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Books on the topic 'Ants – Physiology'

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Author: Grafiati

Published: 10 December 2022

Last updated: 29 January 2023

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1

Clark, Christopher. Oxford activity books for children. Oxford: OUP, 1987.

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2

1943-, Eng Kenneth, and Coppa Gene Francis, eds. Anorectal, presacral, and sacral tumors: Anatomy, physiology, pathogenesis, and management. Philadelphia: Saunders, 1987.

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3

Velvet glove, iron fist: A history of anti-smoking. Ripon, England: Little Dice, 2009.

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4

N, Losso Jack, Shahidi Fereidoon 1951-, and Bagchi Debasis, eds. Anti-angiogenic functional and medicinal foods. Boca Raton: CRC Press, 2007.

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5

Antimicrobial drug resistance. [Totowa, N.J.]: Humana Press, 2009.

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6

Kronhausen, Eberhard. Formula for life: The anti-oxidant, free-radical, detoxification program. New York: Morrow, 1989.

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7

D, Catto Graeme R., ed. Drugs and the kidney. Dordrecht: Kluwer Academic Publishers, 1990.

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8

1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Basel: Birkhäuser, 2001.

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9

Antiarrhythmic drugs: A practical guide. 2nd ed. Malden, Mass: Blackwell Pub., 2007.

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10

Antiarrhythmic drugs: A practical guide. Cambridge, Mass., USA: Blackwell Science, 1997.

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11

Zoppo, Gregory J. Del. Innate inflammation and stroke. Edited by New York Academy of Sciences. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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12

G, Gillissen, and International Symposium on "The Influence of Antibiotics on the Host-Parasite Relationship" (3rd : 1987 : Cologne, Germany), eds. The Influence of antibiotics on the host-parasite relationship III. Berlin: New York, 1989.

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13

Fever and antipyresis: The role of the nervous system. Cambridge: Cambridge University Press, 1995.

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14

1937-, Toyama Junji, Hiraoka Masayasu 1940-, Kodama Itsuo, and Nihon Junkanki Gakkai. Gakujutsu Shukai, eds. Recent progress in electropharmacology of the heart: Proceedings of the international satellite symposium of the 59th Annual Scientific Meeting of the Japanese Circulation Society, Nagoya, April 3-4, 1995. Boca Raton: CRC Press, 1996.

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15

M, Hooper N., ed. Alzheimer's disease: Methods and protocols. Totowa, N.J: Humana Press, 2000.

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16

Pathak, Yashwant, and Jayant N. Lokhande. Nutraceuticals for Aging and Anti-Aging. Taylor & Francis Group, 2021.

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17

Clark, Christopher. Oxford Activity Books for Children: Book 6 (Oxford Activity Books for Children). Oxford University Press, USA, 1993.

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18

Clark, Christopher. Oxford Activity Books for Children: Cards Pack B (Books 4-6) (Oxford Activity Books for Children). Oxford University Press, USA, 1988.

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19

Clark, Christopher. Oxford Activity Books for Children: Book 5 (Oxford Activity Books for Children). Oxford University Press, USA, 1985.

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20

Clark, Christopher. Oxford Activity Books for Children: Cards Pack A (Books 1-3) (Oxford Activity Books for Children). Oxford University Press, USA, 1987.

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21

Localio, Arthur, and Kenneth Eng. Anorectal, Presacral and Sacral Tumors: Anatomy, Physiology, Pathogenesis and Management. W.B. Saunders Company, 1986.

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22

Lokhande, Jayant N., and Yashwant Vishnupant Pathak. Nutraceuticals for Aging and Anti-Aging: Basic Understanding and Clinical Evidence. Taylor & Francis Group, 2021.

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23

Anti-Stress (Powerfoods Series). Gaia Books Ltd, 1999.

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24

Daniel, Goleman, Bennett-Goleman Tara, and Groch Judith, eds. The Relaxed body book: A high-energy anti-tension program. Garden City, N.Y: Doubleday, 1986.

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25

Goleman, Daniel, Tara Bennett-Goleman, and American Health Magazine. The Relaxed Body Book: A High-Energy Anti-Tension Program. Doubleday, 1986.

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26

Tsagareli, Merab G., and Nana Tisklauri. Behavioral Study of 'Non-Opioid' Tolerance'. Nova Biomedical, 2011.

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27

Novel inhibitors of leukotrienes. Basel: Birkhäuser Verlag, 1999.

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28

Novel Inhibitors of Leukotrienes (Progress in Inflammation Research). Birkhäuser Basel, 1999.

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29

Giancarlo, Folco, Murphy Robert C, and Samuelsson Bengt, eds. Novel inhibitors of leukotrienes. Basel: Birkhäuser Verlag, 1999.

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30

Murphy, Robert C., Bengt Samuelsson, and Giancarlo Folco. Novel Inhibitors of Leukotrienes. Birkhäuser Boston, 2012.

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31

Giancarlo, Folco, Samuelsson Bengt, and Murphy Robert C, eds. Novel inhibitors of leukotrienes. Basel: Birkhäuser Verlag, 1999.

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32

(Editor), Giancarlo Folco, Bengt Samuelsson (Editor), and Robert C. Murphy (Editor), eds. Novel Inhibitors of Leukotrienes (Pir (Series).). Birkhauser, 1999.

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33

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0057.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep process by which leucocytes interact with endothelium lining postcapillary venules in order to leave the circulation and migrate towards a site of inflammation is central to the pathology of inflammatory disease. The vasculature is the primary site of injury in several rheumatic diseases, including the vasculitides. It may also be damaged by chronic inflammation, leading to endothelial dysfunction and accelerated atherosclerosis. Thrombosis is also a critical pathological process in several chronic inflammatory diseases, particularly the anti-phospholipid antibody syndrome and Behçet's syndrome. The vascular endothelium is central to angiogenesis, the process of new capillary outgrowth, upon which synovial proliferation in inflammatory arthritis is dependent. Angiogenesis is inhibited by current anti-rheumatic therapies and may become a target for novel anti-rheumatic drugs. An increasing area of research concerns the direct effects of drugs used in the treatment of atherosclerosis and inflammatory disease on the endothelium, and whether these agents are beneficial or harmful. Of particular interest to rheumatologists are the vascular effects of statins, disease-modifying anti-rheumatic drugs (DMARDs), immunosuppressants, and cyclooxygenase inhibitors.

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34

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0057_update_001.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep process by which leucocytes interact with endothelium lining postcapillary venules in order to leave the circulation and migrate towards a site of inflammation is central to the pathology of inflammatory disease. The vasculature is the primary site of injury in several rheumatic diseases, including the vasculitides. It may also be damaged by chronic inflammation, leading to endothelial dysfunction and accelerated atherosclerosis. Thrombosis is also a critical pathological process in several chronic inflammatory diseases, particularly the anti-phospholipid antibody syndrome and Behçet’s syndrome. The vascular endothelium is central to angiogenesis, the process of new capillary outgrowth, upon which synovial proliferation in inflammatory arthritis is dependent. Angiogenesis is inhibited by current anti-rheumatic therapies and may become a target for novel anti-rheumatic drugs. An increasing area of research concerns the direct effects of drugs used in the treatment of atherosclerosis and inflammatory disease on the endothelium, and whether these agents are beneficial or harmful. Of particular interest to rheumatologists are the vascular effects of statins, disease-modifying anti-rheumatic drugs (DMARDs), immunosuppressants, and cyclooxygenase inhibitors.

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35

(Editor), K. E. Andersson, and C.G.A. Persson (Editor), eds. Anti-asthma Xanthines and Adenosine (Current clinical practice series). Elsevier, 1986.

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36

Antibiotics in Laboratory Medicine. LWW, 2014.

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37

Wahed, Ismaeel Abrahim. Testicular toxicity of standard and investigational anti-cancer drugs: Cytotoxic effects ofstandard and investigational anti-cancer drugs on the histology and physiology of the mouse testis. Bradford, 1988.

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38

Sutoresu no kihon rikai to kō-sutoresu shokuhin no kaihatsu: Fundamental concepts of stress and the development of anti-stress foods. 2012.

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39

(Editor), Michael Oettel, and Ekkehard Schillinger (Editor), eds. Physiology and Mechanisms of Action of Estrogens and Antiestrogens (Handbook of Experimental Pharmacology). Springer, 1999.

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40

Berntson, Gary G., Peter J. Gianaros, and Manos Tsakiris. Interoception and the autonomic nervous system: Bottom-up meets top-down. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0001.

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Although the efferent role of the autonomic nervous system (ANS) in homeostasis has long been recognized, afferent aspects of the ANS—especially interoception—are increasingly recognized to be equally important. Interoception is fundamental to the regulation of internal physiology, particularly as it is coordinated with contextually determined and adaptive behavioral processes. A cardinal but often underappreciated feature of interoception is its role in myriad cognitive and affective processes that are integrated in health and disease. This chapter introduces the concept of interoception and outlines its historical origins and applications in multiple domains of psychology and psychobiology. It provides an overview of its peripheral and central neural substrates, and it outlines how this construct is best conceptualized within a multi-system and multi-level regulatory framework.

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41

The Bacteria: Their Origin, Structure, Function and Antibiosis. Springer, 2006.

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42

The Bacteria: Their Origin, Structure, Function and Antibiosis. Springer, 2007.

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43

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology. Birkhäuser, 2013.

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44

1953-, Kurowski M., ed. Adverse reactions to non-steroidal anti-inflammatory drugs: Clinical pharmacoepidemiology. Basel: Birkhäuser Verlag, 1992.

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45

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology. Birkhauser Verlag, 2013.

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46

Kevin M.K. Bottomley (Editor), David Bradshaw (Editor), and John S. Nixon (Editor), eds. Metalloproteinases as Targets for Anti-Inflammatory Drugs (Progress in Inflammation Research). Birkhäuser Basel, 1999.

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47

1939-, Cerny Jan, and Hiernaux Jacques R. J, eds. Idiotypic network and diseases. Washington, D.C: American Society for Microbiology, 1990.

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48

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology (Advances in Pharmacological Sciences). Birkhauser, 1992.

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49

Kurowski, M. Adverse Reactions to Non-Steroidal Anti-Inflammatory Drugs: Clinical Pharmacoepidemiology (Agents and Actions Supplements). Birkhauser, 1992.

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50

Karl-Erik, Andersson, Persson C. G. A, and AB Draco, eds. Anti-asthma xanthines and adenosine: Proceedings of a symposium in Copenhagen, February 22-23, 1985. Amsterdam: Excerpta Medica, 1985.

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