Relevant bibliographies by topics / ASIC

Academic literature on the topic 'ASIC'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 July 2024

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Journal articles on the topic "ASIC"

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Dusenkova, Svetlana, Fei Ru, Lenka Surdenikova, Christina Nassenstein, Jozef Hatok, Robert Dusenka, Peter Banovcin, Jan Kliment, Milos Tatar, and Marian Kollarik. "The expression profile of acid-sensing ion channel (ASIC) subunits ASIC1a, ASIC1b, ASIC2a, ASIC2b, and ASIC3 in the esophageal vagal afferent nerve subtypes." American Journal of Physiology-Gastrointestinal and Liver Physiology 307, no. 9 (November 1, 2014): G922—G930. http://dx.doi.org/10.1152/ajpgi.00129.2014.

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Acid-sensing ion channels (ASICs) have been implicated in esophageal acid sensing and mechanotransduction. However, insufficient knowledge of ASIC subunit expression profile in esophageal afferent nerves hampers the understanding of their role. This knowledge is essential because ASIC subunits form heteromultimeric channels with distinct functional properties. We hypothesized that the esophageal putative nociceptive C-fiber nerves (transient receptor potential vanilloid 1, TRPV1-positive) express multiple ASIC subunits and that the ASIC expression profile differs between the nodose TRPV1-positive subtype developmentally derived from placodes and the jugular TRPV1-positive subtype derived from neural crest. We performed single cell RT-PCR on the vagal afferent neurons retrogradely labeled from the esophagus. In the guinea pig, nearly all (90%–95%) nodose and jugular esophageal TRPV1-positive neurons expressed ASICs, most often in a combination (65–75%). ASIC1, ASIC2, and ASIC3 were expressed in 65–75%, 55–70%, and 70%, respectively, of both nodose and jugular TRPV1-positive neurons. The ASIC1 splice variants ASIC1a and ASIC1b and the ASIC2 splice variant ASIC2b were similarly expressed in both nodose and jugular TRPV1-positive neurons. However, ASIC2a was found exclusively in the nodose neurons. In contrast to guinea pig, ASIC3 was almost absent from the mouse vagal esophageal TRPV1-positive neurons. However, ASIC3 was similarly expressed in the nonnociceptive TRPV1-negative (tension mechanoreceptors) neurons in both species. We conclude that the majority of esophageal vagal nociceptive neurons express multiple ASIC subunits. The placode-derived nodose neurons selectively express ASIC2a, known to substantially reduce acid sensitivity of ASIC heteromultimers. ASIC3 is expressed in the guinea pig but not in the mouse vagal esophageal TRPV1-positive neurons, indicating species differences in ASIC expression.
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Martínez-Barbero, Graciela, Yolanda García-Mesa, Ramón Cobo, Patricia Cuendias, Benjamín Martín-Biedma, Olivia García-Suárez, Jorge Feito, Teresa Cobo, and José A. Vega. "Acid-Sensing Ion Channels’ Immunoreactivity in Nerve Profiles and Glomus Cells of the Human Carotid Body." International Journal of Molecular Sciences 24, no. 24 (December 5, 2023): 17161. http://dx.doi.org/10.3390/ijms242417161.

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The carotid body is a major peripheral chemoreceptor that senses changes in arterial blood oxygen, carbon dioxide, and pH, which is important for the regulation of breathing and cardiovascular function. The mechanisms by which the carotid body senses O2 and CO2 are well known; conversely, the mechanisms by which it senses pH variations are almost unknown. Here, we used immunohistochemistry to investigate how the human carotid body contributes to the detection of acidosis, analyzing whether it expresses acid-sensing ion channels (ASICs) and determining whether these channels are in the chemosensory glomic cells or in the afferent nerves. In ASIC1, ASIC2, and ASIC3, and to a much lesser extent ASIC4, immunoreactivity was detected in subpopulations of type I glomus cells, as well as in the nerves of the carotid body. In addition, immunoreactivity was found for all ASIC subunits in the neurons of the petrosal and superior cervical sympathetic ganglia, where afferent and efferent neurons are located, respectively, innervating the carotid body. This study reports for the first time the occurrence of ASIC proteins in the human carotid body, demonstrating that they are present in glomus chemosensory cells (ASIC1 < ASIC2 > ASIC3 > ASIC4) and nerves, presumably in both the afferent and efferent neurons supplying the organ. These results suggest that the detection of acidosis by the carotid body can be mediated via the ASIC ion channels present in the type I glomus cells or directly via sensory nerve fibers.
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Storozhuk, Maksim, Andrii Cherninskyi, Oleksandr Maximyuk, Dmytro Isaev, and Oleg Krishtal. "Acid-Sensing Ion Channels: Focus on Physiological and Some Pathological Roles in the Brain." Current Neuropharmacology 19, no. 9 (September 14, 2021): 1570–89. http://dx.doi.org/10.2174/1570159x19666210125151824.

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Acid-sensing ion channels (ASICs) are Na+-permeable ion channels activated by protons and predominantly expressed in the nervous system. ASICs act as pH sensors leading to neuronal excitation. At least eight different ASIC subunits (including ASIC1a, ASIC1b, ASIC2a, ASIC2b, ASIC3, ASIC4, ASIC5) are encoded by five genes (ASIC1-ASIC5). Functional ASICs assembled in the plasma membrane are homo- or heteromeric trimers. ASIC1a-containing trimers are of particular interest as, in addition to sodium ions, they also conduct calcium ions and thus can trigger or regulate multiple cellular processes. ASICs are widely but differentially expressed in the central and peripheral nervous systems. In the mammalian brain, a majority of neurons express at least one ASIC subunit. Several recent reviews have summarized findings of the role of ASICs in the peripheral nervous system, particularly in nociception and proprioception, and the structure-function relationship of ASICs. However, there is little coverage on recent findings regarding the role of ASICs in the brain. Here we review and discuss evidence regarding the roles of ASICs: (i) as postsynaptic receptors activated by protons coreleased with glutamate at glutamatergic synapses; (ii) as modulators of synaptic transmission at glutamatergic synapses and GABAergic synapses; (iii) in synaptic plasticity, memory and learning; (iv) in some pathologies such as epilepsy, mood disorders and Alzheimer's disease.
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Jernigan, Nikki L., Michael L. Paffett, Benjimen R. Walker, and Thomas C. Resta. "ASIC1 contributes to pulmonary vascular smooth muscle store-operated Ca2+ entry." American Journal of Physiology-Lung Cellular and Molecular Physiology 297, no. 2 (August 2009): L271—L285. http://dx.doi.org/10.1152/ajplung.00020.2009.

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Acid-sensing ion channels (ASIC) are voltage-insensitive, cationic channels that have recently been identified in vascular smooth muscle (VSM). It is possible that ASIC contribute to vascular reactivity via Na+ and Ca2+ conductance; however, their function in VSM is largely unknown. In pulmonary VSM, store-operated Ca2+ entry (SOCE) plays a significant role in vasoregulatory mechanisms such as hypoxic pulmonary vasoconstriction and receptor-mediated arterial constriction. Therefore, we hypothesized that ASIC contribute to SOCE in pulmonary VSM. We examined SOCE resulting from depletion of intracellular Ca2+ stores with cyclopiazonic acid in isolated small pulmonary arteries and primary cultured pulmonary arterial smooth muscle cells by measuring 1) changes in VSM [Ca2+]i using fura-2 indicator dye, 2) Mn2+ quenching of fura-2 fluorescence, and 3) store-operated Ca2+ and Na+ currents using conventional whole cell patch-clamp configuration in voltage-clamp mode. The role of ASIC was assessed by the use of the ASIC inhibitors, amiloride, benzamil, and psalmotoxin 1, or siRNA directed towards ASIC1, ASIC2, or ASIC3 isoforms. We found that store-operated VSM [Ca2+]i responses, Mn2+ influx, and inward cationic currents were attenuated by either pharmacological ASIC inhibition or treatment with ASIC1 siRNA. These data establish a unique role for ASIC1 in mediating SOCE in pulmonary VSM and provide new insight into mechanisms of VSM Ca2+ entry and pulmonary vasoregulation.
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Hu, Zhuang-Li, Chao Huang, Hui Fu, You Jin, Wen-Ning Wu, Qiu-Ju Xiong, Na Xie, Li-Hong Long, Jian-Guo Chen, and Fang Wang. "Disruption of PICK1 attenuates the function of ASICs and PKC regulation of ASICs." American Journal of Physiology-Cell Physiology 299, no. 6 (December 2010): C1355—C1362. http://dx.doi.org/10.1152/ajpcell.00569.2009.

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Acid-sensing ion channels (ASICs) extensively exist in both central and peripheral neuronal systems and contribute to many physiological and pathological processes. The protein that interacts with C kinase 1 (PICK1) was cloned as one of the proteins interacting with protein kinase C (PKC) and colocalized with ASIC1 and ASIC2. Here, we used PICK1 knockout (PICK1-KO) C57/BL6 mice together with the whole cell patch clamp, calcium imaging, RT-PCR, Western blot, and immunocytochemistry techniques to explore the possible change in ASICs and the regulatory effects of PKC on ASICs. The results showed that PICK1 played a key role in regulation of ASIC functions. In PICK1-KO mouse cortical neurons, both the amplitude of ASIC currents and elevation of [Ca2+]i mediated by acid were decreased, which were attributable to the decreased expression of ASIC1a and ASIC2a proteins in the plasma membrane. PKC, a partner protein of PICK1, regulated ASIC functions via PICK1. The agonist and antagonist of PKC only altered ASIC currents and acid-induced increase in [Ca2+]i in wild-type, but not in KO mice. In conclusion, our data provided the direct evidence from PICK1-KO mice that a novel target protein, PICK1, would regulate ASIC function and membrane expression in the brain. In addition, PICK1 played the bridge role between PKC and ASICs.
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Dymowska, Agnieszka K., Aaron G. Schultz, Salvatore D. Blair, Danuta Chamot, and Greg G. Goss. "Acid-sensing ion channels are involved in epithelial Na+ uptake in the rainbow trout Oncorhynchus mykiss." American Journal of Physiology-Cell Physiology 307, no. 3 (August 1, 2014): C255—C265. http://dx.doi.org/10.1152/ajpcell.00398.2013.

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A role for acid-sensing ion channels (ASICs) to serve as epithelial channels for Na+ uptake by the gill of freshwater rainbow trout was investigated. We found that the ASIC inhibitors 4′,6-diamidino-2-phenylindole and diminazene decreased Na+ uptake in adult rainbow trout in a dose-dependent manner, with IC50 values of 0.12 and 0.96 μM, respectively. Furthermore, we cloned the trout ASIC1 and ASIC4 homologs and demonstrated that they are expressed differentially in the tissues of the rainbow trout, including gills and isolated mitochondrion-rich cells. Immunohistochemical analysis using custom-made anti-zASIC4.2 antibody and the Na+-K+-ATPase (α5-subunit) antibody demonstrated that the trout ASIC localizes to Na+/K+-ATPase-rich cells in the gill. Moreover, three-dimensional rendering of confocal micrographs demonstrated that ASIC is found in the apical region of mitochondrion-rich cells. We present a revised model whereby ASIC4 is proposed as one mechanism for Na+ uptake from dilute freshwater in the gill of rainbow trout.
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Xie, Jinghui, Margaret P. Price, John A. Wemmie, Candice C. Askwith, and Michael J. Welsh. "ASIC3 and ASIC1 Mediate FMRFamide-Related Peptide Enhancement of H+-Gated Currents in Cultured Dorsal Root Ganglion Neurons." Journal of Neurophysiology 89, no. 5 (May 1, 2003): 2459–65. http://dx.doi.org/10.1152/jn.00707.2002.

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The acid-sensing ion channels (ASICs) form cation channels that are transiently activated by extracellular protons. They are expressed in dorsal root ganglia (DRG) neurons and in the periphery where they play a function in nociception and mechanosensation. Previous studies showed that FMRFamide and related peptides potentiate H+-gated currents. To better understand this potentiation, we examined the effect of FMRFamide-related peptides on DRG neurons from wild-type mice and animals missing individual ASIC subunits. We found that FMRFamide and FRRFamide potentiated H+-gated currents of wild-type DRG in a dose-dependent manner. They increased current amplitude and slowed desensitization following a proton stimulus. Deletion of ASIC3 attenuated the response to FMRFamide-related peptides, whereas the loss of ASIC1 increased the response. The loss of ASIC2 had no effect on FMRFamide-dependent enhancement of H+-gated currents. These data suggest that FMRFamide-related peptides modulate DRG H+-gated currents through an effect on both ASIC1 and ASIC3 and that ASIC3 plays the major role. The recent discovery of RFamide-related peptides (RFRP) in mammals suggested that they might also modulate H+-gated current. We found that RFRP-1 slowed desensitization of H+-gated DRG currents, whereas RFRP-2 increased the peak amplitude. COS-7 cells heterologously expressing ASIC1 or ASIC3 showed similar effects. These results suggest that FMRFamide-related peptides, including the newly identified RFRPs, modulate H+-gated DRG currents through ASIC1 and ASIC3. The presence of several ASIC subunits, the diversity of FMRFamide-related peptides, and the distinct effects on H+-gated currents suggest the possibility of substantial complexity in modulation of current in DRG sensory neurons.
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Corrow, Kimberly, Beatrice M. Girard, and Margaret A. Vizzard. "Expression and response of acid-sensing ion channels in urinary bladder to cyclophosphamide-induced cystitis." American Journal of Physiology-Renal Physiology 298, no. 5 (May 2010): F1130—F1139. http://dx.doi.org/10.1152/ajprenal.00618.2009.

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The expression of acid-sensing ion channel (ASIC) isoforms, ASIC1, ASIC2a, and ASIC3, was examined in the urinary bladder after cyclophosphamide (CYP)-induced cystitis of varying duration (4 h, 48 h, and chronic). Immunohistochemical, Western blot, and quantitative PCR approaches were used to evaluate channel expression and effects of CYP-induced cystitis in whole urinary bladder and split-bladder preparations from control (no inflammation) and CYP-treated rats. Quantitative PCR demonstrated significant ( P ≤ 0.01) increases in ASIC2a and ASIC3 transcripts with CYP-induced cystitis (48 h and chronic) in the urothelium but no changes (e.g., ASIC3) or modest changes (e.g., ASIC2a) in detrusor smooth muscle. ASIC1 mRNA expression in the urothelium or detrusor was not affected by CYP-induced cystitis. Immunohistochemistry for ASIC2a and ASIC3 protein expression revealed significant ( P ≤ 0.01) increases in ASIC immunoreactivity in the urothelium and suburothelial plexus with CYP-induced cystitis at all time points examined. Western blotting for ASIC2a and ASIC3 protein expression was complementary and revealed significant ( P ≤ 0.01) increases in ASIC immunoreactivity. For the first time, these studies demonstrate that CYP-induced cystitis alters ASIC2a and ASIC3 expression in the urinary bladder; ASIC1 transcript expression is not altered by CYP-induced cystitis. Future studies are necessary to determine ASIC isoform contributions to micturition reflexes in control and inflamed urinary bladder.
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Lee, Ching-Yu, Tsung-Jen Huang, Meng-Huang Wu, Yen-Yao Li, and Kuan-Der Lee. "High Expression of Acid-Sensing Ion Channel 2 (ASIC2) in Bone Cells in Osteoporotic Vertebral Fractures." BioMed Research International 2019 (August 19, 2019): 1–10. http://dx.doi.org/10.1155/2019/4714279.

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Little is known about the function of acid-sensing ion channels (ASICs) in bone cells or osteoporotic vertebral fractures (OVF). This study delineated ASICs expression in adult human bone marrow-mesenchymal stem cells- (BM-MSC-) derived osteoblasts and in OVF bone cells. Adult BM-MSC-derived osteoblasts were isolated and cultured in different pH values. Osteogenic markers as alkaline phosphatase (ALP), osteopontin (OPN), and osteocalcin (OC) mRNA were assessed. Western blots method was applied to analyze ASICs protein expression in different pH values. Amiloride was added into the osteogenic media to analyze the Na+/K+ ATPase change. We harvested the vertebral cancellous bone through a bone biopsy needle in 26 OVF patients when performing percutaneous vertebroplasty. Six vertebral bone specimens obtained from 4 patients with high-energy vertebral fractures were used as the control. The reverse transcription polymerase chain reaction was performed to analyze the quantitative mRNA expression of ASICs. Osteogenic markers as ALP, OPN, and OC mRNA were higher expressed in increasing pH values throughout osteoblastogenesis. ASIC proteins were higher expressed in lower pH media, especially ASIC3, and ASIC4. The highest protein expression at days 7, 14, and 21 was ASIC2, ASIC4, and ASIC3, respectively. Expression of Na+/K+ ATPase was significantly decreased in cultured osteoblasts by addition of amiloride into the pH 6.9 osteogenic media. ASIC2 mRNA was most highly expressed with a 65.93-fold increase in the biopsied vertebral bone cells in OVF compared with the control. In conclusion, we found osteoblastogenesis was reduced in an acidic environment, and ASIC2, ASIC3, and ASIC4 were most highly expressed in turn during osteoblastogenesis within acidic media. ASIC2 was the most abundantly expressed gene in human bone cells in OVF compared with the control. ASIC2 could be crucial in the pathogenesis of osteoporosis and could serve as a therapeutic target for antiosteoporotic therapies.
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Kusama, Nobuyoshi, Mamta Gautam, Anne Marie S. Harding, Peter M. Snyder, and Christopher J. Benson. "Acid-sensing ion channels (ASICs) are differentially modulated by anions dependent on their subunit composition." American Journal of Physiology-Cell Physiology 304, no. 1 (January 1, 2013): C89—C101. http://dx.doi.org/10.1152/ajpcell.00216.2012.

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Acid-sensing ion channels (ASICs) are sodium channels gated by extracellular protons. ASIC1a channels possess intersubunit Cl−-binding sites in the extracellular domain, which are highly conserved between ASIC subunits. We previously found that anions modulate ASIC1a gating via these sites. Here we investigated the effect of anion substitution on native ASICs in rat sensory neurons and heterologously expressed ASIC2a and ASIC3 channels by whole cell patch clamp. Similar to ASIC1a, anions modulated the kinetics of desensitization of other ASIC channels. However, unlike ASIC1a, anions also modulated the pH dependence of activation. Moreover, the order of efficacy of different anions to modulate ASIC2a and -3 was very different from that of ASIC1a. More surprising, mutations of conserved residues that form an intersubunit Cl−-binding site in ASIC1a only partially abrogated the effects of anion modulation of ASIC2a and had no effect on anion modulation of ASIC3. The effects of anions on native ASICs in rat dorsal root ganglion neurons mimicked those in heterologously expressed ASIC1a/3 heteromeric channels. Our data show that anions modulate a variety of ASIC properties and are dependent on the subunit composition, and the mechanism of modulation for ASIC2a and -3 is distinct from that of ASIC1a. We speculate that modulation of ASIC gating by Cl− is a novel mechanism to sense shifts in extracellular fluid composition.
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Dissertations / Theses on the topic "ASIC"

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Lothian, Angus, Ivar Härnqvist, Adam Jakobsson, Arvid Westerlund, Felix Goding, Jacob Wahlman, Kevin Scott, and Rasmus Karlsson. "B-ASIC - Better ASIC Toolbox : En verktygslåda som förenklar design och optimering av ASIC." Thesis, Linköpings universitet, Institutionen för datavetenskap, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-167069.

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Denna rapport behandlar ett arbete skriven av åtta studenter som läste kursen TDDD96 Kandidatprojekt i programvaruutveckling vid Linköpings universitet under vårterminen 2020. Projektets syfte var att utveckla en verktygslåda i Python och C++ för att konstruera signalbehandlade kretsar. Denna verktygslåda är tänkt att användas inom laborationer i kursen TSTE87 Applikationsspecfika integrerande kretsar vid Linköpings universitet och inom forskning för utveckling av ASIC:s. Projektet resulterade i produkten B-ASIC. B-ASIC är ett bibliotek för programmeringsspråket Python som är skrivet i Python med en underliggande modul i C++. B-ASIC används för design och optimering av ASIC:s. Produkten B-ASIC erbjuder ett grafiskt användargränssnitt där användaren kan interagera med biblioteket utan programmeringskunskaper inom Python. I rapporten beskrivs hur projektarbetet har anpassats för att vara till värde för kunden och hur utvecklingsprocessen har påverkat resultatet av produkten. Projektmedlemmarna har dessutom genomfört egna undersökningar och dessa finns att läsa i slutet av rapporten.
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Arumugam, Prakash. "Investigations into ASIC desensitization." Connect to resource, 2006. http://hdl.handle.net/1811/6036.

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Thesis (Honors)--Ohio State University, 2006.
Title from first page of PDF file. Document formattted into pages: contains 16 p.; also includes graphics. Includes bibliographical references (p. 16). Available online via Ohio State University's Knowledge Bank.
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Ghuman, Parminder, Salman Sheikh, Steve Koubek, Scott Hoy, and Andrew Gray. "High Rate Digital Demodulator ASIC." International Foundation for Telemetering, 1998. http://hdl.handle.net/10150/609676.

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International Telemetering Conference Proceedings / October 26-29, 1998 / Town & Country Resort Hotel and Convention Center, San Diego, California
The architecture of the High Rate (600 Mega-bits per second) Digital Demodulator (HRDD) ASIC capable of demodulating BPSK and QPSK modulated data is presented in this paper. The advantages of all-digital processing include increased flexibility and reliability with reduced reproduction costs. Conventional serial digital processing would require high processing rates necessitating a hardware implementation other than CMOS technology such as Gallium Arsenide (GaAs) which has high cost and power requirements. It is more desirable to use CMOS technology with its lower power requirements and higher gate density. However, digital demodulation of high data rates in CMOS requires parallel algorithms to process the sampled data at a rate lower than the data rate. The parallel processing algorithms described here were developed jointly by NASA’s Goddard Space Flight Center (GSFC) and the Jet Propulsion Laboratory (JPL). The resulting all-digital receiver has the capability to demodulate BPSK, QPSK, OQPSK, and DQPSK at data rates in excess of 300 Mega-bits per second (Mbps) per channel. This paper will provide an overview of the parallel architecture and features of the HRDR ASIC. In addition, this paper will provide an overview of the implementation of the hardware architectures used to create flexibility over conventional high rate analog or hybrid receivers. This flexibility includes a wide range of data rates, modulation schemes, and operating environments. In conclusion it will be shown how this high rate digital demodulator can be used with an off-the-shelf A/D and a flexible analog front end, both of which are numerically computer controlled, to produce a very flexible, low cost high rate digital receiver.
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Ramsten, Johannes, and Markus Klum. "Implementation av fältbuss ASIC i FPGA." Thesis, Halmstad University, School of Information Science, Computer and Electrical Engineering (IDE), 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-4523.

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HMS Industrial Networks AB is in need of changing a communications solution that iscurrently based on an ASIC. This will be achieved by moving the communications solution toa FPGA with the help of the programming language VHDL. By doing this, it is possible toreduce the need for specific circuits, get a more flexible platform and thus get a cheapersolution.

This report describes a solution for how to move a network protocol from an ASIC to anFPGA. The report shows that the network slave device is working under the guidelines forthis project. This means that it is quite realistic to implement a fieldbus protocol on an FPGA,using VHDL and to maintain the same functionality as the earlier communications solution.

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Harrison, Andrew. "ASIC based recorders of electrophysiological signals." Thesis, University of Nottingham, 1995. http://eprints.nottingham.ac.uk/13305/.

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The ability of application specific integrated circuits (ASICs) to minimise the size and power consumption of electronic circuitry, makes their application to the design of ambulatory monitoring equipment, an attractive option. To this end, a multi-purpose mixed analogue and digital ASIC has been fabricated and incorporated into both a long-term recorder of adult heart rate (HR) and a recorder of electrophysiological signals. The adult HR recorder has been employed in a study of long-term daily HR patterns, which verified the ambulatory nature of this instrument, as well as its suitability for investigating HR variability. The electrophysiological signal recorder uses the ASIC to amplify, filter and digitise signals, which are then stored directly into static RAM. The analogue front-end of this instrument is flexible in terms of gain, bandwidth and sampling frequency allowing it be applied to a whole range of signals. This instrument has been used to record the antepartum fetal HR, as part of the development of an ambulatory, ASIC based recorder of fetal HR (FHR). These recordings have shown that a usable signal can be obtained from a mother in her home environment, whilst in various postures. The electrophysiological signal recorder has also been used to record the electrohysterogram (EHG), which is the name given to the electrical activity of the uterus, from abdominal electrodes during labour. A strong correlation was found to exist between tocographs derived from the EHG and tocographs produced by conventional means.
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Dobson, Jonathan M. "ASIC implementations of the Viterbi Algorithm." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/13669.

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The Viterbi Algorithm is a popular method for decoding convolutional codes, receiving signals in the presence of intersymbol-interference, and channel equalization. In 1981 the European Telecommunications Administration (CEPT) created the Groupe Special Mobile (GSM) Committee to devise a unified pan-European digital mobile telephone standard. The proposed GSM receiver structure brings together Viterbi decoding and equilization. This thesis presents three VLSI designs of the Viterbi Algorithm with specific attention paid to the use of such modules within a GSM receiver. The first design uses a technique known as redundant number systems to produce a high speed decoder. The second design uses complementary pass-transistor logic to produce a low-power channel equalizer. The third design is a low area serial equalizer. In describing the three designs, redundant number systems and complementary pass-transistor logic are examined. It is shown that while redundant number systems can offer significant speed advantages over twos complement binary, there are other representations that can perform equally well, if not better. It will also be shown that complementary pass-transistor logic can offer a small improvement for VLSI circuits in terms of power consumption.
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Perumalla, Anvesh Kumar. "A Genetic Algorithm for ASIC Floorplanning." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1484236480221006.

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Hoffman, Joseph A. "VHDL modeling of ASIC power dissipation." Master's thesis, This resource online, 1994. http://scholar.lib.vt.edu/theses/available/etd-10222009-124831/.

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Droste, Dirk. "Realisierung eines Wellenfrontsensors mit einem ASIC." [S.l. : s.n.], 1999. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB8337986.

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Hussain, Waqar Muhammad. "Low Power Implantable ASIC forBio-Impedance Measurements." Thesis, KTH, Skolan för informations- och kommunikationsteknik (ICT), 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-105098.

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Electrical bio-impedance can give a lot of insight into the basic physiological parameters of human body and concentration of glucose is one of those vital parameters.In order to control diabetes mellitus, it is critically essential to maintain blood glucose concentrations within the normal physiological range to avoid diabetes related complications.Consequently, accurate in-vivo and in-vitro measurement of glucose concentration in physiological uids has long been a central goal of bio sensor research.The correlation between glucose levels in human body and bio-impedance is more potently described with invasive measurement solutions and implanting the entire system inside human body can be considered one such technique.Any implantable device needs to be miniature in size as well as consume as little power as possible and since these two factors trade-o directly or indirectly with the accuracy, the solution space of dierent presently existing measurement techniques were implored to find an alternative that can meet all the specied constraints .The resultant application specific integrated circuit (ASIC) is capable of doing electrical bio-impedance measurements in implantable equipment and can handle impedance levels residing in a large dynamic range with very low power consumption, having small size and high accuracy. The circuit is based on 150nm technology, consumes around 530W power and has a worst case accuracy of 94%.
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Books on the topic "ASIC"

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Hoppe, Bernhard. ASIC-Design. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59818-0.

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Peter, Gorman, ed. The ASIC handbook. Upper Saddle River, NJ: Prentice Hall, 2001.

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Bhathagar, Himanshu. Advanced ASIC Chip Synthesis. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4419-8668-9.

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Taraate, Vaibbhav. ASIC Design and Synthesis. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-4642-0.

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Memory, microprocessor, and ASIC. Boca Raton: CRC Press, 2003.

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1934-, Trontelj Lojze, and Shenton Graham 1939-, eds. Analog digital ASIC design. London: McGraw-Hill, 1989.

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Surviving the ASIC experience. Englewood Cliffs, N.J: Prentice Hall, 1992.

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Golshan, Khosrow. ASIC Design Implementation Process. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-58653-8.

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High-performance ASIC design: Using synthesizable domino logic in an ASIC flow. Cambridge: Cambridge University Press, 2008.

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Chadha, Rakesh, and J. Bhasker. An ASIC Low Power Primer. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-4271-4.

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Book chapters on the topic "ASIC"

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Hering, Ekbert, Klaus Bressler, and Jürgen Gutekunst. "ASIC." In Elektronik für Ingenieure, 521–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-06986-8_14.

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Gutekunst, Jürgen. "ASIC." In Elektronik für Ingenieure und Naturwissenschaftler, 659–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-54214-9_14.

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Hering, Ekbert, Klaus Bressler, and Jürgen Gutekunst. "ASIC." In Elektronik für Ingenieure, 521–51. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-662-21862-4_14.

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Gutekunst, Jürgen. "ASIC." In Elektronik für Ingenieure und Naturwissenschaftler, 669–96. Berlin, Heidelberg: Springer Berlin Heidelberg, 2021. http://dx.doi.org/10.1007/978-3-662-62698-6_14.

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Gutekunst, Jürgen. "ASIC." In Elektronik für Ingenieure und Naturwissenschaftler, 655–82. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-05499-0_14.

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Chandrasetty, Vikram Arkalgud. "ASIC Design." In VLSI Design, 47–81. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-1120-8_3.

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Buffa, Cesare. "ASIC Design." In MEMS Lorentz Force Magnetometers, 101–19. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59412-5_9.

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Taraate, Vaibbhav. "Programmable ASIC." In ASIC Design and Synthesis, 271–91. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-4642-0_18.

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Taraate, Vaibbhav. "ASIC Design." In Digital Logic Design Using Verilog, 403–10. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-3199-3_18.

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Golshan, Khosrow. "ASIC Qualification." In ASIC Design Implementation Process, 119–31. Cham: Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-58653-8_7.

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Conference papers on the topic "ASIC"

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"AP-ASIC'99. First IEEE Asia Pacific Conference on ASICs (Cat. No.99EX360)." In Proceedings of AP-ASIC'99: The First IEEE Asia-Pacific Conference on ASICs. IEEE, 1999. http://dx.doi.org/10.1109/apasic.1999.824007.

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Wu, Yuqing, Sofia Brenes, Tejas Totade, Shijin Joshua, Dhaval Damani, and Michel Salim. "ASIC." In Proceeding of the 18th ACM conference. New York, New York, USA: ACM Press, 2009. http://dx.doi.org/10.1145/1645953.1646329.

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Lin, Pingping, Jun Bi, and Hongyu Hu. "ASIC." In the 7th International Conference. New York, New York, USA: ACM Press, 2012. http://dx.doi.org/10.1145/2377310.2377317.

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"Proceedings of Second IEEE Asia Pacific Conference on ASICs. AP-ASIC 2000 (Cat. No.00EX434)." In Proceedings of Second IEEE Asia Pacific Conference on ASICs AP-ASIC 2000. IEEE, 2000. http://dx.doi.org/10.1109/apasic.2000.896892.

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Wong, Jennifer L., Farinaz Kourshanfar, and Miodrag Potkonjak. "Flexible ASIC." In the 42nd annual conference. New York, New York, USA: ACM Press, 2005. http://dx.doi.org/10.1145/1065579.1065818.

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Flynn, M. J., and R. I. Winner. "ASIC microprocessors." In the 22nd annual workshop. New York, New York, USA: ACM Press, 1989. http://dx.doi.org/10.1145/75362.75425.

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Baek, Donkyu, Insup Shin, Seungwhun Paik, and Youngsoo Shin. "Selectively patterned masks: Structured ASIC with asymptotically ASIC performance." In 2011 16th Asia and South Pacific Design Automation Conference ASP-DAC 2011. IEEE, 2011. http://dx.doi.org/10.1109/aspdac.2011.5722217.

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Caminada, Lea Michaela. "ATLAS FEI4 ASIC." In The 21st International Workshop on Vertex Detectors. Trieste, Italy: Sissa Medialab, 2013. http://dx.doi.org/10.22323/1.167.0023.

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Gabriele Saucier. "Tutorial: ASIC Prototyping." In 32nd Design Automation Conference. ACM, 1995. http://dx.doi.org/10.1109/dac.1995.249978.

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"About ASIC Technologies." In 2022 International Conference on IC Design and Technology (ICICDT). IEEE, 2022. http://dx.doi.org/10.1109/icicdt56182.2022.9933090.

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Reports on the topic "ASIC"

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Strasburg, Aaron. ASIC Commitments. Office of Scientific and Technical Information (OSTI), December 2017. http://dx.doi.org/10.2172/1415017.

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Sauers, Aaron G. DITAC FASPAX / ASIC Development. Office of Scientific and Technical Information (OSTI), October 2017. http://dx.doi.org/10.2172/1460391.

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De Geronimo, Gianluigi, and Michael Furey. Low-power CPG ASIC. Office of Scientific and Technical Information (OSTI), October 2009. http://dx.doi.org/10.2172/1001657.

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Nuckolls, L. CMOS ASIC (application specific integrated circuit). Office of Scientific and Technical Information (OSTI), July 1989. http://dx.doi.org/10.2172/5551185.

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Robertson, Perry J., Lyndon George Pierson, and Edward L. Witzke. Data encryption standard ASIC design and development report. Office of Scientific and Technical Information (OSTI), October 2003. http://dx.doi.org/10.2172/918309.

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Brockman, Jay, Peter Kogge, Michael Niemier, and Larry Pileggi. Memory-Based Structured Application Specific Integrated Circuit (ASIC) Study. Fort Belvoir, VA: Defense Technical Information Center, October 2008. http://dx.doi.org/10.21236/ada499474.

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De Geronimo, Gianluigi, and Michael Furey. Low-Power Multichannel ASIC for gamma-ray medical imager. Office of Scientific and Technical Information (OSTI), October 2011. http://dx.doi.org/10.2172/1077986.

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De Geronimo, Gianluigi, and Michael Furey. Low-power multichannel ASIC for Gamma-ray Medical Imager. Office of Scientific and Technical Information (OSTI), February 2013. http://dx.doi.org/10.2172/1077988.

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Nguyen, Du Van. An ASIC Power Analysis System for Digital CMOS Design. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.7249.

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Bolotnikov, Aleksey, Yonggang Cui, Emerson Vernon, and Gianluigi De Geronimo. Upgrading FLIR NanoRaider with the next Generation of CdZnTe Detectors. Goal - Integrate VFG detectors into FLIR R200. Advanced Virtual Grid ASIC (AVG-ASIC). Office of Scientific and Technical Information (OSTI), June 2016. http://dx.doi.org/10.2172/1338589.

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