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Relevant bibliographies by topics / Atrial electrical remodeling

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Academic literature on the topic 'Atrial electrical remodeling'

Author: Grafiati

Published: 14 March 2023

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Journal articles on the topic "Atrial electrical remodeling"

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Liu, Lei, Jianqiang Geng, Hongwei Zhao, et al. "Valsartan Reduced Atrial Fibrillation Susceptibility by Inhibiting Atrial Parasympathetic Remodeling through MAPKs/Neurturin Pathway." Cellular Physiology and Biochemistry 36, no. 5 (2015): 2039–50. http://dx.doi.org/10.1159/000430171.

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Background/Aims: Angiotensin II receptor blockers (ARBs) have been proved to be effective in preventing atrial structural and electrical remodelinq in atrial fibrillation (AF). Previous studies have shown that parasympathetic remodeling plays an important role in AF. However, the effects of ARBs on atrial parasympathetic remodeling in AF and the underlying mechanisms are still unknown. Methods: Canines were divided into sham-operated, pacing and valsartan + pacing groups. Rats and HL-1 cardiomyocytes were divided into control, angiotensin II (Ang II) and Ang II + valsartan groups, respectively

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Goette, Andreas, Clegg Honeycutt, and Jonathan J. Langberg. "Electrical Remodeling in Atrial Fibrillation." Circulation 94, no. 11 (1996): 2968–74. http://dx.doi.org/10.1161/01.cir.94.11.2968.

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Fujiki, Akira. "Electrical remodeling in atrial fibrillation." Journal of Molecular and Cellular Cardiology 45, no. 4 (2008): S3—S4. http://dx.doi.org/10.1016/j.yjmcc.2008.09.600.

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Dobrev, Dobromir. "Electrical Remodeling in Atrial Fibrillation." Herz 31, no. 2 (2006): 108–12. http://dx.doi.org/10.1007/s00059-006-2787-9.

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Dun, Wen, and Penelope A. Boyden. "Aged atria: electrical remodeling conducive to atrial fibrillation." Journal of Interventional Cardiac Electrophysiology 25, no. 1 (2009): 9–18. http://dx.doi.org/10.1007/s10840-008-9358-3.

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Błaszczyk, Robert, and Mateusz Kłoda. "Genetic basis of atrial firbrillation – the role of microRNA." In a good rythm 3, no. 60 (2021): 18–23. http://dx.doi.org/10.5604/01.3001.0015.7297.

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Atrial fibrillation is the most common arrhythmia in adults. In addition to the well-known cardiovascular risk factors, the role of genetic factors in the pathogenesis of atrial fibrillation is emphasized. MicroRNAs are a group of small, endogenous, single-stranded, non-coding RNAs, 20-22 nucleotides long, whose task is to regulate gene expression at the post-transcriptional level. Changes in the expression of microRNAs in circulating blood and tissues lead to the development of cardiovascular diseases, including atrial fibrillation, leading to the remodeling of the heart muscle. Different ty

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Tamargo, Juan, and Eva Delpón. "Vagal Stimulation and Atrial Electrical Remodeling." Revista Española de Cardiología (English Edition) 62, no. 7 (2009): 729–32. http://dx.doi.org/10.1016/s1885-5857(09)72352-7.

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Everett, Thomas H., Emily E. Wilson, Sander Verheule, Jose M. Guerra, Scott Foreman, and Jeffrey E. Olgin. "Structural atrial remodeling alters the substrate and spatiotemporal organization of atrial fibrillation: a comparison in canine models of structural and electrical atrial remodeling." American Journal of Physiology-Heart and Circulatory Physiology 291, no. 6 (2006): H2911—H2923. http://dx.doi.org/10.1152/ajpheart.01128.2005.

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Several animal models of atrial fibrillation (AF) have been developed that demonstrate either atrial structural remodeling or atrial electrical remodeling, but the characteristics and spatiotemporal organization of the AF between the models have not been compared. Thirty-nine dogs were divided into five groups: rapid atrial pacing (RAP), chronic mitral regurgitation (MR), congestive heart failure (CHF), methylcholine (Meth), and control. Right and left atria (RA and LA, respectively) were simultaneously mapped during episodes of AF in each animal using high-density (240 electrodes) epicardial

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Pang, Helen, Ricardo Ronderos, Andrés Ricardo Pérez-Riera, Francisco Femenía, and Adrian Baranchuk. "Reverse atrial electrical remodeling: A systematic review." Cardiology Journal 18, no. 6 (2011): 625–31. http://dx.doi.org/10.5603/cj.2011.0025.

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Hou, Jian, Shaojie Huang, Yan Long, et al. "DACT2 regulates structural and electrical atrial remodeling in atrial fibrillation." Journal of Thoracic Disease 12, no. 5 (2020): 2039–48. http://dx.doi.org/10.21037/jtd-19-4206.

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Dissertations / Theses on the topic "Atrial electrical remodeling"

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Finizola, Francesco. "Clinical aspects and molecular mechanisms of inflammation-driven arrhythmic risk." Doctoral thesis, Università di Siena, 2020. http://hdl.handle.net/11365/1105687.

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Increasing evidence indicates systemic inflammation as a new potential cause of acquired LQTS and Torsade de Pointes and a strong predictor of Atrial Fibrillation, via cytokine-mediated changes in cardiomyocyte ion channels and in gap-junction. We hypothesised that systemic inflammation may represent a novel risk factor contributing to TdP development in the general population and can promotes atrial electric remodelling in-vivo, as a result of cytokine-mediated changes in connexins expression. For this reason, two population studies have been designed to confirm these hypotheses. In the firs

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Hall, Mark Charles Scott. "Effects of angiotensin receptor blockade on atrial electrical remodelling and the 'second factor' in a goat model of lone atrial fibrillation." Thesis, University of Manchester, 2007. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.489010.

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Akar, Joseph Gabriel. "Electrical, structural, and spatiotemporal remodeling in atrial fibrillation /." 2004. http://wwwlib.umi.com/dissertations/fullcit/3144623.

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PANG, HELEN WAI KIU. "Reverse Atrial Electrical Remodeling Induced by Continuous Positive Airway Pressure in Patients with Severe Obstructive Sleep Apnea." Thesis, 2011. http://hdl.handle.net/1974/6625.

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Background: Obstructive sleep apnea (OSA) has been associated with atrial enlargement in response to high arterial and pulmonary pressures and increased sympathetic tone. Continuous positive airway pressure (CPAP) is the gold standard treatment for OSA; its impact on atrial electrical remodeling has not been investigated however. Signal-averaged p-wave (SAPW) is a non-invasive quantitative method to determine p-wave duration, an accepted marker for atrial electrical remodeling. The objective was to determine whether CPAP induces reverse atrial electrical remodeling in patients with severe OSA.

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John, Bobby. "Electrical remodelling of the atria and pulmonary veins due to stretch in rheumatic mitral stenosis." 2008. http://hdl.handle.net/2440/59453.

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Atrial fibrillation is the most common sustained arrhythmia; however, its mechanism is not well understood. Several conditions such as valvular disease, heart failure, and hypertension predispose to atrial fibrillation. Identifying the electrophysiological substrate in these clinical conditions would yield insight into the mechanism of atrial fibrillation and aid in developing strategies to prevent or cure it. Rheumatic mitral stenosis is associated with high prevalence of atrial fibrillation. While atrial stretch itself may be adequate to explain the occurrence of atrial fibrillation in this

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Tsai, Chia-Ti, and 蔡佳醍. "Renin-angiotensin System and Cardiovascular Diseases- Focusing on Mechanisms of Structural and Electrical Remodelings in Atrial Fibrillation and Pharmacological Approach." Thesis, 2006. http://ndltd.ncl.edu.tw/handle/89003535747642363081.

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博士<br>國立臺灣大學<br>臨床醫學研究所<br>95<br>The aim of the present doctoral thesis is to investigate the detailed molecular mechanism by which angiotensin II (AngII) is involved in the pathegenesis of atrial fibrillation (AF). Recently, it has been shown that local atrial rennin-angiotensin system (RAS) is activated with increased tissue AngII level during AF. AngII activates the downstream mitogen-activated protein kinase pathway (MAPK) signaling pathways, resulting in atrial structural remodeling. AngII is also involved in atrial electrical remodeling, and it has also been shown that blockage of endoge

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Books on the topic "Atrial electrical remodeling"

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Billman, George E., ed. Remodeling of cardiac passive electrical properties and susceptibility to ventricular and atrial arrhythmias. Frontiers Media SA, 2015. http://dx.doi.org/10.3389/978-2-88919-647-0.

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Chauhan, Vijay S., Sanjiv M. Narayan, and Atul Verma, eds. Electrical and Structural Remodelling in Atrial Fibrillation: Phenotypes for Personalized Therapy. Frontiers Media SA, 2021. http://dx.doi.org/10.3389/978-2-88971-082-9.

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D’Andrea, Antonello, André La Gerche, and Christine Selton-Suty. Systemic disease and other conditions: athlete’s heart. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0055.

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The term ‘athlete’s heart’ refers to the structural, functional, and electrical adaptations that occur as a result of habitual exercise training. It is characterized by an increase of the internal chamber dimensions and wall thickness of both atria and ventricles. The athlete’s right ventricle also undergoes structural, functional, and electrical remodelling as a result of intense exercise training. Some research suggests that the haemodynamic stress of intense exercise is greater for the right heart and, as a result, right heart remodelling is slightly more profound when compared with the lef

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Book chapters on the topic "Atrial electrical remodeling"

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Wagoner, David R. Van. "Electrical and Structural Remodeling in Atrial Fibrillation." In Atrial Fibrillation. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_5.

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Disertori, M., and M. Marini. "Does Early Echocardiography-Guided Cardioversion of Atrial Fibrillation Prevent Electrical/Mechanical Remodeling of the Atria?" In Cardiac Arrhythmias 2001. Springer Milan, 2002. http://dx.doi.org/10.1007/978-88-470-2103-7_57.

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Yang, Qunhui, Qiufeng Lv, Man Feng, et al. "Taurine Prevents the Electrical Remodeling in Ach-CaCl2 Induced Atrial Fibrillation in Rats." In Advances in Experimental Medicine and Biology. Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-024-1079-2_64.

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Franz, Michael R. "Excitable Gap, Antiarrhythmic Actions, Electrical Remodeling: The Role Of MAP Recording in Atrial Fibrillation And Other Atrial Tachyarrhythmias." In Monophasic Action Potentials. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60851-3_9.

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Schotten, U., and M. A. Allessie. "Electrical and Mechanical Remodeling of the Atria: What Are the Underlying Mechanisms, the Time Course and the Clinical Relevance?" In Cardiac Arrhythmias 2001. Springer Milan, 2002. http://dx.doi.org/10.1007/978-88-470-2103-7_52.

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Van Wagoner, David R. "Electrical Remodeling and Chronic Atrial Fibrillation." In Cardiac Electrophysiology. Elsevier, 2004. http://dx.doi.org/10.1016/b0-7216-0323-8/50044-0.

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Van Wagoner, David R. "Mechanisms of atrial remodelling." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0498.

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The atria serve a combination of reservoir, sensor, and neuroendocrine roles that help the heart to adapt to variations in blood volume, heart rate, and ventricular filling. When stressed by high-rate activity or increased haemodynamic load (due to hypertension, valve disease, or heart failure), the atria respond with increased oxidant production (oxidative stress) that promotes transcriptional changes that reversibly remodel electromechanical activity, with shortened action potential duration and effective refractory period, slowed and heterogeneous conduction, and impaired contractility. When the stresses persistent, the atria undergo persistent structural changes including chamber dilatation and increased interstitial fibrosis. The combination of electrical and structural remodelling leads to increased risk and persistence of atrial fibrillation and stroke. Accumulation of dysfunctional proteins that are normally recycled by the proteasome may contribute to the susceptibility to development of atrial fibrillation. Changes in ion channel expression are most often associated with the development of persistent atrial fibrillation. While many atrial fibrillation therapies have focused on targeting of atrial ion channels, efforts to target atrial proteostasis may have promise as a therapeutic atrial fibrillation treatment or prevention strategy.

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Kalman, Jonathan M., and Gwilym M. Morris. "Sinus node disease: pathophysiology and natural history." In ESC CardioMed, edited by Giuseppe Boriani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0448.

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Sinus node disease is the commonest bradyarrhythmia, often presenting as syncope or exercise limitation and is an important reason for pacemaker implantation. It is usually idiopathic and a disease of ageing with a peak incidence in the seventh decade of life, but may develop secondary to other conditions including heart failure and chronic endurance exercise. The detailed pathophysiology of sinus node disease remains unknown, studies have found evidence of widespread atrial electrical remodelling, and contemporary research suggests that cellular electrical and fibrotic changes may be important mediators of this remodelling. There is an important association between sinus node disease and atrial fibrillation, and the two arrhythmias often coexist, but the nature of this interaction remains a source of debate. This chapter will summarize the current understanding of the natural history and pathophysiology of sinus node disease, with a focus on remodelling and including discussion of theories that may explain the development of coexistent atrial arrhythmia in these patients.

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Lee, Geoffrey, Prashanthan Sanders, Joseph B. Morton, and Jonathan M. Kalman. "Mechanically induced electrical remodelling in human atrium." In Cardiac Mechano-Electric Coupling and Arrhythmias. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199570164.003.0040.

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Conference papers on the topic "Atrial electrical remodeling"

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Bai, Jieyun, Yaosheng Lu, Andy C.Y. Lo, and Jichao Zhao. "PITX2 Overexpression Leads to Atrial Electrical Remodeling Linked to Atrial Fibrillation." In 2019 Computing in Cardiology Conference. Computing in Cardiology, 2019. http://dx.doi.org/10.22489/cinc.2019.002.

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Valinoti, Maddalena, Graziano Vito Lozupone, Paolo Sabbatani, Roberto Mantovan, Stefano Severi, and Cristiana Corsi. "Analysis of the electrical patterns and structural remodeling in atrial fibrillation." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7320012.

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Zhao, Na, Qince Li, Kuanquan Wang, et al. "Effect of Heart Failure:induced Electrical Remodeling on the Initiation of Atrial Arrhythmias." In 2016 Computing in Cardiology Conference. Computing in Cardiology, 2016. http://dx.doi.org/10.22489/cinc.2016.215-160.

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Kharche, Sanjay, and Henggui Zhang. "Simulating the effects of atrial fibrillation induced electrical remodeling: A comprehensive simulation study." In 2008 30th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2008. http://dx.doi.org/10.1109/iembs.2008.4649222.

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KOJIMA, JISHO, SHINICHI NIWANO, DAISUKE SATO, et al. "HETEROGENIC PROCESS OF THE APPEARANCE OF THE ATRIAL ELECTRICAL REMODELING IN CANINE RAPID STIMULATION MODEL." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0014.

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ASHIKAGA, KEIICHI, TAKAO KOBAYASHI, MASAOMI KIMURA, et al. "AMIODARONE NOT ONLY REVERSES ELECTRICAL REMODELING BUT SUPPRESSES MATRIX METALLOPROTEINASES 2 ACTIVITY IN CANINE PACING-INDUCED PERSISTENT ATRIAL FIBRILLATION MODEL." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0013.

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Zhang, H., J. J. Zhu, C. J. Garratt, and A. V. Holden. "Cellular modelling of electrical remodelling in two different models of human atrial myocytes." In Computers in Cardiology, 2003. IEEE, 2003. http://dx.doi.org/10.1109/cic.2003.1291272.

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Bai, Jieyun, Yaosheng Lu, Roshan Sharma, and Jichao Zhao. "In Silico Screening of the Key Electrical Remodelling Targets in Atrial Fibrillation-induced Sinoatrial Node Dysfunction." In 2019 Computing in Cardiology Conference. Computing in Cardiology, 2019. http://dx.doi.org/10.22489/cinc.2019.001.

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