Relevant bibliographies by topics / Ascochyta blight

Academic literature on the topic 'Ascochyta blight'

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Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Ascochyta blight"

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Daba, Ketema, Amit Deokar, Sabine Banniza, Thomas D. Warkentin, and Bunyamin Tar’an. "QTL mapping of early flowering and resistance to ascochyta blight in chickpea." Genome 59, no. 6 (June 2016): 413–25. http://dx.doi.org/10.1139/gen-2016-0036.

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In western Canada, chickpea (Cicer arietinum L.) production is challenged by short growing seasons and infestations with ascochyta blight. Research was conducted to determine the genetic basis of the association between flowering time and reaction to ascochyta blight in chickpea. Ninety-two chickpea recombinant inbred lines (RILs) developed from a cross between ICCV 96029 and CDC Frontier were evaluated for flowering responses and ascochyta blight reactions in growth chambers and fields at multiple locations and during several years. A wide range of variation was exhibited by the RILs for days to flower, days to maturity, node of first flowering, plant height, and ascochyta blight resistance. Moderate to high broad sense heritability was estimated for ascochyta blight reaction (H2 = 0.14–0.34) and for days to flowering (H2 = 0.45–0.87) depending on the environments. Negative correlations were observed among the RILs for days to flowering and ascochyta blight resistance, ranging from r = −0.21 (P < 0.05) to −0.58 (P < 0.0001). A genetic linkage map consisting of eight linkage groups was developed using 349 SNP markers. Seven QTLs for days to flowering were identified that individually explained 9%–44% of the phenotypic variation. Eight QTLs were identified for ascochyta blight resistance that explained phenotypic variation ranging from 10% to 19%. Clusters of QTLs for days to flowering and ascochyta blight resistances were found on chromosome 3 at the interval of 8.6–23.11 cM and on chromosome 8 at the interval of 53.88–62.33 cM.
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Tar’an, B., T. D. Warkentin, A. Tullu, and A. Vandenberg. "Genetic mapping of ascochyta blight resistance in chickpea (Cicer arietinum L.) using a simple sequence repeat linkage map." Genome 50, no. 1 (January 2007): 26–34. http://dx.doi.org/10.1139/g06-137.

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Ascochyta blight, caused by the fungus Ascochyta rabiei (Pass.) Lab., is one of the most devastating diseases of chickpea ( Cicer arietinum L.) worldwide. Research was conducted to map genetic factors for resistance to ascochyta blight using a linkage map constructed with 144 simple sequence repeat markers and 1 morphological marker (fc, flower colour). Stem cutting was used to vegetatively propagate 186 F2 plants derived from a cross between Cicer arietinum L. ‘ICCV96029’ and ‘CDC Frontier’. A total of 556 cutting-derived plants were evaluated for their reaction to ascochyta blight under controlled conditions. Disease reaction of the F1 and F2 plants demonstrated that the resistance was dominantly inherited. A Fain’s test based on the means and variances of the ascochyta blight reaction of the F3 families showed that a few genes were segregating in the population. Composite interval mapping identified 3 genomic regions that were associated with the reaction to ascochyta blight. One quantitative trait locus (QTL) on each of LG3, LG4, and LG6 accounted for 13%, 29%, and 12%, respectively, of the total estimated phenotypic variation for the reaction to ascochyta blight. Together, these loci controlled 56% of the total estimated phenotypic variation. The QTL on LG4 and LG6 were in common with the previously reported QTL for ascochyta blight resistance, whereas the QTL on LG3 was unique to the current population.
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Warkentin, T. D., K. Y. Rashid, and A. G. Xue. "Fungicidal control of ascochyta blight of field pea." Canadian Journal of Plant Science 76, no. 1 (January 1, 1996): 67–71. http://dx.doi.org/10.4141/cjps96-011.

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The use of fungicides for the control of ascochyta blight in field pea was investigated. Four fungicides were applied to the cultivars AC Tamor and Radley at two locations in Manitoba in 1993 and 1994. Fungicides were applied either once, twice, or three times at 10-d intervals, beginning at the initiation of flowering. Chlorothalonil and benomyl were effective m reducing the severity of ascochyta blight and increasing the yield and seed weight of field pea. The triple application of chlorothalonil resulted in a mean yield increase of 33% over that of the untreated control. Iprodione and propiconazole were relatively ineffective in controlling ascochyta blight. The percentage of seedborne ascochyta was not significantly affected by fungicide treatments. The severity of ascochyta blight was greater in 1993 that in 1994, resulting in greater benefits of chlorothalonil and benomyl applications in 1993. Key words: Field pea, Pisum sativum L., ascochyta blight, Mycosphaerella pinodes, fungicide
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Harveson, Robert M., Samuel G. Markell, Rubella Goswami, Carlos A. Urrea, Mary E. Burrows, Frank Dugan, Weidong Chen, and Linnea G. Skoglund. "Ascochyta Blight of Chickpeas." Plant Health Progress 12, no. 1 (January 2011): 30. http://dx.doi.org/10.1094/php-2011-0103-01-dg.

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Although chickpeas are reported to be susceptible to more than 50 pathogens, few diseases are currently recognized as significant economic constraints to production. Ascochyta blight, caused by the fungal pathogen Ascochyta rabiei, is the most serious chickpea disease worldwide. This paper describes the pathogen, symptoms of infection, biological and morphological characteristics, and methods to study the fungus. Accepted for publication 17 November 2010. Published 3 January 2011.
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Skoglund, Linnea G., Robert M. Harveson, Weidong Chen, Frank Dugan, Howard F. Schwartz, Samuel G. Markell, Lyndon Porter, Mary L. Burrows, and Rubella Goswami. "Ascochyta Blight of Peas." Plant Health Progress 12, no. 1 (January 2011): 29. http://dx.doi.org/10.1094/php-2011-0330-01-rs.

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Field pea is an annual, cool-season legume native to northwest to southwest Asia. It was among the first crops cultivated by man. The crop is grown primarily in North Dakota, Washington, Montana, Idaho, Oregon, and southern Canada. Ascochyta blight is a serious disease affecting above ground portions at all growth stages. Stem, crown, pod, and foliar diseases of pea are caused by a complex of Ascochyta pisi, Mycosphaerella pinodes, and Phoma pinodella. This paper reviews the disease and the pathogens involved. Accepted for publication 28 January 2011. Published 30 March 2011.
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Jha, Ambuj B., Krishna K. Gali, Sabine Banniza, and Thomas D. Warkentin. "Validation of SNP markers associated with ascochyta blight resistance in pea." Canadian Journal of Plant Science 99, no. 2 (April 1, 2019): 243–49. http://dx.doi.org/10.1139/cjps-2018-0211.

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Ascochyta blight of pea is an important disease that can cause severe yield loss. Our previous studies identified several closely linked single nucleotide polymorphism (SNP) markers associated with ascochyta blight resistance. The objective of this study was to validate SNP markers in 36 cultivars from the Saskatchewan pea regional variety trial. Ascochyta blight scores ranged from 1.0 to 9.0 at the physiological maturity stage under field conditions across the 25 site–years in Saskatchewan from 2013 to 2017. Based on Kompetitive Allele-Specific PCR assays, six SNP markers were used for an association study. SNP markers RGA-G3Ap103, PsC8780p118, and PsC22609p103 were significantly (P < 0.05) associated with ascochyta blight scores in 2013 and 2016 at Saskatoon. PsC8780p118 was significantly associated with ascochyta blight scores at Milden in 2014 and Rosthern in 2017. Furthermore, RGA-G3Ap103 showed significant association at Milden in 2014. Based on association studies, RGA-G3Ap103 and PsC8780p118 may have some potential as markers for pea breeding.
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Chang, K. F., H. U. Ahmed, S. F. Hwang, B. D. Gossen, R. J. Howard, T. D. Warkentin, S. E. Strelkov, and S. F. Blade. "Impact of cultivar, row spacing and seeding rate on ascochyta blight severity and yield of chickpea." Canadian Journal of Plant Science 87, no. 2 (April 1, 2007): 395–403. http://dx.doi.org/10.4141/p06-067.

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Field trials to assess the impact of chickpea type (desi vs. kabuli), row spacing and seeding rate on ascochyta blight of chickpea were conducted over 2 yr at Brooks, Alberta. A compound-leaved desi chickpea cultivar and unifoliate kabuli cultivar were sown at 20, 30 and 40 cm row spacing, and at three seeding rates (20, 40 and 60 seeds per 3 m row). Most of the variation in disease severity was associated with differences between the cultivars. Seeding rate, row spacing and their interactions had substantially smaller effects on ascochyta blight in comparison with cultivar effects. Late in the growing season, blight severity was consistently lower in the desi than the kabuli cultivar. Wide row spacing and low seeding rate reduced ascochyta blight severity and increased seed yield per plant. Wide row spacing in the first year reduced the seed yield per hectare, but row spacing did not significantly affect yield in 2005. Low in-row seeding rates increased yield only in 2004. There was a positive linear relationship between plant density and blight severity, and a negative relationship between yield per plant and both plant density and disease severity. We conclude that reduced plant population density could be one tool in a program to manage ascochyta blight of chickpea. Key words: Cicer arietinum, plant population density, ascochyta blight, yield
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Gossen, B. D., and D. A. Derksen. "Impact of tillage and crop rotation on ascochyta blight (Ascochyta lentis) of lentil." Canadian Journal of Plant Science 83, no. 2 (April 1, 2003): 411–15. http://dx.doi.org/10.4141/p02-088.

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Two trials were conducted from 1996 to 1999; one at Indian Head, SK, to examine the impact of tillage management on the severity of ascochyta blight of lentil, caused by Ascochyta lentis (teleomorph Didymella lentis), and a second at Saskatoon, SK, to assess the impact of crop rotation. In 1995, the blight-susceptible lentil cv. Eston was seeded across both sites and later inoculated with blight-infested lentil residue to provide a uniform level of infection. Treatments were initiated in the spring of 1996. Ascochyta blight severity was assessed on each lentil plot during the growing season. Seed quality and yield were assessed each year. A split-block design was used to minimize movement of inoculum among plots over years. In the tillage management trial at Indian Head, the main plot treatments were 0, 1, or 2 yr between lentil crops, with spring wheat as the alternate crop; the subplot treatments were zero-till vs. conventional tillage. Ascochyta blight severity was substantially higher under zero-till than under conventional tillage in the continuous lentil treatment when conditions were conducive to blight development. However, tillage management had little effect on severity when there were 2 yr between successive lentil crops. We conclude that tillage management is unlikely to have an important impact on blight severity, except in rotations with short re-cropping intervals. In the crop rotation study at Saskatoon, the main plot treatments were two rotation sequences and the subplot treatments were three crop species (canola, barley, pea) planted in 1996. Rotation 1 was seeded to cv. Eston in 1997 and barley in 1998; Rotation 2 was seeded to barley in 1997 and cv. Eston in 1998. Both rotations were seeded to cv. Eston in 1999. Also, a plot seeded continuously to cv. Eston was included at one end of each replicate block as a control. Blight was more severe in continuous lentil than in the other crop rotations, and ascochyta blight levels in 1999 were lowest where barley followed the 1996 lentil crop for both Rotation 1 and 2. However, the intervening nonhost crop had little impact on seed infection or seed yield. We conclude that at least two nonhost crops between successive lentil crops are required to substantially reduce inoculum of A. lentis following a disease outbreak. Key words: Didymella lentis, zero-till management, fusarium root rot, Lens culinaris, barley, canola, field pea
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Salotti, Irene, and Vittorio Rossi. "A Mechanistic Weather-Driven Model for Ascochyta rabiei Infection and Disease Development in Chickpea." Plants 10, no. 3 (March 1, 2021): 464. http://dx.doi.org/10.3390/plants10030464.

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Ascochyta blight caused by Ascochyta rabiei is an important disease of chickpea. By using systems analysis, we retrieved and analyzed the published information on A. rabiei to develop a mechanistic, weather-driven model for the prediction of Ascochyta blight epidemics. The ability of the model to predict primary infections was evaluated using published data obtained from trials conducted in Washington (USA) in 2004 and 2005, Israel in 1996 and 1998, and Spain from 1988 to 1992. The model showed good accuracy and specificity in predicting primary infections. The probability of correctly predicting infections was 0.838 and the probability that there was no infection when not predicted was 0.776. The model’s ability to predict disease progress during the growing season was also evaluated by using data collected in Australia from 1996 to 1998 and in Southern Italy in 2019; a high concordance correlation coefficient (CCC = 0.947) between predicted and observed data was obtained, with an average distance between real and fitted data of root mean square error (RMSE) = 0.103, indicating that the model was reliable, accurate, and robust in predicting seasonal dynamics of Ascochyta blight epidemics. The model could help growers schedule fungicide treatments to control Ascochyta blight on chickpea.
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Warkentin, Tom, Sabine Banniza, and Albert Vandenberg. "CDC Frontier kabuli chickpea." Canadian Journal of Plant Science 85, no. 4 (October 1, 2005): 909–10. http://dx.doi.org/10.4141/p04-185.

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CDC Frontier, a kabuli chickpea (Cicer arietinum L.) cultivar, was released in 2003 by the Crop Development Centre, University of Saskatchewan, for distribution to Select seed growers in western Canada through the Variety Release Program of the Saskatchewan Pulse Growers. CDC Frontier has a pinnate leaf type, fair ascochyta blight [Ascochyta rabiei (Pass.) Labr.] resistance, medium maturity, medium-large seed size and high yield potential in the Brown and Dark Brown soil zones of the Canadian prairies. Key words: Chickpea, Cicer arietinum L., cultivar description, ascochyta blight
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Dissertations / Theses on the topic "Ascochyta blight"

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Yakop, Uyek Malik. "Resistance of faba beans to Ascochyta blight." Title page, contents and summary only, 1998. http://web4.library.adelaide.edu.au/theses/09APSM/09apsmy15.pdf.

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Bibliography: leaves 111-120. This study investigated various aspects of genetic resistance in fava beans to Ascochta blight (A. fabae) with the objective to facilitate an efficient breeding strategy for long-term control. Pathogenic variability of A. fabae was found to be high, as was genetic variation between resistant fava bean accessions. A number of alternative resistance genes to that of Ascot cultivar were identified.
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Khan, Muhammad Shahid Akhtar. "Epidemiology of ascochyta blight of chickpea in Australia." Title page, contents and summary only, 1999. http://web4.library.adelaide.edu.au/theses/09PH/09phk4455.pdf.

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Bibliography: leaves 182-217. This study was conducted to determine the etiology of a blight disease of chickpea in south-eastern Australia and the factors affecting disease development. The disease had previously been identified as phoma blight. Pathogenicity testing revealed two isolates subsequently identified as Asochyta rabiei, the first conclusive identification in the southern hemisphere. Greenhouse screening of chickpea varieties identified types resistant to ascochyta blight. The effects of plant age and environmental conditions on disease development were investigated under controlled conditions in growth rooms. Seedlings were more susceptible than older plants. The optimum conditions for ascochyta blight were 20° C and a 48-96 h period of leaf wetness. Through field trials it was found that disease intensity increased over time, especially in cv. Desavic. The means of penetration of the chickpea host was established in histological studies. This study provided advance warning of this disease for the expanding chickpea industry, and has allowed the implementation of appropriate disease management strategies. It is recommended that cv. Desavic should not be grown where ascochyta blight is likely to be a problem.
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Lawsawadsiri, Somporn. "Variation in resistance to Ascochyta blight in faba beans." Title page, contents and summary only, 1994. http://web4.library.adelaide.edu.au/theses/09PH/09phl425.pdf.

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Coram, Tristan Edward, and n/a. "Identification and characterisation of genes controlling the resistance response to ascochyta blight (Ascochyta rabiei (Pass.) Labrousse) in chickpea (Cicer arietinum L.)." RMIT University. Applied Science, 2006. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20090715.110720.

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Ascochyta blight, caused by Ascochyta rabiei (Pass.) Labrousse, is one of the most destructive diseases of chickpea (Cicer arietinum L.) worldwide. Despite the existence of highly resistant uncultivated genotypes, attempts to develop cultivars with a high level of durable resistance have been unsuccessful. This study investigated the chickpea defence response to A. rabiei using a functional genomics approach, which has the capacity to improve the overall understanding of the coordinated defence response at a molecular level. An existing cDNA library was used to generate a resource of Expressed Sequence Tags (ESTs) that, after clustering, comprised 516 unigenes. The unigenes were functionally annotated resulting in the identification of 20 specific defence-related unigenes, as well as numerous transcripts with possible involvement in the coordination of defence responses. To explore the expression patterns of the defence-related unigenes in an A. rabiei resistant and susceptible genotype, the unigenes were employed as probes in microarrays. Resulting expression data was analysed to identify differentially expressed unigenes over a time-course after infection. Comparison of the expression profiles from the resistant and susceptible genotype identified three putative genes that were exclusively up-regulated in the resistant genotype, thus may be involved in an effective defence response. Considering that a defence response can involve hundreds of genes, the entire set of chickpea unigenes were used to construct large-scale microarrays. To supplement the chickpea probes, 156 putative defence-related grasspea (Lathyrus sativus L.) ESTs and 41 lentil (Lens culinaris Med.) Resistance Gene Analogs (RGAs) were also included. Expression profiles for three chickpeas and one wild relative were generated over a time course. 97 differentially expressed ESTs were identified using a robust experimental system that included confirmation by quantitative RT-PCR. The results indicated that genes involved in the active defence response were similar to those governed by R-gene mediated resistance, including the production of reactive oxygen species and the hypersensitive response, down-regulation of 'housekeeping' gene expression, and expression of pathogenesis-related proteins. The comparison between resistant and susceptible genotypes identified certain gene expression 'signatures' that may be predictiv e of resistance. To further characterise the regulation of potential defence-related genes, the microarray was used to study expression profiles of the three chickpea genotypes (excluding the wild relative) after treatment with the defence signalling compounds, ethylene (E), salicylic acid (SA), and jasmonate (JA). 425 ESTs were differentially expressed, and comparison between genotypes revealed the presence of a wider range of inducible defence responses in resistant genotypes. Linking the results with the previous microarray results indicated the presence of other pathogen-specific signalling mechanisms in addition to E, SA and JA. The lower arsenal of defence-related gene expression observed in the susceptible genotype may be a result of 'breaks' in the pathways of defence-related gene activation. To draw together the findings of all experiments, a model was constructed for a hypothetical mechanism of chickpea resistance to A. rabiei. The model was synthesised based on the evidence gathered in this study and previously documented defence mechanisms in chickpea, and identified signal transduction as a key to resistance.
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Miranda, Andre Luis Rodrigues. "Genome Mapping and Molecular Markers for Ascochyta Blight Resistance in Pea (Pisum Sativum L.)." Thesis, North Dakota State University, 2012. https://hdl.handle.net/10365/26798.

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Ascochyta blight is the most common disease of economic importance in peas (Pisum sativum L.) in North Dakota. Selection based on molecular markers would greatly facilitate identification of resistant varieties. A mapping population comprised of 394 F7-derived recombinant inbred line (RILs) and derived from the cross `Lifter'/'Radley' was developed to study resistance to Ascochyta blight. A genetic map was developed based on 179 loci including SSR, RAPD, and CAPS markers, distributed on seven linkage groups. Phenotyping for reaction to Ascochyta blight was carried out under greenhouse and field conditions. Five replicate plants were scored using a 0 to 5 scale, where 0 = no disease and 5 = plant death. Forty-three lines showed a high level of resistance and QTL analysis identified ten DNA markers associated with Ascochyta blight resistance genes. This genetic map will provide additional insight to localize disease resistance genes/QTLs and aid development of resistant varieties.
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McCutchan, Jennifer Susan. "Transferring ascochyta blight resistance from Lathyrus sp. into field pea (Pisum sativum L.) via protoplast fusion (somatic hybridisation) /." Connect to thesis, 2001. http://eprints.unimelb.edu.au/archive/00000696.

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Gewiss, Estelle Cecile. "Toxin production by Ascochyta rabiei, the causal agent of ascochyta blight of chickpea (Cicer arietinum L.), and development of a transformation protocol for the fungus." Thesis, University College London (University of London), 2004. http://discovery.ucl.ac.uk/1446852/.

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Ascochyta rabiei is the causal agent of blight of chickpea, an important food legume crop for human populations in Developing Countries. All reliably identified isolates of the fungus produce toxins in culture, known as the solanapyrones, of which solanapyrone A is the most frequently found and also the most toxic. The principal aim of the project was to determine the role of this toxin in the disease syndrome by producing toxin-minus mutants and testing them for virulence. Four transformation techniques were attempted: Restriction Enzyme Mediated Integration (REMI), electroporation, particle bombardment and Agrobacterium tumefaciens-mediated transformation. With the last, employing a T-DNA containing a hygromycin resistance gene, 908 transformants were obtained from germinated pycnidiospores on a selective medium containing hygromycin. Genuine transformants were tested for the production of solanapyrone A using an assay in microtitre plates. Loss of toxin production by transformants was confirmed by reversed phase High Performance Liquid Chromatography. Sixteen transformants produced significantly less solanapyrone A than the wild-type strain. Transformants were also screened for integration events by PCR, using primers specific to the hygromycin resistance gene and homologous hybridisation to a probe consisting of this gene. Among the four transformants tested, three have integrated two copies of T-DNA and one had a single insertion. In order to optimise the production of solanapyrone A so as to provide a source of the compound for screening chickpea genotypes, three types of cultures of A. rabiei were tested: still culture, shake culture and fermenter culture. The toxin was purified from culture filtrates by solvent partitioning followed by flash chromatography. The effect of two safeners on the sensitivity of chickpea shoots to solanapyrone A was tested using bioassays. Dichlormid (300 or 800 g per shoot) and fenclorim (18 g per shoot) decreased the sensitivity of chickpea shoots to solanapyrone A 1.6 and 2.5-fold, respectively.
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Aryamanesh, Nader. "Chickpea improvement through genetic analysis and quantitative trait locus (QTL) mapping of ascochyta blight resistence using wild Cicer species." University of Western Australia. School of Plant Biology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0072.

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[Truncated abstract] The genetics of ascochyta blight resistance was studied in five 5 x 5 half-diallel cross sets involving seven genotypes of chickpea (ICC 3996, Almaz, Lasseter, Kaniva, 24B-Isoline, IG 9337 and Kimberley Large), three accessions of Cicer reticulatum (ILWC 118, ILWC 139 and ILWC 184) and one accession of C. echinospermum (ILWC 181) under field conditions. Both F1 and F2 generations were used in the diallel analysis. Almaz, ICC 3996 and ILWC 118 were the most resistant genotypes. Estimates of genetic parameters, following Hayman's method, showed significant additive and dominant gene actions. The analysis also revealed the involvement of both major and minor genes. Susceptibility was dominant over resistance to ascochyta blight. The recessive alleles were concentrated in the two resistant chickpea parents ICC 3996 and Almaz, and one C. reticulatum genotype ILWC 118. High narrow-sense heritability (ranging from 82 to 86% for F1 generations, and 43 to 63% for F2 generations) indicates that additive gene effects were more important than non-additive gene effects in the inheritance of the trait and greater genetic gain by breeding resistant chickpea cultivars using carefully selected parental genotypes. Current simple leaf varieties are often susceptible to ascochyta blight disease whereas varieties of other leaf types range from resistant to susceptible. The inheritance of ascochyta blight resistance and different leaf types and their correlation were investigated in intraspecific progeny derived from crosses among two resistant genotypes with normal leaf type (ICC 3996 and Almaz), one susceptible simple leaf type (Kimberley Large) and one susceptible multipinnate leaf type (24 B-Isoline). ... An interspecific F2 mapping population derived from a cross between chickpea accession ICC 3996 (resistant to ascochyta blight, early flowering, and semi-erect plant growth habit) and C. reticulatum accession ILWC 184 (susceptible to ascochyta blight, ii late flowering, and prostrate plant growth habit) was used for constructing a genetic linkage map. F2 plants were cloned through stem cuttings taken at pre-flowering stage, treated with plant growth regulator powder (0.5 mg/g indole butyric acid (IBA) and 0.5 mg/g naphthalene acetic acid (NAA)) and grown in a sand + potting mix substrate. Clones were screened for ascochyta blight resistance in controlled environment conditions using a 19 scale. Three quantitative trait loci (QTLs) were found for ascochyta blight resistance in this population. Two linked QTLs, located on linkage group (LG) 4, explained 21.1% and 4.9% of the phenotypic variation. The other QTL, located on LG3, explained 22.7% of the phenotypic variation for ascochyta blight resistance. These QTLs explained almost 49% of the variation for ascochyta blight resistance. LG3 had two major QTLs for days to flowering (explaining 90.2% of phenotypic variation) and a major single QTL for plant growth habit (explaining 95.2% of phenotypic variation). There was a negative correlation between ascochyta blight resistance and days to flowering, and a positive correlation between days to flowering and plant growth habit. The flanking markers for ascochyta blight resistance or other morphological characters can be used in marker-assisted selections to facilitate breeding programs.
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Mittal, Nitin. "Ascochyta Rabiei in North Dakota: Characterization of the Secreted Proteome and Population Genetics." Thesis, North Dakota State University, 2011. https://hdl.handle.net/10365/29857.

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Chickpea is one of the most important leguminous crops grown in regions of southern Europe, Asia, the Middle East, and the United States. Ascochyta blight, caused by Ascochyta rabiei, is the most important foliar disease of chickpea. In favorable conditions, this disease can destroy the entire chickpea field within a few days. In this project the secreted proteins of Ascochyta rabiei have been characterized through one and two-dimensional polyacrylamide gel electrophoresis. This is the first proteomic study of the A. rabiei secretome, and a standardized technique to study the secreted proteome has been developed. A common set of proteins secreted by this pathogen and two isolates that exhibit the maximum and minimum number of secreted proteins when grown in modified Fries and Czapek Dox media have been identified. Population genetic studies of Ascochyta rabiei populations in North Dakota have been conducted using microsatellites and AFLP markers. Population genetic studies have shown that the ascochyta population in North Dakota has not changed genetically in the years 2005, 2006 and 2007, but the North Dakota population is different from the baseline population from the Pacific Northwest. The ascochyta population in North Dakota is a randomly mating population, as shown by the mating type ratio.
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Aryamanesh, Nader. "Chickpea improvement through genetic analysis and quantitative trait locus (QTL) mapping of ascochyta blight resistence using wild Cicer species /." Connect to this title, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0072.

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Books on the topic "Ascochyta blight"

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Pande, S. Host plant resistance to Ascochyta blight of chickpea. Patancheru: International Crops Research Institute for the Semi-arid Tropics, 2010.

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Tivoli, Bernard, Alain Baranger, Fred J. Muehlbauer, and B. M. Cooke, eds. Ascochyta blights of grain legumes. Dordrecht: Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-6065-6.

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Foote, P. Darlene. Identification and utilization of resistance to Ascochyta blight found in wild Cicer species. 1988.

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Kusmenoglu, Ismail. Ascochyta blight of chickpea: Inheritance and relationship to seed size, morphological traits and isozyme variation. 1990.

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Sakr, Bouazza. Inheritance and linkage of genetic markers and resistance to Ascochyta blight in lentil. 1994.

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6

(Editor), Bernard Tivoli, Alain Baranger (Editor), Fred J. Muehlbauer (Editor), and B. M. Cooke (Editor), eds. Ascochyta blights of grain legumes. Springer, 2007.

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Book chapters on the topic "Ascochyta blight"

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Schoeny, Alexandra, Stéphane Jumel, François Rouault, Christophe Le May, and Bernard Tivoli. "Assessment of airborne primary inoculum availability and modelling of disease onset of ascochyta blight in field peas." In Ascochyta blights of grain legumes, 87–97. Dordrecht: Springer Netherlands, 2007. http://dx.doi.org/10.1007/978-1-4020-6065-6_9.

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Taleei, A., H. Kanouni, and M. Baum. "QTL Analysis of Ascochyta Blight Resistance in Chickpea." In Communications in Computer and Information Science, 25–40. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-10236-3_3.

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Taleei, Alireza, Homayoun Kanouni, and Michael Baum. "Genetical Analysis of Ascochyta Blight Resistance in Chickpea." In Bio-Science and Bio-Technology, 31–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-10616-3_5.

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Kaiser, W. J., F. J. Muehlbauer, and R. M. Hannan. "Experience with Ascochyta blight of chickpea in the United States." In Expanding the Production and Use of Cool Season Food Legumes, 849–58. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0798-3_52.

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Conference papers on the topic "Ascochyta blight"

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Taleei, Alireza, Homayoun Kanouni, Michael Baum, Seyed Ali Peyghambari, Seyed Mahmood Okhovat, and Mathew Abang. "Identification and Mapping of QTLs for Resistance to Ascochyta Blight (Pathotype III) in Chickpea." In 2008 Second International Conference on Future Generation Communication and Networking (FGCN). IEEE, 2008. http://dx.doi.org/10.1109/fgcn.2008.184.

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