Relevant bibliographies by topics / Aquatic toxicology

Academic literature on the topic 'Aquatic toxicology'

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Published: 4 June 2021
Last updated: 20 February 2023

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Journal articles on the topic "Aquatic toxicology"

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Moore, Michael R., and Glen Shaw. "Aquatic Toxicology." Therapeutic Drug Monitoring 22, no. 1 (February 2000): 58–60. http://dx.doi.org/10.1097/00007691-200002000-00012.

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Wester, P. W., L. T. M. van der Ven, A. D. Vethaak, G. C. M. Grinwis, and J. G. Vos. "Aquatic toxicology:." Environmental Toxicology and Pharmacology 11, no. 3-4 (July 2002): 289–95. http://dx.doi.org/10.1016/s1382-6689(02)00021-2.

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Mebs, D. "Aquatic Toxicology." Toxicon 23, no. 2 (January 1985): 355–56. http://dx.doi.org/10.1016/0041-0101(85)90164-3.

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Fawcett, H. H. "Aquatic toxicology." Journal of Hazardous Materials 10, no. 1 (February 1985): 154–55. http://dx.doi.org/10.1016/0304-3894(85)80016-9.

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Malins, D. C., and G. K. Ostrander. "Perspectives in Aquatic Toxicology." Annual Review of Pharmacology and Toxicology 31, no. 1 (April 1991): 371–99. http://dx.doi.org/10.1146/annurev.pa.31.040191.002103.

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Cairns, John Jr, and Donald I. Mount. "Aquatic toxicology. Part 2." Environmental Science & Technology 24, no. 2 (February 1990): 154–61. http://dx.doi.org/10.1021/es00072a001.

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Nikinmaa, Mikko, and Daniel Schlenk. "Genomics in Aquatic Toxicology." Aquatic Toxicology 97, no. 3 (May 2010): 173. http://dx.doi.org/10.1016/j.aquatox.2009.12.009.

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Widdows, J. "Fundamentals of aquatic toxicology." Fisheries Research 4, no. 3-4 (December 1986): 355–56. http://dx.doi.org/10.1016/0165-7836(86)90014-7.

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Rico Martínez, Roberto. "An Introduction to Aquatic Toxicology [Introducción a la Toxicología acuática]." Investigación y Ciencia de la Universidad Autónoma de Aguascalientes, no. 72 (November 29, 2017): 131–32. http://dx.doi.org/10.33064/iycuaa201772231.

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Isomaa, Boris, Henrik Lilius, and Christina Råbergh. "Aquatic Toxicology in Vitro: A Brief Review." Alternatives to Laboratory Animals 22, no. 4 (July 1994): 243–53. http://dx.doi.org/10.1177/026119299402200405.

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There is an urgent need for effective in vitro tests in aquatic toxicology, because only a very small proportion of the chemicals in common use have been adequately tested for their toxicity to aquatic organisms and aquatic ecosystems. Toxicity tests with higher animals, besides being time-consuming and expensive, are ethically questionable, which further increases the importance of developing efficient in vitro toxicity tests. In developing in vitro tests for toxicity assessments, aquatic toxicology lags behind mammalian toxicology. Aqueous environmental chemistry is complex, and the sensitivity of the organisms living in a particular aquatic environment may vary considerably. The predictive value of single-species or cell culture tests is therefore generally considered to be low. Nevertheless, single-species tests, utilising bacteria, algae, protozoans and invertebrates, have frequently been used in in vitro toxicity studies of aquatic pollutants (mainly as screening tests). Attempts at large-scale validations are few. Such attempts seem to be hampered by the complexity of the aquatic ecosystem. Although cells from aquatic organisms have been isolated and cultured for many years, the use of isolated or cultured cells in aquatic toxicology has been limited. However, during the last few years, interest in the use of fish cells in toxicity testing has grown rapidly. For aquatic in vitro toxicology to develop further, a more comparative and mechanistic approach needs to be adopted.
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Dissertations / Theses on the topic "Aquatic toxicology"

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Sved, Daniel W. "Monooxygenase induction and lethality as endpoints in aquatic toxicology." W&M ScholarWorks, 1991. https://scholarworks.wm.edu/etd/1539616869.

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Spot, Leiostomus xanthurus, were exposed to suspended sediments (&\approx&20 mg/L) contaminated with polycyclic aromatic hydrocarbons (PAH) in a laboratory flow-through system to evaluate the applicability of hepatic ethoxyresorufin O-deethylase (EROD) induction as an indicator of PAH exposure. PAH sources tested were coal-tar creosote (CTC), a low molecular weight fraction of creosote (LMWF), and a high molecular weight fraction of creosote (HMWF). A standard 96-h acute toxicity test was conducted to ensure that PAH concentrations tested in induction studies were sub-acutely toxic. The 96-h LC50 for spot was 1740 &\mu&g PAH/L (95% confidence interval = 1480-2060 &\mu&g PAH/L). The lowest concentration producing an observable effect in 96 h was 560 &\mu&g PAH/L; no effects were observed for spot exposed to 250 &\mu&g PAH/L for 96 h. Induction of hepatic EROD activity occurred rapidly in fish exposed to high environmentally realistic concentrations of CTC or the HMWF, but not the LMWF. Maximal induction (30-fold) occurred in fish exposed for 48 h to 150 &\mu&g PAH/L. Induction was concentration-dependent up to 150 &\mu&g PAH/L; at 320 &\mu&g PAH/L induction was 14-fold. EROD activity decreased upon further exposure; by day 7, EROD activity was not significantly different than that on day 0. EROD activity in fish exposed to 16 &\mu&g PAH/L was not consistently higher than that in control fish. Spot exposed to at least 70 &\mu&g PAH/L from CTC or the HMWF experienced severe fin erosion, epidermal lesions, and mortality beginning a few days after maximal EROD induction occurred. No relationship between EROD induction and whole animal responses is implied, only that EROD induction did precede any high order effects. These results indicate complications to the use of EROD activity as a sensitive, reliable indicator of PAH exposure. The toxicity of CTC may inhibit or interfere with continued induction of EROD activity, but neither the toxicity nor inducing capability is associated with the LMWF. The lack of exposure-dependent EROD induction indicate there could be difficulties in interpreting field studies, where fish have unknown exposure histories.
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Méndez, Sara I. Semlitsch Raymond D. "Aquatic and terrestrial exposure of amphibians to estrogenic endocrine disrupting contaminants." Diss., Columbia, Mo. : University of Missouri--Columbia, 2009. http://hdl.handle.net/10355/6183.

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Title from PDF of title page (University of Missouri--Columbia, viewed on Feb 15, 2010). The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Dissertation advisor: Raymond D. Semlitsch Vita. Includes bibliographical references.
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Smith, E. C., Phillip R. Scheuerman, and Kurt J. Maier. "Toxicity of Nanomaterials to Aquatic Organisms." Digital Commons @ East Tennessee State University, 2005. https://dc.etsu.edu/etsu-works/2939.

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Webb, Diane. "Assessment of the health of the Swan-Canning river system using biochemical markers of exposure of fish." Thesis, Curtin University, 2005. http://hdl.handle.net/20.500.11937/62.

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Most environmental studies concerning the environmental health of the Swan- Canning River system have focussed on nutrient inputs from both rural and urban catchments that are the cause of algal blooms. On occasions these algal blooms have resulted in fish deaths attributed to oxygen starvation. Relatively few studies have examined whether non-nutrient contamination is affecting the health of the riverine environment. Those studies that have, have concentrated on measuring the levels of heavy metals, organochlorines, organophosphates, and hydrocarbons in the sediments and water of the river system, and in the flesh of the biota. However, chemical analysis often fails to detect chemicals of concern due to high laboratory detection limits. In addition, analysis of the body burden of contaminants within biota does not necessarily convey if exposure is inducing adverse effects at the individual or ecosystem levels. The use of biochemical markers as a tool for the assessment of the health of the Swan-Canning River system was examined under a collaborative research project with the Waters and Rivers Commission, established in response to the recognition of the paucity of information from chemical analyses. The present study focussed on the estuarine portion of the Swan-Canning River system, using the black bream (Acanthopagrus butcheri), an estuarine dependent fish species, as a biomonitoring tool. Prior to the commencement of this study it had been determined that the black bream was a suitable fish species for use as a biomonitoring tool when using mixed function oxygenase (MFO) activity induction under laboratory conditions.Biopsies taken from feral black bream collected from eight sites during the period 2000 to 2002 from the estuary confirmed that the use of MFO induction in this fish species as a biomarker of exposure to organic contaminants is a reliable biomarker. Fish gender was a confounding factor in the interpretation of MFO induction when using the enzyme ethoxyresorufin-O-deethylase (EROD) as EROD activity was suppressed in both pre- and post-spawning female black bream. No such suppression was identified when using the MFO enzyme ethoxycoumarin-O-deethylase (ECOD). However, due to differences in the pattern and intensity of the induction of EROD and ECOD activities it was concluded that ECOD activity was not a substitute for EROD activity to detect certain chemical as ECOD activity represents a different cytochrome P450 pattern to EROD activity. No spatial, seasonal or interannual differences in the level of the enzyme sorbitol dehydrogenase (SDH) in the blood of the black bream were measured indicating that the interpretation of MFO activity induction was not compromised by hepatocellular damage. This study has shown that the black bream in the Swan-Canning Estuary are exposed to, and are metabolising polycyclic aromatic hydrocarbons (PAHs), notwithstanding that the chemical analysis of the contaminant load of these substances in the estuarine waters is consistently below laboratory detection limits. In addition, biomarker responses such as ECOD activity indicate that various other organic pollutants are present and are being metabolised by the black bream.The measurement of biliary metabolites clearly show that, under winter conditions, the comprehensive drainage system of the Swan Coastal Plain contributes PAHs from pyrogenic sources such as burnt fuels into the estuary although the onset and intensity of rainfall events notably impacts on the volume of stormwater inflow. During the summer months, when freshwater flow is minimal, petrogenic sources of PAHs are dominant. Metabolic enzyme analysis points to the black bream being challenged in their aerobic capacities during summer, and that gill tissue was the most suitable tissue to evaluate the aerobic and anaerobic capacity of this fish species. Furthermore, there was a significant negative correlation between stress protein (hsp70) expression and DNA integrity in field-collected fish suggesting that the black bream within the estuary are highly stressed. No gradient of response in biomarker levels was identified in the Swan-Canning Estuary under either winter or summer conditions indicating there are multiple sources of inputs of potential pollutants along the length of the estuary. Stormwater and road runoff are the primary source of pollutant input into the estuary in the winter months, while summer biomarker levels, particularly PAH, appear to reflect the high usage of the estuary for recreational purposes and runoff from poorly irrigated parks and gardens. Significant rainfall events at any time of the year have the potential to adversely impact the biota of the estuary, particularly when these events result in a flush of water from the drains following long dry periods.The study shows that the black bream is a suitable fish species to use under field conditions to detect the presence of bioavailable non-nutrient contamination within the Swan-Canning Estuary. A suite of biomarkers in black bream have been tested seasonally and annually but only a small number of biomarkers have proven suitable for routine monitoring of the health of the Swan-Canning Estuary. This treatise concludes with several recommendations for further investigations into biomarkers of fish health for the purpose of increasing our understanding on the sources and type of contamination entering the estuary, and potential effects on the aquatic biota of the Swan-Canning River system. These recommendations include, but are not limited to: (1) the need to determine baseline levels for the different biomarkers investigated in this study, (2) the examination of the Moore River or the Warren River estuaries as potential reference sites for biomarker studies in the Swan- Canning Estuary, (3) the advantage of identifying a second estuarine-dependent indigenous fish as a biomonitoring tool, (4) the requirement for a targeted study aimed at clarifying the relationship between major drain discharges, biomarker levels and impacts on river biota, and (5) a study of estuarine waters utilising SPMDs be undertaken in tandem with biomarker analysis of field captured fish would be beneficial.
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Gausman, Maria M. "A COMPARISON OF DUCKWEED AND STANDARD ALGAL PHYTOTOXICITY TESTS AS INDICATORS OF AQUATIC TOXICOLOGY." Miami University / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=miami1153752259.

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Wong, Wing-yu, and 黃詠如. "Ecotoxicological effects of selected engineered nano-materials to aquatic organisms in relation to their physicochemicalcharacteristics." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47869410.

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Engineered nanomaterials (NMs), defined as artificially made particles possessing at least one dimension within 1 – 100 nm range, have different physical and biological properties from bulk materials of the same chemistry due to their increased surface areas. Their novel properties have facilitated the prolific growth of commercial NM-incorporating products. NMs may be leached into the aquatic environment during the product life-cycle, but their ecological impacts on marine ecosystems are currently largely unknown. Therefore, this study primarily aimed to investigate the physicochemical characteristics (particle and aggregate sizes, dissolution rate) and in vivo toxicities of commonly-used metallic NMs to marine organisms under various environmental scenarios. First, in vivo ecotoxicity screening tests, using the marine diatom Skeletonema costatum and the rotifer Brachionus sp., were conducted for nine common nano metal oxides which had been applied in various commercial products. Among them, nano zinc oxide (nZnO) and nano magnesia (nMgO) were found to be the most toxic NMs to both organisms, as they induced oxidative stress by increasing reactive oxygen species (ROS) production in the organism. The endocrine disrupting potential of nZnO was revealed by its modulation of transcriptions of the genes for retinoid X receptor (RXR) in the rotifer. Conversely, nano alumina (nAl2O3) and nano indium oxide (nIn2O3) were the least toxic NMs. Due to its high potency, toxicity of nZnO was further evaluated using five marine organisms (i.e., microalgae Thalassiosira pseudonana and S. costatum, copepod Tigriopus japonicus, amphipod Elasmopus rapax, and medaka Oryzias melastigma). Crustaceans were generally more sensitive to nZnO. Toxicity of nZnO was mainly attributed to metal ion dissolution, although nanoparticulate effects such as aggregation and adhesion of nZnO onto the animal’s exoskeleton as well as physical disruption of cell structures could not be discounted. Due to the fact that the dissolution of nZnO decreased from 16 mg Zn L-1 at 4°C to 1.4 mg Zn L-1 at 35°C, and Zn ion was the main contributing factor for nZnO toxicity at 25°C, it was postulated that nZnO toxicity would increase with decreasing temperature. This hypothesis was tested with S. costatum, O. melastigma and the amphipod Melita longidactyla through a factorial design experiment (i.e., 2 concentrations x 4 temperatures). In agreement with the hypothesis, the growth of S. costatum was significantly inhibited by nZnO at the lowest test temperature (15°C). However, contradictive results were observed in the two animal species. For instance, the amphipod could reduce the nZnO uptake and its toxicity by undergoing metabolic depression and dormancy at lower temperatures. As the morphology and coating of NMs utilized in commercial products may differ from those employed in toxicity studies, T. japonicus was exposed to nZnO-containing sunscreens to assess the effects of nZnO and Zn2+ released in seawater during epidermal applications. Based on their genetic biomarker responses, the results suggested that other components in sunscreens could react synergistically or antagonistically on nZnO toxicity. Clearly, there is a need for further study of the combined effects of NMs and other common chemical contaminants to marine organisms.
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Cho, Eun-ah. "Bioturbation as a novel method to characterize the toxicity of aquatic sediment." NCSU, 2005. http://www.lib.ncsu.edu/theses/available/etd-02282005-111535/.

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Bioturbation, the biological process through which many species of infaunal benthic invertebrates suspend bottom sediments into the water column through their burrowing, feeding, respiratory, and locomotor activities, may be a sub-lethal endpoint that can be exploited to assess the toxicity of aquatic sediments. Therefore, we developed a novel test method that used bioturbation (BioTurbTox test) generated by the activities of second in-star Chironomus tentans larvae as the toxicity endpoint (Chapter 2). To validate this method, copper (Cu) and fluoranthene were individually spiked into relatively uncontaminated aquatic sediment to assess changes in bioturbation and mobilization of the chemicals into the overlying water. Turbidity production responded to the chemicals in the sediment in a concentration-dependent manner and was an excellent indicator of sediment toxicity. Moreover, substantial concentrations of Cu were released into the overlying water from the Cu-spiked sediment, whereas little fluoranthene was mobilized into the overlying water from the fluoranthene-spiked sediment. Sediment samples were then collected from the field and used to evaluate the similarity of response of the BioTurbTox test to other more standardized toxicity tests. In the summer of 2003, sediment samples were collected at six sites in the Neuse River of North Carolina tested for toxicity, and analyzed for chemical contaminants (Chapter 3). Atrazine was the most frequently detected current-use pesticide and pyrene and fluoranthene were measured at relatively high concentrations from the Neuse River sites. Concentrations of fluoranthene were correlated with results from the Ceriodaphnia dubia porewater and BioTurbTox tests. We concluded that the new BioTurbTox test was useful as a rapid screening method for sediment toxicity information, but required normalization to the clay content or to the total organic carbon content of field collected sediments. In Chapter 4, the toxicity of environmental pharmaceuticals and personal care products (PPCPs) were evaluated with the BioTurbTox and C. dubia reproductive tests. Fluoxetine and bisphenol A significantly affected bioturbation caused by C. tentans, especially at high concentrations (1-2 mg/L), and the turbidity change induced by caffeine, fluoxetine, and bisphenol A showed a concentration-response relation. Triclosan affected reproduction of C. dubia at relatively low concentrations (IC50: 85.4 ?Ýg/L). However, most of the tested PPCPs were not acutely toxic at environmentally relevant concentrations, but were relatively toxic at high concentrations. In Chapter 5, two sediment-spiking methods (extract mixing vs. whole sediment dilution methods) were compared with the BioTurbTox test and a gradient response was observed from both methods. Based on the similarity of the toxic response, we determined that either of the spiking methods was appropriate for estimating the toxicity of aquatic sediments in screening level assessments. The overall conclusion from this research was that the newly developed BioTurbTox test shows promise as a tool to assess the toxicity and mobilization of contaminants from aquatic sediments.
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Atkinson, Susanna Kate. "The persistence of steroidal estrogens in the aquatic environment." Thesis, University of Ottawa (Canada), 2009. http://hdl.handle.net/10393/28120.

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The presence and fate of the steroidal estrogens, estrone (E1), 17beta-estradiol (E2) and 17alpha-ethinylestradiol (EE2), in Ottawa and Cornwall wastewater treatment plants (WWTP's), drinking water, and the river water used as the drinking water source, were identified. Estrogens were extracted using accelerated solvent extraction, gel permeation chromatography and solid phase extraction, and identified using gas chromatography-mass spectrometry and internal C-13 standards. E1, E2 and EE2 concentrations ranged from 1.8 to 370, 24.3 to 66.9 and 0.4 to 9.8 ng L-1, respectively, and were affected by weather variables such as temperature and precipitation, and WWTP parameters such as daily flow and carbonaceous biochemical oxygen demand. Photodegradation rate constants under ultraviolet B radiation for E1 were directly proportional to radiation intensity and sample thickness, and inversely proportional to dissolved organic carbon concentration, but EE2 was remarkably persistent. A luciferase reporter gene assay found estrogenicity in both sewage effluent and UVB-exposed samples of estrogens, contributed by the degradation products of steroidal estrogens. Finally, EE2 persistence was also seen in a time-course experiment in which goldfish were exposed to 25 ng L-1 EE2. A mass-balance model calculated a bioconcentration factor (BCF) for EE2 in fish blood of 1400, whereas measured data revealed a maximum BCF of only 500.
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Cronin, Mark T. D. "Quantitative structure-activity relationships of comparative toxicity to aquatic organisms." Thesis, Liverpool John Moores University, 1990. http://researchonline.ljmu.ac.uk/4989/.

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Quantitative Structure-Activity relationship (QSAR) attempt statistically to relate the physico-chemical properties of a molecule to its biological activity. A QSAR analysis was performed on the toxicities of up to 75 organic chemicals to two aquatic species, Photobacterium phospherum (known as the Microtox test), and the fathead minnow. To model the toxicities 49 physico-chemical and structural parameters were produced including measures of hydrophobicity, molecular size and electronic effects from techniques such as computational chemistry and the use of molecular connectivity indices. These were reduced to a statistically more manageable number by cluster analysis, principal component analysis, factor analysis, and canonical correlation analysis. The de-correlated data were then used to form relationships with the toxicities. All the techniques were validated using a testing set. Some good predictions of toxicity came from regression analysis of the original de-correlated variables. Although successful in simplifying the complex data matrix, principal component analysis, factor analysis, and canonical content analysis were disappointing as predictors of toxicity. The performance of each of the statistical techniques is discussed. The inter-species relationships of toxicity between four Commonly utilised aquatic endpoints, fathead minnow 96 hour IC50, Microtox 5 minute EC50, Daphnia magna 48 hour IC50, and Tetrahymena pyriformis 60 hour IG50, were investigated. Good relationships was found between the fathead minnow and both T. pyriformis and D. magna toxicities indicating that these species could be used to model fish toxicity. The outliers from individual relationships were assessed in order to elucidate if any molecular features may be causing greater relative toxicity in one species as compared to another. It is concluded that in addition to the intrinsic differences between species, the greater length of the test time for any species may result in increases bioaccumulation, metabolism, and detoxification of certain chemical classes. The relationships involving fish toxicity were moderately improved by the addition of a hydrophobic parameter.
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Duggan, Sam B. "Complex metal mixture reduces apparent protein carbonylation in a tolerant aquatic macroinvertebrate, Arctopsyche grandis." Thesis, Colorado State University, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=1606539.

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Mining is widespread and an economically important industry. Unfortunately, acid mine drainage (AMD) can pollute ecosystems with a cocktail of contaminants too complex for accurately forecasting its health consequences. However, through quantification of fundamental toxic events, the effects of complex mixtures can be observed. This project explored two potentially insightful and convenient endpoints. First, oxygen consumption (MO2), a well-established and sensitive indicator of respiratory impairment was utilized. Second, protein carbonyl content (PCC), an experimental ecological biomarker widely lauded in biomedical circles as a highly conserved indicator of health status was assessed for its utility in a metal tolerant aquatic macroinvertebrate, Arctopsyche grandis. A. grandis were exposed to eight environmentally relevant target concentrations (in duplicate) of AMD for eight days at a temperature controlled greenhouse containing artificial flow-through streams. As expected, MO2 was inversely related to treatment concentration (R 2=0.35, p=0.015). Protein carbonyl content, however, diverged from predictions. Protein carbonyl content analysis detected significantly more oxidative protein injury in control treatments than in metal-rich AMD treatments (p<0.001). Moreover, there was not a significant difference in PCC between different AMD concentrations. Protein carbonyl content’s departure from anticipated results likely is the consequence of dynamic interactions between direct and indirect effects at the chemical, biochemical, physiologic and behavioral levels. The results of this project illustrate flaws of utilizing a single biochemical marker to observe effects of a toxic mixture. Rather, a broad suite of biomarkers should be assayed to determine sublethal toxicity. These results also illustrates how multiple stressors can yield unanticipated outcomes.

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Books on the topic "Aquatic toxicology"

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P, Svensson Elias, ed. Aquatic toxicology research focus. New York: Nova Science Publishers, 2008.

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C, Mothersill, and Austin B. 1951-, eds. In vitro methods in aquatic toxicology. Berlin: Springer, 2003.

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Kent, Ostrander Gary, ed. Techniques in Aquatic Toxicology, Volume 2. Boca Raton, Fla: Lewis Publishers, 1996.

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O, Nriagu Jerome, Lakshminarayana J. S. S, and Workshop on Aquatic Toxicology (13th : 1987? : Moncton, N.B.), eds. Aquatic toxicology and water quality management. New York: Wiley, 1989.

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Landis, WG, and WH van der Schalie, eds. Aquatic Toxicology and Risk Assessment: Thirteenth Volume. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1990. http://dx.doi.org/10.1520/stp1096-eb.

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Mayes, MA, and MG Barron, eds. Aquatic Toxicology and Risk Assessment: Fourteenth Volume. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1991. http://dx.doi.org/10.1520/stp1124-eb.

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Suter, GW, and MA Lewis, eds. Aquatic Toxicology and Environmental Fate: Eleventh Volume. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1988. http://dx.doi.org/10.1520/stp1007-eb.

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Cowgill, UM, and LR Williams, eds. Aquatic Toxicology and Hazard Assessment: 12th Volume. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1989. http://dx.doi.org/10.1520/stp1027-eb.

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Cardwell, RD, R. Purdy, and R. Comotto Bahner, eds. Aquatic Toxicology and Hazard Assessment: Seventh Symposium. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1985. http://dx.doi.org/10.1520/stp854-eb.

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Bahner, RC, and DJ Hansen, eds. Aquatic Toxicology and Hazard Assessment: Eighth Symposium. 100 Barr Harbor Drive, PO Box C700, West Conshohocken, PA 19428-2959: ASTM International, 1985. http://dx.doi.org/10.1520/stp891-eb.

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Book chapters on the topic "Aquatic toxicology"

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Turcotte, Dominique, and André J. Talbot. "Evolutionary Toxicology." In Encyclopedia of Aquatic Ecotoxicology, 511–20. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5704-2_48.

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Zhang, Xiaowei, John P. Giesy, and Markus Hecker. "Cell Lines in Aquatic Toxicology." In Encyclopedia of Aquatic Ecotoxicology, 259–68. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5704-2_25.

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Farré, Marinella, and Damià Barceló. "Biosensors for Aquatic Toxicology Evaluation." In The Handbook of Environmental Chemistry, 115–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-36253-1_5.

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Giesy, John P., Jonathan E. Naile, Jong Seong Khim, Paul D. Jones, and John L. Newsted. "Aquatic Toxicology of Perfluorinated Chemicals." In Reviews of Environmental Contamination and Toxicology, 1–52. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-1157-5_1.

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Stauber, Jenny, and Merrin Adams. "Flow Cytometry Applications in Aquatic Toxicology." In Encyclopedia of Aquatic Ecotoxicology, 521–32. Dordrecht: Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-5704-2_49.

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Rodriguez, Pilar, and Trefor B. Reynoldson. "Toxicology and Laboratory Studies." In The Pollution Biology of Aquatic Oligochaetes, 87–158. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1718-3_4.

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Clary, John J. "General Animal and Aquatic Toxicity." In The Toxicology of Methanol, 73–106. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118353110.ch4.

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e Silva, Francisca A., João A. P. Coutinho, and Sónia P. M. Ventura. "Aquatic Toxicology of Ionic Liquids (ILs)." In Encyclopedia of Ionic Liquids, 1–18. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-10-6739-6_52-1.

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e Silva, Francisca A., João A. P. Coutinho, and Sónia P. M. Ventura. "Aquatic Toxicology of Ionic Liquids (ILs)." In Encyclopedia of Ionic Liquids, 117–33. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-33-4221-7_52.

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Anderson, Julie C., Sarah C. Marteinson, and Ryan S. Prosser. "Correction to: Prioritization of Pesticides for Assessment of Risk to Aquatic Ecosystems in Canada and Identification of Knowledge Gaps." In Reviews of Environmental Contamination and Toxicology Volume 259, C1. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-88342-3_82.

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Abstract:
Correction to: Chapter “Prioritization of Pesticides for Assessment of Risk to Aquatic Ecosystems in Canada and Identification of Knowledge Gaps” in: P. de Voogt (ed.), Reviews of Environmental Contamination and Toxicology Volume 259, Reviews of Environmental Contamination and Toxicology 259, https://doi.org/10.1007/398_2021_81
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Conference papers on the topic "Aquatic toxicology"

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Blaise, C., and J. F. Férard. "Microbiotests in aquatic toxicology: the way forward." In ENVIRONMENTAL TOXICOLOGY 2006. Southampton, UK: WIT Press, 2006. http://dx.doi.org/10.2495/etox060341.

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Papadimitriou, C., V. Evagelopoulos, P. Samaras, A. G. Triantafyllou, S. Zoras, and T. A. Albanis. "Toxicity of atmospheric particulate matter using aquatic bioassays." In ENVIRONMENTAL TOXICOLOGY 2006. Southampton, UK: WIT Press, 2006. http://dx.doi.org/10.2495/etox060041.

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Yusof, Azura Mat, Saadi Ahmad Kamaruddin, Nor Anis Nadhirah Md Nasir, and Irnis Azura Zakarya. "Assessment of aquatic toxicology dataset using MLR." In The 5th Innovation and Analytics Conference & Exhibition (IACE 2021). AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0094883.

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Reports on the topic "Aquatic toxicology"

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Lotufo, Guilherme R., Gunther Rosen, William Wild, and Geoffrey Carton. Summary Review of the Aquatic Toxicology of Munitions Constituents. Fort Belvoir, VA: Defense Technical Information Center, June 2013. http://dx.doi.org/10.21236/ada583083.

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