Academic literature on the topic 'Arteries'

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Journal articles on the topic "Arteries"

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Bahr Ulloa, Sandra, and Katia Guisado Zamora. "TRIPLICIDAD DE ARTERIAS RENALES DERECHAS CON DUPLICIDAD DE IZQUIERDAS: REPORTE DE CASO. Triplicity of right renal arteries and duplicity of left ones: Case report." Revista Argentina de Anatomía Clínica 11, no. 1 (March 24, 2019): 30–36. http://dx.doi.org/10.31051/1852.8023.v11.n1.22011.

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Durante la embriogénesis renal, ocurren fenómenos en su desarrollo que, de persistir en el adulto, se describen como variantes anatómicas. La presencia de arterias renales supernumerarias es una de variantes más frecuentes, pero la triplicidad de estas es rara. Este trabajo tiene como objetivo reportar un caso de arterias renales supernumerarias derechas e izquierdas. El hallazgo se produjo durante una sesión de disección en los laboratorios docentes de anatomía humana de la universidad. El caso consiste en un bloque anatómico de cadáver femenino del cual se aisló el segmento urogenital. El mismo fue disecado en fresco por el método macroscópico directo y luego conservado en formol por el método de Thiel durante 10 días, para luego ser examinado y fotografiado. El bloque consta de dos riñones, derecho e izquierdo, con sus vasos arteriales y venosos unidos a la arteria aorta abdominal y vena cava inferior respectivamente. Las arterias renales halladas fueron cinco, mostrando una triplicidad de arterias renales derechas con duplicidad de izquierdas. Las arterias renales derechas presentan similar calibre y dos de sus ramas son arterias polares superiores, en el caso de las arterias izquierdas la superior presentó mayor calibre. Asociada a esta variante se encontró la vena renal izquierda en posición posterior a la arteria renal inferior izquierda. Es importante para la práctica médica conocer las posibles variantes vasculares renales que pueden presentarse, para efectuar correctos procedimientos diagnósticos radiológicos, así como las planificaciones preoperatorias adecuadas de intervenciones quirúrgicas. During renal embryogenesis, phenomena occur in its development that, if persist in adult age, will be described as anatomical variant. The presence of supernumerary renal arteries is one of the most frequent variants, but the triplicity of these is rare. The objective of this work is to report a case of right and left supernumerary renal arteries. This finding appeared during a dissection session at the university's laboratory of human anatomy. The report case consists on an anatomical block of a female cadaver from which the urogenital piece was isolated. It was dissected fresh by the direct macroscopic method and then preserved in formaldehyde by the Thiel’s method for 10 days, to be examined and photographed. The block consisted on two kidneys, right and left, with their arterial and venous vessels attached to the abdominal aorta and inferior vena cava respectively. After the initial examination, five renal arteries were found, including a triplicity of right renal arteries and duplicity of left one. The right renal arteries have a similar caliber, with two branches as superior polar arteries. In regard to the left arteries, the superior artery presented greater caliber. Associated to these variants, left renal vein was in a posterior position in relation to the artery. It is important for medical practice to know the possible renal vascular variants that may occur, to carry out correct radiological diagnostic procedures and to adequately plan preoperative surgical interventions.
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Gajnitdinova, V. V., A. B. Bakirov, E. Kh Akhmetzyanova, N. F. Berdikaeva, and V. B. Zakirova. "Arterial stiffness of peripheral vasculature in patients with chronic obstructive pulmonary disease and its association with arterial hypertension." Kazan medical journal 94, no. 6 (December 15, 2013): 808–12. http://dx.doi.org/10.17816/kmj1795.

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Aim. To study the structural and functional state of vascular wall, arterial stiffness of large peripheral arteries (common carotid arteries, renal arteries) in patients with severe chronic obstructive pulmonary disease and its association with arterial hypertension. Methods. The study included 67 patients of working age, mainly males, having chronic obstructive pulmonary disease. Among them, 52 patients had severe chronic obstructive pulmonary disease (defined by GOLD III, 2011), 15 had concomitant arterial hypertension of I and II stage. Structural and functional status of common carotid arteries, renal arteries was assessed by measurement of intima-media thickness, arterial stiffness indexes were calculated. Arterial elasticity indices: arterial compliance, elastic index, Young’s elastic modulus were calculated based on the results of ultrasonography of main arterial wall parameters (diameter, arterial wall thickness) and blood pressure measurement. Results. A decrease in common carotid arteries and renal arteries wall elasticity was revealed in patients with chronic obstructive pulmonary disease. Increase of stiffness index in patients with severe chronic obstructive pulmonary disease associated with arterial hypertension, marking the decreased arterial wall elasticity, was registered both in common carotid arteries and renal arteries. Conclusion. In common carotid arteries vascular wall thickness contribute the most in vascular wall stiffness increase, compared to altered hemodynamics in renal arteries. Development of arterial hypertension in these patients is a predicting factor for further large vessel remodeling associated with hypoxia.
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Agematsu, Kota, Mitsugi Nagashima, Yoshiharu Nishimura, and Takashi Higaki. "Corridor technique for coronary arteries from a single arterial sinus." Asian Cardiovascular and Thoracic Annals 28, no. 6 (June 19, 2020): 333–35. http://dx.doi.org/10.1177/0218492320937506.

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The introduction of the arterial switch operation has improved the surgical outcome of transposition of the great arteries. However, coronary anomalies such as intramural coronary arteries, single coronary artery, or coronary arteries originating from a single arterial sinus have been reported as independent risk factors for early mortality and late morbidity after an arterial switch operation. We performed an arterial switch operation using a unique technique for translocation of the coronary arteries originating from a single left-side arterial sinus, to prevent coronary artery distortion and subsequent coronary malperfusion.
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Obeid, Hasan, Catherine Fortier, Charles-Antoine Garneau, Mathilde Pare, Pierre Boutouyrie, Rosa Maria Bruno, Hakim Khettab, Rémi Goupil, and Mohsen Agharazii. "Radial-digital pulse wave velocity: a noninvasive method for assessing stiffness of small conduit arteries." American Journal of Physiology-Heart and Circulatory Physiology 320, no. 4 (April 1, 2021): H1361—H1369. http://dx.doi.org/10.1152/ajpheart.00551.2020.

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Aortic stiffness, a cardiovascular risk factor and a marker of arterial aging, is assessed by pulse wave velocity (PWV) over this arterial segment. The interaction between the stiffness of various arterial segments is important in understanding the behavior of pressure and flow waves along the arterial tree. However, PWV assessment has been limited to large elastic vessels (aorta) or medium-sized arteries (i.e., brachial artery). In this paper, we provide a novel and noninvasive method of assessing the regional stiffness of small conduit arteries using the same piezoelectric sensors used for determination of PWV over large and medium-sized arteries. This development allows for an integrated approach to arterial stiffness from large to medium-sized arteries and now to small conduit arteries in humans.
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Mizutani, Tohru, Hideaki Kojima, and Yoshimasa Miki. "Arterial dissections of penetrating cerebral arteries causing hypertension-induced cerebral hemorrhage." Journal of Neurosurgery 93, no. 5 (November 2000): 859–62. http://dx.doi.org/10.3171/jns.2000.93.5.0859.

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Object. For the past 130 years, it has been believed that hypertension-induced cerebral hemorrhages are the result of ruptures of microaneurysms or ruptures of arteries that have degenerative changes. The majority of previous investigations have focused on autopsied brain. In this study, the authors attempted to verify the cause of hypertension-induced cerebral hemorrhage by using surgical specimens of the penetrating arteries responsible for the hemorrhages.Methods. Between 1997 and 1999, the authors performed pathological studies in surgical specimens of lenticulostriate arteries that had been confirmed during microsurgery to be the cause of hypertension-induced hemorrhage of the putamen. Nineteen lenticulostriate arteries were collected from 12 patients. Fifteen of these arteries were verified as the pathological causes of hemorrhage. They included six arterial dissections, six arterial ruptures with substantial degenerative changes, and three arterial ruptures with few degenerative changes. The pathological findings in the lenticulostriate artery dissections were similar to those of typical arterial dissections in major cerebral arteries.Conclusions. To the best of the authors' knowledge, arterial dissections of lenticulostriate arteries have not been identified as a cause of hypertension-induced cerebral hemorrhages. When penetrating arteries are included as causative vessels, cerebral arterial dissections may be much more common than previously thought.
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Synetos, Andreas, Antonios Karanasos, Stauros Spyropoulos, Maria Gazouli, George Latsios, Konstantinos Toutouzas, Despina Perrea, Dimitrios Tousoulis, and Ioannis A. Chloroyiannis. "Expression of Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 in Human Epicardial and Intramyocardial Coronary Arteries of Male Patients Undergoing Coronary Artery Bypass Grafting." Cardiology 139, no. 4 (2018): 203–7. http://dx.doi.org/10.1159/000486634.

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Objectives: Atherosclerosis is almost absent in intramyocardial coronary arteries, while epicardial coronary arteries may show extensive occlusive disease. Lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1) expression has been implicated in atherogenesis. We aimed to investigate differences in arterial wall LOX-1 expression between human epicardial and intramyocardial coronary arteries. Methods: Epicardial and intramyocardial total artery wall specimens were obtained from 13 male patients (aged 61.9 ± 10.3 years) undergoing coronary artery bypass graft surgery due to 3-vessel coronary artery disease. LOX-1 expression was examined by reverse transcription-polymerase chain reaction. Results: LOX-1 expression was significantly higher in the arterial wall of epicardial coronary arteries compared to intramyocardial coronary arteries. The LOX-1/GAPDH ratio was 0.48 ± 0.07 vs. 0.35 ± 0.03 (p < 0.001). Conclusions: Our findings may partially explain the atheroprotective effect of the intramyocardial course since arterial wall LOX-1 expression was lower in intramyocardial arteries and higher in epicardial coronary arteries.
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Bia, Daniel, Yanina Zócalo, Sandra Wray, and Edmundo I. Cabrera-Fischer. "Comparative in Vivo Analysis of the Role of the Adventitia and the Endothelium on Arterial Mechanical Function: Relevance for Aortic Counterpulsation." International Journal of Artificial Organs 40, no. 6 (May 24, 2017): 286–93. http://dx.doi.org/10.5301/ijao.5000585.

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Purpose The comparative effect of the intimal and adventitial layers on arterial biomechanics control, in basal and altered conditions, remains to be elucidated. This study aimed ( 1 ) to characterize the arterial conduit (CF) and buffering (distensibility) function of the iliac arteries in in vivo animals, in which the intimal and adventitial layers were removed; ( 2 ) to determine the effects of intra-aortic ballon pumping (IABP) on simultaneously de-adventitialized (DA) and de-endothelialized (DE) iliac arteries before and after induced heart failure. Methods Pressure and diameter signals were measured in the iliac arteries of sheep (n = 7) in which the adventitial and intima layer were removed. Intra-aortic balloon pump (IABP) assistance was used in a control state and after heart failure induction. Results Both DE and DA determined significant changes in arterial diameter, distensibility and CF. Changes were higher after DA than after DE in terms of distensibility and CF (p<0.05). DA followed by DE (DA + DE) showed significant increases in arterial diameter and CF, accompanied by a decrease in distensibility (p<0.05) with respect to intact arteries. Heart failure induction caused significant hemodynamic changes without modifying the already impaired local biomechanical parameters. Nonsignificant improvements in the biomechanical parameters of DA+ DE iliac arteries were observed during IABP before and after heart failure induction. Conclusions Biomechanical changes caused by DA of iliac arteries were more important than those observed after DE. The DA + DE arteries showed significant differences with respect to intact arteries and with DA or DE arteries. IABP-related effects on arterial mechanics were absent in DA+ DE arteries.
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Yen Ho, Siew, Gualtiero Catani, and Jeong-Wook Seo. "Arterial supply to the lungs in tetralogy of Fallot with pulmonary atresia or critical pulmonary stenosis." Cardiology in the Young 2, no. 1 (January 1992): 65–72. http://dx.doi.org/10.1017/s1047951100000603.

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SummaryThe arterial supply to the lungs in cases with tetralogy of Fallot with pulmonary atresia or critical pulmonary stenosis is mostly via the arterial duct or else by collateral arteries arising directly or indirectly from the aorta. Nine anatomical specimens with collateral arteries were studied by blunt dissection to determine the arterial supply to each pulmonary segment. The precise supply varied from case to case but, overall, nearly two-thirds of the pulmonary segments were connected to the central pulmonary arteries. A quarter were supplied exclusively by systemic-to-pulmonary collateral arteries, with the remainder having a dual supply. Coexistence of ductal connections and systemic-to-pulmonary collateral arteries was seen in one case. Anastomoses between vessels of different origins were observed up to the segmental level in eight of the nine cases. In four cases, the anastomoses formed arterial rings. The extent of pulmonary parenchyma supplied by arteries from different sources and the presence of arterial anastomoses in these malformations have implications on the planning of surgical repair.
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Lim, Kenneth, Guerman Molostvov, Maria Lubczanska, Simon Fletcher, Rosemary Bland, Thomas F. Hiemstra, and Daniel Zehnder. "Impaired arterial vitamin D signaling occurs in the development of vascular calcification." PLOS ONE 15, no. 11 (November 19, 2020): e0241976. http://dx.doi.org/10.1371/journal.pone.0241976.

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Conflicting data exists as to whether vitamin D receptor agonists (VDRa) are protective of arterial calcification. Confounding this, is the inherent physiological differences between human and animal experimental models and our current fragmented understanding of arterial vitamin D metabolism, their alterations in disease states and responses to VDRa’s. Herein, the study aims to address these problems by leveraging frontiers in human arterial organ culture models. Human arteries were collected from a total of 24 patients (healthy controls, n = 12; end-stage CKD, n = 12). Cross-sectional and interventional studies were performed using arterial organ cultures treated with normal and calcifying (containing 5mmol/L CaCl2 and 5mmol/L β-glycerophosphate) medium, ex vivo. To assess the role of VDRa therapy, arteries were treated with either calcitriol or paricalcitol. We found that human arteries express a functionally active vitamin D system, including the VDR, 1α-hydroxylase and 24-hydroxylase (24-OHase) components and these were dysregulated in CKD arteries. VDRa therapy increased VDR expression in healthy arteries (p<0.01) but not in CKD arteries. Arterial 1α-OHase (p<0.05) and 24-OHase mRNA and protein expression were modulated differentially in healthy and CKD arteries by VDRa therapy. VDRa exposure suppressed Runx2 and MMP-9 expression in CKD arteries, however only paricalcitol suppressed MMP-2. VDRa exposure did not modulate arterial calcification in all organ culture models. However, VDRa reduced expression of senescence associated β-galactosidase (SAβG) staining in human aortic-smooth muscle cells under calcifying conditions, in vitro. In conclusion, maladaptation of arterial vitamin D signaling components occurs in CKD. VDRa exposure can exert vasculo-protective effects and seems critical for the regulation of arterial health in CKD.
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BUND, Stuart J. "Spontaneously hypertensive rat resistance artery structure related to myogenic and mechanical properties." Clinical Science 101, no. 4 (September 14, 2001): 385–93. http://dx.doi.org/10.1042/cs1010385.

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This investigation related arterial structure to myogenic (pressure-dependent) contractile responses in resistance arteries from spontaneously hypertensive rats (SHRs) and Wistar-Kyoto (WKY) normotensive control rats under pressurized conditions in vitro. Femoral and mesenteric resistance arteries from either strain were cannulated and pressurized in an arteriograph for the determination of pressure-diameter relationships under passive and active conditions in the range 5-200mmHg transmural pressure. Arterial geometrical measurements were made under relaxed conditions at 100mmHg. Media thickness/lumen diameter (M/L) ratios were significantly increased in SHR femoral (5.00±0.44% compared with 3.63±0.34%; P<0.05) and mesenteric (4.40±0.29% compared with 2.62±0.23%; P<0.001) arteries compared with those from WKY rats. Maximum myogenic contractions, assessed as minimum normalized diameters, were not significantly different in SHR and WKY rat femoral (0.41±0.03 and 0.40±0.02 respectively) or mesenteric (0.56±0.02 and 0.63±0.03 respectively) arteries. Arterial mechanical analyses demonstrated that incremental elastic modulus is reduced in SHR mesenteric arteries, but is not significantly different in SHR femoral arteries, compared with those from WKY rats. Additionally, wall stress at estimated in vivo pressures under passive and active conditions are similar in SHR and WKY rat arteries. These data demonstrate that increased M/L ratios in resistance arteries from SHRs are not associated with increased maximum pressure-dependent contractile responses. Increased M/L ratios in resistance arteries from SHRs are not accounted for by increased vessel wall stiffness, but the hypertension-associated arterial geometrical abnormalities act to normalize wall stress in the face of increased arterial pressure.
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Dissertations / Theses on the topic "Arteries"

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Bass, David Hyman. "Infra-inguinal arterial bypass procedures at Groote Schuur Hospital, 1977-1983 : analysis and evaluation of results." Master's thesis, University of Cape Town, 1987. http://hdl.handle.net/11427/26248.

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Infra-inguinal arterial bypass is becoming increasingly popular as a means of alleviating incapacitating symptoms of atherosclerosis and other progressive diseases affecting the arteries of the lower limb. The role of this procedure in limb salvage is controversial but an aggressive attitude is emerging from many centres. It is accepted that the majority of atherosclerotic patients have a short life expectancy but reconstructive vascular surgery has an important role to play in improving their quality of life. Progress in infra-inguinal bypass surgery has centred mainly on the development of synthetic grafts but the perfonnance of autologous saphenous vein has not been bettered in terms of longterm results and cost-effectivity. The initial experience of infra-inguinal bypass at Groote Schuur Hospital, Cape Town, is examined retrospectively with the objective of demonstrating the possible influence of patient factors and specific surgical practices on the outcome of results.
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Peng, Siwei. "Medin Amyloid in Human Arteries and its Association with Arterial Diseases." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis (AUU) : Universitetsbiblioteket [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6700.

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Daly, Chris D. "Artificial arteries and bioreactors /." [St. Lucia, Qld.], 2005. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe19028.pdf.

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Evans, Richard James. "Neuronal control mesenteric arteries." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279879.

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Fan, Jinwu. "Dynamic Strength of Porcine Arteries." Thesis, Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/19853.

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The failure behavior of collagenous soft tissues is important for clinical problems of plaque rupture and trauma. Cyclic tests require high frequencies that may affect the strength properties of the soft tissues. Experimental results of mechanical response of blood vessels to physiologic loads can be used to model and predict plaque rupture and direct medical therapy or surgical intervention. The goal of the study is to measure the mechanical failure properties of arteries to determine if they are strain rate and cycle dependant and to measure the progressive damage of arteries with time dependent loading. Ring specimens of porcine carotid arteries were preconditioned and then pulled to failure. In all cases, the intima broke first. Ultimate stress increased as a weak function of increasing strain rates. The ultimate stress at 100 mm/s was 4.54 MPa, greater than the 3.26 MPa at 0.1 mm/s. Strain rates between 1 and 100 mm/s correspond to a cyclic frequency of 0.5 Hz to 5 Hz for fatigue testing. In contrast, ultimate strain in arteries was independent of strain rate over the range tested. The creep tests showed a logarithmic relationship between stress magnitude and stress duration for this soft tissue. The creep testing indicates that damage is accumulating above certain threshold stress levels. The values of ultimate strength showed a 35% increase after 10,000 cycling loading. In contrast, the ultimate strain had a 13% decrease after cycling and the difference was statistically significant with p=0.018. The testing results showed that there were no significant differences on strength among fresh arteries and arteries stored at 5¡ã C for up to two weeks. The test results may be useful for developing a mathematical model to predict the behavior of arterial soft tissues and may be extended to estimate fracture and fatigue in the atherosclerotic plaque cap.
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Chen, Changyi. "Intimal hyperplasia in endarterectomized arteries." Diss., Georgia Institute of Technology, 1996. http://hdl.handle.net/1853/25393.

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Priest, Rachel Michelle. "Vasorelaxant mechanism in pulmonary arteries." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243439.

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Gammack, D. "Blood flow in twisted arteries." Thesis, University of Surrey, 1998. http://epubs.surrey.ac.uk/844008/.

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The motivation for this research into flow in pipes with non-uniform geometry comes from physiological flows. It is now widely believed that haemodynamics plays an important role in the initiation and development of atherosclerosis. Experiments have shown that the preferred sites for atherogenesis are regions of low wall shear stress. The build-up of atherosclerotic plaques in the coronary arteries can lead to arterial blockage and coronary failure. Previous studies have examined uniformly curved pipes and, more recently, uniformly curved and twisted pipes. However, it is well known that the arterial system displays non-uniform, time-dependent geometry. The main objective of this thesis is to describe flow in various pipes with weakly non-uniform curvature and torsion, with a view to understanding the resulting wall shear stress distribution and velocity profiles. The work herein models the flow of an incompressible Newtonian fluid through a pipe whose curvature and torsion vary along the pipe. The governing equations are first derived, then solved for both steady and oscillatory pressure gradients. The solution of these equations involves asymptotic and numerical techniques. The effects due to the non-uniform geometry and possible applications to physiology are discussed. Finally, the effects of torsion upon fluid motion are studied from the Lagrangian viewpoint, using numerical particle tracking.
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Andreeva, V. S. "Arteries of the barrelled part." Thesis, Sumy State University, 2017. http://essuir.sumdu.edu.ua/handle/123456789/53936.

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Introduction. As well as all the mammals investigated earlier, in some sites of a trunk of a brain of the person have a combination rectilinear and a setevidny form in distribution of arterial vessels. Work purpose. To investigate a structure of internal arteries of a barreled part of a brain of the person.
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Shubat, Pamela Jane. "Monocrotaline toxicity and pulmonary arteries." Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184533.

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Monocrotaline is a pyrrolizidine alkaloid found in plants implicated in livestock and human poisoning. Laboratory rats given monocrotaline develop pulmonary hypertension and right heart hypertrophy in the weeks following administration of the chemical. Lung weight increases and right heart hypertrophy correlate with increased pulmonary artery pressure. Rats which consumed monocrotaline drinking water (20 mg/l) for only 4 days developed significant increases in lung and heart weights 14 days after exposure began. This exposure was equivalent to a dose of 15 mg/kg. Other treatment combinations of time (0-10 days exposure) and monocrotaline concentration (5-60 mg/l in drinking water) were tested. The accumulative dose calculated for each of the treatment combinations which produced toxicity was in the range of 15 to 20 mg/kg. Monocrotaline injury appears to be cumulative, but organ weight increases reverse once exposure is stopped. As pulmonary hypertension develops and pulmonary arteries hypertrophy, the force with which isolated pulmonary artery segments contract decreases. This is a loss of efficacy rather than potency to the contracting agents KCl, norepinephrine, and 5-hydroxytryptamine. Relaxation of arteries under conditions of potassium-return (a measure of Na⁺/K⁺ ATPase activity) was also altered by monocrotaline treatment. In vivo monocrotaline treatment had little effect on the force of K⁺-return relaxation. However, the rate at which arteries relaxed was significantly decreased following 4 days ingestion of monocrotaline drinking water (20 mg/l). In vitro ouabain treatment and endothelial injury also decreased the rate of K⁺-return relaxation. Another Na⁺/K⁺ ATPase activity, ⁸⁶Rb⁺ uptake, was decreased following monocrotaline treatment only when 5-hydroxytryptamine was present and only uptake associated with the endothelium was affected. These studies utilized a very low exposure to monocrotaline (4 days ingestion of 20 mg/l monocrotaline drinking water or 15 mg/kg) to produce toxicity in rats. Monocrotaline-induced toxicity measured 20 days after treatment included right heart and lung hypertrophy and decreased contractions of isolated pulmonary arteries. Monocrotaline treatment decreased the rate of Na⁺/K⁺ ATPase-dependent relaxation of isolated pulmonary arteries 4 days after treatment began.
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Books on the topic "Arteries"

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F, Barker Wiley, ed. The Arteries. Austin, TX: R.G. Landes, 1992.

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Halpern, William, John Bevan, Joseph Brayden, Harriet Dustan, Mark Nelson, and George Osol, eds. The Resistance Arteries. Totowa, NJ: Humana Press, 1994. http://dx.doi.org/10.1007/978-1-4757-2296-3.

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Dowe, David A., Massimo Fioranelli, and Paolo Pavone, eds. Imaging Coronary Arteries. Milano: Springer Milan, 2013. http://dx.doi.org/10.1007/978-88-470-2682-7.

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Oskar, Klotz. Fracture of arteries. Boston: [s.n., 1995.

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1869-1940, Abbott Maude E., ed. Patent ductus arteriosus with acute infective pulmonary endarteritis. [Philadelphia?: s.n., 1996.

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Ian, Taylor G., and Tempest Michael N, eds. Arteries of the skin. London: Churchill Livingstone, 1988.

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McColl, Elizabeth. Opening arteries: A novel. Charlotte, N.C: Mint Hill Books, 2010.

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Salmon, Michel. Arteries of the skin. Edited by Taylor G. Ian and Tempest Michael N. London: Churchill Livingstone, 1988.

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Safar, Michel. Arteries in clinical hypertension. Philadelphia: Lippincott-Raven, 1996.

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Rosenberg, Norman. CRC handbook of carotid artery surgery: Facts and figures. 2nd ed. Boca Raton, FL: CRC Press, 1994.

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Book chapters on the topic "Arteries"

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Abrams, David B., J. Rick Turner, Linda C. Baumann, Alyssa Karel, Susan E. Collins, Katie Witkiewitz, Terry Fulmer, et al. "Arteries." In Encyclopedia of Behavioral Medicine, 128. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_82.

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Baumann, Linda C., and Alyssa Ylinen. "Arteries." In Encyclopedia of Behavioral Medicine, 1–2. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6439-6_82-2.

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Baumann, Linda C., and Alyssa Ylinen. "Arteries." In Encyclopedia of Behavioral Medicine, 150–51. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_82.

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Takahashi, Shoki. "Intracranial Arterial System: Infratentorial Arteries." In Neurovascular Imaging, 131–88. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84882-134-7_3.

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Hughes, Graham, and Shirish Sangle. "The Arteries." In Hughes Syndrome: The Antiphospholipid Syndrome, 39–43. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-739-6_10.

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Uhler, Tara. "Ciliary Arteries." In Encyclopedia of Ophthalmology, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-35951-4_74-5.

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Schäberle, Wilhelm. "Peripheral Arteries." In Ultrasonography in Vascular Diagnosis, 45–163. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-02509-9_2.

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Dancygier, Henryk. "Hepatic Arteries." In Clinical Hepatology, 663–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-04519-6_8.

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Klumpp, Sherry A., Harold M. McClure, and Thomas B. Clarkson. "Atherosclerosis, Arteries." In Nonhuman Primates, 105–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84924-4_23.

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Real, Maribel, and Marta Burrel. "Visceral Arteries." In Learning Vascular and Interventional Radiology, 101–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-87997-8_5.

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Conference papers on the topic "Arteries"

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Oltean, Alina, and Victor H. Barocas. "Mechanical Properties Along the Branching Site of the Abdominal Aorta and Iliac Arteries." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19418.

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There is a great interest in studying arteries due to the prevalence of arterial diseases such as atherosclerosis and aneurysms, but much of the focus has been on straight arteries [1,2]. As arteries branch off, the fiber alignment and thickness of the arterial wall change. Smooth muscle cells orient helically in straight segments while multiple orientations have been seen at the branching region. As a result, straight segments of arteries vary mechanically from branched segments [2]. This study focused on the structural and mechanical properties of arteries at branching sites to observe the effect of the structural changes at bifurcations on mechanical behavior.
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Aguado-Sierra, J., K. H. Parker, J. E. Davies, D. Francis, A. D. Hughes, and J. Mayet. "Arterial pulse wave velocity in coronary arteries." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.259375.

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Aguado-Sierra, J., K. H. Parker, J. E. Davies, D. Francis, A. D. Hughes, and J. Mayet. "Arterial pulse wave velocity in coronary arteries." In Conference Proceedings. Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2006. http://dx.doi.org/10.1109/iembs.2006.4397539.

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Fierro, Cesar A., Kurtis Johnson, and Hai-Chao Han. "The Critical Buckling Pressure of Arteries." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176258.

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Artery stability is essential to normal arterial functions. This study examined the critical internal pressure at which arteries buckle. The critical pressure was found to be strongly related to the axial strain in the arteries.
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Early, Michael, and Daniel J. Kelly. "Why Are Rates of Restenosis Higher in Peripheral Arteries Than Coronary Arteries? A Computational Study." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205431.

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The deployment of stents in some arteries outside the heart has long been recognized as problematic. The use of more flexible shape memory alloy (SMA) stents has improved success rates, making stenting a more successful option than balloon angioplasty alone in peripheral arteries. However, restenosis rates are still considerably higher in peripheral arteries, such as the, femoral and popliteal, than in coronary arteries. As well as differences in dimensions, composition and material properties, peripheral arteries can be subjected to adverse mechanical environments. In addition to the pulsatile environment experienced by all arteries, some peripheral arteries experience combinations of compression, twisting and bending [1, 2]. The objective of this study is to investigate the differences between stenting in coronary and peripheral arteries with a view to determining which factors could be responsible for elevating restenosis rates. Specifically, it will be determined which, if any, of differences in (i) geometry and material properties of the arteries, (ii) the type of stent used, and (iii) the pulsatile and flexion driven mechanical environments could be responsible for either fracture of stents or higher arterial stress levels which could explain the higher rates of restenosis seen in peripheral arteries.
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Han, Hai-Chao, Raymond P. Vito, Kristin Michael, and David N. Ku. "Axial Stretch Increases Cell Proliferation in Arteries in Organ Culture." In ASME 2000 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2000. http://dx.doi.org/10.1115/imece2000-2516.

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Abstract To study the effect of axial stretch on vascular function and wall remodeling, porcine carotid arteries were cultured under conditions of physiological flow and elevated axial stretch in an ex vivo organ culture system. Smooth muscle cell proliferation was measured by bromodeoxyuridine index. Results showed that cell proliferation was significantly increased in the highly stretched arteries when compared to the normally stretched arteries. This may indicate the feasibility of stimulating new arterial growth by stretching natural arteries.
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Northcutt, Avione, and Hai-Chao Han. "Finite Element Analysis of Buckling of Arteries With Aneurysms." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206275.

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Tortuosity of arteries occurs when a normally straight artery starts to take on a twisted path. This is often referred to as tortuosity and kinking of arteries. This phenomenon occurs in arteries throughout the body including the aorta and cerebral [1, 2]. Arterial tortuosity is a potentially dangerous condition that can lead to cerebrovascular symptoms, ischemia, and stroke [3].
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Rachev, Alexander, and Rudolph Gleason. "Dynamic Instability of Arteries: Effects of Perivascular Tissue." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206467.

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To accomplish their physiological function, arteries have to remain patent under physiological loads, exhibit sufficient mechanical strength, and be capable of preserving their configuration with a sufficient reserve of safety. The latter requirement means that an artery has to be a mechanically stable structure. Among possible patterns of loss of stability is a global beam-like instability of arterial segments of sufficiently large length/diameter ratio. There are data from in vitro experiments that showed that a straight arterial segment can buckle under appropriate combinations of internal pressure and longitudinal stretch ratio and a few mathematical models were addresses to predict this phenomenon (cf. [1]). Conclusions from these studies were extended to explain the origin of arterial tortuosity observed in vivo that might be associated with serious arterial disorders such as atherosclerosis and hypertension. However, in the living organism arteries are subjected to periodic pressure, and therefore data from static experiments and conclusions from mathematical models using the static criterion for instability, namely the occurrence of bifurcation of equilibrium configuration termed as buckling, cannot be directly related to the response of arteries in vivo. A mathematical model for studying dynamic stability of arteries subjected to a longitudinal extension and a periodic pressure was proposed recently [2]. The dynamic criterion for instability was used, based on analyzing the small transverse vibrations of the vessel considered as a straight beam. Though generalization of the model to include the effect of perivascular tissue was discussed, no analysis had been performed in [2]. The objective of this study is to build a predictive model for examining mechanical stability of relatively long arterial segments subjected to a periodic pressure and longitudinal stretch with an account for the effects of perivascular tissue. The occurrence of instability is interpreted as a plausible cause of certain vascular disorders.
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Jin, Kazuyoshi, Ko Kitamura, Shunji Mugikura, Naoko Mori, Makoto Ohta, and Hitomi Anzai. "Evaluation of Normalization Methods in a Cerebral Artery Atlas for Automatic Labeling." In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-71097.

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Abstract An existence probability atlas has been used for automatic labeling of cerebral arteries. However, the number of arteries varies frequently because of image quality and individual variation of the artery structure. To moderate the influence of number imbalance on labeling accuracy, we propose a new normalized atlas for automatic labeling of cerebral artery centerlines. The number of arteries, which was obtained from magnetic resonance angiography, varies from 11 to 46 among the artery sites. Based on the centerline and diameter, the arterial volume was reconstructed into a voxel space for each subject. After superimposing arteries from 46 subjects, three normalization methods were compared: dividing by the number of subjects (N), by N and the arterial length (L), and by N and the arterial volume (V). To compare the labeling accuracy and precision, the summation of probability and labeling method was also used. The accuracy of all normalization methods was &gt; 85% in all arteries. The precision improved in some parts, with the atlas normalized by N-L and by N-V. The use of N-L and N-V changed the relative value of the existence probability among the parts. Consequently, some normalization methods changed the tendency toward misclassification, which changed the precision.
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Garcia, Justin R., Shawn D. Lamm, and Hai-Chao Han. "Buckling Behavior of Arteries Under Torsion." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53652.

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Arterial tortuosity is a phenomenon which is observed throughout the body and is associated with aging, diabetes, high blood pressure, and other vascular diseases [1]. Tortuous arteries significantly hinder blood flow which may lead to the development of atherosclerotic plaque buildup [2]. Blood vessels may also become twisted or demonstrate 3-D tortuous shapes when subject to large twist deformations such as during surgical implantation of vascular grafts, propeller flap procedures, stent-artery interactions, and sudden movements of the neck or limbs [4–6]. However, the twisting behavior of arteries is poorly understood.
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Reports on the topic "Arteries"

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Alshammari, Mohammed Kanan. Efficacy of Complementary and Alternative Medicine in Peripheral Arterial Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0001.

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Review question / Objective: To explore various CAM therapies available and to generate evidence that these therapies are effective for managing the disease. Condition being studied: Peripheral arterial disease (PAD) is described as the atherosclerotic process of arteries other than cerebral and coronary arteries i.e. the abdominal aorta, iliac, and arteries of the lower limb which leads to the narrowing and blocking of arteries. Information sources: An online systematic literature search will be done from the time of database inception from 5 electronic databases namely PubMed, Cochrane Central Register of Controlled Trials (CENTRAL), Ovid SP, ISI Web of Science, Elsevier Science Direct, and Wiley Online Library.
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Evan Pasha, Evan Pasha. Do healthy arteries equal a healthy brain? Experiment, June 2014. http://dx.doi.org/10.18258/2766.

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Zhang, Ruizhe, and Qingya Xie. A meta-analysis of cholesteryl ester transfer protein(CETP) gene rs708272(G>A) polymorphism in association with cornoary heart disease risk. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2023. http://dx.doi.org/10.37766/inplasy2023.6.0021.

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Review question / Objective: To seek the association of the CETP rs708272 polymorphism with CHD.To figure out if the carriers of allele rs708272-A reduce or increase the risk of CHD in comparison with carriers of allele rs708272-G under allele model, dominant model and recessive model. Condition being studied: The inclusion criteria of CHD:(1)the presence of stenosis≥50% in a minimum of one main segment of coronary arteries (the right coronary artery, left circumfex, or left anterior descending arteries) by coronary angiography.(2) symptoms representing angina pectoris, electrocardiographic changes, and elevations of cardiac enzymes based on the criteria of the World Health Organization. (3) a certifed record of coronary artery bypass graft or percutaneous coronary intervention were included in the study.The exclusion criteria of CHD :patients with congenital heart disease, cardiomyopathy, and valvular disease.Controls:the same populations as the cases and specifed to be without CAD, cardiovascular and cerebrovascular diseases, and peripheral atherosclerotic arterial disease.
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Sabri, Ethan. Atrioventricular Septal Defect (AVSD), Dextro-Transposition of the Great Arteries, and Tricuspid Valve Atresia. Ames (Iowa): Iowa State University, December 2023. http://dx.doi.org/10.31274/cc-20240624-259.

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Yang, Jianguo, Fuyu Zhao, Xinpeng Zhou, Yuying sun, Xueping Lun, Jiaojiao Cao, and Bing Fan. Survival and prognosis analysis of systemic lupus erythematosus-pulmonary hypertension: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2023. http://dx.doi.org/10.37766/inplasy2023.4.0017.

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Review question / Objective: The study aimed to evaluate survival rates and prognosis in systemic lupus erythematosus (SLE) patients with pulmonary hypertension (PH) using meta-analysis. (P: patients with SLE-PH; I: No intervention; C: No comparator; O: survival and prognosis; S: meta-analysis). Condition being studied: Pulmonary hypertension (PH) is a life-threatening condition characterized by elevated pulmonary arteries pressure due to increased pulmonary vascular resistance1. Symptoms of PH are nonspecific but typically include exertional dyspnea and fatigue. Systemic lupus erythematosus (SLE) is characterized by aberrant immune activity leading to variable clinical manifestations ranging from mild fatigue and joint pain to severe and life-threatening organ damage. Recent data from lupus registries have provided more accurate estimates of SLE incidence and prevalence, which showed Lupus is more common in non-white populations.
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Ananev, Vladimir Nikolaevich. THE EFFECT OF FIVE-DAY COLD ADAPTATION ON THE TONE OF THE ARTERIES OF THE MUSCULOSKELETAL REGION AND THE SMALL INTESTINE IN WINTER SPORTS ATHLETES. DOI CODE, 2023. http://dx.doi.org/10.18411/doicode-2023.140.

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Singer, Robert, Brandon Root, and Peter Morone. Arterial Line. Touch Surgery Simulations, June 2015. http://dx.doi.org/10.18556/touchsurgery/2015.s0047.

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Cordis. Carotid Arterial Stenting. Touch Surgery Simulations, 2017. http://dx.doi.org/10.18556/touchsurgery/2017.s0095.

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Young, Stanley, and Dennis So Ting Fong. Arterial Trip Length Characteristics. Purdue University, December 2017. http://dx.doi.org/10.5703/1288284316560.

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Young, Stanley, and Dennis So Ting Fong. Arterial Performance Measures Software. Purdue University, December 2017. http://dx.doi.org/10.5703/1288284316567.

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