Relevant bibliographies by topics / Basal ganglla

Academic literature on the topic 'Basal ganglla'

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Published: 10 December 2022
Last updated: 28 January 2023

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Journal articles on the topic "Basal ganglla"

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Redgrave, Peter. "Basal ganglia." Scholarpedia 2, no. 6 (2007): 1825. http://dx.doi.org/10.4249/scholarpedia.1825.

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Vaštík Kateřina Menšíková Sandra Kurčová, Miroslav, and Michaela Kaiserová Zuzana Matejčíková Petr Kaňovský. "Functional basal ganglia interconnection." Neurologie pro praxi 18, no. 4 (October 1, 2017): 266–70. http://dx.doi.org/10.36290/neu.2017.097.

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Aruna, S. I., S. Sujatha, and E. S. Neenu. "Mathematical Modelling of Basal Ganglia for Parkinson’s Disease: A System Biology Approach." Indian Journal Of Science And Technology 15, no. 36 (September 28, 2022): 1836–41. http://dx.doi.org/10.17485/ijst/v15i36.1397.

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SHARMA, DR VISHAL, DR N. M. KADRI DR. N. M. KADRI, DR CECIL PARMAR, DR MITESH CHANDARANA, DR INDRESH DIXIT, DR SWATI TRIVEDI, and DR KARAN DESAI. "A Case Report of Basal Ganglia Calcification - A Rare Finding of Hypoparathyroidism." International Journal of Scientific Research 3, no. 2 (June 1, 2012): 379–80. http://dx.doi.org/10.15373/22778179/feb2014/123.

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Brüggemann, Norbert, Susanne A. Schneider, Thurid Sander, Christine Klein, and Johann Hagenah. "Distinct basal ganglia hyperechogenicity in idiopathic basal ganglia calcification." Movement Disorders 25, no. 15 (August 27, 2010): 2661–64. http://dx.doi.org/10.1002/mds.23264.

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Tso, Winnie Wan-Yee, Ada Wing-Yan Yung, Hin-Yue Lau, and Godfrey Chi-Fung Chan. "Basal Ganglia Germinoma." Journal of Pediatric Hematology/Oncology 36, no. 7 (October 2014): e443-e447. http://dx.doi.org/10.1097/mph.0000000000000014.

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Patell, R. D., R. V. Dosi, H. K. Joshi, and P. C. Shah. "Basal ganglia calcification." Case Reports 2013, aug13 1 (August 13, 2013): bcr2013200399. http://dx.doi.org/10.1136/bcr-2013-200399.

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Albin, Roger L. "BASAL GANGLIA NEUROTOXINS." Neurologic Clinics 18, no. 3 (August 2000): 665–80. http://dx.doi.org/10.1016/s0733-8619(05)70217-6.

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Graybiel, Ann M. "The basal ganglia." Current Biology 10, no. 14 (July 2000): R509—R511. http://dx.doi.org/10.1016/s0960-9822(00)00593-5.

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Graybiel, Ann M. "The basal ganglia." Trends in Neurosciences 18, no. 2 (February 1995): 60–62. http://dx.doi.org/10.1016/0166-2236(95)80019-x.

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Dissertations / Theses on the topic "Basal ganglla"

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Moss, Jonathan. "Microcircuitry of the Basal Ganglia." Thesis, University of Oxford, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.514971.

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Drinnan, Suzane Loraine. "G proteins in the basal ganglia." Thesis, University of British Columbia, 1990. http://hdl.handle.net/2429/28981.

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G proteins are alpha-beta-gamma heterotrimers in the resting state, bound to GDP and complexed with the unbound receptor. Once the receptor becomes occupied, the alpha subunit exchanges GDP for GTP, becomes activated, and dissociates from the receptor and can stimulate or inhibit many intracellular activities such as phosphorylation and channel conductance. For example, Gs and Golf alpha subunits stimulate and Gi alpha subunits inhibit adenylyl cyclase. Go alpha subunits are abundant in brain, but are of unknown function. cDNAs for the alpha subunit have been cloned. In order to examine the relative distributions of G proteins in the brain, we used in situ hybridization with radiolabelled synthetic oligonucleotide probes. By using a tyrosine hydroxylase antibody, we found that the dopaminergic neurons of the substantia nigra and the noradrenergic neurons of the locus ceruleus express mRNA for the alpha subunits for each of Gi, Go, and Gs. We noted a paucity of Gs mRNA in the striatum. This was surprising because the basal ganglia contain a dopamine-stimulated adenylyl cyclase activity which has been assumed to be transduced by Gs. Also, immunohistochemistry, immunoblotting, and cholera ADP-ribosylation indicated a very high level of Gs alpha-like protein in the striatum. In order to ascertain which specific G protein we were detecting, we made probes to a new G protein previously identified in the olfactory system. Golf is a stimulatory G protein with size and sequence characteristics similar to those of Gs. The cholera toxin ADP-ribosylation site and C-terminal region to which the antibody was made are identical. We made oligonucloetide probes to the translated and untranslated portions of Golf alpha. High levels Golf mRNA and protein were detected in the striatum and nucleus accumbens, in addition to the expected high levels in the olfactory tubercle. Northern blot studies indicated that Golf transcripts are approximately ten-fold more abundant than Gs alpha transcripts in the striatum. These data indicate that Golf in not an olfactory-specific G protein. It is also the major stimulatory G protein in the basal ganglia. The selective expression of high levels of Golf in dopamine-rich forebrain areas suggest that it may couple DI dopamine receptors to adenylyl cyclase. The role of Golf in dopaminergic neurotransmission and neuropsychiatric disease should be considered.
Medicine, Faculty of
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Mogoseanu, Diana. "Basal ganglia connections with orofacial muscles." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260710.

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Smith, Denise P. A. "The Basal Ganglia and Sequential Learning." Kent State University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=kent1353430597.

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van, Albada Sacha. "Mean-field analysis of basal ganglia and thalamocortical dynamics." Connect to full text, 2008. http://ses.library.usyd.edu.au/handle/2123/5124.

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Thesis (Ph. D.)--University of Sydney, 2009.
Includes graphs and tables. Includes list of publications. Title from title screen (viewed June 17, 2009) Submitted in fulfilment of the requirements for the degree of Doctor of Philosophy to the School of Physics, Faculty of Science. Degree awarded 2009; thesis submitted 2008. Includes bibliographical references. Also available in print form.
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Church, Andrew John. "Anti-basal ganglia antibodies in movement disorders." Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1444607/.

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Sydenham's chorea (SC) is a neurological manifestation following group A Streptococcus infection (GABHS) and has been proposed as an antibody-mediated autoimmune disease. Other movement and psychiatric manifestations following GABHS have been recognised and termed Paediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal infections (PANDAS). It is proposed that PANDAS may be caused by the same antibody as SC. As the symptoms of PANDAS are identical to Tourette's syndrome (TS), the possibility that TS might turn out to be an autoimmune disorder has implications for the treatment and understanding of these disorders. Evidence of GABHS was found in all patients with SC and PANDAS and 60% of patients with TS. Autoantibodies against basal ganglia (ABGA) were found in all acute SC and PANDAS patients. Only 25% of TS patients were ABGA positive. There was little evidence for ABGA in controls. There was a higher prevalence of ABGA in systemic diseases associated with GABHS but this did not reach significance. ABGA bound to proteins with molecular weights (40, 45, 60 and 98 kDa) and these responses were variably found in SC, PANDAS and TS. The identification of these antigens proved to be problematic due to contamination with other proteins with the same molecular weights. Neurone specific enolase (NSE) was identified as one of the antigens. As this protein was not specific to basal ganglia it cast doubt as to the specificity of ABGA. Interestingly, however, enolase is also found on the surface of GABHS and has extensive homology with human enolase, thus lending support to the possibility of molecular mimicry derived autoimmunity.
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Brown, Jennifer. "Feedback motor control and the basal ganglia." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648678.

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Noy, G. "Abnormal motor behaviour and the basal ganglia." Thesis, University of Manchester, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.370974.

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Hutton, Elizabeth Anne May. "Somatosensory cortical input to the basal ganglia." Thesis, University of Edinburgh, 1998. http://webex.lib.ed.ac.uk/abstracts/hutton01.pdf.

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Deus, Yela Juan. "Sistema fronto-basal y aprendizaje incidental." Doctoral thesis, Universitat de Barcelona, 1996. http://hdl.handle.net/10803/670405.

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La memoria no es un proceso unitario. Se han definido diferentes subsistemas mnesicos que operan mediante circuitos neuroanatómicos distintos. Tradicionalmente, los estudios de la memoria se han centrado en el aprendizaje intencionado, especificando que el lóbulo temporal y las estructuras diencefálicas son esenciales para este tipo de memoria. Contrariamente, hay pocos estudios sobre el aprendizaje incidental y no han concretado las estructuras anatómicas implicadas en este aprendizaje. Ya que se considera que este último tipo de aprendizaje es un proceso mnésico automático de adquisición de la información, cabe presuponer la posible implicacion del estriado, de forma similar a lo que ocurre con el sistema motor. Se han seleccionado una muestra de 114 sujetos, 70 como pacientes y 44 pertenecen al grupo control de sujetos sanos. La muestra de pacientes está constituida por sujetos afectos de la enfermedad de Alzheimer (N=20), de la enfermedad de Huntington (N=21), pacientes con calcificaciones bilaterales en los ganglios basales (N=18) y pacientes con una lesión vascular unilateral en el estriado (N=11). Se ha diseñado una exploración neuropsicológica que ha evaluado funciones cognitivas generales, visoperceptivas y visoespaciales, aprendizaje intencionado, aprendizaje procedimental y funciones frontales en base a pruebas clínicamente estandarizadas. De forma expresa, se han diseñado tareas para evaluar el aprendizaje incidental del espacio, frecuencia y orden temporal. Nuestros resultados sugieren que el aprendizaje incidental puede estar controlado por circuitos frontoneoestriados bilaterales y puede utilizarse como un indicador del funcionamiento mnésico que permita ayudar a discriminar entre la demencia subcortical y cortical, considerando conjuntamente el funcionamiento de la memoria declarativa y procedimental.
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Books on the topic "Basal ganglla"

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Soghomonian, Jean-Jacques, ed. The Basal Ganglia. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42743-0.

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International Basal Ganglia Society. Symposium. The basal ganglia VII. New York: Kluwer Academic/Plenum Publishers, 2002.

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International Basal Ganglia Society. Symposium. The basal ganglia III. New York: Plenum Press, 1991.

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Meeting, International Basal Ganglia Society. The basal ganglia V. New York: Plenum, 1996.

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Pieter, Voorn, Berendse Henk W, Mulder Antonius B, Cools Alexander Rudolf 1941-, and SpringerLink (Online service), eds. The Basal Ganglia IX. New York, NY: Springer-Verlag New York, 2009.

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P, Bolam J., Ingham C. A, and Magill Peter James, eds. The basal ganglia VIII. New York: Springer, 2006.

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International Basal Ganglia Society. Symposium. The basal ganglia VIII. New York: Springer, 2005.

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Bolam, J. Paul, Cali A. Ingham, and Peter J. Magill, eds. The Basal Ganglia VIII. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-28066-9.

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Percheron, Gérard, John S. McKenzie, and Jean Féger, eds. The Basal Ganglia IV. Boston, MA: Springer US, 1994. http://dx.doi.org/10.1007/978-1-4613-0485-2.

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Carpenter, Malcolm B., and A. Jayaraman, eds. The Basal Ganglia II. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5347-8.

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Book chapters on the topic "Basal ganglla"

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Marmarou, Christina R., Matthew R. Parry, and Ekaterina Dobryakova. "Basal Ganglia." In Encyclopedia of Clinical Neuropsychology, 481–87. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_298.

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Marmarou, Christina R., Matthew R. Parry, and Ekaterina Dobryakova. "Basal Ganglia." In Encyclopedia of Clinical Neuropsychology, 1–7. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_298-2.

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Cho, Youngsun T. "Basal Ganglia." In Encyclopedia of Autism Spectrum Disorders, 1–2. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-6435-8_102059-1.

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McKinlay, Audrey. "Basal Ganglia." In Encyclopedia of Child Behavior and Development, 206–9. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-79061-9_287.

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ten Donkelaar, Hans J., Bart van de Warrenburg, Michèl Willemsen, Benno Küsters, Yoshio Hashizume, and Akira Hori. "Basal Ganglia." In Clinical Neuroanatomy, 591–667. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-41878-6_11.

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Cho, Youngsun T. "Basal Ganglia." In Encyclopedia of Autism Spectrum Disorders, 600–602. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-91280-6_102059.

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Ewert, Jörg-Peter. "Basal Ganglia." In Encyclopedia of Sciences and Religions, 187. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-1-4020-8265-8_200144.

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ten Donkelaar, Hans J., Bart van de Warrenburg, Michèl Willemsen, Benno Küsters, Yoshio Hashizume, and Akira Hori. "Basal Ganglia." In Clinical Neuroanatomy, 495–564. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-19134-3_11.

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Marmarou, Christina R., and Matthew R. Parry. "Basal Ganglia." In Encyclopedia of Clinical Neuropsychology, 346–51. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_298.

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Rubin, Jonathan. "Basal Ganglia: Overview." In Encyclopedia of Computational Neuroscience, 3–7. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4614-6675-8_760.

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Conference papers on the topic "Basal ganglla"

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Miglierini, Marcel, Adriana Lančok, Martin Kopáni, and Roman Boča. "Mössbauer spectroscopy of Basal Ganglia." In MOSSBAUER SPECTROSCOPY IN MATERIALS SCIENCE - 2014. AIP Publishing LLC, 2014. http://dx.doi.org/10.1063/1.4898624.

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Mohagheghi-Nejad, Mohammad Reza, Fariba Bahrami, and Mahyar Janahmadi. "Conductance-based computational model of basal ganglia." In 2014 22nd Iranian Conference on Electrical Engineering (ICEE). IEEE, 2014. http://dx.doi.org/10.1109/iraniancee.2014.6999867.

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Medini, Chaitanya, Anjitha Thekkekuriyadi, Surya Thayyilekandi, Manjusha Nair, Bipin Nair, and Shyam Diwakar. "Modeling basal ganglia microcircuits using spiking neurons." In 2016 International Conference on Advances in Computing, Communications and Informatics (ICACCI). IEEE, 2016. http://dx.doi.org/10.1109/icacci.2016.7732383.

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Gilmour, Timothy P., Constantino Lagoa, W. Kenneth Jenkins, Anand N. Rao, Matthew A. Berk, Kala Venkiteswaran, and Thyagarajan Subramanian. "Transfer entropy between cortical and basal ganglia electrophysiology." In 2012 IEEE Signal Processing in Medicine and Biology Symposium (SPMB). IEEE, 2012. http://dx.doi.org/10.1109/spmb.2012.6469453.

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Song, Enmin, Valerie A. Cardenas, Frank Ezekiel, and Michael W. Weiner. "Automatic boundary modification of warped basal ganglia template." In Medical Imaging 2001, edited by Milan Sonka and Kenneth M. Hanson. SPIE, 2001. http://dx.doi.org/10.1117/12.431023.

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Elibol, Rahmi, and Neslihan Serap Sengor. "Modeling basal ganglia circuits with mass model equations." In 2016 Medical Technologies National Congress (TIPTEKNO). IEEE, 2016. http://dx.doi.org/10.1109/tiptekno.2016.7863131.

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Souza, Felipe Fanine de, Ana Luiza da Silva Wendhausen, Felipe Reinert Avilla Machado, Gustavo Figueiredo da Silva, Maria Eduarda Angelo de Mendonça Fileti, Raddib Eduardo Noleto da Nóbrega de Oliveira, Rafael Pereira Guimarães, et al. "Chorea in a Non-Ketotic Hyperglycemic State: Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.099.

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Context: Non-ketotic hyperglycemia is a rare cause of chorea. Clinical findings are usually unilateral and potentially reversible after treatment for hyperglycemia. Hyperglycemia leads to asymmetric multifocal petechial hemorrhages of the basal ganglia, leading to a dysfunction of neuronal networks that connect the basal ganglia and the motor cortical areas, mainly affecting the subthalamic nucleus and contralateral striatum, which is highlighted by typical hyperdense lesions of the basal ganglia in computed tomography (CT) of the brain. This study aimed to report a case of a patient with choreiform movements due to a rare etiology of hyperglycemia nonketotic in a Hospital Public of Joinville, SC. The study was carried out through the collection and analysis of a patient’s medical record. Case report: Female patient, 54 years old, who presented for 6 days choreiform movements in the face, left upper limb and, discreetly, in the left lower limb. Snake tongue sign and milkmaid’s grip positive, without dysarthria. In the laboratory exam, glucose of 600 mg / dL; without further changes. Cranial tomography showed hyperdensity in the putamen region on the right. The treatment was started to obtain better glycemic control and Risperidone 3 mg / day. Conclusions: It is concluded, then, that non-ketotic hyperglycemia is an uncommon, but reversible cause of chorea, and may manifest itself due to an uncontrolled non-ketotic diabetes mellitus. Its pathogenic mechanism remains to be clarified. In addition, clinical, epidemiological, imaging and laboratory findings, together, corroborate for early diagnosis and proper management.
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Monteiro, Ana Karoline da Costa, Paulo Filho Soares Marcelino, Marcello Holanda de Andrade, Rairis Barbosa Nascimento, Marx Lincoln Lima de Barros Araújo, and Samuel Pinheiro da Silva. "Fahr’s Syndrome: A Case Report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.110.

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Context: Fahr’s syndrome is a clinical entity of primary or secondary causes characterized by neurological and/or psychiatric symptoms associated with abnormal calcifications in basal ganglia, cerebellum and cerebral cortex. Case report: G.M.A, female, 49 years-old, presented athetosis in the distal extremity of the right upper limb (RUL) in December 2020, without seeking for medical help. Known to be diabetic, hypertensive and with diastolic heart failure (HF), in February 2021 she presented decompensated HF associated with worsening of involuntary movements in the RUL. During investigation, abnormalities in serum levels of parathormone (PTH) and calcium were observed, in addition to bilateral calcifications in basal ganglia and thalamus in brain computed tomography (CT) without contrast. In March 2021, she was admitted to the University Hospital of the Federal University of Piauí, with maintenance of athetosis at the distal extremity of the RUL, in addition to oromandibular dyskinesia. Laboratory profile compatible with primary hypoparathyroidism and new brain CT without contrast with calcifications in basal ganglia and pulvinar of the thalamus, bilaterally, were confirmed. Intravenous calcium replacement and use of calcitriol were performed, with partial clinical improvement. Conclusions: Fahr’s syndrome is characterized by the presence of movement disorders (with highlights for parkinsonism and athetosis) and psychiatric symptoms (depression most commonly). Possible etiologies are primary (genetic) and secondary (mainly idiopathic or secondary hypoparathyroidism). Classic finding of symmetrical calcifications in base nuclei is seen on brain CT. Treatment is symptomatic and control of the underlying disease.
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Cavalcante Franca Junior, Marcondes, and Lara GusmÃo Vicente Dos Anjos. "Basal Ganglia abnormalities in ALS: Texture and volumetric analysis." In XXIII Congresso de Iniciação Científica da Unicamp. Campinas - SP, Brazil: Galoá, 2015. http://dx.doi.org/10.19146/pibic-2015-37478.

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Guiyeom Kang and M. M. Lowery. "Conductance-based model of the basal ganglia in Parkinson's Disease." In IET Irish Signals and Systems Conference (ISSC 2009). IET, 2009. http://dx.doi.org/10.1049/cp.2009.1692.

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Reports on the topic "Basal ganglla"

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Arbib, Michael A. Mechanisms of the Basal Ganglia for Arm-Hand Coordination. Fort Belvoir, VA: Defense Technical Information Center, September 1996. http://dx.doi.org/10.21236/ada315470.

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Andrades, Oscar, David Ulloa, Dario Martinez, Francisco Guede, Gustava Muñoz, Luis Javier Chirosa, and Amador García. Effect of the manipulation of the variables that configure the stimulus of strength training on motor symptoms in people with Parkinson's disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0079.

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Review question / Objective: To analyze the evidence on studies that have manipulated the variables that make up the strength training stimulus and its effects on motor symptoms in people with Parkinson's disease. Condition being studied: Parkinson's is a multisystemic neurodegenerative disease that affects the central nervous system and is caused by a loss of dopaminergic neurons in the compact part of the substantia nigra of the basal ganglia of the midbrain. People with Parkinson's disease (PEP) have non-motor and motor clinical symptoms. Classic motor symptoms are rest tremor, joint stiffness, bradykinesia, decreased balance, gait disturbances (speed, temporality, spatiality, support, and freezing) and decreased functional performance.
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Wang, Xiao, Hong Shen, Yujie Liang, Yixin Wang, Meiqi Zhang, and Hongtao Ma. Effectiveness of Tango Intervention on Motor Symptoms in Patients with Parkinson's Disease: A Protocol for Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0009.

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Review question / Objective: Parkinson's disease (PD) is a degenerative neurological disease caused by the loss of dopaminergic neurons in the pars compacta of the substantia nigra of the brain, resulting in lesions in the basal ganglia. The main motor symptoms of PD include resting tremor, rigidity, akinesia or bradykinesia and postural instability. As an exercise intervention based on musical accompaniment, tango dance has shown positive effects on the rehabilitation of motor symptoms in PD patients in recently. In this study, we systematically reviewed the efficacy of tango intervention in alleviating the motor symptoms of patients with PD. Condition being studied: Parkinson. Information sources: The following electronic databases will be searched: PubMed, Cochrane Central Register of Controlled Trials (CENTRAL), Web of Science Core collection, and China National Knowledge Infrastructure Database (CNKI) and WanFang Database.
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Utility of the ADC Map in Identification of Diffusion Restriction on a Background of Basal Ganglia Iron Deposition. Science Repository OU, April 2019. http://dx.doi.org/10.31487/j.rdi.2019.02.01.

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