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Books on the topic 'B-2 B cells'

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Author: Grafiati

Published: 4 June 2021

Last updated: 20 February 2023

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1

Battaglini, Michelina. Determination of the transforming growth factor-B (TGF-B) receptor on the surface of interleukin-2 activated natural killer (IANK) cells. Sudbury, Ont: Laurentian University, 1992.

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2

Lane, Peter John Lockwood. Recruitment of virgin and mature B cells into antibody responses against thymus-dependent (TD) and independent (TI-2) forms of the hapten 2,4 dinitrophenyl (DNP). Birmingham: University of Birmingham, 1986.

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3

Glasier, Mary-Ann M. A role for SHP-1 and Vav in the abrogation of B cell receptor signal transduction by latent membrane protein 2 (LMP2). Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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4

Regulation of the transcription factors Oct-2 and NF-[kappa]B during pre-B cell differentiation. 1993.

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5

Rosado, Maria Manuela, Marcella Visentini, Sven Geissler, Alessandro Camponeschi, and Alaitz Aranburu, eds. The B-Side of B Cells. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-520-6.

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6

Chu, Yiwei, Damo Xu, and Luman Wang, eds. Insights into Regulatory B Cells. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-155-5.

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7

Molecular Biology of B Cells. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-12-053641-2.x5000-x.

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8

Gibbins, Jonathan M., and Martyn P. Mahaut-Smith. Platelets and Megakaryocytes : Volume 2: Perspectives and Techniques. Humana Press, 2010.

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9

Casali, Paolo, ed. Epigenetics of B Cells and Antibody Responses. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-790-3.

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10

Lloyd, Peter, Sarah Doaty, and Bevra H. Hahn. Aetiopathogenesis of systemic lupus erythematosus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198739180.003.0002.

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Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by the presence of immune dysregulation, autoreactive B and T cells, and the production of a broad, heterogeneous group of autoantibodies (autoAb). The pathogenesis of lupus can be divided into three stages: 1) genetic predisposition and environmental exposures, 2) loss of tolerance, and 3) immune activation. In this chapter we will discuss the aetiopathogenesis of systemic lupus erythematosus with emphasis placed on key autoantibodies, cytokines, the innate and adaptive immune system, tolerance, NETosis, genetics and epigenetics, environmental triggers and the role of gender.

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11

Fleischmann, Roy. Signalling pathway inhibitors. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0081.

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Oral, small-molecule signalling pathway inhibitors, including ones that inhibit the JAK and SyK pathways, are currently in development for the treatment of rheumatoid arthritis (RA). Tofacitinib is an orally administered small-molecule inhibitor that targets the intracellular Janus kinase 3 and 1 (JAK1/3) molecules to a greater extent than JAK2 while baricitinib (formerly INCB028050) predominantly inhibits JAK1/2. Many of the proinflammatory cytokines implicated in the pathogenesis of RA utilize cell signalling that involves the JAK-STAT pathways and therefore inhibition of JAK-STAT signalling, by targeting multiple RA-associated cytokine pathways, has the potential to simultaneously reduce inflammation, cellular activation, and proliferation of key immune cells. Fostamatinib disodium is an orally available inhibitor of spleen tyrosine kinase (SyK), which is a cytoplasmic tyrosine kinase that is an important mediator of immunoreceptor signalling in mast cells, macrophages, neutrophils, and B cells. Interruption of SyK signalling may interrupt production of tumour necrosis factor (TNF) and metalloproteinase and therefore affect RA disease activity. Tofacitinib has been investigated in multiple phase 2 and phase 3 trials which have investigated its efficacy (clinical, functional, and radiographic) and safety in patients who have failed disease-modifying anti-inflammatory drugs (DMARDs) as monotherapy or in combination with DMARDs, compared to an inhibitor of tumour necrosis factor alpha (TNFα‎) and in patients who have failed TNFα‎ inhibitors. The efficacy of fostamatinib and baricitinib has been investigated in phase 2 trials; both are in large phase 3 clinical programmes. Each of these medications has demonstrated efficacy; their safety profile has been shown to be different from each other and from currently approved biological agents. This chapter discusses what is currently known and understood about their efficacy and safety.

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12

Gilli, Francesca, Roberta Magliozzi, Laura Piccio, and Enrique Alvarez, eds. B Cells in Inflammatory and Neurodegenerative Diseases of the Central Nervous System. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-638-8.

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13

Chiodi, Francesca, and Gabriella Scarlatti, eds. HIV-Induced Damage of B Cells and Production of HIV Neutralizing Antibodies. Frontiers Media SA, 2018. http://dx.doi.org/10.3389/978-2-88945-461-7.

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14

Dvorak, Antonin. Music Minus One Cello: Dvorak Violoncello Concerto in B minor, op. 104 (Sheet Music & 2 CDs). Music Minus One, 1995.

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15

Music Minus One Cello: Schubert Piano Trio in B-flat major, op. 99, D898 (Sheet Music & 2 CDs). Music Minus One, 1998.

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16

The Role of IL-2 in B cell physiology: Workshop at the Basel Institute for Immunology, October 14-15, 1985. Basel: Editiones Roche, 1986.

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17

Brahms, Johannes. Johannes Brahms Three Quartets: For Two Violins, Viola and Cello, No. 1, Opus 51/1 in C Minor, No. 2, Opus 51/2 in A Minor, No. 3, Opus 67 in B-flat Magor, Miniature Score: Kalmus Cl (Kalmus Edition). Alfred Publishing Company, 1985.

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