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Relevant bibliographies by topics / B-CLL

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Academic literature on the topic 'B-CLL'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "B-CLL"

1

Parker, Anton, Shilu Amin, Tricia Zwiefelhofer, et al. "Epigenetic Regulation of ZAP70 in Chronic Lymphocytic Leukemia." Blood 112, no. 11 (2008): 2246. http://dx.doi.org/10.1182/blood.v112.11.2246.2246.

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Abstract ZAP70 expression has been shown to be involved in enhanced signalling and more aggressive disease in a subset of CLL. Mechanisms regulating ZAP70 expression are unknown. We have shown previously that despite the absence of a 5’ CpG island, the methylation status of a small region of CpG dinucleotides (CpGs) correlates with the transcriptional state of the gene in both normal lymphocytes and B cell leukemias. Quantitative methylation analysis of 605 CpGs across the 28kb genomic region spanning ZAP70 was performed by MassARRAY on a panel of 17 CLL tumor cell samples, 4 lymphoid cell lin

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Silber, R., CM Farber, E. Papadopoulos, et al. "Glutathione depletion in chronic lymphocytic leukemia B lymphocytes." Blood 80, no. 8 (1992): 2038–43. http://dx.doi.org/10.1182/blood.v80.8.2038.2038.

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Abstract Glutathione (GSH) content may be the major determinant of a cell's sensitivity to cytotoxic alkylating agents. In the present study, the GSH concentration was determined in lymphocytes isolated from the blood of normal subjects and patients with chronic lymphocytic leukemia (CLL). Comparable levels were found in both types of cells. Incubation for 20 hours led to a decrease in GSH to 51% of baseline values in CLL B cells. Under the same conditions, normal B- or T-lymphocyte GSH content remained constant. GSH depletion was shown to be a characteristic of the B-CLL B lymphocyte. It was

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Silber, R., CM Farber, E. Papadopoulos, et al. "Glutathione depletion in chronic lymphocytic leukemia B lymphocytes." Blood 80, no. 8 (1992): 2038–43. http://dx.doi.org/10.1182/blood.v80.8.2038.bloodjournal8082038.

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Glutathione (GSH) content may be the major determinant of a cell's sensitivity to cytotoxic alkylating agents. In the present study, the GSH concentration was determined in lymphocytes isolated from the blood of normal subjects and patients with chronic lymphocytic leukemia (CLL). Comparable levels were found in both types of cells. Incubation for 20 hours led to a decrease in GSH to 51% of baseline values in CLL B cells. Under the same conditions, normal B- or T-lymphocyte GSH content remained constant. GSH depletion was shown to be a characteristic of the B-CLL B lymphocyte. It was not found

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Goodman, Alice. "B-CLL." Oncology Times &NA;, Supplement (2006): 21. http://dx.doi.org/10.1097/01.cot.0000316106.04463.a9.

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Silber, R., B. Degar, D. Costin, et al. "Chemosensitivity of lymphocytes from patients with B-cell chronic lymphocytic leukemia to chlorambucil, fludarabine, and camptothecin analogs." Blood 84, no. 10 (1994): 3440–46. http://dx.doi.org/10.1182/blood.v84.10.3440.3440.

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Abstract Chemosensitivity of B lymphocytes, obtained from 65 patients with B- cell chronic lymphocytic leukemia (B-CLL), Rai stages 0 through IV, was determined using the MTT assay. The results were expressed by the drug concentration required for 50% inhibition of cell viability (IC50). The cytotoxicity of chlorambucil (CLB) was compared with that of fludarabine and the DNA topoisomerase I inhibitors, camptothecin, 9- aminocamptothecin, 10,11-methylenedioxy-20(S)-camptothecin (10,11-MDC) and 9-amino-10,11-methylenedioxy-20(S)-campthothecin (9-A-10,11-MDC), and topotecan. Considerable heteroge

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Silber, R., B. Degar, D. Costin, et al. "Chemosensitivity of lymphocytes from patients with B-cell chronic lymphocytic leukemia to chlorambucil, fludarabine, and camptothecin analogs." Blood 84, no. 10 (1994): 3440–46. http://dx.doi.org/10.1182/blood.v84.10.3440.bloodjournal84103440.

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Chemosensitivity of B lymphocytes, obtained from 65 patients with B- cell chronic lymphocytic leukemia (B-CLL), Rai stages 0 through IV, was determined using the MTT assay. The results were expressed by the drug concentration required for 50% inhibition of cell viability (IC50). The cytotoxicity of chlorambucil (CLB) was compared with that of fludarabine and the DNA topoisomerase I inhibitors, camptothecin, 9- aminocamptothecin, 10,11-methylenedioxy-20(S)-camptothecin (10,11-MDC) and 9-amino-10,11-methylenedioxy-20(S)-campthothecin (9-A-10,11-MDC), and topotecan. Considerable heterogeneity in

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7

Matvieieva, A. S., L. M. Kovalevska, and E. V. Kashuba. "The SMAD4 transcription factor shows cytoplasmic retention in B-cells of patients with chronic lymphocytic leukemia (CLL)." Faktori eksperimental'noi evolucii organizmiv 22 (September 9, 2018): 144–48. http://dx.doi.org/10.7124/feeo.v22.939.

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Aim. To illuminate the reason of inactivity of the TGFB-SMAD2/3 pathways in CLL cells. Methods. CLL cells were isolated from peripheral blood of CLL patients, using gradient centrifugation at the ficoll. Expression and cellular localization of SMAD2, 3 and 4 proteins were analyzed by fluorescence microscopy, using specific antibodies. Results. The SMAD2 protein was basically not expressed in CLL cells, in contrast to B cells, isolated from the peripheral blood of a healthy donor. Moreover, the SMAD3 and SMAD4 proteins were localized exclusively in the cytoplasm (a proportion of SMAD3 was detec

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Santiago-Schwarz, F., C. Panagiotopoulos, A. Sawitsky, and KR Rai. "Distinct characteristics of lymphokine-activated killer (LAK) cells derived from patients with B-cell chronic lymphocytic leukemia (B-CLL). A factor in B-CLL serum promotes natural killer cell-like LAK cell growth." Blood 76, no. 7 (1990): 1355–60. http://dx.doi.org/10.1182/blood.v76.7.1355.1355.

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Abstract We show that lymphokine-activated killer (LAK) cell precursors derived from patients with B-cell chronic lymphocytic leukemia (B-CLL) and cultured in the presence of recombinant interleukin-2 and normal human serum (NHS), develop into primarily NK cell-like (CD 57+) LAK cells, whereas identically prepared LAK cell precursors from normal subjects develop into mainly T cell-like (CD 3+, CD 8+) LAK cells. B-CLL LAK cells exhibited greater proliferative capacity than did normal LAK cells. When normal LAK cells were grown in B-CLL serum instead of NHS, their proliferation increased; NK cel

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Santiago-Schwarz, F., C. Panagiotopoulos, A. Sawitsky, and KR Rai. "Distinct characteristics of lymphokine-activated killer (LAK) cells derived from patients with B-cell chronic lymphocytic leukemia (B-CLL). A factor in B-CLL serum promotes natural killer cell-like LAK cell growth." Blood 76, no. 7 (1990): 1355–60. http://dx.doi.org/10.1182/blood.v76.7.1355.bloodjournal7671355.

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We show that lymphokine-activated killer (LAK) cell precursors derived from patients with B-cell chronic lymphocytic leukemia (B-CLL) and cultured in the presence of recombinant interleukin-2 and normal human serum (NHS), develop into primarily NK cell-like (CD 57+) LAK cells, whereas identically prepared LAK cell precursors from normal subjects develop into mainly T cell-like (CD 3+, CD 8+) LAK cells. B-CLL LAK cells exhibited greater proliferative capacity than did normal LAK cells. When normal LAK cells were grown in B-CLL serum instead of NHS, their proliferation increased; NK cell levels

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Tummala, Hemanth, Alexey Goltsov, Hilal S. Khalil, et al. "Advocating the need of a systems biology approach for personalised prognosis and treatment of B-CLL patients." BioDiscovery 6 (December 30, 2012): e8939. https://doi.org/10.7750/BioDiscovery.2012.6.4.

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The clinical course of B-CLL is heterogeneous. This heterogeneity leads to a clinical dilemma: can we identify those patients who will benefit from early treatment and predict the survival? In recent years, mathematical modelling has contributed significantly in understanding the complexity of diseases. In order to build a mathematical model for determining prognosis of B-CLL one has to identify, characterise and quantify key molecules involved in the disease. Here we discuss the need and role of mathematical modelling in predicting B-CLL disease pathogenesis and suggest a new systems biology

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Dissertations / Theses on the topic "B-CLL"

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Schlaak, Max Simon. "Differentielles Genexpressionsmuster in chronischen lymphatischen Leukämiezellen vom B-Zell-Typ (B-CLL-Zellen)." [S.l.] : [s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=966320069.

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Schlaak, Max Simon. "Differentielles Genexpressionsmuster in chronischen lymphatischen Leukämiezellen vom B-Zell-Typ (B-CLL-Zellen)." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2003. http://dx.doi.org/10.18452/14817.

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Die B-CLL ist eine niedrigmaligne Erkrankung, die im höheren Lebensalter auftritt und für die es weiterhin keine dauerhaft kurative Therapie gibt. Die Erforschung der genetischen Grundlagen dieser Krankheit könnte Erkenntnisse über die Funktionsmechanismen und neue Diagnose- und Therapieansätze erbringen. Ziel dieser Arbeit war es, eine Genbank von CLL-Patienten bzw. gesunden Spendern zu erstellen, um Gene, die für die Entstehung der Erkrankung mitverantwortlich sein könnten, zu identifizieren. Die subtraktive suppressive Hybridisierung (SSH) wurde als Methode eingesetzt, um cDNA-Mischungen z

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Kurzeder, Christian. "Gentransfer in primäre B-CLL-Zellen mittels EBV abgeleiteter Genvektoren." [S.l.] : [s.n.], 2004. http://edoc.ub.uni-muenchen.de/archive/00004955.

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Kurzeder, Christian. "Gentransfer in primäre B-CLL-Zellen mittels EBV abgeleiteter Genvektoren." Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-49552.

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Bund, Dagmar. "hTERT, CD23 und CD229 als Tumorantigene bei der B-CLL." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-145282.

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Almond, Jason Baron. "Mechanisms of proteasome inhibitor-induced apoptosis of B-cell chronic lymphocytic leukaemia (B-CLL) cells." Thesis, University of Leicester, 2002. http://hdl.handle.net/2381/30761.

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Proteasome inhibitors, including lactacystin, LLnL (N-acetyl-N-leucinyl-L-leucinyl-L- norleucinal) and MG132 (carbobenzoxyl-leucinyl-leucinyl-leucinal), potently induce apoptosis in leukaemic B-cells from patients with B-cell chronic lymphocytic leukaemia (B-CLL). This pro-apoptotic effect occurs in cells from patients at all stages of the disease, including those resistant to conventional chemotherapy, suggesting that proteasome inhibitors may be useful for treatment of B-CLL. Following initial inhibition of proteasomal activity and an increase in ubiquitinated proteins, these agents induce m

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Walton, Alexander James. "CD4+PF+ T Cells in B-CLL Patients & Normal Controls." Thesis, University of Westminster, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.502405.

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Our group has previously shown that CD4+Perforin (PFt T cells with cytotoxic potential are expanded in patients with B-CLL, accounting for up to 50% of CD4+ T cells in this disease. This study confinns the finding of increased percentages of CD4+PF+ T cells in a new cohort of B-CLL patients. However, the significance of this subset in B-CLL remains unclear. Evidence has accumulated for the potential role of CD4+ cytotoxic T cells in controlling cytomegalovirus (CMV). Therefore, a potential relationship between chronic CMV infection and CD4+PF+ T cell expansion in B-CLL was investigated. CMV se

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Munoz-Ritchie, Varinia Graciela. "P-glycoprotein-associated anthracycline resistance in B-CLL : potential for cytokine modulation." Thesis, University of Plymouth, 2001. http://hdl.handle.net/10026.1/2809.

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The phenomenon of multidrug resistance (MDR) in cancer cells is generally associated with P-glycoprotein (P-gp) expression and presents an obstacle to successful chemotherapy. Attempts to overcome P-gp-associated MDR using P-gp modulators, such as verapamil, have been hindered by their intrinsic in vivo toxicity. In 1991, however, Scala et al. demonstrated the alteration of P-gp function by interferon-alpha (IFN-α) in vitro at non-toxic in vivo concentrations, suggesting a basis for the use of IFN-α clinically in patients exhibiting P-gp-associated MDR. Drug resistance in B-CLL has been linked

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Hellqvist, Eva. "Antigen interaction with B cells in two proliferative disorders : CLL and MGUS." Doctoral thesis, Linköping : Department of Clinical and Experimental Medicine, Linköping University, 2010. http://www2.bibl.liu.se/liupubl/disp/disp2010/med1158s.pdf.

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Gorgone, Ausilia Giuseppa. "Correlazione tra dati biologici e prognosi in pazieti affetti da B-CLL." Doctoral thesis, Università di Catania, 2012. http://hdl.handle.net/10761/1138.

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Gli studi di questi anni hanno dimostrato che i fattori biologici giocano un ruolo fondamentale per la stratificazione del rischio come elementi predittivi del treatment-free survival e dell' overall survival nei pazienti affetti da CLL in stadio iniziale.La maggioranza di questi markers prognostici ,anche se ormai in uso quotidiano nella pratica clinica, non è ancora incluso nelle linee guida internazionali che si basano ancora su criteri esclusivamente clinici e individuano la valutazione biologica da usare in combinazione ai parametri clinici.Il loro impiego può certamente contribuire al m

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Books on the topic "B-CLL"

1

Sztuka Fabryka (Group of artists). B/W - CLR. Redfoxpress, 2010.

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Dawson, Alma. Development of a core library collection for library automation: Report to the Council on Library Resources on Project CLR #4026-B. Produced at the Research Center Annex, School of Library and Information Science, Louisiana State University, 1989.

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Flohr, T. Regulation von Zytoinproduktion und Zellproliferation durch Zytokine bei B-CLL-Zellen in vitro. 2000.

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Brass, Ute. Der Einfluss von Interferon-alpha auf durch CD40-Antikörper stimulierte B-CLL-Zellen. 1996.

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Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Leukaemia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0004.

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Acute myeloblastic leukaemia (AML) - Acute lymphoblastic leukaemia (ALL) - Chronic myeloid leukaemia (CML) - Chronic lymphocytic leukaemia (B-CLL) - Prolymphocytic leukaemia (PLL) - Hairy cell leukaemia and variant - Large granular lymphocyte leukaemia (LGLL) - Adult T-cell leukaemia-lymphoma (ATLL)

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Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Hassan Al-Sader. Leukaemia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0004_update_001.

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Acute myeloblastic leukaemia (AML) - Acute lymphoblastic leukaemia (ALL) - Chronic myeloid leukaemia (CML) - Chronic lymphocytic leukaemia (B-CLL) - Prolymphocytic leukaemia (PLL) - Hairy cell leukaemia and variant - Large granular lymphocyte leukaemia (LGLL) - Adult T-cell leukaemia-lymphoma (ATLL)

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Magin, Thomas O. H. Durchflusszytometrische Immunphänotypisierung maligner B-CLL Lymphozyten: Korrelation zu Klinik und prognostischen Parametern der chronisch lymphatischen Leukämie. 1995.

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8

Woelke, Corinna. Ex-vivo Inkubation von immunomagnetisch aufgereinigten autologen Stammzelltransplantaten bie Patienten mit B-CLL zur Eliminierung residueller kontaminierender Tumorzellen. 2003.

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Carton, James. Haematopathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0015.

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This chapter discusses haematopathology, including iron deficiency anaemia, anaemia of chronic disease, megaloblastic anaemias, hereditary spherocytosis, glucose-6-phosphate dehydrogenase deficiency, thalassaemias, sickle-cell disorders, idiopathic thrombocytopenic purpura (ITP), thrombotic thrombocytopenic purpura (TTP), von Willebrand disease, haemophilia, thrombophilia, acute B-lymphoblastic leukaemia, acute myeloid leukaemias, chronic lymphocytic leukaemia (CLL), chronic myelogenous leukaemia, polycythaemia vera (PV), essential thrombocythaemia (ET), primary myelofibrosis (PMF), myelodyspl

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Steensma, David P. Malignant Hematology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0296.

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The hematologic neoplasms include lymphoproliferative disorders (eg, chronic lymphocytic leukemia [CLL]/small lymphocytic lymphoma [SLL], large granular lymphocyte leukemia, hairy cell leukemia [HCL], Hodgkin lymphoma, non-Hodgkin lymphoma), plasma cell disorders (multiple myeloma, light chain amyloidosis, Waldenström macroglobulinemia, POEMS syndrome, heavy chain disease, plasmacytoma), chronic myeloid neoplasms (chronic myeloid leukemia, the BCR/ABL-negative myeloproliferative neoplasms, myelodysplastic syndromes), and acute leukemia (acute myeloid leukemia, acute lymphocytic leukemia). In a

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Book chapters on the topic "B-CLL"

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Stilgenbauer, Stephan, Hartmut Döhner, and Peter Lichter. "Chronisch lymphatische Leukämie vom B-Zell-Typ (B-CLL)." In Molekularmedizinische Grundlagen von hämatologischen Neoplasien. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59343-7_13.

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Agathangelidis, Andreas, Stavroula Ntoufa, and Kostas Stamatopoulos. "B Cell Receptor and Antigens in CLL." In Advances in Experimental Medicine and Biology. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8051-8_1.

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Faguet, Guy B. "Clonal Evolution and Second Malignancies in B-CLL." In Chronic Lymphocytic Leukemia. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-412-2_21.

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Hamblin, Terry. "The Heterogeneous Origin of the B-CLL Cell." In Chronic Lymphocytic Leukemia. Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-412-2_4.

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Genetet, Noelle, Dominique Bourel, Bernard Grosbois, et al. "Heterogeneity of B-CLL Cells Defined by Monoclonal Antibodies." In Leukocyte Typing II. Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4612-4848-4_32.

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Schetelig, Johannes, and Peter Dreger. "Chronic Lymphocytic Leukemia." In The EBMT Handbook. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-44080-9_85.

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AbstractCLL is a rare indication for HCT/Cellular Therapy since it usually follows an indolent course. Allogeneic HCT is considered as standard of care in eligible high-risk patients who have failed at least two classes of modern pathway inhibitor-based therapy, and in select patients with CLL transformed in to an aggressive B-cell lymphoma (Richter transformation). Except for Richter transformation, there is no role for autologous HCT in CLL. In the absence of a labeled indication, CAR T-cells should not be used outside of clinical trials.

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Lanza, F., and G. L. Castoldi. "Flow Cytochemical Analysis of Lymphoid Cells in B-Chronic Lymphocytic Leukemia (B-CLL)." In Leukemias. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77083-8_56.

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Phipps, R. P., S. J. Pollock, K. Kaur, et al. "Expression of Cyclooxygenase-2 and Prostaglandins by B-1 Cells and B-CLL Cells." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-57284-5_30.

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Okkenhaug, Klaus, and Jan A. Burger. "PI3K Signaling in Normal B Cells and Chronic Lymphocytic Leukemia (CLL)." In Current Topics in Microbiology and Immunology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/82_2015_484.

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Gorczyca, Wojciech. "Chronic Lymphocytic Leukemia/Small Lymphocytic Lymphoma (CLL/SLL) and B-Cell Prolymphocytic Leukemia (B-PLL)." In Atlas of Differential Diagnosis in Neoplastic Hematopathology, 4th ed. CRC Press, 2021. http://dx.doi.org/10.1201/9781003120445-09.

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Conference papers on the topic "B-CLL"

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Chambers, Brian, Russ Kane, and Mark Yunovich. "Implications of Temperature and Buffering Systems for Laboratory Testing of Alloy Steel and 13Cr Materials in Oil and Gas Production Environments." In CORROSION 2011. NACE International, 2011. https://doi.org/10.5006/c2011-11096.

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Abstract This paper describes the results of a laboratory study evaluating cracking resistance of 4130 (UNS G41300), 13Cr-L80 (UNS S42000), and two modified “13-5-2” type 13Cr alloys (UNS S41426) with 110 ksi (758 MPa) specified minimum yield strength in three different oil and gas well environments. Sulfide stress cracking resistance was evaluated using tensile specimens stressed to 90% actual yield strength and double cantilever beam specimens. Three environments were evaluated that included: (a) NACE Solution A with 15 psia (0.1 MPa) H2S; (b) a simulated oil well with 100,000 mg/L Cl−, pH 4

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Ritter, S., and H. P. Seifert. "Stress Corrosion Cracking Behavior of Low-Alloy Reactor Pressure Vessel Steels and of a Weld Filler Material under Simulated BWR Environment." In CORROSION 2003. NACE International, 2003. https://doi.org/10.5006/c2003-03664.

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Abstract The stress corrosion cracking (SCC) behavior of three different nuclear grade reactor pressure vessel (RPV) steels (SA 533 B Cl.1, SA 508 Cl.2, 20 MnMoNi 5 5) and of a RPV weld filler material was characterized under simulated boiling water reactor (BWR)/normal water chemistry (NWC) conditions by constant and ripple load tests with pre-cracked fracture mechanics specimens. The experiments were performed in oxygenated high-temperature water at temperatures of either 288, 250, 200 or 150 °C. Modern high-temperature water loops, on-line crack growth monitoring (DCPD) and fractographical

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Mansfeld, F., and Y. Wang. "Corrosion Protection of High-Copper Aluminum Alloys Using Green Technology." In CORROSION 1995. NACE International, 1995. https://doi.org/10.5006/c1995-95041.

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Abstract The concept of surface modification as a new method of corrosion protection using chemicals without toxic problems is described for the Al alloys Al 6061, Al 7075-T6 and Al 2024-T3. In the Ce-Mo process Ce and Mo are incorporated into the original oxide film by chemical and electrochemical processes. The resulting surfaces are resistant to pitting in aggressive solutions such as 0.5 N NaCl. Surface modified Al 6013 has passed the salt spray test according to ASTM B 117. For Al 6061 and 7075, hot solutions of CeCl3 and Ce(NO3)3 are used, while for Al 2024 CeCl3 is replaced by Ce acetat

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Efird, K. D., E. J. Wright, J. A. Boros, and T. G. Hailey. "Experimental Correlation of Steel Corrosion in Pipe Flow with Jet Impingement and Rotating Cylinder Laboratory Tests." In CORROSION 1993. NACE International, 1993. https://doi.org/10.5006/c1993-93081.

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Abstract The results from the first phase of research to quantify the relationship of laboratory fluid flow corrosion test techniques to field applications and the parameters required to effectively apply laboratory data to plant operations are presented. Single phase, aqueous, sweet corrosion of steel in turbulent pipe flow (12.7 and 25.4 mm diameter) is correlated to corrosion in jet impingement and rotating cylinder tests. All tests were simultaneously conducted using the same test fluid (3.0% NaCl + 1000 ppm HCO3− solution at pH 6.0 with 20-30 ppb O2 and 1.3 bar (20 psia) CO2 at 50°C) to m

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Andrianopoulou, S., M. Schmitt, V. Grüßinger, B. Lippert, and U. Martens. "Simultanes kutanes Plattenepithelkarzinom und B-CLL: Grenzen innovativer Therapiekonzepte." In Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685815.

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Deng, Jiusheng, Jonathon Cohen, Andrea Pennati, et al. "Abstract A11: GIFT4-reprogrammed leukemic B cells for CLL immunotherapy." In Abstracts: AACR Special Conference on Hematologic Malignancies: Translating Discoveries to Novel Therapies; September 20-23, 2014; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1557-3265.hemmal14-a11.

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Davis, Joanne, Kylie Mason, Chia Sharpe, et al. "Abstract B054: Can CLL-B cells treated with novel combination therapies be used to promote anti-CLL immune responses?" In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-b054.

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Al Sharifi, Liqaa, Haider Abdul Ridha, Ahmed Rashid, Sinan Muhsin, and Teeb Jaafer. "Role of CD200 and CD43 in Diagnosis and Prognosis of CLL and NHL Patients." In 5th International Conference on Biomedical and Health Sciences. Cihan University-Erbil, 2024. http://dx.doi.org/10.24086/biohs2024/paper.1403.

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Background: Chronic lymphoproliferative disorder (LPD), is a malignant disease of lymphocytes in the blood and lymphatic tissue. Chronic lymphocytic leukaemia, it is the commonest type of chronic lymphoproliferative disorder. Scoring by immunophenotyping is used to differentiate B-cell chronic lymphocytic leukemia from other B-Non-Hodgkin lymphomas. CD200 (OX2) is a glycoprotein of membrane, it is related to type I superfamily of immunoglobulin . CD43 (Sialophorin) is a sialoglycoprotein that is present on the surface of T lymphocytes, some B lymphocytes, granulocytes and monocytes, that play

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Santanam, Urmila, Nicola Zanesi, Alexey Efanov, et al. "Abstract LB-352: B-CLL phenotype in Eµ-miR-29 transgenic mice." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-352.

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Lai, Hsien, Suping Zhang, Christina Wu, et al. "Abstract 950: Selective cytotoxicity of A6 peptide against ZAP-70 expressing CLL B-cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-950.

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Reports on the topic "B-CLL"

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CALS TEST NETWORK WRIGHT-PATTERSON AFB OH. TMSS Parsing Test MIL-M-9977J. Appendix J. NATO Stage B Cross-Servicing Checklist (CL2) Document Type Definition. Defense Technical Information Center, 1994. http://dx.doi.org/10.21236/ada306309.

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CALS TEST NETWORK WRIGHT-PATTERSON AFB OH. TMSS Parsing Test MIL-M-9977J. Appendix I. NATO Stage B Cross-Servicing Checklist (CL1) Document Type Definition. Defense Technical Information Center, 1994. http://dx.doi.org/10.21236/ada306310.

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CALS TEST NETWORK WRIGHT-PATTERSON AFB OH. TMSS Parsing Test, MIL-M-9977J, Appendix J, NATO Stage B Cross-Servicing Checklist (CL2), Document Type Definition. Defense Technical Information Center, 1994. http://dx.doi.org/10.21236/ada309304.

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Reid, M. S., X. Wang, N. Utting, and C. Jiang. Comparison of water chemistry of hydraulic-fracturing flowback water from two geological locations at the Duvernay Formation, Alberta, Canada. Natural Resources Canada/CMSS/Information Management, 2021. http://dx.doi.org/10.4095/329276.

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We analyzed and compared the water chemistry between 17 Fox Creek region samples, each from a different well, and 23 Three Hills region samples from a single well. Overall, the two regions were similar in chemical composition but showed small differences in some lower abundance dissolved elements. Additionally, we investigated changes in water chemistry of FPW over time from a single well. The majority of water quality parameters and water chemistry remained constant over the 7-month sampling time. Major ion chemistry showed increasing concentrations of Ca and Mg, and a decreasing concentratio

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Jauvin, Nathalie, François Aubry, Francis Ethridge, et al. Recherche-action visant le développement d’un modèle d’intervention préventive en SST par et pour les préposés aux bénéficiaires en CHSLD. IRSST, 2024. http://dx.doi.org/10.70010/nkup8051.

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Contexte Ce rapport a pour objectif de présenter les résultats d’une recherche-action visant le développement d’un modèle d’intervention préventive en santé et sécurité du travail (SST) par et pour les préposés aux bénéficiaires (PAB) dans les centres d’hébergement et de soins de longue durée (CHSLD) du Québec. Les PAB sont au cœur des soins prodigués dans les milieux gériatriques. Malheureusement, l’augmentation du nombre de blessures et l’accentuation des problématiques de santé psychologique subies par ce personnel depuis quelques années fragilisent leur situation, ce qui se prouve en terme

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Guy, Charles, Gozal Ben-Hayyim, Gloria Moore, Doron Holland, and Yuval Eshdat. Common Mechanisms of Response to the Stresses of High Salinity and Low Temperature and Genetic Mapping of Stress Tolerance Loci in Citrus. United States Department of Agriculture, 1995. http://dx.doi.org/10.32747/1995.7613013.bard.

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The objectives that were outlined in our original proposal have largely been achieved or will be so by the end of the project in February 1995 with one exception; that of mapping cold tolerance loci based on the segregation of tolerance in the BC1 progeny population. Briefly, our goals were to 1) construct a densely populated linkage map of the citrus genome: 2) map loci important in cold and/or salt stress tolerance; and 3) characterize the expression of genes responsive to cold land salt stress. As can be seen by the preceding listing of accomplishments, our original objectives A and B have

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