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Academic literature on the topic 'Awareness of cognitive decline'

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Published: 25 May 2024

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Journal articles on the topic "Awareness of cognitive decline"

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Vogel, Asmus, Frans Boch Waldorff, and Gunhild Waldemar. "Longitudinal changes in awareness over 36 months in patients with mild Alzheimer's disease." International Psychogeriatrics 27, no. 1 (July 30, 2014): 95–102. http://dx.doi.org/10.1017/s1041610214001562.

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ABSTRACTBackground:Longitudinal changes in awareness in dementia have been studied with short follow-up time and mostly in small patient groups (including patients with moderate dementia). We investigated awareness in patients with mild Alzheimer's disease (AD) over 36 months and studied if a decline in awareness was associated with decline in cognition and increase in neuropsychiatric symptoms.Methods:Awareness was measured on a categorical scale in 95 AD patients (age ≥50 years, Mini-Mental State Examination (MMSE) score ≥20). Awareness was rated at three time points (follow-up at 12 and 36 months) where MMSE, Neuropsychiatric Inventory (NPI-Q), and Cornell scale for Depression in Dementia also were applied.Results:At 12 months, 26% had lower awareness rating as compared to baseline and at 36 months lower awareness ratings were found in 39%. At both visits, 16% had higher awareness rating as compared to baseline. Patients with lower awareness at 36 months as compared to baseline had a more rapid increase in NPI-Q score (p = 0.002) over 36 months as compared to patients with stable or improved awareness over 36 months. A more rapid decline in MMSE score was observed for patients with lower awareness at 36 months (as compared to baseline) but only when compared to patients in whom awareness improved over time.Conclusions:The results show essentially no clear relationship between cognitive decline over three years and awareness. In some cases, awareness remained stable or even improved despite significant cognitive decline. In the subgroup where awareness declined over time, overall ratings of neuropsychiatric symptoms declined more rapidly than in the remaining patients.
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Tahmasebi, R., S. Zehetmayer, E. Stögmann, and Johann Lehrner. "Awareness of Olfactory Dysfunction in Subjective Cognitive Decline, Mild Cognitive Decline, and Alzheimer’s Disease." Chemosensory Perception 13, no. 1 (June 24, 2019): 59–70. http://dx.doi.org/10.1007/s12078-019-09267-7.

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Ameriks, John, Andrew Caplin, Minjoon Lee, Matthew D. Shapiro, and Christopher Tonetti. "Cognitive Decline, Limited Awareness, Imperfect Agency, and Financial Well-Being." American Economic Review: Insights 5, no. 1 (March 1, 2023): 125–40. http://dx.doi.org/10.1257/aeri.20210711.

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Cognitive decline may lead older Americans to make poor financial decisions. Preventing poor decisions may require timely transfer of financial control to a reliable agent. Cognitive decline, however, can develop unnoticed, creating the possibility of suboptimal timing of the transfer of control. This paper presents survey-based evidence that older Americans with significant wealth regard suboptimal timing of the transfer of control, in particular delay due to unnoticed cognitive decline, as a substantial risk to financial well-being. This paper provides a theoretical framework to model such a lack of awareness and the resulting welfare loss. (JEL G51, G53, H55, J14, J26, J32)
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Therriault, Joseph, Kok Pin Ng, Tharick A. Pascoal, Sulantha Mathotaarachchi, Min Su Kang, Hanne Struyfs, Monica Shin, et al. "Anosognosia predicts default mode network hypometabolism and clinical progression to dementia." Neurology 90, no. 11 (February 14, 2018): e932-e939. http://dx.doi.org/10.1212/wnl.0000000000005120.

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ObjectiveTo identify the pathophysiologic mechanisms and clinical significance of anosognosia for cognitive decline in mild cognitive impairment.MethodsWe stratified 468 patients with amnestic mild cognitive impairment into intact and impaired awareness groups, determined by the discrepancy between the patient and the informant score on the Everyday Cognition questionnaire. Voxel-based linear regression models evaluated the associations between self-awareness status and baseline β-amyloid load, measured by [18F]florbetapir, and the relationships between awareness status and regional brain glucose metabolism measured by [18F]fluorodeoxyglucose at baseline and at 24-month follow-up. Multivariate logistic regression tested the association of awareness status with conversion from amnestic mild cognitive impairment to dementia.ResultsWe found that participants with impaired awareness had lower [18F]fluorodeoxyglucose uptake and increased [18F]florbetapir uptake in the posterior cingulate cortex at baseline. In addition, impaired awareness in mild cognitive impairment predicted [18F]fluorodeoxyglucose hypometabolism in the posterior cingulate cortex, left basal forebrain, bilateral medial temporal lobes, and right lateral temporal lobe over 24 months. Furthermore, participants with impaired awareness had a nearly 3-fold increase in likelihood of conversion to dementia within a 2-year time frame.ConclusionsOur results suggest that anosognosia is linked to Alzheimer disease pathophysiology in vulnerable structures, and predicts subsequent hypometabolism in the default mode network, accompanied by an increased risk of progression to dementia. This highlights the importance of assessing awareness of cognitive decline in the clinical evaluation and management of individuals with amnestic mild cognitive impairment.
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Lehrner, Johann, Sandra Kogler, Claus Lamm, Doris Moser, Stefanie Klug, Gisela Pusswald, Peter Dal-Bianco, Walter Pirker, and Eduard Auff. "Awareness of memory deficits in subjective cognitive decline, mild cognitive impairment, Alzheimer's disease and Parkinson's disease." International Psychogeriatrics 27, no. 3 (November 10, 2014): 357–66. http://dx.doi.org/10.1017/s1041610214002245.

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ABSTRACTBackground:Impaired awareness of memory deficits has been recognized as a common phenomenon in Alzheimer's disease (AD) and research is now increasingly focusing on awareness in groups at risk for future dementia. This study aimed to determine whether levels of awareness differ among healthy elderly people and patients with subjective cognitive decline (SCD), amnestic and non-amnestic subtypes of mild cognitive impairment (aMCI, naMCI), Alzheimer's disease (AD) and Parkinson's disease (PD), to explore correlates of awareness and to establish frequencies of memory over- and underestimation within each diagnostic group.Methods:756 consecutive outpatients of a memory clinic and 211 healthy controls underwent thorough neuropsychological testing. Impairment of awareness was measured as the difference between subjective memory appraisals (16-item questionnaire on current memory-related problems in everyday life) and objective memory performance (15-item delayed recall task). Subgroups of over- and underestimators were classified using percentile ranks of controls.Results:At group level, awareness significantly decreased along the naMCI→aMCI→AD continuum, with naMCI patients showing a tendency towards overestimation of memory dysfunction. PD patients showed accurate self-appraisals as long as memory function was largely unaffected. However, there was a considerable between-group overlap in awareness scores. Furthermore, different correlates of awareness were observed depending on the diagnostic group. In general, unawareness seems to be associated with decreased cognitive performance in various domains (especially memory), higher age and lower levels of depression and self-reported functional impairment.Conclusion:Impaired awareness is an important symptom in aMCI. Yet, given the considerable variability in awareness scores, longitudinal studies are required to evaluate their predictive power.
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Dourado, M., M. F. B. Sousa, R. L. Santos, P. Simões, T. Belfort, M. Nogueira, R. Dias, B. Torres, M. A. T. Baptista, and I. B. Lacerda. "The Independence Between Awareness and Cognitive Decline in Alzheimer's Disease." European Psychiatry 30 (March 2015): 452. http://dx.doi.org/10.1016/s0924-9338(15)30357-6.

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Costa, Amy, Christina McCrae, Jennifer O'Brien, and Ashley Curtis. "0083 You Don’t Snooze, You Lose (Awareness): Sleep’s Role on Awareness of Cognition in Mild Cognitive Impairment." SLEEP 47, Supplement_1 (April 20, 2024): A36—A37. http://dx.doi.org/10.1093/sleep/zsae067.0083.

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Abstract Introduction In adults with Mild Cognitive Impairment (prodromal phase of Alzheimer’s disease; MCI), the level of awareness of cognitive functioning varies significantly. A lack of awareness of cognitive functioning may interfere with monitoring of disease progression and opportunities for intervention. Therefore, understanding mechanisms associated with this relationship is critical. Given age-related changes in sleep and known relationships between sleep health and cognition, this pilot tested whether sleep moderates associations between subjective and objective cognition in older adults with MCI. Methods Older adults with MCI (N=46, Mage=68.2 years, SD=6.1 years, 33 women) completed one week of sleep diaries [averages of sleep onset latency (SOL), total sleep time (TST), sleep efficiency (SE)], Cognitive Failures Questionnaire (sub-scores CFQ-memory, CFQ-distractibility, CFQ-blunders), and cognitive tasks [Cambridge Brain Sciences; Polygons (processing speed), Feature Match (attention), Double Trouble (attention/inhibition), Digit Span (working memory)]. Multiple regressions tested if sleep parameters moderated associations between subjective and objective cognition, covarying for depressive symptoms and number of medical conditions. Results SE moderated associations between Polygons and CFQ-blunders (R2-change=.08, p=.03). Specifically, at highest SE, slower processing speed was associated with more blunder complaints (B=-.10, p=.04). Additionally, SE moderated associations between Digit Span and CFQ-distractibility (R2-change=.06, p=.03) and CFQ-blunders (R2-change=.12, p=.004). Specifically, at lowest SE, worse working memory was associated with less distractibility (B=3.01, p=.002) and blunders (B=2.45, p<.001) complaints, while at highest SE worse working memory was associated with more blunder complaints (B=-2.48, p=.04). Conclusion Preliminary findings suggest in older adults with MCI, lower sleep efficiency may exacerbate discrepancies between objective and subjective cognition, while higher sleep efficiency may converge this relationship. Sleep efficiency should be considered to fully understand the level of awareness of cognitive functioning in older adults with MCI. Critically, these findings may help identify those at risk of further cognitive decline. While future prospective studies are warranted, present findings suggest a potential next step is to examine whether behavioral treatments for insomnia (e.g., Cognitive Behavioral Therapy for Insomnia, cognitive training) may also change the relationship between objective/subjective cognition. Support (if any) This research was supported in part by the University of Missouri School of Medicine TRIUMPH award (PI: Curtis).
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Taler, Vanessa, Cassandra Morrison, and Christine Sheppard. "Cognitive Performance in Older Adults With Subjective Cognitive Decline." Innovation in Aging 4, Supplement_1 (December 1, 2020): 291. http://dx.doi.org/10.1093/geroni/igaa057.932.

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Abstract Subjective cognitive decline (SCD) refers to a perceived decline in cognitive function in the absence of neuropsychological deficits. Older adults with SCD are at increased of subsequent development of mild cognitive impairment or dementia. We had 224 adults aged 65+ complete questionnaires assessing their subjective appraisal of their cognitive function, including questions about word-finding difficulty, memory, and attention/concentration. Participants also completed the Montreal Cognitive Assessment (MoCA). All participants exhibited cognitive performance that was within normal limits for age and education. In total, 29.5% of participants reported word-finding difficulties, 16.5% reported difficulties with remembering things, and 8.5% reported difficulties with attention/concentration. We found that (1) self-reported word-finding difficulties were associated with lower performance on delayed word recall, and (2) self-reported difficulties in concentration/attention or memory were associated with lower performance on the abstraction subtask in the MoCA. No other MoCA subtasks were associated with self-reported cognitive function. A subset of the participants (n=69) also completed a battery of tasks assessing semantic function, including picture naming, associative matching tasks, identification of semantic features, and semantic questions. Again, self-reported word-finding difficulty predicted lower performance on semantic tasks. These results suggest that older adults may be aware of changes in their cognitive performance prior to objective neuropsychological impairment. Moreover, their awareness appears to be domain-specific: self-reported language difficulty is associated with lower performance on language-based tasks, while self-reported difficulty in memory, attention, or concentration is associated with lower performance on an abstraction task.
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Lautenschlager, Nicola T. "Awareness of memory deficits in subjective cognitive decline, mild cognitive impairment, Alzheimer's disease and Parkinson's disease." International Psychogeriatrics 27, no. 3 (February 2, 2015): 355–56. http://dx.doi.org/10.1017/s1041610214002786.

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Anosognosia or reduced awareness of cognitive and functional problems is a known clinical phenomenon in patients experiencing cognitive decline. It is common even in mild dementia syndromes and is often reported to increase with clinical disease progression (Aalten et al., 2006). More recently it also has been described in older adults with cognitive-impairment-no-dementia (CIND) and mild cognitive impairment (MCI) (Greenop et al., 2011; Maki et al., 2012). The clinical significance of awareness is a topic of controversy in the literature and determining its biological correlates in brain function and structure is an ongoing challenge (Zamboni et al., 2013; Ford et al., 2014).
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Gigi, Ariela, and Merav Papirovitz. "Association of Anxiety Awareness with Risk Factors of Cognitive Decline in MCI." Brain Sciences 11, no. 2 (January 21, 2021): 135. http://dx.doi.org/10.3390/brainsci11020135.

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Studies demonstrate that anxiety is a risk factor for cognitive decline. However, there are also study findings regarding anxiety incidence among people with mild cognitive impairment (MCI), which mostly examined general anxiety evaluated by subjective questionnaires. This study aimed to compare subjective and objective anxiety (using autonomic measures) and anxiety as a general tendency and anxiety as a reaction to memory examination. Participants were 50 adults aged 59–82 years who were divided into two groups: MCI group and control group, according to their objective cognitive performance in the Rey Auditory Verbal Learning Test. Objective changes in the anxiety response were measured by skin conductivity in all tests and questionnaires. To evaluate subjective anxiety as a reaction to memory loss, a questionnaire on “state-anxiety” was used immediately after completing memory tests. Our main finding was that although both healthy and memory-impaired participants exhibited elevations in physiological arousal during the memory test, only healthy participants reported an enhanced state anxiety (p = 0.025). Our results suggest that people with MCI have impaired awareness of their emotional state.
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Dissertations / Theses on the topic "Awareness of cognitive decline"

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Cacciamani, Federica. "Awareness of cognitive decline in early-stage alzheimer's disease : implications for diagnosis, patient management and research." Electronic Thesis or Diss., Sorbonne université, 2021. http://www.theses.fr/2021SORUS295.

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Nous décrivons 6 études originales, menées sur le cohorte française INSIGHT-PreAD et la cohorte américaine ADNI, qui abordent différents aspects de l'association entre le niveau de conscience des patients du déclin cognitif (« awareness of cognitive decline » en anglais, ou ACD) et leur risque de maladie d'Alzheimer (MA). Nous avons utilisé des méthodes statistiques classiques et l'apprentissage automatique. Nous montrons qu'un déclin subtil est déjà présent dans la MA préclinique. Les patients peuvent remarquer ces changements précoces alors que ni son entourage ni les tests cognitifs ne le font. Cependant, l’entourage et les tests deviennent très vite des sources d'information plus fiables que le patient lui-même : au cours de la progression du stade préclinique au stade prodromal, l'ACD commence à décliner. Dans la MA prodromale, le patient est légèrement anosognosique, jusqu'à ce qu'il atteigne une anosognosie franche dans la démence MA. Le clinicien doit tenir compte de la plainte du patient, mais aussi la comparer à d'autres sources d'information plus fiables. Cela peut également aider à une meilleure sélection des sujets pour les études ciblant la MA pré-démentielle
We describe 6 original studies, conducted on INSIGHT-PreAD and ADNI cohorts, that address different aspects of the association between patients' level of awareness of cognitive decline (ACD) and their risk of Alzheimer’s disease (AD). We used both classical statistical methods and machine learning. We show that a subtle decline is already present in preclinical AD. Patients may notice these early changes when neither the informant nor cognitive tests do. However, the informant and tests very quickly become more reliable sources of information than the patient him/herself: during the progression from the preclinical to the prodromal stage, ACD begins to decline. In prodromal AD, the patient is mildly anosognosic, until he/she usually reaches clear anosognosia in AD dementia. The clinician should consider the patient's complaint but also compare it to other, more reliable sources of information. This may also help in a better subject selection for studies targeting pre-dementia AD
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McAulay, Vincent. "Cognitive decline during acute hypoglycaemia." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/24922.

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The initial chapters of this thesis describe the clinical and physiological aspects of hypoglycaemia, followed by a review of the literature on the effects of acute hypoglycaemia on cognitive function. The subsequent chapters describe original research studies in subjects with and without diabetes, which examine the effects of acute hypoglycaemia on aspects of cognitive function and the prevention of hypoglycaemia. In Studies 1 to 3, a hyperinsulinemic glucose clamp was used to either maintain euglycemia (blood glucose 4,5 mmol/l) or induce hypoglycaemia (2.6 mmol/l) in both healthy adults (n=20), and subjects with type 1 diabetes (n=16). A cognitive test battery was administered to examine aspects of attention, intelligence, motivation, affect and subjective cognition. Hypoglycaemia induced a significant deterioration in tests sensitive to both visual and auditory selective attention, and attentional flexibility deteriorated (Studies 1 and 2). Intelligence scores did not deteriorate during hypoglycaemia (Studies 1 and 2). In Study 3, hypoglycaemia increased task-irrelevant interference and self-focus of attention, but motivation declined to a similar extent during both study conditions. Hypoglycaemia produced a negative mood state with a significant fall in energy levels and a concomitant rise in anxiety (Study 3). Study 4 was an open-label, comparative study of the post-prandial glucodynamics of insulin lispro, when administered either 5 minutes before or 20 minutes after a high fat/high solid phase meal, in twelve subjects with type 1 diabetes. Administration of insulin lispro after the meal reduced the risk of early postprandial hypoglycaemia, without compromising postprandial glycaemic control. Therefore, the work in this thesis has demonstrated a different deterioration of attentional function in humans during hypoglycaemia with no effect on non-verbal reasoning skills. Furthermore, it would appear that the brain is not only less cognitively competent and more dysphoric during hypoglycaemia, it is also more self-aware and distracted when required to perform effortful processing.
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De, Marco Matteo. "Cognitive and physical stimulation, genetic risk and cognitive decline." Thesis, University of Sheffield, 2012. http://etheses.whiterose.ac.uk/3379/.

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Miralbell, Blanch Júlia. "Biomarkers of cognitive decline and dementia." Doctoral thesis, Universitat de Barcelona, 2012. http://hdl.handle.net/10803/91068.

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Cognitive impairment in the elderly encompasses many forms, ranging from subtle impairments in otherwise cognitively healthy individuals through mild cognitive impairment and dementia. Brain structural and functional changes underlie the observed cognitive impairment. Complementary to the clinical observation, biomarkers have been proposed as in vivo indicators of the underlying pathophysiology and neurobiological changes in a sufficiently reliable manner that they could be used to detect, track, and predict the disease course over time. In this thesis we used a combination of epidemiological and clinic-based approaches to investigate the mechanisms underlying vascular cognitive impairment (VCI) and Alzheimer’s disease (AD) and to identify possible biomarkers that could help early diagnosis of such conditions. To do so, a set of circulating and cerebrospinal fluid (CSF) biomarkers were studied in healthy and cognitively impaired subjects. Then, these measures were related to grey matter (GM) volumes, white matter (WM) integrity and cognition. The first two studies are part of the population-based Barcelona-ASIA neuropsychology study. Study I aimed to compare the cognitive patterns of risk markers for cerebrovascular disease (CVD) with the cognitive profile in relation to novel and traditional vascular risk factors (VRF) in a community-dwelling sample. Biomarkers of inflammation, endothelial dysfunction and vascular thrombosis were selected. Results showed that VRF and circulating markers of inflammation and endothelial dysfunction predicted performance in several cognitive domains. Cognitive patterns of inflammatory markers overlapped those related to VRF. Markers of endothelial dysfunction predicted lower performance in verbal memory. Study II was designed to further explore the structural changes mediating the relationships between risk markers of CVD and cognition. For that purpose the same set of markers of risk for CVD were related to GM atrophy and WM integrity and cognition. The main finding was an association of inflammation and vascular thrombosis with WM integrity loss in cortico-subcortical pathways and association fibres of frontal and temporal lobes. As expected, none of the biomarkers was related to GM volume changes. Vascular thrombosis also predicted lower performance in processing speed. The third study is a memory clinic-based investigation that was conducted aiming to test the potential use of CSF biomarkers cut-offs as components for the diagnostic work-up in AD. We assessed GM and cognitive patterns in cognitively impaired subjects using CSF Aβ1-42, t-tau and p-tau181 cut-offs as grouping criteria. Results indicated that patients with abnormal CSF levels of t-tau and p-tau (but not Aβ1-42) showed impairment and signs of regional GM atrophy in brain regions characteristic for AD, compared to those with normal levels. More specifically, GM volume differences were found in temporal, inferior parietal, lateral occipital and widespread prefrontal regions. Studies I and II show that risk markers of inflammation and vascular thrombosis are related to a VCI profile for both cognitive patterns and structural brain changes. A microvascular damage of WM projections in fronto-subcortical pathways, but not GM atrophy, could mediate the association between these pathogenic processes and cognitive performance. Markers of endothelial dysfunction are related to a different cognitive pattern which is characteristic of both vascular and neurodegenerative mechanisms. Study III provides evidence that patients with abnormal CSF levels of t-tau and p-tau (but not Aβ1-42) show cognitive an AD profile according to GM density patterns and cognitive impairment. Taken together, these results suggest that, complementary to the clinical observation, plasma and CSF markers and structural imaging are well placed to improve early diagnosis of both VCI and AD.
El terme deteriorament cognitiu (DC) es refereix al contínuum de canvis cognitius associats a l’envelliment sa i patològic. El diagnòstic precoç de les persones amb DC és clau, ja que els tractaments són més eficaços quan s’inicien als inicis de la malaltia. Els biomarcadors s’han proposat com a eines pel diagnòstic precoç del DC i la demència. Es consideren indicadors in vivo de la patologia i s’han plantejat com a possibles eines pel diagnòstic, pronòstic i seguiment del DC i la patologia subjacent. L’objectiu general de la present tesi era explorar els mecanismes patofisiològics subjacents al deteriorament cognitiu vascular (DCV) i la (MA). Per aquest motiu, vàrem mesurar diversos biomarcadors sanguinis i de LCR en persones sanes i en persones amb diagnòstic de deteriorament cognitiu i vàrem relacionar-los amb canvis de l’estructura cerebral i de la cognició. L’objectiu final era identificar possibles biomarcadors pel diagnòstic precoç d’aquestes malalties. Els estudis I i II s’emmarquen dins del projecte Barcelona-ASIA Neuropsicologia i tenien com a objectiu estudiar la relació entre biomarcadors en plasma de malaltia vascular cerebral (MVC) i canvis estructurals i cognitius. Els resultats obtinguts mostren que els biomarcadors d’inflamació i trombosi vascular es relacionen amb un perfil de deteriorament cognitiu vascular tant a nivell cognitiu com estructural. La lesió microvascular dels tractes de SB còrtico-subcorticals mediaria l’associació entre aquests mecanismes i la cognició. Els marcadors de disfunció endotelial es relacionen amb un perfil cognitiu diferent, que és característic tant de processos vasculars com neurodegeneratius. L’estudi III té com a objectiu valorar el possible ús dels biomarcadors de líquid cefaloraquidi pel diagnòstic de la MA. En concret, vàrem estudiar els perfils estructurals i cognitius en persones amb deteriorament cognitiu emprant punts de tall de líquid cefaloraquidi com a criteri d’agrupació. Els resultats mostren que pacients amb DC i amb nivells patològics de t-tau i p-tau al LCR (però no d’Aβ1-42) presenten un perfil cognitiu i estructural de MA. En conclusió, els resultats obtinguts en la present tesi suggereixen que, complementaris a l’observació clínica, els biomarcadors de LCR i plasma, així com els indicadors de morfologia cerebral podrien ser d’ús pel diagnòstic precoç del DCL i la demència.
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Lu, Zhengdong. "Constrained clustering and cognitive decline detection /." Full text open access at:, 2008. http://content.ohsu.edu/u?/etd,650.

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Marston, Kieran John. "Preventing cognitive decline through resistance training." Thesis, Marston, Kieran John (2019) Preventing cognitive decline through resistance training. PhD thesis, Murdoch University, 2019. https://researchrepository.murdoch.edu.au/id/eprint/51337/.

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The foundations for later-life cognitive health are often laid decades prior to the first symptoms of cognitive decline, therefore preventative strategies should be implemented early in life. Structured resistance training shows promise at reducing dementia risk and has been associated with enhanced cognitive function and biomarker profiles. More specifically, intense acute resistance exercise and chronic resistance training may increase levels of growth factors, such as brain-derived neurotrophic factor (BDNF), insulin-like growth factor (IGF-1) and vascular endothelial growth factor (VEGF), which influence the proliferation and maintenance of neural and vascular tissue. Furthermore, biomarkers associated with suboptimal cognitive health (e.g., homocysteine) are decreased following longer-term resistance training. Although promising, the current literature is inconsistent, with varying resistance exercise regimens implemented, contributing to conflicting reported effects on cognition and biomarkers. The aim of the current thesis was to evaluate the impact of different strength training protocols on cognitive health. Four studies were conducted to examine acute response in growth factors following resistance exercise in young and older adults, and the chronic response in neurotrophic growth factors and cognitive function following resistance training in older adults. Chapters Four and Five explored the acute response in neurotrophic growth factors to intense resistance exercise in young or late middle-aged adults. Sixteen young adults were recruited (Chapter Four) to perform to-fatigue (i.e., maximal), high-volume acute resistance exercise bouts: i) traditional hypertrophy-based resistance exercise (i.e., three sets, 10 repetitions at 100% of 10 repetition maximum [RM]), or ii) traditional strength-based resistance exercise (i.e., five sets, five repetitions at 100% of 5RM). Levels of serum BDNF and blood lactate concentration (i.e., physiological marker of session intensity) were measured prior to and following exercise. Serum BDNF was increased (p<0.01; d=0.52) immediately post-hypertrophy resistance exercise when compared to strength resistance exercise. Change in serum BDNF levels were positively correlated (r=0.70; p<0.01) with change in blood lactate concentration immediately following hypertrophy-based resistance exercise only. The acute BDNF response to resistance exercise may not be consistent across the lifespan, and to-fatigue and high-volume resistance exercise is not practical in ageing adults. For this reason, Chapter Five explored the acute growth factor response to intense, yet pragmatic resistance exercise (i.e., submaximal and lower-volume) in 29 late middle-aged adults. Two resistance exercise sessions i) moderate-load (i.e., three sets, 10 repetitions at 70% of 1RM), or ii) high-load (i.e., five sets, five repetitions at 85% of 1RM) were performed in parallel groups (Chapter Six and Seven). Session intensity was determined by change in blood lactate concentration, and session rating of perceived exertion (sRPE). Serum samples were taken prior to and following exercise for later BDNF, IGF-1, and VEGF analysis. No acute changes in BDNF, IGF-1 or VEGF were observed. Changes in BDNF, IGF-1 or VEGF were not associated with changes in blood lactate concentration or sRPE. My findings provide evidence that to-fatigue, high-volume resistance exercise can increase acute levels of BDNF; however, under more practical resistance training scenarios (e.g., moderate volume, submaximal resistance exercise) this outcome is less likely to occur. Resting levels of BDNF, IGF-1, VEGF and homocysteine are important predictors for later life cognitive function in ageing adults; thus, Chapter Six explored changes in resting blood markers following a 12-week period of intense, lower-volume resistance training twice per week in 45 late middle-aged adults. Participants were randomised into one of three groups; i) moderate-load (i.e., three sets, 10 repetitions at 70% of 1RM), ii) high-load (i.e., five sets, five repetitions at 85% of 1RM), or iii) a non-exercising (i.e., no intervention) control group for comparison. Fasted levels of serum BDNF, IGF-1, VEGF, and plasma homocysteine were quantified from blood samples collected before and after the 12-week intervention. No differences were observed in BDNF, IGF-1, VEGF, or plasma homocysteine from pre- to post- intervention between groups. Despite an intense training stimulus, it is possible that the protocols implemented here were not of high enough volume to enhance growth factors. However, high-intensity and high-volume resistance exercise is not a practical training model in ageing adults. Chapters Four-Six placed a strong emphasis on the importance of physiology, and the levels of certain biomarkers, in regards to cognitive health. Therefore, it is essential to evaluate the functional outcomes of resistance training on cognitive health. As part of the randomised controlled trial introduced in Chapter Six, Chapter Seven explored the response in cognitive function following 12 weeks of intense resistance training in 45 late middle-aged adults. Participants were randomised into either i) moderate-load resistance training, ii) high-load resistance training, or iii) a non-exercising control group. Cognitive function was assessed using the CogState computerised cognitive battery, which evaluates performance within several cognitive domains. Greater delayed short-term memory (p=0.02) was observed in high-load and moderate-load groups when compared to the control, with no difference observed between resistance training groups. No other differences for changes in cognitive function between groups were observed. In cognitively healthy adults, 12 weeks of intense resistance training enhances short-term memory, a finding supporting the use of structured resistance training to promote physical and cognitive health. In conclusion, the findings presented in this thesis provide evidence that resistance exercise needs to be to-fatigue and high in volume to enhance BDNF levels. Intense resistance exercise in late middle-aged adults influences neither the acute nor chronic response in neurotrophic growth factors or homocysteine when resistance exercise is reduced in total volume. However, 12 weeks of intense resistance training enhances short-term memory in cognitively healthy adults, a relevant outcome that supports the hypothesis that resistance training can contribute to reducing dementia risk. These findings support the use of structured resistance training to promote full body health inclusive of physical and cognitive health.
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Fortman, James Alexander. "Computer-Based Cognitive Training for Age-Related Cognitive Decline and Mild Cognitive Impairment." Antioch University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=antioch1353454752.

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Hogervorst, Eva. "Age-related cognitive decline and cognition enhancers." Maastricht : Maastricht : Neuropsych Publishers ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=6058.

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Saxton, Judith. "Cognitive and functional decline in Alzheimer's disease." Thesis, University of Reading, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254512.

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Morrell, Lucy. "Informant reported cognitive decline in older adults." Thesis, Canterbury Christ Church University, 2017. http://create.canterbury.ac.uk/16424/.

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Objectives: Gaining an informant’s perspective on cognitive decline has become an increasingly popular, and recommended practice, in the assessment of dementia. However, concern regarding the accuracy of such reports has been documented. The current study aimed to explore factors that might influence such reports, with a particular focus on informant burden. Design: Using a cross-sectional, single-group design, routinely collected data from 82 patient-informant dyads within a memory assessment service, was analysed. Univariate and multivariate analyses explored associations between informant-reported cognitive decline, demographic characteristics and clinical variables (including burden). Results: None of the demographic characteristics explored were associated with informant-reported cognitive decline. Informant reports were associated with patient cognitive functioning, as assessed by a standardised psychometric measure, and the final outcome of the assessment. Patient affective state and informant-reported burden interacted in influencing informant-reported cognitive decline. Informant-reported burden did not mediate the relationship between informant-reported cognitive decline and patient performance on a standardised psychometric measure. Conclusions: Findings suggest that informant subjective burden predicts informant-reported cognitive decline, and that patient affective state interacts with subjective burden in doing so. Clinical and empirical implications are discussed.
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Books on the topic "Awareness of cognitive decline"

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Leshner, Alan I., Story Landis, Clare Stroud, and Autumn Downey, eds. Preventing Cognitive Decline and Dementia. Washington, D.C.: National Academies Press, 2017. http://dx.doi.org/10.17226/24782.

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Howard, Fillit, and Butler Robert N. 1927-, eds. Cognitive decline: Strategies for prevention. 2nd ed. London: Greenwich Medical Media, 1997.

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1956-, Brown Don, ed. Dementia and age-related cognitive decline. Seattle, WA: Natural Product Research Consultants, 1997.

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United States. Agency for Healthcare Research and Quality and Duke University Evidence-based Practice Center, eds. Preventing Alzheimer's disease and cognitive decline. Rockville, MD: Agency for Healthcare Research and Quality, 2010.

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Meditation and Mental Health: An introduction to Awareness Based Cognitive Therapy. Whitstable [England]: New Yoga Publications, 2010.

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F, Bornstein Robert, and Pittman Thane S, eds. Perception without awareness: Cognitive, clinical, and social perspectives. New York: Guilford Press, 1992.

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Harris, Don, ed. Engineering Psychology and Cognitive Ergonomics: Performance, Emotion and Situation Awareness. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-58472-0.

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Janicki, Karol. Against essentialism: Toward language awareness. München: Lincom Europa, 1999.

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Tomc, Gregor. Geni, nevroni & jeziki: Duševnost kot flogiston sodobne kognitivne znanosti? Ljubljana: Fakulteta za družbene vede, 2011.

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Čok, Tina. Cognitive Implications for Raising Cross-language Awareness in Foreign Language Acquisition. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-27829-7.

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Book chapters on the topic "Awareness of cognitive decline"

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Polidori, Maria Cristina, Gereon Nelles, Umberto Senin, and Patrizia Mecocci. "Cognitive Decline." In Practical Issues in Geriatrics, 67–80. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-61997-2_7.

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Hedges, Dawson, Thomas J. Farrer, Erin D. Bigler, and Ramona O. Hopkins. "Postoperative Cognitive Decline." In The Brain at Risk, 139–54. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-14260-5_11.

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Xu, Hanzhang, Matthew E. Dupre, and James R. Burke. "Subjective Cognitive Decline." In Encyclopedia of Gerontology and Population Aging, 4798–801. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-22009-9_1136.

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Xu, Hanzhang, Matthew E. Dupre, and James R. Burke. "Subjective Cognitive Decline." In Encyclopedia of Gerontology and Population Aging, 1–4. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-69892-2_1136-1.

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Monsch, Andreas U., Panagiota Mistridis, and Alessandra Thomann. "Postponing Cognitive Decline." In Practical Issues in Geriatrics, 117–27. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-96529-1_13.

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Theisler, Charles. "Dementia/Cognitive Decline." In Adjuvant Medical Care, 90–91. New York: CRC Press, 2022. http://dx.doi.org/10.1201/b22898-103.

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Abrams, David B., J. Rick Turner, Linda C. Baumann, Alyssa Karel, Susan E. Collins, Katie Witkiewitz, Terry Fulmer, et al. "Age-Related Cognitive Decline." In Encyclopedia of Behavioral Medicine, 52. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_100050.

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Lugavere, Max, Alon Seifan, and Richard S. Isaacson. "Prevention of Cognitive Decline." In Handbook on the Neuropsychology of Aging and Dementia, 205–29. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-93497-6_14.

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Gutierrez, Jose, and Richard S. Isaacson. "Prevention of Cognitive Decline." In Handbook on the Neuropsychology of Aging and Dementia, 167–92. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3106-0_12.

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Larroy, Cristina, and Rosa Vera. "Cognitive Decline in Menopause." In Nutrition and Diet in Menopause, 347–57. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-373-2_26.

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Conference papers on the topic "Awareness of cognitive decline"

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Sundarapperuma, T. D., P. N. Kariyawasam, K. De Silva, H. Madushanth, and M. K. Gamage. "Informal Caregivers’ Perception of Barriers for Cognitive Health Promotion Activities for Older Adults in Galle District." In SLIIT International Conference on Advancements in Sciences and Humanities 2023. Faculty of Humanities and Sciences, SLIIT, 2023. http://dx.doi.org/10.54389/bgtk7766.

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Health promotion activities to improve cognition have shown a significant impact on the prevention of cognitive decline in elderly people. Limited studies are available on the caregivers’ perceptions of barriers to health promotion interventions to improve the cognitive health of older adults. A qualitative descriptive explorative study was conducted with the participation of 17 caregivers. Data were collected through focus group discussions, and all the sessions were audio recorded and transcribed verbatim. The thematic analysis was used for data analysis. The mean age ±SD of the participants was 40.24±4.7 years and 9 participants out of 17 were female. Five themes were identified based on discussions with caregivers. These themes were lack of awareness of cognitive decline and preventive interventions, the physical discomfort of older people, lack of financial independence, resource deficiency, and intransigent behavior. The findings revealed that the majority of caregivers do not have an appropriate understanding of cognitive health promotion interventions. Therefore, a well-structured, culturally sensitive, and person centered intervention programme would be beneficial to introduce cognitive health promotion activities for older adults and caregivers.
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Escudeiro, Paula, Márcia Campos, Francisca Escudeiro, and Nuno Escudeiro. "A serious game for the cognitive stimulation of seniors." In Intelligent Human Systems Integration (IHSI 2024) Integrating People and Intelligent Systems. AHFE International, 2024. http://dx.doi.org/10.54941/ahfe1004479.

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With the increasing number of elderly individuals in the Portuguese population and the consequent rise in cognitive decline associated with normal aging, there is a growing need to invest in mental health. This investment can be realized through the promotion of active aging and cognitive stimulation. Developing interactive tools that provide stimulation and motivation for everyday activities proves to be an effective strategy in slowing down cognitive decline. These tools aim to simulate familiar everyday scenarios for end-users, thereby encouraging regular use. This project focuses on the design and implementation of a platform to assist in evaluating and training the cognitive capacities of adults. The platform facilitates early awareness of cognitive deficiencies and stimulates users with pre-clinical symptoms, eliminating the need for visits to a medical office. It presents an innovative alternative to traditional tests conducted in clinical environments. Specifically, the platform recreates scenarios encountered during appointments with psychologists, transforming them into a game consisting of minigames. These minigames mimic real-world tasks, enabling a seamless integration of users' daily life results and enhancing their interaction with the environment. This approach aims to prevent the stress often associated with traditional cognitive stimulation programs and yield more accurate results without the pressure of a clinical setting. The primary goal is to develop a serious game that efficiently links simulation results to the daily activities of the targeted audience. This game adapts a scientifically validated cognitive training program to an Information Technology (IT) platform. The most significant outcome of this work is the cognitive stimulation of users and the effective integration of stimulation results. Through this serious game, the project strives to promote active aging, cognitive stimulation, and mental health using an interactive platform tailored to individual everyday life and activities.
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Liu, Yuhan, and Baosheng Wang. "Promoting indigenous cultural awareness through participatory game design with children." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1002406.

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As urbanization progresses in China's rural areas, so do the severity of social issues, including the decline of social assets, the recession of agricultural industries, the lack of community cohesion, and a weak sense of belonging. A decline in cultural awareness is the reason behind such phenomena, which stems from changes to residents' lifestyles and a lack of cultural beliefs. This issue also results in insufficient cultural awareness, weak cultural inheritance, and neglect of cultural values among community residents. To this end, this paper aims to examine an educational model to enhance the cultural awareness of local community residents.At present, there are two types of education methods to enhance cultural awareness: passive types and active types. For example, passive education refers to the enhancement of participants’ cultural qualities through the problem-solving style lesson and ‘implicit’ curriculum, while an active education might use reflective writing or PD to promote cultural awareness. Of the two, active education, represented by PD, is more conducive to participants' acceptance of cultural knowledge. PD is also an effective method for developing humanitarianism in developing countries. It can be applied to the special scenario of rural communities in China as a new solution for raising the cultural awareness of residents. This paper shares a specific case study of enhancing residents' cultural awareness in community collective memory using participatory game design.A total of eight subjects were selected in this study. Since children are the future of the community's cultural development, the subjects included 6 children and 2 adults. Unlike traditional PD, this study focused on attracting the interests of subjects and enhancing their abilities to inherit traditional culture through participatory game design. The study consisted of three workshops: the cultural exploration workshop, the game design workshop, and the game testing workshop. Activity theory was used as a basis to guide the choice of time, location, and power dynamics, from which a framework of participatory activities covering the four approaches of "probing", "telling", "acting", and "making" was developed for the workshops. To further enhance collaboration, participants were also provided with a complete set of toolkits during the three workshops, including role-playing tools, game idea cards, house of cards, scaffolding, etc. At the end of each workshop, the Cultural Awareness Scale, which contains the three elements of cultural cognition, cultural heritage, and cultural values, was administered to measure the change in cultural awareness of the subjects. A mixed methods approach was used in analysis to uncover underlying cultural associations. The study qualitatively analyzed the transcribed spoken words and behaviors of the subjects using multimodal analysis, and quantitatively analyzed the variations in the word count of the text and the level of detail in the elaboration. In summary, this case study is important for examining cultural education models and improving the cultural awareness of the population. It also provides a framework of activities for participatory design workshops, which can serve as a reference for further research.
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Cristina Barbosa Medeiros, Ana. "Designing Positive User Experiences to Encourage Older Adults’ Self-care." In 13th International Conference on Applied Human Factors and Ergonomics (AHFE 2022). AHFE International, 2022. http://dx.doi.org/10.54941/ahfe1001662.

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User experience can be defined as the sum of responses elicited from the user by a succession of events that take place during the interaction with a device or system, where not only the users’ capabilities are considered, but also their needs and attitudes. Older users are more likely to make decisions based on emotion and past experiences, as their other capabilities decline due to the inevitable ageing of the body tissues. In a simplistic view, user’s capabilities can be divided into four components: the sensory level is related to the inputs from the environment, whilst cognitive and affective levels judge and mediate what the most appropriate physical output for an interaction should be, according to the context in which the overall user experience takes place. In young adults, these responses tend to happen almost simultaneously. With ageing, there is a natural decay of the sensory, cognitive and physical levels. The slow-down of sensory conduction speed and a decrease in the intensity of sensation translate into a partial awareness of the world around us. Cognitive decline impacts negatively on information processing which progressively takes longer. On top of that, motor abilities are compromised, and physical responses are delayed. The affective level, on the other hand, becomes a stronger component of users’ capabilities, compared to the other three levels. Gradually, it serves more and more as an aiding tool for decision making, prior to, and during user experience. However, unless a vigilant design process that addresses older adults’ requirements is in place, the consequences of sensory, cognitive and physical ageing result in a slow-paced, hesitant interaction and an unsatisfying user experience. User-centred design in healthcare should aim at realising the optimal embodiment of user requirements to deliver the best possible experience and encourage consistent adherence to health treatment or monitoring routines, especially in the home environment. The product has not only to be useful and usable; it also needs to be desirable in ways that transcend aesthetics. Its design has to communicate to users that the product matches their capabilities, meets their expectations, and provides obvious information about its utility. The ultimate goal is to help improve the physical and emotional aspects of older patients’ well-being by removing, as much as possible, any negative elements from the use process to facilitate an inviting, engaging and – why not – fulfilling experience. This work focuses on better understanding older adults’ needs, capabilities and attitudes, and emphasises the necessity to design for their inclusion by involving this user group in human factors activities throughout the medical device development process.
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Mathur, Akhil, and Fahim Kawsar. "Towards cognitive awareness." In UbiComp '17: The 2017 ACM International Joint Conference on Pervasive and Ubiquitous Computing. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3123024.3124565.

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Blackler, Alethea, Doug Mahar, and Vesna Popovic. "Older adults, interface experience and cognitive decline." In the 22nd Conference of the Computer-Human Interaction Special Interest Group of Australia. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1952222.1952257.

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Loukas, Vasileios S., Thomas Kassiotis, Ignacio Lamata Martinez, Lefteris Koumakis, Jeroen Bruinsma, Roberto Pasciuti, Monica Balatresi, et al. "LETHE: A Digital Intervention for Cognitive Decline*." In 2023 45th Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC). IEEE, 2023. http://dx.doi.org/10.1109/embc40787.2023.10340897.

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Alvarado-Contreras, Rocio E., Jessica Beltrán, Valeria Soto-Mendoza, Yohandra Herrera-Díaz, Gabriela Rodríguez-Ruiz, Nadia Cavazos-Lumbreras, and Mayra Mireles-Esquivel. "Towards a Cognitive Assessment Companion: Empowering Therapists in Evaluating Cognitive Decline." In 2023 Mexican International Conference on Computer Science (ENC). IEEE, 2023. http://dx.doi.org/10.1109/enc60556.2023.10508653.

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Fu, Xingguang, and Dirk Soeffker. "Cognitive Awareness of Intelligent Vehicles." In SAE 2010 World Congress & Exhibition. 400 Commonwealth Drive, Warrendale, PA, United States: SAE International, 2010. http://dx.doi.org/10.4271/2010-01-0465.

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France, Grace, Mark W. Orme, Neil J. Greening, Michael C. Steiner, and Sally J. Singh. "Cognitive decline following an acute exacerbation of COPD (AECOPD)." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3291.

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Reports on the topic "Awareness of cognitive decline"

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Ameriks, John, Andrew Caplin, Minjoon Lee, Matthew Shapiro, and Christopher Tonetti. Cognitive Decline, Limited Awareness, Imperfect Agency, and Financial Well-being. Cambridge, MA: National Bureau of Economic Research, January 2022. http://dx.doi.org/10.3386/w29634.

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Darden, Michael. Cognitive Decline and Dynamic Selection. Cambridge, MA: National Bureau of Economic Research, November 2022. http://dx.doi.org/10.3386/w30679.

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Tirre, William C., and Leo J. Gugerty. A Cognitive Correlates Analysis of Situation Awareness. Fort Belvoir, VA: Defense Technical Information Center, April 1999. http://dx.doi.org/10.21236/ada459961.

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Kane, M.D., Robert L., Mary Butler, Ph.D., M.B.A., and Howard A. Fink, M.D., M.P.H. Interventions To Prevent Age-Related Cognitive Decline, Mild Cognitive Impairment, and Clinical Alzheimer’s-Type Dementia. Agency for Healthcare Research and Quality (AHRQ), 2017. http://dx.doi.org/10.23970/ahrqepccer188.

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Fillipin, Federico, Pamela Seron, and Ruvistay Gutierrez-Arias. Effectiveness of antihypertensive drugs to prevent cognitive decline, mild cognitive impairment, and dementia. An overview of systematic reviews. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0057.

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Review question / Objective: To determine effectiveness of antihypertensive drugs to prevent different dementia subtypes such as Alzheimer’s disease and vascular dementia. Condition being studied: Dementia is a global health burden, with the number of affected individuals increasing. A recent meta-analysis reported that the prevalence of all-type dementia was 697 per 10,000 people and the prevalence of Alzheimer’s disease was 324 per 10,000 people. The SHEP and SYST-EUR were the two first randomized controlled trials to show that hypertension treatment reduces dementia risk.
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Fremouw, Thane, Maxwell Hennings, Christy Fessler, Hawk Cambron, Teresa Collins, and Moriah Greer. Chemotherapy, Neurotoxicity, and Cognitive Decline: Developing a Mouse Model and Potential Interventions. Fort Belvoir, VA: Defense Technical Information Center, September 2012. http://dx.doi.org/10.21236/ada568167.

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Fremouw, Thane. Chemotheraphy, Neurotoxicity, and Cognitive Decline: Developing a Mouse Model and Potential Interventions. Fort Belvoir, VA: Defense Technical Information Center, September 2011. http://dx.doi.org/10.21236/ada555413.

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Qin, wenjiao, and chunli chen. Risk factors for cognitive decline in elderly patients with type 2 diabetes. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2023. http://dx.doi.org/10.37766/inplasy2023.4.0056.

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Sureshbabu, Keertana, Egbe-Etu Etu, Susan Summerville, Ankur Parmar, and Gaojian Huang. Exploring the Use of Public Transportation Among Older Adults During the COVID-19 Pandemic: A National Survey. Mineta Transportation Institute, November 2022. http://dx.doi.org/10.31979/mti.2022.2204.

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Public transportation is an essential part of many older adults’ lives, but the pandemic presented new challenges for the vulnerable population. Adults aged 65 years and older experienced additional challenges, such as limited mobility options (e.g., lack of buses or trains in service due a combination of government lockdowns, fear of contracting or spreading the virus, and driver shortages in certain areas) because of the pandemic, which may have resulted in more age-related declines in perceptual, cognitive, and physical functioning. This study explores how older adults living in major metropolitan cities in the United States used and perceived public transportation during the COVID-19 pandemic. The research team conducted an online survey through the Amazon Mechanical Turk (MTurk) crowdsourcing marketplace, a platform that offers opportunities to recruit a larger number of participants from diverse geographic locations. 260 respondents completed the survey. Eligibility included: (1) residing in the United States, (2) being aged 55 years or older (the oldest age that can be selected on MTurk), and (3) having an approval rating of 90% or above (i.e., the percentage of the workers’ submitted tasks approved by survey requesters, offered by the MTurk platform). Overall, older adults reported that they had changed travel patterns since the onset of the COVID-19 pandemic, experienced challenges in using public transportation, and expressed concerns about catching the SARS-CoV-2 virus while using public transportation. Mobile technology (e.g., a transportation navigation app) was perceived as a good option for finding public transportation information, but needs improved user experience and accessibility. These findings may help transit agencies develop effective strategies for improving transportation services and increasing policymakers’ awareness of older adults’ need for accessible public transportation.
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Hu, XinYi, JingXuan Hao, and HangYue Wang. Improvement of Environmental enrichment on Cognitive Functions in Patients and animals : A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2022. http://dx.doi.org/10.37766/inplasy2022.12.0014.

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Review question / Objective: To study the relationship between environmental enrichment and cognitive function through a meta-analysis of the literature, analyze its effects on the improvement of cognitive function in patients and animals, and evaluate the effects of different environmental enrichment measures on cognitive function improvement. Condition being studied: Cognitive decline refers to an individual's memory, language, reasoning and other aspects of cognitive function showing obvious, measurable decline or abnormal. The causes of cognitive decline are various, including neurodegeneration, cerebrovascular disease, infection, trauma, and depression. Alzheimer's disease and stroke are the most common.
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