Academic literature on the topic 'Bilateral Vestibulopathy'

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Journal articles on the topic "Bilateral Vestibulopathy"

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Kim, Minbum, and Kyu-Sung Kim. "Bilateral Vestibulopathy." Korean Journal of Otorhinolaryngology-Head and Neck Surgery 56, no. 3 (2013): 131. http://dx.doi.org/10.3342/kjorl-hns.2013.56.3.131.

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Kim, Seonhye, Young-Mi Oh, Ja-Won Koo, and Ji Soo Kim. "Bilateral Vestibulopathy." Otology & Neurotology 32, no. 5 (July 2011): 812–17. http://dx.doi.org/10.1097/mao.0b013e31821a3b7d.

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Fujimoto, Chisato. "Idiopathic bilateral vestibulopathy." Equilibrium Research 72, no. 6 (2013): 501–3. http://dx.doi.org/10.3757/jser.72.501.

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Baloh, R. W., K. Jacobson, and V. Honrubia. "Idiopathic bilateral vestibulopathy." Neurology 39, no. 2 (February 1, 1989): 272. http://dx.doi.org/10.1212/wnl.39.2.272.

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Zhiznevskiy, D. V., M. V. Zamergrad, O. S. Levin, and A. A. Azimova. "Bilateral peripheral vestibulopathy." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 123, no. 4 (2023): 24. http://dx.doi.org/10.17116/jnevro202312304124.

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Young, Allison S., Rachael L. Taylor, Leigh A. McGarvie, G. M. Halmagyi, and Miriam S. Welgampola. "Bilateral sequential peripheral vestibulopathy." Neurology 86, no. 15 (March 11, 2016): 1454–56. http://dx.doi.org/10.1212/wnl.0000000000002563.

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Gürkov, Robert, Leonardo Manzari, Alexander Blödow, Angela Wenzel, Dusan Pavlovic, and Leonal Luis. "Amiodarone-associated bilateral vestibulopathy." European Archives of Oto-Rhino-Laryngology 275, no. 3 (December 27, 2017): 823–25. http://dx.doi.org/10.1007/s00405-017-4858-3.

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Ravishankar, Uma, Rithvik Ramesh, Shankar Venkatasubramanian, and Sundar Shanmugam. "Bilateral vestibulopathy presaging clinically probable multisystem atrophy." BMJ Case Reports 16, no. 6 (June 2023): e254472. http://dx.doi.org/10.1136/bcr-2022-254472.

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Bilateral vestibulopathy is a clinical diagnosis backed by investigative confirmation, which can be masked by the lack of lateralising signs. It has a broad aetiological spectrum including neurodegenerative conditions, though many such cases also have unknown aetiology. We present the case of an elderly gentleman who presented with progressive bilateral vestibulopathy nearly 1.5 years prior to his eventual diagnosis of clinically probable multisystem atrophy. This case highlights the need to serially re-evaluate for parkinsonism and cerebellar signs in idiopathic bilateral vestibulopathy and raises a possibility that bilateral vestibulopathy, similar to constipation or anosmia, could be an early syndrome presaging the onset of overt extrapyramidal or cerebellar symptoms in patients with multisystem atrophy.
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Gu, Huanhuan, Yixin Xu, Jin Xu, and Jianhua Zhuang. "A rare case report of bilateral vestibulopathy due to otosyphilis." Medicine 103, no. 20 (May 17, 2024): e38149. http://dx.doi.org/10.1097/md.0000000000038149.

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Rationale: Bilateral vestibulopathy is an important cause of imbalance. There are multiple etiologies of bilateral vestibulopathy (BVP), but reports of BVP due to otosyphilis are rare. Patient concerns: A 39-year-old male was referred to our medical center due to vertigo, persistent dizziness and gait disturbance for 2 months. Diagnoses: Bilateral vestibulopathy due to otosyphilis was considered in this case, as confirmed through analyses of vestibular function, laboratory tests, and penicillin treatment. Interventions: The patient was was treated with a high dose of penicillin G (24 × 106 IU/d) for 14 days. Outcomes: The patient’s symptoms had improved greatly following treatment, with dizziness and gait disturbance having completely resolved at 3 months following hospital discharge. Lessons: Bilateral vestibulopathy should be considered when evaluating patients with acute or subacute persistent dizziness. Clinicians should also be aware of the potential for otosyphilis among patients who report BVP.
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Iwasaki, Shinichi. "Novel Treatments for Bilateral Vestibulopathy." Practica Oto-Rhino-Laryngologica 113, no. 6 (2020): 335–44. http://dx.doi.org/10.5631/jibirin.113.335.

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Dissertations / Theses on the topic "Bilateral Vestibulopathy"

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Navarro, Morales Deborah. "Τhe influence οf the vestibular system οn time perceptiοn." Electronic Thesis or Diss., Normandie, 2025. https://theses.hal.science/tel-05000089.

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Au niveau perceptif, le temps n’est pas une métrique constante régie par le tic-tac d’une horloge. Les distorsions temporelles surviennent en raison de divers facteurs comme le manque d’attention, les émotions, le manque de sommeil, l’excitation, le mouvement, entre autres. Cette thèse vise à explorer comment le système vestibulaire influence la perception du temps. Dans le premier axe de cette thèse deux études ont été réalisées lors des missions spatiales de longue durée. Nous avons trouvé que les astronautes à bord de la Station Spatiale Internationale estiment correctement les intervalles de temps dans l’échelle de jours, ils surestiment le temps dans les durées de secondes à minutes et sous-estiment le temps dans l’échelle des heures. Etant donné que les distorsions temporelles dans l’espace peuvent provenir de plusieurs sources, nous avons décidé de mener une tâche spécifique du système vestibulaire. Le deuxième axe comprend deux études sur la perception du temps lors de stimulations vestibulaires, en se concentrant sur les rotations corporelles. Dans la première étude, nous avons constaté que le temps perçu durant les rotations est sous-estimé par rapport aux conditions statiques chez les sujets en bonne santé. Dans la deuxième étude, nous avons confirmé que cet effet est vestibulaire, car il est absent chez les patients atteints de vestibulopathie bilatérale. Nos résultats suggèrent que la perception du temps dépend des entrées vestibulaires : lorsque ces entrées diminuent, le temps perçu est surestimé ; lorsque ces entrées augmentent (stimulées), le temps perçu est sous-estimé
At the perceptual level, time is not a constant metric defined by the ticks of a clock. Distortions in time perception occur due to various factors, including attention deficits, emotions, sleep deprivation, arousal, motion, and others. This thesis explores how the vestibular system influences time perception. In the first part of the thesis, two studies were conducted during long-term space missions. We found that astronauts aboard the International Space Station accurately estimate short time delays over days. However, they tend to overestimate durations ranging from seconds to minutes and underestimate durations on the scale of hours. Given that time distortions in space can arise from multiple sources, we conducted a specific vestibular task to isolate the vestibular contribution. The second part of the thesis includes two studies on time perception during vestibular stimulation, focusing on whole-body rotations. In the first study, we found that time during rotations is underestimated compared to static conditions in healthy subjects. In the second study, we confirmed that this time underestimation was vestibular, as it was absent in Bilateral Vestibulopathy patients. Our findings suggest that time perception depends on vestibular inputs: when vestibular inputs are decreased, perceived time is overestimated; when vestibular inputs are increased (stimulated), perceived time is underestimated
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Kirchner, Hanni. "Cerebelläre Ataxie und bilaterale Vestibulopathie." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-164181.

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Aus den Erfahrungen in der Schwindelambulanz des Universitätsklinikums München, Campus Großhadern, und vorangegangener Publikationen [46, 62] hatten sich Hinweise auf einen möglichen pathophysiologischen Zusammenhang zwischen dem Bestehen einer (idopathischen) bilateralen Vestibulopathie (BV) und dem Auftreten cerebellärer Symptome ergeben. Nach Zingler et. al 2007 wiesen 25% aller Patienten mit BV ebenfalls cerebelläre Symptome auf und von diesen weiterhin 32% zusätzliche Anzeichen einer peripheren Polyneuropathie (PNP). Dabei waren vor allem die Patienten mit idiopathischer BV von diesem Syndrom betroffen. Ziel dieser Studie war es, das sog. CABV (cerebelläre Ataxie und bilaterale Vestibulopathie)- Syndrom durch klinische, elektrophysiologische und zusätzliche apparative Untersuchungen näher zu untersuchen und zu beschreiben, damit darauf aufbauend ggf. neue Diagnose- und Therapiemöglichkeiten entwickelt werden können. Dazu wurden aus dem Patientenkollektiv der Schwindelambulanz der vergangenen Jahre 31 Patienten ausgewählt, die an einem klinisch diagnostizierten CABV-Syndrom erkrankt waren, welches definiert war als das gleichzeitige Vorhandensein eines bds. pathologischen Kopfimpulstests als Ausdruck eines bilateralen, vestibulären Defizits und cerebellärer Symptomatik (Gang-, Stand-, Extremitätenataxie, cerebellärer Okulomotorikstörung und/oder Dysarthrie). Alle 31 Patienten erhielten eine kalorische Testung mittels Elektronystagmographie (ENG) oder Videookulographie (VOG), und wurden abhängig davon in zwei Gruppen eingeteilt: CACR- (= Cerebellar Ataxia; Caloric Responsiveness - : Patienten mit bds. pathologischem Kopfimpulstest und pathologischer Kalorik) und CACR+ (Cerebellar Ataxia; Caloric Responsiveness + : Patienten mit bds. pathologischem Kopfimpulstest ohne pathologische Kalorik). Alle Patienten erhielten eine ausführliche klinisch-neurologische und neuroophthalmologische Untersuchung, sowie weiterführende apparative Untersuchungen (EMG/NLG, cVEMP, Audiometrie, VBM, Search-Coil). Die Untersuchungsergebnisse der beiden Gruppen wurden dabei jeweils miteinander verglichen. Folgende Kernaussagen können basierend auf diesen Studien getroffen werden: 1. Aus den Messungen mittels Search-Coil-Technik ergab sich, dass einige der Patienten mit pathologischem Kopfimpulstest in der klinischen Untersuchung bei der apparativen Untersuchung des Kopfimpulstests einen normalen Gain aufwiesen. Da sich der Kopfimpulstest als Bedside-Test zur Diagnose einer BV bisher als geeignetes Verfahren durchgesetzt hatte, gehen wir davon aus, dass der falsch pathologische Kopfimpulstest möglicherweise durch das cerebelläre Syndrom beeinflusst worden sein könnte. 2. Beim Vergleich beider Studiengruppen zeigte sich in der Search-Coil-Untersuchung des Kopfimpulstests (horizontal head impulses, hHIT) ein signifikanter Unterschied im hHIT-Gain nach 80 und 100 ms, nicht aber bei 40 und nur teilweise bei 60 ms. Während in der Anfangsphase der VOR bei beiden Gruppen noch funktionierte, kam es in der CACR- Gruppe nach ca. 80 ms zu einem Zusammenbruch des Systems und zur Auslösung von Ausgleichssakkaden, während die CACR+ Patienten ihren hohen Gain aufrecht erhalten konnten. 3. Bei den CACR+ Patienten kam es signifikant später (nach ca. 200 ms) als bei den CACR- Patienten zur Generierung einer der Kopfbewegung entgegen gesetzten Augenbewegung. Diese könnte Ausdruck eines Blickhaltedefizits bei cerebellären bzw. flokkulären Läsionen sein und eine Korrektursakkade im Bedside-Kopfimpulstest vortäuschen und somit zu einem falsch-pathologischen Kopfimpulstest nach Halmagyi und Curthoys führen. 4. Zudem ist aufgrund zahlreicher anatomischer Verbindungen zwischen Vestibulocerebellum und der am VOR beteiligten Neuronen vorstellbar, dass der VOR durch das Cerebellum moduliert wird und sich auch cerebelläre Störungen in VOR-Defiziten bemerkbar machen können. 5. Bei cerebellären Patienten ist der Kopfimpulstest nach Halmagyi und Curthoys nicht ausreichend aussagekräftig und kann zu falsch-pathologischen Ergebnissen führen. Eine zusätzliche Diagnostik (Kalorik) ist zur Diagnose eines CABV-Syndroms notwendig. 6. Die cVEMP können bei cerebellären Patienten einen weiteren Hinweis auf das Vorliegen eines „echten“ CABV-Syndroms liefern, sind aber vor allem bei älteren Patienten (> 60 Jahre) nur eingeschränkt beurteilbar. 7. Die Beiteilung peripherer Nerven in Form einer Polyneuropathie ist regelmäßig bei CABV-Patienten nachzuweisen. Eine ausführliche PNP-Diagnostik sollte demnach Bestandteil der CABV-Diagnostik sein. 8. Die Hörnerven können ebenfalls im Rahmen eines CABV-Syndrom betroffen sein, weshalb die Durchführung einer Audiometrie sinnvoll erscheint. 9. Im Vergleich mit einem Normalkollektiv weisen CABV-Patienten wie zu erwarten eine bilaterale Kleinhirnatrophie auf, welche sowohl die Vermis als auch die Kleinhirnhemisphären betrifft.
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Fernandes, Virgínia Oliveira. "Prevalência de alterações vestibulares em indivíduos com síndrome metabólica e sua relação com risco cardiometabólico." reponame:Repositório Institucional da UFC, 2009. http://www.repositorio.ufc.br/handle/riufc/26841.

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FERNANDES, V. O. Prevalência de alterações vestibulares em indivíduos com síndrome metabólica e sua relação com risco cardiometabólico. 2009. 110 f. Dissertação (Mestrado em Saúde Pública) - Faculdade de Medicina, Universidade Federal do Ceará, Fortaleza, 2009.
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Metabolic Syndrome (MS) is a prevalent disorder that increases cardiovascular risk (CVR) and increases the risk to develop DM. Metabolic disorders are also related to vestibular symptoms as dizziness and vertigo. The inner ear spends much energy for its appropriate operation. Due to the high sensibility of the vestibular system, is very frequently the relationship between vestibular symptoms and metabolic abnormalities. This study intent to verify the occurrence of abnormalities in vestibular system using the vectoelectronystamography (VENG) in patients with metabolic disorders and its relationship with CVR. After a campaign to detect metabolic syndrome in a Health Center of Fortaleza, 78 individuals were evaluated. They were not diabetics or were using any medication. The age was 38.1 ± 10.7 years old and 74.4% were female. After clinical and laboratory evaluation, 38% were diagnosed with MS and 62% did not. Those groups were matched for sex and age. In the analysis of MS components, measures of BMI, waist circumference, systolic BP, diastolic BP, triglycerides, fasting glucose, glucose at 120 minutes were significantly higher in MS patients, whereas HDL-C was lower. Other metabolic parameters evalueted as uric acid GT, ALT, AST, and HOMA-IR were significantly higher in MS group. In the comparative analysis of both groups, the presence of vestibular disorders symptoms were found in 33.3% of the group without MS and in 63.3% of the MS group. When specific symptoms were analyzed, it was observed that the MS group reported most frequently dizziness and / or vertigo compared with the group without SM (p = 0.01). The findings of VENG in the MS group showed that 66.7% had anormal findings and Just against 20.8% of the group without MS. In MS group, we found a significantly higher prevalence of irritative disease than deficiency disease (p <0.0001). We conclude that patients with MS have a higher prevalence of vestibular-cochlear abnormalities comparing to individuals without MS. It occurs even in patients recently diagnosed and without other complications. That suggests that vestibular disorders are an early manifestation of MS. Thus, patients with vestibular complaints should be routinely investigated for metabolic abnormalities.
A Síndrome Metabólica (SM) é uma condição de alta prevalência que aumenta o risco cardiovascular (RCV) e de desenvolvimento de DM. Os distúrbios metabólicos também são relacionados a sintomas vestibulares como tontura e vertigem. A orelha interna despende muita energia para seu adequado funcionamento, sendo freqüente a relação de sintomas vestibulares com alterações do sistema metabólico. Esse estudo objetivou verificar a prevalência de alterações do sistema vestibular diagnosticada pela vectoeletronistagmografia (VENG) em portadores de SM, correlacionando-os com fatores de RCV. Foram avaliados 78 indivíduos atendidos em uma Campanha para detecção de Síndrome Metabólica em um Centro de Saúde de Fortaleza. Nenhum deles era diabético ou fazia uso de qualquer medicação. A idade variou de 38,1±10,7 anos e 74,4% eram do sexo feminino. Após a avaliação clínica e laboratorial, observou-se que 38% eram portadores de SM e 62% não. Os grupos estavam pareados para sexo, idade. Na análise quanto aos componentes da SM, as medidas de IMC, circunferência abdominal, PA sistólica, PA diastólica, triglicerídeos, glicemia de jejum, glicemia aos 120 minutos foram significativamente mais elevadas no com SM, enquanto o HDL-C foi mais baixo. Observou-se também que outros parâmetros metabólicos avaliados, como ácido úrico, GT, TGO, TGP, e HOMA-IR estavam significativamente mais elevados no grupo com SM. Na análise comparativa dos grupos quanto à presença de sintomas sugestivos de vestibulopatia, encontrou-se que 33,3% do grupo sem SM e 63,3% do grupo com SM apresentavam alguma sintomatologia. Quando analisados os sintomas específicos, observou-se que os indivíduos com SM referiram com maior frequência tontura e/ou vertigem, quando comparados com o grupo sem SM (p=0,01). Analisando os achados da VENG, encontrou-se que no grupo com SM 66,7% apresentavam VENG alterada contra 20,8% dos indivíduos sem SM. Nos indivíduos com SM, encontrou-se uma prevalência significativamente maior de doença irritativa que de doença deficitária (p<0,0001). Conclui-se que portadores de SM apresentam maior prevalência de alterações no aparelho vestibulo-coclear quando comparados a indivíduos sem SM, mesmo em pacientes com diagnóstico recente e sem outras complicações, sugerindo que as alterações no aparelho vestibular aparecem precocemente. Assim, pacientes com queixas de vestibulopatia deveriam ser rotineiramente investigados para a possibilidade de apresentarem alterações metabólicas
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Kirchner, Hanni [Verfasser], and Michael [Akademischer Betreuer] Strupp. "Cerebelläre Ataxie und bilaterale Vestibulopathie : Definition eines neuen Syndroms / Hanni Kirchner. Betreuer: Michael Strupp." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2013. http://d-nb.info/1046503162/34.

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Schmidt, Caroline. "Molekulargenetische Charakterisierung von Mutationen der mitochondrialen 12S rRNA bei Patienten mit bilateraler Vestibulopathie." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-77386.

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Nusser, Eva [Verfasser], and Klaus [Akademischer Betreuer] Jahn. "Einfluss einer galvanischen vestibulären Rauschstimulation auf die Gangstabilität bei gesunden Probanden und bei Patienten mit bilateraler Vestibulopathie / Eva Nusser ; Betreuer: Klaus Jahn." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2021. http://d-nb.info/123320064X/34.

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Müller, Karin. "Untersuchungen zur beidseitigen Vestibularisschädigung (Bilaterale Vestibulopathie) /." 2005. http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&doc_number=014169570&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA.

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Müller, Karin [Verfasser]. "Untersuchungen zur beidseitigen Vestibularisschädigung (Bilaterale Vestibulopathie) / von Karin Müller." 2005. http://d-nb.info/977752569/34.

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Carvalho, António Baptista. "Implantes vestibulares : uma nova abordagem terapêutica para a hipofunção vestibular bilateral." Master's thesis, 2019. http://hdl.handle.net/10451/42815.

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Trabalho Final do Curso de Mestrado Integrado em Medicina, Faculdade de Medicina, Universidade de Lisboa, 2019
O balanço, considerado um sexto sentido, está essencialmente à responsabilidade do aparelho vestibular e das suas múltiplas conexões, fazendo parte das suas variadas funções o controlo e estabilização postural, movimentos reflexos e perceção do movimento próprio. Assim, uma lesão deste sistema, a nível central ou periférico, poderá resultar numa assimetria das aferências nos centros vestibulares que, clinicamente, se poderá traduzir por sinais e sintomas como tonturas, vertigem, nistagmo, náuseas e vómitos. Atualmente, em relação às lesões que condicionam perda funcional bilateral dos órgãos vestibulares, as opções terapêuticas existentes são muito limitadas, cingindo-se essencialmente a fisioterapia associada a promoção e ativação dos sistemas compensatórios centrais e adaptação comportamental. Sendo evidente o impacto significativo que estes défices provocam ao nível da qualidade de vida destes doentes, é relevante e necessário procurar diferentes opções de tratamento com vista na melhoria da perspetiva futura para estes doentes. Como novas opções encontram-se a ser desenvolvidas próteses vestibulares, dispositivos que estimulam eletricamente o aparelho vestibular de forma a tentar restaurar a sua função. Apesar de se encontrarem ainda no início do seu desenvolvimento, o seu potencial e resultados em estudos preliminares em animais e humanos são promissores quanto a uma nova forma restituir parte da função vestibular e promover o bem-estar dos doentes com hipofunção bilateral.
The balance, considered a sixth sense, is essentially the responsibility of the vestibular apparatus and its multiple connections. Its functions include postural control and stabilization, reflex movements and perception of the movement itself. The balance, considered a sixth sense, is essentially the responsibility of the vestibular apparatus and its multiple connections, being part of its varied functions control and postural stabilization, reflex movements and perception of the movement itself. Thus, an injury to this system, at a central or peripheral level, may result in an asymmetry of the afferents in the vestibular centers, which, clinically, may be translated into signs and symptoms such as dizziness, vertigo, nystagmus, nausea and vomiting. Currently, in relation to the lesions that condition bilateral functional loss of the vestibular organs, the existing therapeutic options are very limited, essentially encompassing physiotherapy associated with the promotion and activation of central compensatory systems and behavioral adaptation. The significant impact of these deficits on the quality of life of these patients is evident, it is relevant and necessary to look at different treatment options with a view to improving the future prospects for these patients. As new options, vestibular prostheses are being developed, devices that electrically stimulate the vestibular apparatus to try to restore its function. Although they are still early in their development, their potential and results in preliminary animal and human studies are promising in a new way to restore part of the vestibular function and promote the well-being of patients with bilateral hypofunction.
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Schmidt, Caroline [Verfasser]. "Molekulargenetische Charakterisierung von Mutationen der mitochondrialen 12S-rRNA bei Patienten mit bilateraler Vestibulopathie / vorgelegt von Caroline Schmidt." 2007. http://d-nb.info/986865095/34.

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Books on the topic "Bilateral Vestibulopathy"

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Ward, Bryan K., and Alexander A. Tarnutzer, eds. Bilateral Vestibulopathy - Current Knowledge and Future Directions to Improve its Diagnosis and Treatment. Frontiers Media SA, 2018. http://dx.doi.org/10.3389/978-2-88945-628-4.

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Book chapters on the topic "Bilateral Vestibulopathy"

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Brandt, Thomas. "Bilateral vestibulopathy." In Vertigo, 127–41. New York, NY: Springer New York, 2003. http://dx.doi.org/10.1007/978-1-4757-3801-8_8.

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Brandt, Thomas. "Bilateral vestibulopathy." In Vertigo, 127–41. London: Springer London, 1999. http://dx.doi.org/10.1007/978-1-4471-0527-5_8.

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Albernaz, Pedro Luiz Mangabeira, Francisco Zuma e Maia, Sergio Carmona, Renato Valério Rodrigues Cal, and Guillermo Zalazar. "Bilateral Vestibulopathy." In The New Neurotology, 175–80. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11283-7_12.

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Zhang, Lei, and Yuanxing Chen. "Bilateral Vestibulopathy." In Experts' Perspectives on Medical Advances, 37–39. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-99-6995-1_9.

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Strupp, Michael, Thomas Brandt, and Marianne Dieterich. "Bilateral Vestibulopathy." In Vertigo and Dizziness, 99–118. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-030-78260-3_7.

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Albernaz, Pedro Luiz Mangabeira, Francisco Zuma e Maia, Sergio Carmona, Renato Valério Rodrigues Cal, and Guillermo Zalazar. "Bilaterale Vestibulopathie." In Die neue Neurootologie, 191–96. Cham: Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-51382-4_12.

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Strupp, Michael, F. Schautzer, D. A. Hamilton, R. Brüning, H. J. Markowitsch, R. Kalla, C. Darlington, P. Smith, and Thomas Brandt. "Chronische Bilaterale Vestibulopathie führt zu Störungen des räumlichen Gedächtnisses und einer beidseitigen Atrophie des Hippocampus." In Der Gleichgewichtssinn, 103–7. Vienna: Springer Vienna, 2008. http://dx.doi.org/10.1007/978-3-211-75432-0_12.

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"Bilateral Vestibulopathy." In Encyclopedia of Otolaryngology, Head and Neck Surgery, 327. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_100127.

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Strupp, M., K. Feil, M. Dieterich, and T. Brandt. "Bilateral vestibulopathy." In Handbook of Clinical Neurology, 235–40. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-444-63437-5.00017-0.

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Black, F. Owen. "Acquired bilateral peripheral vestibulopathy." In Vertigo and Imbalance: Clinical Neurophysiologyof the Vestibular System, 333–52. Elsevier, 2010. http://dx.doi.org/10.1016/s1567-4231(10)09026-x.

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Conference papers on the topic "Bilateral Vestibulopathy"

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Bohmann, Simon, Maximilian Oberste, and Claudia Rudack. "Differential diagnosis of a bilateral vestibulopathy." In 94th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1767463.

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Jadeed, R., A. Aljaber, J. Ilgner, and M. Westhofen. "Otolith dysfunction in bilateral vestibulopathys." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686410.

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Bohmann, Simon, Maximilian Oberste, and Claudia Rudack. "Differentialdiagnose einer bilateralen Vestibulopathie." In 94. Jahresversammlung Deutsche Gesellschaft für Hals-Nasen-Ohren-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1766825.

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Häußler, Marie Sophia, Katharina Stölzel, and Cristian Mihnea Trache. "Akute bilaterale Cochleo- und Vestibulopathie einer Patientin mit Anti-PD-L1-Antikörper-Therapie eines metastasierten nicht-kleinzelligen Bronchialkarzinoms." In 94. Jahresversammlung Deutsche Gesellschaft für Hals-Nasen-Ohren-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1766826.

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