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Journal articles on the topic 'Basal ganglla'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Redgrave, Peter. "Basal ganglia." Scholarpedia 2, no. 6 (2007): 1825. http://dx.doi.org/10.4249/scholarpedia.1825.

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2

Vaštík Kateřina Menšíková Sandra Kurčová, Miroslav, and Michaela Kaiserová Zuzana Matejčíková Petr Kaňovský. "Functional basal ganglia interconnection." Neurologie pro praxi 18, no. 4 (October 1, 2017): 266–70. http://dx.doi.org/10.36290/neu.2017.097.

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3

Aruna, S. I., S. Sujatha, and E. S. Neenu. "Mathematical Modelling of Basal Ganglia for Parkinson’s Disease: A System Biology Approach." Indian Journal Of Science And Technology 15, no. 36 (September 28, 2022): 1836–41. http://dx.doi.org/10.17485/ijst/v15i36.1397.

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4

SHARMA, DR VISHAL, DR N. M. KADRI DR. N. M. KADRI, DR CECIL PARMAR, DR MITESH CHANDARANA, DR INDRESH DIXIT, DR SWATI TRIVEDI, and DR KARAN DESAI. "A Case Report of Basal Ganglia Calcification - A Rare Finding of Hypoparathyroidism." International Journal of Scientific Research 3, no. 2 (June 1, 2012): 379–80. http://dx.doi.org/10.15373/22778179/feb2014/123.

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5

Brüggemann, Norbert, Susanne A. Schneider, Thurid Sander, Christine Klein, and Johann Hagenah. "Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification." Movement Disorders 25, no. 15 (August 27, 2010): 2661–64. http://dx.doi.org/10.1002/mds.23264.

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6

Tso, Winnie Wan-Yee, Ada Wing-Yan Yung, Hin-Yue Lau, and Godfrey Chi-Fung Chan. "Basal Ganglia Germinoma." Journal of Pediatric Hematology/Oncology 36, no. 7 (October 2014): e443-e447. http://dx.doi.org/10.1097/mph.0000000000000014.

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7

Patell, R. D., R. V. Dosi, H. K. Joshi, and P. C. Shah. "Basal ganglia calcification." Case Reports 2013, aug13 1 (August 13, 2013): bcr2013200399. http://dx.doi.org/10.1136/bcr-2013-200399.

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8

Albin, Roger L. "BASAL GANGLIA NEUROTOXINS." Neurologic Clinics 18, no. 3 (August 2000): 665–80. http://dx.doi.org/10.1016/s0733-8619(05)70217-6.

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9

Graybiel, Ann M. "The basal ganglia." Current Biology 10, no. 14 (July 2000): R509—R511. http://dx.doi.org/10.1016/s0960-9822(00)00593-5.

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10

Graybiel, Ann M. "The basal ganglia." Trends in Neurosciences 18, no. 2 (February 1995): 60–62. http://dx.doi.org/10.1016/0166-2236(95)80019-x.

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11

KONSTANTINOU, GERASIMOS, ANASTASIA STAVRINOU, SAVVINA MYLONA, GEORGE PASCHALAKIS, and POLYXENI VASILOPOULOU. "Basal Ganglia Calcification." Journal of Psychiatric Practice 25, no. 5 (September 2019): 391–94. http://dx.doi.org/10.1097/pra.0000000000000410.

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12

Braunlich, Kurt, and Carol Seger. "The basal ganglia." Wiley Interdisciplinary Reviews: Cognitive Science 4, no. 2 (December 21, 2012): 135–48. http://dx.doi.org/10.1002/wcs.1217.

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13

Wright, Christopher I., Bradley S. Peterson, and Scott L. Rauch. "Neuroimaging Studies in Tourette Syndrome." CNS Spectrums 4, no. 3 (March 1999): 54–61. http://dx.doi.org/10.1017/s1092852900000821.

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ABSTRACTTourette syndrome (TS) is a complex neurobehavioral disorder principally characterized by motor and vocal tics. However, features of obsessive-compulsive and attention-deficit/hyperactivity disorders are often present. The basal ganglia and associated brain structures have been implicated in the pathophysiology of TS, as well as in these related conditions. Specifically, it is believed that the neuroanatomically and functionally defined basal ganglia thalamocortical loops are involved in TS. These loops are composed of a sequence of connections originating in the cortex and passing in series through the striatum (caudate and putamen), globus pallidus, and thalamus before returning to the cortical areas of origin. This review concentrates on the neuroimaging findings in Ts, particularly as they relate to alterations in components of the basal ganglia thalamocortical circuits. These neuroimaging data suggest that the major abnormalities in TS involve striatal or cortical dysfunction, as well as dysfunction of dopaminergic systems that regulate basas ganglia neurotransmission.

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14

Persad, A., and V. Mehta. "P.112 Permanent isolated micrographia from traumatic basal ganglia injury." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 46, s1 (June 2019): S43. http://dx.doi.org/10.1017/cjn.2019.205.

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Background: Micrographia is a rare neurological finding in isolation. Most cases of isolated micrographia have been found in association with focal ischemia of the left basal ganglia. Methods: We present a case of post-traumatic micrographia stemming from contusion to the left basal ganglia. We performed a detailed analysis of the patient’s writing at three-year follow-up. Results: A halthy 15 year old male was admitted following a BM accident. CT showed contusion to the left basall ganglia/external capsule. MRI was negative for underlying lesion. He had a short stay in the ICU and then was discharged. Two years later, he expressed concern regarding difficulty with sma, cramped writing at school. Writing analysis revealed micrographia with spontaneous printing as well as printing to dictation, but not with copied English nor Japanese writing. Conclusions: Isolated micrographia is a rare neurological finding. We present the incidence of this symptom folllowing gliding contusion to the et basal ganglia and external capsule.

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15

Farries, Michael A. "How Basal' Are the Basal Ganglia?" Brain, Behavior and Evolution 82, no. 4 (2013): 211–14. http://dx.doi.org/10.1159/000356101.

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16

Sharma, Vikas, and Amitabh Goel. "Case of Bilateral MCA Infarct following Basal Ganglia Hemorrhage: A Rare Coincidence Case Report." International Journal of Neurology and Neurosurgery 10, no. 3 (2018): 280–82. http://dx.doi.org/10.21088/ijnns.0975.0223.10318.24.

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17

Mete, Ahmet. "Posttraumatic basal ganglia infarction: A rare complication in a child after mild head injury." Dicle Medical Journal 41, no. 4 (December 1, 2014): 746–47. http://dx.doi.org/10.5798/diclemedj.0921.2014.04.0511.

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18

Dogan, Ibrahim, Serdar Kahvecioglu, Unal Kurtoglu, Demet Yildiz, and Abdulmecit Yildiz. "Acute Chorea Characterized by Bilateral Basal Ganglia Lesions in a Patient with Diabetic Nephropathy." Turkish Nephrology Dialysis Transplantation 24, no. 01 (January 26, 2015): 110–12. http://dx.doi.org/10.5262/tndt.2015.1001.16.

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19

Wu, Min-Chi, Chiun-Li Chin, Wen-Chi Chin, Jian-Shiun Wu, Wan-Jou Li, and Ting-Yu Liu. "The Recognition of Basal Ganglia Area in the Brain MRI Image with Hybrid Classifier." International Journal of Information and Electronics Engineering 7, no. 1 (2017): 11–17. http://dx.doi.org/10.18178/ijiee.2017.7.1.654.

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20

Dale, Russell C., and Fabienne Brilot. "Autoimmune Basal Ganglia Disorders." Journal of Child Neurology 27, no. 11 (July 25, 2012): 1470–81. http://dx.doi.org/10.1177/0883073812451327.

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The basal ganglia are deep nuclei in the brain that include the caudate, putamen, globus pallidus, and substantia nigra. Pathological processes involving the basal ganglia often result in disorders of movement and behavior. A number of different autoimmune disorders predominantly involve the basal ganglia and can result in movement and psychiatric disorders. The classic basal ganglia autoimmune disorder is Sydenham chorea, a poststreptococcal neuropsychiatric disorder. Resurgence in the interest in Sydenham chorea is the result of the descriptions of other poststreptococcal neuropsychiatric disorders including tics and obsessive-compulsive disorder, broadly termed pediatric autoimmune neuropsychiatric disorders associated with streptococcal infection. Encephalitic processes affecting the basal ganglia are also described including the syndromes basal ganglia encephalitis, encephalitis lethargica, and bilateral striatal necrosis. Last, systemic autoimmune disorders such as systemic lupus erythematosus and antiphospholipid syndrome can result in chorea or parkinsonism. Using paradigms learned from other autoantibody associated disorders, the authors discuss the autoantibody hypothesis and the role of systemic inflammation in autoimmune basal ganglia disorders. Identification of these entities is important as the clinician has an increasing therapeutic repertoire to modulate or suppress the aberrant immune system.

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21

Hikosaka, Okihide. "Dopamine and Basal Ganglia." Psychiatry and Clinical Neurosciences 45, no. 2 (June 1991): 511. http://dx.doi.org/10.1111/j.1440-1819.1991.tb02535.x.

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22

Jellinger, K. "Traumatic basal ganglia hemorrhage." Neurology 40, no. 5 (May 1, 1990): 862. http://dx.doi.org/10.1212/wnl.40.5.862-a.

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23

Katz, D. I., and M. P. Alexander. "Traumatic basal ganglia hemorrhage." Neurology 40, no. 5 (May 1, 1990): 862. http://dx.doi.org/10.1212/wnl.40.5.862-b.

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24

BROOKS, DAVID J. "Imaging basal ganglia function." Journal of Anatomy 196, no. 4 (May 2000): 543–54. http://dx.doi.org/10.1046/j.1469-7580.2000.19640543.x.

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25

Jellinger, K. A. "The Basal Ganglia IX." European Journal of Neurology 17, no. 2 (December 7, 2009): e11-e11. http://dx.doi.org/10.1111/j.1468-1331.2009.02881.x.

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26

Alexander, Garrett E. "Basal Ganglia-Thalamocortical Circuits." Journal of Clinical Neurophysiology 11, no. 4 (July 1994): 420–31. http://dx.doi.org/10.1097/00004691-199407000-00004.

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27

Alexander B. Norinsky, D. O., and Vidhi J. Parikh. "Bilateral basal ganglia lesions." Visual Journal of Emergency Medicine 6 (January 2017): 29–30. http://dx.doi.org/10.1016/j.visj.2016.07.006.

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28

Chaudhuri, Abhijit, and Peter O. Behan. "Fatigue and basal ganglia." Journal of the Neurological Sciences 179, no. 1-2 (October 2000): 34–42. http://dx.doi.org/10.1016/s0022-510x(00)00411-1.

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29

Houk, Jim. "Models of basal ganglia." Scholarpedia 2, no. 10 (2007): 1633. http://dx.doi.org/10.4249/scholarpedia.1633.

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30

Alloway, Kevin. "Vibrissal basal ganglia circuits." Scholarpedia 8, no. 9 (2013): 7279. http://dx.doi.org/10.4249/scholarpedia.7279.

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31

Siderowf, A. "The Basal Ganglia V." Archives of Neurology 55, no. 2 (February 1, 1998): 255. http://dx.doi.org/10.1001/archneur.55.2.255.

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32

Malwitz, Kari, Judith H. Crossett, and Susan K. Schultz. "Idiopathic Basal Ganglia Calcification." American Journal of Geriatric Psychiatry 25, no. 3 (March 2017): S83—S84. http://dx.doi.org/10.1016/j.jagp.2017.01.095.

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33

Miocinovic, Svjetlana. "Beyond the Basal Ganglia." JAMA Neurology 71, no. 1 (January 1, 2014): 8. http://dx.doi.org/10.1001/jamaneurol.2013.4643.

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34

Da Cunha, Claudio, Suelen L. Boschen, Alexander Gómez-A, Erika K. Ross, William S. J. Gibson, Hoon-Ki Min, Kendall H. Lee, and Charles D. Blaha. "Toward sophisticated basal ganglia neuromodulation: Review on basal ganglia deep brain stimulation." Neuroscience & Biobehavioral Reviews 58 (November 2015): 186–210. http://dx.doi.org/10.1016/j.neubiorev.2015.02.003.

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35

Lou, Jau-Shin. "Pathophysiology of Basal Ganglia Disorders." CNS Spectrums 3, no. 2 (February 1998): 36–40. http://dx.doi.org/10.1017/s1092852900005514.

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AbstractParkinson's disease is the most common basal ganglia disorder that is caused by the degeneration of dopaminergic neurons in the substantia nigra. This article reviews the normal physiology of the basal ganglia in the normal state, as well as the pathophysiology of Parkinson's disease (PD) and other movement disorders associated with the basal ganglia. Also discussed is the pathophysiological basis for the surgical treatment of PD.

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36

Groenewegen, Henk J. "The Basal Ganglia and Motor Control." Neural Plasticity 10, no. 1-2 (2003): 107–20. http://dx.doi.org/10.1155/np.2003.107.

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This paper briefly reviews the functional anatomy of the basal ganglia and their relationships with the thalamocortical system. The basal ganglia, including the striatum, pallidum, subthalamic nucleus, and substantia nigra, are involved in a number of parallel, functionally segregated cortical-subcortical circuits. These circuits support a wide range of sensorimotor, cognitive and emotional-motivational brain functions. A main role of the basal ganglia is the learning and selection of the most appropriate motor or behavioral programs. The internal functional organization of the basal ganglia is very well suited for such selection mechanisms, both in development and in adulthood. The question of whether clumsiness may be, at least in part, attributed to dysfunction of the basal ganglia is discussed in the context of the differential, complementary, or interactive roles of the basal ganglia and the cerebellum in the development of motor control.

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37

Coleman, Melissa J., and Stephanie A. White. "Basal ganglia: Bursting with song." Current Biology 31, no. 12 (June 2021): R791—R793. http://dx.doi.org/10.1016/j.cub.2021.04.064.

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38

Ugawa, Yoshikazu. "Sensory input and basal ganglia." Rinsho Shinkeigaku 52, no. 11 (2012): 862–65. http://dx.doi.org/10.5692/clinicalneurol.52.862.

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39

Millichap, J. Gordon. "Basal Ganglia and Thalamic Infarction." Pediatric Neurology Briefs 11, no. 1 (January 1, 1997): 1. http://dx.doi.org/10.15844/pedneurbriefs-11-1-1.

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40

Maleki, Nasim, Lino Becerra, Lauren Nutile, Gautam Pendse, Jennifer Brawn, Marcelo Bigal, Rami Burstein, and David Borsook. "Migraine Attacks the Basal Ganglia." Molecular Pain 7 (January 2011): 1744–8069. http://dx.doi.org/10.1186/1744-8069-7-71.

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41

Brown, P. "Oscillations in the Basal Ganglia." Neuromodulation: Technology at the Neural Interface 6, no. 3 (June 26, 2003): 191. http://dx.doi.org/10.1046/j.1525-1403.2003.03027_2.x.

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42

Madu, Ezemonye, and Hamid Alam. "Chorea, Hyperglycemia, Basal Ganglia Syndrome." Journal of the American Osteopathic Association 115, no. 7 (July 1, 2015): 465. http://dx.doi.org/10.7556/jaoa.2015.099.

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43

Barker, Roger A. "The Basal Ganglia and Pain." International Journal of Neuroscience 41, no. 1-2 (January 1988): 29–34. http://dx.doi.org/10.3109/00207458808985739.

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44

Wichmann, Thomas, and Mahlon R. DeLong. "Oscillations in the basal ganglia." Nature 400, no. 6745 (August 1999): 621–22. http://dx.doi.org/10.1038/23148.

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45

Fernández-Ruiz, J., I. Lastres-Becker, A. Cabranes, S. González, and J. A. Ramos. "Endocannabinoids and basal ganglia functionality." Prostaglandins, Leukotrienes and Essential Fatty Acids (PLEFA) 66, no. 2-3 (February 2002): 257–67. http://dx.doi.org/10.1054/plef.2001.0350.

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46

Inzelberg, R., N. M. Bornstein, I. Reider, and A. D. Korczyn. "Basal Ganglia Lacunes and Parkinsonism." Neuroepidemiology 13, no. 3 (1994): 108–12. http://dx.doi.org/10.1159/000110367.

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47

Md Noh, M. S. F., and A. M. Abdul Rashid. "The disappearing basal ganglia sign." QJM: An International Journal of Medicine 111, no. 5 (December 7, 2017): 343. http://dx.doi.org/10.1093/qjmed/hcx240.

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48

Takeda, Naoya, Katsuzo Fujita, Shigenori Katayama, Yoshito Uchihashi, Yusuke Okamura, Hiroyuki Nigami, Kimio Hashimoto, and Eiji Kohmura. "Germinoma of the Basal Ganglia." Pediatric Neurosurgery 40, no. 6 (2004): 306–11. http://dx.doi.org/10.1159/000083745.

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49

Saenz, Rocky C. "The Disappearing Basal Ganglia Sign." Radiology 234, no. 1 (January 2005): 242–43. http://dx.doi.org/10.1148/radiol.2341030252.

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50

Song, Cheng-Yuan, Zhen-Xiang Zhao, Wei Li, Cong-Cong Sun, and Yi-Ming Liu. "Pseudohypoparathyroidism with basal ganglia calcification." Medicine 96, no. 11 (March 2017): e6312. http://dx.doi.org/10.1097/md.0000000000006312.

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