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1

Harald, Kropshofer, and Vogt Anne B, eds. Antigen presenting cells: From mechanisms to drug development. Weinheim: Wiley-VCH, 2005.

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2

Chang, Nan-Hua. Selective elimination of antigen-specific T cells by antigen-targeted drug-labeled antigen-presenting cell membranes. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1997.

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3

Z, Atassi M., and Abbott Laboratories, eds. Immunobiology of proteins and peptides IV: T-cell recognition and antigen presentation. New York: Plenum Press, 1987.

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4

Antigen-presenting cells. Oxford: IRL Press at Oxford University Press, 1989.

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5

Benvenuto, Pernis, Silverstein Samuel C, and Vogel Henry J. 1920-, eds. Processing and presentation of antigens. San Diego: Academic Press, 1988.

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6

James, McCluskey, ed. Antigen processing and recognition. Boca Raton: CRC Press, 1991.

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7

1953-, Angelov D. N., ed. The cerebral perivascular cells. Berlin: Springer, 1998.

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8

Edward, Humphreys Robert, and Pierce Susan K, eds. Antigen processing and presentation. San Diego: Academic Press, 1994.

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9

Kang, Chʻang-yul. Pairŏsŭ pektʻŏ ro hyŏngjil toiptoen hangwŏn chesi sepʻo ŭi myŏnyŏk chʻiryoje yuhyosŏng pʻyŏngka mit sihŏmpŏp yŏnʼgu =: Development and estimation of immunotherapeutic cell-based vaccine approaches using antigen presenting cells transduced with viral vector. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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10

Viner, Nicholas John. Antigen presenting cells in inflammatory arthritis. Birmingham: University of Birmingham, 1992.

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11

Harding, Clifford V. MHC molecules and antigen processing. Austin, Tex: R.G. Landes Co., 1997.

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12

Dr, Fernandez Nelson, and Butcher G, eds. MHC: A practical approach. Oxford: Oxford University Press, 1997.

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13

1941-, Thomas D. Brian, ed. Viruses and the cellular immune response. New York: Marcel Dekker, 1993.

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14

Atassi, M. Zouhair, and Howard L. Bachrach. Immunobiology of Proteins and Peptides IV: T-Cell Recognition and Antigen Presentation. Springer London, Limited, 2012.

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15

Atassi, M. Zouhair. Immunobiology of Proteins and Peptides Iv: T-Cell Recognition And Antigen Presentation. Springer, 2012.

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16

B, Schook Lawrence, Tew John G, and International RES Symposium (1987 : Richmond, Va.), eds. Antigen presenting cells: Diversity, differentiation, and regulation : proceedings of a symposium held in Richmond, Virginia, March 26-29, 1987. New York: Liss, 1988.

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17

Immunology: From Cell Biology to Disease. Wiley-VCH, 2007.

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18

Ortega, Henry William. Mechanisms of accessory cell function in rainbow trout (Oncorhynchus mykiss). 1993.

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19

Lubor, Fornusek, and Větvička Václav 1954-, eds. Immune system accessory cells. Boca Raton, FL: CRC Press, 1992.

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20

Dörner, Thomas, and Peter E. Lipsky. Cellular side of acquired immunity (B cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0050.

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B cells have gained interest in rheumatoid arthritis (RA) beyond being the precursors of antibody-producing plasma cells since they are also a broader component of the adaptive immune system. They are capable of functioning as antigen-presenting cells for T-cell activation and can produce an array of cytokines. Disturbances of peripheral B-cell homeostasis together with the formation of ectopic lymphoid neogenesis within the inflamed synovium appears to be a characteristic of patients with RA. Enhanced generation of memory B cells and autoreactive plasma cells producing IgM-RF and ACPA-IgG antibodies together with formation of immune complexes contribute to the maintenance of RA, whereas treatment with B-cell-directed anti-CD20 therapy provides clinical benefit.
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21

Kropshofer, Harald, and Anne B. Vogt, eds. Antigen Presenting Cells. Wiley, 2005. http://dx.doi.org/10.1002/3527607021.

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22

(Editor), Harald Kropshofer, and Anne B. Vogt (Editor), eds. Antigen Presenting Cells: From Mechanisms to Drug Development. Wiley-VCH, 2005.

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23

Harald, Kropshofer, and Vogt Anne B, eds. Antigen presenting cells: From mechanisms to drug development. Weinheim: Wiley-VCH, 2005.

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24

Antigen presenting cells: From mechanisms to drug development. Weinheim: Wiley-VCH, 2005.

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25

Kaiserlian, Dominique. Antigen Presentation by Intestinal Epithelal Cells. Landes Bioscience, 1996.

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26

1955-, Kaiserlian Dominique, ed. Antigen presentation by intestinal epithelial cells. New York: Springer, 1996.

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27

Atassi, M. Zouhair, and Howard L. Bachrach. Immunobiology of Proteins and Peptides (Advances in Experimental Medicine and Biology). Springer, 1988.

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28

Loss, George E. MHC class II presentation of an endogenous antigen. 1993.

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29

Zierhut, Manfred, Hans-Georg Rammensee, and Wayne Streilein. Antigen-Presenting Cells and the Eye. Taylor & Francis Group, 2007.

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30

Antigen-Presenting Cells and the Eye. Informa Healthcare, 2007.

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31

Zierhut, Manfred, Hans-Georg Rammensee, and Wayne Streilein. Antigen-Presenting Cells and the Eye. Taylor & Francis Group, 2007.

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32

SCHOOK, LB. Schook: Antigen Presenting Cells - Diversity Differentiation & Regulation. John Wiley & Sons Inc, 1988.

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33

Clark, Georgina, Natasha Mireille Rogers, and I. Jolanda M. De Vries, eds. The Therapeutic Potential of Antigen Presenting Cells. Frontiers Media SA, 2022. http://dx.doi.org/10.3389/978-2-88976-392-4.

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34

Bruijnzeel-Koomen, C. A. F. M. and Hoefsmit Elizabeth C. M, eds. Immunopharmacology of macrophages and other antigen-presenting cells. London: Academic Press, 1994.

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35

Carla A.F.M. Bruijnzeel-Koomen (Editor), Elisabeth C.M. Hoefsmit (Series Editor), and Clive Page (Series Editor), eds. Immunopharmacology of Macrophages and Other Antigen-Presenting Cells (Handbook of Immunopharmacology) (Handbook of Immunopharmacology). Academic Press, 1994.

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36

Carla A.F.M. Bruijnzeel-Koomen (Editor), Elisabeth C.M. Hoefsmit (Series Editor), and Clive Page (Series Editor), eds. Immunopharmacology of Macrophages and Other Antigen-Presenting Cells (Handbook of Immunopharmacology) (Handbook of Immunopharmacology). Academic Press, 1994.

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37

Hoefsmit, Elisabeth C. M., Clive Page, and Carla A. F. M. Bruijnzeel-Koomen. Immunopharmacology of Macrophages and Other Antigen-Presenting Cells. Elsevier Science & Technology Books, 1994.

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38

Kropshofer, Harald, and Anne B. Vogt. Antigen Presenting Cells: From Mechanisms to Drug Development. Wiley-VCH Verlag GmbH, 2006.

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39

Kropshofer, Harald, and Anne B. Vogt. Antigen Presenting Cells: From Mechanisms to Drug Development. Wiley & Sons, Incorporated, John, 2006.

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40

Antigen Processing and Presentation Protocols. Humana Press, 2001.

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41

Solheim, Joyce C. Antigen Processing and Presentation Protocols. Humana Press, 2010.

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42

Whitton, J. Lindsay. Antigen Presentation (Current Topics in Microbiology and Immunology). Springer, 1998.

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43

van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient is diagnosed with SLE. Apoptotic debris is present in the extracellular matrix and circulation of patients with SLE due to an aberrant process of apoptosis and/or insufficient clearance of apoptotic cells and apoptotic debris. The non-cleared apoptotic debris in patients with SLE may lead to activation of both the innate (myeloid and plasmacytoid dendritic cells) and adaptive (T and B cells) immune system. In addition to the activation by apoptotic debris and immune complexes, the immune system in SLE may be deregulated at the level of (a) presentation of self-peptides by antigen-presenting cells, (b) selection processes for both B and T cells, and (c) regulatory processes of B- and T-cell responses. Lupus nephritis may be classified in different classes based on histological findings in renal biopsies. The chromatin-containing immune complexes deposit in the capillary filter, most likely due to the interaction of chromatin with the polysaccharide heparan sulphate. A decreased renal expression of the endonuclease DNaseI further contributes to the glomerular persistence of chromatin and the development of glomerulonephritis.Current treatment of lupus nephritis is not specific and aims to reduce the inflammatory response with general immunosuppressive therapies. However, research has revealed novel potential therapeutic candidates at the level of dendritic cells, B cells, and T cells.
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44

Isaacs, John, and Catharien Hilkens, eds. Tolerogenic Antigen-Presenting Cells – Modulating Unwanted Immune Response at Their Core. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88963-176-6.

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45

Immunobiology of bacterial CpG-DNA. Berlin: Springer, 2000.

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46

MHC Volume 1: A Practical Approach (Practical Approach Series). Oxford University Press, USA, 1997.

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47

(Editor), N. Fernandez, and G. Butcher (Editor), eds. MHC: Volume 2: A Practical Approach (Practical Approach Series, 180, etc.). Oxford University Press, USA, 1998.

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48

MHC, Volume 2: A Practical Approach. Oxford University Press, USA, 1998.

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49

(Editor), N. Fernandez, and G. Butcher (Editor), eds. MHC: A Practical Approach 2-Volume Set (Practical Approach Series). Oxford University Press, USA, 1998.

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50

MHC: A Practical Approach 2-Volume Set (Practical Approach Series). Oxford University Press, USA, 1998.

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