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Journal articles on the topic 'ARRHYTHMOGENIC'

Author: Grafiati

Published: 1 April 2023

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1

Podgoršek, Blaž, Gregor Poglajen, Andraž Cerar, Matjaž Šinkovec, and Bojan Vrtovec. "Arrhythmogenic Cardiomyopathy." Slovenian Medical Journal 87, no. 11-12 (January 4, 2019): 599–618. http://dx.doi.org/10.6016/zdravvestn.2723.

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Arrhythmogenic cardiomyopathy (AC) is a genetic disease of the myocardium characterized by fibro-fatty replacement of the apoptotic myocardium. It primarily affects the right ventricle, however in advanced stages of the disease the left ventricle can also be significantly affected. AC is a challenging diagnosis, especially in the early stages of the disease, and should be considered in all patients presenting with palpitations, syncope or sudden cardiac death when other, more common causes of these symptoms/signs are excluded. In patients with suspected AC, evaluation according to the current Task Force Criteria should be applied to achieve optimal diagnostic yield. The main therapeutic concern in AC patients is the prevention of SCD, and thus all patients with established diagnosis have to be evaluated for potential ICD implantation, which is indicated in the majority of symptomatic patients. In this narrative review we aim to outline current knowledge on the pathophysiology, diagnosis and treatment strategies of AC.

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2

Priori, Silvia G., and Demetrio J. Santiago. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 12 (December 8, 2017): 1296–98. http://dx.doi.org/10.1161/circresaha.117.312211.

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3

Abraham, Terri. "Arrhythmogenic Mechanisms." AACN Advanced Critical Care 3, no. 1 (February 1, 1992): 157–65. http://dx.doi.org/10.4037/15597768-1992-1020.

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The arrhythmogenic mechanisms are the basis for the genesis of a wide variety of complex dysrhythmias that can arise in both pacemaker and nonpacemaker cells. Automaticity, or the ability to rhythmically and spontaneously depolarize cardiac cells, is normally the domain of the sinus node. Altered automaticity takes place when conduction is enhanced or abnormal. A second mechanism, reentry, refers to a phenomenon that occurs when an impulse is delayed within a pathway of slow conduction and then reenters surrounding tissue and produces another impulse. One-way conduction is necessary to produce a return route for the reentrant circuit. Lastly, late potentials are fragmented, low-amplitude electrical currents that occur at the terminal portion of the QRS complex or during the ST segment. Supraventricular and ventricular beats and tachydysrhythmias are the consequences of these mechanisms. Common contributing factors include but are not limited to hypoxia, hypercapnia, electrolyte disturbance, catecholamines, and pharmacotherapy

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4

Corrado, Domenico, Cristina Basso, and Daniel P. Judge. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 7 (September 15, 2017): 784–802. http://dx.doi.org/10.1161/circresaha.117.309345.

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5

Beffagna, Giorgia, Alessandro Zorzi, Kalliopi Pilichou, Martina Perazzolo Marra, Ilaria Rigato, Domenico Corrado, Federico Migliore, et al. "Arrhythmogenic Cardiomyopathy." European Heart Journal 41, no. 47 (November 9, 2020): 4457–62. http://dx.doi.org/10.1093/eurheartj/ehaa719.

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6

Mestroni, Luisa, and Orfeo Sbaizero. "Arrhythmogenic Cardiomyopathy." Circulation 137, no. 15 (April 10, 2018): 1611–13. http://dx.doi.org/10.1161/circulationaha.118.033558.

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7

Pilichou, Kalliopi, Connie R. Bezzina, Gaetano Thiene, and Cristina Basso. "Arrhythmogenic Cardiomyopathy." Circulation: Cardiovascular Genetics 4, no. 3 (June 2011): 318–26. http://dx.doi.org/10.1161/circgenetics.110.959031.

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8

Jallon, P. "Arrhythmogenic Seizures." Epilepsia 38 (November 1997): S43—S47. http://dx.doi.org/10.1111/j.1528-1157.1997.tb06127.x.

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9

Rawal, Aranyak S., Tara VanCleave, Neeraja Yedlapati, Jeffery E. Saffitz, William James Craigen, and John L. Jefferies. "Arrhythmogenic Ventricular Cardiomyopathy." JACC: Case Reports 3, no. 3 (March 2021): 438–42. http://dx.doi.org/10.1016/j.jaccas.2020.12.012.

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10

Priori, Silvia G. "Inherited Arrhythmogenic Diseases." Circulation Research 94, no. 2 (February 6, 2004): 140–45. http://dx.doi.org/10.1161/01.res.0000115750.12807.7e.

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11

Topaz, On. "Arrhythmogenic RV dysplasia." American Heart Journal 112, no. 6 (December 1986): 1343. http://dx.doi.org/10.1016/0002-8703(86)90380-7.

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12

Millane, Teri A., David E. Ward, and A. John Camm. "Is hypomagnesemia arrhythmogenic?" Clinical Cardiology 15, no. 2 (February 1992): 103–8. http://dx.doi.org/10.1002/clc.4960150210.

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13

Sadredini, Mani, Marie Haugsten Hansen, Michael Frisk, William E. Louch, Stephan E. Lehnart, Ivar Sjaastad, and Mathis Korseberg Stokke. "CaMKII inhibition has dual effects on spontaneous Ca2+ release and Ca2+ alternans in ventricular cardiomyocytes from mice with a gain-of-function RyR2 mutation." American Journal of Physiology-Heart and Circulatory Physiology 321, no. 2 (August 1, 2021): H446—H460. http://dx.doi.org/10.1152/ajpheart.00011.2021.

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Genetically increased RyR2 activity promotes arrhythmogenic Ca2+ release. Inhibition of CaMKII suppresses RyR2 activity and arrhythmogenic Ca2+ release. Suppression of RyR2 activity prolongs refractoriness of Ca2+ release. Prolonged refractoriness of Ca2+ release leads to arrhythmogenic Ca2+ alternans. CaMKII inhibition promotes Ca2+ alternans by prolonging Ca2+ release refractoriness.

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14

Mirmomen, Seyedeh Mojdeh, Andrew Jay Bradley, Andrew Ernest Arai, and Arlene Sirajuddin. "Arrhythmogenic left ventricular cardiomyopathy." BJR|case reports 6, no. 1 (March 2020): 20190079. http://dx.doi.org/10.1259/bjrcr.20190079.

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Arrhythmogenic ventricular cardiomyopathy (AVC) is a heritable heart muscle disorder characterized by fibrofatty infiltration of the myocardium. Intramyocardial fat deposition is considered arrhythmogenic and predisposes patients to life-threatening arrhythmias and sudden cardiac death. The classic subtype of AVC is characterized by fibrofatty replacement of the right ventricular myocardium (i.e. arrhythmogenic right ventricular cardiomyopathy). In advanced cases of arrhythmogenic right ventricular cardiomyopathy, the left ventricle may be involved as well. Predominantly left ventricular involvement by AVC is exceedingly rare and lack of specific diagnostic criteria as well as its potential cardiotoxic effect make its diagnosis challenging and of high importance.

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15

Tapoi, Laura, Alexandra Clement, Rodica Radu, and Radu Sascau. "Multimodality imaging in arrhythmogenic cardiomyopathy." Romanian Journal of Cardiology 31, no. 1 (March 31, 2021): 10–16. http://dx.doi.org/10.47803/rjc.2021.31.1.10.

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Arrhythmogenic cardiomyopathy, as it has been recently redefi ned, is characterized by progressive myocyte loss with fibrosis and fat infiltration of the myocardium, which finally leads to a broad clinical spectrum ranging from heart failure symptoms to sudden cardiac death. The diagnosis of arrhythmogenic cardiomyopathy is challenging particularly because of its heterogeneity in presentation, which varies from focal right ventricular involvement to biventricular or prominent left ventricular phenotype. In the past decades, the development of new electrocardiographic and imaging diagnostic criteria for arrhythmogenic cardiomyopathy constituted an important area of research and resulted in the elaboration of the Padua criteria. However, even with the widespread availability of modern imaging techniques, there is still a lack of awareness in the health care community and this pathology persist in being under-or misdiagnosed. Given the limited indication of endomyocardial biopsy for the diagnosis of arrhythmogenic cardiomyopathy, one can conclude that the progress that has been made in the last few years in the multimodality imaging field is of utmost importance for the early detection and proper treatment of patients with arrhythmogenic cardiomyopathy, providing valuable prognostic information.

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16

Murakawa, Y., K. Sezaki, T. Yamashita, Y. Kanese, and M. Omata. "Three-dimensional activation sequence of cesium-induced ventricular arrhythmias." American Journal of Physiology-Heart and Circulatory Physiology 273, no. 3 (September 1, 1997): H1377—H1385. http://dx.doi.org/10.1152/ajpheart.1997.273.3.h1377.

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To investigate the electrophysiological and electrocardiographic characteristics of ventricular arrhythmia due to abnormal repolarization, we studied the three-dimensional activation sequence of cesium-induced ventricular tachycardia (VT) in 10 anesthetized dogs using a 384-channel recording system. Seventeen monomorphic VT (mVT) and eight polymorphic VT (pVT) episodes induced by cesium chloride (2 or 3 mM/kg) were analyzed. Only a single arrhythmogenic focus was detected in most beats of VT, whereas two competing foci were temporarily observed in two episodes of pVT. The site of arrhythmogenic focus of mVT was the endocardium (5 of 17), the midmyocardium (4 of 17), or undetermined (8 of 17). Both endocardial and midmyocardial arrhythmogenic foci were also found in pVT, and most pVT (6 of 8) were associated with the transition of the site of arrhythmogenic focus. These results are consistent with the view that both myocardial muscle fibers and Purkinje cells can cause ventricular arrhythmia due to abnormal repolarization and that changing the site of arrhythmogenic focus is the main mechanism of pVT.

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17

Wichter, Thomas, Peter Milberg, Henry D. Wichter, and Dirk G. Dechering. "Pregnancy in arrhythmogenic cardiomyopathy." Herzschrittmachertherapie + Elektrophysiologie 32, no. 2 (May 25, 2021): 186–98. http://dx.doi.org/10.1007/s00399-021-00770-7.

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AbstractArrhythmogenic cardiomyopathy (AC) is a rare heart muscle disease with a genetic background and autosomal dominant mode of transmission. The clinical manifestation is characterized by ventricular arrhythmias (VA), heart failure (HF) and the risk of sudden cardiac death (SCD). Pregnancy in young female patients with AC represents a challenging condition for the life and family planning of young affected women. In addition to genetic mechanisms that influence the complex pathophysiology of AC, experimental and clinical data have confirmed the pathogenetic role of strenuous exercise and competitive sports in the early onset and rapid progression of AC symptoms and complications. Pregnancy and exercise share a number of physiological aspects of adaptation. In AC, both result in ventricular volume overload and myocardial stretch. Therefore, pregnancy has been postulated as a potential risk factor for HF, VA, SCD, and pregnancy-related obstetric complications in patients with AC. However, the available evidence on pregnancy in AC does not confirm this hypothesis. In most women with AC, pregnancies are well tolerated, uneventful, and follow a benign course. Pregnancy-related symptoms (VA, syncope, HF) and mortality, as well as obstetric complications, are uncommon in AC patients and range in the order of background populations and cohorts with AC and no pregnancy. The number of completed pregnancies is not associated with an acceleration of AC pathology or an increased risk of VA or HF during pregnancy and follow-up. Accordingly, there is no medical indication to advise against pregnancy in patients with AC. Preconditions include stability of rhythm and hemodynamics at baseline, as well as clinical follow-ups and the availability of multidisciplinary expert consultation during pregnancy and postpartum. Genetic counseling is recommended prior to pregnancy for all couples and their families affected by AC.

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18

Bhardwaj, Parveen, Minoo Sharma, and Neeraj Ganju. "Arrhythmogenic right ventricular cardiomyopathy." Nigerian Journal of Cardiology 12, no. 2 (2015): 142. http://dx.doi.org/10.4103/0189-7969.152035.

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19

Yu, Chao, Tian-Gang Zhu, Wen-Ling Liu, and Wen-Feng Huang. "Left-dominant Arrhythmogenic Cardiomyopathy." Chinese Medical Journal 129, no. 14 (July 2016): 1763–64. http://dx.doi.org/10.4103/0366-6999.185874.

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20

Bockeria, O. L., and T. G. Le. "Arrhythmogenic right ventricular dysplasia." Annaly aritmologii 12, no. 2 (June 30, 2015): 89–99. http://dx.doi.org/10.15275/annaritmol.2015.2.4.

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21

Chaulin, A. M., and D. V. Duplyakov. "Arrhythmogenic effects of doxorubicin." Complex Issues of Cardiovascular Diseases 9, no. 3 (September 28, 2020): 69–80. http://dx.doi.org/10.17802/2306-1278-2020-9-3-69-80.

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The article discusses the adverse arrhythmogenic effects of an antitumor drug – doxorubicin.Doxorubicin has a significant effect on the action potentials and ion currents of cardiomyocytes, the dynamics of intracellular calcium concentration.Oncological diseases are the leading causes of death and disability of the population, causing extremely high socio-economic damage. Among the many currently available drugs for the treatment of cancer, an important place is taken by the anthracycline antibiotic – doxorubicin. However, adverse concomitant effects on several organs and systems of the human body, in particular on the cardiovascular system, do not allow the full use of the high potential of doxorubicin`s antitumor effectiveness. Cardiotoxicity of doxorubicin is manifested in the form of electrocardiographic abnormalities and arrhythmias, degenerative cardiomyopathy and chronic heart failure. The authors consider the following arrhythmogenic effects of doxorubicin: the mechanisms of influence of doxorubicin on electrocardiographic parameters, the action potential of cardiomyocytes, cardiac ion currents and the dynamics of intracellular calcium concentration. The study and assessment of specific pathophysiological mechanisms of arrhythmogenic effects of doxorubicin is necessary for the development and justified use of cardioprotective measures.

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22

McCullough, Jocelyn, and Rohan G. Perera. "Arrhythmogenic Right Ventricular Cardiomyopathy." New England Journal of Medicine 387, no. 10 (September 8, 2022): e20. http://dx.doi.org/10.1056/nejmicm2119255.

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23

Jug, Juraj, and Martina Lovrić Benčić. "Arrhythmogenic right ventricular cardiomyopathy." Cardiologia Croatica 14, no. 1-2 (March 2019): 12–20. http://dx.doi.org/10.15836/ccar2019.12.

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24

Tseluyko, V. Y., and O. O. Butko. "Right ventricular arrhythmogenic cardiomyopathy." Medicine of Ukraine, no. 3(249) (May 7, 2021): 38–42. http://dx.doi.org/10.37987/1997-9894.2021.3(249).238044.

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The article describes the questions of prevalence, etiology and pathogenesis, clinical presentation, instrumental diagnostics of arrhythmogenic right ventricular cardiomyopathy (ARVC). Diagnostic criteria, sudden cardiac death risk stratification in patients with ARVC and basic approaches in the treatment of this disease are proposed.

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25

Grishina, N. V. "Arrhythmogenic right ventricular cardiomyopathy." PULMONOLOGIYA 32, no. 2 (April 13, 2022): 47–53. http://dx.doi.org/10.18093/0869-0189-2022-32-2s-47-53.

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The article provides current information about the clinical manifestations and diagnosis of arrhythmogenic right ventricular cardiomyopathy, highlights the genetic aspects of the disease, and analyzes the recent scientific research. A clinical case of diagnosis of this rare disease is given.

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26

Krahn, Andrew D., Arthur A. M. Wilde, Hugh Calkins, Andre La Gerche, Julia Cadrin-Tourigny, Jason D. Roberts, and Hui-Chen Han. "Arrhythmogenic Right Ventricular Cardiomyopathy." JACC: Clinical Electrophysiology 8, no. 4 (April 2022): 533–53. http://dx.doi.org/10.1016/j.jacep.2021.12.002.

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27

Nogami, Akihiko. "Arrhythmogenic Right Ventricular Cardiomyopathy." Japanese Journal of Electrocardiology 34, no. 3 (2014): 245–63. http://dx.doi.org/10.5105/jse.34.245.

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28

Gallo, Cristina, Alessandro Blandino, Carla Giustetto, Matteo Anselmino, Davide Castagno, Elena Richiardi, and Fiorenzo Gaita. "Arrhythmogenic right ventricular cardiomyopathy." Journal of Cardiovascular Medicine 17, no. 6 (June 2016): 418–24. http://dx.doi.org/10.2459/jcm.0000000000000354.

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29

Poloni, Giulia, Marzia De Bortoli, Martina Calore, Alessandra Rampazzo, and Alessandra Lorenzon. "Arrhythmogenic right-ventricular cardiomyopathy." Journal of Cardiovascular Medicine 17, no. 6 (June 2016): 399–407. http://dx.doi.org/10.2459/jcm.0000000000000385.

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30

Graziosi, Maddalena, and Claudio Rapezzi. "Right ventricular arrhythmogenic cardiomyopathy." Journal of Cardiovascular Medicine 18 (January 2017): e157-e160. http://dx.doi.org/10.2459/jcm.0000000000000470.

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31

Basso, Cristina, Domenico Corrado, Barbara Bauce, and Gaetano Thiene. "Arrhythmogenic Right Ventricular Cardiomyopathy." Circulation: Arrhythmia and Electrophysiology 5, no. 6 (December 2012): 1233–46. http://dx.doi.org/10.1161/circep.111.962035.

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32

Stoughton, Samuel Joseph. "Arrhythmogenic Right Ventricular Cardiomyopathy." Journal of Diagnostic Medical Sonography 25, no. 5 (August 25, 2009): 263–66. http://dx.doi.org/10.1177/8756479309344098.

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33

Corrado, Domenico, Mark S. Link, and Hugh Calkins. "Arrhythmogenic Right Ventricular Cardiomyopathy." New England Journal of Medicine 376, no. 1 (January 5, 2017): 61–72. http://dx.doi.org/10.1056/nejmra1509267.

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34

Dalal, Darshan, Khurram Nasir, Chandra Bomma, Kalpana Prakasa, Harikrishna Tandri, Jonathan Piccini, Ariel Roguin, et al. "Arrhythmogenic Right Ventricular Dysplasia." Circulation 112, no. 25 (December 20, 2005): 3823–32. http://dx.doi.org/10.1161/circulationaha.105.542266.

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35

HAMILTON, ROBERT M. "Arrhythmogenic Right Ventricular Cardiomyopathy." Pacing and Clinical Electrophysiology 32 (July 2009): S44—S51. http://dx.doi.org/10.1111/j.1540-8159.2009.02384.x.

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36

Johansson, Bengt, Christer Backman, Erik Tossavainen, and Michael Henein. "Unusual arrhythmogenic myocardial disease." International Cardiovascular Forum Journal 1, no. 4 (April 7, 2015): 195. http://dx.doi.org/10.17987/icfj.v1i4.52.

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37

Krejčí, Jan. "Arrhythmogenic right ventricular cardiomyopathy." Cor et Vasa 52, no. 7-8 (July 1, 2010): 405–8. http://dx.doi.org/10.33678/cor.2010.110.

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38

Widimský, Petr, and Rostislav Polášek. "Arrhythmogenic left ventricular dysplasia." Cor et Vasa 52, Suppl. (December 1, 2010): 12–14. http://dx.doi.org/10.33678/cor.2010.197.

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39

Ferrari, Victor A., and Craig H. Scott. "Arrhythmogenic Right Ventricular Cardiomyopathy:." Journal of Cardiovascular Electrophysiology 14, no. 5 (May 2003): 483–84. http://dx.doi.org/10.1046/j.1540-8167.2003.03130.x.

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40

Huynh, Karina. "Ketogenesis in arrhythmogenic cardiomyopathy." Nature Reviews Cardiology 17, no. 5 (February 25, 2020): 266. http://dx.doi.org/10.1038/s41569-020-0356-1.

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41

Fontaine, Guy. "Arrhythmogenic right ventricular dysplasia." Current Opinion in Cardiology 10, no. 1 (January 1995): 16–20. http://dx.doi.org/10.1097/00001573-199501000-00004.

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42

Fontaine, G., F. Fontaliran, J. L. Hébert, D. Chemla, O. Zenati, Y. Lecarpentier, and R. Frank. "ARRHYTHMOGENIC RIGHT VENTRICULAR DYSPLASIA." Annual Review of Medicine 50, no. 1 (February 1999): 17–35. http://dx.doi.org/10.1146/annurev.med.50.1.17.

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43

Fatkin, D., C. Thorburn, J. Hickie, and D. Kuchar. "Arrhythmogenic right ventricular dysplasia." Australian and New Zealand Journal of Medicine 21, no. 4 (August 1991): 451–53. http://dx.doi.org/10.1111/j.1445-5994.1991.tb01352.x.

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44

Fontaine, G., F. Fontaliran, and R. Frank. "Arrhythmogenic Right Ventricular Cardiomyopathies." Circulation 97, no. 16 (April 28, 1998): 1532–35. http://dx.doi.org/10.1161/01.cir.97.16.1532.

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45

Laptev, Dmitriy Nikitich, and Irina Anatol'evna Shmushkovich. "Arrhythmogenic effects of hypoglycemia." Diabetes mellitus 15, no. 1 (March 15, 2012): 25–30. http://dx.doi.org/10.14341/2072-0351-5975.

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Hypoglycemia is a frequent event in patients on insulin therapy. Current clinical and experimental evidence shows hypoglycemia tobe a cause of arrhythmia and, possibly, a link to increased mortality risk in patients with diabetes mellitus. This review addressesprobable mechanisms and pathogenic factors of arrhythmia development due to hypoglycemic events. We adduce data accumulatedon rates of hypoglycemia, as well as their correlation with cardiovascular and general mortality according to ACCORD, ADVANCEand VADT trials.

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46

John, A. S. "Arrhythmogenic right ventricular cardiomyopathy." Heart 90, no. 9 (September 1, 2004): 1102. http://dx.doi.org/10.1136/hrt.2003.030841.

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47

Basso, Cristina, Gaetano Thiene, Domenico Corrado, Annalisa Angelini, Andrea Nava, and Marialuisa Valente. "Arrhythmogenic Right Ventricular Cardiomyopathy." Circulation 94, no. 5 (September 1996): 983–91. http://dx.doi.org/10.1161/01.cir.94.5.983.

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48

Fontaine, G. "Arrhythmogenic right ventricular dysplasia." Heart 70, no. 3 (September 1, 1993): 293–94. http://dx.doi.org/10.1136/hrt.70.3.293.

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49

Gutierrez, P. S. "Arrhythmogenic right ventricular dysplasia." Heart 70, no. 3 (September 1, 1993): 294. http://dx.doi.org/10.1136/hrt.70.3.294.

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50

Basso, Cristina, Domenico Corrado, Frank I. Marcus, Andrea Nava, and Gaetano Thiene. "Arrhythmogenic right ventricular cardiomyopathy." Lancet 373, no. 9671 (April 2009): 1289–300. http://dx.doi.org/10.1016/s0140-6736(09)60256-7.

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