Dissertations / Theses on the topic 'Anxiety disorder'
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Shaw, Allan. "Functional bowel disorders in anxiety disorder out patients." Thesis, London South Bank University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288174.
Full textCowart, Maria Jane Whitmore. "Generalized Anxiety Disorder and Social Anxiety Disorder in Youth: Are They Distinguishable?" Diss., Virginia Tech, 2009. http://hdl.handle.net/10919/37645.
Full textPh. D.
Hammel, Jacinda Celeste McGlynn F. Dudley. "Meta worry and generalized anxiety disorder." Auburn, Ala., 2006. http://repo.lib.auburn.edu/2006%20Summer/Dissertations/HAMMEL_JACINDA_58.pdf.
Full textO'Garro-Moore, Jared K. "AN ETIOLOGICAL UNDERSTANDING OF BIPOLAR DISORDER-ANXIETY DISORDER COMORBIDITY: THE ROLE OF ANXIETY SENSITIVITY AND TRAIT ANXIETY." Diss., Temple University Libraries, 2018. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/512695.
Full textPh.D.
Little to no research has evaluated factors that explain the manifestation and maintenance of bipolar disorder-anxiety disorder (BD-AD) comorbidity. The literature has shown that disruption of social and circadian rhythms is associated with mood episode onset. This association is especially pronounced among individuals who have a sensitive behavioral approach system (BAS). Inasmuch as anxiety sensitivity and trait anxiety have been associated both with BD and social rhythm disruption, it is worth examining whether anxiety sensitivity and trait anxiety confer risk for mood episode onset. The aims of this project were to: 1) evaluate trait anxiety and anxiety sensitivity as predictors of social rhythm disruption and BD-AD comorbidity, 2) examine social rhythm disruption (SRD) as a mediator of the association between trait anxiety and anxiety sensitivity and BD-AD comorbidity status, and 3) explore behavioral approach system sensitivity in these processes as contributing to the vulnerability to BD-AD comorbidity. A sample of 156 young adults participated in a multi-wave study in which they completed diagnostic interviews, symptom measures, and life event interviews which assessed the occurrence of positive and negative life events and the degree of SRD following these events every six months. Partial support for the hypotheses was found. Initial anxiety sensitivity, but not trait anxiety, positively predicted SRD for rewarding life events and follow-up bipolar symptoms. Additionally, SRD following positive life events predicted increases in depressive symptoms, but not hypomanic symptoms. SRD mediated the relationship between anxiety sensitivity and depressive symptoms. Furthermore, this relationship was stronger for healthy controls than for those with a bipolar disorder (BD) diagnosis or at-risk for developing BD. Moreover, individuals with a comorbid BD-AD diagnosis tended to have greater social rhythm disruption following negative life events than BD only or healthy individuals. Unexpectedly, individuals with comorbid BD-AD did not exhibit greater anxiety sensitivity or trait anxiety. Overall, the results suggest that anxiety sensitivity is a factor that may help to identify individuals who are vulnerable to bipolar symptoms. Furthermore, SRD is a mechanism that may partially explain this relationship.
Temple University--Theses
MacCallam, Jackie. "Cognitive appraisals in obsessive-compulsive disorder & other anxiety disorders." Thesis, University of Plymouth, 1997. http://hdl.handle.net/10026.1/1138.
Full textRück, Christian. "Capsulotomy in anxiety disorders /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-769-3/.
Full textPlasencia, Melissa Leili. "Safety behaviours and social anxiety disorder." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/24201.
Full textHattingh, Coenraad Jacobus. "Neurobiological aspects of social anxiety disorder." Master's thesis, University of Cape Town, 2011. http://hdl.handle.net/11427/10865.
Full textFlynn, Jessica Jane. "Daily Fear in Social Anxiety Disorder." Kent State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=kent1470046109.
Full textEgic, Milica. "Social anxiety disorder : SSRI vs. placebo." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-20230.
Full textEnright, Simon James. "Obsessive-compulsive disorder: anxiety disorder or schizotype? : a questionnaire and experimental investigation." Thesis, University of Reading, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357850.
Full textPercy, Ray. "Maternal verbal communication and the treatment of children with anxiety disorders in the context of maternal anxiety disorder." Thesis, University of Southampton, 2014. https://eprints.soton.ac.uk/370404/.
Full textHommersen, Paul. "Separation Anxiety Disorder and Oppositional Defiant Disorder : perceived comorbidity between disorders resulting from ambiguous items and halo effects." Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/31331.
Full textArts, Faculty of
Psychology, Department of
Graduate
Gräsbeck, Anne. "The epidemiology of anxiety and depressive syndromes a prospective, longitudinal study of a geographically defined, total population : the Lundby study /." Lund : Dept. of Psychiatry, Lund University Hospital, 1996. http://books.google.com/books?id=sw9sAAAAMAAJ.
Full textKujanpää, T. (Tero). "Generalized anxiety disorder and health care utilization." Doctoral thesis, Oulun yliopisto, 2016. http://urn.fi/urn:isbn:9789526212715.
Full textTiivistelmä Yleistynyt ahdistuneisuushäiriö on mielenterveyden häiriö, jolle on ominaista ylenmääräinen ahdistuneisuus ja huolestuneisuus, joita on vaikea kontrolloida. Väestöstä noin 2 % on kärsinyt siitä vuoden aikana ja 5 % elinaikanaan. Perusterveydenhuoltoon hakeutuvilla potilailla sen on todettu olevan muuta väestöä yleisempi n. 5-8 %:n täyttäessä diagnostiset kriteerit. Aiemmat tutkimukset ovat viitanneet yleistyneestä ahdistuneisuushäiriöstä kärsivien käyttävän usein runsaasti terveyspalveluita. Tämän tutkimuksen tarkoituksena oli selvittää yleistyneen ahdistuneisuushäiriön esiintyvyyttä terveyspalveluita paljon käyttävillä suomalaisilla henkilöillä, yleistyneen ahdistuneisuushäiriön yhteyttä erilaisten terveyspalveluiden käyttöön väestötasolla, yleistyneen ahdistuneisuushäiriön ja somaattisten oireiden yhteyttä terveyspalveluiden suurkäyttöön ja yleistyneeseen ahdistuneisuushäiriöön erikoissairaanhoidossa liittyviä kustannuksia vertaillen niitä masennukseen liittyviin kustannuksiin. Samalla validoitiin yleistyneen ahdistuneisuushäiriön seulontaan kehitetyn GAD-7 seulan suomenkielinen käännös. Joukossa pohjoissuomalaisia (n=150) terveyspalveluita paljon käyttäviä henkilöitä 4 %:lla todettiin yleistynyt ahdistuneisuushäiriö. Heillä GAD-7-kysely osoittautui toimivaksi seulontatyökaluksi. Pohjois-Suomen 1966 syntymäkohortissa väestötasolla tutkittaessa todettiin yleistyneeseen ahdistuneisuushäiriöön liittyvän runsasta terveyspalveluiden käyttöä. GAD-7-kyselyssä positiivisen testituloksen saaneilla oli kokonaisuudessaan 112 % enemmän terveyspalveluiden käyttöä. Tulokset olivat tilastollisesti merkitseviä myös huomioitaessa mahdolliset sekoittavat tekijät. Lisäksi sekä yleistynyt ahdistuneisuushäiriö että somaattiset oireet liittyivät terveyspalveluiden suurkäyttöön. Sairaaloiden poistoilmoitusrekisteriä ja kansallista sairaaloiden vertailutietokantaa hyödyntäen laskettiin uusien yleistyneen ahdistuneisuushäiriön ja masennuksen diagnoosin saaneiden potilaiden erikoissairaanhoidon kokonaiskustannukset 2 vuotta ennen ja jälkeen diagnoosin. Erityisen suuret kustannukset olivat niillä uuden diagnoosin saaneilla, joilla oli historiassa aiempi masennus tai ahdistuneisuushäiriö. Suurimmat keskimääräiset erikoissairaanhoidon kustannukset (19 538 €) todettiin niillä yliestyneen ahdistuneisuushäiriön diagnoosin saaneilla, joilla aiemmin oli todettu jokin muu ahdistuneisuushäiriö tai masennus
Wittchen, Hans-Ulrich. "The many faces of social anxiety disorder." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-88859.
Full textNyroos, Mikaela. "Childhood Test Anxiety - Booster, detainer, disability, disorder." Thesis, Umeå universitet, Barn- och ungdomspsykiatri, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-90530.
Full textWittchen, Hans-Ulrich. "The many faces of social anxiety disorder." Lippincott Williams & Wilkins, 2000. https://tud.qucosa.de/id/qucosa%3A26036.
Full textGrace, Caitlin Claire. "Daily emotional functioning in social anxiety disorder." Thesis, Australian Catholic University, 2020. https://acuresearchbank.acu.edu.au/download/23c87a3ccf87b7ce7728c9888ab2357b6717eec01a257150bb8e67fdb20452a0/16161055/Grace_2020_Daily_emotional_functioning_in_social_anxiety_Redacted.pdf.
Full textThompson, Rachel Diane. "A Taxometric Investigation of Generalized Anxiety Disorder." University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1185819016.
Full textFällmark, Amanda. "Social anxiety disorder : Amygdala activation and connectivity." Thesis, Högskolan i Skövde, Institutionen för biovetenskap, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-20176.
Full textCrawley, Sarah. "Somatic Complaints in Anxious Youth." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/155885.
Full textPh.D.
Objective: This study examined (a) the distribution of physical symptoms in youth with specific primary anxiety disorders (i.e. separation anxiety disorder [SAD], generalized anxiety disorder [GAD], and social phobia [SP]) and (b) their response to treatment with cognitive-behavioral therapy (CBT; 14 sessions of CBT over the course of 12 weeks), medication, combination therapy (CBT + medication), or pill placebo in a sample. Method: Anxiety disordered youth (N = 488, age 7-17) who met criteria for a primary diagnosis of GAD, SAD, and/or SP as part of the Child/Adolescent Anxiety Multimodal Study (CAMS; Walkup et al. 2008) were included in this study. The sample was diverse and included children with comorbid secondary diagnoses. Results: The most common somatic complaints were headache, stomach pain or aches, feeling drowsy or too sleepy, head cold or sniffles, and sleeplessness. The distribution of these complaints did not differ across diagnostic groups. The number and severity of physical symptoms decreased over the course of treatment. Treatment condition, including placebo, was unrelated to the number and severity of physical symptoms posttreatment. Conclusions: Treatment of anxiety leads to a decrease in the number and severity of physical symptoms experienced in anxiety-disordered youth, irrespective of treatment type.
Temple University--Theses
Yngve, Adam. "Resilience against social anxiety : The role of social networks in social anxiety disorder." Thesis, Linköpings universitet, Institutionen för beteendevetenskap och lärande, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-131140.
Full textSargeant, Elizabeth. "Attentional bias modification training for generalised anxiety disorder." Thesis, University of Southampton, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.617827.
Full textAlvares, Gail Alviza. "Behavioral and autonomic inflexibility in social anxiety disorder." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/12639.
Full textKerns, Connor Morrow. "Typical and Atypical Anxiety in Autism Spectrum Disorder." Diss., Temple University Libraries, 2013. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/214770.
Full textPh.D.
Objective. There is confusion regarding the presentation and correct classification of anxiety symptoms in autism spectrum disorders (ASD) as comorbid anxiety disorders, core ASD symptoms or a separate syndrome. The present study examined the degree to which Diagnostic Statistical Manual-Fourth Edition (DSM-IV) anxiety disorders ("typical anxiety") can be reliably distinguished from atypical presentations in ASD ("atypical anxiety"). To inform their classification, the study also assessed how these typical and atypical presentations were differentially related to child characteristics (i.e. IQ, language ability) and features of anxiety disorders (i.e., anxious self-talk) and ASDs (i.e., ASD symptomology, sensory abnormalities). Method. Youth (N = 59; 7 - 17 years; IQ > 60) diagnosed with ASD and their parents completed a semi-structured diagnostic interview (ADIS-C/P) adapted to measure both typical and atypical presentations of anxiety as well as self- and parent-report measures. Results. Seventeen percent of the sample met criteria for a typical anxiety disorder, 15% for an atypical anxiety disorder and 31% for both typical and atypical disorder variants. Whereas IQ, language ability, anxious self-talk and sensory sensitivity significantly predicted typical anxiety, atypical anxiety was significantly associated with anxious self-talk and ASD symptomology. Conclusions. Results suggest that ASD youth display two, phenomenological distinct classes of anxiety. These typical and atypical presentations likely reflect comorbid anxiety and a novel variant of anxiety, which may be missed by current, unmodified anxiety measures. How these presentations differentially respond to interventions and contribute to the range of results regarding the prevalence and presentation of anxiety in ASD warrants investigation.
Temple University--Theses
DeVore, Bethany Rebekah. "Two Short Stories About Anxiety Disorder and Their Psychological Analyses." Miami University Honors Theses / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=muhonors1114104198.
Full textHenje, Blom Eva. "Anxiety and depression in adolescent females autonomic regulation and differentiation /." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-807-5/.
Full textHearn, Cate. "What’s the Worry with Social Anxiety? An Investigation of Worry and its Associated Cognitive Variables In Social Anxiety Disorder in Children and Adolescents." Thesis, Griffith University, 2017. http://hdl.handle.net/10072/365746.
Full textThesis (PhD Doctorate)
Doctor of Philosophy (PhD)
School of Applied Psychology
Griffith Health
Full Text
McGregor, Nathaniel Wade. "The identification of novel susceptibility genes involved in anxiety disorders." Thesis, Stellenbosch : Stellenbosch University, 2014. http://hdl.handle.net/10019.1/95859.
Full textENGLISH ABSTRACT: The etiology of anxiety disorders remains incompletely understood. Clear evidence for a genetic component has been proposed; however, there is also an increasing focus on environmental factors and the interaction between these and the genetic components that may mediate (anxiety) disorder pathogenesis. No single gene or genetic component has been explicitly identified as being involved in the development of anxiety disorders. This is most likely due to a number of reasons, which include, for example, the heterogeneity of anxiety disorders, the contribution of environmental factors and methodological limitations (e.g. small sample size) of research studies. Until now, genetic association studies usually focused on one particular psychiatric disorder at a time. However, with the difficulty in identifying susceptibility genes and/or loci in heterogeneous disorders like obsessive-compulsive disorder and other conditions in the anxiety spectrum, it is perhaps timely to consider multivariate genetics and epidemiological studies in a number of disorders sharing a core characteristic – such as anxiety. In addition to genetic underpinnings, a number of environmental variables have also been identified as risk factors for pathological anxiety, including adverse life events like childhood physical and sexual abuse. The hypothesis for this project is that a pre-existing genetic vulnerability (or genetic risk) interacts with the impact of adverse life events to result in the development of one or more anxiety disorder(s). Considering phenotypic overlap amongst the anxiety disorders, it is likely that diverse networks of genes and/ or interacting pathways are responsible for the phenotypic manifestations observed. Sprague Dawley rats exhibiting behaviours indicative of anxiety in the context of environmental stressors (maternal separation and restraint stress) were used as model for the identification of novel susceptibility genes for anxiety disorders in humans. The striatum has previously been implicated as a candidate in the brain architecture of anxiety pathogenicity, and is also a site exhibiting a high degree of synaptic plasticity. The synaptic plasticity pathway was investigated using the dorsal striatum of the rat brain and several genes were identified to be aberrantly expressed in “anxious” rats relative to controls (Mmp9, Bdnf, Ntf4, Egr2, Egr4, Grm2 and Arc). In humans, it was found that the severity of early adversity was significantly and positively associated with the presence of an anxiety disorder in adulthood. When the human homologues of the susceptibility candidate genes that were identified using the animal model were screened in a human cohort of patients with obsessive-compulsive disorder (OCD), panic disorder (PD) or social anxiety disorder (SAD) (relative to controls), five single nucleotide polymorphisms (SNPs) were found to be significantly associated with these conditions. Four of these SNPs were also found to significantly interact with the severity of childhood trauma. Haplotype analysis of variants within the identified susceptibility candidates revealed novel haplotype associations, four of which are located in the MMP9 gene. Notably, this the first study to link these particular mutations in the MMP9 gene with anxiety disorders and this finding is consistent with previous work suggesting that MMP9 is involved in conditions like cardiovascular disease and cancer which have been associated with increased prevalence of anxiety disorders. In conclusion, this project yielded important findings pertaining to the etiology of anxiety disorders. The use of a combined anxiety disorders cohort (OCD, PD and SAD) may suggest that the associations found here may hold true for anxiety disorders in general and not only for a particular clinically delineated condition. Childhood trauma was confirmed as an increased susceptibility risk for anxiety disorders. Also, this research contributed several novel susceptibility genes (MMP9, EGR2, EGR4, NTF4, and ARC), five significant SNP associations, four significant SNP-environment interactions and five haplotype associations (within MMP9 and BDNF) as candidates for anxiety pathogenicity. The identified polymorphisms and haplotypes were demonstrated to be associated with susceptibility to anxiety disorders in a gene-environment correlation and gene-environment interaction.
AFRIKAANSE OPSOMMING: Die oorsake van angssteurings word steeds nie volledig verstaan nie. Daar is duidelike bewyse vir 'n genetiese komponent, maar daar is ook toenemende fokus op omgewingsfaktore en die interaksie tussen hierdie omgewingsfaktore en genetiese komponente by angssteurings. Geen enkele geen of genetiese komponent is al geïdentifiseer as diè wat betrokke is by die ontwikkeling van angssteurings nie. Dit is waarskynlik weens 'n aantal redes, wat byvoorbeeld, die heterogeneïteit van angssteurings, die bydrae van omgewingsfaktore en metodologiese beperkings (bv. klein steekproef) van die navorsingstudies, insluit. Verder het genetiese assosiasiestudies tot nou toe gewoonlik net op een spesifieke psigiatriese versteuring op 'n slag gefokus. Maar, gegewe die uitdaging om vatbaarheidsgene en / of loci in heterogene steurings soos obsessief – kompulsiewe steuring (OKV) en ander toestande op die angsspektrum te identifiseer, is dit tyd om genetiese en kliniese studies in ‘n aantal steurings - met ‘n oorvleuende kern-element soos angs -, gesamentlik te oorweeg. Bykomend tot die genetiese boustene, is ‘n aantal omgewingsveranderlikes soos traumatiese lewenservarings tydens die kinderjare as risikofaktore vir patologiese angs geidentifiseer. Die hipotese vir hierdie projek is dat daar 'n interaksie tussen genetiese kwesbaarheid (of genetiese risiko) en traumatiese lewensevarings is en dat dit tot die ontwikkeling van 'n / veelvoudige angssteuring(s) kan lei. Inaggenome die fenotipiese oorvleueling tussen die angssteurings, is dit waarskynlik dat diverse netwerke van gene en / of interaktiewe geen-paaie vir die manifestasie van hierdie toestande verantwoordelik is. Sprague Dawley-rotte met gedragswyses aanduidend van angs, in die konteks van omgewingstressore (d.i. skeiding van die ma-rot en bedwang-stres [restraint stress]), is as model gebruik vir die identifisering van nuwe vatbaarheidsgene vir angssteurings in mense. Die striatum is voorheen as ‘n kandidaat in die brein-argitektuur van patologiese angs voorgehou, en is ook ‘n plek met ‘n hoë mate van sinaptiese plastisiteit. Die sinaptiese plastisiteit is ondersoek deur te fokus op die dorsale striatum van die rotbrein en daar is verskeie gene gevind wat anders is in “angstige” rotte in vergelyking met kontroles (Mmp9, Bdnf, Ntf4, Egr2, Egr4, Grm2 en Arc). In mense is daar gevind dat die ernstigheidsgraad van vroeë trauma beduidend en positief met die teenwoordigheid van ‘n angssteuring tydens volwassenheid verband hou. Toe die menslike ekwivalente van die vatbaarheidsgene wat met die dieremodel geïdentifiseer is in ‘n mens-kohort met obsessief-kompulsiewe steuring (OKS), panieksteuring (PS) en sosiale angssteuring (SAS) ondersoek is, is gevind dat daar 5 enkele nukleotied polimorfismes (ENPs) is wat met die toestande verband hou. Daar is ook gevind dat vier van hierdie ENPs beduidend verband hou met die ernstigheidsgraad van trauma tydens die kinderjare. Haplotipe analise van variante binne die geïdentifiseerde vatbaarheidsgene het op nuwe haplotipe assosiasies – waarvan 4 op die MMP9-geen geleë is – gedui. Hierdie is dus die eerste studie wat gevind het dat dié spesifieke mutasies van die MMP9-geen met angssteurings verband hou. Hierdie bevinding strook met vorige werk wat daarop dui dat die MMP9-geen by toestande soos kardiovaskulêre siekte en kanker wat ook met verhoogde voorkoms van angssteurings verband hou, betrokke is. Ter afsluiting kan ons sê dat hierdie projek belangrike bevindinge oor die oorsake van angssteurings gemaak het. Die gebruik van ‘n gekombineerde angssteurings-kohort (OKS. PS en SAS) kan moontlik suggereer dat die assosiasies wat ons hier gevind het, waar is vir alle angssteurings en nie net vir ‘n spesifieke afgebakende toestand nie. Traumatiese ervarings tydens die kinderjare is ook bevestig as ‘n risiko vir die ontwikkeling van angssteurings. Hierdie navorsing het ook verskeie nuwe vatbaarheidsgene (MMP9, EGR2, EGR4, NTF4, en ARC), 5 beduidende ENP assosiasies, 4 beduidende ENP-omgewings-interaksies en 5 haplotipe assosiasies (by MMP9 en BDNF) geïdentifiseer as moontlike kandidate wat ‘n rol speel by die ontstaan van patologiese angs. Daar is ook gevind dat die geïdentifiseerde polimorfismes en haplotipes met vatbaarheid vir angssteurings in ‘n geen-omgewing- korrelasie en geen-omgewing- interaksie verband hou. Stellenbosch University http://scholar.sun.ac.za
Airaksinen, Eija. "Cognitive functions in depression and anxiety disorders : findings from a population-based study /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-954-8/.
Full textHalldorsdottir, Thorhildur. "Comorbid Oppositional Defiant Disorder and Anxiety Disorders in Boys and Girls: Relations to Perceptual Bias." Thesis, Virginia Tech, 2011. http://hdl.handle.net/10919/76947.
Full textMaster of Science
Young, Laura. "The lived experience of adults with generalised anxiety disorder." Thesis, City, University of London, 2019. http://openaccess.city.ac.uk/21343/.
Full textMcKenna, Ian. "Cognitive behavioural therapies for social anxiety disorder (SAnD) review." Thesis, University of Exeter, 2013. http://hdl.handle.net/10871/13623.
Full textLupardus, Joshua. "Anxiety and Depression| Temporal Manifestations of the Same Disorder?" Thesis, Southern Illinois University at Edwardsville, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10807938.
Full textAnxiety and depression are some of the most common forms of mental illness. About 66 percent of the population diagnosed with generalized anxiety disorder will also be diagnosed with a form of depression in their lifetimes (Kaufman & Charney, 2000). While historically anxiety and depression have been seen as inherently separate conditions, recent research has indicated that the two disorders may be more closely linked. Watson and Clark’s (1991) tripartite model explains that the comorbidity rates of the disorders and overlapping symptomology could be due to an underlying, shared factor that they called negative affect. Eysenck, Payne, and Santos (2006) found evidence to suggest that anxiety and depression may be temporal manifestations of the same disorder which they called negative affect in reference to the tripartite model. Two other studies have since found evidence for this temporal differentiation (Pomerantz & Rose, 2014; Rinaldi, Locati, Parolin, & Girelli, 2017). Results of this study indicate that there is evidence to suggest a temporal component as a potential associated factor which could help determine whether anxiety or depression manifests in individuals with high levels of negative affect.
Nilzon, Kjell R. "Childhood depressive disorder social withdrawal, anxiety and familial aspects /." Göteborg, Sweden : Dept. of Psychology, Göteborg University, 1996. http://catalog.hathitrust.org/api/volumes/oclc/35143427.html.
Full textO'Leary, Emily. "Cognitive processing characteristics in obsessive-compulsive disorder subtypes." Thesis, University of Canterbury. Psychology, 2005. http://hdl.handle.net/10092/1359.
Full textFaria, Vanda. "Mind really does matter : The Neurobiology of Placebo-induced Anxiety Relief in Social Anxiety Disorder." Doctoral thesis, Uppsala universitet, Institutionen för psykologi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-181548.
Full textKenny, Lucy Margaret. "Memory processes in posttraumatic stress disorder." [New South Wales : University of New South Wales], 2006. http://www.library.unsw.edu.au/~thesis/adt-NUN/uploads/approved/adt-NUN20061110.142022/public/02whole.pdf.
Full textBubier, Jennifer L. "Co-occurrence of Oppositional Defiant Disorder with Generalized and Separation Anxiety Disorders Among Inner-city Children." Diss., Temple University Libraries, 2010. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/48936.
Full textPh.D.
There is a paucity of research that has examined co-occurring oppositional defiant disorder and generalized anxiety disorder (ODD+GAD) symptoms and oppositional defiant disorder and separation anxiety disorder (ODD+SAD) symptoms among children. To address this gap, I investigated multiple explanations for the co-occurrence of ODD+GAD and ODD+SAD. Specifically, I investigated whether (a) GAD symptoms prospectively predicted ODD symptoms and SAD symptoms prospectively predicted ODD symptoms (Explanation 1), (b) ODD symptoms prospectively predicted GAD symptoms and ODD symptoms prospectively predicted SAD symptoms (Explanation 2), and (c) shared risk processes accounted for the co-occurrence of ODD+GAD and ODD+SAD (Explanation 3). Participants were an ethnic minority, inner-city sample of first through fourth grade children (N = 88, 51% male) and their primary caregivers. I used data collected at the baseline and 1-year follow-up assessments of the Child Health and Behavior Study, a longitudinal survey of families residing in North Philadelphia. Findings provided support for Explanation 2 and Explanation 3 in the development of co-occurring ODD+GAD symptoms and support for Explanation 3 in the development of co-occurring ODD+SAD symptoms. This study contributes to the extant literature by providing the first empirical examination of these multiple explanations in an ethnic minority, inner city sample of children.
Temple University--Theses
Whitaker, Juanita Angela. "African American Males Coping With Anxiety Disorder: Treatment-Seeking Behavior." ScholarWorks, 2018. http://scholarworks.waldenu.edu/dissertations/4896.
Full textPalm, Marisha Emily. "The neurochemistry, neuropsychology and functional neuroanatomy of generalised anxiety disorder." Thesis, University of Manchester, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492844.
Full textKerr, Natalie. "Exploring emotional bias, anxiety and attentional deficits in bipolar disorder." Thesis, King's College London (University of London), 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.497542.
Full textMesa, Franklin. "Psychopathology and Functional Impairment in Adolescents with Social Anxiety Disorder." Master's thesis, University of Central Florida, 2013. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/5822.
Full textM.S.
Masters
Psychology
Sciences
Psychology Clinical
Owens, Maryann. "Does Virtual Reality Elicit Physiological Arousal in Social Anxiety Disorder." Master's thesis, University of Central Florida, 2013. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/5832.
Full textM.S.
Masters
Psychology
Sciences
Psychology Clinical
Ferrell, Susanna S. "Pattern and Disorder: Anxiety and the Art of Yayoi Kusama." Scholarship @ Claremont, 2015. http://scholarship.claremont.edu/scripps_theses/554.
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Full textSchwob, Jeremy T. "A Smartphone Application for the Treatment of Generalized Anxiety Disorder." University of Dayton / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1461176493.
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