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Relevant bibliographies by topics / Auto-immune blistering diseases/dermatology

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Academic literature on the topic 'Auto-immune blistering diseases/dermatology'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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Journal articles on the topic "Auto-immune blistering diseases/dermatology"

1

GHOHESTANI, R. "Sub-epidermal auto-immune blistering skin diseases and newly identified antigens." Journal of the European Academy of Dermatology and Venereology 11 (September 1998): S121. http://dx.doi.org/10.1016/s0926-9959(98)94988-1.

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2

ROUSSELLE, P. "Biological investigation of adhesion molecules involved in auto-immune blistering skin diseases." Journal of the European Academy of Dermatology and Venereology 11 (September 1998): S120—S121. http://dx.doi.org/10.1016/s0926-9959(98)94985-6.

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3

Gopalakrishnan, Geetha K., V. Bindu, and Najeeba Riyaz. "Clinicopathological and immunofluorescence study of vesiculobullous disorders." International Journal of Research in Dermatology 5, no. 2 (April 26, 2019): 281. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20190912.

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<p class="abstract"><strong>Background:</strong> Vesiculobullous diseases are mostly immune mediated and diagnosed based on the clinical features, histology and Immunofluorescence. The aim of the study was to identify the immunofluorescence pattern in auto immune vesiculobullous diseases and correlate it with the clinical profile and histology.</p><p class="abstract"><strong>Methods:</strong> Patients attending the dermatology outpatient department in a tertiary hospital with vesiculobullous diseases, suggestive of auto immune aetiology were evaluated clinically. Histopathology and direct immuno-fluorescence were done in all patients.<strong></strong></p><p class="abstract"><strong>Results:</strong> During the one year period from June 2008 to July 2009, 40 patients with vesiculobullous disorders clinically suggestive of auto immune aetiology attended the outpatient department. Out of the 40 patients, 22 (55%) patients were diagnosed to have intraepidermal with female preponderance and 18 patients (45%) sub epidermal blistering diseases. Bullous pemphigoid was the commonest sub epidermal disease, seen in 8 patients.</p><p class="abstract"><strong>Conclusions:</strong> In all cases diagnosed clinically as pemphigus a histological diagnosis of pemphigus was made (100%). The clinical variants of pemphigus could also be diagnosed in all cases histologically (100%). The positive and negative predictive value was 100% in pemphigus group cases. Histology of all patients showed subepidermal bulla (100%). A specific diagnosis could be made in 18 patients with sub epidermal disease (100%). DIF was found to be an invaluable tool in diagnosing different diseases belonging to the sub epidermal group, but it was not of much help in sub classifying variants of pemphigus.</p><p class="abstract"> </p>

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4

Harman, K. E., and M. M. Black. "High-dose intravenous immune globulin for the treatment of autoimmune blistering diseases: an evaluation of its use in 14 cases." British Journal of Dermatology 140, no. 5 (May 24, 1999): 865–74. http://dx.doi.org/10.1046/j.1365-2133.1999.02817.x.

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5

Mydlarski, P. Régine, Vincent Ho, and Neil H. Shear. "Canadian Consensus Statement on the Use of Intravenous Immunoglobulin Therapy in Dermatology." Journal of Cutaneous Medicine and Surgery 10, no. 5 (September 2006): 205–21. http://dx.doi.org/10.2310/7750.2006.00048.

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Background: As a safe, well-tolerated, and potentially beneficial therapy, intravenous immunoglobulin (IVIG) has been increasingly used by dermatologists to treat immune-mediated skin disease. However, practical and comprehensive guidelines for the use of IVIG have yet to be established. Objective: To develop the first Canadian consensus statement on the use of IVIG therapy in skin disease. Methods: A group of Canadian dermatologists convened to discuss current issues in IVIG therapy. The participants reviewed and evaluated the literature and shared clinical experience. Using a modified Delphi process, a consensus statement was developed. Results: Herein we provide a brief overview of pemphigus vulgaris, pemphigus foliaceus, bullous pemphigoid, mucous membrane pemphigoid, epidermolysis bullosa acquisita, Stevens-Johnson syndrome, and toxic epidermal necrolysis. Recommendations for the management of these diseases are detailed, and therapeutic algorithms for the treatment of various autoimmune mucocutaneous blistering diseases are presented. The appropriate use of IVIG therapy is placed in context for each disease. Conclusion: Although preliminary data suggest that IVIG is a safe and effective therapy for many skin disorders, uncontrolled clinical trials, case series, and anecdotal case reports dominate the literature. Collaborative randomized controlled trials are required to firmly establish the role of IVIG in dermatology.

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6

Korman, N. J. "Immune-mediated subepithelial blistering diseases of the mucous membranes. Improving the detection of circulating autoantibodies by the use of concentrated serum samples." Archives of Dermatology 132, no. 10 (October 1, 1996): 1194–98. http://dx.doi.org/10.1001/archderm.132.10.1194.

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7

Chan, L. S. "Immune-mediated subepithelial blistering diseases of mucous membranes. Pure ocular cicatricial pemphigoid is a unique clinical and immunopathological entity distinct from bullous pemphigoid and other subsets identified by antigenic specificity of autoantibodies." Archives of Dermatology 129, no. 4 (April 1, 1993): 448–55. http://dx.doi.org/10.1001/archderm.129.4.448.

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8

BARTH, J. H., V. A. VENNING, and F. WOJNAROWSKA. "Palmo-plantar involvement in auto-immune blistering disorders-pemphigoid, linear IgA disease and herpes gestationis." Clinical and Experimental Dermatology 13, no. 2 (March 1988): 85–86. http://dx.doi.org/10.1111/j.1365-2230.1988.tb00664.x.

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9

Takahashi, Hayato, Hisato Iriki, Miho Mukai, Aki Kamata, Hisashi Nomura, Jun Yamagami, and Masayuki Amagai. "Autoimmunity and immunological tolerance in autoimmune bullous diseases." International Immunology 31, no. 7 (March 19, 2019): 431–37. http://dx.doi.org/10.1093/intimm/dxz030.

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Abstract Autoimmune diseases are devastating conditions in which the immune system is directed against the host, leading to life-threatening destruction of organs. Although autoantigens are ill-defined in most autoimmune diseases, this is not the case in the skin. Autoimmune bullous diseases have been extensively studied with detailed characterization of autoantigens, the epitopes that are targeted, and the mechanisms of action that mediate autoimmune tissue destruction. Pemphigus is an autoimmune bullous disease caused by circulating IgG that targets two desmosomal proteins, desmoglein 1 and 3, which are crucial for cell–cell adhesion of keratinocytes. Binding of auto-antibodies to desmogleins impairs keratinocyte adhesion, leading to severe blistering disease. Mouse models that recapitulate the human disease have been instrumental in elucidating the detailed pathophysiology. Taking advantage of the fact that desmogleins are specifically targeted in pemphigus, studying humoral and cellular autoimmunity against these autoantigens provides us with an opportunity to understand not only the effector mechanisms of B and T cells in mediating pathology but also how autoreactive lymphocytes are regulated during development in the thymus and post-development in the periphery. This review introduces pemphigus and its subtypes as prototypic autoimmune diseases from which recent basic and translational developments should provide insight into how autoimmunity develops.

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10

JONKMAN, M. "Fluorescence overlay antigen mapping and the use of antigen-deficient substrates in the fine diagnosis of auto-immune bullous dermatoses." Journal of the European Academy of Dermatology and Venereology 11 (September 1998): S122. http://dx.doi.org/10.1016/s0926-9959(98)94991-1.

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Dissertations / Theses on the topic "Auto-immune blistering diseases/dermatology"

1

Venning, Vanessa Ann. "Bullous pemphigoid : clinical and pathogenetic studies." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334938.

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Book chapters on the topic "Auto-immune blistering diseases/dermatology"

1

Maruthappu, Thiviyani, and David P. Kelsell. "Inherited skin disease." In Oxford Textbook of Medicine, edited by Roderick J. Hay, 5602–11. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0552.

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Considerable advances in our understanding of inherited skin diseases have been made over the last decade as a result of high throughput sequencing technologies, including next generation sequencing and whole exome sequencing. The genetic basis of a myriad of monogenic epidermal disorders and syndromes including blistering diseases, ichthyoses, palmoplantar keratodermas, and the ectodermal dysplasias have now been elucidated. However, most patients referred from primary care to the dermatology clinic will be seeking treatment for a few common skin disorders such as psoriasis, eczema, and acne. The genetic basis of these disorders is rather more complex, but progress has been made through genome-wide association studies, which, for example, have linked susceptibility variants in the gene for filaggrin (FLG) and SPINK5 to atopic eczema, and IL23R and many other immune-related genes to psoriasis.

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