Relevant bibliographies by topics / ARRHYTHMOGENIC

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'ARRHYTHMOGENIC'

Author: Grafiati
Published: 1 April 2023
Last updated: 25 July 2025

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Journal articles on the topic "ARRHYTHMOGENIC"

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Podgoršek, Blaž, Gregor Poglajen, Andraž Cerar, Matjaž Šinkovec, and Bojan Vrtovec. "Arrhythmogenic Cardiomyopathy." Slovenian Medical Journal 87, no. 11-12 (2019): 599–618. http://dx.doi.org/10.6016/zdravvestn.2723.

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Arrhythmogenic cardiomyopathy (AC) is a genetic disease of the myocardium characterized by fibro-fatty replacement of the apoptotic myocardium. It primarily affects the right ventricle, however in advanced stages of the disease the left ventricle can also be significantly affected.
 AC is a challenging diagnosis, especially in the early stages of the disease, and should be considered in all patients presenting with palpitations, syncope or sudden cardiac death when other, more common causes of these symptoms/signs are excluded. In patients with suspected AC, evaluation according to the cu
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Priori, Silvia G., and Demetrio J. Santiago. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 12 (2017): 1296–98. http://dx.doi.org/10.1161/circresaha.117.312211.

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Abraham, Terri. "Arrhythmogenic Mechanisms." AACN Advanced Critical Care 3, no. 1 (1992): 157–65. http://dx.doi.org/10.4037/15597768-1992-1020.

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The arrhythmogenic mechanisms are the basis for the genesis of a wide variety of complex dysrhythmias that can arise in both pacemaker and nonpacemaker cells. Automaticity, or the ability to rhythmically and spontaneously depolarize cardiac cells, is normally the domain of the sinus node. Altered automaticity takes place when conduction is enhanced or abnormal. A second mechanism, reentry, refers to a phenomenon that occurs when an impulse is delayed within a pathway of slow conduction and then reenters surrounding tissue and produces another impulse. One-way conduction is necessary to produce
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Corrado, Domenico, Cristina Basso, and Daniel P. Judge. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 7 (2017): 784–802. http://dx.doi.org/10.1161/circresaha.117.309345.

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Beffagna, Giorgia, Alessandro Zorzi, Kalliopi Pilichou, et al. "Arrhythmogenic Cardiomyopathy." European Heart Journal 41, no. 47 (2020): 4457–62. http://dx.doi.org/10.1093/eurheartj/ehaa719.

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Mestroni, Luisa, and Orfeo Sbaizero. "Arrhythmogenic Cardiomyopathy." Circulation 137, no. 15 (2018): 1611–13. http://dx.doi.org/10.1161/circulationaha.118.033558.

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Pilichou, Kalliopi, Connie R. Bezzina, Gaetano Thiene, and Cristina Basso. "Arrhythmogenic Cardiomyopathy." Circulation: Cardiovascular Genetics 4, no. 3 (2011): 318–26. http://dx.doi.org/10.1161/circgenetics.110.959031.

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Jallon, P. "Arrhythmogenic Seizures." Epilepsia 38 (November 1997): S43—S47. http://dx.doi.org/10.1111/j.1528-1157.1997.tb06127.x.

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Olivetti, Natalia, Luciana Sacilotto, and Marcelo Luiz Campos Vieira. "Arrhythmogenic Cardiomyopathy." JACC: Asia 5, no. 7 (2025): 924–26. https://doi.org/10.1016/j.jacasi.2025.05.010.

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Sadredini, Mani, Marie Haugsten Hansen, Michael Frisk, et al. "CaMKII inhibition has dual effects on spontaneous Ca2+ release and Ca2+ alternans in ventricular cardiomyocytes from mice with a gain-of-function RyR2 mutation." American Journal of Physiology-Heart and Circulatory Physiology 321, no. 2 (2021): H446—H460. http://dx.doi.org/10.1152/ajpheart.00011.2021.

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Genetically increased RyR2 activity promotes arrhythmogenic Ca2+ release. Inhibition of CaMKII suppresses RyR2 activity and arrhythmogenic Ca2+ release. Suppression of RyR2 activity prolongs refractoriness of Ca2+ release. Prolonged refractoriness of Ca2+ release leads to arrhythmogenic Ca2+ alternans. CaMKII inhibition promotes Ca2+ alternans by prolonging Ca2+ release refractoriness.
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Dissertations / Theses on the topic "ARRHYTHMOGENIC"

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BERNARDI, JOYCE. "Arrhythmogenic mechanisms in genetic channelopathies." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/153192.

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Introduzione: Recentemente, varianti minori di SNP del gene NOS1AP sono stati associati a prolungamento del QT e aumentata incidenza di morte improvvisa in pazienti LQT1. Il gene NOS1AP codifica per la proteina CAPON, che localizza NOS1 in prossimità del reticolo sarcoplasmico (SR). L' attività di NOS1 è importante per la modulazione mediate da NO di ICaL, dei canali RyR2 e di SERCA, interferendo così con la regolazione dell’handling del Ca2+ e la stabilità del SR. Perciò abbiamo ipotizzato che gli SNPs di NOS1AP possano alterare la localizzazione/funzione di NOS1 diminuendo la stabilità de
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Egdell, Robin Michael. "Arrhythmogenic phenomena in isolated cardiac myocytes." Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322380.

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Åström, Aneq Meriam. "Arrhythmogenic right ventricular cardiomyopathy : Is it right?" Doctoral thesis, Linköpings universitet, Klinisk fysiologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-70403.

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Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an inherited heart disease, where sudden cardiac death in young seemingly healthy persons may be the first symptom. There is a need for more sensitive and accurate diagnostic methods to detect signs of disease, at an early stage and in relatives of affected individuals. The aim of this thesis is the evaluation of new non-invasive modalities in assessment of right ventricular (RV) volume and function with focus on patients with ARVC. Clinical and non-invasive follow-up of fifteen patients with ARVC during a mean period of 8 years permitt
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Scridon, Alina. "Atrial fibrillation : insights concerning the arrhythmogenic substrate." Phd thesis, Université Claude Bernard - Lyon I, 2012. http://tel.archives-ouvertes.fr/tel-00933537.

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Atrial fibrillation is the most prevalent form of cardiac arrhythmia. Studies in animal modelshave provided important insights into arrhythmia mechanisms. However, to date, we do not dispose ofanimal models of spontaneous atrial arrhythmia.Thus, we aimed to develop a model of spontaneous atrial arrhythmia in rats and to assesspathophysiological mechanisms of these arrhythmias by using a multidisciplinary approach. We alsoaimed to assess the presence and the extent of inflammation and endothelial dysfunction, incriminatedin atrial fibrillation-related complications such as stroke, in atrial fib
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King, James Harmsworth. "Arrhythmogenic mechanisms in RYR2-P2328S murine hearts." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648837.

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Celeghin, Rudy. "Genomics in Arrhythmogenic Cardiomyopathy: exploring the complexity." Doctoral thesis, Università degli studi di Padova, 2019. http://hdl.handle.net/11577/3421844.

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Background Arrhythmogenic cardiomyopathy (AC) is a rare heart muscle disease characterized by fibrofatty myocardial replacement, prominent impairment of ventricular systolic function and arrhythmias. AC phenotypic spectrum was revealed wider than previously thought thanks to genotype–phenotype correlation. Combining multiple sources of clinical information, such as genetic, electrocardiographic, arrhythmic, morphofunctional, and histopathologic findings resulted the best approach to untangle the complexity of this disease. Aim The aim of our study was to assess AC genetic heterogeneity, app
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Cason, Marco. "Application of omic technologies in Arrhythmogenic Cardiomyopathy." Doctoral thesis, Università degli studi di Padova, 2018. http://hdl.handle.net/11577/3427282.

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Background. Arrhythmogenic cardiomyopathy (AC) is an inherited myocardial disease characterized by fibro-fatty replacement of the myocardium and life-threatening arrhythmias. This genetically and phenotypically heterogeneous condition, caused mainly by mutations in desmosomal genes (JUP, DSP, PKP2, DSG2 and DSC2), exhibits reduced penetrance making challenging the diagnosis and the identification of a molecular mechanism underlying disease pathogenesis. Aims. (1) To identify one or more altered molecular pathways in AC carriers of desmosomal mutations; (2) to evaluate the diagnostic role of J
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Lazzarini, Elisabetta. "Diagnostic Implications of Arrhythmogenic Cardiomyopathy Genetic Testing." Doctoral thesis, Università degli studi di Padova, 2015. http://hdl.handle.net/11577/3424161.

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Background & Aims: Arrhythmogenic Cardiomyopathy (AC) is a rare inherited heart muscle disease associated with mutations in genes encoding mainly components of the cardiac desmosome. We performed a comprehensive study of genetic variants in a cohort of AC subjects and the assessment of Next Generation Sequencing (NGS) strategies for molecular diagnosis of AC. Methods: Ninety-nine unrelated index cases, of which 26 sudden cardiac death cases, underwent genetic screening for 5 desmosomal genes by denaturing high performance liquid chromatography and direct sequencing, whereas 46 probands were a
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Spier, Alan W. "The electrocardiographic evaluation of arrhythmogenic cardiomyopathy in boxers /." The Ohio State University, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=osu1486462702466123.

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Bueno, Marinas Maria. "MicroRNA profiling in Arrhythmogenic Cardiomyopathy and prognostic markers." Doctoral thesis, Università degli studi di Padova, 2018. http://hdl.handle.net/11577/3427265.

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Background: Arrhythmogenic cardiomyopathy (AC) is a clinically and genetically heterogeneous myocardial disease, characterised by a progressive myocardial dystrophy with fibro-fatty replacement, and represents one of the major causes of sudden cardiac death in the young and athletes. Although half of AC patients harbour private desmosomal gene mutations, their low and age-dependent penetrance suggests the involvement of other regulatory molecules. MicroRNAs (miRNAs) are a group of endogenous short noncoding RNAs that regulate gene expression by sequence-specific recognition of their target tra
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Books on the topic "ARRHYTHMOGENIC"

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Markus, Frank I., Andrea Nava, and Gaetano Thiene, eds. Arrhythmogenic RV Cardiomyopathy/Dysplasia. Springer Milan, 2007. http://dx.doi.org/10.1007/978-88-470-0490-0.

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Andrea, Nava, Rossi Lino, and Thiene Gaetano, eds. Arrhythmogenic right ventricular cardiomyopathy/dysplasia: Proceedings of the 1st International Symposium on [T.B.A.]. Elsevier, 1997.

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Harmon, Jordan, Tufts-New England Medical Center. Evidence-based Practice Center., and United States. Agency for Healthcare Research and Quality., eds. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2004.

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United States. Agency for Healthcare Research and Quality, ed. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, 2004.

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United States. Agency for Healthcare Research and Quality., ed. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, 2004.

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Harmon, Jordan, Tufts-New England Medical Center. Evidence-based Practice Center, and United States. Agency for Healthcare Research and Quality, eds. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2004.

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Harmon, Jordan, Tufts-New England Medical Center. Evidence-based Practice Center, and United States. Agency for Healthcare Research and Quality, eds. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2004.

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Harmon, Jordan, Tufts-New England Medical Center. Evidence-based Practice Center., and United States. Agency for Healthcare Research and Quality., eds. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2004.

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Harmon, Jordan, Tufts-New England Medical Center. Evidence-based Practice Center., and United States. Agency for Healthcare Research and Quality., eds. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, U.S. Dept. of Health and Human Services, 2004.

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United States. Agency for Healthcare Research and Quality., ed. Effects of omega-3 fatty acids on arrhythmogenic mechanisms in animal and isolated organ/cell culture studies. Agency for Healthcare Research and Quality, 2004.

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Book chapters on the topic "ARRHYTHMOGENIC"

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Proost, V. M., and Arthur A. Wilde. "Arrhythmogenic Cardiomyopathy." In Electrocardiography of Inherited Arrhythmias and Cardiomyopathies. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-52173-8_7.

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Cox, Moniek G. P. J., Ardan M. Saguner, Anneline S. te Riele, et al. "Arrhythmogenic Cardiomyopathy." In Clinical Cardiogenetics. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-44203-7_6.

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Cox, Moniek G. P. J., Anneline S. J. M. te Riele, and Richard N. W. Hauer. "Arrhythmogenic Cardiomyopathy." In Clinical Cardiogenetics. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-45457-9_6.

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Vaideeswar, Pradeep. "Arrhythmogenic Cardiomyopathy." In Tropical Cardiovascular Pathology. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3720-0_44.

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Cipriani, Alberto, Antonio De Luca, Antonio Curcio, and Martina Perazzolo Marra. "Arrhythmogenic Cardiomyopathies." In Case-based Atlas of Cardiovascular Magnetic Resonance. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-32593-9_8.

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Horacek, B. Milan, Terrence J. Montague, Martin J. Gardner, and Eldon R. Smith. "Arrhythmogenic Conditions." In Body Surface Electrocardiographic Mapping. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4613-1769-2_13.

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Scharnagl, Hubert, Winfried März, Markus Böhm, et al. "Arrhythmogenic Cardiomyopathy." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_7470.

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Basso, Cristina, Monica De Gaspari, Stefania Rizzo, and Gaetano Thiene. "Arrhythmogenic Cardiomyopathy." In Sport-related sudden cardiac death. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80447-3_5.

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Hedrich, Olaf, Gianfranco Buja, Domenico Corrado, Mark S. Link, Thomas Wichter, and N. A. Mark Estes. "The Role of the Implantable Cardiac Defibrillator in the Management." In Arrhythmogenic RV Cardiomyopathy/Dysplasia. Springer Milan, 2007. http://dx.doi.org/10.1007/978-88-470-0490-0_21.

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Czarnowska, Elzbieta, Mila Della Barbera, Gaetano Thiene, Marialuisa Valente, and Cristina Basso. "Ultrastructural Substrates." In Arrhythmogenic RV Cardiomyopathy/Dysplasia. Springer Milan, 2007. http://dx.doi.org/10.1007/978-88-470-0490-0_7.

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Conference papers on the topic "ARRHYTHMOGENIC"

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A, Manjunatha Sathish, S. Jayanthy, Judeson Antony Kovilpillai J, Viyaskrishna S, Senthamil Arasi T, and Sudharsan S. "AC-GAN and Machine learning based Arrhythmogenic Right Ventricular Dysplasia detection system." In 2024 4th Asian Conference on Innovation in Technology (ASIANCON). IEEE, 2024. https://doi.org/10.1109/asiancon62057.2024.10838090.

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Iacono, Francesca Lo, Anna Slättman, Uzma Chaudhry, Pyotr P. Platonov, and Valentina D. A. Corino. "An ECG-based model to distinguish cardiac sarcoidosis from arrhythmogenic right ventricular cardiomyopathy." In 2024 13th Conference of the European Study Group on Cardiovascular Oscillations (ESGCO). IEEE, 2024. http://dx.doi.org/10.1109/esgco63003.2024.10767069.

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Nasir, U., T. A. Waheed, K. Syed, and R. Reddy. "Pulmonary Arterial Hypertension and Arrhythmogenic Right Ventricular Cardiomyopathy." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a3517.

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Vives-Gilabert, Yolanda, Jorge Sanz, Antonio Cebrián, et al. "Dyssynchrony Assessment in Arrhythmogenic Cardiomyopathy With Left Ventricular Involvement." In 2018 Computing in Cardiology Conference. Computing in Cardiology, 2018. http://dx.doi.org/10.22489/cinc.2018.073.

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CHEN, YAO-CHANG, YI-JEN CHEN, SHIH-ANN CHEN, and CHENG-I. LIN. "LOSARTAN DECREASES THE ARRHYTHMOGENIC ACTIVITY OF PULMONARY VEIN CARDIOMYOCYTE." In Proceedings of the 31st International Congress on Electrocardiology. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702234_0011.

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Ščurek, Martin, Martin Pešl, Jan Přibyl, et al. "Arrhythmogenic and chronotropic effects of bronchodilator drugs on human cardiomyocytes." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3297.

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Cohen, Mark H., Giora Ben-Shachar, Stanley D. Beder, Mark Sivakoff, and Thomas A. Riemenschneider. "Epicardial Application Of Laser Energy In Vivo: Acute Arrhythmogenic Potential." In 29th Annual Technical Symposium, edited by Abraham Katzir. SPIE, 1986. http://dx.doi.org/10.1117/12.950733.

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Vives-Gilabert, Yolanda, Bego�a Igual, Santiago Jim�nez-Serrano, et al. "Bayesian Classification Applied to Strain in Arrhythmogenic Left-Ventricle Cardiomyopathy." In 2017 Computing in Cardiology Conference. Computing in Cardiology, 2017. http://dx.doi.org/10.22489/cinc.2017.079-088.

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Bocharin, I., A. Martusevich, and L. Dilenyan. "COMPLEX SCRINING OF SPECIALTIES OF HEART RATE VARIABILITY IN STUDENTS AT MEGAPOLIS." In XIV International Scientific Conference "System Analysis in Medicine". Far Eastern Scientific Center of Physiology and Pathology of Respiration, 2020. http://dx.doi.org/10.12737/conferencearticle_5fe01d9c9d03c8.92975024.

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The aim of the study was to study the state of systemic hemodynamics in students of the main higher educational institutions of Nizhny Novgorod during the inter-sessional period. It was found that this group of individuals has good adaptive reserves, but some of the surveyed students have signs of sympathicotonia and a moderate risk of arrhythmogenic events
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Wonham, Andrew, and Jan Till. "1396 Arrhythmogenic flecainide toxicity in Neonatal Atrio-ventricular Re-entry Tachycardia." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference, Liverpool, 28–30 June 2022. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2022. http://dx.doi.org/10.1136/archdischild-2022-rcpch.695.

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Reports on the topic "ARRHYTHMOGENIC"

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Shalganov, Tchavdar, Milko Stoyanov, and Vassil Traykov. Outcomes following catheter ablation for ventricular tachycardia in adult patients with structural heart disease and implantable cardioverter-defibrillator: protocol for an updated systematic review and meta-analysis of randomized studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.6.0080.

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Review question / Objective: Does catheter ablation for scar-related monomorphic ventricular tachycardia improve outcomes (defined as any appropriate ICD therapy, appropriate ICD shocks, all-cause mortality, VT storm, cardiovascular mortality, cardiovascular hospitalizations, complications) in adult patients with ischemic or non-ischemic cardiomyopathy and implantable cardioverter-defibrillator? Condition being studied: Ventricular tachycardia in patients with structural heart disease is usually an arrhythmia using the myocardial scar as a substrate for reentry. It poses a risk of syncope and
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