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Books on the topic 'Antidepressants Mechanisms of action'

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Author: Grafiati

Published: 10 December 2022

Last updated: 29 January 2023

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1

Polgár, László. Mechanisms of protease action. Boca Raton, Fla: CRC Press, 1989.

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2

Saltiel, Alan R., and Jeffrey E. Pessin. Mechanisms of Insulin Action. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72204-7.

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3

Woodruff, G. N., ed. Mechanisms of Drug Action. London: Palgrave Macmillan UK, 1986. http://dx.doi.org/10.1007/978-1-349-08026-7.

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4

Sakamoto, Yukiya. Glucocorticoid hormone: Mechanisms of action. Tokyo: JapanScientific Societies Press, 1986.

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5

Molecular mechanisms of drug action. London: Taylor & Francis, 1988.

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6

Gehring, Ulrich, Ernst J. M. Helmreich, and Günter Schultz, eds. Molecular Mechanisms of Hormone Action. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-75022-9.

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7

1936-, Gehring Ulrich, Helmreich, E. J. M. 1922-, and Schultz G. 1936-, eds. Molecular mechanisms of hormone action. Berlin: Springer-Verlag, 1989.

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8

Dipple, Anthony. Mechanisms of action of chemical carcinogens. Birmingham: University of Birmingham, 1987.

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9

Williams, Graham R. Molecular mechanisms of thyroid hormone action. Birmingham: University of Birmingham, 1993.

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10

White, H. Steve. Mechanisms of action of antiepileptic drugs. West Islip, NY: Professional Communications, Inc., 2010.

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11

L, Wynne Clive D., and Staddon J. E. R, eds. Models of action: Mechanisms for adaptive behavior. Mahwah, N.J: Lawrence Erlbaum Associates, 1998.

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12

Pelt, Annemarie C. Glucocorticoids: Effects, action mechanisms, and therapeutic uses. Hauppauge, N.Y: Nova Science, 2011.

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13

Lombardini, John B. Taurine: Nutritional Value and Mechanisms of Action. Boston, MA: Springer US, 1992.

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14

Barry, Lombardini J., Schaffer S. W, Azuma Junichi, Waltham Symposium on Taurine and Cat Nutrition (1991 : Orange Beach, Ala.), and International Taurine Symposium: New Dimensions on its Mechanisms of Action (1991 : Orange Beach, Ala.), eds. Taurine: Nutritional value and mechanisms of action. New York: Plenum Press, 1992.

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15

Understanding insulin action: Principles and molecular mechanisms. Chichester: E. Horwood, 1989.

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16

Yamada, Hiromasa, and Kintaro Takahashi. Ghrelin: Production, action mechanisms and physiological effects. New York: Nova Biomedical, Nova Science Publishers, Inc., 2012.

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17

Akinsanya, A. A. The molecular mechanisms of haemopoietic growth factor action. Manchester: UMIST, 1992.

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18

Eberle, A. The melanotropins: Chemistry, physiology, and mechanisms of action. Basel: Karger, 1988.

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19

Johnston, Heather Patricia. The mechanisms of action of thiopurine nucleotide prodrugs. Norwich: University of East Anglia, 1985.

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20

Preskorn, Sheldon H., and Matthew Macaluso. Antidepressants: From Biogenic Amines to New Mechanisms of Action. Springer, 2019.

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21

Norman, Trevor, and James S. Olver. Duloxetine: Clinical Uses, Mechanism of Action and Efficacy. Nova Science Publishers, Incorporated, 2018.

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22

Bipolar medications : mechanisms of action. Washington, DC: American Psychiatric Press, 2000.

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23

(Editor), Husseini K. Manji, Charles L. Bowden (Editor), and Robert H., M.D. Belmaker (Editor), eds. Bipolar Medications: Mechanisms of Action. American Psychiatric Publishing, Inc., 2000.

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24

Duman, Ronald S. Neurotrophic Mechanisms of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0027.

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Early theories of depression and treatment response were centered on the monoamine neurotransmitters, but more recent work has focused on functional and structural synaptic plasticity and the role of neurotrophic factors, particularly brain derived neurotrophic factor (BDNF). Neurotrophic factors regulate all aspects of neuronal function, including adaptive plasticity, synapse formation, and neuronal survival. Chronic stress and depression cause reductions in levels of BDNF and other key factors, including vascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2), in cortical regions that contribute to atrophy and loss of neurons observed in depressed patients and rodent stress models. In contrast, these neurotrophic factors are upregulated by chronic administration of typical antidepressants and are required for antidepressant responses. Moreover, fast acting, highly efficacious antidepressant agents such as ketamine rapidly increase BDNF release and synapse formation, paving the way for a new generation of medications for the treatment of depression.

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25

Hendrickson, Rebecca C., and Murray A. Raskind. Pharmacological Treatment of Nightmares, Sleep Disturbance, and Daytime Hyperarousal in PTSD: The Role of Prazosin, Other Noradrenergic Modulators, and Sedative Hypnotics or Commonly Used Sedating Medications. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0035.

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Disruption of stress-response systems contributes to the pathophysiology of post-traumatic stress disorder (PTSD). Consistent with this, daytime hyperarousal and nighttime sleep disruption, including trauma-related nightmares, are core symptoms of the disorder, often requiring targeted pharmacologic treatment. Although a variety of medications that target sleep–wake and arousal mechanisms are commonly used for this purpose, there remains the best empirical support for prazosin, a brain-active antagonist of the α‎1 noradrenaline receptor, with emerging evidence for doxazosin, a longer-acting medication with the same mechanism of action. This chapter reviews the evidence for use of prazosin and doxazosin as well as for the sedative hypnotics (benzodiazepines, nonbenzodiazepine hypnotics, and related medications), antihistamines, and sedating antidepressants trazodone and nefazodone to address hyperarousal symptoms and trauma-associated nightmares in PTSD. Clinical recommendations for the use of prazosin in PTSD, as well as a discussion of emerging pharmacologic treatments, are also included.

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26

Krystal, John H., and Dennis S. Charney. Current Treatments for Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0031.

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Depression is among the most common and disabling medical disorders worldwide. Pharmacotherapy plays an important role in its treatment, although only half of all depressed patients show full remission with currently available therapies. This chapter reviews the most common pharmacotherapies with respect to their mechanisms of action, efficacy, tolerability, and safety. It also considers pharmacologic approaches to treatment-resistant symptoms of depression including adjunctive pharmacotherapies and the emerging rapid-acting antidepressants. An important focus of current research is to devise biological measures that direct a given patient to an effective form of treatment. Depression research is at a very exciting phase that will have important consequences for affected patients and for society overall.

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27

Cozza, Kelly L., Rita Rein, Gary H. Wynn, and Eric G. Meyer. Psychopharmacology of Depression as a Systemic Illness for Primary and Specialty Care Clinicians. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603342.003.0010.

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There are nearly 4 million patients with depression followed by primary care in the United States, with nearly 80% of prescriptions for antidepressants written by non-psychiatrists (Mark et al. 2009). Understanding and utilizing psychopharmacology is a critical skill for primary care physicians, who are often initial or sole prescribers. Persons with medical illnesses and depression are often prescribed a multitude of medications, necessitating attention to pharmacodynamic, pharmacokinetics, and an understanding of intended effects, side effects, toxicities, and drug interactions. This chapter begins with a brief review of drug mechanisms of action, metabolism, and interaction principles; addressing the interplay between depression, the medications used to treat depression, co-prescribed medications, and medical illness. The chapter includes a discussion of drugs used to treat depression in text and table format, highlighted with case examples. Details about mechanism of action, common side effects and adverse reactions, drug interactions, and other clinical implications are provided.

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28

A, Montgomery S., and Corn Timothy H, eds. Psychopharmacology of depression. Oxford: Oxford University Press, 1994.

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29

(Editor), N. B. Finter, and R. M. Friedman (Editor), eds. Mechanisms Produc & Action: (Mechanisms of Production & Action). Elsevier Science & Technology, 1985.

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30

Mechanisms of Drug Action. Palgrave Macmillan, 1986.

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31

Mechanisms of insulin action. Austin, Tex: Landes Bioscience, 2007.

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32

Phytochemicals: Mechanisms of Action. CRC, 2003.

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33

Lavrik, O. I. Mechanisms of Enzyme Action. Routledge, 1989.

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34

Bidlack, Wayne R., R. Keith Randolph, Mark S. Meskin, Audra J. Davies, and Douglas S. Lewis. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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35

Bidlack, Wayne R., R. Keith Randolph, Mark S. Meskin, Audra J. Davies, and Douglas S. Lewis. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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36

Meskin, Mark S. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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37

Bidlack, Wayne R., R. Keith Randolph, Mark S. Meskin, Audra J. Davies, and Douglas S. Lewis. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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38

Mechanisms of drug action. London: Macmillan, 1986.

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39

Wolstenholme, G. E. W., and Maeve O'Connor. Carcinogenesis: Mechanisms of Action. Wiley & Sons, Incorporated, John, 2009.

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40

Phytochemicals: Mechanisms of action. Boca Raton, FL: CRC Press, 2003.

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41

Saltiel, Alan. Mechanisms of Insulin Action. Eurekah.Com Inc, 2005.

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42

Mechanisms of Adiponectin Action. MDPI, 2019. http://dx.doi.org/10.3390/books978-3-03921-246-0.

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43

Bidlack, Wayne R., R. Keith Randolph, Mark S. Meskin, Audra J. Davies, and Douglas S. Lewis. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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44

Saltiel, Alan R., and Jeffrey E. Pessin. Mechanisms of Insulin Action. Springer London, Limited, 2007.

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45

Saltiel, Alan, Alan R. Saltiel, and Jeffrey E. Pessin. Mechanisms of Insulin Action. Springer, 2011.

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46

Bidlack, Wayne R., R. Keith Randolph, Mark S. Meskin, Audra J. Davies, and Douglas S. Lewis. Phytochemicals: Mechanisms of Action. Taylor & Francis Group, 2003.

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47

Staff, CIBA Foundation Symposium. Carcinogenesis - Mechanisms of Action. Wiley & Sons, Limited, John, 2008.

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48

1960-, Ozawa H., Saito Toshikazu, Takahata Naohiko 1932-, Nihon Seishin Shinkei Gakkai, and Symposium on Affective Disorders and Neuronal Signal Transduction (1996 : Sapporo-shi, Japan), eds. Signal transduction in affective disorders. Tokyo: Springer, 1998.

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49

Casey, Patricia. Treatment of adjustment disorders (DRAFT). Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198786214.003.0007.

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There are few randomized controlled studies of the treatment of AD. This is due to the transience of the symptoms, the difficulty obtaining a homogeneous population owing to the absence of diagnostic criteria, and the variable nature of the stressor, among others. Clinical guidelines specify that brief psychological interventions are preferred, and these incorporate elements from the many approaches now available on the assumption that their mechanism of action will also be effective in AD. Few have been tested specifically in AD. Low-intensity therapies specific to AD are being developed, such as biblio-therapy and online self-help. Pharmacological interventions have not been tested in quality trials, but despite this, antidepressants continue to be frequently prescribed. A few trials of anxiolytics have found some benefit for all the agents examined, but none used placebo. EMDR was found to be beneficial in a small pilot study and is worthy of further study.

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50

Serotonin: Reshaping the treatment of depression. Oxford [England]: Medicine Pub. Foundation, 1992.

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