Relevant bibliographies by topics / Atrial differentiation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Atrial differentiation'

Author: Grafiati
Published: 9 March 2023
Last updated: 31 July 2025

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Journal articles on the topic "Atrial differentiation"

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Mesquita, Fernanda C. P., Jacquelynn Morrissey, Po-Feng Lee, et al. "Cues from human atrial extracellular matrix enrich the atrial differentiation of human induced pluripotent stem cell-derived cardiomyocytes." Biomaterials Science 9, no. 10 (2021): 3737–49. http://dx.doi.org/10.1039/d0bm01686a.

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Decellularized extracellular matrix (dECM) from human atria preserves key native components that directed the cardiac differentiation of hiPSCs to an atrial-like phenotype, yielding a twofold increase of functional atrial-like cells.
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Rautaharju, Pentti M. "Differentiation of atrial flutter from atrial fibrillation." Journal of Electrocardiology 33, no. 2 (2000): 203. http://dx.doi.org/10.1016/s0022-0736(00)80078-3.

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Davies, M. J., J. Rode, N. Woolf, and D. M. Krikler. "NEUROENDOCRINE DIFFERENTIATION IN ATRIAL MYXOMAS." Lancet 330, no. 8562 (1987): 800. http://dx.doi.org/10.1016/s0140-6736(87)92533-5.

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Knight, Bradley P., Gregory F. Michaud, S. Adam Strickberger, and Fred Morady. "Electrocardiographic differentiation of atrial flutter from atrial fibrillation by physicians." Journal of Electrocardiology 32, no. 4 (1999): 315–19. http://dx.doi.org/10.1016/s0022-0736(99)90002-x.

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Muir, T. M., J. Hair, G. C. Inglis, J. W. Dow, G. B. Lindop, and B. J. Leckie. "Dexamethasone-induced differentiation of atrial myocytes in culture." American Journal of Physiology-Heart and Circulatory Physiology 263, no. 3 (1992): H722—H729. http://dx.doi.org/10.1152/ajpheart.1992.263.3.h722.

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Atrial and ventricular myocytes from fetal and newborn rats were cultured in medium supplemented with fetal or newborn calf serum with and without glucocorticoid. Myocyte morphology was examined by light and electron microscopy, and the amount of stored and secreted atrial natriuretic peptide (ANP) was measured. Without dexamethasone, neonatal atrial myocytes cultured for 7 days contained myofibrils organized into sarcomeres and numerous endocrine granules containing immunostainable ANP. Secretion of immunoreactive ANP reached a peak between days 7 and 9 of culture. Myocytes from fetal rats se
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Schwach, Verena, Carla Cofiño-Fabres, Simone A. ten Den та Robert Passier. "Improved Atrial Differentiation of Human Pluripotent Stem Cells by Activation of Retinoic Acid Receptor Alpha (RARα)". Journal of Personalized Medicine 12, № 4 (2022): 628. http://dx.doi.org/10.3390/jpm12040628.

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Human pluripotent stem cell (hPSC)-derived cardiomyocytes have proven valuable for modeling disease and as a drug screening platform. Here, we depict an optimized protocol for the directed differentiation of hPSCs toward cardiomyocytes with an atrial identity by modulating the retinoic acid signaling cascade in spin embryoid bodies. The crucial steps of the protocol, including hPSC maintenance, embryoid body (EB) differentiation, the induction of cardiac mesoderm, direction toward the atrial phenotype, as well as molecular and functional characterization of the cardiomyocytes, are described. A
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Yao, Yao, Amanda N. Marra, and Deborah Yelon. "Pathways Regulating Establishment and Maintenance of Cardiac Chamber Identity in Zebrafish." Journal of Cardiovascular Development and Disease 8, no. 2 (2021): 13. http://dx.doi.org/10.3390/jcdd8020013.

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The vertebrate heart is comprised of two types of chambers—ventricles and atria—that have unique morphological and physiological properties. Effective cardiac function depends upon the distinct characteristics of ventricular and atrial cardiomyocytes, raising interest in the genetic pathways that regulate chamber-specific traits. Chamber identity seems to be specified in the early embryo by signals that establish ventricular and atrial progenitor populations and trigger distinct differentiation pathways. Intriguingly, chamber-specific features appear to require active reinforcement, even after
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Wolke, Carmen, Elmer Antileo, and Uwe Lendeckel. "WNT signaling in atrial fibrillation." Experimental Biology and Medicine 246, no. 9 (2021): 1112–20. http://dx.doi.org/10.1177/1535370221994086.

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The Wnt signaling pathway regulates physiological processes such as cell proliferation and differentiation, cell fate decisions, and stem cell maintenance and, thus, plays essential roles in embryonic development, but also in adult tissue homeostasis and repair. The Wnt signaling pathway has been associated with heart development and repair and has been shown to be crucially involved in proliferation and differentiation of progenitor cells into cardiomyocytes. The investigation of the role of the Wnt signaling pathway and the regulation of its expression/activity in atrial fibrillation has onl
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Hotchkiss, Adam, Tiam Feridooni, Mark Baguma-Nibasheka, Kathleen McNeil, Sarita Chinni, and Kishore B. S. Pasumarthi. "Atrial natriuretic peptide inhibits cell cycle activity of embryonic cardiac progenitor cells via its NPRA receptor signaling axis." American Journal of Physiology-Cell Physiology 308, no. 7 (2015): C557—C569. http://dx.doi.org/10.1152/ajpcell.00323.2014.

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The biological effects of atrial natriuretic peptide (ANP) are mediated by natriuretic peptide receptors (NPRs), which can either activate guanylyl cyclase (NPRA and NPRB) or inhibit adenylyl cyclase (NPRC) to modulate intracellular cGMP or cAMP, respectively. During cardiac development, ANP serves as an early maker of differentiating atrial and ventricular chamber myocardium. As development proceeds, expression of ANP persists in the atria but declines in the ventricles. Currently, it is not known whether ANP is secreted or the ANP-NPR signaling system plays any active role in the developing
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Suffee, Nadine, Thomas Moore-Morris, Patrick Farahmand, et al. "Atrial natriuretic peptide regulates adipose tissue accumulation in adult atria." Proceedings of the National Academy of Sciences 114, no. 5 (2017): E771—E780. http://dx.doi.org/10.1073/pnas.1610968114.

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The abundance of epicardial adipose tissue (EAT) is associated with atrial fibrillation (AF), the most frequent cardiac arrhythmia. However, both the origin and the factors involved in EAT expansion are unknown. Here, we found that adult human atrial epicardial cells were highly adipogenic through an epithelial–mesenchymal transition both in vitro and in vivo. In a genetic lineage tracing the WT1CreERT2+/−RosatdT+/−mouse model subjected to a high-fat diet, adipocytes of atrial EAT derived from a subset of epicardial progenitors. Atrial myocardium secretome induces the adipogenic differentiatio
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Dissertations / Theses on the topic "Atrial differentiation"

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Jambi, Majed. "Differentiation of Human Atrial Myocytes from Endothelial Progenitor Cell-Derived Induced Pluripotent Stem Cells." Thèse, Université d'Ottawa / University of Ottawa, 2014. http://hdl.handle.net/10393/31158.

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Recent advances in cellular reprogramming have enabled the generation of embryoniclike cells from virtually any cell of the body. These inducible pluripotent stem cells (iPSCs) are capable of indefinite self-renewal while maintaining the ability to differentiate into all cell types. Nowhere will this technology have a greater impact than in the ability to generate disease and patient-specific cell lines. Here we explore the capacity of human iPSCs reprogrammed from peripheral blood endothelial progenitor cells lines to differentiate into atrial myocytes for the study of patient specific atrial
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GIANNETTI, FEDERICA. "HIPS AND MES AS A CELLULAR MODEL TO STUDY THE MECHANISMS BEHIND ARRHYTHMIAS." Doctoral thesis, Università degli Studi di Milano, 2022. http://hdl.handle.net/2434/903405.

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Mouse embryonic stem cells (mESc) and human induced pluripotent stem cells (hiPSc) are commonly exploited in research for their ability to potentially differentiate into any cell subtype. Regarding cardiac differentiation, today's existing protocols are well advanced in recapitulating and promoting cardiomyocytes (CMs) differentiation, making CMs derived from mESc and hiPSc a good in vitro model for analyzing heart pathologies. In this thesis, these models have been used to characterize and better comprehend the functional output caused by genetic alterations. In the first part, mESc-derived
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Huber, Adrian Thomas. "Multi-organ non-invasive tissue characterization of fibrosis, adipose tissue, edema and inflammation with magnetic resonance (MR) imaging : applications to myocardium, skeletal muscle and liver interactions Cardiac MR strain: a noninvasive biomarker of fibro-fatty remodeling of the left atrial myocardium Comparison of MR T1 and T2 mapping parameters to characterize myocardial and skeletal muscle involvement in systemic Idiopathic Inflammatory Myopathy (IIM) Non-invasive differentiation of acute viral myocarditis and idiopathic inflammatory myopathy with cardiac involvement using magnetic resonance imaging T1 and T2 mapping CT predicts liver fibrosis: Prospective evaluation of morphology- and attenuationbased quantitative scores in routine portal venous abdominal scans." Thesis, Sorbonne université, 2019. http://www.theses.fr/2019SORUS135.

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Cette thèse réalise une preuve de concept pour quantifier la déformation de l’oreillette gauche (OG) en IRM, ainsi que la relaxométrie IRM dans le myocarde, dans les muscles squelettiques et dans le foie. Grâce à l’interaction entre radiologues et ingénieurs, deux logiciels différents ont été développés, appliqués et validés pour l'analyse de la déformation myocardique multi-chambre et pour la cartographie quantitative du T1 multi-organes. La première publication a montré une forte corrélation de la déformation de l’OG, avec le degré de remplacement fibro-graisseux en histologie. Ce biomarqueu
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Baik, Hayeon. "Role of the SUMO pathway in Acute Myeloid Leukemias response to treatments." Thesis, Montpellier, 2017. http://www.theses.fr/2017MONTT016/document.

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Les thérapies de différenciation sont une alternative prometteuse aux drogues génotoxiques utilisées en chimiothérapie pour le traitement de nombreux cancers. En particulier, l’acide tout-trans rétinoïque (ATRA) est utilisé avec succès pour traiter la leucémie aiguë promyélocytaire, un sous-type des leucémies aiguës myéloïdes (LAM). Malheureusement, son efficacité clinique est limitée dans les autres sous-types des LAM. Cela est en particulier du à une répression épigénétique des gènes de réponse à l’ATRA. Les SUMO constituent une famille de modificateurs post-traductionnels apparentés à l’ubi
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Salvatico, Jose. "The Expression of MKRN1, an E3 Ubiquitin Ligase for Telomerase Reverse Transcriptase, Is Induced with Differentiation Therapy in Leukemia." Master's thesis, University of Central Florida, 2009. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/3744.

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Telomeres are important structural and functional components of chromosomes, serving to provide stability and enabling full replication of the chromosomes. However, a shortening of the telomeres occurs with each cell division that can be fixed by a polymerase activity provided by telomerase, preventing this loss which would otherwise eventually lead to chromosome end-to-end fusions, senescence and cell death. The telomerase activity is present in stem cells and germ line cells, but absent or barely noticeable in adult somatic cells. However, in approximately 80-90% of transformed somatic cells
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Kårehed, Karin. "Signal Transduction in Malignant Cells – Transformation, Activation and Differentiation." Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6346.

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<p>All aspects of cell life are regulated by signal transduction mechanisms. This thesis describes the regulatory roles of a few key signal transduction molecules involved in three major biological responses. The studied pathways include platelet derived growth factor (PDGF)-BB induced transformation of murine fibroblasts, interferon (IFN)-γ stimulated monocyte activation and all-trans retinoic acid (ATRA) induced myeloid differentiation. </p><p>We found that intact phosphoinositide 3OH-kinase (PI3K) activity is essential in the signaling pathway that leads to the morphological alterations an
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De, Felice Dario. "Retinoic acid is required for prostate luminal lineage differentiation and epithelial integrity via Foxa1 expression." Doctoral thesis, Università degli studi di Trento, 2021. http://hdl.handle.net/11572/324422.

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Retinoids are a class of compounds derived from the metabolism of vitamin A and β-carotene. Retinoid signaling has vital functions in both vertebrate and invertebrate embryogenesis such as the formation of body axes and the control of organogenesis. Genetic evidence suggests a role of retinoids in cell fate decision, maturation and homeostasis of the prostate epithelium. Knockout (KO) of the retinoic acid receptor gamma (RARG) gene in mice leads to growth deficits and male sterility due to squamous metaplasia and keratinization of the seminal vesicles and prostate. Noteworthy, synergistic anti
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Dimberg, Anna. "The Role of Stat1 in Retinoic Acid-induced Myelomonocytic Differentiation of Human Leukemia Cells." Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2002. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1669.

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<p>All-trans retinoic acid (ATRA), a biologically active metabolite of vitamin A, is a powerful inducer of terminal differentiation and growth arrest of several myeloid cell lines <i>in vitro</i>. Although the efficacy of ATRA as an anti-cancer drug has been demonstrated by the successful treatment of acute promyelocytic leukemia (APL), knowledge concerning the molecular mechanisms directing ATRA-induced differentiation and cell cycle arrest of myeloid cells is lacking. Our results show, for the first time, that the complex regulation of cell cycle proteins and myeloid-specific transcription f
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Hotchkiss, Adam Gordon. "The Effects of Calcium Channel Blockade and Atrial Natriuretic Peptide Signalling on Proliferation and Differentiation of Cardiac Progenitor Cells." 2013. http://hdl.handle.net/10222/36236.

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Cardiac progenitor cells (CPCs) are abundant in the embryonic heart and have hallmark features which include a rapid rate of cell division and the ability to differentiate into mature heart muscle cells (cardiomyocytes). Based on these features, CPCs are considered an attractive candidate cell type for transplantation therapies which aim to replenish the diseased heart muscle tissue (myocardium) with new muscle forming cells. A better understanding of how pharmacological drugs and endogenous hormones/signalling molecules modulate the balance between proliferation and differentiation of CPCs
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Li, I.-Ting, and 李伊婷. "The anti-inflammatory role of apoptotic ATRA-NB4 cells in the resolution of differentiation syndrome." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/12833815224409163853.

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碩士<br>國立陽明大學<br>生理學研究所<br>100<br>All- trans retinoic acid (ATRA) has been used successfully in the treatment of acute promyelocytic leukemia (APL). However, this treatment may complicate with differentiation syndrome (DS) in 30% of patients, which manifests with clinical picture of acute lung injury and massive infiltration of ATRA-treated APL (ATRA-APL) cells into alveolar spaces. It is not clear how these ATRA-APL cells were cleared during resolution phase of DS. Previous studies have reported that cross-talk between apoptotic ATRA-APL cells and alveolar macrophage is crucial during the re
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Books on the topic "Atrial differentiation"

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Jones, Michael, Norman Qureshi, and Kim Rajappan. Multifocal atrial tachycardia. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0113.

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Multifocal atrial tachycardia (MAT) is an atrial arrhythmia arising in the left or right atrium, or both, with multiple different P wave morphologies (at least three), with an atrial rate usually faster than 100 min−1. The atrial rhythm may be irregular; however, the defining difference between MAT and atrial fibrillation is the presence of a P wave prior to each QRS complex in MAT (but the absence of P waves in atrial fibrillation). MAT may be compared to sinus rhythm with very frequent polymorphic atrial ectopic beats, and in fact similar pathophysiologic mechanisms underlie both conditions;
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Banerjee, Ashis, and Clara Oliver. Cardiac emergencies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198786870.003.0009.

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Chest pain is a common presenting complaint for patients in the emergency department. This chapter focuses on the management and recent changes to non-ST-segment elevation myocardial infarction (NSTEMI) and STEMI pathways, in keeping with national guidance. Arrhythmia management including atrial fibrillation as well as the use of scoring systems as the CHADVASC score also commonly appears in the short-answer question (SAQ) paper, which is covered in this chapter in line with current NICE guidance. In addition, there is also a section on the diagnosis and differentiation on managing a patient w
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Campione, Marina, Amelia Aranega, and Diego Franco. Cardiac looping and laterality. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, et al. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0014.

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Dextral looping is a complex process which progresses concomitantly with cardiac chamber differentiation and ultimately leads to the final alignment of the cardiac regions. Generation of cardiac asymmetry is crucial to ensure the proper form and consequent function of the heart and thus is a highly regulated process. Molecular signals originate long before morphological asymmetry and therefore can direct it; a complex regulatory network has been characterized which invariably converges on the Tgf-β‎ signalling molecule Nodal and its downstream target, the homeobox transcription factor Pitx2. W
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Heidbuchel, Hein, Mattias Duytschaever, and Haran Burri. LAA or LSPV? Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198766377.003.0034.

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D’Andrea, Antonello, André La Gerche, and Christine Selton-Suty. Systemic disease and other conditions: athlete’s heart. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0055.

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The term ‘athlete’s heart’ refers to the structural, functional, and electrical adaptations that occur as a result of habitual exercise training. It is characterized by an increase of the internal chamber dimensions and wall thickness of both atria and ventricles. The athlete’s right ventricle also undergoes structural, functional, and electrical remodelling as a result of intense exercise training. Some research suggests that the haemodynamic stress of intense exercise is greater for the right heart and, as a result, right heart remodelling is slightly more profound when compared with the lef
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Karatasakis, G., and G. D. Athanassopoulos. Cardiomyopathies. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0019.

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Echocardiography is a key diagnostic method in the management of patients with cardiomyopathies.The main echocardiographic findings of hypertrophic cardiomyopathy are asymmetric hypertrophy of the septum, increased echogenicity of the myocardium, systolic anterior motion, turbulent left ventricular (LV) outflow tract blood flow, intracavitary gradient of dynamic nature, mid-systolic closure of the aortic valve and mitral regurgitation. The degree of hypertrophy and the magnitude of the obstruction have prognostic meaning. Echocardiography plays a fundamental role not only in diagnostic process
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Book chapters on the topic "Atrial differentiation"

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Degos, Laurent. "ATRA Therapy in Acute Promyelocytic Leukemia a Model for Differentiation Therapy." In Cancer Therapy. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84613-7_8.

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Nalliah, Chrishan, and Jonathan Kalman. "Focal Atrial Tachycardia and its Differentiation from Macroreentrant Atrial Tachycardia." In Practical Cardiac Electrophysiology. Jaypee Brothers Medical Publishers (P) Ltd., 2017. http://dx.doi.org/10.5005/jp/books/13028_22.

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Lip, Gregory Y. H. "Epidemiology of supraventricular tachycardias." In ESC CardioMed. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0476.

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The precise description of the epidemiology of supraventricular tachycardias is difficult as the published data often has poor differentiation between atrial fibrillation, atrial flutter, and other supraventricular arrhythmias. In contrast to the extensive epidemiology on atrial fibrillation, a specific focus on supraventricular tachycardia population epidemiology is sparse, especially in the general population (rather than observational cohorts from specialized centres).
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Rudya, A., A. Kibarskis, and V. Shlleikis. "Differentiation of atrial rhythms by differentially amplified ECG." In Electrocardiology ’87. De Gruyter, 1988. http://dx.doi.org/10.1515/9783112484289-116.

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Rudys, A., A. Kibarskis, and V. Shileikis. "Differentiation of atrial rhythms by differentially amplified ECG." In Electrocardiology ’87. De Gruyter, 1988. http://dx.doi.org/10.1515/9783112542385-116.

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Kelly, Robert G. "Cardiac embryogenesis." In ESC CardioMed, edited by Miguel Torres. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0004.

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The embryonic heart forms in anterior lateral splanchnic mesoderm and is derived from Mesp1-expressing progenitor cells. During embryonic folding, the earliest differentiating progenitor cells form the linear heart tube in the ventral midline. The heart tube extends in length and loops to the right as new myocardium is progressively added at the venous and arterial poles from multipotent second heart field cardiovascular progenitor cells in contiguous pharyngeal mesoderm. While the linear heart tube gives rise to the left ventricle, the right ventricle, outflow tract, and a large part of atria
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Kelly, Robert G. "Cardiac embryogenesis." In ESC CardioMed, edited by Miguel Torres. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0004_update_001.

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The embryonic heart forms in anterior lateral splanchnic mesoderm and is derived from Mesp1-expressing progenitor cells. During embryonic folding, the earliest differentiating progenitor cells form the linear heart tube in the ventral midline. The heart tube extends in length and loops to the right as new myocardium is progressively added at the venous and arterial poles from multipotent second heart field cardiovascular progenitor cells in contiguous pharyngeal mesoderm. While the linear heart tube gives rise to the left ventricle, the right ventricle, outflow tract, and a large part of atria
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Kelly, Robert G. "Cardiac embryogenesis." In ESC CardioMed, edited by Miguel Torres. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0004_update_002.

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The embryonic heart forms in anterior lateral splanchnic mesoderm and is derived from Mesp1-expressing progenitor cells. During embryonic folding, the earliest differentiating progenitor cells form the linear heart tube in the ventral midline. The heart tube extends in length and loops to the right as new myocardium is progressively added at the venous and arterial poles from multipotent second heart field cardiovascular progenitor cells in contiguous pharyngeal mesoderm. While the linear heart tube gives rise to the left ventricle, the right ventricle, outflow tract, and a large part of atria
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Tardos, Jonathan, Nawal Aamir, Dhaval Desai, Amanda Chajkowski, and Amit H. Patel. "Frozen Hearts: The Emerging Role of Cryoablation for Pulmonary Vein Isolation." In Atrial Fibrillation - Diagnosis and Management in the 21st Century [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.105885.

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The cornerstone for the modern treatment of paroxysmal atrial fibrillation (AF) is pulmonary vein isolation, also called an AF ablation. Various ablation technologies exist to accomplish this goal with specific advantages. This chapter will focus on the unique attributes of cryoablation for pulmonary vein isolation. Specifically, we will summarize the trial data and outcomes of cryoablation in patients with paroxysmal and persistent AF from the initial FDA approval studies to novel uses beyond the pulmonary veins. Readers will have an appreciation of the unique characteristics differentiating
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Sareban, Mahdi, and Josef Niebauer. "Ambulatory (24-hour Holter monitoring, event recorders) and signal-averaged ECG for arrhythmia registration in the athlete’s heart." In The ESC Textbook of Sports Cardiology, edited by Antonio Pelliccia, Hein Heidbuchel, Domenico Corrado, Mats Börjesson, and Sanjay Sharma. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198779742.003.0012.

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Systematic physical exercise leads to structural, functional, and electrical cardiovascular changes summarized in the term ‘athlete’s heart’. Arrhythmias that are common features in the resting ECG of otherwise healthy athletes may be an expression of the athlete’s heart, but on the other hand may be caused by underlying cardiac pathology, opening up a grey zone of diagnostic uncertainty. Differentiating adaptive changes from pathological cardiac conditions is of great clinical importance because some cardiomyopathies are leading causes of sudden cardiac death in athletes. In addition, there i
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Conference papers on the topic "Atrial differentiation"

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Chen, Darren, Dong Han, Luís R. Mercado-Díaz, Jihye Moon, and Ki H. Chon. "Smartwatch Photoplethysmogram-Based Atrial Fibrillation Detection with Premature Atrial and Ventricular Contraction Differentiation Using Densely Connected Convolutional Neural Networks." In 2024 IEEE 20th International Conference on Body Sensor Networks (BSN). IEEE, 2024. https://doi.org/10.1109/bsn63547.2024.10780734.

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Razzaq, Nauman, Shafa-at Ali Sheikh, Tahir Zaidi, Imran Akhtar, and Syed Hassaan Ahmed. "Automated Differentiation between Normal Sinus Rhythm, Atrial Tachycardia, Atrial Flutter and Atrial Fibrillation during Electrophysiology." In 2017 IEEE 17th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2017. http://dx.doi.org/10.1109/bibe.2017.00-43.

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Zaidi, Syed Hassan, Shafa-at Ali Sheikh, Imran Akhtar, and Tahir Zaidi. "Differentiation between Atrial Fibrillation and Atrial Flutter using 1D Poincare maps based on endocardial bipolar intracardiac electrograms extracted from the Right Atria." In 2016 13th International Bhurban Conference on Applied Sciences and Technology (IBCAST). IEEE, 2016. http://dx.doi.org/10.1109/ibcast.2016.7429858.

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Ugarte, Juan P., Catalina Tobón, and Andrés Hernández. "Potassium Currents Affect the Rotor Dynamics in a Fractional-order Model of Atrial Fibrillation." In 2023 International Conference on Fractional Differentiation and Its Applications (ICFDA). IEEE, 2023. http://dx.doi.org/10.1109/icfda58234.2023.10153219.

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Byram, Brett, Lauren Oliveri, Patrick Wolf, and Gregg Trahey. "Acute and chronic myocardial infarct differentiation using atrial kick induced strain (AKIS) imaging." In 2013 IEEE International Ultrasonics Symposium (IUS). IEEE, 2013. http://dx.doi.org/10.1109/ultsym.2013.0027.

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Buś, Szymon, Konrad Jędrzejewski, and Przemysław Guzik. "Comparison of Effectiveness of Heart Rate Variability Indices in Differentiation of Atrial Fibrillation from Sinus Rhythm." In 2023 Signal Processing Symposium (SPSympo). IEEE, 2023. http://dx.doi.org/10.23919/spsympo57300.2023.10302689.

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Nopp, S., O. Königsbrügge, D. Kraemmer, I. Pabinger, and C. Ay. "Growth differentiation factor-15 predicts major cardiac adverse events and all-cause mortality in patients with atrial fibrillation." In 65th Annual Meeting of the Society of Thrombosis and Haemostasis Research. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728087.

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Yang, Guang, Xiahai Zhuang, Habib Khan, et al. "Differentiation of pre-ablation and post-ablation late gadolinium-enhanced cardiac MRI scans of longstanding persistent atrial fibrillation patients." In SPIE Medical Imaging, edited by Samuel G. Armato and Nicholas A. Petrick. SPIE, 2017. http://dx.doi.org/10.1117/12.2250910.

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Liu, Yingjuan, Mun-kit Choy, Gennadiy Tenin, Sabu Abraham, Graeme Black, and Bernard Keavney. "BS2 STX18-AS1 is a long noncoding rna governing in vitro cardiomyocyte differentiation and predisposing to atrial septal defect via downregulation of NKX2-5." In British Cardiovascular Society Annual Conference ‘Digital Health Revolution’ 3–5 June 2019. BMJ Publishing Group Ltd and British Cardiovascular Society, 2019. http://dx.doi.org/10.1136/heartjnl-2019-bcs.166.

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Maji, U., M. Mitra, and S. Pal. "Differentiating normal sinus rhythm and atrial fibrillation in ECG signal: A phase rectified signal averaging based approach." In 2014 International Conference on Control, Instrumentation, Energy and Communication (CIEC). IEEE, 2014. http://dx.doi.org/10.1109/ciec.2014.6959073.

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