Academic literature on the topic 'Aviara'
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Journal articles on the topic "Aviara"
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Ubachukwu, N. N., and C. N. Emeribe. "The 2012 Flooding in Selected Parts of Isoko South, Delta State: Assessment of Socio-Economic Impacts." Mediterranean Journal of Social Sciences 8, no. 1 (January 26, 2017): 353–58. http://dx.doi.org/10.5901/mjss.2017.v8n1p353.
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Abstract The study investigated the socio-economic impacts of the 2012 flooding in the riverine areas Isoko south Local Government area, Delta State. Two communities Oleh and Aviara which were greatly affected by the flood event were selected for the study. Both purposive and simple random sampling techniques were adopted in the survey. A total of two hundred questionnaire were distributed to household heads, famers, community and religious leaders, one hundred per community. Impacts on building/household property, financial cost of damage, impact on small farm holders, damage on school infrastructures, diseases distributed were used as impact indicators. The study showed that displacement of family members for a period of 3-4months recorded the highest impact with Oleh and Aviara communities scoring impact level of 34% and 36.6% respectively. This was followed by submergence of farmland/lost of valuable household property, 27.7% in Oleh community and 23.6% Aviara community. Death/major injury during the flood event recorded least impact of 8.5% in Oleh and 3.2% in Aviara communities. On the average these impacts were valued at between one and two million naira (N). Analysis of impact on small farm holders showed that monthly income groups between <50,000 and 100,000 naira(N) were worst hit by the flood event. These groups are mostly subsistent farmers including snail farmers, poultry owners. The study revealed that schools were closed down for a period of 2-3months while collapsed of bridges was a leading cause of disruption in school activities with impact scores of 22.2% and 18.8% in Oleh and Avaira communities respectively. Analysis of disease distribution showed that malaria fever ranked highest, 33.1% in Oleh and 41.9% in Avaira respectively. This was followed by Diarrhoea, 22.6% in Oleh and Typhoid fever, 22.1% in Aviara. Bearing the losses was a common response approach to the flood disaster in the sampled communities with scores of 30.9% in Oleh and 27.6% in Aviara community. Structural modification ranked second with impact score of 16.5% in Oleh and migration to alternative location 16.3% in Avaira. Least response approach was construction of Monkey Bridge, 4.2% in Oleh and 4.1% in Avaira. Analysis of underlying cause of vulnerability showed that over reliance on government intervention thus doing little was main cause of vulnerability in Oleh, 25%, while in Avaira the main cause of vulnerability was attitude/cultural belief, 20.4%. The geographical locations of these communities also increase their vulnerability to flood by 22.9% in Oleh and 19.4% in Aviara. Result of student t test at 0.05 level of significance showed that the impact of the 2012 flooding in study area was similar in terms of damage to physical property, financial cost of impact, damage to school infrastructures and disease distribution. However, significant variation was observed in the two communities in terms of impacts on small farm holders. The study recommends land zoning in the framework of urban planning and regulation of urban development with a view to reducing the vulnerability of future flooding especially in the light recent global warming and climate change.
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Meghann B. Humphries and Robert E. Ricklefs. "Alpha-globin variation does not predict avian malaria infection in the West Indian Bananaquit (Coereba flaveola)." Journal of Caribbean Ornithology 36 (February 6, 2023): 1–16. http://dx.doi.org/10.55431/jco.2023.36.1-16.
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Abstract Infection by lineages of avian malaria parasites (Apicomplexa: Haemosporida) varies geographically, and some lineages exhibit disjunct distributions. These patterns might be related to differential resistance among host populations reflecting intrinsic characteristics of both the host and the pathogen. In hematophagous parasites in particular, the structure of host hemoglobin can influence parasite development and reproduction. Though variation in avian hemoglobin has been documented across altitudinal gradients, little is known about hemoglobin variation as it relates to infection by the parasites causing avian malaria. We sequenced the αA-globin subunit of the Bananaquit (Coereba flaveola), and related sequence variation to avian malaria infection frequency and parasite lineage identity. We found no association between αA-globin haplotype and infection by particular parasite lineages among all locations, nor any protective association between globin haplotype frequency and the proportion of individuals infected within populations. Phylogeographic structure and genetic variation at the αA-globin locus, including a highly variable intron, is largely concordant with the mitochondrial cytochrome b locus for these same populations, supporting this marker as an independent and variable target with potential application in biogeographic analyses.
Keywords alpha-globin, avian malaria, Bananaquit, Coereba flaveola, West Indies
Resumen La variación de la globina alfa no predice la infección por malaria aviar en Coereba flaveola • La infección por linajes de parásitos de la malaria aviar (Apicomplexa: Haemosporida) varía geográficamente y algunos linajes exhiben distribuciones discontinuas. Estos patrones podrían estar relacionados con la resistencia diferencial entre las poblaciones de huéspedes, y reflejan las características intrínsecas tanto del huésped como del patógeno. En los parásitos hematófagos, en particular, la estructura de la hemoglobina del huésped puede influir en el desarrollo y la reproducción del parásito. Aunque se ha documentado la variación de la hemoglobina aviar a través de gradientes altitudinales, se sabe poco sobre su variación en relación con la infección por los parásitos que causan la malaria aviar. Secuenciamos la subunidad de globina αA de Coereba flaveola y relacionamos la variación de la secuencia con la frecuencia de infección por malaria aviar y la identidad del linaje del parásito. No encontramos ninguna asociación entre el haplotipo de globina αA y la infección por determinados linajes de parásitos entre localidades; ni ninguna asociación protectora entre la frecuencia del haplotipo de globina y la proporción de individuos infectados dentro de las poblaciones. La estructura filogeográfica y la variación genética en el locus de la globina αA, que incluye un intrón muy variable, concuerdan en gran medida con el locus del citocromo b mitocondrial para estas mismas poblaciones; lo que respalda a este marcador como un objetivo independiente y variable con aplicación potencial en análisis biogeográficos.
Palabras clave Caribe Insular, Coereba flaveola, globina alfa, malaria aviar
Résumé La variation de l’alpha-globine ne permet pas de prédire l’infection par le paludisme aviaire chez le Sucrier à ventre jaune (Coereba flaveola) • L’infection par des lignées de parasites responsables du paludisme aviaire (Apicomplexa: Haemosporida) varie géographiquement, et certaines lignées présentent des répartitions disjointes. Ces schémas pourraient être liés à une résistance différentielle entre les populations d’hôtes reflétant des caractéristiques intrinsèques à la fois de l’hôte et du pathogène. Dans le cas particulier des parasites hématophages, la structure de l’hémoglobine de l’hôte peut influer sur le développe- ment et la reproduction du parasite. Bien que la variation de l’hémoglobine aviaire ait été documentée en fonction des gradients altitudinaux, peu de choses sont connues sur la variation de l’hémoglobine en relation avec l’infection par les parasites responsables du paludisme aviaire. Nous avons séquencé la sous-unité αA-globine du Sucrier à ventre jaune (Coereba flaveola), et nous avons mis en relation la variation de la séquence avec la fréquence d’infection du paludisme aviaire et l’identité de la lignée parasitaire. Nous n’avons trouvé aucune association entre l’haplotype αA-globine et l’infection par des lignées parasitaires particulières en fonction des sites, ni aucune association protectrice entre la fréquence de l’haplotype globine et la proportion d’individus infectés au sein des populations. La structure phylogéographique et la variation génétique au locus de l’αA-globine, y compris un intron hautement variable, est largement concordante avec le locus du cytochrome b mitochondrial pour ces mêmes populations, indiquant ce marqueur comme une cible indépendante et variable pouvant avoir une application potentielle dans les analyses biogéographiques.
Mots clés alpha-globine, Antilles, Coereba flaveola, paludisme aviaire, Sucrier à ventre jaune
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Content, Rob. "Aviary." Antioch Review 46, no. 4 (1988): 488. http://dx.doi.org/10.2307/4611957.
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Underwood, Susan O'Dell. "Aviary." Appalachian Heritage 31, no. 1 (2003): 89–91. http://dx.doi.org/10.1353/aph.2003.0020.
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Belcher, Philip. "Aviary." Appalachian Heritage 45, no. 2 (2017): 112. http://dx.doi.org/10.1353/aph.2017.0019.
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Huarcaya R., Freshia, Sonia Calle E., Juan Siuce M., André Sedano S., Jhonatan Huamaní P., Arturo García B., Luis Álvarez V., and Sofía Gonzales M. "Serotipificación y detección genética de Salmonella spp de origen aviar." Revista de Investigaciones Veterinarias del Perú 33, no. 3 (June 29, 2022): e22893. http://dx.doi.org/10.15381/rivep.v33i3.22893.
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El objetivo del presente estudio fue identificar molecular y serológicamente Salmonella spp presentes en aislados de huevos, canales y vísceras aviares. Se evaluaron 46 aislados de origen aviar identificados como Salmonella spp mediante cultivos y pruebas bioquímicas en el periodo 2012-2017, procedentes de varios distritos de la ciudad de Lima. Estos aislados son conservados en el cepario de Laboratorio de Bacteriología de la Universidad Nacional Mayor de San Marcos. Se reactivaron las cepas, y se reconfirmaron las colonias sospechosas a Salmonella spp mediante el cultivo en medios selectivos. Posteriormente se realizó el PCR a las muestras sospechosas como método de diagnóstico genético de Salmonella. Se detectó el gen de invasividad invA, gen involucrado con la virulencia de Salmonella. Se realizó la serotipificación con antisueros polivalentes y monovalentes para serogrupo y serovar en el Laboratorio de Enteropatógenos del Instituto Nacional de Salud (INS), y finalmente se determinaron las serovariedades de acuerdo con el esquema propuesto por Kauffmann-White. Las 46 cepas evidenciaron bandas de peso molecular correspondientes al gen invA (244 pb). Todas las cepas fueron identificadas mediante serotipificación, obteniendo: S. Infantis (34.8%), S. Pullorum (34.8%), S. Gallinarum (15.2%), S. Enteritidis (8.7%) y S. Typhimurium (6.5%). Los resultados revelan la predominancia de los serovares circulantes en muestras aviares, así como su implicancia en la salud pública.
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Maillé, Yves. "Tuberculinum aviare." Revista Médica de Homeopatía 4, no. 3 (September 2011): 111–15. http://dx.doi.org/10.1016/s1888-8526(11)70112-6.
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Pérez, Alberto A. "Influenza Aviar." El Hornero 29, no. 1 (August 1, 2014): 46–47. http://dx.doi.org/10.56178/eh.v29i1.626.
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Barrett, Paul. "The Mesozoic Aviary." American Scientist 95, no. 6 (2007): 531. http://dx.doi.org/10.1511/2007.68.531.
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Machado, Aditi. "Outside the Aviary." New England Review 33, no. 2 (2012): 6. http://dx.doi.org/10.1353/ner.2012.0041.
Full textDissertations / Theses on the topic "Aviara"
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Silveira, Flávio 1986. "Caracterização molecular de isolados brasileiros de Escherichia coli aviária." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317368.
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Orientador: Wanderley Dias da SilveiraDissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia
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Mestrado
Microbiologia
Mestre em Genética e Biologia Molecular
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Macacu, Alina. "Modélisation du risque Influenza Aviaire dans l'écosystème de la Dombes, France." Thesis, Grenoble, 2014. http://www.theses.fr/2014GRENS008/document.
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Influenza aviaire est une maladie infectieuse contagieuse, due à des virus de la famille des Orthomyxoviridae, les virus Influenza A, touchant les oiseaux et susceptible d'entraîner une mortalité extrêmement élevée, notamment chez les volailles. Les souches hautement pathogènes des virus influenza aviaire provoquent une maladie sévère et mortelle chez les volailles et constituent un risque pour la santé publique. En février 2006, la France a été touchée par une épizootie d'influenza aviaire H5N1 hautement pathogène, qui s'est répandu dans l'avifaune sauvage de la zone humide la Dombes. La Dombes est une zone humide, mosaïque de plus de mille étangs, et une zone d'importance ornithologique internationale, abritant des milliers d'oiseaux d'eau. C'est une zone à risque pour la propagation de l'influenza aviaire de part à la fois la présence des populations d'oiseaux sauvages sur les étangs et de la proximité de ces populations aux élevages de volailles qui parsèment la région. L'objectif principal de ce travail est la modélisation du risque influenza aviaire dans la région de la Dombes. Ce travail est axé sur l'avifaune sauvage et sur le risque d'infection et de transmission de l'infection par les oiseaux sauvages. Nous avons, dans un premier temps, identifié les caractéristiques particulières à la Dombes, pouvant influer sur l'épidémiologie de l'influenza aviaire dans la région. Les trois acteurs principaux de l'écosystème de la Dombes sont les étangs, les populations d'oiseaux sauvages et les élevages de volailles. Ensuite, le risque influenza aviaire dans la Dombes a été décliné à deux niveaux : 1) dans l'écosystème d'un seul étang ; et 2) au sein d'un réseau d'étangs comme caractérisés en 1), où la propagation de l'infection peut se faire d'étang à étang. Au niveau étang, le risque influenza aviaire est modélisé, d'une part, par le temps de persistance des virus influenza aviaire dans l'eau de l'étang, et d'autre part, par la dynamique de l'infection au sein des populations d'oiseaux sauvages par transmission directe entre oiseaux et indirecte via l'eau de l'étang. Au niveau réseau d'étangs, l'analyse du risque permet d'identifier des groupements spatiaux d'étangs à risque élevé d'infection aux virus influenza aviaire. Quant au risque d'exposition des élevages aux virus influenza aviaire d'origine de l'avifaune sauvage, il est calculé en tenant compte à la fois de la proximité des élevages aux étangs susceptibles d'être infectés et de la probabilité de ces étangs à être infectés en cas d'épizootieAvian influenza is a contagious infectious disease caused by viruses of the family Orthomyxoviridae, influenza A viruses, affecting birds and known to cause extremely high mortality, especially in poultry. Highly pathogenic strains of avian influenza viruses cause severe and fatal disease in poultry and pose a risk to public health. In February 2006, France was hit by a highly pathogenic H5N1 avian influenza outbreak, affecting aquatic wildfowl in Dombes wetland. The Dombes is a wetland mosaic of more than a thousand ponds and an area of international ornithological importance, home to thousands of waterfowl. This is an area at risk for the spread of avian influenza viruses due to both the presence of wild birds on the ponds and the proximity of these populations to poultry farms that are present in the area. The main objective of this work is the modeling of the avian influenza risk in the Dombes region. This work is focused on the wildfowl and the risk of infection and transmission of infection by wild birds. As a first step, we have identified the specific characteristics of the Dombes area, which may affect the epidemiology of avian influenza in the region. The three main players in the Dombes ecosystem are the ponds, the wildfowl populations and the poultry farms. Secondly, the avian influenza risk in the Dombes was declined at two levels: 1) in the ecosystem of a single pond; and 2) within a network of ponds as characterized by 1), where the infection is spread from pond to pond. At the pond level, the avian influenza risk is modeled, on the one hand, by the persistence time of avian influenza viruses in the water of the pond, and, on the other hand, by the infection dynamics within wild bird populations by direct transmission between birds and indirectly through the water of the pond. At the ponds network, the risk analysis identifies spatial clusters of ponds at high risk of infection with avian influenza viruses. Concerning the risk of exposure of poultry farms to avian influenza viruses of wildfowl origin, it is calculated taking into account both the proximity of the farms to ponds likely to be infected and the probability of these ponds to become infected during an outbreak
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Quel, Natália Galdi. "Escherichia coli Vacuolating Factor (ECVF) como fator associado a celulite aviária." Universidade de São Paulo, 2014. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-27062014-171721/.
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E. coli isoladas de lesões de celulite aviária em frangos de corte produzem uma citotoxina, denominada ECVF (E. coli Vacuolating Factor), que causa intensa vacuolização citoplasmática em células aviárias, mas não em células mamárias. A importância de ECVF na patogenia da celulite foi avaliada neste estudo. ECVF purificado foi inoculado subcutaneamente em frangos de corte, e induziu sinais de inflamação nos tecidos subcutâneo, adiposo e conjuntivo. Em ensaios de citotoxicidade, foi verificado que ECVF induz alterações citoplasmáticas e nucleares que podem afetar diretamente o metabolismo celular, entre elas condensação da cromatina e fragmentação nuclear, intensa vacuolização citoplasmática e desorganização do citoesqueleto, conduzindo à apoptose. Também foi verificada interação de ECVF com proteínas de células aviárias, em detrimento das de células de mamíferos, sugerindo uma especificidade da toxina a este tipo celular. Nossos resultados, apoiados por dados de estudos anteriores, permitem sugerir um importante papel de ECVF na patogenia da celulite aviária.E. coli isolated from cellulitis lesions in broiler chickens produce a citotoxin, called ECVF (Escherichia coli Vacuolating Factor), which causes intense cytoplasm vacuolization in avian cells, but not in mammalian cells. The importance of ECVF in the pathogenesis of avian cellulitis was assessed in this study. Purified ECVF was inoculated subcutaneously in broiler chickens, and induced signs of inflammation on subcutaneous, adipose and connective tissues. In citotoxicity assays, we verified that ECVF induced cytoplasmic and nuclear alterations, which can affect cellular metabolism directly, such as chromatin condensation and nuclear fragmentation, intense cytoplasm vacuolization, and disorganization of cytoskeleton, leading to apoptosis. It was also verified the interaction of ECVF with proteins of avian cells, instead of those from mammalian cells, suggesting the specificity of this toxin to this cells. Our results, supported by data from previous studies, suggest an important role of ECVF in the pathogenesis of avian cellulitis.
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Borzi, Mariana Monezi [UNESP]. "Produção da nucleoproteína recombinante do vírus da influenza aviária para aplicação no imunodiagnóstico." Universidade Estadual Paulista (UNESP), 2015. http://hdl.handle.net/11449/128008.
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000849126.pdf: 1214857 bytes, checksum: a2d47dd516d137071a1aa19d9f8af738 (MD5)A nucleoproteína (NP) do Vírus da Influenza Aviária (VIA) é um importante alvo antigênico no imunodiagnóstico desta doença, devido à sua baixa variabilidade entre as diferentes estirpes do VIA, resultando em uma elevada reatividade cruzada, e por ser também uma proteína altamente imunogênica para hospedeiros vertebrados. Neste estudo, o gene codificador da NP do VIA foi parcialmente clonado e expresso em Escherichia coli como uma proteína recombinante fusionada ao polipeptídeo SUMO e uma etiqueta de poli-histidina para seu uso no desenvolvimento de um ensaio de ELISA indireto para a detecção de anticorpos específicos contra o VIA. A NP recombinante foi expressada na fração solúvel e foi mais facilmente purificada. Após análise em relação aos seus principais sítios de antigenicidade e caracterização por meio de Western blotting, a NP recombinante foi utilizada como uma preparação antigênica no ELISA indireto para detecção de anticorpos contra o VIA presentes em amostras de soro de galinha. A análise comparativa do teste desenvolvido no presente estudo com um ELISA comercial apresentou valores de 95%, 97% e 96,7% de sensibilidade, especificidade e acurácia, respectivamente e um índice κappa de 0,88. Os resultados permitem concluir que a NP recombinante do VIA desenvolvida neste estudo possui características favoráveis para ser aplicada como antígeno no ELISA indireto, constituindo-se em um método sensível e específico para o imunodiagnóstico da Influenza Aviária em galinhas
The nucleoprotein (NP) of Avian Influenza Virus (AIV) is an important antigenic target for immunodiagnosis of this disease, due to its low variability among different AIV strains, resulting in high cross-reactivity, and the also highly immunogenic for vertebrate hosts. In this study, the gene enconding NP of AIV was cloned and expressed in Escherichia coli as a recombinant protein fused to SUMO polypeptide with a polyhistidine tag and used to develop an indirect ELISA for the detection of AIV-specific antibodies. The recombinant NP was expressed in the soluble fraction and easily purified. After Analysis of the main sites of antigenicity and characterization in Western-Blotting, the recombinant NP was optimized as an antigen preparation for indirect ELISA to detect anti-AIV antibodies in chicken serum samples. The comparative analysis of this ELISA with a commercial ELISA showed values of 95%, 97%, 96.7% of sensitivity, specificity and accuracy, respectively, and an agreement of k=0.88. In conclusion, the results indicated that the recombinant NP of AIV produced in this study is a good source of antigen for indirect ELISA and provides a sensitive and specific method for the immunodiagnosis of Avian Influenza in chickens
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Maturana, Victor Gonçalves. "Subpatótipos de Escherichia coli patogênica para aves (APEC) podem estar associados às síndromes infecciosas infecciosas causadas no hospedeiro." [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317393.
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Orientadores: Wanderley Dias da Silveira, Marcelo Palma SirciliDissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Biologia
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Resumo: Escherichia coli patogênica para aves (APEC) causa diferentes tipos de infecções sistêmicas extraintestinais nestes hospedeiros, coletivamente denominadas colibaciloses, causando grandes prejuízos econômicos à indústria aviária. Essas doenças incluem, dentre outras, septicemia, onfalite, celulite e síndrome da cabeça inchada. Entretanto, não há até o momento wna descrição de genes ou características que permitam classificar as linhagens aviárias em patótipos responsáveis por causar doenças específicas em seus hospedeiros, a semelhança do que ocorre para linhagens de E. co/i patogênicas para seres humanos. O objetivo deste estudo foi caracterizar linhagens de Escherichia coli de origem aviána representantes de 4 grupos, sendo um grupo de linhagens comensais (AFEC - stgla em inglês para "Avian Fecal Escherichia co/i") e três grupos de linhagens patogêmcas, causadoras de três sindromes diferentes em seus hospedeiros (septicemia, síndrome da cabeça inchada e onfalite). Para o trabalho, as características biológicas estudadas foram: adesão em células eucarióticas, formação de biofilme, produção de molécula sinalizadora de quorum sensing, presença de genes de ilhas de patogenicidade, dose de letalidade (LD50), grupo filogenético e presença de genes de virulência. A comparação entre as diferentes linhagens com base nestes traços genotípicos e fenotípicos, por meio de diferentes ferramentas de estatística multivariada além de redes complexas, permitiu inferir a estrutura populacional do grupo estudado. Os resultados indicam que APEC não constitui um grupo homogêneo, mas um conjunto estruturado de diferentes subgrupos, cada um associado a uma síndrome infecciosa específica causada no hospedeiro, possivelmente definindo diferentes patótipos ou subpatótipos dentro de linhagens APEC. Assim, sugerimos a existência de um subpatótipo associado à onfalite, com características de letal idade semelhantes as de linhagens AFEC, mas com um padrão de adesão diferente, que pode ser atribuído a uma especialização do grupo relacionada a colonização de um nicho particular, o saco da gema do ovo. E um subpatótipo associado à síndrome da cabeça inchada, igualmente adaptado à patogenicidade, mas com características mais "agressivas" evidenciadas pelos altos índices de letalidade, grande número de genes de virulência e altos índices de adesão. Linhagens associadas à septicemia, contudo, não constituem um grupo coeso, sugerindo tratar-se de uma miscelânea de linhagens que podem pertencer a diferentes subpatótipos e que em última instância geram a síndrome sistêmica (septicemia), devido à evolução do quadro clínico e/ou às condições imunológicas do hospedeiro. Este trabalho é pioneiro em demonstrar a existência de subpatótipos dentro de linhagens APEC, relacionando diferentes síndromes infecciosas com grupos específicos de linhagens as quais possuem características fenotípicas e genotípicas particulares. Tais resultados abrem novas possibilidades no estudo de genes responsáveis pelos diferentes processos de patogênese em APEC, bem como no desenvolvimento de vacinas. Talvez seja importante considerar estes subgrupos no desenvolvimento de vacinas, com o intuito de produzir vacinas com proteção cruzada, o que ainda não foi atingido com sucesso para linhagens APEC.
Abstract: Avian Pathogenic Escherichia coli (APEC) cause different types of systemic extraintestinal infections in poultry, which are collectively termed colibacillosis, imposing significant economic casses for the avian industry Among these diseases are septicaemia, omphalitis, cellulites and swollen head syndrome. However, to the date, there is no description of genes or characteristics which allow us to classify avian strains in pathotypes responsible for causing specific diseases in their hosts, as there are for human pathogenic E. co/i strains. In this study we aimed to characterize avian pathogenic E. co/i strains representing 4 groups, one of commensal strains (AFEC - Avian Fecal Escherichia co/i) and 3 groups of pathogenic strains responsible for causing 3 different syndromes in their hosts (septicaemia, omphalitis and swollen head syndrome). The biological characteristics studied were: adhesion to eukaryotic cells, biofilm formation, capacity of synthesizing quorum sensing s1gnaling molecule, presence of pathogenicity island, pathogenicity levels according to lethal dose (50%) assay, phylogenetic group and presence of virulence genes. The comparison between strains based on these genotypic and phenotypic traits, by different multivariate statistics tools and complex network, allowed us to infer. the population structure of the studied group. The results indicate that APEC do not constitute a unique homogeneous group, but a structured set of different subgroups, each one associated to a specific infectious syndrome inflicted to the host, possibly defining pathotypes or subpathotypes within APEC strains. Thus, we suggest the existence of a subpathotype associated to omphalitis, with lethality characteristics similar to AFEC strains, but with a different adhesion pattem, which may be due to a specialization related to the colonization of a particular niche, the egg's yolk sac. Anda subpathotype associated to the swollen head syndrome, equally adapted to pathogenicity, but with more "aggressive" characteristics demonstrated by the high lethality and adhesion levels. Septicaemic strains, however, do not constitute a cohesive group, which suggests a constellation of strains associated to different subpathotypes and capable of, eventually, cause a systemic syndrome (sepsis), due to the clinical evolution of the illness ami/or host immunological conditions. This work is pioneer in demonstrating the existence of subpathotypes within APEC strains, relating different infectious syndromes to specific groups of strains possessing particular genotypic and phenotypic characteristics. These results offer new possibilities in studying the genes responsible for different pathogenesis processes within APEC and for the vaccine developing. It may be important to consider these subgroups in the process of vaccine developing, in the efforts for obtain cross protection, which had not yet being accomplished successfully concerning APEC strains.
Mestrado
Genetica de Microorganismos
Mestre em Genética e Biologia Molecular
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Rojas, Thaís Cabrera Galvão 1980. "Detecção de genes sob seleção positiva em linhagens de Escherichia coli patogênicas para aves (APEC) e para humanos." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317369.
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Orientador: Wanderley Dias da SilveiraTese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
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Resumo: A bactéria Escherichia coli coloniza o trato intestinal de aves e humanos, de maneira comensal sem causar processos infecciosos. No entanto alguns clones adquiriram fatores de virulência específicos, permitindo o desenvolvimento de diferentes doenças como infecção do trato urinário, diarréia e meningite em humanos e colibacilose em aves. As linhagens que causam doença em aves são tipicamente denominadas APEC (Avian Pathogenic Escherichia coli). Neste trabalho foram sequenciados e anotados os genomas de quatro linhagens APECs (SCI-07, SEPT362, S17 e O8)que, juntamente com mais nove genomas referentes a linhagens de Escherichia coli patogênicas para aves e patogênicas para humanos foram utilizados para a busca de genes sob seleção positiva. Os genes homólogos foram agrupados,e posteriormente submetidos ao alinhamento de códons e das sequencias protéicas correspondentes. Uma árvore filogenética foi gerada para cada grupo de proteínas homólogas. Testes estatísticos determinaram qual entre os modelos de seleção neutra ou seleção positiva melhor explicou os dados existentes (alinhamentos de códons e árvores filogenéticas). Essas análises detectaram duzentas e cinquenta e quatro grupos de genes homólogos com evidência de seleção positiva. Para cada grupo foi realizado um teste de recombinação para verificar se o aumento na variação das sequencias não era devido à conversão gênica, resultando em cento e dezesseis grupos de genes homólogos sob seleção positiva. A proteína correspondente a um gene de cada grupo de genes homólogos foi identificada, por meio da ferramenta Blast. Diversos fatores de virulência, já conhecidos, e proteínas regulatórias puderem ser detectados. Os genes sob seleção positiva, também foram submetidos à anotação considerando o termo GO (Gene Ontology),apenas da categoria processo biológico. Dos cento e dezesseis genes apenas cinquenta e sete puderam ser identificados por meio dessa metodologia. O resultado da classificação dos genes dentro da classe GO, considerando o terceiro nível hierárquico,mostrou que a maioria dos genes anotados (31) tinha relação com o metabolismo primário.As proteínas cuja identificação, por meio do blast, não foi possível (proteínas hipotéticas)foram submetidas à análise de predição de localização subcelular e de peptídeo sinal. Essas análises revelaram que três proteínas desconhecidas (hypothetical proteinECIAI39_1028, hypothetical proteinZ0639e hypothetical proteinEC042_3791) são potenciais alvos para estudos que visam à busca de novos fatores de virulência de Escherichia coli patogênicas
Abstract: The bacterium Escherichia coli colonizesthe intestinal tract of birds and humans, in a commensal relationship without causing infection. However, some clones have acquired specific virulence factors allowing the development of various diseases such as urinary tract infection, diarrhea and meningitis in humans and colibacillosis in poultry. The strains that cause disease in birds are typically named APEC (Avian Pathogenic Escherichia coli). In this study we sequenced and annotated the genomes of four APECs strains (SCI-07, SEPT362, S17 and O8). These genomes and nine others avian pathogenic Escherichia coli and humans pathogenic strains genomes were used for studying genes under positive selection. The homologous genes were grouped and then subjected to codons and corresponding protein sequences alignment. A phylogenetic tree was generated for each group of homologous proteins. Statistical tests determined which among neutral or positive selection models best explains the existing data (codon alignments and phylogenetic trees). This analyzes detected two hundred fifty-four groups of homologous genes with positive selection evidence. For each group a recombination test was conducted to verify if the variation increase in the sequences was not due to gene conversion, resulting in one hundred and sixteen groups of homologous genes under positive selection. The protein corresponding to a gene of each group of homologous genes under positive selection was identified through Blast tool. Genes under positive selection were annotated considering the GO term (Gene Ontology), just for the biological process category. Only fifty-seven genes could be identified using this methodology. The gene classification within the GO classes, considering only the third hierarchical level showed that most of the annotated genes (31) were related with the primary metabolism. Proteins which blast identification was not possible (hypothetical proteins) were subjected to sub cellular localization and signal peptide prediction analyzes. These analyzes revealed that three unknown proteins (hypothetical protein ECIAI39_1028, hypothetical protein Z0639e hypothetical protein EC042_3791) are potential targets for studies, in order to search for new virulence factors of pathogenic Escherichia coli
Doutorado
Microbiologia
Doutora em Genética e Biologia Molecular
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Scanavini, Luciana Simeoni. "Desenvolvimento de uma reação em cadeia pela polimerase para a detecção de coronavírus de perus (TCOV), por meio de um fragmento da região S2 de seu genoma." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-02122013-160012/.
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A partir de amostras de campo de diferentes regiões do país, o TCoV foi identificado em aves de 13-84 dias, em órgãos como intestino, rins e traqueia, por uma reação em cadeia pela polimerase para a região 3\' UTR e o gene 3. A presença deste coronavírus nesses diferentes órgãos, que não o intestino, sugere a presença de uma diferente estirpe, com características fenotípicas mais próximas em relação ao vírus da Bronquite Infecciosa das Galinhas (BIG), o IBV. Ao mesmo tempo, um perfil considerando a idade das aves positivas foi desenhado por um teste T de duas amostras, que mostrou maior positividade em aves mais jovens. Com a finalidade de desenvolver um novo ensaio de PCR, várias sequências do TCoV disponíveis no GenBank foram alinhadas, um segmento gênico foi desenhado e submetido a diferentes condições de reação. Um fragmento de 436 pb foi obtido, sequenciado e apresentou 100% de identidade quando sua sequência foi comparada a sequência completa do TCoV. A reação mostrou boa repetibilidade, bem como boa reprodutibilidade, e algumas amostras de campo não se apresentaram positivas.TCOV was identified from field samples of birds with 13-84 days, from different regions of the country, and from organs such as intestine, kidneys and trachea, by polymerase chain reaction for 3\' UTR region and gene 3. The presence of this coronavirus in these different organs, not intestine, suggest a different strain, with similar phenotype observed in Infectious Bronchitis Virus (IBV). At the same time, a profile considering age of positive birds was designed by a Two sample T-test, which showed higher positivity in younger poults. Several TCOV sequences obtained from GenBank were aligned, and a genic segment was designed and submitted to different conditions in reaction in pursuit to develop a new PCR assay. A 436bp fragment was obtained, sequenced and presented 100% ID when compared to a TCOV complete sequence. The reaction featured good repetition, and reproduction as well, and some field samples did not turn out on positive results.
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Matter, Leticia Beatriz. "Interação entre Escherichia coli patogência aviária (APEC) e células não fagocitárias." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2011. http://hdl.handle.net/10183/60558.
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Neste trabalho foi estuda a interação da E. coli patogênica aviária (APEC), agente etiológico da colibacilose aviária, e células não fagocitárias. A APEC é uma ExPEC (E. coli extra-intestinais), grupo que também inclui a UPEC (E. coli uropatogênica) e a NMEC (E. coli de meningite neonatal). Foi analisado o comportamento de 8 cepas APEC - MT78, IMT2470, A2363, UEL31, UEL13, UEL17, IMT5155, UEL29 - frente a duas linhagens de células não-fagocitárias, fibroblastos aviários CEC-32 e células endoteliais humanas EAhy926. Foi realizada a genotipagem de 33 genes associados à virulência, verificou-se capacidade de associação (adesão e invasão), de invasão e de multiplicação intracelular, de citotoxicidade e de ativação das caspases 3/7 das cepas após infecção de fibroblastos aviários. Foi observado que enquanto todas as cepas foram capazes de aderir aos fibroblastos aviários, somente a cepa MT78 foi capaz de invadí-los em níveis comparáveis à bactéria invasiva Salmonella Typhymurium SL1344. As cepas APEC não induziram ativação de capases 3/7, nem foram citotóxicas aos fibroblastos. Uma vez que a cepa invasiva, MT78, e a não invasiva, IMT2470, apresentam genótipos de virulência muito similares, foi realizado o estudo da expressão por RT-PCR dos genes de virulência da bactéria crescida com e sem fibroblastos aviários, por 3 h. Os resultados mostraram a expressão de adesinas, sideróforos e protectinas/estruturas de resistência ao soro para ambas as cepas na ausência e presença de fibroblastos. A análise da expressão dos genes fimH, ompA, ibeA e gimB pela técnica RT-qPCR revelou a repressão do gene fimH na MT78, mas indução do mesmo na IMT2470. Já as invasinas, gimB e ibeA, estavam induzidas em MT78 e reprimidas em IMT2470, enquanto o gene ompA estava sendo expresso em ambas as cepas. A expressão das invasinas gimB e ibeA explica em parte o fenótipo invasivo da MT78. No presente estudo também foi analisado o comportamento das 8 cepas APEC em relação ao perfil de associação, invasão e multiplicação intracelular ao infectarem células EAhy926. As cepas não mostraram capacidade de invadir as células mas apresentaram um nível de associação muito superior ao dos fibroblastos aviários (até 14 vezes superior para algumas cepas). Este trabalho mostrou que o ensaio in vitro com CEC-32 é um modelo adequado para o estudo da interação celular entre APEC e células eucarióticas e agregou mais conhecimento sobre o patotipo.In this work the interaction between avian pathogenic E. coli (APEC), the etiological agent of avian colibacillosis, and non-phagocytic cells was studied. APEC is an ExPEC (extraintestinal E. coli), a group that also includes UPEC (uropathogenic E. coli) and NMEC (E. coli neonatal meningitis). We analysed the behavior of 8 APEC strains - MT78, IMT2470, A2363, UEL31, UEL13, UEL17, IMT5155, UEL29 - against two non-phagocytic cell lines, avian fibroblasts (CEC-32) and human endothelial cells (Eahy926). Strains were genotyped for 33 virulence associated genes, and investigated the association capacity (adhesion and invasion), the invasion ability, intracellular multiplication, cytotoxicity and activation of caspases 3/7 of the strains after infecting avian fibroblasts. While all strains were able to adhere to avian fibroblasts, only the strain MT78 was able to invade them at levels comparable to the invasive bacterium Salmonella Typhymurium SL1344. APEC strains could not induce activation of caspases 3/7, nor were cytotoxic to fibroblasts. Since the invasive MT78 and the non-invasive IMT2470 strains presented very similar virulence genotypes, the expression of virulence genes of the bacteria grown in the absence and presence of avian fibroblasts by 3 h was analysed by RT-PCR. Results showed the expression of adhesins, siderophores and protectins/serum resistance structures for both strains in the two culture conditions, with and without fibroblasts. Analysis of the expression of fimH, ompA, ibeA and gimB by RT-qPCR revealed the repression of fimH in MT78, but the induction in IMT2470. In relation to ibeA and gimB invasins, they were induced in MT78 but repressed in IMT2470, while the ompA gene was expressed in both strains. The expression of invasins partly explains the invasive phenotype of MT78. It was also analysed the behaviour of the strains in relation to the association profile, invasion and intracellular multiplication when infecting EAhy926 human endothelial cells. The strains showed no ability to invade endothelial cells but showed a high level of association (up to 14 times higher than for fibroblasts cells for some strains). This study showed that the CEC-32 in vitro model is suitable for the study of cellular interaction between APEC and eukaryotic cells, and added more knowledge about the pathotype.
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Zélé, Flore. "Interaction entre la bactérie endosymbiotique Wolbachia et le parasite responsable de la malaria aviaire, Plasmodium relictum, chez le moustique Culex pipiens." Thesis, Montpellier 2, 2012. http://www.theses.fr/2012MON20102.
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Les interactions hôtes-parasites sont classiquement vues comme un équilibre dynamique entre seulement deux partenaires ; cependant, ce paradigme a progressivement changé ces dernières années avec la découverte de nombreux hôtes naturellement co-infectés par de multiples parasites. L'étude des co-infections s'avère particulièrement pertinente lorsque l'hôte est un vecteur de maladies, dans la mesure où les infections multiples peuvent avoir d'importantes conséquences sur la transmission d'un parasite, à la fois à des échelles écologiques et évolutives. Wolbachia pipientis est la bactérie endosymbiotique la plus commune chez les insectes, et de fait, suscite un intérêt particulier pour comprendre le rôle des co-infections sur l'issue des infections parasitaires. Afin de déterminer si l'infection naturelle par Wolbachia affecte la capacité des moustiques à transmettre la malaria, nous avons étudié une triade qui partage une histoire évolutive commune : le parasite de la malaria aviaire P. relictum SGS1, son vecteur naturel Cx. pipiens, et un ensemble de souches wPip de Wolbachia naturellement présentes chez ce moustique. Nous nous sommes tout d'abord intéressé à l'impact de différents types de wPip sur la prévalence et la diversité des parasites responsables de la malaria aviaires dans les populations naturelles de Cx. pipiens de la région de Montpellier. Puis, par l'utilisation de différentes lignées isogéniques de moustiques, infectés ou non par Wolbachia, nous avons étudiés l'impact de la présence du symbiote sur différents traits d'histoire de vie du moustique essentiels pour la transmission de Plasmodium. Nous montrons ainsi que Wolbachia profite à Cx. pipiens, mais également à Plasmodium: elle améliore plusieurs traits d'histoire de vie des moustiques, tels que leur longévité et leur fécondité, leur tolérance à l'infection par P. relictum, et facilite l'infection par ce parasite à la fois qualitativement et quantitativement (i.e. elle augmente à la fois la prévalence et l'intensité de l'infection). Bien que les mécanismes impliqués dans cette interaction tripartite restent encore inconnus, ces résultats suggèrent que Wolbachia peut avoir d'importantes implications sur la transmission de la malaria dans la nature. De plus, ces résultats suggèrent la nécessité de réévaluer l'utilisation de Wolbachia comme moyen de lutte contre les pathogènes et soulignent la nécessité de mieux comprendre les interactions multipartitesIn recent years, there has been a shift in the one host one parasite paradigm with the realization that, in the field, most hosts are co-infected with multiple parasites. Coinfections are particularly relevant when the host is a vector of diseases, because multiple infections can have drastic consequences for parasite transmission at both the ecological and evolutionary time scales. Wolbachia pipientis is the most common parasitic microorganism in insects and as such it is of special interest for understanding the role of coinfections in the outcome of parasite infections. This thesis investigates whether a natural Wolbachia infection can alter the quality of mosquitoes as vectors of malaria. To address this issue, we used a Wolbachia-mosquito-Plasmodium triad with a common evolutionary history. Our experimental system consists in the avian malaria parasite P. relictum SGS1 and its natural vector, the mosquito Cx. pipiens, which naturally harbours several wPip Wolbachia strains. First, we investigated the impact of different wPip groups on the prevalence and diversity on avian malaria in natural populations of Cx. pipiens mosquitoes in the Montpellier region. Second, using different isogenic laboratory mosquito strains harboring or not Wolbachia, we investigated the impact of the presence of Wolbachia on several mosquito and Plasmodium life history traits relevant for malaria transmission. We show that Wolbachia benefits both Cx. pipiens and Plasmodium: it enhances several mosquito life history traits, such as longevity and fecundity, increases their tolerance to P. relictum (i.e. compensates for a Plasmodium-induced mortality) and facilitates P. relictum infection both qualitatively (increases infection prevalence) and quantitatively (increases infection intensity). Although the mechanisms involved in the mosquito-Wolbachia-Plasmodium interaction remain elusive, these results suggest that Wolbachia may have important implications on the transmission of malaria in nature. This is consistent with the high prevalence and diversity of avian malaria parasites found in natural populations of Cx. pipiens. Further, these results suggest the need to reassess the use of Wolbachia as a way to fight pathogens and highlight the need to better understand parasite multipartite interactions
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Cappelle, Julien. "Evaluation éco-épidémiologique du risque d'émergence du virus Influenza Aviaire Hautement Pathogène H5N1 dans le Delta Intérieur du Niger au Mali via l'avifaune sauvage." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210000.
Full textAbstract:
Cette thèse évalue le risque d’émergence d’un pathogène via l’avifaune sauvage dans une région indemne en combinant deux approches :(1) L’étude de pathogènes partageant des caractéristiques éco-épidémiologiques communes avec le pathogène émergeant ;et (2) L’utilisation de données écologiques disponibles dans la région indemne.\
Doctorat en Sciences agronomiques et ingénierie biologique
info:eu-repo/semantics/nonPublished
Books on the topic "Aviara"
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Cornel, Udrea, ed. Domnișoara aviara: Antologie de proză umoristică românească. Cluj-Napoca: Grinta, 2007.
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Peutz, Mike. Introducing Aviary. New York: Friends of ED, 2009.
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Peutz, Mike. Introducing Aviary. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7.
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Hall, Gaston. Alphabet aviary. Kenilworth: Ritchie, 1986.
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O'Dell, Kathleen. The aviary. New York: Alfred A. Knopf, 2011.
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ill, Doty Andrea, ed. The children's aviary. West Hartford, Conn: Frog Books, 1998.
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Conn, Stewart. At the aviary. Plumstead, South Africa: Snailpress, 1995.
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Gillian, Cummings. My dim aviary. [United States]: Black Lawrence Press, 2016.
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Hickman, Katie. The aviary gate. Bath: Windsor, 2008.
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Kelsey, Karla. Knowledge, forms, the aviary. Boise: Ahsahta Press, Boise State University, 2006.
Find full textBook chapters on the topic "Aviara"
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Peutz, Mike. "Welcome to the Aviary." In Introducing Aviary, 1–7. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_1.
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Peutz, Mike. "Getting to Know Aviary.com." In Introducing Aviary, 555–89. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_10.
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Peutz, Mike. "Creating Images with Phoenix." In Introducing Aviary, 9–77. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_2.
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Peutz, Mike. "Image Manipulation with Phoenix." In Introducing Aviary, 79–151. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_3.
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Peutz, Mike. "Raven—Vector Editing." In Introducing Aviary, 153–264. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_4.
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Peutz, Mike. "Creating Images with Peacock." In Introducing Aviary, 265–358. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_5.
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Peutz, Mike. "Creating Textures and Tiles in Peacock." In Introducing Aviary, 359–444. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_6.
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Peutz, Mike. "Selecting and Managing Colors with Toucan." In Introducing Aviary, 445–74. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_7.
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Peutz, Mike. "Screen Capture and Markup with Talon and Falcon." In Introducing Aviary, 475–504. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_8.
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Peutz, Mike. "Application Switching." In Introducing Aviary, 505–53. Berkeley, CA: Apress, 2009. http://dx.doi.org/10.1007/978-1-4302-7200-7_9.
Full textConference papers on the topic "Aviara"
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DeLand, Trevor S., Ross E. Dudgeon, Michael W. Orth, Darrin M. Karcher, and Roger C. Haut. "Effect of Housing System on Properties of Pullet Bones." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80631.
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Recently, attention has been brought to the welfare of laying hens and the benefits provided by progressive housing systems [3]. Conventional battery cage (CC) systems provide each bird with access to feed and water at all times with room to move. One of the new housing types being implemented, referred to as a cage-free aviary system (AV), is much larger than conventional cages and houses more birds. Aviary systems comprise multiple levels providing more opportunity for movement and exercise. Hens can forage and dust bathe in an open communal area, have access to perches, and nest boxes. However, for mature hens to fully utilize an AV, pullets (pre-egg production birds) must be reared in a similar environment.
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Arruda, Rafaela Oliveira de, and LUANA HELEN GONÇALVES SLAUTA. "CLAMIDIOSE AVIÁRIA." In II Congresso Brasileiro On-line de Clínica Médica Veterinária. Revista Multidisciplinar em Saúde, 2023. http://dx.doi.org/10.51161/convet2023/24812.
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AGUIAR, RAYANE OLIVEIRA PASCHOAL, and SHIHANE MOHAMAD COSTA MENDES. "Influenza aviária." In Congresso Iberoamericano de Saúde Pública Veterinária - 5ª Edição. Congresse.me, 2023. http://dx.doi.org/10.54265/beao9608.
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Fattal, Laura. "Aviary Aspirations: Modeling Dance and Science Pedagogies." In AERA 2022. USA: AERA, 2022. http://dx.doi.org/10.3102/ip.22.1880276.
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Fattal, Laura. "Aviary Aspirations: Modeling Dance and Science Pedagogies." In 2022 AERA Annual Meeting. Washington DC: AERA, 2022. http://dx.doi.org/10.3102/1880276.
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Norton, Thomas T. "Experimental myopia and emmetropization in mammals." In OSA Annual Meeting. Washington, D.C.: Optica Publishing Group, 1991. http://dx.doi.org/10.1364/oam.1991.tuy3.
Full textAbstract:
Studies on developing mammalian as well as avian eyes have suggested the existence of an emmetropization process that matches axial length to focal length. A vision-dependent signal may slow axial elongation to hold the retina in the focal plane: deprivation of clear images on the retina produces an elongated, myopic eye in humans, most monkeys, and in tree shrews. If deprivation is removed in young, experimentally myopic tree shrews, recovery can occur due partially to slowing of axial expansion relative to the continued increase in focal length of the developing eye. In at least some mammals, it seems that a retinal signal is transmitted directly to the choroid and/or sclera; deprivation-induced myopia can develop despite blocking ganglion cell output to central brain structures. In tree shrews, as in avians, this signal seems local: deprivation in part of the visual field produces elongation and myopia selectively in the deprived region of the eye. Although accommodation may participate in normal ocular development, experimental myopia can develop in tree shrews despite atropine sulfate administration. In mammals it is still unclear whether the emmetropization process controls ocular growth, stretch, or both. How emmetropization is disrupted in human myopia also remains uncertain.
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Gockley, Rachel, Michael Marotta, Carin Rogoff, and Adrian Tang. "AVIVA." In CHI '06 extended abstracts. New York, New York, USA: ACM Press, 2006. http://dx.doi.org/10.1145/1125451.1125796.
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MARTINEZ, CRISTINA, MARIA TRIGUEZ, and NURIA ZAMORA. "CELIACA SIN AVISAR." In 37 Congreso Nacional de la Sociedad Española de Pediatría y Atención Primaria - SEPEAP 2023. Grupo Pacífico, 2023. http://dx.doi.org/10.48158/sepeap2023.pd046.
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Deak, Michael D., and Scott C. McKenzie. "A COMPARISON OF NON-AVIAN THEROPOD AND BASAL AVIALAN LOCALITIES IN CHINA, GERMANY, AND ALBERTA WITH THE POTENTIAL FOR FEATHER PRESERVATION." In GSA Annual Meeting in Indianapolis, Indiana, USA - 2018. Geological Society of America, 2018. http://dx.doi.org/10.1130/abs/2018am-316030.
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Morgan Davis Hayes, Hongwei Xin, Hong Li, Timothy Shepherd, Yang Zhao, and John Paul Stinn. "Bioenergetics of Hy-Line Brown Hens in Aviary Houses." In 2012 IX International Livestock Environment Symposium (ILES IX). St. Joseph, MI: American Society of Agricultural and Biological Engineers, 2012. http://dx.doi.org/10.13031/2013.41576.
Full textReports on the topic "Aviara"
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Medici, André. Financial Support to Developing Countries to Face the Challenges Associated with Avian Influenza. Inter-American Development Bank, July 2006. http://dx.doi.org/10.18235/0006878.
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This presentation discusses financial support mechanisms for developing countries to deal with Avian Influenza consequences. It examines potential impacts of the disease, reasons to offer international financial support, what to finance in the pre-pandemic phase and what to finance in the pandemic phase, mechanisms of financing, and available international resources, including the role of the IDB. This paper was created for the Seminar Los Medios de Comunicación en America Latina frente a la Amenaza de la Gripe Aviar held in Washington D.C., on July 11th, 2006.
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Zhao, Yang, and Hongwei Xin. Ammonia Concentrations and Emissions of Aviary Hen Houses. Ames (Iowa): Iowa State University, January 2013. http://dx.doi.org/10.31274/ans_air-180814-740.
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Jenkins, Jessica D., Rebecca L. Parsons, Morgan Hayes, Hongwei Xin, and Suzanne T. Millman. Litter Use in an Aviary Laying Hen Housing System. Ames (Iowa): Iowa State University, January 2012. http://dx.doi.org/10.31274/ans_air-180814-1365.
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Chai, Lilong, Hongwei Xin, Yang Zhao, Tong Wang, and Michelle L. Soupir. Air Emissions Mitigation from Aviary Cage-free Hen Litter. Ames (Iowa): Iowa State University, January 2018. http://dx.doi.org/10.31274/ans_air-180814-390.
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Majo Masferrer, Natalia. La influenza aviar: una enfermedad en constante evolución. Sociedad Española de Bioquímica y Biología Molecular, March 2024. http://dx.doi.org/10.18567/sebbmrev_219.202403.dc002.
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Falconi, César, and Alejandro Nin Pratt. Impacto económico potencial de la influenza aviar en el sector avícola de América Latina y el Caribe. Inter-American Development Bank, September 2006. http://dx.doi.org/10.18235/0010293.
Full textAbstract:
Este estudio realiza un análisis económico del potencial impacto de la influenza aviar sobre el sector avícola y sus posibles encadenamientos en los países latinoamericanos, utilizando como referencia el impacto de esta enfermedad en distintas economías del Este y Sudeste Asiático. Dada la importancia del sector avícola en la región, el impacto de un brote de influenza aviar puede ser muy significativo tanto para el propio sector avícola como para el sector agropecuario. El presente estudio concluye que la inversión en el corto plazo para fortalecer los sistemas de sanidad animal en América Latina resultaría en significativos beneficios económicos para la región. Según los resultados de los diferentes escenarios elaborados, se determina que una inversión realizada a tiempo en los sistemas de sanidad animal para la preparación y la prevención de la influenza aviar es conveniente y puede ahorrarle a la región por lo menos 1.200 millones de dólares de pérdidas en el sector avícola, reduciendo al mismo tiempo la probabilidad de aparición de una pandemia humana.
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Franzreb, Kathleen E. A Mobile Aviary Design to Allow the Soft Release of Cavity Nesting Birds. Asheville, NC: U.S. Department of Agriculture, Forest Service, Southern Research Station, 1997. http://dx.doi.org/10.2737/srs-rn-005.
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Franzreb, Kathleen E. A Mobile Aviary Design to Allow the Soft Release of Cavity Nesting Birds. Asheville, NC: U.S. Department of Agriculture, Forest Service, Southern Research Station, 1997. http://dx.doi.org/10.2737/srs-rn-5.
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Hapner, Ralph W. Avian Influenza/Pandemic Influenza Program. Fort Belvoir, VA: Defense Technical Information Center, October 2007. http://dx.doi.org/10.21236/ada501658.
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Bradley, David Sherman. Avian Diagnostic and Therapeutic Antibodies. Office of Scientific and Technical Information (OSTI), December 2012. http://dx.doi.org/10.2172/1114116.
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