Relevant bibliographies by topics / Arthritis

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Arthritis'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Arthritis"

1

SMITH, JAMES L. "Arthritis and Foodborne Bacteria." Journal of Food Protection 57, no. 10 (October 1, 1994): 935–41. http://dx.doi.org/10.4315/0362-028x-57.10.935.

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Diarrheic episodes caused by the foodborne pathogens Campylobacter, Salmonella, Shigella or Yersinia may lead to a sterile arthritis such as reactive arthritis, Reiter's syndrome or ankylosing spondylitis. Reiter's syndrome and reactive arthritis have been shown to be sequelae in a few well-studied bacterial food poisoning outbreaks. Reactive arthritis, Reiter's syndrome and ankylosing spondylitis show strong familial association related to the gene for HLA-B27 (HLA = human leucocyte antigen) antigen. Why HLA-B27-positive individuals are more susceptible to arthritis is not known, but molecular mimicry between the HLA-B27 antigen and antigens of triggering bacteria has been demonstrated and this mimicry has been proposed as a mechanism involved in etiology of the arthritides. Antigens from bacteria that triggered the arthritis are present in arthritic joints but bacterial cells are not found. Antibodies and T-cells specific for the triggering bacteria have been demonstrated in arthritic patients. T-cells present in synovial joints respond specifically to the particular arthritic triggering pathogen. The cells that respond to bacterial antigens belong to the T-cell subset TH1 that secrete a limited number of cytokines but it is not known if cytokines are involved in arthritis. A few studies have demonstrated that T-cells from the joints of arthritic patients respond to both bacterial and human heat shock proteins indicating that autoimmunity may be involved in causation of arthritis. While only about 2% of a population exposed to a triggering infection will acquire arthritis, these individuals undergo pain and suffering as well as economic hardships as a result of their disease.
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ODEWUSI, OO, MJ ABDULMUMIN, and OO OLANIYAN. "AN ASSESSMENT OF AUTOIMMUNITY IN ARTHRITIS PATIENTS." International Journal of Medical Laboratory Research 07, no. 01 (2022): 53–61. http://dx.doi.org/10.35503/ijmlr.2022.7108.

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Objectives: The goal of this study is to estimate autoimmune biomarkers that characterize the development and severity of arthritis, but probably normalize following successful therapy. Materials and methods: In this study a total of 109 subjects were used out of which treated and untreated arthritics were 48 and 44 respectively, the remaining 17 were healthy individuals which were used as control. Samples were collected from patients attending Rheumatology and Orthopedic clinic of Federal Teaching Hospital Ido-ekiti, Ekiti State Nigeria. Antinuclear antibody was estimated using Enzyme Linked Immunosorbent Assay (ELISA) while Lupus Erythematosus cells were ascertained microscopically using Leishman staining technique. All parameters were assessed in treated and untreated arthritic patients relative to healthy subjects. Body mass index was also calculated. Statistical analysis was done using SPSS. Results: Body mass index and Antinuclear antibodies were significantly higher in treated and untreated arthritics compared to control (P<0.05). When treated and untreated arthritics were compared, Body mass index and Antinuclear antibody were found to be significantly higher in untreated arthritics (P<0.05). Antinuclear antibody and Age correlated directly in untreated arthritics. Lupus Erythematosus cell prevalence was found to be higher in untreated arthritics having a percentage Lupus Erythematosus test positivity of 6.8% compared to the 2.1% seen in treated arthritics. Conclusion: It was found that Autoimmunity in arthritics can be significantly lowered through treatment with Arthritic drugs, diets, life style modifications over a period of time. The study suggests that Antinuclear antibody and Lupus Erythematosus estimations could be adopted as markers of diagnosis, prognosis and monitoring of arthritis.
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Castillero, Estíbaliz, María Paz Nieto-Bona, Carmen Fernández-Galaz, Ana Isabel Martín, María López-Menduiña, Miriam Granado, María Angeles Villanúa, and Asunción López-Calderón. "Fenofibrate, a PPARα agonist, decreases atrogenes and myostatin expression and improves arthritis-induced skeletal muscle atrophy." American Journal of Physiology-Endocrinology and Metabolism 300, no. 5 (May 2011): E790—E799. http://dx.doi.org/10.1152/ajpendo.00590.2010.

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Arthritis is a chronic inflammatory illness that induces cachexia, which has a direct impact on morbidity and mortality. Fenofibrate, a selective PPARα activator prescribed to treat human dyslipidemia, has been reported to decrease inflammation in rheumatoid arthritis patients. The aim of this study was to elucidate whether fenofibrate is able to ameliorate skeletal muscle wasting in adjuvant-induced arthritis, an experimental model of rheumatoid arthritis. On day 4 after adjuvant injection, control and arthritic rats were treated with 300 mg/kg fenofibrate until day 15, when all rats were euthanized. Fenofibrate decreased external signs of arthritis and liver TNFα and blocked arthritis-induced decreased in PPARα expression in the gastrocnemius muscle. Arthritis decreased gastrocnemius weight, which results from a decrease in cross-section area and myofiber size, whereas fenofibrate administration to arthritic rats attenuated the decrease in both gastrocnemius weight and fast myofiber size. Fenofibrate treatment prevented arthritis-induced increase in atrogin-1 and MuRF1 expression in the gastrocnemius. Neither arthritis nor fenofibrate administration modify Akt-FoxO3 signaling. Myostatin expression was not modified by arthritis, but fenofibrate decreased myostatin expression in the gastrocnemius of arthritic rats. Arthritis increased muscle expression of MyoD, PCNA, and myogenin in the rats treated with vehicle but not in those treated with fenofibrate. The results indicate that, in experimental arthritis, fenofibrate decreases skeletal muscle atrophy through inhibition of the ubiquitin-proteasome system and myostatin.
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Baillet, Athan, Candice Trocmé, Xavier Romand, Chuong M. V. Nguyen, Anais Courtier, Bertrand Toussaint, Philippe Gaudin, and Olivier Epaulard. "Calprotectin discriminates septic arthritis from pseudogout and rheumatoid arthritis." Rheumatology 58, no. 9 (March 28, 2019): 1644–48. http://dx.doi.org/10.1093/rheumatology/kez098.

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Abstract Objective We aimed to determine whether calprotectin and α-defensins could discriminate septic from other inflammatory arthritides. Methods Synovial fluids with a predominance of neutrophils from patients with septic arthritis, pseudogout and RA were prospectively collected. Neutrophil-related proteins calprotectin and human neutrophil α-defensins levels were assessed in synovial fluids. Demographic parameters and biomarkers with P-value ⩽0.05 for differentiating septic from non-septic arthritis were included in a multivariable model. Multivariable logistic regression with stepwise selection was performed to build the final combined model. Results A total of 74 patients were included: septic arthritis (n = 26), pseudogout (n = 28) and RA (n = 20). Patients with septic arthritis were more likely to be male and young, and to display higher synovial neutrophil count. Calprotectin was significantly increased in patients with septic arthritis. The multivariable model included calprotectin, synovial fluid neutrophil count and gender. Calprotectin was the only biomarker that discriminated septic arthritis from non-septic inflammatory arthritides, with 76% sensitivity, 94% specificity and a positive likelihood ratio = 12.2 at the threshold for calprotectin of 150 mg/l. Conclusion Synovial fluid calprotectin is a relevant biomarker to discriminate septic arthritis from other inflammatory arthritides. This biomarker should be tested in an independent cohort.
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Gómez-SanMiguel, Ana Belén, Ana Isabel Martín, Maria Paz Nieto-Bona, Carmen Fernández-Galaz, María López-Menduiña, María Ángeles Villanúa, and Asunción López-Calderón. "Systemic α-melanocyte-stimulating hormone administration decreases arthritis-induced anorexia and muscle wasting." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 304, no. 10 (May 15, 2013): R877—R886. http://dx.doi.org/10.1152/ajpregu.00447.2012.

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Rheumatoid cachexia is associated with rheumatoid arthritis and it increases mortality and morbidity. Adjuvant-induced arthritis is an experimental model of rheumatoid arthritis that causes anorexia and muscle wasting. α-Melanocyte-stimulating hormone (α-MSH) has anti-inflammatory actions, and it is able to decrease inflammation in several inflammatory diseases including experimental arthritis. In this study we tested whether systemic α-MSH treatment is able to ameliorate cachexia in arthritic rats. On day 8 after adjuvant injection control and arthritic rats were treated with α-MSH (50 μg/rat ip) twice a day, until day 16 when all rats were euthanized. Arthritis decreased food intake, but it increased hypothalamic expression of neuropeptide Y (NPY) and Agouti-related peptides (AgRP) as well as interleukin-1β (IL-1β) and cyclooxygenase-2 (COX-2) mRNA. In arthritic rats, α-MSH decreased the external signs of arthritis and increased food intake ( P < 0.01). In addition, α-MSH decreased hypothalamic expression of IL-1β, COX-2, proopiomelanocortin, and prohormone-converting (PC) enzymes PC1/3 and PC2 mRNA in arthritic rats. In control rats, α-MSH did not modify food intake or hypothalamic expression of aforementioned mRNA. α-MSH prevented arthritis-induced increase in gastrocnemius COX-2, muscle-specific RING-finger protein-1 (MuRF1), and atrogin-1 expression, and it increased fast myofiber size. In conclusion our data show that in arthritic rats peripheral α-MSH treatment has an anti-cachectic action increasing food intake and decreasing muscle wasting.
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Dalix, E., M. Maalouf, A. Vanden-Bossche, M. Paul, and H. Marotte. "OP0208 MECHANICAL UNLOADING PREVENTED ARTHRITIS IN THE RAT ADJUVANT-INDUCED ARTHRITIS MODEL." Annals of the Rheumatic Diseases 82, Suppl 1 (May 30, 2023): 137.1–137. http://dx.doi.org/10.1136/annrheumdis-2023-eular.3762.

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BackgroundRheumatoid arthritis (RA) is the most common chronic inflammatory rheumatic disease, characterized by synovitis associated with progressive bone loss and joint swelling. The adjuvant-induced arthritis (AIA) model mimics the pathophysiological features of RA, with a very high prevalence and reproducibility. YAP plays a key role in synovial hyperplasia. It is a transcription factor which can be activated in response to inflammation but also by a mechanical stimulus as tissue stiffening. We already showed a decrease arthritis severity by inhibiting YAP in the AIA model [1]. Then, different mechanical challenges could potentially impact arthritis development by regulating YAP.ObjectivesThe objective of this study was to investigate the impact of mechanical loading and unloading on the development of arthritis in the AIA model.MethodsArthritis was induced in female Lewis rats by injection of the adjuvantMycobacterium butyricum, defining day (D)0. The AIA model normally develops arthritis at D10, with a peak of inflammation at D17 [2]. Rats were randomized into three groups: an AIA+mechanical loading group (n=11) with free access to an activity wheel from D0 to D17, an AIA+mechanical unloading group (n=11) by tail suspension from D0 to D17, and an AIA-only group (n=11) as positive control. Daily clinical monitoring (arthritic index and ankle circumference) was used to follow the progression and severity of arthritis. At D17, the ankles of the rats were collected to perform RT-qPCR.ResultsArthritis onset was observed at the same time (D10) in the AIA control and AIA+mechanical loading groups with the same kinetic of arthritis index and ankle circumferences. However, the majority of rats in the AIA+mechanical unloading group did not develop any signs of arthritis. At D17, gene expression of YAP and CYR61 (YAP target gene), IL1B, IL6, RANKL, ACP5 (encoding TRAPc), CTSK (encoding cathepsin K), MMP9, and MMP13 was decreased in the ankle of AIA+mechanical unloading rats compared to other groups. In the AIA+ mechanical loading group, rat stopped their physical activity at D10 which may explain the lack of clinical and molecular differences between the AIA and AIA+mechanical loading groups at D17.Figure 1.Arthritic index from day(D) 0 to D17 in the AIA control (CTR) group (n=11), the AIA+mechanical loading (WHEEL) group (n=11) and the AIA+mechanical unloading (SUSP) group (n=11).ConclusionMechanical unloading of the hindpaws by the suspension system strongly prevented arthritis by a reduced expression of pro-inflammatory genes, bone resorption, and bone degradation genes. The decrease in inflammation may be partly explained by the decrease in YAP transcriptional activity. Mechanical stress is therefore a key factor in inflammation during AIA. The precise mechanisms linking these two mechanisms are still under investigation.References[1]Caire R, Audoux E, Courbon G, Michaud E, Petit C, Dalix E, et al. YAP/TAZ: Key Players for Rheumatoid Arthritis Severity by Driving Fibroblast Like Synoviocytes Phenotype and Fibro-Inflammatory Response.Front Immunol2021;12:791907.[2]Courbon G, Cleret D, Linossier M-T, Vico L, Marotte H. Early Subchondral Bone Loss at Arthritis Onset Predicted Late Arthritis Severity in a Rat Arthritis Model: EARLY BONE LOSS AT ARTHRITIS.J Cell Physiol2017;232:1318–1325. Available at:http://doi.wiley.com/10.1002/jcp.25601. Accessed October 20, 2020.Acknowledgements:NIL.Disclosure of InterestsNone Declared.
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Gómez-SanMiguel, Ana Belén, Carolina Gomez-Moreira, María Paz Nieto-Bona, Carmen Fernández-Galaz, Maria Ángeles Villanúa, Ana Isabel Martín, and Asunción López-Calderón. "Formoterol decreases muscle wasting as well as inflammation in the rat model of rheumatoid arthritis." American Journal of Physiology-Endocrinology and Metabolism 310, no. 11 (June 1, 2016): E925—E937. http://dx.doi.org/10.1152/ajpendo.00503.2015.

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Adjuvant-induced arthritis is an experimental model of rheumatoid arthritis that is associated with body weight loss and muscle wasting. β2-adrenergic receptor agonists are powerful anabolic agents that trigger skeletal muscle hypertrophy and have been proposed as a promising treatment for muscle wasting in human patients. The aim of this work was to determine whether formoterol, a selective β2-adrenoreceptor agonist, is able to ameliorate muscle wasting in arthritic rats. Arthritis was induced in male Wistar rats by intradermal injection of Freund's adjuvant. Control and arthritic rats were injected daily with 50 μg/kg sc formoterol or saline for 12 days. Body weight change, food intake, and arthritis index were analyzed. After euthanasia, in the gastrocnemius mRNA was analyzed by PCR, and proteins were analyzed by Western blotting. Arthritis decreased gastrocnemius weight, cross-sectional area, and myofiber size, whereas formoterol increased those variables in both arthritic and control rats. Formoterol decreased the external signs of arthritis as well as NF-κB(p65) activation, TNFα, and COX-2 levels in the gastrocnemius of arthritic and control rats. Those effects of formoterol were associated with a decreased expression of myostatin, atrogin-1, and MuRF1 and in LC3b lipidation. Arthritis increased the expression of MyoD, myogenin, IGF-I, and IGFBP-3 and -5 in the gastrocnemius. In control and in arthritic rats, treatment with formoterol increased Akt phosphorylation and myogenin levels, whereas it decreased IGFBP-3 expression in the gastrocnemius. These data suggest that formoterol has an anti-inflammatory effect and decreases muscle wasting in arthritic rats through increasing Akt activity and myogenin and decreasing myostatin, the p-NF-κB(p65)/TNF pathway, and IGFBP-3.
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Jacobs, C., D. Young, S. Tyler, G. Callis, S. Gillis, and P. J. Conlon. "In vivo treatment with IL-1 reduces the severity and duration of antigen-induced arthritis in rats." Journal of Immunology 141, no. 9 (November 1, 1988): 2967–74. http://dx.doi.org/10.4049/jimmunol.141.9.2967.

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Abstract IL-1 has been implicated in the pathogenesis of arthritis. Although IL-1 injected in vivo into normal joints results in a transient inflammatory reaction, we have shown that three weekly repetitive injections of IL-1 do not produce a progressive inflammatory condition suggestive of chronic arthritis. In fact, priming normal joints with three weekly IL-1 intraarticular injections results in a significant reduction in joint swelling and diminished histopathologic alterations/lesions caused by subsequent methylated BSA-induced arthritis. Similarly, post treatment with IL-1 intraarticular injection after arthritis induction reduced arthritic swelling and joint injury if IL-1 was given during the developing stage of arthritis. Our results suggest that IL-1 might limit arthritic inflammation and progressive cartilage destruction through an, as yet, undetermined mechanism(s). Further in vivo investigations are required to determine the therapeutic utility of IL-1 in reducing early arthritic inflammation.
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Zhang, Kevin, June Liu, Jilin Deng, Liangtang Chang, Jian Shao, Jun Lu, Alison Bendele, Yunfeng Fu, and Jeff Duan. "Modeling human rheumatoid arthritis in NHP: Type II collagen induced arthritis in cynomolgus macaques (167.4)." Journal of Immunology 186, no. 1_Supplement (April 1, 2011): 167.4. http://dx.doi.org/10.4049/jimmunol.186.supp.167.4.

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Abstract Collagen induced arthritis (CIA) rodent models have been extensively used in rheumatoid arthritis (RA) research. An RA model in non-human primate (NHP) is particularly demanded because of the close phylogenesis that provides the cross-reactivity to human for different development compounds using most modern drug technologies. However, NHP RA model has been reported extremely difficult because of the low and inconsistent disease incidence. We studied type II collagen induced arthritis in Cynomolgus monkeys. Following immunization with collagen, the disease progression was monitored for 8 weeks. Overall the arthritic incidence reached 87% and the average arthritic incidence of proximal interphalangeal (PIP) joint reached near 90%, significantly higher than what was previously reported. The average swelling of PIP joint increased approximately by 45%. Radiography, histopathology and histomorphometry analysis of the joint bones well supported the arthritic disease with the similar characteristics of human RA joints. The average arthritic score was significantly reduced with the single agent treatment of Methotrexate or Dexamethasone. Our results demonstrated the successful establishment of an reliable CIA in Cynomolgus monkeys, providing a valuable tool for studies of RA disease in pathogenesis, biomarker, translational research, and most importantly, anti-arthritic therapeutics as well as other relevant diseases, such as anemia of chronic disease and arthritic pain.
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Faustino, Célia, Noélia Duarte, and Lídia Pinheiro. "Triterpenes Drug Delivery Systems, a Modern Approach for Arthritis Targeted Therapy." Pharmaceuticals 17, no. 1 (December 28, 2023): 54. http://dx.doi.org/10.3390/ph17010054.

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Arthritis is a major cause of disability. Currently available anti-arthritic drugs, such as disease-modifying anti-rheumatic drugs (DMARDs), have serious side-effects associated with long-term use. Triterpenoids are natural products with known anti-inflammatory properties, and many have revealed efficiency against arthritis both in vitro and in vivo in several animal models, with negligible cytotoxicity. However, poor bioavailability due to low water solubility and extensive metabolism upon oral administration hinder the therapeutic use of anti-arthritic triterpenoids. Therefore, drug delivery systems (DDSs) able to improve the pharmacokinetic profile of triterpenoids and achieve sustained drug release are useful alternatives for targeted delivery in arthritis treatment. Several DDSs have been described in the literature for triterpenoid delivery, including microparticulate and nanoparticulate DDSs, such as polymeric micro and nanoparticles (NPs), polymeric micelles, liposomes, micro and nanoemulsions, and hydrogels. These systems have shown superior therapeutic effects in arthritis compared to the free drugs and are similar to currently available anti-arthritic drugs without significant side-effects. This review focuses on nanocarriers for triterpenoid delivery in arthritis therapy, including osteoarthritis (OA), rheumatoid arthritis (RA) and gout that appeared in the literature in the last ten years.
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Dissertations / Theses on the topic "Arthritis"

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Riding, S. Barbara. "The arthritic pain experience of children with juvenile rheumatoid arthritis." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/27731.

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This study was designed to investigate the experience of having arthritic pain from the children's perspective. Previous research on how Canadian children perceive and manage arthritic pain and how it affects their daily lives is nonexistent. Therefore the purpose of this qualitative descriptive study was to explore and describe the arthritic pain experience of school age children with juvenile rheumatoid arthritis (JRA) and to understand the impact/influence of various factors on the construction of that experience. Ten children, aged 10 to 13 years, with either early (at 2 to 4 years) or late (at 7 to 11 years) onset arthritis participated in this study. Descriptive data were obtained during two open-ended in depth interviews with the children in their homes. Using content analysis, data were analyzed for themes and their elements. An analytical framework of themes and their elements was developed that reflected the children's descriptions of and explanations for arthritic pain in the context of their day to day in the context of their day to day living with arthritis, both in the past and currently. The children perceived pain to be synonymous with arthritis and the mediating factor in how they functioned. They described arthritic pain in relation to distinguishing factors: intensity, duration, and frequency. Intermittent arthritic pain was attributed to cessation of medications, arthritis "flare-ups," inactivity, and activity. A current concern for most children was pain attributed to activity because it meant limitations in activities with peers. The children identified strategies they used to manage pain and cope with pain's unpredictability. The findings of this study were discussed in relation to selected research studies that either supported or refuted the findings of this study. Implications for nursing practice and research were addressed.
Applied Science, Faculty of
Nursing, School of
Graduate
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Spilis, Angelica Abby. "Dancing With Arthritis." Master's thesis, Temple University Libraries, 2015. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/327134.

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Dance
M.A.
This Master of Arts thesis is based on research that I conducted on dancers who have the auto-immune disease of Rheumatoid Arthritis. Rheumatoid Arthritis is a long-term autoimmune disease that causes inflammation in the joints and the surrounding tissues. Dancers with arthritis feel pain the joints that can be minor or severe, depending on how they are moving their bodies. This research investigates how dancers with an arthritic body can dance without the experiencing pain in their joints. Arthritis impairs movement because it is a disease that affects the joints. In this thesis, I created movements that could enable arthritic dancers the opportunity to continue dancing. I have identified a movement vocabulary, movement methods, and strategies for arthritic dancers who want and need to move with minimal pain. Movements have been created specifically for the arthritic body. I use my own experiences and challenges as an arthritic dancer to inform this study. My experiences helped me to create movements specifically for arthritic dancers because I am an advocate for those who suffer from arthritis.
Temple University--Theses
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Hermann, Kay-Geert. "Die rheumatoide Arthritis." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2000. http://dx.doi.org/10.18452/14542.

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Multimedia - Wort des Jahres 1995 - taucht als populärer Begriff in allen Bereichen unserer Gesellschaft auf. Auch an Universitäten erhofft man sich durch die Einführung von computerunterstützten Lernformen eine höhere Qualität der Lehre sowie Kosteneinsparungen. Nach ersten Versuchen in den 60er Jahren war das Neue in den 90ern die realitätsnahe, multimediale Simulation von Entscheidungssituationen. Auf dem Gebiet der Rheumatologie ist derzeit jedoch noch ein Mangel an deutschsprachigen Softwaretiteln zu erkennen. Ziel war die Erstellung eines multimedialen Kompendiums über die rheumatoide Arthritis für den Einsatz in der universitären und postgraduierten Lehre. Das System soll als elektronisches Nachschlagewerk und als Basis für interaktive Diashows geeignet sein. Mit Hilfe eines Apple Macintosh und der Autorensoftware Macromedia Director wurde eine CD-ROM entwickelt, die sowohl für Macintosh- als auch für Windows-Computer geeignet ist. Die Beschreibung der Symptome der rheumatoiden Arthritis und der erforderlichen Untersuchungstechniken nimmt mit 31% der Bildschirmseiten den größten Teil des vorliegenden Multimedia-Kompendiums ein. Weitere Schwerpunkte wurden auf Pathogenese (19%), bildgebende Verfahren (14%), Differentialdiagnosen (11%), Therapie (10%) und Laboruntersuchungen (7%) gelegt. Videos und Animationen dienen der Illustration zellulärer Vorgänge und der Zusammenfassung klinischer Untersuchungstechniken. Etablierte Kriterienkataloge für elektronische Medien dienten der Qualitätssicherung im Entwicklungsprozeß. Eine parallel durchgeführte formative Evaluation lieferte erste Erkenntnisse über Praxistauglichkeit und Stabilität des Programmes, ohne jedoch eine fundierte summative Evaluation ersetzen zu können. Multimedia-Lehrbücher wie das vorliegende Kompendium stellen für den konventionellen Unterricht eine ideale Ergänzung zum klassischen Lehrbuch dar und dienen für die problemorientierte Ausbildung als schnell zur Verfügung stehende Wissensbasis. Jedoch blieben bei der fakultativen Nutzung von computerbasierten Lernmöglichkeiten in Lernzentren die Ergebnisse bisher hinter den Erwartungen zurück. Es ist zu diskutieren, inwieweit die Vorteile der Multimedia-Technologie durch gezielte Integration in das Curriculum an deutschen Hochschulen zu Kosten- und Zeitersparnissen führen können.
Multimedia - word of the year 1995 in Germany - is a popular term cropping up in all areas of society. Universities, too, hope to improve the quality of teaching and to cut costs by introducing computer-based forms of learning. Following initial attempts in the sixties, a new aspect introduced in the nineties was the life-like multimedia simulation of decision-making situations. In medicine, there still is a lack of German-language software packages in rheumatology. The aim of the present project was to develop a multimedia compendium on rheumatoid arthritis for teaching at the university and postgraduate level. The system was intended to serve both as an electronic work of reference and as a basis for interactive slide presentations. Using the authoring tool Macromedia Director on an Apple Macintosh computer, a CD-ROM was developed that can be run on Macintosh and Windows computers alike. The largest part of the multimedia compendium now available (31% of the screen pages) is dedicated to the description of the symptoms of rheumatoid arthritis and examination techniques. Other main areas are pathogenesis (19%), imaging modalities (14%), differential diagnoses (11%), therapy (10%), and laboratory tests (7%). Videos and animations serve to illustrate cellular processes and to summarize the clinical examination techniques. Catalogues of established criteria for electronic media were adhered to during development to assure quality. A simultaneously performed formative evaluation yielded initial results about the practicability and stability of the program but cannot replace a thorough summative evaluation. Multimedia textbooks such as the compendium presented here are ideal supplements to classical textbooks in conventional teaching, providing a rapidly accessible knowledge base for problem-oriented training. However, the results achieved with computer-based learning tools available for optional use at teaching centers have so far lagged behind expectations. It remains to be discussed to what extent the advantages of multimedia technology can save both cost and time by being selectively integrated into the curriculum at German universities.
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Matthee, Pierre Armand. "The quality of life of arthritis patients taking biologic arthritis medication." Diss., University of Pretoria, 2014. http://hdl.handle.net/2263/46176.

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The goal of the study was to explore and describe how arthritis patients experience the influence of arthritis on their quality of life after commencing with biologic arthritis medication. In order to achieve this goal, a qualitative research approach was adopted in an attempt to understand what the arthritis patients experience their quality of life to be while taking biologic arthritis medication. To achieve the research goal, the collective case study guided the research. Individual interviews, specifically semi-structured one-to-one interviews, with an interview schedule, were used as method of data collection. The researcher made use of Creswell’s (1998) qualitative data analysis process to analyse and to interpret the qualitative data. The trustworthiness of the data interpretation was confirmed through credibility, transferability, dependability and conformability. An analysis of the literature and transcripts of interviews was undertaken to answer the following research question: What is the influence of biologic arthritis medication on the quality of life of arthritis patients? The key findings of the study can be stipulated as follows: (1) Several signs and symptoms are associated with an arthritis patient, such as pain and fatigue; (2) Arthritis patients often visit several medical practitioners which can even be over a period of years before an official diagnosis of arthritis can be made; (3) A decrease of arthritis symptoms may be experienced during pregnancy, but increases again after the delivery; (4) Several types of arthritis treatments are available for managing the arthritis condition, ranging from traditional methods, such as anti-inflammatory medication, to biologic medication, such as Adalimumab (Humira); (5) A lack of knowledge pertaining to certain levels of medical doctors causes a mistrust amongst patients towards medical personnel; (6) Quality of life is a variable construct that is influenced by a patient’s culture and values; (7) Arthritis affects the patient’s ability to perform daily activities, such as washing; (8) Employment is important in making the patient feel human and therefore patients tend to hide their condition or to make adjustments at work, just to keep on working; (9) Biologic medication is seen as a miracle drug, albeit not totally without side effects, as patients are able to do things that were previously impossible; (10) Relationships are important in the life of an arthritis patient to enable coping, whether it be family, friends or the relationship with their medical practitioner; (11) Support provided by patients amongst themselves was also found to be important in order to facilitate coping with the disease; (12) Arthritis patients are encouraged to participate in physical exercise as it increases joint mobility; and lastly, (13) Biologic medication is quite expensive and patients are reliant on medical aids for funding of the treatment. In strengthening the role of social workers to assist arthritis patients to manage their disease better, the following recommendations are offered: (1) An awareness campaign facilitated by social workers, with experience in arthritic conditions, in collaboration with other health care workers in order to create awareness at different levels of society; (2) Social workers working in the field of arthritis should continuously strive toward improving the quality of life of arthritis patients they work with by setting up support networks and facilitating programmes that aim toward empowering those living with arthritis; (3) Social workers are encouraged to partner with rheumatology practices during which social workers are able to support newly diagnosed patients from the point of diagnosis. Social work support services could be in the form of counselling, or group support programmes; (4) A “fit for life” programme is recommended that is facilitated by social workers working with patients suffering from arthritic conditions with the goal of providing a safe environment where patients can be encouraged to be physically active. The aim will be to improve the patients’ quality of life experience, but also to create a safe environment for patients to support each other. (5) It is recommended that social workers working in the field of arthritis set up a database of patients that have proved to be involved in support programmes and shared their desire to provide guidance to newly diagnosed arthritis patients. The aim is then to partner a newly diagnosed arthritis patient with a more “senior” patient with a similar diagnosis and characteristics in order to establish a buddy support system. A context can then be created where the “senior” patient can share surviving techniques to the newly diagnosed patient but also provide assistance, for example picking up the children from school. Social workers are encouraged to then work closely with these buddies in order to provide further therapeutic support should it be required (6) Social workers working in the field of arthritis should always seek to advocate for arthritis patients when presenting at conferences and workshops (7) Investigate current, refine, or develop, policy related to the management and treatment of arthritis. Such policy should address aspects, including but not limited to, the employment conditions of people living with arthritis and securing the employment of people once diagnosed with the disease, medical aid and the requirements patients need to comply with in order to receive the full benefit provided to patients by medical aids, and thirdly, aspects related to the pricing (i.e., affordability) of disability insurance (8) The design and implementation of a continuous professional development programme is recommended to enable all health care workers to be continuously up to date with the latest developments related to arthritis research and management, in order to ensure that first line practitioners, for example physio-therapists and general practitioners, be equipped with the necessary skills to identify possible arthritis signs and symptoms which will ensure that patients are referred to specialist intervention as soon as possible. The sooner the patient receives the adequate level of care, the less joint deterioration may be sustained, and the higher the possibility to enjoy a good quality of life Future research could focus on initiating a research study that covers a more extensive geographical area, which is also more representative of the ethnic diversity in South Africa. Such a study could also cover more of the biologic medication used in the treatment of arthritis in order to reach a more holistic picture.
Dissertation (MSW)--University of Pretoria, 2014.
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Goertz, Christine [Verfasser], and Stephan [Gutachter] Meller. "Evaluation verkürzter MRT-Scores bei Patienten mit Rheumatoider Arthritis (Rheumatoid Arthritis MR Imaging Score (RAMRIS)) und Psoriasis-Arthritis (Psoriatic Arthritis MR Imaging Score (PsAMRIS)) / Christine Goertz ; Gutachter: Stephan Meller." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2021. http://d-nb.info/1233007572/34.

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Calander, Ann-Marie. "Proteases in staphylococcal arthritis /." Göteborg : Department of Rheumatology and Inflammation Research, Sahlgrenska Academy, Göteborg University, 2007. http://hdl.handle.net/2077/2584.

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Ulfgren, Ann-Kristin. "Cytokines in rheumatoid arthritis /." Stockholm, 2000. http://diss.kib.ki.se/2000/91-628-3823-7/.

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DeLaura, Angela. "Rheumatoid arthritis : an overview /." Online version of thesis, 1989. http://hdl.handle.net/1850/11502.

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Cruwys, Simon Charles. "Neurogenic influences on arthritis." Thesis, Queen Mary, University of London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243318.

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Edwards, Bryan Michael. "Collagen epitopes in arthritis." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.265003.

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Books on the topic "Arthritis"

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Powers, Mary C. Arthritis. New York: Chelsea House, 1992.

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Gold, Susan Dudley. Arthritis. Parsippany, N.J: Crestwood House, 1997.

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Publishing, DK. Arthritis. London: Dorling Kindersley Limited, 2006.

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Wyles, Mandy. Arthritis. London: Office of Health Economics, 1992.

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Terrass, Stephen. Arthritis. Oxford: ISIS, 1995.

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Chaitow, Leon. Arthritis. London: Thorsons, 1997.

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Schwarz, Shelley Peterman. Arthritis. New York: Demos Medical Publishing, 2009.

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1944-, Canfield Jack, Hansen Mark Victor, and Pisetsky David S, eds. Arthritis. Deerfield Beach, Fla: Health Communications, 2006.

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Dieppe, P. A. Arthritis. Wellingborough: Equation, 1988.

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Coleman, Vernon. Arthritis. Bath: Chivers, 1988.

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Book chapters on the topic "Arthritis"

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Order, Stanley E., and Sarah S. Donaldson. "Arthritis." In Radiation Therapy of Benign Diseases, 38. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-58719-1_19.

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Ash, Zoe, Sibel Zehra Aydin, Ai Lyn Tan, and Dennis McGonagle. "Arthritis." In Nail Psoriasis, 33–42. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-08810-5_5.

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Gilbert, Patricia. "Arthritis." In The A-Z Reference Book of Childhood Conditions, 16–21. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-7098-5_5.

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Viegas, Steven F. "Arthritis." In Hand Surgery Study Guide, 93–101. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-1910-1_10.

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Kuchynski, Marie. "Arthritis." In Encyclopedia of Women’s Health, 112–14. Boston, MA: Springer US, 2004. http://dx.doi.org/10.1007/978-0-306-48113-0_38.

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Bährle-Rapp, Marina. "Arthritis." In Springer Lexikon Kosmetik und Körperpflege, 48. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_823.

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O’Connor, Philip James, J. Farrant, Richard Hodgson, Kay-Geert A. Hermann, Nathalie Boutry, Xavier Demondion, Chadi Khalil, et al. "Arthritis." In Measurements in Musculoskeletal Radiology, 719–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-540-68897-6_18.

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Schroeder, Beth. "Arthritis." In Encyclopedia of Behavioral Medicine, 151–52. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1629.

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Abrams, David B., J. Rick Turner, Linda C. Baumann, Alyssa Karel, Susan E. Collins, Katie Witkiewitz, Terry Fulmer, et al. "Arthritis." In Encyclopedia of Behavioral Medicine, 128–29. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1629.

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Valencia, Elizabeth M. "Arthritis." In Encyclopedia of Immigrant Health, 217–19. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-5659-0_50.

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Conference papers on the topic "Arthritis"

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Chandran, Vinod. "Pathogenesis of Psoriatic Arthritis." In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/c9ea6682.

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Reis Gama, Cipriano, Heitor Furlan Giordano, Victor Matheus Ostrovski Souza Santos, Ana Cristina de Medeiros Ribeiro, and Karina Rossi Bonfiglioli. "Arthritis by Paracoccidioides Brasiliensis in rheumatoid arthritis." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1930.

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Prinz, Jörg C. "Inflammatory diseases – a new way of thinking." In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/a93901e6.

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McGrath, Conn, Mark Yates, Zenas Yiu, Sinead Langan, Teresa Tsakok, Nick Dand, Kayleigh Mason, et al. "Psoriasis and COVID-19: findings from PsoProtectMe." In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/819f9152.

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Sommer, Rachel, and Matthias Augustin. "How to improve people-centred healthcare in dermatology?" In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/27076e04.

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Bandyopadhyay, Dr Anisha, Dr Khawar Hussain, and Professor Anthony P. Bewley. "Psychodermatology and Psoriasis." In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/1be7e178.

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Bandyopadhyay, Dr Anisha, Dr Khawar Hussain, and Professor Anthony P. Bewley. "Psychodermatology and Psoriasis." In The 6th IFPA World Psoriasis and Psoriatic Arthritis Congress. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/bddfbdc3.

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Chen, Xi, and Fan Li. "Effects of Arthrigia on adjuvant arthritis rats' serum TNF-α." In 2011 International Conference on Human Health and Biomedical Engineering (HHBE). IEEE, 2011. http://dx.doi.org/10.1109/hhbe.2011.6027993.

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YAMAUCHI, Morio, Kazuhisa NAKANO, Yoshiya TANAKA, and Keiichi HORIO. "Predicting Disease Activity for Biologic Selection in Rheumatoid Arthritis." In 9th International Conference on Signal, Image Processing and Pattern Recognition (SPPR 2020). AIRCC Publishing Corporation, 2020. http://dx.doi.org/10.5121/csit.2020.101913.

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In this article, we implemented a regression model and conducted experiments for predicting disease activity using data from 1929 rheumatoid arthritis patients to assist in the selection of biologics for rheumatoid arthritis. On modelling, the missing variables in the data were completed by three different methods, mean value, self-organizing map and random value. Experimental results showed that the prediction error of the regression model was large regardless of the missing completion method, making it difficult to predict the prognosis of rheumatoid arthritis patients.
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Waller, Alexander J., Terence E. McIff, Mehmet Bilgen, E. Bruce Toby, and Kenneth J. Fischer. "Validation of MRI-Based Contact Modeling for Analysis of In Vivo Radiocarpal Mechanics." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176741.

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Arthritis is a pervasive problem and over 15% of the total population of the United States has been doctor-diagnosed with arthritis. Even more Americans have symptoms. Clearly, understanding the pathogenesis of arthritis, developing effective treatments and/or finding ways to prevent it are all important goals.
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Reports on the topic "Arthritis"

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Chen, Cheng-Che, and Chung-Jen Chen. New-Onset Inflammatory Arthritis After Covid-19 Vaccination. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, July 2022. http://dx.doi.org/10.37766/inplasy2022.7.0128.

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Review question / Objective: Investigate the new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic diseases and analyze their clinical patterns. Condition being studied: To help the readers to understand the clinical patterns of new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic diseases. Eligibility criteria: Inclusion criteria: publications of new-onset inflammatory arthritis after Covid-19 vaccination in patients without pre-existing autoimmune nor rheumatic diseases between January 2020 to March 2022. Exclusion criteria: cases with arthritis after SARS-CoV-2 infection and arthritis reactivation in those with underlying or history of arthritis-associated or autoimmune diseases.
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Kalinina, E. V., A. R. Babaeva, A. V. Levickaya, and M. S. Zvonorencko. MULTIMORBIDITY IN RHEUMATOID ARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-184-186.

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Fox, David A. Citrullinated Chemokines in Rheumatoid Arthritis. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada611994.

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Cockburn, Iain, and Aslam Anis. Hedonic Analysis of Arthritis Drugs. Cambridge, MA: National Bureau of Economic Research, May 1998. http://dx.doi.org/10.3386/w6574.

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Kalinina, E. V., E. V. Krivospitskaya, A. R. Babaeva, and M. S. Zvonorenko. COMORBIDITY IN PATIENTS WITH RHEUMATOID ARTHRITIS. "PLANET", 2019. http://dx.doi.org/10.18411/978-5-907192-54-6-2019-xxxvi-112-119.

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Deane, Keivn D. Pathogenesis and Prediction of Future Rheumatoid Arthritis. Fort Belvoir, VA: Defense Technical Information Center, October 2014. http://dx.doi.org/10.21236/ada613196.

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Zhigulina, K. V., and S. S. Spitsina. Carbohydrate imbalance in patients with gouty arthritis. DOI CODE, 2021. http://dx.doi.org/10.18411/wco-iof-esceo-2021-392-2.

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Russell, Mark, James Galloway, Sumera Qureshi, Joanna Ledingham, Arti Mahto, Andrew Rutherford, Maryam Adas, et al. Incidence and management of inflammatory arthritis in England before and during the COVID-19 pandemic. OpenSAFELY, January 2023. http://dx.doi.org/10.53764/rpt.ca5bce7991.

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The National Early Inflammatory Arthritis Audit (NEIAA) is the largest audit of its kind globally, reporting on care delivered across rheumatology services in the NHS in England. Clinical researchers from King’s College London are collaborating with OpenSAFELY to recreate key aspects of NEIAA, and benchmark the quality of care for people with inflammatory arthritis in England. This report will be updated on a regular basis.
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Mozgovaya, E. E., A. S. Trofimenko, M. A. Mamus, E. A. Tikhomirova, S. A. Bedina, and S. S. Spitsma. FORMATION OF MONOCYTE EXTRACELLULAR TRAPS IN RHEUMATOID ARTHRITIS. Academy of Natural Knowledge, 2019. http://dx.doi.org/10.18411/1996-3955-2019-10-86-89.

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Papichev, E. V., L. E. Seewordova, Y. R. Akhverdyan, Y. V. Polyakova, B. V. Zavodovsky, and O. D. Korolik. CLINICAL SIGNIFICANCE OF FETUIN-A IN RHEUMATOID ARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-219-225.

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