Relevant bibliographies by topics / ARRHYTHMOGENIC

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  1. Journal articles

Academic literature on the topic 'ARRHYTHMOGENIC'

Author: Grafiati
Published: 1 April 2023
Last updated: 12 June 2025

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Journal articles on the topic "ARRHYTHMOGENIC"

1

Podgoršek, Blaž, Gregor Poglajen, Andraž Cerar, Matjaž Šinkovec, and Bojan Vrtovec. "Arrhythmogenic Cardiomyopathy." Slovenian Medical Journal 87, no. 11-12 (2019): 599–618. http://dx.doi.org/10.6016/zdravvestn.2723.

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Abstract:
Arrhythmogenic cardiomyopathy (AC) is a genetic disease of the myocardium characterized by fibro-fatty replacement of the apoptotic myocardium. It primarily affects the right ventricle, however in advanced stages of the disease the left ventricle can also be significantly affected.
 AC is a challenging diagnosis, especially in the early stages of the disease, and should be considered in all patients presenting with palpitations, syncope or sudden cardiac death when other, more common causes of these symptoms/signs are excluded. In patients with suspected AC, evaluation according to the current Task Force Criteria should be applied to achieve optimal diagnostic yield.
 The main therapeutic concern in AC patients is the prevention of SCD, and thus all patients with established diagnosis have to be evaluated for potential ICD implantation, which is indicated in the majority of symptomatic patients.
 In this narrative review we aim to outline current knowledge on the pathophysiology, diagnosis and treatment strategies of AC.
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2

Priori, Silvia G., and Demetrio J. Santiago. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 12 (2017): 1296–98. http://dx.doi.org/10.1161/circresaha.117.312211.

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3

Abraham, Terri. "Arrhythmogenic Mechanisms." AACN Advanced Critical Care 3, no. 1 (1992): 157–65. http://dx.doi.org/10.4037/15597768-1992-1020.

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The arrhythmogenic mechanisms are the basis for the genesis of a wide variety of complex dysrhythmias that can arise in both pacemaker and nonpacemaker cells. Automaticity, or the ability to rhythmically and spontaneously depolarize cardiac cells, is normally the domain of the sinus node. Altered automaticity takes place when conduction is enhanced or abnormal. A second mechanism, reentry, refers to a phenomenon that occurs when an impulse is delayed within a pathway of slow conduction and then reenters surrounding tissue and produces another impulse. One-way conduction is necessary to produce a return route for the reentrant circuit. Lastly, late potentials are fragmented, low-amplitude electrical currents that occur at the terminal portion of the QRS complex or during the ST segment. Supraventricular and ventricular beats and tachydysrhythmias are the consequences of these mechanisms. Common contributing factors include but are not limited to hypoxia, hypercapnia, electrolyte disturbance, catecholamines, and pharmacotherapy
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4

Corrado, Domenico, Cristina Basso, and Daniel P. Judge. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 7 (2017): 784–802. http://dx.doi.org/10.1161/circresaha.117.309345.

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5

Beffagna, Giorgia, Alessandro Zorzi, Kalliopi Pilichou, et al. "Arrhythmogenic Cardiomyopathy." European Heart Journal 41, no. 47 (2020): 4457–62. http://dx.doi.org/10.1093/eurheartj/ehaa719.

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6

Mestroni, Luisa, and Orfeo Sbaizero. "Arrhythmogenic Cardiomyopathy." Circulation 137, no. 15 (2018): 1611–13. http://dx.doi.org/10.1161/circulationaha.118.033558.

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7

Pilichou, Kalliopi, Connie R. Bezzina, Gaetano Thiene, and Cristina Basso. "Arrhythmogenic Cardiomyopathy." Circulation: Cardiovascular Genetics 4, no. 3 (2011): 318–26. http://dx.doi.org/10.1161/circgenetics.110.959031.

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8

Jallon, P. "Arrhythmogenic Seizures." Epilepsia 38 (November 1997): S43—S47. http://dx.doi.org/10.1111/j.1528-1157.1997.tb06127.x.

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9

Rawal, Aranyak S., Tara VanCleave, Neeraja Yedlapati, Jeffery E. Saffitz, William James Craigen, and John L. Jefferies. "Arrhythmogenic Ventricular Cardiomyopathy." JACC: Case Reports 3, no. 3 (2021): 438–42. http://dx.doi.org/10.1016/j.jaccas.2020.12.012.

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10

Priori, Silvia G. "Inherited Arrhythmogenic Diseases." Circulation Research 94, no. 2 (2004): 140–45. http://dx.doi.org/10.1161/01.res.0000115750.12807.7e.

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Journal articles
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