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Journal articles on the topic 'Autonomic changes'

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Published: 4 June 2021
Last updated: 19 February 2022

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1

Delamont, Robert S., and Matthew C. Walker. "Pre-ictal autonomic changes." Epilepsy Research 97, no. 3 (December 2011): 267–72. http://dx.doi.org/10.1016/j.eplepsyres.2011.10.016.

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2

Jin, Heung Yong, Hong Sun Baek, and Tae Sun Park. "Morphologic Changes in Autonomic Nerves in Diabetic Autonomic Neuropathy." Diabetes & Metabolism Journal 39, no. 6 (2015): 461. http://dx.doi.org/10.4093/dmj.2015.39.6.461.

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3

Heydarpour, F. "SODIUM CHANGES AUTONOMIC NERVOUS RESPONSE." Journal of Hypertension 22, Suppl. 2 (June 2004): S210. http://dx.doi.org/10.1097/00004872-200406002-00733.

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4

Cook, Glen A. "Toenail changes in autonomic neuropathy." Clinical Autonomic Research 28, no. 4 (June 19, 2018): 437–38. http://dx.doi.org/10.1007/s10286-018-0538-7.

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5

Igamova, Saodat, and Aziza Djurabekova. "CHANGES OF PSYCHOMOTOR DEVELOPMENT IN CHILDREN WITH PERINATAL BRAIN HYPOXIA." JOURNAL OF NEUROLOGY AND NEUROSURGICAL RESEARCH 3, no. 1 (March 30, 2020): 35–37. http://dx.doi.org/10.26739/2181-0982-2020-3-6.

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The central nervous system is the main mechanism that determines the nature of the reactivity and adaptation of the body to a set of environmental factors. At the same time, autonomic imbalance acts as the root cause of the pathological process or asa predisposing factor, and it is advisable to use the cardiovascular system as an indicator of neurohumoral regulationits reactions are associated with the activity of the central nervous system, autonomic nervous system and subcortical centers
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6

De Caprio, Lorenzo, Marco Papa, Domenico Acanfora, Carlo Vigorito, Vincenzo Canonico, Pasquale Abete, Sergio Cuomo, Fernando Gallucci, and Franco Rengo. "Autonomic Tone Changes during Isometric Exercise." American Journal of Noninvasive Cardiology 3, no. 1 (1989): 58–63. http://dx.doi.org/10.1159/000470584.

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7

Reinsberger, Claus, Rani Sarkis, Christos Papadelis, Chiran Doshi, David L. Perez, Gaston Baslet, Tobias Loddenkemper, and Barbara A. Dworetzky. "Autonomic Changes in Psychogenic Nonepileptic Seizures." Clinical EEG and Neuroscience 46, no. 1 (January 2015): 16–25. http://dx.doi.org/10.1177/1550059414567739.

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8

Moseley, Brian D. "Seizure-Related Autonomic Changes in Children." Journal of Clinical Neurophysiology 32, no. 1 (February 2015): 5–9. http://dx.doi.org/10.1097/wnp.0000000000000138.

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9

Yun, WH, SW Min, J. Huh, YJ Ro, and CS Kim. "Autonomic Changes in Preoperative Uncomplicated Diabetic Patients with Postural Changes." Journal of International Medical Research 38, no. 5 (October 2010): 1764–71. http://dx.doi.org/10.1177/147323001003800522.

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10

Schultz, Jordan L., and Peg C. Nopoulos. "Autonomic Changes in Juvenile-Onset Huntington’s Disease." Brain Sciences 10, no. 9 (August 26, 2020): 589. http://dx.doi.org/10.3390/brainsci10090589.

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Patients with adult-onset Huntington’s Disease (AOHD) have been found to have dysfunction of the autonomic nervous system that is thought to be secondary to neurodegeneration causing dysfunction of the brain–heart axis. However, this relationship has not been investigated in patients with juvenile-onset HD (JOHD). The aim of this study was to compare simple physiologic measures between patients with JOHD (n = 27 participants with 64 visits) and participants without the gene expansion that causes HD (GNE group; n = 259 participants with 395 visits). Using data from the Kids-JOHD study, we compared mean resting heart rate (rHR), systolic blood pressure (SBP), and diastolic blood pressure (DBP) between the JOHD and GNE groups. We also divided the JOHD group into those with childhood-onset JOHD (motor diagnosis received before the age of 13, [n = 16]) and those with adolescent-onset JOHD (motor diagnosis received at or after the age of 13 [n = 11]). We used linear mixed-effects models to compare the group means while controlling for age, sex, and parental socioeconomic status and including a random effect per participant and family. For the primary analysis, we found that the JOHD group had significant increases in their rHR compared to the GNE group. Conversely, the JOHD group had significantly lower SBP compared to the GNE group. The JOHD group also had lower DBP compared to the GNE group, but the results did not reach significance. SBP and DBP decreased as disease duration of JOHD increased, but rHR did not continue to increase. Resting heart rate is more sensitive to changes in autonomic function as compared to SBP. Therefore, these results seem to indicate that early neurodegenerative changes of the central autonomic network likely lead to an increase in rHR while later progression of JOHD leads to changes in blood pressure. We hypothesize that these later changes in blood pressure are secondary to neurodegeneration in brainstem regions such as the medulla.
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11

Mohammad, Samia Ashour, Ahmed Abdul Monem Gaber, and Lobna Mohammad Elnabil. "Autonomic Cardiac Changes in Temporal Lobe Epilepsy." Egyptian Journal of Hospital Medicine 64 (July 2016): 450–53. http://dx.doi.org/10.12816/0029037.

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12

Shusterman, Vladimir, Anna Beigel, S. Ismail Shah, Benhur Aysin, Raul Weiss, Venkateshwar K. Gottipaty, David Schwartzman, and Kelley P. Anderson. "Changes in autonomic activity and ventricular repolarization." Journal of Electrocardiology 32 (January 1999): 185–92. http://dx.doi.org/10.1016/s0022-0736(99)90078-x.

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13

Bergen, Donna C. "In a Heartbeat: Autonomic Changes during Seizures." Epilepsy Currents 5, no. 5 (September 2005): 194–96. http://dx.doi.org/10.1111/j.1535-7511.2005.00062.x.

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Abnormalities in Cardiac and Respiratory Function Observed during Seizures in Childhood O'Regan ME, Brown JK Dev Med Child Neurol 2005;47:4–9 The aim of this study was to observe any changes in cardiac and respiratory function that occur during seizures. Thirty-seven children (20 boys, 17 girls; median age, 7 years 6 months; range, 1 year 6 months to 15 years 6 months) were studied. We recorded electroencephalograms, respiratory rate, heart rate, electrocardiograms, blood pressure, oxygen saturation, heart rate variability (time-domain analysis), and cardiac vagal tone. A respiratory pause was defined as an interruption in respiration lasting more than 3 seconds but less than 15 seconds. Apnea was defined as absence of respiration for more than 15 seconds. Tachypnea was defined as a 10% increase in respiratory rate from the preictal baseline. Bradypnea was defined as a 10% decrease in respiratory rate from the preictal baseline. Significant hypoxia was defined as a saturation of less than 85%. A significant change in heart rate was taken as a 10% increase or decrease below the baseline rate. Data were obtained from 101 seizures: 40 focal seizures, 21 generalized seizures, and 40 absences. Focal seizures were frequently associated with significant respiratory abnormalities, tachypnea in 56%, apnea in 30%, frequent respiratory pauses in 70%, and significant hypoxemia in 40%. The changes seen in respiratory rate were statistically significant. Changes in cardiac parameters, an increase or decrease in heart rate, were observed in only 26% of focal seizures and 48% of generalized seizures. We conclude that seizure activity can disrupt normal physiologic regulation and control of respiratory and cardiac activity. Cardiac Arrhythmias in Focal Epilepsy: A Prospective Long-term Study Rugg-Gunn FJ, Simister RJ, Squirrell M, Holdright DR, Duncan JS Lancet 2004;364:2212–2219 Purpose Patients with epilepsy are at risk of sudden unexpected death. Neurogenic cardiac arrhythmias have been postulated as a cause. Electrocardiograms (ECGs) can be monitored by use of an implantable loop recorder for up to 18 months. We aimed to determine the frequency of cardiac arrhythmias in patients with refractory focal seizures over an extended period. Methods Twenty patients received an implantable loop recorder at one hospital in the United Kingdom. Devices were programmed to record automatically if bradycardia (<40 beats/min) or tachycardia ( >140 beats/min) was detected. Additionally, in the event of a seizure, patients and relatives could initiate ECG recording with an external activator device. Data were analyzed at regular intervals and correlated with seizure diaries. Results More than 220,000 patient-hours were monitored over a 24-month period, during which ECGs were captured on implantable loop recorders in 377 seizures. One patient withdrew from the study. In 16 patients, median heart rate during habitual seizures exceeded 100 beats/min. Ictal bradycardia (<40 beats/min) was rare, occurring in eight (2.1%) recorded events, in seven patients. Four patients (21%) had bradycardia or periods of asystole with subsequent permanent pacemaker insertion. Three of these four (16% of total) had potentially fatal asystole. Conclusions Clinical characteristics of patients with periictal cardiac abnormalities are closely similar to those at greatest risk of sudden unexpected death in epilepsy. Asystole might underlie many of these deaths, which would have important implications for the investigation of similar patients and affect present cardiac-pacing policies.
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14

Mehlsen, J., M. N. Kaijer, and A. B. Mehlsen. "Autonomic and electrocardiographic changes in cardioinhibitory syncope." Europace 10, no. 1 (December 12, 2007): 91–95. http://dx.doi.org/10.1093/europace/eum237.

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15

KOCHIADAKIS, GEORGE E., EVANGELOS A. PAPADIMITRIOU, MARY E. MARKETOU, STAVROS I. CHRYSOSTOMAKIS, EMMANUEL N. SIMANTIRAKIS, and PANOS E. VARDAS. "Autonomic Nervous System Changes in Vasovagal Syncope:." Pacing and Clinical Electrophysiology 27, no. 10 (October 2004): 1371–77. http://dx.doi.org/10.1111/j.1540-8159.2004.00641.x.

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16

Sevcencu, Cristian, and Johannes J. Struijk. "Autonomic alterations and cardiac changes in epilepsy." Epilepsia 51, no. 5 (January 7, 2010): 725–37. http://dx.doi.org/10.1111/j.1528-1167.2009.02479.x.

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17

Mano, T. "Age-related changes in human autonomic functions." Journal of the Autonomic Nervous System 43 (April 1993): 35–36. http://dx.doi.org/10.1016/0165-1838(93)90146-l.

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18

Pruneti, C., F. Fontana, and C. Fante. "Autonomic changes and stress response in psychopathology." International Journal of Psychophysiology 69, no. 3 (September 2008): 224–25. http://dx.doi.org/10.1016/j.ijpsycho.2008.05.069.

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19

Meretskyy, V. M., and M. M. Korda. "The features of neuroendocrine changes in cranial injury associated with diabetes mellitus." Kazan medical journal 94, no. 4 (December 15, 2013): 560–65. http://dx.doi.org/10.17816/kmj1971.

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Aim. To study the features of neuroendocrine regulation in cranial injury associated with diabetes mellitus. Methods. Experiments were carried out on 100 male white rats that were divided into the following groups: the first group (n=10) - control group consisted of 10 intact animals, the second group (n=40) - rats with simulated traumatic cranial injury, the third group (n=10) - rats with experimental diabetes mellitus, the fourth group (n=40) - rats with simulated traumatic cranial injury and experimental diabetes mellitus. Experimental diabetes mellitus was induced by a single streptozotocin solution intraperitoneal injection. Animals were withdrawn from the experiment at 3, 24 hours, 5 and 14 days after the traumatic cranial injury. The mathematical analysis of the cardiac rhythm variability was performed by ECG intervals data analysis. Corticosterone serum levels were measured using ELISA. Results. Based on a comprehensive ECG intervals data analysis, it was found that after experimental traumatic cranial injury the imbalance of autonomic nervous system with increased sympathetic autonomic nervous system function occurs, manifesting as a deregulation of cholinergic and adrenergic effects on sinoatrial node. In diabetes, an increase of the role of sympathetic effects on the sinus node functioning also occurred. Revealed autonomic nervous system deregulation after experimental traumatic cranial injury associated with experimental diabetes mellitus were associated with sympathetic autonomic nervous system over-function, parasympathetic autonomic nervous system exhausting and decreased recovery speed. There was a relevant increase in corticosterone serum concentration reaching its maximum 24 hours after traumatic cranial injury. Conclusion. A significant autonomic nervous system sympathetic shift and autonomic regulation intensifying characterizes the clinical course in traumatic cranial injury associated with diabetes mellitus, a positive correlation of corticosterone serum concentration and sympathetic autonomic nervous system over-function was revealed in animals with experimental traumatic cranial injury, particularly when associated with experimental diabetes mellitus.
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20

van Vliet, JA, AA Vein, MD Ferrari, and JG van Dijk. "Cardiovascular Autonomic Function Tests in Cluster Headache." Cephalalgia 26, no. 3 (March 2006): 329–31. http://dx.doi.org/10.1111/j.1468-2982.2006.01004.x.

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While facial autonomic signs are prominent during cluster headache (CH) attacks, cardiovascular autonomic changes have been described in few CH patients. Cardiovascular autonomic function tests (AFT) can be used to assess general autonomic function in CH patients in different stages of the disease. We aimed to assess whether general autonomic function is changed in CH patients during a cluster period. AFT was performed both during a cluster period, but outside an actual attack, and outside a cluster period in 18 patients. Heart rate variability was studied at rest, during deep breathing, after standing up and during a Valsalva manoeuvre. Blood pressure (BP) changes were recorded at rest, during standing up and during sustained handgrip. Measurements during and outside the cluster period were compared using the paired t-test. AFT measurements revealed no significant differences between the two measurements, except for diastolic BP in rest, which was higher during the cluster period [80.3 (SD 12.2) vs. 74.8 (SD 9.0), P = 0.04]. Autonomic dysfunction during a cluster period, but outside an attack, does not include systemic cardiovascular control.
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21

Kelbaek, H., O. Munck, N. J. Christensen, and J. Godtfredsen. "Autonomic nervous control of postprandial hemodynamic changes at rest and upright exercise." Journal of Applied Physiology 63, no. 5 (November 1, 1987): 1862–65. http://dx.doi.org/10.1152/jappl.1987.63.5.1862.

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Postprandial hemodynamic changes were studied in healthy subjects at rest and during exercise in the upright position with and without autonomic blockade of the heart. At rest cardiac output increased 61% mostly because of a stroke volume increase accomplished by left ventricular end-diastolic dilation. These changes seemed to be dependent on the autonomic nervous system, whereas the postprandial heart rate increase did not. During exercise cardiac output was 23% higher after food intake due to a rise in both stroke volume and heart rate. These changes were apparently under influence of the autonomic nervous system, whereas left ventricular dilation was not. The present findings indicate that most of the postprandial changes in the central circulation are under control of the autonomic nervous system.
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22

Podolskyi, Vl V., V. V. Podolskyi, V. K. Tishchenko, and S. K. Strizhak. "Oxidative stress as an important pathogenetic mechanism of changes in the reproductive health of women with somatoform diseases and violation of autonomic homeostasis." HEALTH OF WOMAN, no. 3(119) (May 3, 2017): 23–27. http://dx.doi.org/10.15574/hw.2017.119.23.

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The objective: to investigate the status of phospholipids and neutral lipids of blood cells in fertile aged women with violation of autonomic homeostasis and changes in reproductive health. Patients and methods. We examined 360 women with violation of autonomic homeostasis in the form of autonomic dysfunction syndrome and somatofom dysfunction of the autonomic nervous system of hypertonic, hypotonic and cardiac type and changes in reproductive health such as: artificial abortions, infertility and uterine leiomyoma. Results. The article presents data from a study of lipid peroxidation of cell membranes in fertile aged women with violation of autonomic homeostasis and changes in reproductive health. The obtained data of decay products concentrations of phospholipids and neutral lipids of cellular membranes indicate the ravages of free radical compounds to form intermediates and final products of lipid peroxidation. The results confirm the role of oxidative stress as an important pathogenetic mechanism of changes in the reproductive health of women with somatoform disorders and violations of autonomic homeostasis. Conclusion. Changes in the concentration of phospholipids, neutral lipids and lipid peroxidation products indicate a destabilizing effect on cell membranes in women with violation of autonomic homeostasis and reproductive health changes. Increasing the number of phospholipid degradation products show a decrease in the liquid component of cell membranes, which in turn changes the nature receptor interaction, as evidenced by a study hormonal and leads to disruption of feedback mechanisms concentration of hormones in the blood. Key words: oxidative stress, lipid peroxidation, women of fertile age, violations of autonomic homeostasis, changes in reproductive health.
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23

Moodithaya, Shailaja, and Sandhya T. Avadhany. "Gender Differences in Age-Related Changes in Cardiac Autonomic Nervous Function." Journal of Aging Research 2012 (2012): 1–7. http://dx.doi.org/10.1155/2012/679345.

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Ageing is associated with changes in cardiac autonomic control as measured by Heart Rate Variability (HRV). Not many studies have explored the influence of gender on age-related changes in cardiac autonomic regulation. This study evaluated the gender differences in age-associated changes in cardiac autonomic nervous activity by assessing HRV using frequency domain analysis of short-term stationary R-R intervals. HRV was studied in healthy males and females ranging in age from 6 to 55 years. Total power and absolute power in High-Frequency (HF) and Low-Frequency (LF) components as well as HF in normalized unit declined significantly with ageing. The HF/LF ratio was significantly higher in the adolescent and adult females compared to male of these age groups. This study suggests that gender differences exist in age-related changes in HRV. The finding that gender differences are limited to adolescent and adult age groups may indicate a role for female sex hormones in cardiac autonomic modulation.
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24

Lacuey, Nuria, Vasant Garg, Barbara Bangert, Johnson P. Hampson, Jonathan Miller, and Samden Lhatoo. "Insular resection may lead to autonomic function changes." Epilepsy & Behavior 97 (August 2019): 260–64. http://dx.doi.org/10.1016/j.yebeh.2019.04.035.

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25

Lacuey, Nuria, and Samden Lhatoo. "Insular resection may lead to autonomic function changes." Epilepsy & Behavior 99 (October 2019): 106475. http://dx.doi.org/10.1016/j.yebeh.2019.106475.

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26

Antipova, O. S., V. N. Krasnov, and O. S. Trofimova. "Changes in Autonomic Regulation in Moderate Depressive Disorders." Neuroscience and Behavioral Physiology 45, no. 4 (April 14, 2015): 404–12. http://dx.doi.org/10.1007/s11055-015-0089-5.

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27

Kuo, C. D., G. Y. Chen, M. J. Yang, H. M. Lo, and Y. S. Tsai. "Biphasic changes in autonomic nervous activity during pregnancy." British Journal of Anaesthesia 84, no. 3 (March 2000): 323–29. http://dx.doi.org/10.1093/oxfordjournals.bja.a013433.

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28

Latson, T. W., V. A. Hyndman, and M. A. Mirhej. "RAPID CHANGES IN AUTONOMIC REFLEXES INDUCED BY PENTOTHAL." Anesthesia & Analgesia 70, Supplement (February 1990): S226. http://dx.doi.org/10.1213/00000539-199002001-00226.

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29

De Angelis, K., R. B. Wichi, W. R. A. Jesus, E. D. Moreira, M. Morris, E. M. Krieger, and M. C. Irigoyen. "Exercise training changes autonomic cardiovascular balance in mice." Journal of Applied Physiology 96, no. 6 (June 2004): 2174–78. http://dx.doi.org/10.1152/japplphysiol.00870.2003.

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Experiments were performed to investigate the influence of exercise training on cardiovascular function in mice. Heart rate, arterial pressure, baroreflex sensitivity, and autonomic control of heart rate were measured in conscious, unrestrained male C57/6J sedentary ( n = 8) and trained mice ( n = 8). The exercise training protocol used a treadmill (1 h/day; 5 days/wk for 4 wk). Baroreflex sensitivity was evaluated by the tachycardic and bradycardic responses induced by sodium nitroprusside and phenylephrine, respectively. Autonomic control of heart rate and intrinsic heart rate were determined by use of methylatropine and propranolol. Resting bradycardia was observed in trained mice compared with sedentary animals [485 ± 9 vs. 612 ± 5 beats/min (bpm)], whereas mean arterial pressure was not different between the groups (106 ± 2 vs. 108 ± 3 mmHg). Baroreflex-mediated tachycardia was significantly enhanced in the trained group (6.97 ± 0.97 vs. 1.6 ± 0.21 bpm/mmHg, trained vs. sedentary), whereas baroreflex-mediated bradycardia was not altered by training. The tachycardia induced by methylatropine was significantly increased in trained animals (139 ± 12 vs. 40 ± 9 bpm, trained vs. sedentary), whereas the propranolol effect was significantly reduced in the trained group (49 ± 11 vs. 97 ± 11 bpm, trained vs. sedentary). Intrinsic heart rate was similar between groups. In conclusion, dynamic exercise training in mice induced a resting bradycardia and an improvement in baroreflex-mediated tachycardia. These changes are likely related to an increased vagal and decreased sympathetic tone, similar to the exercise response observed in humans.
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30

Morris, Mariana, Mary P. Key, and Vera Farah. "Sarin produces delayed cardiac and central autonomic changes." Experimental Neurology 203, no. 1 (January 2007): 110–15. http://dx.doi.org/10.1016/j.expneurol.2006.07.027.

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31

Ekholm, E. M. K., R. U. Erkkola, S. J. Piha, J. O. Jalonen, T. H. Metsälä, and K. J. Antila. "Changes in autonomic cardiovascular control in mid-pregnancy." Clinical Physiology 12, no. 5 (September 1992): 527–36. http://dx.doi.org/10.1111/j.1475-097x.1992.tb00356.x.

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32

&NA;. "Biphasic Changes in Autonomic Nervous Activity During Pregnancy." Survey of Anesthesiology 45, no. 1 (February 2001): 33. http://dx.doi.org/10.1097/00132586-200102000-00030.

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33

Sumner, M. G., and P. A. Hwang. "1. Autonomic changes following traumatic brain injury (tbi)." Clinical Neurophysiology 120, no. 8 (August 2009): e181. http://dx.doi.org/10.1016/j.clinph.2009.05.021.

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34

El-Sawy, Noha, Gihan El-Tantawi, Ghada Abdel Hadi Achmawi, Hussein Sultan, and Safaa Younis. "Autonomic changes in fibromyalgia: Clinical and electrophysiological study." Alexandria Journal of Medicine 48, no. 3 (September 1, 2012): 215–22. http://dx.doi.org/10.1016/j.ajme.2012.02.007.

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35

Sedova, Ksenia A., Sergey L. Goshka, Vladimir A. Vityazev, Dmitriy N. Shmakov, and Jan E. Azarov. "Load-induced changes in ventricular repolarization: evidence of autonomic modulation." Canadian Journal of Physiology and Pharmacology 89, no. 12 (December 2011): 935–44. http://dx.doi.org/10.1139/y11-098.

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Augmented hemodynamic load increases the risk of arrhythmogenesis by modulating cardiac repolarization duration. We hypothesized that the intervention on the autonomic tone may affect the load-dependent changes in ventricular repolarization. Activation–recovery intervals were measured in unipolar electrograms simultaneously recorded from 64 ventricular epicardial leads, in a total of 26 chinchilla rabbits in resting conditions, and after 1 and 10 min of aortic stenosis. Eleven animals were given atropine and propranolol before the loading. The short-term stenosis decreased the activation–recovery intervals in the right ventricle, whereas the prolonged overload increased the repolarization duration in both ventricles. The treatment with the β-adrenergic and M-cholinergic blockers prolonged the activation–recovery intervals, especially at the left ventricle, attenuating the apicobasal and interventricular gradients of repolarization duration seen in the baseline state. Further ventricular loading shortened the repolarization duration in both ventricles in animals with autonomic blockade. Thus, the autonomic tone was shown to be essential for the development of repolarization heterogeneity across the ventricles. The autonomic blockade transformed the biphasic changes of activation–recovery intervals into their monophasic shortening at aortic stenosis.
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36

Subramanian, Sandya, Patrick L. Purdon, Riccardo Barbieri, and Emery N. Brown. "Quantitative assessment of the relationship between behavioral and autonomic dynamics during propofol-induced unconsciousness." PLOS ONE 16, no. 8 (August 11, 2021): e0254053. http://dx.doi.org/10.1371/journal.pone.0254053.

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During general anesthesia, both behavioral and autonomic changes are caused by the administration of anesthetics such as propofol. Propofol produces unconsciousness by creating highly structured oscillations in brain circuits. The anesthetic also has autonomic effects due to its actions as a vasodilator and myocardial depressant. Understanding how autonomic dynamics change in relation to propofol-induced unconsciousness is an important scientific and clinical question since anesthesiologists often infer changes in level of unconsciousness from changes in autonomic dynamics. Therefore, we present a framework combining physiology-based statistical models that have been developed specifically for heart rate variability and electrodermal activity with a robust statistical tool to compare behavioral and multimodal autonomic changes before, during, and after propofol-induced unconsciousness. We tested this framework on physiological data recorded from nine healthy volunteers during computer-controlled administration of propofol. We studied how autonomic dynamics related to behavioral markers of unconsciousness: 1) overall, 2) during the transitions of loss and recovery of consciousness, and 3) before and after anesthesia as a whole. Our results show a strong relationship between behavioral state of consciousness and autonomic dynamics. All of our prediction models showed areas under the curve greater than 0.75 despite the presence of non-monotonic relationships among the variables during the transition periods. Our analysis highlighted the specific roles played by fast versus slow changes, parasympathetic vs sympathetic activity, heart rate variability vs electrodermal activity, and even pulse rate vs pulse amplitude information within electrodermal activity. Further advancement upon this work can quantify the complex and subject-specific relationship between behavioral changes and autonomic dynamics before, during, and after anesthesia. However, this work demonstrates the potential of a multimodal, physiologically-informed, statistical approach to characterize autonomic dynamics.
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37

Rodrigues, Bruno, Fabio S. Lira, Fernanda M. Consolim-Colombo, Juraci A. Rocha, Erico C. Caperuto, Kátia De Angelis, and Maria-Cláudia Irigoyen. "Role of Exercise Training on Autonomic Changes and Inflammatory Profile Induced by Myocardial Infarction." Mediators of Inflammation 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/702473.

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The cardiovascular autonomic imbalance in patients after myocardial infarction (MI) provides a significant increase in mortality rate, and seems to precede metabolic, hormonal, and immunological changes. Moreover, the reduction in the parasympathetic function has been associated with inflammatory response in different pathological conditions. Over the years, most of the studies have indicated the exercise training (ET) as an important nonpharmacological tool in the management of autonomic dysfunction and reduction in inflammatory profile after a myocardial infarction. In this work, we reviewed the effects of ET on autonomic imbalance after MI, and its consequences, particularly, in the post-MI inflammatory profile. Clinical and experimental evidence regarding relationship between alterations in autonomic regulation and local or systemic inflammation response after MI were also discussed.
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38

Kim, Jung Bin, Hayom Kim, Chan-Nyung Lee, Kun-Woo Park, and Byung-Jo Kim. "Regional Gray Matter Volume Changes in Parkinson’s Disease with Orthostatic Hypotension." Brain Sciences 11, no. 3 (February 26, 2021): 294. http://dx.doi.org/10.3390/brainsci11030294.

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Neurodegenerative change in the central nervous system has been suggested as one of the pathophysiological mechanisms of autonomic nervous system dysfunction in Parkinson’s disease (PD). We analyzed gray matter (GM) volume changes and clinical parameters in patients with PD to investigate any involvement in the brain structures responsible for autonomic control in patients with PD having orthostatic hypotension (OH). Voxel-based morphometry was applied to compare regional GM volumes between PD patients with and without OH. Multivariate logistic regression analysis using a hierarchical model was carried out to identify clinical factors independently contributing to the regional GM volume changes in PD patients with OH. The Sobel test was used to analyze mediation effects between the independent contributing factors to the GM volume changes. PD patients with OH had more severe autonomic dysfunction and reduction in volume in the right inferior temporal cortex than those without OH. The right inferior temporal volume was positively correlated with the Qualitative Scoring MMSE Pentagon Test (QSPT) score, reflecting visuospatial/visuoperceptual function, and negatively correlated with the Composite Autonomic Severity Score (CASS). The CASS and QSPT scores were found to be factors independently contributing to regional volume changes in the right inferior temporal cortex. The QSPT score was identified as a mediator in which regional GM volume predicts the CASS. Our findings suggest that a decrease in the visuospatial/visuoperceptual process may be involved in the presentation of autonomic nervous system dysfunction in PD patients.
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39

Edwards, Ian J. "HIGHLIGHTS IN BASIC AUTONOMIC NEUROSCIENCES: CHANGES TO THE AUTONOMIC NERVOUS SYSTEM ASSOCIATED WITH HEALTHY AGEING." Autonomic Neuroscience 183 (July 2014): 1–3. http://dx.doi.org/10.1016/j.autneu.2014.04.004.

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40

Al-Shargabi, Tareq, Daniel Reich, R. Govindan, Somya Shankar, Marina Metzler, Caitlin Cristante, Robert McCarter, Anthony Sandler, Mariam Said, and Adre Plessis. "Changes in Autonomic Tone in Premature Infants Developing Necrotizing Enterocolitis." American Journal of Perinatology 35, no. 11 (April 2, 2018): 1079–86. http://dx.doi.org/10.1055/s-0038-1639339.

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Background Necrotizing enterocolitis (NEC) is a complication of prematurity with a high mortality rate. Currently, there are no reliable biomarkers capable of identifying infants at risk for developing NEC. We sought to determine the autonomic nervous system antecedents of NEC in premature infants, using heart rate variability (HRV). Materials and Methods HRV was quantified by retrieving archived electrocardiogram (EKG) data from 30 premature infants from 4 days prior, through 4 days after, the clinical NEC diagnosis. HRV metrics were compared with those on the diagnosis day using the receiver operating characteristic (ROC) analysis. Results HRV metrics showed a depression of autonomic tone that preceded the clinical NEC diagnosis by 2 days, and which recovered to baseline by 2 days after diagnosis (area under the curve [AUC] < 0.7). The pattern of HRV change was significantly associated with the clinical severity of NEC (stage II vs. stage III). Conclusion Our studies suggest that readily accessible metrics of autonomic depression might expedite the diagnosis of NEC and its severity in a clinically meaningful manner. Clearly, these studies need to be extended prospectively to determine the diagnostic utility of this approach.
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41

Hazarika, Arpana, and Archana Sarma. "Effect of Life Style Changes on Cardiovascular Autonomic Function." International Journal of Physiology 5, no. 1 (2017): 46. http://dx.doi.org/10.5958/2320-608x.2017.00010.5.

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42

Amar, David, Hao Zhang, Saul Miodownik, and Alan H. Kadish. "Autonomic changes preceding the onset of postoperative atrial fibrillation." Journal of the American College of Cardiology 41, no. 6 (March 2003): 101. http://dx.doi.org/10.1016/s0735-1097(03)80650-2.

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43

Fukudo, S., M. Kano, T. Hamaguchi, and Y. Sagami. "Brain processing and autonomic changes in perception-induced emotion." Journal of Psychosomatic Research 55, no. 2 (August 2003): 120. http://dx.doi.org/10.1016/s0022-3999(03)00435-5.

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44

Fukudo, S., Y. Shimada, Y. Sagami, J. Tayama, M. Kano, M. Kanazawa, T. Nomura, and M. Hongo. "Brain processing and autonomic changes in perception-induced emotion." Journal of Psychosomatic Research 55, no. 2 (August 2003): 120. http://dx.doi.org/10.1016/s0022-3999(03)00436-7.

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45

Critchley, Hugo D., Catriona D. Good, Richard S. J. Frackowiak, Christopher J. Mathias, and Raymond J. Dolan. "Changes in cerebral morphology consequent to peripheral autonomic denervation." NeuroImage 13, no. 6 (June 2001): 780. http://dx.doi.org/10.1016/s1053-8119(01)92122-6.

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46

Critchley, Hugo D., Catriona D. Good, John Ashburner, Richard S. Frackowiak, Christopher J. Mathias, and Raymond J. Dolan. "Changes in cerebral morphology consequent to peripheral autonomic denervation." NeuroImage 18, no. 4 (April 2003): 908–16. http://dx.doi.org/10.1016/s1053-8119(03)00011-9.

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47

Brandenberger, Gabrielle, Antoine U. Viola, Jean Ehrhart, Anne Charloux, Bernard Geny, FranCois Piquard, and Chantal Simon. "Age-related changes in cardiac autonomic control during sleep." Journal of Sleep Research 12, no. 3 (September 2003): 173–80. http://dx.doi.org/10.1046/j.1365-2869.2003.00353.x.

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48

Hautala, Arto, Mikko P. Tulppo, Timo H. Mäkikallio, Raija Laukkanen, Seppo Nissilä, and Heikki V. Huikuri. "Changes in cardiac autonomic regulation after prolonged maximal exercise." Clinical Physiology 21, no. 2 (March 2001): 238–45. http://dx.doi.org/10.1046/j.1365-2281.2001.00309.x.

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49

Staber, M., S. Hansen, and P. OO Julu. "Changes in brainstem autonomic function following induction of anaesthesia." European Journal of Anaesthesiology 24, Supplement 39 (June 2007): 74. http://dx.doi.org/10.1097/00003643-200706001-00271.

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50

OBERLANDER, T. F., C. B. BERDE, K. H. LAM, L. A. RAPPAPORT, and J. P. SAUL. "Infants Tolerate Spinal Anesthesia with Minimal Overall Autonomic Changes." Survey of Anesthesiology 39, no. 5 (October 1995): 293. http://dx.doi.org/10.1097/00132586-199510000-00023.

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