absence of glucose the cellular ATP turnover is seriously repressed surprisingly cellular survival is rather improved. These results raise therefore the question of the possible contribution of the hyperosmotic conditions caused by inflammation in the acquisition of the inflammatory metabolic profile
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Mould, Joanne. "Bioenergetic modelling of heat loss in endotherms." Thesis, University of Glasgow, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443428.

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Spickett, Corinne Michelle. "NMR studies of cellular bioenergetics." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.257961.

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Palmer, John. "Chemostat growth studies and bioenergetic aspects of Methanosarcina barkeri." Thesis, University of Kent, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279877.

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Lennon, Adrian Michael. "Bioenergetic and developmental aspects of plant mitochondrial protein import." Thesis, University of Sussex, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386188.

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Bloch, Katarzyna. "Structural and bioenergetic changes in tumour spheroids during growth." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:1d7b8669-b62a-4554-bb66-157f54e3ded2.

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Multicellular tumour spheroids (TS) are an in vitro model of avascular tumours, and have been widely used to investigate tumour growth, metabolism and hypoxia. The geometry of the TS lends itself to mathematical representation, and theoretical models of TS growth and the development of hypoxia are abundant. With some notable exceptions however, these models have been developed independently of the biological data collection process and are overwhelmingly based upon data from multiple sources. Thus, whilst mathematical modeling has the potential to help explain and guide biological experiments, without reliable data it is unlikely to live up to this expectation. In this thesis, a combination of experimental and theoretical approaches was used to characterize the relationship between proliferation, hypoxia and metabolism during the growth of TS derived from the DLD-1 human colon adenocarcinoma cell line. Experimental data were collected over the entire period of TS growth, generating a high volume of predominantly imaging data. To facilitate the extraction of quantitative information from this, a suite of image analysis software, which is readily applicable to other data sets, was developed. During growth, the DLD-1 TS maintained a macroscopic spherical geometry but at the microscale level the TS boundary was increasingly irregular, with TS disintegrating rapidly after 20 days. Immunofluorescence (IF) studies showed that hypoxia developed soon after TS initiation, followed by the characteristic onset of necrosis. Reduced proliferation was found to be concomitant with the development of hypoxia, although some cells retained proliferative capacity even under severely hypoxic conditions. Towards the end of culture, TS were primarily comprised of severely hypoxic and necrotic cells, a probable cause of disintegration. Mathematical simulation of oxygen gradients in TS using literature-based values for the maximal rate of oxygen consumption was used to estimate the partial oxygen pressure (pO2) at which the IF marker of hypoxia was bound. Assuming a spatially-invariant rate of oxygen consumption, the model predicted that the onset of hypoxic binding occurs at pO2 levels similar to those reported in the literature, however the onset of necrosis was overestimated. Mathematical simulations predicted that oxygen consumption decreases as TSs increase in size, supporting previous observations. The Warburg Effect, where glucose metabolism is favoured even under aerobic conditions, is a hallmark of tumours. Although development of the glycolytic phenotype during TS growth was observed in the form of an elevated activity of the lactate dehydrogenase V (LDHV) enzyme, the activity and expression of other glycolytic enzymes, such as hexokinase II (HKII), was unaltered. Whilst the spatial distribution of HKII was unrestricted throughout the TS's viable fraction, LDHV expression was elevated in regions of hypoxia, suggesting constant adaptation of tumour cells to their microenvironment. In addition to the above findings, the data generated have been collected and analysed in the context of the requirements of theoretical modelling at each step; thus, they can be used to parameterise and inform more sophisticated models of tumour metabolism.
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Lindgård, Ann. "Improved bioenergetic recovery during experimental ischemia and reperfusion by irradiation /." Göteborg : Göteborg University, Bioenergetics Group, Department of Surgery, Wallenberg Laboratory & Lundberg Laboratory for Bioanalysis, Sahlgrenska Academy, Göteborg University, 2007. http://hdl.handle.net/2077/7505.

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Morinville, Geneviève R. "The bioenergetic basis of anadromy in brook trout (Salvelinus fontinalis) /." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=85942.

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Migratory and resident forms of salmonid fishes, including brook trout (Salvelinus fontinalis), coexist in many river systems, but little is known about the ecological basis of these important variations in life history. This thesis elucidates the bioenergetic basis of anadromy (migration from freshwater spawning sites to the sea) in populations of brook trout. By focusing on the early stages, I provide support for the idea that variation in energy allocation leads to the adoption of migratory or resident strategies. More specifically, I demonstrate that juvenile anadromous brook trout, in the year(s) prior to migration, exhibit higher food consumption rates but lower growth efficiencies compared to residents indicating that they have higher metabolic costs. Higher metabolic costs of migratory fish are associated with the exploitation of higher current velocity habitats that provide more food but at a higher cost. This conclusion is supported by differences in delta13C (migrants have more negative delta13 C compared to residents), morphology (migrants are more streamlined than residents), and field observations (brook trout inhabiting streams with both forms exploit a wider range of habitats than those inhabiting 'pure' resident streams). Brook trout thus appear to migrate in response to energetic limitations in their local habitat. The estuary to which they migrate has better feeding opportunities, as the prey spectrum at sea is both larger and wider than that found in freshwater. This permits them to undergo diet shifts to larger prey, reducing their foraging costs, and thus most likely contributes to the trout's rapid growth rates experienced at sea. Importantly, the results of this thesis indicate that the persistence of migrant and resident strategies in the same system suggest a trade-off between local adaptability and the ability to exploit large-scale environmental heterogeneity.
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Books on the topic "Bioenergetic"

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Bioenergetic landscape: La progettazione del giardino terapeutico bioenergetico. Napoli: Sistemi editoriali, 2009.

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Lowen, Alexander. Joy: The surrender to the body and to life. New York: Arkana, 1995.

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Stefanatos, Joanne. Bioenergetic veterinary medicine: This seminar is entirely on bioenergetic veterinary medicine. [S.l: s.n., 1988.

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Peschek, Guenter A., Christian Obinger, and Gernot Renger, eds. Bioenergetic Processes of Cyanobacteria. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0388-9.

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Giuseppe, Carzedda, Fauser Wera, Heinrich-Clauer Vita, Helferich Christoph, Schroeter Vincentia Hrsg, Koemeda-Lutz Margit Hrsg, Nascimento Maê Hrsg, and Psychosozial-Verlag, eds. Bioenergetic Analysis: The Clinical Journal of the International Institute for Bioenergetic Analysis (25/2015). Gießen, Lahn: Psychosozial-Verlag, 2015.

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Stefanatos, Joanne. Bioenergetic medicine: Homeopathy and acupuncture for animals. [s.l.]: American Holistic Veterinary Medicine Association, 1991.

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J, Vanrenen Louis, ed. Bioenergetic medicines east and west: Acupuncture and homeopathy. Berkeley, Calif: North Atlantic Books, 1988.

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Narcissism: Denial of the true self. New York: Collier Books, 1985.

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Lowen, Alexander. Narcissism: Denial of the true self. New York: Touchstone, 1997.

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Lowen, Alexander. Bioenergetics. New York: Penguin/Arkana, 1994.

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Book chapters on the topic "Bioenergetic"

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Zeiger, Stephanie L. H., Jeannette N. Stankowski, and BethAnn McLaughlin. "Assessing Neuronal Bioenergetic Status." In Methods in Molecular Biology, 215–35. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-170-3_15.

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Skulachev, Vladimir P. "Membrane Bioenergetic Studies: An Outlook." In Membrane Bioenergetics, 327–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-72978-2_8.

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Singer, M. "Dysfunction of the Bioenergetic Pathway." In Update in Intensive Care and Emergency Medicine, 299–310. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/3-540-30328-6_21.

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Krulwich, Terry Ann, David B. Hicks, Talia Swartz, and Masahiro Ito. "Bioenergetic Adaptations That Support Alkaliphily." In Physiology and Biochemistry of Extremophiles, 311–29. Washington, DC, USA: ASM Press, 2014. http://dx.doi.org/10.1128/9781555815813.ch24.

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Halling, Jens Frey, Anders Gudiksen, Henriette Pilegaard, and P. Darrell Neufer. "Exercise: Thermodynamic and Bioenergetic Principles." In Physiology in Health and Disease, 27–50. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-94305-9_3.

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Kazak, Lawrence. "Bioenergetic Analyses in Adipose Tissue." In Thermogenic Fat, 125–34. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6820-6_12.

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Peschek, Günter A., Margit Bernroitner, Samira Sari, Martin Pairer, and Christian Obinger. "Life Implies Work: A Holistic Account of Our Microbial Biosphere Focussing on the Bioenergetic Processes of Cyanobacteria, the Ecologically Most Successful Organisms on Our Earth." In Bioenergetic Processes of Cyanobacteria, 3–70. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0388-9_1.

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Bald, Dirk. "ATP Synthase: Structure, Function and Regulation of a Complex Machine." In Bioenergetic Processes of Cyanobacteria, 239–61. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0388-9_10.

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Drews, Gerhart. "The Evolution of Cyanobacteria and Photosynthesis." In Bioenergetic Processes of Cyanobacteria, 265–84. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0388-9_11.

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Fromme, Petra, and Ingo Grotjohann. "Structure of Cyanobacterial Photosystems I and II." In Bioenergetic Processes of Cyanobacteria, 285–335. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0388-9_12.

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Conference papers on the topic "Bioenergetic"

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Rezende, Maria Clara Lopes, Maria Luiza Franco de Oliveira, Júlia Campos Fabri, Maria Júlia Filgueiras Granato, Mariana Vanon Moreira, and Leandro Vespoli Campos. "Neuroprotective Effects of Creatine Supplementation in Neurodegenerative Diseases." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.234.

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Introduction: Creatine is important in providing energy for the resynthesis of adenosine triphosphate (ATP) and in the deposition of intracellular energy, being present mainly in muscle fibers and in the brain. Supplementation with exogenous creatine can be used in neurodegenerative disorders that are related to bioenergetic deficits in the etiology and progression of the disease. Objective: Highlight the neuroprotective mechanisms of creatine supplementation in neurodegenerative diseases. Methods: In April 2021, a search was carried out on MEDLINE, with the descriptors: “Creatine” and “Neuroprotection”; and its variations, obtained in MeSH. Studies published in the last five years were included. Results: Of the 122 articles found, four were used in this work. They concluded that creatine supplementation contributes to brain bioenergetics by increasing phosphocreatine deposits, restoring mitochondrial functions and decreasing susceptibility to apoptosis. In addition, creatine intake shortly after the diagnosis of Huntington’s and Parkinson’s Diseases can be used as a complementary therapy, because improve performance in tasks of memory and intelligence. Finally, it buffers cellular concentrations of ATP, being a possible therapeutic strategy to delay or stop neurodegeneration diseases. Conclusion: Creatine promote important neuroprotective effect, but further studies on the subject are needed.
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Babushkin, D. D., and S. А. Zaytsev. "BIOENERGETIC EFFICIENCY OF SEED PRODUCTION OF CORN HYBRIDS." In 11-я Всероссийская конференция молодых учёных и специалистов «Актуальные вопросы биологии, селекции, технологии возделывания и переработки сельскохозяйственных культур». V.S. Pustovoit All-Russian Research Institute of Oil Crops, 2021. http://dx.doi.org/10.25230/conf11-2021-15-18.

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We conducted the comparative analysis of bioenergetic efficiency of production of seeds of corn hybrids developed in breeding centers – participants of the Coordination Council on breeding and seed production of corn, included into the State register of breeding achievements permitted for production in the Russian Federation. We revealed corn hybrids with relatively high yield of gross energy: Neon 147 МV (71.4 GJ/ha), Katerina SV (68.4 GJ/ha), and crude protein: Neon 147 МV (0.45 t/ha), RNIISK-1 (0.43 t/ha), Baykal (0.44 t/ha).
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Nguyen, Q., M. Simon, and S. Shiva. "Right and Left Ventricle Cardiomyocytes Show Distinct Mitochondrial Bioenergetic Profiles." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a7400.

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Hanlon, M. M., T. McGarry, M. Canavan, C. Lowe, S. Wade, D. J. Veale, and U. Fearon. "SAT0007 Distinct macrophage phenotype and bioenergetic profiles in rheumatoid arthritis." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.2195.

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TU, S., A. GEHRING, and P. IRWIN. "BIOENERGETIC CONFIRMATION OF VIABLE PATHOGENS IN FOODS BY ATP-BIOLUMINESCENCE." In Proceedings of the 13th International Symposium. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702203_0106.

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Mendonça, Bárbara Gazolla de, Lara Lopardi de Souza Leite, Carolina Falconi Amorim, Flávio Welinton Martins Cruz, and Gustavo Cosendey Portes. "The relation between the menopause transition with higher rates of Alzheimer in the female gender: a literature review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.577.

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Introduction: The reproductive senescence and the complete exhaustion of the germ cells result in processes capable of provoking changes in the hormone profile of women. The decrease in the bioenergetic metabolism during the menopause transition (MT), due to modifications in the estrogen levels, can be substrate for neurological dysfunctions. The physiopathological mechanisms of the Alzheimer’s Disease (AD) are activated years before the symptoms and coincide with MT, making the female gender a risk factor. The review aims on analyzing the higher rates of AD in the female gender, based on physiological changes that occur in the MT. Methods: Literature review based on articles from the PubMed database. Results: Were compared results from cerebral images of women in MT with cognitively normal men with the same age. In the women were found alterations such as abnormalities in the biomarkers of AD and reduction of the cerebral metabolic rate. It was noticed that women in the post menopause presented hypometabolism in the same cerebral regions as patients with AD and a reduction of the mitochondrial cytochrome oxidase of the platelets. Conclusion: The study presented evident bioenergetic factors that corroborate to the relation of MT and higher incidence of AD in the female gender. This way, such transition represents a window of opportunity for possible therapeutic interventions.
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Hanlon, MM, T. McGarry, M. Canavan, C. Low, DJ Veale, and U. Fearon. "P022/O02 Distinct macrophage phenotype and bioenergetic profiles in rheumatoid arthritis." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.16.

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Diers, Anne R., Michael A. Kiebish, Arleide Lee, Rakibou Ouro-Djobo, Stephane Gesta, Vivek K. Vishnudas, Rangaprasad Sarangarajan, and Niven R. Narain. "Abstract 3051: Functional bioenergetic signature predicts therapeutic responses to BPM 31510." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3051.

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Chhina, J., S. Dar, M. Deshpande, S. Giri, A. Munkarah, and R. Rattan. "Abstract POSTER-BIOL-1307: Bioenergetic adaptations in chemoresistant ovarian cancer cells." In Abstracts: 10th Biennial Ovarian Cancer Research Symposium; September 8-9, 2014; Seattle, WA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.ovcasymp14-poster-biol-1307.

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Henchey, Elizabeth M., Sallie S. Schneider, D. Joseph Jerry, and Nagendra Yadava. "Abstract A28: Bioenergetic analysis of primary human mammary epithelial cells (hMECs)." In Abstracts: AACR Special Conference: Metabolism and Cancer; June 7-10, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.metca15-a28.

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Reports on the topic "Bioenergetic"

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Beal, M. F. Bioenergetic Approaches and Inflammation in MPTP Toxicity. Fort Belvoir, VA: Defense Technical Information Center, September 2005. http://dx.doi.org/10.21236/ada439263.

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Beal, M. F. Bioenergetic Approaches and inflammation of MPTP Toxicity. Fort Belvoir, VA: Defense Technical Information Center, September 2008. http://dx.doi.org/10.21236/ada488708.

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Beal, M. F. Bioenergetic Approaches and Inflammation of MPTP Toxicity. Fort Belvoir, VA: Defense Technical Information Center, September 2009. http://dx.doi.org/10.21236/ada508622.

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Beal, M. F. Bioenergetic Approaches and Inflammation of MPTP Toxicity. Fort Belvoir, VA: Defense Technical Information Center, September 2007. http://dx.doi.org/10.21236/ada482978.

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Kelly, R. M. Bioenergetic and physiological studies of hyperthermophilic archaea. Final report. Office of Scientific and Technical Information (OSTI), March 1999. http://dx.doi.org/10.2172/325744.

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Dudkin, I. V., and T. A. Dudkina. Bioenergetic efficiency of growing crops in grain cultivating crop rotation. Курская государственная сельскохозяйственная академия, 2018. http://dx.doi.org/10.18411/issn1997-0749.2018-05-13-18.

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Ludden, P. W. The Biochemistry, Bioenergetic, and Physiology of the Co-Dependent Growth of Rhodosprillium. Office of Scientific and Technical Information (OSTI), March 2002. http://dx.doi.org/10.2172/1183434.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Fort Belvoir, VA: Defense Technical Information Center, May 2004. http://dx.doi.org/10.21236/ada430585.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Fort Belvoir, VA: Defense Technical Information Center, May 2003. http://dx.doi.org/10.21236/ada419306.

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Browne, Susan E. Bioenergetic Defects and Oxidative Damage in Transgenic Mouse Models of Neurodegenerative Disorders. Fort Belvoir, VA: Defense Technical Information Center, June 2005. http://dx.doi.org/10.21236/ada460659.

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