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Journal articles on the topic 'Atrial fibrillation; myocardial infarction'

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Author: Grafiati

Published: 10 December 2022

Last updated: 28 January 2023

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1

Vermond, Rob A., Isabelle C. Van Gelder, Harry J. Crijns, and Michiel Rienstra. "Does Myocardial Infarction Beget Atrial Fibrillation and Atrial Fibrillation Beget Myocardial Infarction?" Circulation 131, no. 21 (May 26, 2015): 1824–26. http://dx.doi.org/10.1161/circulationaha.115.016595.

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2

Bhattarai, Radha, and Sergey Anatolevich Sayganov. "Atrial Fibrillation in Acute Myocardial Infarction." Nepalese Heart Journal 12, no. 1 (March 24, 2015): 15–20. http://dx.doi.org/10.3126/njh.v12i1.12327.

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Background and Aim: New-onset atrial fibrillationfrequently complicates acute myocardial infarction.The incidence ranges from 6 - 21% “1”.We aim todetermine the incidence of atrial fibrillation in thesetting of acute myocardial infarction.Methods: This was a single center prospective study,conducted in the coronary care unit of Saint-PetersburgPokrovskaya city hospital, Russia, during the period,June 2013 to June 2014. Sixty consecutive patientsof acute myocardial infarction with atrial fibrillationwere included in this study. Onset, duration, and modeof termination of atrial fibrillation, clinical factorsassociated with its presentation and its relation withpatient outcome were evaluated.Results: Among the 60 patients 33 (55%) had inferiorwall myocardial infarction and 27 (45%) patientshad anterior wall myocardial infarction. In patientswith inferior wall myocardial infarction the onsetof atrial fibrillation occurred within 24 hours in 30(91%) patients, after 24 hour in 3 (9%) patients. Theepisode lasted for less than 24 hours in 12 (36%), andmore than 24 hours in 21 (64%) patients. In anteriorwall myocardial infarction atrial fibrillation occurredwithin 24 hours in 2 (7%) patients, on the second dayin 25 (93%). The episode lasted less than 24 hoursin 3 (11%), 48 hours in (85%), 72 hours in 1 (4%)patients. There was a significant difference in theonset and duration of atrial fibrillation in relation tothe location of infarction (P < 0.0001). Anterior wallmyocardial infarction was associated with late onsetof atrial fibrillation, increased frequency of heartfailure and higher CCU mortality.Conclusion: The onset of atrial fibrillation in anteriorwall myocardial infarction occurred later and lastedlonger in comparison to inferior wall myocardialinfarction. Increased incidence of heart failure andhigher CCU mortality was associated with anteriorwall myocardial infarction.DOI: http://dx.doi.org/10.3126/njh.v12i1.12327 Nepalese Heart Journal Vol.12(1) 2015: 15-20

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3

Nambiar, Dr Supriya D. "Cardiac and Extra Cardiac Predictors and Complications of Acute Atrial Fibrillation Complicating ST Elevation Myocardial Infarction (STEMI) ST Elevation myocardial infarction Acute Atrial Fibrillation (STAAF) Study." Journal of Medical Science And clinical Research 05, no. 05 (May 23, 2017): 22124–34. http://dx.doi.org/10.18535/jmscr/v5i5.139.

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4

Jortveit, Jarle, Are H. Pripp, Jørund Langørgen, and Sigrun Halvorsen. "Poor adherence to guideline recommendations among patients with atrial fibrillation and acute myocardial infarction." European Journal of Preventive Cardiology 26, no. 13 (April 9, 2019): 1373–82. http://dx.doi.org/10.1177/2047487319841940.

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Background The prevalence of atrial fibrillation in patients with acute myocardial infarction is largely unknown. The aims of the present study were to assess the prevalence of atrial fibrillation in a nationwide cohort of patients with acute myocardial infarction, to assess the prescription of anticoagulation therapy, and to study the long-term outcomes. Design A nationwide registry-based cohort study. Methods All patients registered in the Norwegian Myocardial Infarction Registry between 2013 and 2016 were included and followed up through 2017. Stroke rates during follow-up were obtained through linkage with the Norwegian Patient Registry. Results In total, 47,204 patients were registered in the Norwegian Myocardial Infarction Registry. Atrial fibrillation on admission was recorded in 5393 (11%) patients, and 2190 (5%) additional patients developed atrial fibrillation during their hospital stay. Only 45% of patients with atrial fibrillation on admission and CHA2DS2-VASc score ≥ 2 were treated with anticoagulation therapy prior to myocardial infarction, and 56% of patients with atrial fibrillation and CHA2DS2-VASc score ≥ 2 were prescribed anticoagulation therapy at discharge. Patients with myocardial infarction and atrial fibrillation had an increased risk of stroke or death during 822 (426, 1278) days of follow-up compared with patients without atrial fibrillation (multivariate adjusted hazard ratio 1.4, 95% confidence interval 1.3–1.4). Conclusions Almost half of patients with atrial fibrillation and myocardial infarction were not prescribed the guideline recommended treatment with anticoagulation therapy at discharge, and their long-term risk of stroke and death was increased compared with patients without atrial fibrillation. Increased efforts to improve the treatment of patients with myocardial infarction and atrial fibrillation are needed.

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Alasady, Muayad, Nicholas J. Shipp, Anthony G. Brooks, Han S. Lim, Dennis H. Lau, David Barlow, Pawel Kuklik, et al. "Myocardial Infarction and Atrial Fibrillation." Circulation: Arrhythmia and Electrophysiology 6, no. 4 (August 2013): 738–45. http://dx.doi.org/10.1161/circep.113.000163.

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6

Dukes, Jonathan W., and Gregory M. Marcus. "Atrial Fibrillation Begets Myocardial Infarction." JAMA Internal Medicine 174, no. 1 (January 1, 2014): 5. http://dx.doi.org/10.1001/jamainternmed.2013.11392.

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7

Neobutov, Nikolai, and Sergey Kolbasnikov. "The state of the myocardium, the vascular wall and the severity of anxiety-depressive disorders in patients with myocardial infarction and atrial fibrillation." Archiv Euromedica 9, no. 1 (April 30, 2019): 109–12. http://dx.doi.org/10.35630/2199-885x/2019/9/1/109.

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In patients with myocardial infarction with atrial fibrillation, the relationship between vascular wall parameters, structural and functional changes in the myocardium, and the severity of emotional disorders were clarified. 138 inpatients of the cardiology department were closely monitored. It turned out that patients with myocardial infarction and a constant form of atrial fibrillation, unlike patients with paroxysmal form, show significant structural changes in the myocardium, endothelial dysfunction with a reduction in wall elasticity, which must be considered during therapeutic and preventive, rehabilitation measure

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8

Tomcsányi, János, Katalin Takó, and Balázs Sármán. "Recidív akut myocardialis infarctus pitvarfibrilláció miatt." Orvosi Hetilap 157, no. 5 (January 2016): 191–93. http://dx.doi.org/10.1556/650.2016.30349.

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Coronary thromboembolism with subsequent myocardial infarction is a rare complication of atrial fibrillation. The authors present the history of a 55-year-old male with a history of acute myocardial infarction caused by thromboembolism in the distal part of left anterior descending coronary artery and paroxysmal atrial fibrillation, who presented one year later with new chest pain, ST-segment elevation and atrial fibrillation. Coronarography confirmed the presence of thrombus in the circumflex coronary artery. Transesophageal echocardiogram showed left atrial appendage thrombus. To the knowledge of the authors this is the first report of recurrent myocardial infarction caused by atrial fibrillation. Orv. Hetil., 2016, 157(5), 191–193.

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9

Cheng, Liting, and Yongquan Wu. "Mesenteric Ischemia and Myocardial Infarction Associated with Atrial Fibrillation." Case Reports in Cardiology 2018 (2018): 1–3. http://dx.doi.org/10.1155/2018/7860397.

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Atrial fibrillation is a common disease correlated with embolism incidents. However, there is lack of report on atrial fibrillation causing myocardial infarction and mesenteric ischemia at the same time. Our patient is a 69-year-old woman who had undergone thoracic surgery a month before presented to our hospital with newly discovered atrial fibrillation, abdominal pain, and ST-elevated myocardial infarction. This is a rare case that atrial fibrillation took place one month after surgery and caused embolism incidents in both coronary artery and mesenteric artery.

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10

Radha, B., S. A. Sayganov, and T. Y. Gromiko. "ATRIAL FIBRILLATION IN PATIENTS WITH INFERIOR MYOCARDIAL INFARCTION." HERALD of North-Western State Medical University named after I.I. Mechnikov 7, no. 1 (March 15, 2015): 46–52. http://dx.doi.org/10.17816/mechnikov20157146-52.

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Objective: To elucidate the mechanism of atrial fibrillation and evaluate left atrium function after restoration of sinus rhythm in patients with acute posterior wall myocardial infarction ( MI). Materials and Methods: The study included 53 patients with posterior wall MI.All patients were divided into 2 groups. The first group consisted of 33 a people with paroxysms of atrial fibrillation (AF), and the second included 20 control subjects without arrhythmia. All percutaneous intervention was performed within the first 24 hours. Patients were evaluated for time and duration of paroxysms, the size of the heart chambers and the recovery time of the left atrium (LA)function. Results: Patients with posterior wall myocardial infarction developed AF in the early stages of the disease (in 91% on the first day), with short duration of paroxysms, stopped spontaneously and often within 1 hour (in 11 people). There were no significant differences in the size of the heart chambers, left ventricular contractility and hemodynamic disturbances in patients of both groups. AF in most cases developed in patients without left ventricular failure (in 27 people; 82%). Wherein the proximal right coronary artery occlusion was observed more frequently in patients with atrial fibrillation, than in the control group (17 vs 2; p <0,001). Approximately half patients(16 ) with AF before the appearance of atrial fibrillation bradysystolya of atria (less than 50 in 1 min) was recorded, due to acute sinus node dysfunction. After the reversion of sinus rhythm mechanical function of the LA was absent in only 4 people with left ventricular failure. Effective systole of LA was restored only 7 days after reversion to sinus rhythm. The rare occurrence of mechanical dysfunction after discontinuation of arrhythmia indicates a low probability of thrombosis and embolism in the systemic circulation. Conclusion: In cases of patients with posterior wall localization of MI main causes of AF include acute ischemia of atria due to occlusion of the right coronary artery above the branches supplying atrium. Atrial bradysystolya due to acute sinus node dysfunction often contributes to the development of AF as a substitute atrial rate (acute syndrome of tachy-bradycardia). In case of patients with posterior wall MI AF episodes were rarely accompanied by hemodynamic disturbances and the risk of systemic thromboembolism after reversion to sinus rhythm was low.

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11

Helmers, C., T. Lundman, L. Mogensen, E. Orinius, A. Sjögren, and P. O. Wester. "ATRIAL FIBRILLATION IN ACUTE MYOCARDIAL INFARCTION." Acta Medica Scandinavica 193, no. 1-6 (April 24, 2009): 39–44. http://dx.doi.org/10.1111/j.0954-6820.1973.tb10535.x.

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12

Sugiura, Tetsuro, Toshiji Iwasaka, Akio Ogawa, Yoshihachiro Shiroyama, Hisako Tsuji, Hideki Onoyama, and Mitsuo Inada. "Atrial fibrillation in acute myocardial infarction." American Journal of Cardiology 56, no. 1 (July 1985): 27–29. http://dx.doi.org/10.1016/0002-9149(85)90560-0.

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13

Prystowsky, Eric N., and Edward T. Fry. "Atrial Fibrillation and Incident Myocardial Infarction." JAMA 312, no. 10 (September 10, 2014): 1049. http://dx.doi.org/10.1001/jama.2014.5742.

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14

El-Shetry, Mohamed, Ragab Mahfouz, Abdel-Fattah Frere, and Mohamed Abdeldayem. "The interplay between atrial fibrillation and acute myocardial infarction." British Journal of Hospital Medicine 82, no. 2 (February 2, 2021): 1–9. http://dx.doi.org/10.12968/hmed.2020.0584.

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Atrial fibrillation is the most frequently occurring supraventricular arrhythmia in patients presenting with acute myocardial infarction. It is associated with worse outcomes when it coexists with acute myocardial infarction and results in increased morbidity and mortality. Both conditions are closely related to each other and share similar pathophysiological pathways. The management of atrial fibrillation in patients with acute myocardial infarction is challenging since triple antithrombotic therapy is indicated, but this results in a markedly increased risk of bleeding events and mortality. This review addresses the interactions between both conditions including common risk factors, possible mechanisms through which acute myocardial infarction contributes to development of atrial fibrillation and vice versa, and the problem of using anticoagulation in the management of these patients.

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15

Tatarsky, B. A., N. V. Kazennova, and D. A. Napalkov. "The Risk of Myocardial Infarction in Patients with Atrial Fibrillation Taking a Direct Thrombin Inhibitor: Myths and Reality." Rational Pharmacotherapy in Cardiology 16, no. 2 (May 2, 2020): 301–6. http://dx.doi.org/10.20996/1819-6446-2020-04-17.

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The purpose of this review is to analyze the results of randomized clinical trials, meta-analyses of cohort and observational studies in real clinical practice on the influence of dabigatran etexilate on the risk of myocardial infarction in patients with atrial fibrillation. A pivotal RE-LY study on dabigatran use in patients with atrial fibrillation did not show statistically significant differences in the frequency of myocardial infarction between any of the doses of dabigatran and warfarin, and the risk of coronary events did not depend on the presence of coronary heart disease or myocardial infarction in the patient's history. Subsequently, a number of meta-analyses have reported an increased risk of myocardial infarction when dabigatran was administered to patients with atrial fibrillation. In general, these studies were characterized by conflicting data, which did not allow to draw any definite conclusions regarding the use of dabigatran in relation to the risk of myocardial infarction. Two FDA cohort observational studies were published in 2014 and 2017, and the former was significantly criticized by experts, and the results of the second study did not provide a definitive answer to the question about the importance of the effect of dabigatran on the development of myocardial infarction in patients with atrial fibrillation. Even more "confusing" the problem arose after the publication of meta-analyses of randomized trials, which showed that the risk of myocardial infarction was increased in patients treated with direct oral anticoagulants compared to patients treated with warfarin. This review provides high quality evidence for the efficacy of dabigatran in preventing myocardial infarction and other vascular complications in patients with atrial fibrillation.

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16

Neobutov, N. N., and S. V. Kolbasnikov. "State of myocardium, stiffness of vascular wall and severity of emotional disorders in patients with myocardial infarction with atrial fibrillation." Medical alphabet, no. 7 (June 16, 2020): 45–48. http://dx.doi.org/10.33667/2078-5631-2020-7-45-48.

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The widespread prevalence of myocardial infarction and the frequency of complications in the form of atrial fibrillation, makes it relevant to study its pathogenesis and effect on hemodynamics, significantly aggravating the patient’s condition, leading to a deterioration in hemodynamic parameters and the appearance of recurrent myocardial ischemia. At present, a large amount of data has been accumulated on the causes, electrophysiological mechanisms, and hemodynamic consequences of the development of atrial fibrillation during myocardial infarction. This circumstance necessitates both further study of the features of the development and course of atrial fibrillation, and the search for new approaches in organizing and optimizing the treatment of this complication. It is necessary to study in detail the structure of anxiety-depressive disorders, the state of the elastotonic properties of the vascular wall, endothelial function and heart remodeling in patients with myocardial infarction with various forms of atrial fibrillation.

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17

Abdelmaseih, Ramy. "A Rare Case of Evolocumab Induced Atrial Fibrillation." Clinical Cardiology and Cardiovascular Interventions 04, no. 10 (March 19, 2021): 01. http://dx.doi.org/10.31579/2641-0419/160.

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Evolocumab, a proprotein convertase subtilisin/kexin type 9 inhibitor (PCSK9i) is a novel low-density lipoprotein (LDL) lowering agent that has been recently approved by the FDA to reduce the risk of myocardial infarction, stroke, and coronary revascularization in individuals with established atherosclerotic cardiovascular disease, alone or in combination with other lipid-lowering agents, and for treatment of patients with primary hyperlipidemia including familial hypercholesterolemia

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18

Lorem, Geir Fagerjord, Ida Marie Opdal, Tom Wilsgaard, Henrik Schirmer, Maja-Lisa Løchen, Ingrid Petrikke Olsen, Terje Steigen, and Kamilla Rognmo. "Assessment of mental health trajectories before and after myocardial infarction, atrial fibrillation or stroke: analysis of a cohort study in Tromsø, Norway (Tromsø Study, 1994–2016)." BMJ Open 12, no. 4 (April 2022): e052948. http://dx.doi.org/10.1136/bmjopen-2021-052948.

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ObjectivesThe increased survival rate of cardiovascular disease (CVD) implies a higher proportion of individuals who live with CVD. Using data from the Tromsø Study, we aimed to investigate mental health symptom trajectories before and after myocardial infarction, atrial fibrillation or stroke in a general population and to explore factors that contribute to the association.DesignCohort study.SettingSample drawn from inhabitants of the municipality of Tromsø, Norway, who participated in the Tromsø Study (1994–2016).ParticipantsA total of 18 719 participants (52.3% women) were included, and of these 2098 (32.9% women) were diagnosed with myocardial infarction, 1896 (41.9% women) with atrial fibrillation and 1263 (42.9% women) with stroke.Primary outcome measuresMental health symptoms were assessed using the Hopkins Symptom Checklist-10 and the Conor Mental Health Index.ResultsThe participants who were diagnosed with either myocardial infarction or stroke had a significant monotonous increase in mental health symptoms before myocardial infarction (p=0.029) and stroke (p=0.029) that intensified at the time of diagnosis. After the event, the study found a higher prevalence of mental health symptoms with a decline in symptom levels over time for myocardial infarction (p<0.001) and stroke (p=0.004), but not for atrial fibrillation (before: p=0.180, after: p=0.410). The risk of elevated mental health symptoms with myocardial infarction, atrial fibrillation and stroke was associated with sex (p<0.001), age (p<0.01), physical activity (p<0.001), diabetes (p<0.05) and other comorbidities (p<0.001).ConclusionThe study indicates that mental health problems among individuals with myocardial infarction, atrial fibrillation and stroke may have started to develop several years before the cardiovascular event and suggests that successful CVD rehabilitation may need to consider previous life factors. Future research is recommended to examine whether health promotion measures in a general population also create mental health resilience after a CVD event.

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Gursul, Erdal, Serdar Bayata, Ercan Aksit, and Basak Ugurlu. "Development of ST Elevation Myocardial Infarction and Atrial Fibrillation after an Electrical Injury." Case Reports in Emergency Medicine 2015 (2015): 1–3. http://dx.doi.org/10.1155/2015/953102.

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Electrical energy is a type of energy that is commonly used in daily life. Ventricular premature beats, ventricular tachycardia, ventricular fibrillation, atrial tachycardia, atrial fibrillation, bundle branch blocks, and AV block are arrhythmic complications that are encountered in case of electric shocks. Myocardial infarction is one of the rarely seen complications of electric shocks yet it has fatal outcomes. Coronary arteries were detected to be normal in most of the patients who had myocardial infarction following an electric shock. So, etiology of myocardial infarction is thought to be unrelated to coronary atherosclerosis in these cases. Coronary artery vasospasm is thought to be the primary etiological cause. In our case report, we presented a patient who developed ST elevation MI with atrial fibrillation after an electric shock.

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20

Leonova, I., S. Boldueva, M. Ryzhikova, and D. Oblavatsky. "ATRIAL FIBRILLATION AMONG PATIENTS WITH MYOCARDIAL INFARCTION." Journal of Hypertension 37 (July 2019): e185. http://dx.doi.org/10.1097/01.hjh.0000572384.47522.33.

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21

Geeren, M., T. Pop, Th Noll, R. Erbel, N. Treese, A. Faure, and J. Meyer. "Atrial fibrillation in acute-phase myocardial infarction." Journal of Electrocardiology 22, no. 3 (July 1989): 265. http://dx.doi.org/10.1016/0022-0736(89)90046-0.

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22

Jabre, Patricia, Xavier Jouven, Frédéric Adnet, Gabriel Thabut, Suzette J. Bielinski, Susan A. Weston, and Véronique L. Roger. "Atrial Fibrillation and Death After Myocardial Infarction." Circulation 123, no. 19 (May 17, 2011): 2094–100. http://dx.doi.org/10.1161/circulationaha.110.990192.

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23

Lubitz, Steven A., Jared W. Magnani, Patrick T. Ellinor, and Emelia J. Benjamin. "Atrial Fibrillation and Death After Myocardial Infarction." Circulation 123, no. 19 (May 17, 2011): 2063–65. http://dx.doi.org/10.1161/circulationaha.111.030171.

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24

Somasundaram, K., and J. Ball. "Medical emergencies: atrial fibrillation and myocardial infarction." Anaesthesia 68 (December 4, 2012): 84–101. http://dx.doi.org/10.1111/anae.12050.

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25

Asanin, M., M. Matic, J. Perunicic, P. Mitrovic, and Z. Vasiljevic. "Atrial fibrillation/-flutter following acute myocardial infarction." European Journal of Heart Failure 2 (June 2000): 35. http://dx.doi.org/10.1016/s1388-9842(00)80126-9.

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26

Ahamed H.B., Irfan, Bilal Bin Abdullah, Mohammed Ismail, and Syed Aman Jagirdar. "Association of comorbidities in atrial fibrillation in acute myocardial infarction." International Journal of Advances in Medicine 4, no. 1 (January 23, 2017): 143. http://dx.doi.org/10.18203/2349-3933.ijam20170098.

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Background: Atrial fibrillation is a most common arrhythmia in patients with and without structural heart disease with an increasing incidence mainly due to the aging population. As the population ages, one can expect that AF will remain a frequent and troublesome complication of AMI with comorbidities. Therefore, the present study was undertaken to investigate the association of co-morbidities in atrial fibrillation in acute myocardial infarction.Methods: The study was performed after the institutional ethical clearance and consent from all the patients. Heart rate, atrial fibrillation, blood pressure, ventricular fibrillation and stroke after acute myocardial infarction were recorded. The blood sugar and serum lipid levels were also measured using commercially available kit as per the manufacturer’s guidelines. The data was analyzed for statistical significance using univariate analysis and comparison was performed by Fisher Exact test and by using SPSS Version 20.Results: In our study, all the patients who developed AF after AMI were more than 60 years of age. Higher heart rate was more than or equal to 100 in 62.5% of the patients. 25% of patients had diabetes mellitus 75% of 8 patients had atrial fibrillation after acute myocardial infarction, 77% of patients without atrial fibrillation were known hypertensive’s. Out of the patients who had atrial fibrillation, 50% had hyperlipidemia and developed ventricular fibrillation.Conclusions: Higher heart rate (>100 bpm) at time of presentation is a risk factor for AF following to AMI. Patients with new onset AF after AMI had more complication during hospital stay.

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MacKenzie, Ross. "Infarction or Pseudo-infarction?" Journal of Insurance Medicine 47, no. 1 (January 1, 2017): 50–54. http://dx.doi.org/10.17849/insm-47-01-50-54.1.

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An applicant with a history of paroxysmal atrial fibrillation is found to have QS waves in leads III and AVF suggestive of a prior inferior wall myocardial infarction. Using the relationship between Q wave and T wave vectors in the inferior leads, an alternative explanation is explored.

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Neobutov, N. N., and S. V. Kolbasnikov. "Structure of risk factors, comorbid conditions and emotional disorders in patients with myocardial infarction with atrial fibrillation." Meditsinskiy sovet = Medical Council, no. 11 (August 8, 2020): 26–31. http://dx.doi.org/10.21518/2079-701x-2020-11-26-31.

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Introduction: the majority of patients with cardiovascular diseases in real medical practice are characterized by a combination of two or more diseases and conditions, that is, comorbidity. This places additional demands on the observation and treatment of this category of patients. Objective: to assess risk factors, the structure of comorbid conditions and the severity of psychoemotional disorders in patients with myocardial infarction with various forms of atrial fibrillation. Materials and methods: 138 patients (63 men, 75 women) who were hospitalized in the cardiology department of the City Clinical Hospital No. 7 GBUZ with a diagnosis of myocardial infarction with atrial fibrillation were examined. Depending on the form of atrial fibrillation, the patients were divided into 2 groups: the 1st consisted of 83 (60,1%) patients with a paroxysmal form, the 2nd group – 55 (39,9%) with a constant form. The examination was carried out in the first three days of hospitalization of patients. Results: in patients with myocardial infarction with a paroxysmal form of atrial fibrillation, the structure of risk factors is dominated by an average degree of nicotine addiction, dangerous alcohol consumption, high salt intake and excess body weight, and with a constant form of atrial fibrillation, a mostly weak and high degree of nicotine addiction, harmful alcohol consumption and obesity. Conclusion: in patients with myocardial infarction with a paroxysmal form of atrial fibrillation, the predominant risk factors are: dangerous alcohol consumption, high salt intake and overweight combined with depressive disorders in the form of subclinical and clinical depression, and with a constant form of atrial fibrillation, a high degree of nicotinic dependence and mild anxiety disorders: subclinically and clinically severe anxiety, which must be taken into account when carrying out preventive, rehabilitation measures and the solution of expert issues.

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Al-Obaidi, F., T. Al-Kinani, M. H. Al-Ali, and M. H. Al-Myahi. "New-Onset Atrial Fibrillation in the Post-Primary PCI Setting: A Systematic Review." Acta Medica Bulgarica 46, no. 2 (May 1, 2019): 62–70. http://dx.doi.org/10.2478/amb-2019-0021.

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Abstract Background: Atrial fibrillation is a relatively common complication of acute myocardial infarction with significant impact on the short and long-term prognosis. Methods: A systematic literature review was done through Pubmed and CENTRAL to extract data related to new-onset atrial fibrillation following primary PCI. Results: Searching resulted in twenty-one matched studies. Extraction of data showed an incidence rate of new-onset atrial fibrillation (2.8%-58%). A negative impact was found on the outcomes of patients treated with primary PCI with increased short and long-term mortality and morbidity. Conclusion: New-onset atrial fibrillation is an adverse prognostic marker in patients with acute myocardial infarction treated invasively. Preventive measures and anticoagulant therapy should be considered more intensively in this subset of patients.

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Neverauskaitė-Piliponienė, Gintarė, Rasa Kūgienė, Žaneta Petrulionienė, and Pranas Šerpytis. "Retrospective analysis of complications and survival in patients with acute inferior myocardial infarction accompanied by right ventricular myocardial infarction." Seminars in Cardiovascular Medicine 25, no. 1 (January 1, 2019): 9–13. http://dx.doi.org/10.2478/semcard-2019-0003.

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Summary Right ventricular myocardial infarction (RVMI) accompanies about 30–50% of inferior wall myocardial infarction. RVMI is associated with higher rates of cardiogenic shock, atrioventricular block, atrial fibrillation, increased mortality rates. The topic requires a scientific update, as only a few studies have been made on RVMI during the past decade. We aimed to analyse the impact of RVMI on inferior myocardial infarction. Design and methods: Retrospective study included 310 patients with documented inferior myocardial infarction (with and without RVMI) between January 2013 and January 2014. Data on baseline characteristics, mortality, in-hospital complications: cardiogenic shock and rhythm and conduction disorders was collected. Results: In 102 (32.9%) patients with inferior myocardial infarction, RVMI was present and 208 (67.1%) cases were without RVMI involvement. RVMI patients had higher rate of rhythm and conduction disturbances than patients without RVMI involvement: atrioventricular block (OR 3.8, 95% CI 2.0–7.1, p < 0.001), atrial fibrillation (OR 1.6, 95% CI 0.9–2.9, p = 0.001), also higher incidence of cardiogenic shock (OR 2.6, 95% CI 1.7–3.9, p < 0.001). Mortality rates after 24 months were higher in RVMI group (OR 1.8, 95% CI 1.2–3.8, p = 0.034). No significant difference was found on in-hospital mortality. Conclusions: Right ventricular involvement complicates the long-term mortality and outcomes after inferior myocardial infarction. It is related to a higher incidence of in-hospital complications, especially I–III degree AV block and atrial fibrillation. However, influence on long-term mortality needs further investigation.

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Fang, Dingfeng, Jiabin Xu, Bo Bai, and Haibo Chen. "Acute myocardial infarction occurring after hypertrophic obstructive cardiomyopathy with paroxysmal atrial fibrillation: a case report." Journal of International Medical Research 50, no. 10 (October 2022): 030006052211337. http://dx.doi.org/10.1177/03000605221133702.

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Atheromatous plaque rupture and coronary artery stenosis/occlusion are leading causes of acute myocardial infarction (AMI). Other reasons for AMI are frequently overlooked. We report a rare case of AMI occurring after hypertrophic obstructive cardiomyopathy with paroxysmal atrial fibrillation. Although the patient showed multiple lead ST-T changes on an electrocardiogram and elevated circulating cardiac troponin I concentrations, no stenotic lesions were observed by repeated invasive coronary angiography examinations. Hypertrophic obstructive cardiomyopathy with paroxysmal atrial fibrillation was suspected as the primary cause of AMI. The patient received radiofrequency catheter ablation therapy and was free of atrial fibrillation and myocardial infarction 1 year postoperatively. The clinical management and outcome of the patient are also discussed.

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Vukmirović, Mihailo, Aneta Bošković, Irena Tomašević Vukmirović, Radoje Vujadinovic, Nikola Fatić, Zoran Bukumirić, and Filip Vukmirović. "Predictions and outcomes of atrial fibrillation in the patients with acute myocardial infarction." Open Medicine 12, no. 1 (May 2, 2017): 115–24. http://dx.doi.org/10.1515/med-2017-0018.

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AbstractThe large epidemiological studies demonstrated that atrial fibrillation is correlated with high mortality and adverse events in patients with acute myocardial infarction. The aim of this study was to determinate predictors of atrial fibrillation develop during the hospital period in patients with acute myocardial infarction as well as short- and long-term mortality depending on the atrial fibrillation presentation. The 600 patients with an acute myocardial infarction were included in the study and follow-up 84 months. Atrial fibrillation develops during the hospital period was registered in 48 patients (8%). After adjustment by logistic regression model the strongest predictor of atrial fibrillation develop during the hospital period was older age, particularly more than 70 years (odds ratio 2.37, CI 1.23-4.58, p=0.010), followed by increased of Body Mass Index (odds ratio 1.17, CI 1.04-1.33, p=0.012), enlarged diameter of left atrium (LA) (odds ratio 1,18, CI 1,03-1,33, p=0,015) presentation of mitral regurgitation (odds ratio 3.56, CI 1.25-10.32, p=0.018) and B-type natriuretic peptide (odds ratio 2.12, CI 1.24-3.33, p=0.048).Patients with atrial fibrillation develop during the hospital period had a higher mortality during the hospital course (10.4% vs. 5.6%) p=0.179. as well as follow-up period of 84 months than patients without it (64.6% vs. 39.1%) p=0.569, than patients without it, but without statistically significance. Patients with AF develop during the hospital period had higher mortality during the hospital course as well as follow up period of 84 months than patients without it, but without statistically significance.

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McDonald, Mark, Nirav Desai, Chi-Hyan You, and L. Julian Haywood. "Influence of atrial fibrillation on acute myocardial infarction." Health 06, no. 01 (2014): 86–89. http://dx.doi.org/10.4236/health.2014.61013.

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34

Aronson, Doron. "Clinical significance of atrial fibrillation after myocardial infarction." Expert Review of Cardiovascular Therapy 9, no. 9 (September 2011): 1111–13. http://dx.doi.org/10.1586/erc.11.101.

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35

Soliman, Elsayed Z., Monika M. Safford, Paul Muntner, Yulia Khodneva, Farah Z. Dawood, Neil A. Zakai, Elan L. Thacker, et al. "Atrial Fibrillation and the Risk of Myocardial Infarction." Survey of Anesthesiology 59, no. 2 (April 2015): 105. http://dx.doi.org/10.1097/sa.0000000000000127.

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36

Tilling, L., and B. Clapp. "Atrial fibrillation in myocardial infarction: predictors and prognosis." International Journal of Clinical Practice 63, no. 5 (May 2009): 680–82. http://dx.doi.org/10.1111/j.1742-1241.2009.02061.x.

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Halperin, Jonathan L. "Preventing Myocardial Infarction in Patients With Atrial Fibrillation." Journal of the American College of Cardiology 69, no. 24 (June 2017): 2910–12. http://dx.doi.org/10.1016/j.jacc.2017.05.006.

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Siu, Chung-Wah, Man-Hong Jim, Hee-Hwa Ho, Raymond Miu, Stephen W. L. Lee, Chu-Pak Lau, and Hung-Fat Tse. "Transient Atrial Fibrillation Complicating Acute Inferior Myocardial Infarction." Chest 132, no. 1 (July 2007): 44–49. http://dx.doi.org/10.1378/chest.06-2733.

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Saczynski, Jane S., David McManus, Zheng Zhou, Frederick Spencer, Jorge Yarzebski, Darleen Lessard, Joel M. Gore, and Robert J. Goldberg. "Trends in Atrial Fibrillation Complicating Acute Myocardial Infarction." American Journal of Cardiology 104, no. 2 (July 2009): 169–74. http://dx.doi.org/10.1016/j.amjcard.2009.03.011.

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40

Soliman, Elsayed Z., Monika M. Safford, Paul Muntner, Yulia Khodneva, Farah Z. Dawood, Neil A. Zakai, Evan L. Thacker, et al. "Atrial Fibrillation and the Risk of Myocardial Infarction." JAMA Internal Medicine 174, no. 1 (January 1, 2014): 107. http://dx.doi.org/10.1001/jamainternmed.2013.11912.

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41

Alasady, M., W. Abhayaratna, A. Brooks, L. Mackenzie, M. Worthley, D. Chew, and P. Sanders. "Atrial Ischemia Predicts Risk of Atrial Fibrillation in Acute Myocardial Infarction." Heart, Lung and Circulation 18 (2009): S140. http://dx.doi.org/10.1016/j.hlc.2009.05.317.

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Trujillo Garcia, Anival, Enrique Verdugo Castro, and Juan Manuel Palacios Rodriguez. "Acute Myocardial Infarction of the Left Main Coronary Artery Secondary to Embolism." Clinical Cardiology and Cardiovascular Interventions 5, no. 9 (December 5, 2022): 01–04. http://dx.doi.org/10.31579/2641-0419/282.

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There are few reported cases of left main artery embolism and is usually fatal, the importance of atrial fibrillation as a cause of acute myocardial infarction and the treatment these patients should be offered, this case highlights the challenges faced during the management of this rare condition due to lack of clear-cut guidelines describing an evidence-based approach [1]. We describe the case of of LMCA thrombus that presented as ST-elevation myocardial infarction (STEMI) in a patient with paroxysmal atrial fibrillation who was successfully treated with thrombus aspiration is presented. We highlight the importance of intravascular imaging to determine the underlying putative mechanism.

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Aksoy, Fatih, and Ali Bagcı. "Predictive value of ATRIA risk score for contrast-induced nephropathy after percutaneous coronary intervention for ST-segment elevation myocardial infarction." Revista da Associação Médica Brasileira 65, no. 11 (November 2019): 1384–90. http://dx.doi.org/10.1590/1806-9282.65.11.1384.

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SUMMARY BACKGROUND: The AnTicoagulation and Risk factors In Atrial fibrillation (ATRIA) risk score used to detect the thromboembolic and hemorrhagic risk in atrial fibrillation patients has been shown recently to predict poor clinical outcomes in patients with acute myocardial infarction (ACS), regardless of having atrial fibrillation (AF). We aimed to analyze the relationship between different risk scores and contrast-induced nephropathy (CIN) development in patients with ACS who underwent urgent percutaneous coronary intervention (PCI) and compare the predictive ability of the ATRIA risk score with the MEHRAN risk score. METHODS: We analyzed 429 patients having St-segment Elevation Myocardial Infarction (STEMI) who underwent urgent PCI between January 2016 and February 2017. Patients were divided into two groups: those with and those without CIN and both groups were compared according to clinical, laboratory, and demographic features, including the CHA2DS2-VASc and ATRIA risk score. Predictors of CIN were determined by multivariate regression analysis. Receiver operating characteristics (ROC) curve analysis was used to analyze the prognostic value of CHA2DS2-VASc and ATRIA risk score for CIN, following STEMI. RESULTS: Multivariate regression analysis showed that Athe TRIA risk score, Opaque/Creatinine Clearance ratio, and low left ventricular ejection fraction was an independent predictor of CIN. The C-statistics for the ATRIA risk score and CHA2DS2-VASC risk score were 0.66 and 0.64 (p<0.001, and p<0.001), respectively. A pair-wise comparison of ROC curves showed that both scores were not inferior to the MEHRAN score in predicting CIN. CONCLUSION: The ATRIA and CHA2DS2-VASC scoring systems were useful for detecting CIN following STEMI.

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Yanovskiy, K. G., and L. A. Ivanova. "Cardiac Autonomic Neuropathy as Predictor of Development of Atrial Fibrillation in Patients with Type 2 Diabetes Mellitus after Myocardial Infarction." NAUKA MOLODYKH (ERUDITIO JUVENIUM) 10, no. 3 (September 30, 2022): 265–76. http://dx.doi.org/10.23888/hmj2022103265-276.

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BACKGROUND: Type 2 diabetes mellitus is a widespread chronic disease having many factors that negatively affect the course of cardiovascular events. One of these factors is cardiac autonomic neuropathy. The possibility of assessing the effect of cardiac autonomic neuropathy on the risk of development of atrial fibrillation after myocardial infarction may help in choosing the management tactics for patients with myocardial infarction and type 2 diabetes mellitus. AIM: To evaluate the effect of cardiac autonomic neuropathy on the risk of development of atrial fibrillation in patients with past myocardial infarction. To develop a method for assessing the risk of atrial fibrillation in 12-month period in patients with type 2 diabetes mellitus. MATERIALS AND METHODS: The study involved 60 individuals with type 2 diabetes mellitus and 60 individuals with newly diagnosed type 2 diabetes hospitalized with anterior and anteroseptal myocardial infarction with Q wave. At the initial stage of the study, the formed groups were homogeneous in age, gender, and all general clinical research methods. The diagnosis of type 2 diabetes mellitus was made on the basis of anamnesis data and available medical documentation confirmed by examination of glycosylated hemoglobin and glycemic profile. The newly found diabetes mellitus was diagnosed based on the data of glycosylated hemoglobin > 6.5%, blood glucose > 11.1 mmol/l recorded two or more times, according to national clinical guidelines. On the 3rd day, in 3 and 12 months after hospitalization, a set of tests for detecting cardiac autonomic neuropathy was performed; the presence of atrial fibrillation, the dynamics of left ventricular hypertrophy were assessed by electrocardiography and echocardiography. Statistical processing of the obtained results was performed using Microsoft Office Excel 2019, SPSS Statistics Standart ver. 28 programs for Windows. In order to choose the method of analysis, the distribution was estimated using Kolmogorov–Smirnov test. Comparison of the groups was carried out using Mann–Whitney test. The risk of atrial fibrillation was assessed using logistic, multifactorial and ROC analysis. RESULTS: Prognostic models were formed to determine the risk of development of atrial fibrillation in patients with type 2 diabetes mellitus. The model for diabetes mellitus had determination coefficient (R2 = 0.7), Hosmer–Lemeshow criterion 0.83, and the area under the curve 0.93. The model for newly diagnosed diabetes mellitus had R2 coefficient 0.72, Hosmer–Lemeshow goodness-of-fit test 0.85, and the area under the curve 0.95. CONCLUSION: Use of the developed models based on the results of tests aimed at detecting cardiac autonomic neuropathy, permits to predict the development of atrial fibrillation in patients with existing and newly diagnosed type 2 diabetes mellitus in combination with diabetic polyneuropathy in 12-month period after anterior, anteroseptal myocardial infarction with high accuracy.

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Bhattarai, R., S. A. Sayganov, and E. V. Trofimova. "ATRIAL FIBRILLATION ASSOCIATED WITH MYOCARDIAL INFARCTION OF DIFFERENT LOCALIZATIONS." Rational Pharmacotherapy in Cardiology 11, no. 1 (January 1, 2015): 25–30. http://dx.doi.org/10.20996/1819-6446-2015-11-1-25-30.

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Kolodgie, Frank D., Renu Virmani, Aloke V. Finn, and Maria E. Romero. "Embolic Myocardial Infarction as a Consequence of Atrial Fibrillation." Circulation 132, no. 4 (July 28, 2015): 223–26. http://dx.doi.org/10.1161/circulationaha.115.017534.

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Bayturan, Ozgur, Rishi Puri, E. Murat Tuzcu, Mingyuan Shao, Kathy Wolski, Paul Schoenhagen, Samir Kapadia, Steven E. Nissen, Prashanthan Sanders, and Stephen J. Nicholls. "Atrial fibrillation, progression of coronary atherosclerosis and myocardial infarction." European Journal of Preventive Cardiology 24, no. 4 (November 12, 2016): 373–81. http://dx.doi.org/10.1177/2047487316679265.

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48

Bhatia, Gurbir S., and Gregory Y. H. Lip. "Atrial fibrillation post-myocardial infarction: Frequency, consequences, and management." Current Heart Failure Reports 1, no. 4 (December 2004): 149–55. http://dx.doi.org/10.1007/s11897-004-0002-y.

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van den Berg, N. W. E., and J. R. de Groot. "Myocardial infarction, atrial fibrillation and mortality: timing is everything." Netherlands Heart Journal 23, no. 9 (June 5, 2015): 428–29. http://dx.doi.org/10.1007/s12471-015-0710-9.

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Bertolet, B. D., J. A. Hill, R. A. Kerensky, and L. Belardinelli. "Myocardial infarction related atrial fibrillation: role of endogenous adenosine." Heart 78, no. 1 (July 1, 1997): 88–90. http://dx.doi.org/10.1136/hrt.78.1.88.

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