Relevant bibliographies by topics / Asbestos pleural disease

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Asbestos pleural disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Asbestos pleural disease"

1

Perić, Irena, Katarina Novak, Igor Barišić, Kornelija Miše, Maja Vučković, Stipan Janković, and Jadranka Tocilj. "Interobserver Variations in Diagnosing Asbestosis According to the ILO Classification." Archives of Industrial Hygiene and Toxicology 60, no. 2 (June 1, 2009): 191–95. http://dx.doi.org/10.2478/10004-1254-60-2009-1904.

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Interobserver Variations in Diagnosing Asbestosis According to the ILO ClassificationInhalation of asbestos fibres leads to asbestosis of the pleura and the lung, with possible progression to lung cancer and malignant pleural or peritoneal mesothelioma. Asbestosis remains difficult to diagnose, especially in its early stages. The most important role in its diagnosis is that of chest radiographs. The aim of this cross-sectional study was to address interobserver variations in interpreting chest radiographs in asbestos workers, which remain to be an issue, despite improvements in the International Labour Office (ILO) classification system. In our ten-year study, we investigated 318 workers occupationally exposed to asbestos, and in 210 workers with diagnosed asbestos-related changes we compared interpretations of chest radiographs according to ILO by two independent radiologists. The apparent degree of interobserver variation in classifying lung fibrosis was 26.66% for the diameter of changes and 42.2% for the profusion of the changes. In cases with diffuse pleural thickening, the interobserver variation using ILO procedures was 34.93%. This investigation raises the issue of standardisation and objectivity of interpretation of asbestosis according to the ILO classification system. This study has revealed a significant disagreement in the estimated degree of pleural and parenchymal asbestos pulmonary disease. This is why we believe high-resolution computed tomography (HRCT) should also be used as a part of international classification.
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Franko, Alenka, Katja Goricar, Metoda Dodic Fikfak, Viljem Kovac, and Vita Dolzan. "The role of polymorphisms in glutathione-related genes in asbestos-related diseases." Radiology and Oncology 55, no. 2 (January 26, 2021): 179–86. http://dx.doi.org/10.2478/raon-2021-0002.

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Abstract Background The study investigated the influence of GCLC, GCLM, GSTM1, GSTT1 and GSTP1 polymorphisms, as well as the influence of interactions between polymorphism and interactions between polymorphisms and asbestos exposure, on the risk of developing pleural plaques, asbestosis and malignant mesothelioma (MM). Subjects and methods The cross sectional study included 940 asbestos-exposed subjects, among them 390 subjects with pleural plaques, 147 subjects with asbestosis, 225 subjects with MM and 178 subjects with no asbestos-related disease. GCLC rs17883901, GCLM rs41303970, GSTM1 null, GSTT1 null, GSTP1 rs1695 and GSTP1 rs1138272 genotypes were determined using PCR based methods. In statistical analysis, logistic regression was used. Results GSTT1 null genotype was associated with the decreased risk for pleural plaques (OR = 0.63; 95% CI = 0.40–0.98; p = 0.026) and asbestosis (OR = 0.51; 95% CI = 0.28–0.93; p = 0.028), but not for MM. A positive association was found between GSTP1 rs1695 AG + GG vs. AA genotypes for MM when compared to pleural plaques (OR = 1.39; 95% CI = 1.00–1.94; p = 0.049). The interactions between different polymorphisms showed no significant influence on the risk of investigated asbestos-related diseases. The interaction between GSTT1 null polymorphism and asbestos exposure decreased the MM risk (OR = 0.17; 95% CI = 0.03–0.85; p = 0.031). Conclusions Our findings suggest that GSTT1 null genotype may be associated with a decreased risk for pleural plaques and asbestosis, may modify the association between asbestos exposure and MM and may consequently act protectively on MM risk. This study also revealed a protective effect of the interaction between GSTP1 rs1695 polymorphism and asbestos exposure on MM risk.
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Roggli, Victor L. "The Contributions Of Analytical Electron Microscopy to the Detection and Quantification of Asbestos in Human Lung Samples." Proceedings, annual meeting, Electron Microscopy Society of America 48, no. 2 (August 12, 1990): 340–41. http://dx.doi.org/10.1017/s0424820100135307.

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Analytical electron microscopy has contributed a great deal to our understanding of asbestosrelated diseases. Exposure to the various forms of asbestos, which include the serpentine form known as chrysotile asbestos, and the amphibole forms referred to as amosite, crocidolite, tremolite, anthophyllite, and actinolite asbestos, has been associated with the development of a number of diseases in man. These include asbestosis (scarring of the lung parenchyma), pleural plaques (scarring of the pleura), malignant mesothelioma of the pleura and peritoneum, and carcinoma of the lung, especially among those who also smoke cigarettes.Analysis of the mineral fiber content of the lung in patients with these various diseases has provided a powerful investigative tool to researchers interested in the relationship between fiber burdens and disease. Such studies have shown that when sufficiently sensitive digestion concentration techniques are employed, some asbestos can be found in lung tissue from virtually every adult in the general population.
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Nishimura, Stephen L., and V. Courtney Broaddus. "ASBESTOS-INDUCED PLEURAL DISEASE." Clinics in Chest Medicine 19, no. 2 (June 1998): 311–29. http://dx.doi.org/10.1016/s0272-5231(05)70079-4.

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Hillerdal, Gunnar. "Asbestos-Related Pleural Disease." Seminars in Respiratory and Critical Care Medicine 9, no. 01 (July 1987): 65–74. http://dx.doi.org/10.1055/s-2007-1012690.

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6

Smith, Dorsett D. "Asbestos-Related Pleural Disease." Clinical Pulmonary Medicine 1, no. 5 (September 1994): 289–300. http://dx.doi.org/10.1097/00045413-199409000-00003.

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7

Myers, Renelle. "Asbestos-related pleural disease." Current Opinion in Pulmonary Medicine 18, no. 4 (July 2012): 377–81. http://dx.doi.org/10.1097/mcp.0b013e328354acfe.

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8

Weber, Marc-André, Michael Bock, Christian Plathow, Klaus Wasser, Christian Fink, Ivan Zuna, Astrid Schmähl, Irina Berger, Hans-Ulrich Kauczor, and Stefan O. Schoenberg. "Asbestos-Related Pleural Disease." Investigative Radiology 39, no. 9 (September 2004): 554–64. http://dx.doi.org/10.1097/01.rli.0000131888.39636.c5.

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Solbes, Eduardo, and Richart W. Harper. "Biological responses to asbestos inhalation and pathogenesis of asbestos-related benign and malignant disease." Journal of Investigative Medicine 66, no. 4 (January 6, 2018): 721–27. http://dx.doi.org/10.1136/jim-2017-000628.

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Asbestos comprises a group of fibrous minerals that are naturally occurring in the environment. Because of its natural properties, asbestos gained popularity for commercial applications in the late 19th century and was used throughout the majority of the 20th century, with predominant use in the construction, automotive, and shipbuilding industries. Asbestos has been linked to a spectrum of pulmonary diseases, such as pleural fibrosis and plaques, asbestosis, benign asbestos pleural effusion, small cell lung carcinoma, non-small cell lung carcinoma, and malignant mesothelioma. There are several mechanisms through which asbestos can lead to both benign and malignant disease, and they include alterations at the chromosomal level, activation of oncogenes, loss of tumor suppressor genes, alterations in cellular signal transduction pathways, generation of reactive oxygen and nitrogen species, and direct mechanical damage to cells from asbestos fibers. While known risk factors exist for the development of asbestos-related malignancies, there are currently no effective means to determine which asbestos-exposed patients will develop malignancy and which will not. There are also no established screening strategies to detect asbestos-related malignancies in patients who have a history of asbestos exposure. In this article, we present a case that highlights the different biological responses in human hosts to asbestos exposure.
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Beritić, Tihomil, and Silvija Kovač. "Asbestos-related disease without asbestosis — why not pleural asbestosis?" American Journal of Industrial Medicine 8, no. 6 (1985): 517–20. http://dx.doi.org/10.1002/ajim.4700080603.

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Dissertations / Theses on the topic "Asbestos pleural disease"

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Singh, Bhajan. "The function of the human diaphragm as a volume pump and measurement of its efficiency." University of Western Australia. School of Biomedical and Chemical Sciences, 2004. http://theses.library.uwa.edu.au/adt-WU2004.0029.

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[Truncated abstract] The function of the diaphragm as a volume pump has not been adequately evaluated because there are no accurate methods to measure the volume displaced by diaphragm motion (ΔVdi). As a consequence, the work done, power output and efficiency of the diaphragm have not been measured. Efficiency of the diaphragm could be measured by relating the power output of the diaphragm to its neural activation. The aims of this thesis were to (a) develop a new biplanar radiographic method to measure ΔVdi and use this to evaluate the effect of costophrenic fibrosis and emphysema on ΔVdi, (b) develop a new fluoroscopic method to enable breath-by-breath measurements of ΔVdi, (c) evaluate a method for quantifying neural activation of the diaphragm, and (d) combine measurements of transdiaphragmatic pressure, ΔVdi, inspiratory duration and neural activation of the diaphragm to quantify the neuromechanical efficiency of the diaphragm
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Books on the topic "Asbestos pleural disease"

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1930-, Greenberg S. Donald, and Pratt Philip C, eds. Pathology of asbestos-associated diseases. Boston: Little, Brown, 1992.

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2

Millar, Professor Ann B., Dr Richard Leach, Dr Rebecca Preston, Dr Richard Leach, Dr Richard Leach, Dr Wei Shen Lim, Dr Richard Leach, et al. Respiratory diseases and respiratory failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0005.

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Chapter 5 covers respiratory diseases and respiratory failure, including clinical presentations of respiratory disease, assessment of diffuse lung disease, hypoxaemia, respiratory failure, and oxygen therapy, pneumonia, mycobacterial infection, asthma, chronic obstructive pulmonary disease (COPD), lung cancer, mediastinal lesions, pneumothorax, pleural disease, asbestos-related lung disease, diffuse parenchymal (interstitial) lung disease, sarcoidosis, pulmonary hypertension, acute respiratory distress syndrome, bronchiectasis and cystic fibrosis, bronchiolitis, eosinophilic lung disease, airways obstruction, aspiration syndromes, and near-drowning, pulmonary vasculitis, the immunocompromised host, sleep apnoea, and rare pulmonary diseases.
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Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Chest medicine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0011.

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This chapter provides information on the ageing lung, respiratory infections, influenza, pneumonia, pneumonia treatment, vaccinating against pneumonia and influenza, pulmonary fibrosis, rib fractures, pleural effusions, pulmonary embolism, aspiration pneumonia/pneumonitis, lung cancers, chronic cough, presentation of tuberculosis, tuberculosis investigation, treatment of tuberculosis, assessment of asthma and chronic obstructive pulmonary disease (COPD), drug treatment of asthma and COPD, non-drug treatment of asthma and COPD, oxygen therapy, and asbestos-related disease.
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Oury, Tim D., Thomas A. Sporn, and Victor L. Roggli. Pathology of Asbestos-Associated Diseases. Springer, 2016.

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5

(Editor), Victor L. Roggli, Tim D. Oury (Editor), and Thomas A. Sporn (Editor), eds. Pathology of Asbestos-Associated Diseases. 2nd ed. Springer, 2004.

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6

Ken, O'Byrne, and Rusch Valerie W, eds. Malignant pleural mesothelioma. Oxford: Oxford University Press, 2006.

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7

(Editor), Kenneth O'Byrne, and Valerie Rusch (Editor), eds. Malignant Pleural Mesothelioma. Oxford University Press, USA, 2006.

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8

E, Craighead John, and Gibbs A. R, eds. Asbestos and its diseases. New York: Oxford University Press, 2008.

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9

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, Gareth Morris-Stiff, and Madhumita Bhattacharyya. Breast cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0014_update_001.

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Thoracic cancer examines the epidemiology, aetiology, and role of screening and prevention in the reduction of deaths from lung cancer, the majority caused by cigarette smoking. The pathology and genetics of lung cancer, with particular note of the driver mutations, are followed by the symptoms and signs of the disease. Appropriate investigations are described to stage the tumour. The optimum treatment for localised non-small cell lung cancer (NSCLC) is surgical resection, followed in some cases by adjuvant chemotherapy. However, most cases present with disease too advanced for surgery, and for these chemotherapy and radiotherapy are appropriate. Metastatic NSCLC can be treated with platinum based doublet chemotherapy with modest palliative benefits. Metastatic NSCLC with specific driver mutations are amenable to control by targeted therapy. Locally advanced NSCLC is often treated with similar chemotherapy and radiotherapy, ideally administered concurrently, to achieve symptom relief but also improved survival rates. Short course simple radiotherapy offers symptom relief in patients not fit for chemotherapy. Patients with localised NSCLC who are not fit for surgery, may benefit from radical radiotherapy, particularly stereotactic radiotherapy. Small cell lung cancer (SCLC) is characterised by almost universal systemic spread, so that surgery is rarely appropriate. Staging is similar to NSCLC, and chemotherapy is the mainstay of treatment, usually cisplatin or carboplatin combined with etoposide. When possible, this is combined with concurrent thoracic irradiation covering all radiological sites of disease. Prophylactic cranial irradiation reduces the risk of CNS disease. Malignant pleural mesothelioma is caused by occupational asbestos exposure. Symptoms and signs, investigation and staging, and management are discussed. Thymic tumours, their pathology, presenting symptoms including paraneoplastic syndromes, investigation, staging and treatment are reviewed.
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Malhotra, Jyoti, Paolo Boffetta, and Lorelei Mucci. Cancer of the Lung, Larynx, and Pleura. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0014.

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Lung cancer is the most commonly diagnosed cancer among men in most countries, and is the primary cause of cancer death in men and women. Its epidemic increase in incidence began in the first half of the twentieth century, paralleling the uptake of cigarette smoking that occurred 20 years before. A series of landmark studies beginning in 1950 established tobacco as the primary cause of lung cancer. Current smokers have a 10- to 20-fold higher lung cancer risk compared to never smokers. Important for prevention, former smokers substantially reduce this excess risk 5 years after smoking cessation. Exposure to secondhand smoke, a well-established risk factor for lung cancer, has a 20%–25% higher risk for those exposed. There are several occupational exposures associated with lung cancer, including asbestos. Despite the success in defining lung cancer’s etiology, this highly preventable disease remains among the most common and most lethal cancers globally.
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Book chapters on the topic "Asbestos pleural disease"

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Allen, Timothy Craig. "Asbestos-Induced Pleural Disease." In Encyclopedia of Pathology, 21–22. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-66796-6_4294.

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Allen, Timothy Craig. "Asbestos-Induced Pleural Disease." In Encyclopedia of Pathology, 1–3. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28845-1_4294-1.

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Manni, Michelle L., and Tim D. Oury. "Benign Asbestos-Related Pleural Disease." In Pathology of Asbestos-Associated Diseases, 141–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-41193-9_6.

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4

Musk, Arthur William, and Jennie Hui. "Non-malignant pleural disease from asbestos and malignant pelural mesothelioma." In Occupational and Environmental Lung Disease, 141–49. Sheffield, United Kingdom: European Respiratory Society, 2020. http://dx.doi.org/10.1183/2312508x.10034719.

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5

Nishimura, Yasumitsu, Naoko Kumagai-Takei, Suni Lee, Kei Yoshitome, Tatsuo Ito, and Takemi Otsuki. "Asbestos Fiber and Immunological Effects: Do Immunological Effects Play Any Role in Asbestos-Related Diseases?" In Malignant Pleural Mesothelioma, 33–41. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-9158-7_3.

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6

"Asbestos-Induced Pleural Disease." In Asbestos, 497–516. CRC Press, 2011. http://dx.doi.org/10.1201/b10958-15.

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7

Azok, Joseph T. "Asbestosis." In Chest Imaging, 383–86. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199858064.003.0066.

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Asbestosis is a fibrotic pneumoconiosis resulting from the inhalation of asbestos fibers, most commonly from occupational exposure. Chest radiographs and high-resolution chest CT can detect asbestos-related disease. Pleural abnormalities include pleural plaques, pleural effusions, pleural thickening, and mesothelioma. Pleural plaques serve as a marker of asbestos exposure and are the most common imaging abnormality found in patients exposed to asbestos. Parenchymal-induced lung disease includes pulmonary fibrosis, known as asbestosis, rounded atelectasis, and lung cancer. Asbestos exposure leads to an increased risk of both lung cancer and especially mesothelioma, which is rare in the absence of asbestos exposure.
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8

"Benign Asbestos-Related Pleural Disease." In Pleural Disease, 568–92. CRC Press, 2004. http://dx.doi.org/10.1201/b14205-35.

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9

"Benign Asbestos–Related Pleural Disease." In Pleural Disease, 524–44. CRC Press, 2009. http://dx.doi.org/10.3109/9781420077391-33.

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Smith, Dorsett D. "Asbestos-related pleural disease." In The Health Effects of Asbestos, 109–28. CRC Press, 2015. http://dx.doi.org/10.1201/b19413-8.

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Conference papers on the topic "Asbestos pleural disease"

1

Scarlata, Simone, Panaiotis Finamore, Gilda Giannunzio, Simona Santangelo, and Raffaele Antonelli Incalzi. "Chest ultrasonography in health surveillance of asbestos related pleural disease." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa410.

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Rojano-Broz, B., J. Rodriguez-Portal, E. Rodriguez-Becerra, D. Rodriguez, I. Alfageme, A. Quero Martinez, C. Diego, A. Leon-Jimenez, I. Isidro Montes, and P. Cebollero. "Serum Levels of Soluble Mesothelin-Related Peptides in Malignant and Non-Malignant Asbestos-Related Pleural Disease: Relation with past Asbestos Exposure." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3928.

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Girardi, Paolo, Anna Somigliana, Pietro Gino Barbieri, and Enzo Merler. "0362 Risk of pleural mm and residual asbestos burden in the lung: a retrospective case-control study." In Eliminating Occupational Disease: Translating Research into Action, EPICOH 2017, EPICOH 2017, 28–31 August 2017, Edinburgh, UK. BMJ Publishing Group Ltd, 2017. http://dx.doi.org/10.1136/oemed-2017-104636.297.

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