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Academic literature on the topic 'Barhl1'

Author: Grafiati

Published: 25 May 2024

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Journal articles on the topic "Barhl1"

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Hou, Kun, Hui Jiang, Md Rezaul Karim, Chao Zhong, Zhouwen Xu, Lin Liu, Minxin Guan, Jianzhong Shao, and Xiao Huang. "A Critical E-box in Barhl1 3′ Enhancer Is Essential for Auditory Hair Cell Differentiation." Cells 8, no. 5 (May 15, 2019): 458. http://dx.doi.org/10.3390/cells8050458.

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Barhl1, a mouse homologous gene of Drosophila BarH class homeobox genes, is highly expressed within the inner ear and crucial for the long-term maintenance of auditory hair cells that mediate hearing and balance, yet little is known about the molecular events underlying Barhl1 regulation and function in hair cells. In this study, through data mining and in vitro report assay, we firstly identified Barhl1 as a direct target gene of Atoh1 and one E-box (E3) in Barhl1 3’ enhancer is crucial for Atoh1-mediated Barhl1 activation. Then we generated a mouse embryonic stem cell (mESC) line carrying disruptions on this E3 site E-box (CAGCTG) using CRISPR/Cas9 technology and this E3 mutated mESC line is further subjected to an efficient stepwise hair cell differentiation strategy in vitro. Disruptions on this E3 site caused dramatic loss of Barhl1 expression and significantly reduced the number of induced hair cell-like cells, while no affections on the differentiation toward early primitive ectoderm-like cells and otic progenitors. Finally, through RNA-seq profiling and gene ontology (GO) enrichment analysis, we found that this E3 box was indispensable for Barhl1 expression to maintain hair cell development and normal functions. We also compared the transcriptional profiles of induced cells from CDS mutated and E3 mutated mESCs, respectively, and got very consistent results except the Barhl1 transcript itself. These observations indicated that Atoh1-mediated Barhl1 expression could have important roles during auditory hair cell development. In brief, our findings delineate the detail molecular mechanism of Barhl1 expression regulation in auditory hair cell differentiation.

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Chellappa, Ramesh, Shengguo Li, Sarah Pauley, Israt Jahan, Kangxin Jin, and Mengqing Xiang. "Barhl1 Regulatory Sequences Required for Cell-Specific Gene Expression and Autoregulation in the Inner Ear and Central Nervous System." Molecular and Cellular Biology 28, no. 6 (January 22, 2008): 1905–14. http://dx.doi.org/10.1128/mcb.01454-07.

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ABSTRACT The development of the nervous system requires the concerted actions of multiple transcription factors, yet the molecular events leading to their expression remain poorly understood. Barhl1, a mammalian homeodomain transcription factor of the BarH class, is expressed by developing inner ear hair cells, cerebellar granule cells, precerebellar neurons, and collicular neurons. Targeted gene inactivation has demonstrated a crucial role for Barhl1 in the survival and/or migration of these sensory cells and neurons. Here we report the regulatory sequences of Barhl1 necessary for directing its proper spatiotemporal expression pattern in the inner ear and central nervous system (CNS). Using a transgenic approach, we have found that high-level and cell-specific expression of Barhl1 within the inner ear and CNS depends on both its 5′ promoter and 3′ enhancer sequences. Further transcriptional, binding, and mutational analyses of the 5′ promoter have identified two homeoprotein binding motifs that can be occupied and activated by Barhl1. Moreover, proper Barhl1 expression in inner ear hair cells and cerebellar and precerebellar neurons requires the presence of Atoh1. Together, these data delineate useful Barhl1 regulatory sequences that direct strong and specific gene expression to inner ear hair cells and CNS sensory neurons, establish a role for autoregulation in the maintenance of Barhl1 expression, and identify Atoh1 as a key upstream regulator.

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Li, Shengguo, Sandy M. Price, Hugh Cahill, David K. Ryugo, Michael M. Shen, and Mengqing Xiang. "Hearing loss caused by progressive degeneration of cochlear hair cells in mice deficient for the Barhl1 homeobox gene." Development 129, no. 14 (July 15, 2002): 3523–32. http://dx.doi.org/10.1242/dev.129.14.3523.

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The cochlea of the mammalian inner ear contains three rows of outer hair cells and a single row of inner hair cells. These hair cell receptors reside in the organ of Corti and function to transduce mechanical stimuli into electrical signals that mediate hearing. To date, the molecular mechanisms underlying the maintenance of these delicate sensory hair cells are unknown. We report that targeted disruption of Barhl1, a mouse homolog of the Drosophila BarH homeobox genes, results in severe to profound hearing loss, providing a unique model for the study of age-related human deafness disorders. Barhl1 is expressed in all sensory hair cells during inner ear development, 2 days after the onset of hair cell generation. Loss of Barhl1 function in mice results in age-related progressive degeneration of both outer and inner hair cells in the organ of Corti, following two reciprocal longitudinal gradients. Our data together indicate an essential role for Barhl1 in the long-term maintenance of cochlear hair cells, but not in the determination or differentiation of these cells.

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Lopes, Carmela, Anne-Lise Delezoide, Jean-Maurice Delabar, and Mohammed Rachidi. "BARHL1 homeogene, the human ortholog of the mouse Barhl1 involved in cerebellum development, shows regional and cellular specificities in restricted domains of developing human central nervous system." Biochemical and Biophysical Research Communications 339, no. 1 (January 2006): 296–304. http://dx.doi.org/10.1016/j.bbrc.2005.11.021.

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Dong, Hongyan, Carole L. Yauk, and Michael G. Wade. "Barhl1 is directly regulated by thyroid hormone in the developing cerebellum of mice." Biochemical and Biophysical Research Communications 415, no. 1 (November 2011): 157–62. http://dx.doi.org/10.1016/j.bbrc.2011.10.041.

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Pöschl, J., A. Lorenz, W. Hartmann, A. O. von Bueren, M. Kool, S. Li, A. Peraud, et al. "Expression of BARHL1 in medulloblastoma is associated with prolonged survival in mice and humans." Oncogene 30, no. 47 (May 23, 2011): 4721–30. http://dx.doi.org/10.1038/onc.2011.173.

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Sud, Richa, Chris M. Jones, Sandro Banfi, and Sally J. Dawson. "Transcriptional regulation by Barhl1 and Brn-3c in organ of corti derived cell lines." Molecular Brain Research 141, no. 2 (November 2005): 174–80. http://dx.doi.org/10.1016/j.molbrainres.2005.09.007.

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Barh, Debmalya, María García-Solano, Sandeep Tiwari, Antaripa Bhattacharya, Neha Jain, Daniel Torres-Moreno, Belén Ferri, et al. "BARHL1 Is Downregulated in Alzheimer’s Disease and May Regulate Cognitive Functions through ESR1 and Multiple Pathways." Genes 8, no. 10 (September 28, 2017): 245. http://dx.doi.org/10.3390/genes8100245.

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Li, S. "Barhl1 Regulates Migration and Survival of Cerebellar Granule Cells by Controlling Expression of the Neurotrophin-3 Gene." Journal of Neuroscience 24, no. 12 (March 24, 2004): 3104–14. http://dx.doi.org/10.1523/jneurosci.4444-03.2004.

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Li, Shengguo, and Mengqing Xiang. "Barhl1 is required for maintenance of a large population of neurons in the zonal layer of the superior colliculus." Developmental Dynamics 235, no. 8 (2006): 2260–65. http://dx.doi.org/10.1002/dvdy.20858.

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Dissertations / Theses on the topic "Barhl1"

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Pöschl, Julia. "The role of the transcription factor BARHL1 in medulloblastoma." Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-137752.

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Pöschl, Julia [Verfasser], and Ulrich [Akademischer Betreuer] Schüller. "The role of the transcription factor BARHL1 in medulloblastoma / Julia Pöschl. Betreuer: Ulrich Schüller." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2011. http://d-nb.info/1018615717/34.

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Bou-Rouphaël, Johnny. "A new role for Barhl1 in a cerebellar germinative zone as inhibitor of T-cell factors transcriptional activity." Electronic Thesis or Diss., Sorbonne université, 2023. http://www.theses.fr/2023SORUS009.

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Le cervelet humain contient plus de 50 % des neurones cérébraux. Les neurones granulaires cérébelleux représentent la population neuronale majeure. Les progéniteurs des neurones granulaires (GNP), définis par l’expression de Atoh1, émergent à partir de la lèvre rhombique supérieure (URL), une zone germinative située dans le territoire cérébelleux. Au cours du développement, les GNP prolifèrent, migrent et se différencient. Chacun de ces processus est régulé par un certain nombre de facteurs de transcription et de voies de signalisation. Les « T-Cell Factors » (Tcf/Lef) » sont des effecteurs transcriptionnels agissant en aval de la signalisation Wnt/β-caténine. Bien que les Tcf soient transcriptionnellement actifs dans la URL, leur(s) fonction(s) et leur(s) régulateur(s) développementaux n'ont été étudié. Le facteur de transcription « BarH-like 1 » (Barhl1) est exprimé dans les GNPs engagés, situés dans des zones dépourvues d'une activité transcriptionnelle Tcf. Par conséquent, les objectifs de cette thèse étaient d'étudier les fonctions de Tcf et Barhl1 en tant que régulateurs du développement des GNPs chez le Xénope. Les données présentées dans cette thèse englobent une analyse approfondie des marqueurs majeurs impliqués dans le développement des GNPs chez les amphibiens, et une étude des fonctions de Barhl1 et Tcf dans ce processus développemental. Nos expériences de gain et de perte de fonction, ainsi que l'analyse transcriptomique en absence de Barhl1 dans le rhombomère 1 valident un rôle clé de Tcf en tant qu'activateur transcriptionnel de atoh1 et en tant qu'inducteur du territoire cérébelleux, et un rôle pour Barhl1 en tant qu'inhibiteur développemental de l’activité Tcf, permettant aux GNPs de sortir de l'URL. Nous avons identifié des gènes cibles clés inhibés par Barhl1, et impliqués dans le maintien d’une zone germinative
The human cerebellum hosts more than 50% of all brain neurons. Cerebellar granule neurons are the smallest and most abundant neurons. atonal homologue 1 (Atoh1)-expressing granule neuron progenitors (GNPs) emerge from the upper rhombic lip (URL), a germinative zone located in the cerebellar primordium and displaying features of a niche of neural stem cells. GNPs proliferate, migrate, and differentiate to settle into the internal granule layer. These processes are tightly regulated by a number of transcription factors and signaling pathways. T-Cell Factor/Lymphoid Enhancer-binding Factor (Tcf/Lef) are transcriptional effectors acting downstream of Wnt/β-catenin signaling. Although Tcf is transcriptionally active in the URL, neither its function(s) nor its developmental regulator(s) have been addressed in this area. The transcription factor BarH-like 1 (Barhl1) is expressed in committed GNPs located in areas devoid of Tcf transcriptional activity. The aims of this thesis were to investigate the functions of Tcf and of Barhl1 as regulators of GNPs development using amphibian as experimental model. The data presented in this work encompass a thorough analysis of the spatial and temporal expressions of key markers involved in GNP development in amphibian, and an investigation of Barhl1 and Tcf functions in this developmental process. Our gain and loss of function experiments, together with the transcriptomic analysis of Barhl1 depletion in the rhombomere 1 validate a key role for Tcf as a transcriptional activator of atoh1 and as an inducer of the URL territory, and for Barhl1 as a developmental inhibitor of Tcf activity allowing GNPs to exit the URL. We identified key genes inhibited by Barhl1 and involved in the maintenance of URL germinative zone

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Betin, Cansu. "Barely Transitive Groups." Phd thesis, METU, 2007. http://etd.lib.metu.edu.tr/upload/3/12608605/index.pdf.

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A group G is called a barely transitive group if it acts transitively and faithfully on an infinite set and every orbit of every proper subgroup is finite. A subgroup H of a group G is called a permutable subgroup, if H commutes with every subgroup of G. We showed that if an infinitely generated barely transitive group G has a permutable point stabilizer, then G is locally finite. We proved that if a barely transitive group G has an abelian point stabilizer H, then G is isomorphic to one of the followings: (i) G is a metabelian locally finite p-group, (ii) G is a finitely generated quasi-finite group (in particular H is finite), (iii) G is a finitely generated group with a maximal normal subgroup N where N is a locally finite metabelian group. In particular, G=N is a quasi-finite simple group. In all of the three cases, G is periodic.

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Sena, Elena. "The Transcription Factor Barhl2 Inhibits Wnt Canonical Signaling during Xenopus Embryogenesis." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS090/document.

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Le développement embryonnaire est un processus hautement contrôlé où différentes voies de signalisation se coordonnent pour la construction d'un organisme. L'une des principales voies de signalisation impliquées dans ce processus est la voie canonique Wnt. La longue quête pour comprendre la cascade de signalisation Wnt/β-catenine a révélé que la réponse transcriptionelle induite par le signal Wnt/β-catenine est dépendante du contexte, ou compétence, cellulaire. Peu de choses sont connues sur les évènements moléculaires qui influencent cette compétence cellulaire. Dans les embryons de X. laevis Wnt/β-catenine est le signal inducteur pour l'Organisateur de Spemann. On ne sait pas ce qui limite l'activité Wnt dans ce territoire et par voie de conséquence la taille de l'Organisateur. Les résultats présentés dans ce manuscrit de thèse montrent que le facteur de transcription Barhl2 affecte le développement de l'organisateur de Spemann. Nous démontrons que Barhl2 inhibe l'activité Wnt via son interaction avec le corépresseur Groucho et le facteur de transcription Tcf, et mobilise l'activité de Hdac1 qui agit sur la structure chromatinienne. En utilisant des expériences in vitro et in vivo sur des cellules en culture et des embryons de Xénope nous démontrons que la régulation de Barhl2 sur les activités Groucho-Tcf est maintenue pendant l'embryogenèse et joue un rôle dans le confinement des progéniteurs neuraux dans le cerveau. Ensemble, nos résultats fournissent un mécanisme nouveau et important agissant sur le contrôle de l'activité transcriptionelle Wnt et la compétence des cellules à répondre à ce signal
Embryonic development is a highly controlled process where different signaling pathways participate into the elaboration of an organism. One of the main signaling pathways is the Wnt canonical pathway. The long-lasting search to understand Wnt/β-catenin transduction cascade revealed that the net transcriptional read out of Wnt/β-catenin signaling is highly dependent on the cellular context. In X. laevis embryos Wnt/β-catenin signaling is the informative signal for the Spemann Organizer induction. However little is known on what limits Wnt activity in this territory and consequently the size of the Spemann Organizer. The results presented in this manuscript provide evidence that the evolutionarily conserved transcription factor Barhl2 limits the development of the Spemann organizer. In this territory Barhl2 inhibits Wnt activity via its interaction with the co-repressor Groucho and the transcription factor Tcf. It participates to the recruitment of the chromatin remodeling enzyme, Hdac1 that represses the expression of Spemann organizer genes. Using a Xenopus tropicalis Tcf reporter line we demonstrate that Barhl2 inhibitory effect on Groucho-Tcf activities is maintained during embryogenesis and plays a role in the confinement of neural progenitors in the brain. Together, our results provide a new and important mechanism for the control of Wnt transcriptional activity

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Kuzucuoglu, M. "Barely transitive permutation groups." Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233097.

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Parish, Elisa Victoria. "Interactions between Pax6, Barhl2 and Shh in the early patterning of the mammalian diencephalon." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/23387.

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Diencephalic development requires the transcription factors Pax6 and Barhl2 in order to proceed correctly. Both genes are necessary for the normal development of the organizer region known as the zona limitans intrathalamica (ZLI). The ZLI goes on to pattern the diencephalon via its secretion of the morphogen Shh, which inhibits the expression of Pax6. These findings suggest that interactions between Pax6, Barhl2 and Shh may be involved in the control of diencephalic development. This project aims to characterise these interactions and investigate their roles. The expression domains of Pax6 and Barhl2 were mapped during the early development of the mouse diencephalon. Qualitative approaches were employed to confirm the high complementarity of their expression domains and obtain evidence of a mutually repressive relationship existing between the two genes. The findings from a quantitative analysis suggested that this inhibition is incomplete within the thalamus. Investigations using the Pax6-null mutant mouse confirmed that in the absence of Pax6 the thalamic Barhl2 expression domain expands beyond the ventricular zone, the site of thalamic neurogenesis. The influence of Shh signalling on the expression of Pax6 and Barhl2 was investigated via a gain-of-function approach utilising in utero electroporation to activate the Shh pathway. This led to a downregulation of both Pax6 and Barhl2 within the thalamus. In Shh loss-of-function experiments drug treatment with the Shh antagonist vismodegib led to an upregulation of Barhl2 and the loss of the GABAergic pTh-R in the Pax6-null mutant thalamus, but not in the wild type thalamus, suggesting that Pax6 and Shh may be required to inhibit Barhl2 in order for GABAergic neurogenesis to proceed. Barhl2 expression was detected in the Shh-null mutant mouse confirming that, in contrast with their homologues in Drosophila, Shh may be expressed downstream of Barhl2. Together these findings have been used to develop a novel model of thalamic development in which Barhl2 induces ZLI development, inhibition of Barhl2 by Pax6 restricts its expansion, and secretion of Shh by the ZLI then goes on to inhibit Pax6 and Barhl2 in the pTh-R while mutual repression between Pax6 and Barhl2 modulates neurogenesis in the more caudal regions of the thalamic neuroepithelium.

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Juraver-Geslin, Hugo. "Barhl2, un inhibiteur prolifératif des progéniteurs neuraux dans le développement du diencéphale caudal." Paris 6, 2013. http://www.theses.fr/2013PA066101.

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Notre équipe s’intéresse à la mise en place de la partie caudale du cerveau antérieur qui génère le thalamus, un processeur de l’information vers le cortex, et son centre organisateur, la zona limitans intrathalamica (ZLI). La ZLI, via la sécrétion des facteurs Wnt et Sonic HedgeHog (Shh), contrôle l’acquisition d’identité et la croissance de toute la partie caudale du cerveau antérieur. Wnt et Shh sont essentiels dans le contrôle de la survie et de la prolifération neurale au cours du développement. Chez les amphibiens, et les rongeurs, le gène à homéodomaine Barhl2 est exprimé dans le neuroépithelium du cerveau antérieur avec des ligands Wnt et Shh. Chez le xénope, Barhl2 limite la prolifération des progéniteurs neuraux du diencéphale et contrôle l’organisation du neuroépithélium. Barhl2 agit via une fonction non apoptotique de la Caspase-3, qui inhibe l’accumulation et l’activation de la caténine, l’intermédiaire majeur de la voie Wnt. En parallèle, Barhl2 est crucial dans l’élaboration du champ histogénique qui génère la ZLI et le thalamus. Un réseau dynamique impliquant barhl2, iroquois (irx), otx et pax6 est en jeu pour l’établissement de ces territoires présomptifs. Mon travail, ainsi que des études chez le poisson zèbre, démontre que barhl2, otx2 et irx3 sont des gènes maîtres dans le prépatterning de la ZLI et que ce rôle est conservé chez les vertébrés
Our team focuses on the development of the caudal forebrain that generates the thalamus, a processor of information to the cortex, and its organizing center, the zona limitans intrathalamica (ZLI). ZLI, through the secretion of Sonic Hedgehog (Shh) and Wnt ligands controls the acquisition of identity and the growth of the entire caudal forebrain. Shh and Wnt are the main pahways that controls the survival and proliferation of neural progenitors during development. In amphibians and rodents, the homeodomain containing gene Barhl2 is expressed with Shh and Wnt ligands in the forebrain neuroepithelium. In Xenopus, Barhl2 limit the proliferation of diencephalic neural progenitors and controls the neuroepithelium architecture. Barhl2 acts through an unconventional function of Caspase-3, the major effector of apoptosis, which inhibits the accumulation and activation of βcaténine. My work also showed that Barhl2 is crucial in the development of the histogenic field that generates the ZLI and thalamus. A dynamic network involving barhl2, iroquois (irx), otx and pax6 is at play in establishing the presumptive territories. My work, and studies in zebrafish, shows that barhl2, otx2 and irx3 are masters prepatterning genes of the ZLI and this role is conserved in vertebrates

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Olivesi, Dominique. "Virgile Barel : 1889-1979 /." Nice : Serre, 1996. http://catalogue.bnf.fr/ark:/12148/cb36688081n.

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Lewis, Colin A. "Barkly East bells and the British Empire." The Ringing World, 2002. http://www.ringingworld.co.uk.

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Colin Lewis was Professor of Geography at Rhodes University, Grahamstown, South Africa from 1989 until his retirement at the end of 2007. In 1990, with the strong support of the incumbent Vice-Chancellor, Dr Derek Henderson, he instigated the Certificate in Change Ringing (Church Bell Ringing) in the Rhodes University Department of Music and Musicology - the first such course to be offered in Africa. Since that date he has lectured in the basic theory, and taught the practice of change ringing. He is the Ringing Master of the Cathedral of St Michael and St George, Grahamstown, South Africa.

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Books on the topic "Barhl1"

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Hirēmaṭha, Ār Si. Uri barali-siri barali. Beḷagāvi: Vīraśaiva Adhyayana Akāḍemi, Śrī Nāganūru Rudrākṣimaṭha, 1995.

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Barolo. Lincoln: University of Nebraska Press, 2010.

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Frank, Matthew Gavin. Barolo. Lincoln: University of Nebraska Press, 2010.

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Frank, Matthew Gavin. Barolo. Lincoln: University of Nebraska Press, 2010.

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LaBrecque, Jennifer. Barely behaving. Toronto, Ont: Harlequin, 2003.

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Carr, Tom. Barely there. Madrid, Spain: Galeria Astarté, 2011.

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Frost, Kimberly. Barely bewitched. New York: Berkley Books, 2009.

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Dalapati, Murmu, ed. The Barela. Calcutta: Anthropological Survey of India, Ministry of Human Resource Development, Dept. of Culture, Govt. of India, 1995.

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Donnelly, Shannon. Barely Proper. New York, N.Y: Zebra Books, 2003.

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Rebecca, Donovan. Barely breathing. Las Vegas, NV: Skyscape, 2013.

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Book chapters on the topic "Barhl1"

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Pingel, Susan F. "Barely There." In Sports and the Law, 283–87. New York: Routledge, 2021. http://dx.doi.org/10.4324/9781003249931-53.

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Irminger-Finger, Irmgard. "BARD1." In Encyclopedia of Cancer, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_526-2.

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Irminger-Finger, Irmgard. "BARD1." In Encyclopedia of Cancer, 428–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_526.

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Irminger-Finger, Irmgard. "BARD1." In Encyclopedia of Cancer, 339–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_526.

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de Kiewiet, C. W. "Sir Bartle Frere." In The Imperial Factor in South Africa, 125–47. London: Routledge, 2022. http://dx.doi.org/10.4324/9781003306832-6.

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Schad, John, and Fred Dalmasso. "Barely a Film." In Derrida | Benjamin, 183–91. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-49807-8_4.

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Azimova, Shakhnoza S., and Anna I. Glushenkova. "Barosma betulina Bartl." In Lipids, Lipophilic Components and Essential Oils from Plant Sources, 785–86. London: Springer London, 2012. http://dx.doi.org/10.1007/978-0-85729-323-7_2585.

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Probst, Helga. "Gotthelf, Jeremias: Barthli der Korber." In Kindlers Literatur Lexikon (KLL), 1–2. Stuttgart: J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_6515-1.

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Komm, Dennis, and Richard Královič. "Advice Complexity and Barely Random Algorithms." In SOFSEM 2011: Theory and Practice of Computer Science, 332–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-18381-2_28.

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"barhak." In The Fairchild Books Dictionary of Textiles. Fairchild Books, 2021. http://dx.doi.org/10.5040/9781501365072.1138.

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Conference papers on the topic "Barhl1"

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"Tourism Impacts; Threats and Opportunities in Barili, Cebu." In Jan. 29-30, 2019 Cebu (Philippines). Emirates Research Publishing, 2019. http://dx.doi.org/10.17758/erpub3.ea01191018.

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Southby, Chris. "The hidden Proterozoic successions of the Barkly region." In Central Australian Basins Symposium IV. Petroleum Exploration Society of Australia (PESA), 2022. http://dx.doi.org/10.36404/qpnu9817.

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The Barkly 2D Deep Crustal Reflection Seismic Survey (L212) was acquired in 2019 by Geoscience Australia as a major objective of the Australian Governments’ multi-year $225m Exploring for the Future (EFTF) program in partnership with, and co-funded by, the Northern Territory Geological Survey under the Resourcing the Territory initiative.

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Cai, Heng, Shumeng Li, Lei Qi, Qian Yu, Yinghuan Shi, and Yang Gao. "Orthogonal Annotation Benefits Barely-supervised Medical Image Segmentation." In 2023 IEEE/CVF Conference on Computer Vision and Pattern Recognition (CVPR). IEEE, 2023. http://dx.doi.org/10.1109/cvpr52729.2023.00322.

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4

Feld, Michael S. "Barely dressed single atoms in an optical resonator." In OSA Annual Meeting. Washington, D.C.: Optica Publishing Group, 1989. http://dx.doi.org/10.1364/oam.1989.wdd3.

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5

Tong, Xin, and Andreas Moshovos. "BarTLB: Barren page resistant TLB for managed runtime languages." In 2014 32nd IEEE International Conference on Computer Design (ICCD). IEEE, 2014. http://dx.doi.org/10.1109/iccd.2014.6974692.

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6

SPROAT, W., and W. LEWIS. "Barely Visible Impact Damage (BVID) detection in aircraft composites." In 2nd Aerospace Maintenance Conference. Reston, Virigina: American Institute of Aeronautics and Astronautics, 1986. http://dx.doi.org/10.2514/6.1986-1142.

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7

Singh, Abhendra, Barry Davidson, David Eisenberg, Mike Czabaj, and Alan Zehnder. "Barely Visible Impact Damage Evaluation of Composite Sandwich Structures." In 51st AIAA/ASME/ASCE/AHS/ASC Structures, Structural Dynamics, and Materials Conference
18th AIAA/ASME/AHS Adaptive Structures Conference
12th. Reston, Virigina: American Institute of Aeronautics and Astronautics, 2010. http://dx.doi.org/10.2514/6.2010-2770.

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8

Smith, Tony, Riccardo Tarroni, Dennis Clouthier, and Gretchen Rothschopf. "BARELY FLUORESCENT MOLECULES I. TWIN DISCHARGE JET SPECTROSCOPY OF HSnCl." In 2020 International Symposium on Molecular Spectroscopy. Urbana, Illinois: University of Illinois at Urbana-Champaign, 2020. http://dx.doi.org/10.15278/isms.2020.tf02.

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Sunahori, Fumie, Riccardo Tarroni, Dennis Clouthier, Gretchen Rothschopf, and Tony Smith. "BARELY FLUORESCENT MOLECULES II. TWIN DISCHARGE JET SPECTROSCOPY OF HSnBr." In 2020 International Symposium on Molecular Spectroscopy. Urbana, Illinois: University of Illinois at Urbana-Champaign, 2020. http://dx.doi.org/10.15278/isms.2020.tf03.

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Ren, Feifei, Ilias N. Giannakeas, Zahra Sharif Khodaei, and Ferri M. H. Aliabadi. "The influence of temperature on barely visible impact damage detection." In ADVANCES IN FRACTURE AND DAMAGE MECHANICS XX. AIP Publishing, 2023. http://dx.doi.org/10.1063/5.0150876.

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Reports on the topic "Barhl1"

1

Lefgren, Lars, Brennan Platt, and Joseph Price. Sticking with What (Barely) Worked. Cambridge, MA: National Bureau of Economic Research, October 2011. http://dx.doi.org/10.3386/w17477.

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2

Bednarski, J. M. Surficial geology, Baril Lake, Alberta. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1999. http://dx.doi.org/10.4095/210481.

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3

Modi, Ami. Checkpoint Functions of the BRCA1/BARD1 Tumor Suppressor. Fort Belvoir, VA: Defense Technical Information Center, July 2007. http://dx.doi.org/10.21236/ada484036.

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Modi, Ami. Checkpoint Functions of the BRCA1/BARD1 Tumor Suppressor. Fort Belvoir, VA: Defense Technical Information Center, July 2008. http://dx.doi.org/10.21236/ada493385.

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5

Southby, C., C. J. Carson, T. Fomin, N. Rollet, P. A. Henson, L. K. Carr, M. P. Doublier, and D. Close. Exploring for the Future - The 2019 Barkly Reflection Seismic Survey (L212). Geoscience Australia, 2022. http://dx.doi.org/10.11636/record.2022.009.

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Kürşat Önder, Yasin, Maria Alejandra Ruiz-Sanchez, Sara Restrepo-Tamayo, and Mauricio Villamizar-Villegas. Government Borrowing and Crowding Out. Banco de la República, December 2021. http://dx.doi.org/10.32468/be.1182.

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We investigate the impact of fiscal expansions on firm investment by exploiting firms that have multiple banking relationships. Further, we conduct a localized RDD approach and compare the lending behavior of banks that barely met and missed the criteria of being a primary dealer, as well as barely winners and losers at government auctions. Our results indicate that a 1 percentage point increase in banks’ bonds-to-assets ratio decreases loans by up to 0.4%, which leads to significant declines in firm investment, profits and wages. Our findings are grounded in a quantitative model with financial and real sectors with which we undertake a welfare analysis and compute the cost of government borrowing on the overall economy.

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7

Joukov, Vladimir. The Role of BRCA1/BARD1 Heterodimers in the Mitosis-Interphase Transition. Fort Belvoir, VA: Defense Technical Information Center, May 2007. http://dx.doi.org/10.21236/ada471801.

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de Caritat, P., E. Bastrakov, A. T. Walker, and B. I. A. McInnes. The Heavy Mineral Map of Australia Project – Data Release 2: The Barkly-Isa-Georgetown Region. Geoscience Australia, 2022. http://dx.doi.org/10.11636/record.2022.043.

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9

Gray, D. H. A commentary on the authenticity of the name 'Pointe au Baril' (Ontario). Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1989. http://dx.doi.org/10.4095/298310.

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Harris, Jeremy, Alejandro Ramos Martínez, and Paolo Giordano. INTrade: Latin American Trade Trend Estimates: 2013. Inter-American Development Bank, December 2013. http://dx.doi.org/10.18235/0008278.

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Latin American Trade Trend Estimates is an annual study published by the Integration and Trade Sector of the Inter-American Development Bank that analyses the region's trade performance based on available national and international data for selected countries. For 2013, the study reports that Latin American exports stagnated, posting a second straight year of sluggish growth. Export increases were barely over 0%, for a total value of slightly more than $1 trillion. It is also noted that the region's exports actually declined in the first months of the year, continuing a negative trend that began in late 2012. However, in the third quarter, exports began to grow again, and current estimates point to a small positive overall growth rate for the year. Results varied considerably among countries. Brazil, Colombia, Peru, and Venezuela all posted declines in exports, while Argentina, Bolivia, Chile, Ecuador, Mexico, Paraguay and Uruguay posted increases. Results in Central America were mixed, with exports barely growing in Costa Rica and Guatemala, declining in Honduras and Nicaragua, but expanding in El Salvador, and Panama.

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